ライフサイエンスコーパス: basilar

1 y examination revealed wheezes and decreased basilar air entry.

2 phase-contrast magnetic resonance imaging of basilar and carotid arteries to measure cerebral blood f

3 Basilar and posterior communicating arteries (PCAs) were

4 e for the cochlear resonance, presumably the basilar and tectorial membranes, move together in phase

5 s specific to a compartment either apical or basilar and that an additional mechanism may be required

6 occlusion (defined as intracranial carotid, basilar, and M1 segment of middle cerebral artery occlus

7 oss-sectional areas of the internal carotid, basilar, and middle cerebral arteries on the first day a

8 sham controls, with reductions of 33% in the basilar arbor and 28% in the apical arbor.

9 n lesioned and sham rats in either apical or basilar arbors; however, the proportion of immature to m

10 ive fungi, with extensive involvement of the basilar arterial circulation of the brain.

11 ChR agonists induced exclusive relaxation of basilar arterial rings without endothelium.

12 etic-dependent nitrergic dilation of porcine basilar arterial rings.

13 rculation with emphasis on the vertebral and basilar arteries (the posterior cerebral circulation).

14 ic stenosis or occlusion in vertebral and/or basilar arteries underwent large-vessel flow measurement

15 Rabbit basilar arteries were exposed to autologous EPCs ( appro

16 Changes of middle cerebral arteries and basilar arteries were extremely rare, thus we can say th

17 intracisternal delivery of autologous EPCs, basilar arteries were isolated and expression of vasoreg

18 roximal occlusions (internal carotid, M1, or basilar arteries).

19 asal cisterns and subsequent invasion of the basilar arteries.

20 It involves mostly vertebral and basilar arteries.

21 d wide middle cerebral, internal carotid and basilar arteries.

22 l arteries, posterior cerebral arteries, and basilar arteries.

23 er normal values for the middle cerebral and basilar arteries.

24 ertebral artery (40 patients, 12 bilateral), basilar artery (46 patients).

25 longed obesity mouse model that suffers from basilar artery (BA) abnormalities, we find that microgli

26 We examined the vascular responses of the basilar artery (BA) and its side branches through a cran

27 on in endovascular stroke treatment (EVT) of basilar artery (BA) occlusion.

28 lood vessels [internal carotid artery (ICA), basilar artery (BA), middle cerebral artery (MCA)], the

29 corrected p=0.010) and decreased flux in the basilar artery (mean difference -0.9 mL/s, 95% CI -1.5 t

30 = 0.05) and relative signal intensity in the basilar artery (p = 0.04; sham-group unchanged).

31 Patients with isolated tip of basilar artery (TBA) occlusion had significantly more BP

32 One of the patients with a basilar artery aneurysm also had coarctation of the aort

33 Two had basilar artery aneurysms, 7 had internal carotid artery

34 ed for WMLs (Fazekas scale), infarctions and basilar artery diameter (BAD).

35 ired acetylcholine-induced dilatation of the basilar artery during diabetes mellitus can be restored

36 ther cxcl12b or cxcr4a results in defects in basilar artery formation, showing that the assembly and

37 The basilar artery geometry strongly influenced both skewing

38 a direct acidifier of the cell, dilates rat basilar artery in K(ATP)-dependent fashion.

39 nM) produced dose-related dilatation of the basilar artery in non-alcohol-fed and alcohol-fed rats.

40 We measured the diameter of the basilar artery in non-diabetic rats, diabetic (streptozo

41 roM) produced dose-related dilatation of the basilar artery in non-diabetic rats, which was inhibited

42 A only partially inhibited dilatation of the basilar artery in response to acetylcholine in diabetic

43 ine were not affected in either aorta or the basilar artery in vitro.

44 Measurements of the basilar artery indicated a considerable hypertrophy, ind

45 Basilar artery maximal diameter was greater in SF mice (

46 Acute basilar artery occlusion (BAO) is a rare but potentially

47 itive results also apply to the patient with basilar artery occlusion (BAO).

48 le to reperfusion therapy; 11 patients had a basilar artery occlusion and were excluded, leaving 100

49 ic mechanism, distal territory location, and basilar artery occlusive disease carried the poorest pro

50 d dilation that was markedly impaired in the basilar artery of mice expressing dominant-negative muta

51 ial recanalization of the middle cerebral or basilar artery on TCD were studied.

52 nce to that molecule and markedly suppressed basilar artery spasm after subarachnoid hemorrhage.

53 any benefit in the treatment of vertebral or basilar artery stenosis.

54 A 6-year-old male with vertebral-basilar artery thrombosis was recognized to have high-mo

55 g posterior fossa lesions, the management of basilar artery thrombosis, which may have a longer time

56 logical parameters that were associated with basilar artery tip aneurysms (BTA) in a location-specifi

57 We examined NT in basilar artery vascular smooth muscle cells (VSMCs) from

58 The prevalence of basilar artery vasospasm in children with moderate traum

59 rt of the posterior cerebral artery (P1) and basilar artery was observed in the ligated group, as wel

60 The responses to acetylcholine in basilar artery were restored to normal after treatment w

61 In vitro or in vivo treatment of basilar artery with conditioned media from EPCs also cau

62 Unlike basilar artery, 12 weeks of a high-fat diet was not suff

63 he major artery supplying the hindbrain, the basilar artery, runs along the ventral keel of the hindb

64 f freshly isolated contractile VSMC from rat basilar artery, we found that EGF (100 ng ml(-1)) caused

65 d one patient (1.02%) had an aneurysm of the basilar artery.

66 s coexisting with a fusiform aneurysm of the basilar artery.

67 keel immediately adjacent to the assembling basilar artery.

68 -type Ca2+ channels in native VSMCs from rat basilar artery.

69 tery, the posterior cerebral artery, and the basilar artery.

70 days (+/- 2 d) and 1.5 days (+/- 1 d) in the basilar artery.

71 intracranial internal carotid artery and the basilar artery.

72 ebral arteries and 5 days (+/- 2.5 d) in the basilar artery.

73 Animals underwent a basilar cistern inoculation of group B Streptococci to i

74 Animals underwent a basilar cistern tap receiving either sterile saline as a

75 y partially or completely disappeared in the basilar cisterns (P <.001) and cerebral sulcal subarachn

76 ts presented with significantly more effaced basilar cisterns (p = 0.0008).

77 pines, and a previously unreported cell with basilar dendrites and complex, spiny apical processes (t

78 Basilar dendrites exhibited idiosyncratic geometry acros

79 C (Brodmann area 46) were Golgi-stained, and basilar dendrites of pyramidal cells in the deep half of

80 ntified in second-, third-, and fourth-order basilar dendrites of pyramidal neurons in layer II-III o

81 across compartments, between the apical and basilar dendrites, follows different rules and requires

82 pyramidal neurons with elongated "tap root" basilar dendrites.

83 characterized by large somata and extensive basilar dendrites.

84 and was specific to the apical, but not the basilar, dendrites of CA1.

85 tion of terminal branches of apical, but not basilar, dendrites of IL neurons.

86 t capture of L-LTP can also occur within the basilar dendritic compartment and that the tagging signa

87 compartment, can also take place within the basilar dendritic compartment and, if so, whether captur

88 After Golgi impregnation, basilar dendritic field size was estimated for layer V p

89 Smaller basilar dendritic field size was evident in proximal and

90 lation delivered in a restricted part of the basilar dendritic tree invariably produced sustained pla

91 ior circulation events and had vertebral and basilar imaging, of whom 37 (26.2%) had > or = 50% verte

92 s, severe and diffuse in nine, posterior and basilar in three, and limited to the apices in one (not

93 increased the wall thickness in the apex and basilar infarct regions compared with the control infarc

94 ne anomalies seen in paediatric patients are basilar invagination, C1-C2 instability, atlantoaxial ro

95 th zones were positioned periodically in the basilar layer to enhance branching of barb ridges.

96 to a constant suppressor displacement on the basilar membrane (as in experiments with wild-type anima

97 body vibrations normal to the surface of the basilar membrane (BM) at 0.8 (d(1)), 5.8 (d(2)), 15.6 (d

98 The mammalian basilar membrane (BM) consists of two collagen-fiber lay

99 Sound-evoked vibrations of the basilar membrane (BM) in anaesthetised guinea-pigs are s

100 Using a laser velocimeter, basilar membrane (BM) responses to tones were measured i

101 -otoacoustic-emissions (DPOAEs), and passive basilar membrane (BM) responses.

102 ounts of energy as passive, pressure-driven, basilar membrane (BM) traveling waves.

103 served to propagate longitudinally along the basilar membrane (BM) ultimately stimulate the mechano-s

104 h in vivo physiological measurements for the basilar membrane (BM) velocity, V(BM), frequency tuning

105 IHCs respond to basilar membrane (BM) vibration by producing a transduce

106 rimentally tested in this study by measuring basilar membrane (BM) vibrations at the cubic distortion

107 ween internal structures such as hair cells, basilar membrane (BM), and modiolus with external surfac

108 ty, harmonic distortion, and DC shift on the basilar membrane (BM), tectorial membrane (TM), and OHC

109 situated within the organ of Corti atop the basilar membrane (BM), which supports sound-evoked trave

110 m to be observed in vivo at the level of the basilar membrane (BM).

111 the cochlear organ of Corti that rest on the basilar membrane (BM).

112 rwise damped sound-induced vibrations of the basilar membrane [2-4], a mechanism known as negative da

113 erilymphatic chloride level manipulations of basilar membrane amplification in the living guinea pig.

114 by the passive mechanical properties of the basilar membrane and active feedback from the outer hair

115 ound stimuli from hair cell receptors in the basilar membrane and are arranged tonotopically.

116 ncy ratio, and measured as vibrations at the basilar membrane and at the stapes, and as sound pressur

117 of OHC electrical activity, pressure at the basilar membrane and basilar membrane displacement gave

118 , likely by the mechanical properties of the basilar membrane and organ of Corti.

119 ed by measuring the phase difference between basilar membrane and stapes vibrations.

120 ugh the fluid-tissue interaction between the basilar membrane and the fluid in scala tympani (ST) has

121 pled fluid-structure interaction between the basilar membrane and the scala fluids.

122 partition and active frequency tuning of the basilar membrane are enhanced in the cochleae of CD-1Cx3

123 ibited considerable re-growth of PAFs in the basilar membrane area.

124 s as compression waves rather than along the basilar membrane as backward-traveling waves.

125 timulation of the cochlear bone vibrates the basilar membrane as well.

126 the extracellular voltage referenced to the basilar membrane at a frequency approximately one-half o

127 isplacements of the reticular lamina and the basilar membrane at the 19 kHz characteristic place in g

128 nds on not only the passive mechanics of the basilar membrane but also an active amplification of the

129 duced comparable amplitudes of motion of the basilar membrane but differed in the polarity of their f

130 gy is that tuned mechanical vibration of the basilar membrane defines the frequency response of the i

131 tivity, pressure at the basilar membrane and basilar membrane displacement gave direct evidence for p

132 as been considered to be proportional to the basilar membrane displacement or velocity.

133 generated extracellular voltage relative the basilar membrane displacement, which was initiated at a

134 ve relations between transducer currents and basilar membrane displacements are lacking, as well as t

135 sured acoustically and electrically elicited basilar membrane displacements from the cochleae of wild

136 Such faint sounds produce 0.1-nm basilar membrane displacements, a distance smaller than

137 to cochlear filtering that is independent of basilar membrane filtering.

138 hick fibers that coursed radially across the basilar membrane in small fascicles, gave off small bran

139 on (reflecting the nonlinear response of the basilar membrane in the cochlea), followed by linear sum

140 A hearing sensation arises when the elastic basilar membrane inside the cochlea vibrates.

141 A simple monophasic vibratory mode of the basilar membrane is found at both ends of the cochlea.

142 OHCs at various cochlear locations while the basilar membrane is mechanically stimulated.

143 The basilar membrane is typically set into motion through ai

144 s at the stapes earlier than at the measured basilar membrane location.

145 furosemide injection, the vibrations of the basilar membrane lost the best frequency (BF) peak and s

146 man speech, is not principally determined by basilar membrane mechanics.

147 are characterized here through recordings of basilar membrane motion and hair cell extracellular rece

148 was found by taking the phase difference of basilar membrane motion between two longitudinally space

149 sducer currents (or receptor potentials) and basilar membrane motion in an excised and bisected cochl

150 istortion product otoacoustic emissions, and basilar membrane motion indicate that the TM remains fun

151 The model is fit to in vivo basilar membrane motion with one free parameter for the

152 energy at the appropriate moment to enhance basilar membrane motion.

153 cells that detect and amplify sound-induced basilar membrane motions.

154 have demonstrated that efference suppresses basilar membrane movement, there is still much unknown a

155 odel could be tested by measuring TTS on the basilar membrane of the Otoa(EGFP/EGFP) mice to improve

156 The detectable basilar membrane response to a low-level 16-kHz tone occ

157 In a sensitive cochlea, the basilar membrane response to transient excitation of any

158 he amplification and frequency tuning of the basilar membrane responses to sounds are almost normal.

159 ation results agree with 2TS measurements of basilar membrane responses.

160 compared with that at the commonly measured basilar membrane side.

161 tion result from longitudinal differences in basilar membrane stiffness and numerous individual grada

162 itude of the crista stiffness was similar to basilar membrane stiffness in mammals, and as in mammals

163 lfrog's amphibian papilla lacks the flexible basilar membrane that effects tuning in mammals, its aff

164 ane in hearing: it enables the motion of the basilar membrane to optimally drive the inner hair cells

165 vibration collaboratively interacts with the basilar membrane traveling wave primarily through the co

166 hlear outer hair cells (OHCs) to amplify the basilar membrane traveling wave; however, it is unclear

167 Cochlear amplification of basilar membrane traveling waves is thought to occur bet

168 Compared with basilar membrane traveling waves, tectorial membrane tra

169 lar lamina motion is not directly coupled to basilar membrane traveling waves.

170 sms within outer hair cells that amplify the basilar membrane travelling wave.

171 peaked at a higher frequency than transverse basilar membrane tuning in the passive, postmortem condi

172 apical ends of outer hair cells and from the basilar membrane using a custom-built heterodyne low-coh

173 in the cochlea, as shown by measurements of basilar membrane velocity and auditory nerve responses t

174 ived electromotile force near to that of the basilar membrane velocity at frequencies above the shift

175 The basilar membrane velocity was measured through the trans

176 hlear responses are amplified during maximum basilar membrane velocity.

177 n discrepancies between previously published basilar membrane vibration and auditory nerve single uni

178 Data also show that basilar membrane vibration at the emission frequency is

179 The phase relation of reticular lamina to basilar membrane vibration changes with frequency by up

180 ate that outer hair cells do not amplify the basilar membrane vibration directly through a local feed

181 ear amplifier from available measurements of basilar membrane vibration in sensitive mammalian cochle

182 best to different sound frequencies because basilar membrane vibration is mechanically tuned to diff

183 sions showed no significant change while the basilar membrane vibration nearly disappeared.

184 a vibration is substantially larger than the basilar membrane vibration not only at the best frequenc

185 The current data indicate that basilar membrane vibration was not involved in the backw

186 ve yielded ever-better descriptions of gross basilar membrane vibration, the internal workings of the

187 ically been limited to point measurements of basilar membrane vibration.

188 ase differences between reticular lamina and basilar membrane vibrations are absent in postmortem coc

189 electrically in the second turn and measured basilar membrane vibrations at two longitudinal location

190 n longitudinal location, electrically evoked basilar membrane vibrations showed the same tuning and p

191 generate forces for amplifying sound-induced basilar membrane vibrations, yet how cellular forces amp

192 ructures range from a few hundred (e.g., the basilar membrane) to a few micrometers (e.g., the stereo

193 al coupling between adjacent sections of the basilar membrane, and such coupling may be critical for

194 o Deiters' cells, the sulcus epithelium, the basilar membrane, and the surface of the spiral limbus.

195 ratchet: Sound-evoked forces, acting on the basilar membrane, are transmitted to the hair bundles, w

196 h constrain outer hair cells standing on the basilar membrane, causes a leftward shift in outer hair

197 efrom, may not involve the usual wave on the basilar membrane, suggesting that additional cochlear st

198 zing their responses to the vibration of the basilar membrane, the radial vibrations of the tectorial

199 n physical and geometrical properties of the basilar membrane, the sensitivity or gain of the hearing

200 ning which OHCs enhance the vibration of the basilar membrane, thereby tuning the gain of cochlear am

201 uter hair cells that generates forces on the basilar membrane, we demonstrate that these forces inter

202 bing amplification in narrow segments of the basilar membrane, we further show that a cochlear travel

203 echanical filters analogous to the cochlea's basilar membrane, which deconstructs complex sounds into

204 cells that effectively couple energy to the basilar membrane, which reduces sensitivity.

205 interferometry to measure vibrations of the basilar membrane.

206 and were enhanced compared with those at the basilar membrane.

207 f sensory and supporting cells riding on the basilar membrane.

208 ng the organ of Corti than in displacing the basilar membrane.

209 nge well below the resonant frequency of the basilar membrane.

210 decouples active hair-bundle forces from the basilar membrane.

211 ar to be related in part to the width of the basilar membrane.

212 ts along the cochlea, similar to that of the basilar membrane.

213 traveling wave to amplify the motion of the basilar membrane.

214 nd that the stapes vibrates earlier than the basilar membrane.

215 amina, but only a slow tuned response of the basilar membrane.

216 ously measured close to the sensory tissue's basilar membrane.

217 increases the sound-evoked vibrations of the basilar membrane.

218 fer of electrical to mechanical power at the basilar membrane.

219 line in motion amplitude occurred across the basilar membrane.

220 ular lamina, to the transverse motion of the basilar membrane.

221 on caused motion of a minimal portion of the basilar membrane.

222 seen when the beam was directed towards the basilar membrane.

223 her than an immediate local vibration of the basilar membrane; this travelling wave vibrates in phase

224 hese mice have a low-frequency hearing loss, basilar-membrane and neural tuning are both significantl

225 ries are very similar to the trajectories of basilar-membrane peak velocity toward scala tympani.

226 10], we show that the nonlinear component of basilar-membrane responses to sound stimulation leads th

227 Whether measured by basilar-membrane vibration, nerve-fiber activity, or per

228 usion of the intracranial internal carotid, \basilar, or middle cerebral artery were included less th

229 g the developing tonotopic axis of the chick basilar papilla (BP) identified a gradient of Bmp7.

230 ly phases of cell orientation in the chicken basilar papilla (BP), Vangl2 is present at supporting ce

231 of efferent fibers that innervate the chick basilar papilla (BP).

232 o the cochlear nerve, cochlear ganglion, and basilar papilla (i.e., avian cochlea) in fixed tissue an

233 a macula, whereas SFRP2 is maintained in the basilar papilla along with Fzd10 and Wnt7b.

234 mately characteristic frequency) between the basilar papilla and NM is established as cochlear nerve

235 Flanking the basilar papilla are Wnt7a, Wnt9a, Wnt11, and SFRP2 on th

236 f the substructural alterations in the chick basilar papilla at the earliest signs of hair cell degen

237 ation pathways among supporting cells in the basilar papilla but not in the utricular macula.

238 ed in most support cells in the mature chick basilar papilla but not in vestibular organs of the chic

239 ment sufficient to destroy hair cells in the basilar papilla causes a rapid, transient downregulation

240 ll ears tested, even in non-TM species whose basilar papilla contained as few as 50-60 hair cells.

241 The avian basilar papilla contains tall and short hair cells, with

242 Interestingly, hair cells in the avian basilar papilla demonstrate both electrical resonance an

243 cells that reenter the mitotic cycle in the basilar papilla do not express detectable levels of FGFR

244 s after drug-induced hair cell damage to the basilar papilla in an opposite way to that found in the

245 ations within the apical half of the chicken basilar papilla in vivo and found broadly-tuned travelli

246 in the middle region along the length of the basilar papilla in which, in one cell, the terminals occ

247 re, frequency tuning within the apical avian basilar papilla is not mechanical, and likely derives fr

248 onotopic mapping observed between NM and the basilar papilla later in development.

249 In the chicken cochlea (the basilar papilla or BP), dying hair cells are extruded fr

250 ut patches from hair cells along the chicken basilar papilla revealed 'tonotopic' gradations in calci

251 A semi-intact preparation of the chick basilar papilla was developed to study calcium-dependent

252 Most notably, the basilar papilla, an auditory organ, contained infected s

253 neural edge of the avian auditory organ, the basilar papilla, by embryonic day 5 (E5).

254 Finally, in contrast to the chick basilar papilla, ectopic activation of Notch signaling d

255 the position of their cell bodies along the basilar papilla, foreshadowing the tonotopic mapping obs

256 junctions connecting supporting cells of the basilar papilla, in which its immunofluorescence colocal

257 In the basilar papilla, nuclear cProx1 expression is down-regul

258 uditory sensory organ (the lagena macula and basilar papilla, respectively), which each have a distin

259 ontribute to the structural integrity of the basilar papilla, the maintenance of the ionic barrier at

260 evels within the chicken auditory organ: the basilar papilla.

261 auditory subdivision, the cochlear duct, or basilar papilla.

262 r cell ejection from the proximal end of the basilar papilla.

263 bryonic day 11 (E11) of the developing chick basilar papilla.

264 is expressed early in the development of the basilar papilla.

265 cells from the frog sacculus and the turtle basilar papilla.

266 lication of ACh to hair cells in the chicken basilar papilla.

267 The basilar papillae were studied by conventional transmissi

268 Similarly, in cultured chicken basilar papillae, supporting cell proliferation in respo

269 hout HNSCC specimens, when compared with the basilar pattern of protein expression and minimal inflam

270 addition, loss of Nfib results in defects in basilar pons formation and hippocampus development that

271 In addition, the basilar pons forms normally in dystrophin-deficient mice

272 n in the Large glycosyltransferase gene, the basilar pons is absent.

273 ts from lesions involving the neurons of the basilar pons that link the ipsilateral cerebral cortex w

274 ed in 25 patients with focal infarcts in the basilar pons to determine whether pontine lacunar syndro

275 e required for the normal development of the basilar pons, one of several precerebellar nuclei of the

276 aw region projects mainly to the ipsilateral basilar pons, the MI whisker region has significantly mo

277 culation are localized to rostral and medial basilar pons; hand coordination is medial and ventral in

278 se but not dystrophin is required for normal basilar pontine development.

279 dbrain indicates that the development of the basilar pontine nuclei is delayed, with pontine neurons

280 One such population, the neurons of the basilar pontine nuclei, expresses high levels of Nfi pro

281 increased axonal growth in the deafferented basilar pontine nuclei.

282 rpositus nucleus (IPN) of the cerebellum and basilar pontine nucleus (PN) during different phases of

283 terograde tracing experiments identified the basilar pontine nucleus at the confluence of outputs fro

284 ction from the medial central nucleus to the basilar pontine nucleus.

285 We also provide evidence that the convergent basilar pontine pathways carry corollary discharges from

286 rate sensory (upper body proprioceptive) and basilar pontine pathways onto individual granule cells a

287 esions (pure motor hemiplegia) to incomplete basilar pontine syndrome and restricted deficits after s

288 There was a basilar predominance of interstitial lung disease at CT.

289 n the stria vascularis, spiral ligament, sub-basilar region, stromal tissue, and the spiral and vesti

290 ng, non-severe injury mechanism, no signs of basilar skull fracture, and no severe headache) had a ne

291 The prevalence of > or = 50% vertebral and basilar stenosis in posterior circulation transient isch

292 r = 50% apparently symptomatic vertebral and basilar stenosis using contrast-enhanced MRA in consecut

293 circulation events, > or = 50% vertebral and basilar stenosis was associated multiple transient ischa

294 Presence of > or = 50% vertebral and basilar stenosis was unrelated to age, sex or vascular r

295 whom 37 (26.2%) had > or = 50% vertebral and basilar stenosis, compared with 41 (11.5%) patients with

296 are) CA1 spine density, as well as increased basilar (stratum oriens) CA3 spine density.

297 s, manifested initially as meningitis and/or basilar stroke.

298 Each participant was asked to identify a basilar tip aneurysm in a validated model head.

299 l neurons of the parietal cortex and the CA1 basilar tree of the hippocampus and quantified dendritic

300 white matter tracts and lower volume in the basilar (ventral) pons, cerebellar white matter and visu