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1 roximal occlusions (internal carotid, M1, or basilar arteries).
2 g and in the moments after a blockage of the basilar artery.
3 days (+/- 2 d) and 1.5 days (+/- 1 d) in the basilar artery.
4 ebral arteries and 5 days (+/- 2.5 d) in the basilar artery.
5 intracranial internal carotid artery and the basilar artery.
6 d one patient (1.02%) had an aneurysm of the basilar artery.
7 s coexisting with a fusiform aneurysm of the basilar artery.
8 keel immediately adjacent to the assembling basilar artery.
9 -type Ca2+ channels in native VSMCs from rat basilar artery.
10 tery, the posterior cerebral artery, and the basilar artery.
11 ht account for the effects of alcohol on the basilar artery.
12 e in modulating constrictor responses of the basilar artery.
13 ormed over the ventral medulla to expose the basilar artery.
14 er normal values for the middle cerebral and basilar arteries.
15 asal cisterns and subsequent invasion of the basilar arteries.
16 It involves mostly vertebral and basilar arteries.
17 d wide middle cerebral, internal carotid and basilar arteries.
18 l arteries, posterior cerebral arteries, and basilar arteries.
19 junction conductances in SMC pairs from rat basilar arteries.
21 osterior cerebral arteries, the VAs, and the basilar artery; 4) intracranial atheromatous branch dise
23 h muscle cells were isolated from guinea-pig basilar artery and conventional whole-cell recordings of
26 c oxide synthase-dependent reactivity of the basilar artery and to determine a potential mechanism wh
29 longed obesity mouse model that suffers from basilar artery (BA) abnormalities, we find that microgli
30 We examined the vascular responses of the basilar artery (BA) and its side branches through a cran
33 lood vessels [internal carotid artery (ICA), basilar artery (BA), middle cerebral artery (MCA)], the
34 of this study was to measure the response of basilar artery blood flow to hyperventilation in patient
35 e is augmented in hypertension was tested in basilar artery cells from Wistar rats exhibiting stable
38 ired acetylcholine-induced dilatation of the basilar artery during diabetes mellitus can be restored
39 c oxide synthase-dependent dilatation of the basilar artery during diabetes mellitus may be related,
41 Doppler ultrasonography was used to measure basilar artery flow during rest and after hyperventilati
42 ther cxcl12b or cxcr4a results in defects in basilar artery formation, showing that the assembly and
46 e examined whether impaired responses of the basilar artery in alcohol-fed rats in response to acetyl
48 n of L-NMMA did not affect dilatation of the basilar artery in diabetic rats in response to acetylcho
49 c oxide synthase-dependent dilatation of the basilar artery in diabetic rats towards that observed in
51 RB induced a fusiform-like dilatation of the basilar artery in mice, which was blocked by systemic ad
52 nM) produced dose-related dilatation of the basilar artery in non-alcohol-fed and alcohol-fed rats.
54 roM) produced dose-related dilatation of the basilar artery in non-diabetic rats, which was inhibited
60 A only partially inhibited dilatation of the basilar artery in response to acetylcholine in diabetic
61 ynthase, may contribute to dilatation of the basilar artery in response to acetylcholine in rats trea
62 lin treated diabetic rats, dilatation of the basilar artery in response to acetylcholine was signific
63 ital microscopy, we measured the diameter of basilar artery in response to nitric oxide synthase-depe
72 e Scale [NIHSS] score of 5 or less) and LVO (basilar artery, internal carotid artery, first [M1] or s
75 corrected p=0.010) and decreased flux in the basilar artery (mean difference -0.9 mL/s, 95% CI -1.5 t
76 uently, the Endovascular Treatment for Acute Basilar Artery Occlusion (ATTENTION) and the Basilar Art
79 y perfusion (CTP) parameters and outcomes in basilar artery occlusion (BAO), and to select patients w
82 le to reperfusion therapy; 11 patients had a basilar artery occlusion and were excluded, leaving 100
83 Basilar Artery Occlusion (ATTENTION) and the Basilar Artery Occlusion Chinese Endovascular (BAOCHE) t
84 rnational Cooperation Study (BASICS) and the Basilar Artery Occlusion Endovascular Intervention versu
86 r endovascular thrombectomy is beneficial in basilar artery occlusion now appears to be settled in pa
87 f endovascular thrombectomy in patients with basilar artery occlusion was unclear until recently, bec
88 Five patients undergoing thrombectomy with basilar artery occlusion were excluded due to insufficie
89 late window (>6 h from symptom onset), with basilar artery occlusion, and with large ischaemic core
90 the benefit of thrombectomy in patients with basilar artery occlusion, did not find significant evide
94 e estimated time of onset of a stroke due to basilar-artery occlusion, in a 1:1 ratio, to receive end
96 ic mechanism, distal territory location, and basilar artery occlusive disease carried the poorest pro
97 d dilation that was markedly impaired in the basilar artery of mice expressing dominant-negative muta
98 e smooth muscle cells were isolated from the basilar artery of the guinea-pig and, within 10 h, inwar
100 ral carotid ligation to increase flow in the basilar artery or sham surgery (n = 2 ligated, n = 2 sha
101 ; proximal, middle, or distal segment of the basilar artery; or P1 segment of the posterior cerebral
103 he major artery supplying the hindbrain, the basilar artery, runs along the ventral keel of the hindb
107 nce to that molecule and markedly suppressed basilar artery spasm after subarachnoid hemorrhage.
110 rculation with emphasis on the vertebral and basilar arteries (the posterior cerebral circulation).
112 g posterior fossa lesions, the management of basilar artery thrombosis, which may have a longer time
113 logical parameters that were associated with basilar artery tip aneurysms (BTA) in a location-specifi
114 oxide modulates constrictor responses of the basilar artery to angiotensin II, arginine vasopressin,
115 mann method, allows the 3D flow entering the basilar artery to be finely controlled to replicate the
117 fed rats, and did not alter responses of the basilar artery to nitric oxide synthase-dependent or -in
118 ide dismutase did not alter responses of the basilar artery to nitroglycerin in alcohol-fed rats, and
119 ic stenosis or occlusion in vertebral and/or basilar arteries underwent large-vessel flow measurement
124 rt of the posterior cerebral artery (P1) and basilar artery was observed in the ligated group, as wel
125 f freshly isolated contractile VSMC from rat basilar artery, we found that EGF (100 ng ml(-1)) caused
127 Changes of middle cerebral arteries and basilar arteries were extremely rare, thus we can say th
128 intracisternal delivery of autologous EPCs, basilar arteries were isolated and expression of vasoreg
129 Elongated, ectatic, tortuous vertebral and basilar arteries were the most common angiographic and p
132 uced marked dose-related constriction of the basilar artery which also was similar in nondiabetic and
133 uced only minimal changes in diameter of the basilar artery which were similar in nondiabetic and dia