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1 roximal occlusions (internal carotid, M1, or basilar arteries).
2 g and in the moments after a blockage of the basilar artery.
3 days (+/- 2 d) and 1.5 days (+/- 1 d) in the basilar artery.
4 ebral arteries and 5 days (+/- 2.5 d) in the basilar artery.
5 intracranial internal carotid artery and the basilar artery.
6 d one patient (1.02%) had an aneurysm of the basilar artery.
7 s coexisting with a fusiform aneurysm of the basilar artery.
8  keel immediately adjacent to the assembling basilar artery.
9 -type Ca2+ channels in native VSMCs from rat basilar artery.
10 tery, the posterior cerebral artery, and the basilar artery.
11 ht account for the effects of alcohol on the basilar artery.
12 e in modulating constrictor responses of the basilar artery.
13 ormed over the ventral medulla to expose the basilar artery.
14 er normal values for the middle cerebral and basilar arteries.
15 asal cisterns and subsequent invasion of the basilar arteries.
16             It involves mostly vertebral and basilar arteries.
17 d wide middle cerebral, internal carotid and basilar arteries.
18 l arteries, posterior cerebral arteries, and basilar arteries.
19  junction conductances in SMC pairs from rat basilar arteries.
20                                       Unlike basilar artery, 12 weeks of a high-fat diet was not suff
21 osterior cerebral arteries, the VAs, and the basilar artery; 4) intracranial atheromatous branch dise
22 ertebral artery (40 patients, 12 bilateral), basilar artery (46 patients).
23 h muscle cells were isolated from guinea-pig basilar artery and conventional whole-cell recordings of
24       Carotid ligation increased flow in the basilar artery and terminus and caused differential expr
25                       24 h post-surgery, the basilar artery and terminus were embedded for sectioning
26 c oxide synthase-dependent reactivity of the basilar artery and to determine a potential mechanism wh
27                   One of the patients with a basilar artery aneurysm also had coarctation of the aort
28                                      Two had basilar artery aneurysms, 7 had internal carotid artery
29 longed obesity mouse model that suffers from basilar artery (BA) abnormalities, we find that microgli
30    We examined the vascular responses of the basilar artery (BA) and its side branches through a cran
31 on in endovascular stroke treatment (EVT) of basilar artery (BA) occlusion.
32      Phase-contrast MRA measured flow in the basilar artery (BA), internal carotid arteries (ICA), an
33 lood vessels [internal carotid artery (ICA), basilar artery (BA), middle cerebral artery (MCA)], the
34 of this study was to measure the response of basilar artery blood flow to hyperventilation in patient
35 e is augmented in hypertension was tested in basilar artery cells from Wistar rats exhibiting stable
36 ed for WMLs (Fazekas scale), infarctions and basilar artery diameter (BAD).
37 ad wider posterior communicating but smaller basilar artery diameters.
38 ired acetylcholine-induced dilatation of the basilar artery during diabetes mellitus can be restored
39 c oxide synthase-dependent dilatation of the basilar artery during diabetes mellitus may be related,
40 ic oxide synthase-mediated dilatation of the basilar artery during diabetes mellitus.
41  Doppler ultrasonography was used to measure basilar artery flow during rest and after hyperventilati
42 ther cxcl12b or cxcr4a results in defects in basilar artery formation, showing that the assembly and
43                                          The basilar artery geometry strongly influenced both skewing
44 uding vertebral occlusion extending into the basilar artery, have shown inconsistent results.
45 ), M1 in 207 (28.4%), M2 in 395 (54.2%), and basilar artery in 30 (4.1%).
46 e examined whether impaired responses of the basilar artery in alcohol-fed rats in response to acetyl
47 itric oxide synthase-dependent dilatation of basilar artery in alcohol-fed rats.
48 n of L-NMMA did not affect dilatation of the basilar artery in diabetic rats in response to acetylcho
49 c oxide synthase-dependent dilatation of the basilar artery in diabetic rats towards that observed in
50  a direct acidifier of the cell, dilates rat basilar artery in K(ATP)-dependent fashion.
51 RB induced a fusiform-like dilatation of the basilar artery in mice, which was blocked by systemic ad
52  nM) produced dose-related dilatation of the basilar artery in non-alcohol-fed and alcohol-fed rats.
53              We measured the diameter of the basilar artery in non-diabetic rats, diabetic (streptozo
54 roM) produced dose-related dilatation of the basilar artery in non-diabetic rats, which was inhibited
55 ) did not alter constrictor responses of the basilar artery in nondiabetic and diabetic rats.
56 duced similar dose-related dilatation of the basilar artery in nondiabetic and diabetic rats.
57 U/ml) did not alter baseline diameter of the basilar artery in nondiabetic and diabetic rats.
58 roM) produced dose-related dilatation of the basilar artery in nondiabetic and diabetic rats.
59 de dismutase did not alter dilatation of the basilar artery in nondiabetic rats.
60 A only partially inhibited dilatation of the basilar artery in response to acetylcholine in diabetic
61 ynthase, may contribute to dilatation of the basilar artery in response to acetylcholine in rats trea
62 lin treated diabetic rats, dilatation of the basilar artery in response to acetylcholine was signific
63 ital microscopy, we measured the diameter of basilar artery in response to nitric oxide synthase-depe
64               In contrast, dilatation of the basilar artery in response to nitroglycerin was similar
65                            Dilatation of the basilar artery in response to nitroglycerin was similar
66 c oxide played a role in constriction of the basilar artery in response to the agonists.
67 ine were not affected in either aorta or the basilar artery in vitro.
68              We measured the diameter of the basilar artery in vivo in nondiabetic and diabetic rats
69 c oxide synthase-dependent reactivity of the basilar artery in vivo.
70 tes mellitus on constrictor responses of the basilar artery in vivo.
71                          Measurements of the basilar artery indicated a considerable hypertrophy, ind
72 e Scale [NIHSS] score of 5 or less) and LVO (basilar artery, internal carotid artery, first [M1] or s
73                                          The Basilar Artery International Cooperation Study (BASICS)
74                                              Basilar artery maximal diameter was greater in SF mice (
75 corrected p=0.010) and decreased flux in the basilar artery (mean difference -0.9 mL/s, 95% CI -1.5 t
76 uently, the Endovascular Treatment for Acute Basilar Artery Occlusion (ATTENTION) and the Basilar Art
77                                              Basilar artery occlusion (BAO) is a devastating conditio
78                                        Acute basilar artery occlusion (BAO) is a rare but potentially
79 y perfusion (CTP) parameters and outcomes in basilar artery occlusion (BAO), and to select patients w
80 itive results also apply to the patient with basilar artery occlusion (BAO).
81 ase (TPA) increases the likelihood of FPE in basilar artery occlusion (BAO).
82 le to reperfusion therapy; 11 patients had a basilar artery occlusion and were excluded, leaving 100
83 Basilar Artery Occlusion (ATTENTION) and the Basilar Artery Occlusion Chinese Endovascular (BAOCHE) t
84 rnational Cooperation Study (BASICS) and the Basilar Artery Occlusion Endovascular Intervention versu
85                                              Basilar artery occlusion is a rare and severe condition.
86 r endovascular thrombectomy is beneficial in basilar artery occlusion now appears to be settled in pa
87 f endovascular thrombectomy in patients with basilar artery occlusion was unclear until recently, bec
88   Five patients undergoing thrombectomy with basilar artery occlusion were excluded due to insufficie
89  late window (>6 h from symptom onset), with basilar artery occlusion, and with large ischaemic core
90 the benefit of thrombectomy in patients with basilar artery occlusion, did not find significant evide
91           Trials of endovascular therapy for basilar artery occlusion, including vertebral occlusion
92 ar therapy in patients with stroke caused by basilar-artery occlusion has not been well studied.
93              Among patients with stroke from basilar-artery occlusion, endovascular therapy and medic
94 e estimated time of onset of a stroke due to basilar-artery occlusion, in a 1:1 ratio, to receive end
95 icacy and safety of endovascular therapy for basilar-artery occlusion.
96 ic mechanism, distal territory location, and basilar artery occlusive disease carried the poorest pro
97 d dilation that was markedly impaired in the basilar artery of mice expressing dominant-negative muta
98 e smooth muscle cells were isolated from the basilar artery of the guinea-pig and, within 10 h, inwar
99 ial recanalization of the middle cerebral or basilar artery on TCD were studied.
100 ral carotid ligation to increase flow in the basilar artery or sham surgery (n = 2 ligated, n = 2 sha
101 ; proximal, middle, or distal segment of the basilar artery; or P1 segment of the posterior cerebral
102 = 0.05) and relative signal intensity in the basilar artery (p = 0.04; sham-group unchanged).
103 he major artery supplying the hindbrain, the basilar artery, runs along the ventral keel of the hindb
104                                  The greater basilar artery sensitivity to hyperventilation shown by
105                       Our data indicate that basilar artery SMCs are coupled in vivo in a richly comp
106 ry effects on voltage-gated Ca2+ channels of basilar artery smooth muscle cells.
107 nce to that molecule and markedly suppressed basilar artery spasm after subarachnoid hemorrhage.
108 any benefit in the treatment of vertebral or basilar artery stenosis.
109                Patients with isolated tip of basilar artery (TBA) occlusion had significantly more BP
110 rculation with emphasis on the vertebral and basilar arteries (the posterior cerebral circulation).
111             A 6-year-old male with vertebral-basilar artery thrombosis was recognized to have high-mo
112 g posterior fossa lesions, the management of basilar artery thrombosis, which may have a longer time
113 logical parameters that were associated with basilar artery tip aneurysms (BTA) in a location-specifi
114 oxide modulates constrictor responses of the basilar artery to angiotensin II, arginine vasopressin,
115 mann method, allows the 3D flow entering the basilar artery to be finely controlled to replicate the
116                       Thus, responses of the basilar artery to important vasoactive agonists are not
117 fed rats, and did not alter responses of the basilar artery to nitric oxide synthase-dependent or -in
118 ide dismutase did not alter responses of the basilar artery to nitroglycerin in alcohol-fed rats, and
119 ic stenosis or occlusion in vertebral and/or basilar arteries underwent large-vessel flow measurement
120                            We examined NT in basilar artery vascular smooth muscle cells (VSMCs) from
121                            The prevalence of basilar artery vasospasm in children with moderate traum
122                            Reactivity of the basilar artery was measured 2-3 months after injection o
123                          The diameter of the basilar artery was measured using intravital microscopy
124 rt of the posterior cerebral artery (P1) and basilar artery was observed in the ligated group, as wel
125 f freshly isolated contractile VSMC from rat basilar artery, we found that EGF (100 ng ml(-1)) caused
126                                       Rabbit basilar arteries were exposed to autologous EPCs ( appro
127      Changes of middle cerebral arteries and basilar arteries were extremely rare, thus we can say th
128  intracisternal delivery of autologous EPCs, basilar arteries were isolated and expression of vasoreg
129   Elongated, ectatic, tortuous vertebral and basilar arteries were the most common angiographic and p
130                              Rings of canine basilar artery were incubated with increasing titers of
131            The responses to acetylcholine in basilar artery were restored to normal after treatment w
132 uced marked dose-related constriction of the basilar artery which also was similar in nondiabetic and
133 uced only minimal changes in diameter of the basilar artery which were similar in nondiabetic and dia
134             In vitro or in vivo treatment of basilar artery with conditioned media from EPCs also cau

 
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