ライフサイエンスコーパス: basket cell

1 ectively suppress the output of one class of basket cell.

2 are highly enriched in hilar mossy cells and basket cells.

3 gly, significantly later than downstream CA1 basket cells.

4 he synaptic output of parvalbumin-expressing basket cells.

5 parvalbumin- and cholecystokinin-expressing basket cells.

6 ly modulated, including parvalbumin-positive basket cells.

7 lso enhance the distinct network function of basket cells.

8 ctive cationic conductance to depolarize PV+ basket cells.

9 ucing inhibition in fast-spiking parvalbumin basket cells.

10 induced depression of GABA release from CCK+ basket cells.

11 sing GABA release from CCK expressing (CCK+) basket cells.

12 nct classes of GABAergic interneurons called basket cells.

13 n-containing GABAergic interneurons known as basket cells.

14 required for the terminal differentiation of basket cells.

15 eep Layer II principal neurons and Layer III basket cells.

16 excitation (DSE) is present in both SCs and basket cells.

17 I markedly depends on the activity levels of basket cells.

18 extent, perikarya, which resembled GABAergic basket cells.

19 normally excite insult-resistant inhibitory basket cells.

20 arly fast-spiking parvalbumin-positive (PV+) basket cells.

21 lcium binding proteins that are expressed by basket cells.

22 type and certain morphological attributes of basket cells.

23 from synaptic terminals of mouse cerebellar basket cells.

24 ebellum, in particular pyramidal neurons and basket cells.

25 tly expressed in the terminals of cerebellar basket cells.

26 eurons, specifically of parvalbumin-positive basket cells.

27 r to require the involvement of fast-spiking basket cells.

28 ule cells (GCs) by parvalbumin (PV)-positive basket cells.

29 rent from the dendritic arborizations of CA1 basket cells.

30 or the bistratified cells (1:7) than for the basket cells (1:22).

31 By suppressing sIPSCs from CCK-expressing basket cells, a CB(1) agonist reverted the stimulatory e

32 und that the spike timing of CB1R-expressing basket cells, a major target for cannabinoids in the rat

33 pal CA3 regions, parvalbumin (PV)-expressing basket cells, activated by ACh and glutamatergic agonist

34 ited pyramidal cells, resulting in increased basket cell activation and gamma power.

35 on of parallel fibers but not in response to basket cell activity.

36 contribute to the precisely timed phasing of basket cell activity.

37 1 (CB(1)), another marker for CCK-containing basket cells, also labeled fewer boutons in pilocarpine-

38 elonging to Purkinje, granule, stellate, and basket cells, although Purkinje cell dendrites, includin

39 lls at the gamma peak but lag at trough; (5) basket cells amplify theta rhythms; (6) ketamine alters

40 fast kinetics and, whenever tested (n = 5; 1 basket cell and 4 Schaffer-associated interneurons), wer

41 tage deflection at steady state) was 1.69 in basket cells and 0.23 in stellate cells.

42 lls), whereas parvalbumin(+) subpopulations (basket cells and chandelier cells) seem to be unaffected

43 tions at two subtypes of PV(+) interneurons: basket cells and chandelier cells.

44 ich primarily affected Purkinje cells (PCs), basket cells and climbing fibres, in individuals with ET

45 action of CCK on GABA release both from CCK+ basket cells and dendritically projecting, CCK+ Schaffer

46 Fast-spiking inhibitory basket cells and excitatory pyramidal neurons received s

47 n arborization preceding the terminal of the basket cells and is therefore a potential candidate for

48 In contrast, regular-spiking basket cells and ivy cells were less likely to be innerv

49 pulations of neocortical inhibitory neurons: basket cells and Martinotti cells.

50 ally targeting parvalbumin (PARV)-containing basket cells and neuropeptide Y (NPY) interneurons.

51 large decrease in intrinsic excitability of basket cells and oriens-lacunosum moleculare interneuron

52 MORs are abundantly expressed by basket cells and other inhibitory interneurons of CA1.

53 aired recording experiments from presynaptic basket cells and postsynaptic CA1 pyramidal cells in acu

54 a26YFP mice, were anatomically identified as basket cells and PV bistratified cells in the stratum py

55 n neurons into three classes: bipolar cells, basket cells and radial cells, respectively.

56 osynaptic mossy fiber inputs to fast-spiking basket cells and spiny stratum lucidum cells were found

57 ivision of molecular layer neurons into deep basket cells and superficial stellate cells is not suppo

58 inhibitory synapses made by CCK(+)VGlut3(+) basket cells and the inhibitory drive they exerted on py

59 Excitatory synaptic input to basket cells and unitary IPSCs (uIPSCs) from basket cell

60 number of direct neighbors to be ~4 for rat basket cells and ~1 for rat stellate cells.

61 rcuit inhibitory interneuron, the so-called 'basket cells' and 'Schaffer collateral-associated' cells

62 receive input from different combinations of basket cells, and a close association between axonal mor

63 yramidal cells, parvalbumin-positive (PV(+)) basket cells, and an unidentified class of anti-SWR inte

64 ells, neurogliaform cells, irregular spiking basket cells, and regular spiking presumptive basket cel

65 (+) interneurons in addition to the expected basket cells, and their extensive circuit innervation pr

66 segments, few cholecystokinin (CCK)-positive basket cells, and very low gamma-aminobutyric acid trans

67 ing interneurons compared to PARV-containing basket cells; and (2) may provide a more powerful (i.e.,

68 Cholecystokinin-positive (CCK+) basket cells are a major source of perisomatic GABAergic

69 of CA1 pyramidal cells, these two classes of basket cells are active at different times.

70 Networks of parvalbumin (PV)-expressing basket cells are implicated in synchronizing cortical ne

71 Parvalbumin-expressing, fast-spiking basket cells are important for the generation of synchro

72 Basket cells are important inhibitors of granule cells.

73 We also find that basket cells are integrated into specific inhibitory sub

74 , supporting the view that PV(+) fast-firing basket cells are more likely to exhibit class 2 excitabi

75 e not consistently vulnerable and inhibitory basket cells are not consistently seizure resistant.

76 ced selective and powerful excitation of PV+ basket cells are not understood.

77 Here, we show that basket cells are selectively modulated by either opioids

78 Parvalbumin-positive (PV+) fast-spiking basket cells are thought to play key roles in network fu

79 gment oscillations; (4) pyramidal cells lead basket cells at the gamma peak but lag at trough; (5) ba

80 ADAM11 spares spontaneous GABA release from basket cells at the perisomatic synapse yet eliminates u

81 ntly differed between parvalbumin-containing basket cells, axoaxonic cells, and type 1 cannabinoid re

82 resis of l-aspartate, an NMDAR agonist, onto basket cell axon collaterals had no effect on evoked IPS

83 n for functional innervation by CCK-positive basket cell axon terminals was confirmed by reduced freq

84 tic synapse formation impairs the process of basket cell axonal branching and bouton formation.

85 teral formation and hypertrophy of GABAergic basket cell axonal processes, could be compensatory resp

86 ar structure formed by descending GABA-ergic basket cell axonal terminals converging on the initial a

87 u are not understood because the majority of basket cell axons fail to make identifiable synaptic con

88 ic vicinity of PCs in which large boutons of basket cell axons form synapses on the PC soma.

89 rmally densely clustered at the terminals of basket cell axons in the cerebellar cortex.

90 er collaterals in the hippocampal formation, basket cell axons in the cerebellar pinceau, and granule

91 nd the number of synapses made by inhibitory basket cell axons is reduced to 38%.

92 o the type II GABAergic synapses made by the basket cell axons on Purkinje cell somata.

93 ease machinery to test for NMDAR activity in basket cell axons.

94 d strong, selective localization of Kv3.2 to basket cell axons.

95 Kv1.2 is also expressed in cerebellar basket cell (BC) axon terminals, where its blockade incr

96 lar Purkinje cells and hippocampal pyramidal basket cells, BCATc is concentrated in cell bodies.

97 re, we find that excessive GABA release from basket cells (BCs) attenuates the firing frequency of Pu

98 ll (PC) inputs to Martinotti cells (MCs) and basket cells (BCs) in layer 5 of the developing mouse vi

99 essing (VIP) and parvalbumin-expressing (PV) basket cells (BCs) in mouse hippocampal CA1.

100 In cerebellum, basket cells (BCs) innervate the soma and axon initial s

101 ramidal neurons, chandelier cells (ChCs) and basket cells (BCs), are generally thought to have the sa

102 ound in the PFC, chandelier cells (ChCs) and basket cells (BCs), are thought to play different roles

103 f PV(+) neurons, chandelier cells (ChCs) and basket cells (BCs), has not been determined.

104 mp and 2-photon laser scanning microscopy of basket cells (BCs), we found that classical excitatory p

105 ive (PV+) or cholecystokinin-positive (CCK+) basket cells (BCs), we tested the hypothesis that neuroc

106 bumin (PV)-expressing perisomatic inhibitory basket cells (BCs), whereas BCs and HICAPs rarely target

107 ltage-gated sodium currents were impaired in basket cells (BCs).

108 ervate pyramidal neuron perisomatic regions (basket cells, BCs) were depolarized by muscarinic recept

109 kinin- (CCK) and parvalbumin (PV)-containing basket cells, beginning with differences at the level of

110 urogliaform cells, double bouquet cells, net basket cells, bitufted cells, and regular-spiking pyrami

111 A cerebellar basket cell bouton, a pinceau surrounding a Purkinje cel

112 TK (250 nm) abolished GABA release from PV+ basket cells, but it had no effect on the CCK-induced de

113 Kv1.1 deletion enhances excitability of the basket cells by selectively enhancing the likelihood of

114 Two major subclasses of CA1 basket cells can be identified based on their expression

115 ppressing neurotransmitter release in single basket cells can have completely opposite effects depend

116 uting of inhibitory cholecystokinin-positive basket cells (CCK(+) BCs), through enhanced inhibition o

117 cteristics of the cholecystokinin-expressing basket cells (CCK-BC).

118 interneuron, the cholecystokinin-expressing basket cell (CCKBC), is particularly well suited to inte

119 findings suggest dysfunction of the dentate basket cell circuit could contribute to hyperexcitabilit

120 his review explores the dichotomy of the two basket cell classes, cholecystokinin- (CCK) and parvalbu

121 apses interconnecting parvalbumin-expressing basket cells contained a 3.6 times higher overall densit

122 sleep and quiet wakefulness, suggesting that basket cells coordinate cell assemblies in a behavioral

123 ifferential modulation of these two types of basket cells could hence be important for regulating the

124 and GABAergic neurons, which means that the basket cells could play a key role in the generation of

125 ng events, CCK2 receptors on neighboring PV+ basket cells couple to an unusual, pertussis-toxin-sensi

126 ree other cell types, including fast-spiking basket cells, CR-negative Martinotti cells, and bipolar

127 o altered parvalbumin expression rather than basket cell death, because substance P receptor-positive

128 Basket cells, defined by axons that preferentially conta

129 Basket cells did not contribute to gamma oscillations ge

130 The phases of stellate and basket cell discharge suggests that their activity could

131 Granule, unipolar brush, stellate, and basket cells discharge in phase with ipsilateral roll ti

132 The distinct spike timing of basket cells during oscillations in CA1 and CA2/3 sugges

133 hoc identified cholecystokinin-positive CA1 basket cells elicited IPSCs in the postsynaptic pyramida

134 We found that both stellate and basket cells engaged in synchronized waves of calcium ac

135 ermore, CCK decreased GABA release from CCK+ basket cells even in the presence of the GABAB receptor

136 6 mutant cortical interneurons revealed that basket cells, even when mispositioned, retain characteri

137 aspects of somatic inhibition from different basket cells, evidence that different principal cell pop

138 dings, action potentials in presynaptic CCK+ basket cells evoked large IPSCs with fast kinetics in py

139 Although basket cells exert a powerful inhibitory influence on th

140 Fast-spiking, parvalbumin-expressing, basket cells express muORs, but circumstantial evidence

141 nit variant, whereas cerebellar stellate and basket cells express only the beta2 as the beta subunit.

142 normal integration into cortical circuits of basket cells expressing CCK and vesicular glutamate tran

143 somatic inhibition of pyramidal neurons from basket cells expressing cholecystokinin (CCK(b) cells) a

144 During theta oscillations, CA2/3 basket cells fired on the same phase as putative pyramid

145 Area CA2/3 basket cells fired phase locked to both CA2/3 and CA1 ga

146 tude or frequency of miniature IPSCs and the basket cell firing frequency did not differ between grou

147 comprehensively investigated, the pattern of basket cell firing was strongly influenced by the activi

148 Axons of surviving NeuroD2-deficient basket cells follow irregular trajectories and their inh

149 It is argued that the diversity in basket cell form in the piriform cortex, as in other are

150 Basket cells form dense inhibitory plexuses that wrap Pu

151 tes inhibitory feedback between fast-spiking basket cells (FS cells) and star pyramidal neurons (star

152 ange projection pattern in the definition of basket cell function.

153 Selective depletion of basket cell GABAergic neurotransmission increases the fr

154 compare how the lack of stellate cell versus basket cell GABAergic neurotransmission sculpts the firi

155 igh-frequency trains of action potentials in basket cells generated uIPSCs in granule cells to evalua

156 Basket cells had a synaptic target preference for somata

157 hibitory neuron, parvalbumin-expressing (PV) basket cells, have selectively reduced activity in a mod

158 The "dormant basket cell" hypothesis suggests that postinjury hippoca

159 unitary synaptic connections between GCs and basket cells in acute cerebellar slices from wild-type a

160 d the spike timing of parvalbumin-expressing basket cells in areas CA2/3 of anesthetized rats in rela

161 dependent generation of fast oscillations by basket cells in CA1 and CA2/3.

162 ts suggest that normal integration of CCK(+) basket cells in cortical networks is key to support spat

163 In contrast, the number of CCK and PV-IR basket cells in epileptic animals was similar to that in

164 ramidal cell innervation from CCK-expressing basket cells in mice with spontaneous seizures and a gre

165 ransmitter release, in mouse cortical single basket cells in slice cultures decreases the number of i

166 ctedly large dendritic arborization of CA2/3 basket cells in stratum lacunosum moleculare (33% of len

167 timing of identified parvalbumin-expressing basket cells in the CA1 hippocampus of anesthetized rats

168 The networks of PV basket cells in the DG are regulated by vesicular releas

169 c acid neurons (i.e., parvalbumin-containing basket cells) in layer 3 of the DLPFC.

170 parvalbumin-expressing interneurons (mostly basket cells) in sector CA1/subiculum is sufficient to i

171 projecting fast-spiking and regular-spiking basket cells, in addition to the dendritically projectin

172 proposing that the CCK-induced firing of PV+ basket cells increases the release of GABA, which, in tu

173 ng receptors in synapses made by PV-negative basket cells, indicate that the number and subtypes of G

174 ompatible to the arborization range of small basket cells (inhibitory neurons) in AI.

175 Cortical basket cells innervate hundreds of pyramidal cell somata

176 scopic investigations to show that, although basket cells innervate the entire somato-denditic membra

177 Broad CA2 basket cells innervated all three CA subfields and could

178 In the cerebellum, the basket cell innervation on Purkinje cells provides a maj

179 dendritic synapses indicating the absence of basket cell innervation.

180 a significant increase in nNOS expression in basket cell interneurons in both mutant mice.

181 of inhibitory control accomplished by single basket cells is also variable.

182 icient control of principal neuron firing by basket cells is critical for information processing in c

183 risomatic inhibition mediated by PV-positive basket cells is pruned by sensory deprivation.

184 Spontaneous firing rate of the basket cells is unaltered.

185 sults reveal that GABA release from CA1 CCK+ basket cells is under homosynaptic tonic inhibition by e

186 not known whether GABA release from CA1 CCK+ basket cells is under tonic endocannabinoid inhibition.

187 cal types of somata were postsynaptic to the basket cells: large (20-30-microm) oval cells with dark

188 Thus, PV-positive basket cells mainly act through alpha1 subunit-containin

189 Parvalbumin-expressing basket cells, making GABAergic synapses onto cell bodies

190 (CCK) is able to selectively depolarize PV+ basket cells, making these cells sensitive biosensors fo

191 Thus, alterations in basket cells may underlie the cortical oscillation defic

192 Excitatory synaptic drive to basket cells, measured by mean charge transfer and frequ

193 h number of GABA(A) receptors in synapses on basket cells might contribute to the precisely timed pha

194 Particularly, basket cells might serve as target for innovative therap

195 receptors than synapses made by PV-negative basket cells, most of which were immunonegative.

196 bumin-expressing interneurons, including the basket cell network which is fundamental to gamma oscill

197 ramidal neurons, including chandelier cells, basket cells, neurogliaform cells, double bouquet cells,

198 ar granule cell parallel fiber terminals and basket cell neurons where they serve to inhibit the rele

199 cular layer interneurons (MLIs, stellate and basket cells) of the cerebellar cortex are linked togeth

200 th synapses formed by boutons of PV-positive basket cells on pyramidal cells.

201 Synapses formed by PV-positive basket cells on the somata of pyramidal cells had severa

202 As the innervation patterns of individual basket cells on their different postsynaptic partners sh

203 fired with high frequency and in-phase with basket cells, on average 1-2 ms after the discharges in

204 s of cholecystokinin-expressing, PV-negative basket cells onto pyramidal cells.

205 parvalbumin- and cholecystokinin-expressing basket cells onto pyramidal neurons.

206 nd axonal properties that were distinct from basket cells or interneurons expressing various calcium-

207 ecordings from CCK-positive perisomatically (basket cells) or dendritically projecting (Schaffer coll

208 By contrast, parvalbumin (PV)-expressing basket cells originate mostly from the rostral MGE, wher

209 A specialized axonal ending, the basket cell "pinceau," encapsulates the Purkinje cell ax

210 SD95 and GAD67 unexpectedly mark patterns of basket cell pinceaux that map onto Purkinje cell functio

211 Perisomatic inhibition from basket cells plays an important role in regulating pyram

212 r specializations in the synapses of the two basket cell populations, we used quantitative electron m

213 In both neurochemical classes basket cells predominate, but both groups included subpo

214 scillations from intrinsic network dynamics: basket cells primarily generated gamma and amplified the

215 uit features he exemplified using cerebellar basket cell projections.

216 cholecystokinin- and parvalbumin-containing basket cells provide equally potent control of principal

217 Normally, hippocampal basket cells provide strong and reliable synaptic inhibi

218 lbumin- and cholecystokinin (CCK)-expressing basket cells provide two parallel, functionally distinct

219 In contrast, VIP-positive basket cells provided perisomatic inhibition to CA1 pyra

220 ine the time course of release at inhibitory basket cell-Purkinje cell synapses and show that it is i

221 ators reveal that channel-sensor coupling at basket cell-Purkinje cell synapses is very tight, with a

222 ts to pyramidal neurons from the parvalbumin basket cell (PVBC) class of GABAergic neurons.

223 pplied this approach to quantify parvalbumin basket cell (PVBC) inputs in area 9 of the dorsolateral

224 isomatically projecting parvalbumin-positive basket cells (PVBCs) and distal dendritically projecting

225 deling, we found that parvalbumin-expressing basket cells (PVBCs) evoked greater inhibition in CA1 PC

226 nvestigated GABAergic parvalbumin-expressing basket cells (pvBCs) in layer 2/3 (L2/3) in human neocor

227 decrease in excitatory synaptic drive to PV basket cells (PVBCs) likely underlies reduced function.

228 eurons include chandelier cells (PVChCs) and basket cells (PVBCs), which innervate the axon initial s

229 a 46% spike rate reduction during SWRs in PV basket cells (PVBCs), while PV bistratified and axo-axon

230 bistratified cells (range, 0.5-9 mV) than in basket cells (range, 0.15-3.6 mV) and the probability of

231 Therefore, in epileptic animals, basket cells receive less excitatory synaptic drive, the

232 asket cells, and regular spiking presumptive basket cells, received strong excitatory input from laye

233 ata set of excitatory neurons and inhibitory basket cells reconstructed from cat primary visual corte

234 However, the presynaptic basket cells recovered from the cannabinoid agonist-indu

235 in the cerebellum, stellate cells (SCs) and basket cells, regulate the strength of parallel fiber (P

236 In contrast, CB1R-positive basket cells responded later and did not follow repetiti

237 CB1R-negative basket cells responded reliably and immediately to subtl

238 e (3.9-11.2 ms) were slightly longer than in basket cells (rise times, 0.4-1.6 ms; half-widths, 2.2-9

239 s generated around stratum pyramidale, where basket cells selectively innervate pyramidal cells with

240 abinoid type 1 receptor-expressing GABAergic basket cells selectively innervated principal cells in l

241 with classical GABA(A,fast) IPSCs evoked by basket cells, single spikes in neurogliaform cells evoke

242 Whole-cell recordings from basket cell somata also revealed an I(h) current, which

243 terneurons, which include the CCK-expressing basket cells, strongly suppressed inhibitory oscillation

244 s and in presynaptic terminals of cerebellar basket cells, structures with a high density of I(h).

245 In three basket cells studied at the ultrastructural level 6 of 6

246 voked by an action potential in the recorded basket cells, suggested that coupled interneurones, poss

247 Basket cell synapses predominantly express beta2-subunit

248 ions suggest that subcellular alterations at basket cell synapses rather than chandelier cell synapse

249 s plasticity was observed at somatodendritic basket cell synapses, but not at distal dendritic stella

250 ate is coreleased with GABA from hippocampal basket cell-synapses to act on NMDA receptors.

251 axons, axon initial segments, and cerebellar basket cell terminals (BCTs).

252 .1 and Kv1.2 localize normally in cerebellar basket cell terminals and the juxtaparanodal region of m

253 pyramidal cells caused loss of CCK-positive basket cell terminals in hippocampus and neocortex.

254 isually targeted patch-clamp recordings from basket cell terminals of mice harbouring an ataxia-assoc

255 inhibits GABA release from neighbouring CCK+ basket cell terminals through presynaptic GABAB receptor

256 PV-positive basket cell terminals were unaffected in mutant mice, de

257 positive hippocampal interneurons and in CA1 basket cell terminals.

258 , electrical communication is stronger among basket cells than among stellate cells.

259 tors are coexpressed in parvalbumin-positive basket cells that are critical for gamma oscillations.

260 the perisomatic region of pyramidal cells by basket cells that coexpress the cannabinoid type 1 recep

261 particularly true for a major population of basket cells that express the neuropeptide cholecystokin

262 struct a terminal differentiation program in basket cells that regulates targeted axon growth and inh

263 aptic depolarization, although in two (of 4) basket cells the voltage relation was conventional.

264 s, was distinct from the other main group of basket cells, the CB1R-negative.

265 Furthermore, in basket cells, these receptors were associated with parti

266 basket cells and unitary IPSCs (uIPSCs) from basket cells to granule cells were evaluated in hippocam

267 fast calcium-permeable AMPA receptors enable basket cells to respond rapidly, such that they promptly

268 impaired inhibition in epileptic animals at basket cell-to-granule cell (BC-->GC) synapses, which no

269 In a rat model of temporal lobe epilepsy, basket cell-to-granule cell (BC-->GC) synaptic transmiss

270 and a trend toward lower amplitude uIPSCs at basket cell-to-granule cell synapses in epileptic rats.

271 les are smaller, and transmission failure at basket cell-to-granule cell synapses is increased.

272 However, quantal size at basket cell-to-granule cell synapses was larger and RRP

273 zed in synaptic vesicles in the terminals of basket cells together with GABA-containing vesicles.

274 Spike timing of basket cells tuned the phase and amplitude of gamma osci

275 Our results show that both basket cell types can powerfully regulate the activity i

276 mporal separation in the activity of the two basket cell types generated distinct epochs of somatic i

277 to Martinotti cells, while leaving those to basket cells unaffected.

278 Furthermore, calcium indicators in basket cell varicosities did not report any change in in

279 We find no evidence for functional NMDARs in basket cell varicosities.

280 SCs (sIPSCs) originating from CCK-containing basket cells was accordingly reduced in CA1 pyramidal ce

281 associated with sharp waves, firing of CA2/3 basket cells was phase locked only to local but not CA1

282 ells and two types of interneurons (O-LM and basket cells), we show here that the O-LM interneurons l

283 piking bistratified, and 3 of 5 burst-firing basket cells were also PV-IR.

284 , parvalbumin- or cholecystokinin-expressing basket cells were found to be similar.

285 Basket cells were identified by electrophysiological and

286 Two types of basket cells were identified.

287 g interneuron groups; parvalbumin-expressing basket cells were one of the most active GABAergic cells

288 ic inputs from murine parvalbumin-expressing basket cells were selectively modulated by the membrane

289 type 1 cannabinoid receptor (CB1)-expressing basket cells, which might explain their distinct recruit

290 modulation of CA3 and CA1 pyramidal cells by basket cells, which receive different inputs.

291 reciprocally connected parvalbumin-positive basket cells, which start ripple-frequency spiking that

292 ly depolarizing parvalbumin expressing (PV+) basket cells while indirectly depressing GABA release fr

293 s the output from rat parvalbumin-expressing basket cells, while concurrently suppressing GABA releas

294 Basket cells, whose axon terminals surround principal ce

295 Er81 protein levels define a spectrum of FS basket cells with different properties, whose relative p

296 their axonal arbours in the SP, and included basket cells with somata in the SP (6), SO (3), and SR (

297 Of 36 PV-IR cells, 29 were basket cells, with most of their axonal arbours in the s

298 Of 27 CCK-IR neurones, 13 were basket cells, with most of their axonal arbours in the S

299 To determine the allocation of stellate/basket cells within the ML, we examined their migration

300 T2) mouse conditional allele, we reveal that basket cell zones comprise different sizes of pinceaux.