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1 pus but did not prevent hyperactivity in the basolateral amygdala.
2 y transmission in the PFC at inputs from the basolateral amygdala.
3 contralateral cortex, nucleus accumbens, and basolateral amygdala.
4 l hippocampus, and projection neurons in the basolateral amygdala.
5 mmunofluorescence, and RNA sequencing of the basolateral amygdala.
6 cortex via the paraventricular thalamus and basolateral amygdala.
7 ippocampal-entorhinal cortex network and the basolateral amygdala.
8 INs, and principal excitatory neurons in the basolateral amygdala.
9 exclusively within excitatory neurons of the basolateral amygdala.
10 mbens, the medial prefrontal cortex, and the basolateral amygdala.
11 gulation of AMPA-receptor endocytosis in the basolateral amygdala.
12 in emotion-related circuits anchored in the basolateral amygdala.
13 d striatum and remained intact in cortex and basolateral amygdala.
14 eceptors on output projection neurons of the basolateral amygdala.
15 enlargement is localized specifically to the basolateral amygdala.
16 ses and theta oscillations recorded from the basolateral amygdala.
17 ion, and these effects are most prominent in basolateral amygdala.
18 ering protein expression within the adjacent basolateral amygdala.
19 rentially modulated through the vSub and the basolateral amygdala.
20 a direct pathway between anterior insula and basolateral amygdala.
21 h a marker of synaptic plasticity within the basolateral amygdala.
22 y transmission in the PFC at inputs from the basolateral amygdala.
23 in GABA- and glutamate-positive cells in the basolateral amygdala.
24 lective potentiator CIQ bilaterally into the basolateral amygdala (3, 10, or 30 mug/side) following e
25 focused on the primary output neurons of the basolateral amygdala, a brain region that plays a key ro
26 chronized theta oscillations between V2L and basolateral amygdala, a physiological correlate of succe
27 dal neurons such as hippocampal area CA1 and basolateral amygdala, a slow afterhyperpolarization (sAH
28 eported neural correlates of salience in the basolateral amygdala (ABL) of rats during learning in an
32 norepinephrine (NE) administration into the basolateral amygdala after training on an inhibitory avo
33 cortical nucleus, whereas in the lateral and basolateral amygdala alpha1, alpha2, alpha5, beta1, beta
37 et, PTEN, colocalized with miR-144-3p in the basolateral amygdala and showed functional downregulatio
40 scillatory neuronal interactions between the basolateral amygdala and the rostral anterior cingulate
42 s (accumbens shell, ventral hippocampus, and basolateral amygdala) and differences (medial prefrontal
43 terior cingulate cortex, infralimbic cortex, basolateral amygdala, and habenula inhibited social play
44 e thalamus, bed nucleus of stria terminalis, basolateral amygdala, and medial prefrontal cortex as pr
46 revealed that functional Hcn channels in the basolateral amygdala are necessary for conditioned fear
47 macology, revealing that Hcn channels in the basolateral amygdala are required for fear acquisition a
51 valbumin-expressing (PV) interneurons in the basolateral amygdala (BLA) a dedicated role in the selec
52 he dorsomedial prefrontal cortex (dmPFC) and basolateral amygdala (BLA) and altered synaptic transmis
53 ing the intrinsic functional connectivity of basolateral amygdala (BLA) and centromedial amygdala (CM
54 question, we recorded activity in the mPFC, basolateral amygdala (BLA) and dorsal and ventral hippoc
56 aversive unconditioned stimuli (USs) in the basolateral amygdala (BLA) and have examined their role
57 that NgR1 expression is required in both the basolateral amygdala (BLA) and infralimbic (IL) cortex t
59 itioned fear requires neural activity in the basolateral amygdala (BLA) and medial prefrontal cortex
60 ated with distinct oscillatory states in the basolateral amygdala (BLA) and medial prefrontal cortex
61 ation of specific brain regions, such as the basolateral amygdala (BLA) and nucleus accumbens (NAc),
63 ered activity in brain systems including the basolateral amygdala (BLA) and orbitofrontal cortex (OFC
64 , we investigate causal contributions of the basolateral amygdala (BLA) and orbitofrontal cortex (OFC
65 agonistic relationship may exist between the basolateral amygdala (BLA) and PFC during emotional proc
66 ions directly stimulate GIRK currents in the basolateral amygdala (BLA) and potentiate baclofen-induc
67 c properties of ventral hippocampus (vHipp), basolateral amygdala (BLA) and prefrontal cortex (PFC) i
68 ctions from the ventral hippocampus (vHipp,) basolateral amygdala (BLA) and prefrontal cortex (PFC) o
69 mbic (IL) cortex neurons that project to the basolateral amygdala (BLA) and reduced CD response compa
70 gion of the dorsal hippocampus (CA1) and the basolateral amygdala (BLA) and that of dopaminergic, nor
72 nderlie learning and memory deficits via the basolateral amygdala (BLA) and the hippocampus; however,
73 epend on functional interactions between the basolateral amygdala (BLA) and the nucleus accumbens cor
74 in the higher-order auditory cortex Te2 and basolateral amygdala (BLA) and their crosstalk during th
75 e of the medial prefrontal cortex (mPFC) and basolateral amygdala (BLA) and their reciprocal inhibito
78 (vHPC), medial prefrontal cortex (mPFC), and basolateral amygdala (BLA) are each required for the exp
79 Reciprocal circuits between the mPFC and basolateral amygdala (BLA) are particularly important fo
80 depression (LTD) in principal neurons of the basolateral amygdala (BLA) are thought to underlie the a
81 eir synaptic connections with neurons in the basolateral amygdala (BLA) at neonatal to adult developm
82 deling of relative excitatory drive from the basolateral amygdala (BLA) away from corticotropin relea
83 had stronger functional connectivity of the basolateral amygdala (BLA) complex with the pregenual an
87 lcohol consumption, whereas knockdown in the basolateral amygdala (BLA) decreased alcohol consumption
88 glutamatergic terminals from the PeF to the basolateral amygdala (BLA) enhanced the acquisition, del
89 a dampening of neuronal excitability in the basolateral amygdala (BLA) ex vivo and in vivo, and were
91 by miR-19b, Adrb1 levels were reduced in the basolateral amygdala (BLA) following chronic stress.
93 pharmacological manipulations, we found that basolateral amygdala (BLA) glutamatergic activity tracks
94 imbic medial prefrontal cortex (PL-mPFC) and basolateral amygdala (BLA) have been implicated in the e
95 By contrast, inhibitory Gi-DREADDs in the basolateral amygdala (BLA) impaired the acquisition of b
96 rtance of the orbitofrontal cortex (OFC) and basolateral amygdala (BLA) in acquisition of the rGT and
97 udies implicate cholinergic signaling in the basolateral amygdala (BLA) in behaviors related to stres
98 the FAAH inhibitor URB597, directly into the basolateral amygdala (BLA) in conjunction with memory re
99 we identified a role for miR-34a within the basolateral amygdala (BLA) in fear memory consolidation.
100 young and aged rats to test the role of the basolateral amygdala (BLA) in intertemporal decision mak
101 nd (3) excitatory synaptic physiology in the basolateral amygdala (BLA) in male Sprague Dawley rats.
102 n between the roles of the central (CeA) and basolateral amygdala (BLA) in regulating social behavior
103 r insular cortex (aIC) that project into the basolateral amygdala (BLA) in response to conditioned ta
104 earch has established a crucial role for the basolateral amygdala (BLA) in social experiential learni
105 neurons of mice integrate auditory cues and basolateral amygdala (BLA) inputs non-linearly in a NMDA
107 is structure, and the insular cortex and the basolateral amygdala (BLA) interact during CTA formation
112 s of E2 signaling.SIGNIFICANCE STATEMENT The basolateral amygdala (BLA) is a key structure of the fea
121 formed and stored in a genetically distinct basolateral amygdala (BLA) neuronal population that driv
122 dator) induces long-lasting sensitization of basolateral amygdala (BLA) noradrenergic (NE) receptors
123 either central nucleus of amygdala (CeA) or basolateral amygdala (BLA) of female rats with one parti
124 ve onto excitatory projection neurons in the basolateral amygdala (BLA) of juvenile Fmr1(-/y) knockou
126 by specifically overexpressing FKBP51 in the basolateral amygdala (BLA) or central amygdala resulted
127 divergent downstream structures such as the basolateral amygdala (BLA) or nucleus accumbens (NAc).
128 memory but was ineffective when given in the basolateral amygdala (BLA) or the ventral medial prefron
129 ore, this effect was not mediated via the LC-basolateral amygdala (BLA) pathway, because BLA inactiva
130 and associated with anatomical variation in basolateral amygdala (BLA) perineuronal nets, which are
131 ow that neuropeptide-receptor systems in the basolateral amygdala (BLA) play an important role in the
138 The lateral orbitofrontal cortex (lOFC) and basolateral amygdala (BLA) promote cocaine-seeking behav
139 version of diazepam (CD) that was tested on basolateral amygdala (BLa) pyramidal cells in mouse brai
140 as negatively related to both aggression and basolateral amygdala (BLA) reactivity to angry faces, wh
142 ions between the prefrontal cortex (PFC) and basolateral amygdala (BLA) regulate emotional behaviors.
149 that an increase in calcineurin (CaN) in the basolateral amygdala (BLA) supports the shift from fear
150 tified distinct neuronal circuits within the basolateral amygdala (BLA) that differentially mediate f
151 Y1R+ neurons projecting from the mPFC to the basolateral amygdala (BLA) this efferent population was
152 f the medial prefrontal cortex (mPFC) or the basolateral amygdala (BLA) to examine the effects of bet
153 supports unidirectional gustatory input from basolateral amygdala (BLA) to hypothalamus in sated subj
154 AMPAR)-silent excitatory synapses within the basolateral amygdala (BLA) to nucleus accumbens (NAc) pr
155 were analyzed with anterograde tracing from basolateral amygdala (BLA) to PFC to identify sex-specif
156 g factor (CRF) modulate the responses of the basolateral amygdala (BLA) to stress and are associated
157 t involve activation of projections from the basolateral amygdala (BLA) to the medial prefrontal cort
158 s coeruleus (LC) neurons that project to the basolateral amygdala (BLA) using a DREADD (designer rece
160 cannabinoid 1 receptor (Cnr1) and CCK in the basolateral amygdala (BLA), a brain region critical for
161 the BLA evokes norepinephrine release in the basolateral amygdala (BLA), alters BLA neuronal activity
162 fy the roles of the dorsal hippocampus (DH), basolateral amygdala (BLA), and medial prefrontal cortex
163 r learning, disengaged mesolimbic circuitry, basolateral amygdala (BLA), and plasticity-related AMPA
164 ess effects on the dorsal hippocampus (HPC), basolateral amygdala (BLA), and somatosensory cortex (SS
165 This avoidance requires prelimbic (PL) PFC, basolateral amygdala (BLA), and ventral striatum (VS).
166 ginate from medial prefrontal cortex (mPFC), basolateral amygdala (BLA), and ventral subiculum of the
167 ehavior, with a focus on the NACc and on the basolateral amygdala (BLA), another important locus for
168 onmuscle myosin II inhibition (NMIIi) in the basolateral amygdala (BLA), but not dorsal hippocampus (
169 ommunication between the hippocampus and the basolateral amygdala (BLA), but the mechanisms of this p
170 ties in the medial prefrontal cortex (mPFC), basolateral amygdala (BLA), dorsomedial striatum (DMS) a
171 s, including those examining the role of the basolateral amygdala (BLA), have traditionally used tech
174 excitatory input from pyramidal cells of the basolateral amygdala (BLA), neurons that are activated b
175 ess in BLA.SIGNIFICANCE STATEMENT Within the basolateral amygdala (BLA), neuropeptide Y (NPY) is asso
176 ue-guided alcohol seeking is mediated by the basolateral amygdala (BLA), nucleus accumbens (NAc), and
177 omous receptor of neuregulin 1 (NRG1) in the basolateral amygdala (BLA), was expressed in GABAergic n
178 ctivation in the prefrontal cortex (PFC) and basolateral amygdala (BLA), we examined electron microsc
179 we visualized robust c-fos expression in the basolateral amygdala (BLA), which was reduced in mice pr
180 ronal activity in one key limbic region, the basolateral amygdala (BLA), whose activity fluctuates ac
205 ronic stress contributes to hyperactivity of basolateral amygdala (BLA; comprised of basal, lateral,
206 e, selectively control PCs projecting to the basolateral amygdala (BLAPC) compared to those projectin
207 n at inputs from the ventral hippocampus and basolateral amygdala but not from the mediodorsal nucleu
210 xpression in the infralimbic cortex (IL) and basolateral amygdala complex (BLA) that were associated
212 ck requires de novo protein synthesis in the basolateral amygdala complex (BLA), whereas consolidatio
214 y enhanced memory and heightened cFos in the basolateral amygdala complex (BLC) with retrieval of the
216 gests that these changes limited only to the basolateral amygdala complex may not be sufficient to in
217 , including the lateral nucleus (LAT) of the basolateral amygdala complex, is sensitive to stress.
218 entromedial prefrontal cortex or hippocampal-basolateral amygdala connectivity-differentiated between
219 ur results indicate that anterior insula and basolateral amygdala constitute a network part that is p
220 on how distinct interneuron subtypes in the basolateral amygdala contribute to the acquisition and e
221 ion in the nucleus accumbens and central and basolateral amygdala correlated with genotype-related di
223 uding the locus coeruleus, and much later in basolateral amygdala, dorsal raphe nucleus, and the subs
224 intracellular signaling pathways within the basolateral amygdala during stress-induced reinstatement
225 insic functional connectivity, with the left basolateral amygdala emerging as the strongest predictor
226 show that increasing neural activity in the basolateral amygdala enhances both conditioned anticipat
229 ctify this, we monitored, in near-real time, basolateral amygdala glutamate concentration changes dur
230 e we combined chemogenetic excitation of rat basolateral amygdala glutamatergic neurons with a variet
231 ndamide and CRF1 signaling exhibited blunted basolateral amygdala habituation, which further mediated
233 egions, namely the medial prefrontal cortex, basolateral amygdala, hippocampus, anterior cingulate co
234 sory cortex, and increased activation of the basolateral amygdala in response to repeated whisker sti
237 anyl induced similar oxygen decreases in the basolateral amygdala, indicating that brain hypoxia coul
239 and medial prefrontal cortices as well as at basolateral amygdala inputs and striatal cholinergic int
240 ation were specific to MD inputs; activating basolateral amygdala inputs produced opposite effects.
242 tral amygdala, but MJN110 infusions into the basolateral amygdala, interfered with the naloxone-preci
243 nduction of beta-adrenergic signaling in the basolateral amygdala is sufficient to induce acute and s
245 Activation of noradrenergic circuitry in the basolateral amygdala is thought to have a role in stress
248 ate gyrus, we find that knockdown of PTEN in basolateral amygdala leads to a significant decrease in
249 D1 neuron projections implicates the medial basolateral amygdala (mBLA) as a downstream target of th
250 unction of the ventral striatum, whereas the basolateral amygdala mediates processing of the threat c
252 fect of acute stress occurred independent of basolateral amygdala neural input and was mimicked by tr
253 ed the effects of shRNA knockdown of PTEN in basolateral amygdala neurons on synaptic spine density a
254 revealed that these arrests are effected by basolateral amygdala neurons projecting to the central a
255 nsic excitability and synaptic plasticity in basolateral amygdala neurons that give rise to temporall
257 re) produces a long-lasting sensitization of basolateral amygdala noradrenergic substrates [via a cor
258 d that in vivo optogenetic activation of the basolateral amygdala-nucleus accumbens (BLA-NAc) glutama
260 In contrast, FosB remained elevated in the basolateral amygdala of mice with resolved nociception a
261 was also seen in the infralimbic cortex and basolateral amygdala of stress-susceptible male mice aft
262 ), with little AT(2)R-eGFP expression in the basolateral amygdala or lateral division of the central
263 togenetically stimulated mPFC projections to basolateral amygdala or nucleus accumbens, two subcortic
267 umber of Fos-positive neurons in central and basolateral amygdala, orbitofrontal cortex, ventral and
269 s (NAc) shell from ventral subiculum (vSub), basolateral amygdala, paraventricular thalamus, and vent
270 mPFC projections to the NAcS, but not to the basolateral amygdala, partially reversed suppression of
272 complex, and local plasticity in excitatory basolateral amygdala principal neurons is considered to
273 nstrate that C1ql3-expressing neurons in the basolateral amygdala project to the medial prefrontal co
278 entation of anandamide hydrolysis within the basolateral amygdala reduces behavioral indices of stres
281 idate, miR-144-3p, robustly expressed in the basolateral amygdala, showed specific extinction-induced
282 sory cortex, and increased activation of the basolateral amygdala, suggesting that impaired somatosen
283 identified a distinct neural ensemble in the basolateral amygdala that encodes the negative affective
284 projection-defined neuronal ensemble in the basolateral amygdala that is active during self-paced be
285 malian brain, such as the hippocampus or the basolateral amygdala, the clustering of the scaffolding
286 ns of a protein synthesis inhibitor into the basolateral amygdala, the subsequent fear response was a
289 ogical investigation of projections from the basolateral amygdala to the medial prefrontal cortex and
290 absent or labile, in the projection from the basolateral amygdala to the NAc in incubation of cocaine
291 olateral prefrontal cortex, hippocampus, and basolateral amygdala) to identify imaging predictors of
293 , we report that transient inhibition of the basolateral amygdala triggered a profound increase in so
294 l, and lateral shell; anterior and posterior basolateral amygdala; ventral pallidum; and periaqueduct
295 strocytes depressed excitatory synapses from basolateral amygdala via A1 adenosine receptor activatio
296 sing paired recordings obtained in the mouse basolateral amygdala, we found that AACs powerfully inhi
297 cortex, but not to the infralimbic cortex or basolateral amygdala, were more active to safety and com
298 trengthened the coupling between the cmA and basolateral amygdala, whereas LZP increased the interpla
299 ch as the nucleus accumbens shell (NAcS) and basolateral amygdala, which encode positive and negative
300 became silent with time, engram cells in the basolateral amygdala, which were necessary for fear memo