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1 specially children whose mothers continue to be depressed.
2 d less belonging support were more likely to be depressed.
3 three times the odds that a nonsmoker would be depressed.
4 are, if anything, more likely than girls to be depressed.
5 puts with high release probability tended to be depressed.
6 GAP knockout mice, whereas P38 MAPK function is depressed.
7 F cells discharge and sensory responsiveness is depressed.
8 s the input to the reward-related structures is depressed.
9 c firing rate information before the synapse is depressed.
10 eart failure, especially when renal function is depressed.
11 to the high-copper-mobility superionic phase is depressed.
12 of interventions for youth with diabetes who are depressed.
13 terviews would help to identify patients who are depressed.
14 by NMDA receptors that include NR2A subunits are depressed.
15 tured RPE, the mitotic activity of the cells was depressed.
16 and brachial artery flow-mediated dilatation was depressed.
17 ncreased with Ni exposure and calcium influx was depressed.
18 or shock whether or not ventricular function was depressed.
19 ence of calsyntenin-1, synaptic transmission was depressed.
20 nts were anxious and stressed than when they were depressed.
21 ion, sarcoplasmic reticulum (SR) Ca2+ stores were depressed.
22 intracellularly, inspiratory drive currents were depressed.
23 low: 11.9% had heightened anxiety, and 9.8% were depressed.
24 led receptors into the postsynaptic membrane were depressed.
25 was accelerated, whereas HF ventricular CaTs were depressed.
26 f sickness per depressed worker per month of being depressed.
27 did not see the infant were least likely to be depressed (13 of 33, 39%, vs three of 14, 21%, vs one
28 e that senescent cardiac angiogenic activity was depressed (2 of 17 allografts were viable in 18-mont
30 ,S)-SAM to ACC and methylthioadenosine (MTA) are depressed 630- and 25-fold for the E47Q and E47D enz
35 by activation of CRF1 receptors with CRF but was depressed after activation of CRF2 receptors with Uc
36 e excitatory neurotransmission in LA neurons was depressed after ischemia via presynaptic mechanisms.
38 both resting-state field potentials and CBF were depressed after cocaine administration (19.8+/-4.7%
40 ll unclear because some DA neurons appear to be depressed and others excited by aversive stimuli.
41 fferentiation, because synaptic transmission is depressed and fails entirely, in a cyclical manner, a
42 at the contractility of failing human hearts is depressed and that abnormalities of basal Ca2+ regula
43 Basal left ventricular (LV) contractility is depressed and the force-frequency relationship is blu
46 Contractile function of isolated myocytes was depressed and preceded left ventricular dilation and
47 ion closure, peak oxygen consumption (VO(2)) was depressed and there was systemic desaturation at res
49 ts remained significant both in patients who were depressed and in those who were nondepressed at enr
50 nd motor output curves in small hand muscles were depressed and motor thresholds were elevated compar
53 ines in response to an inflammatory stimulus is depressed, and this may contribute to delayed recover
54 okine release from FLAP knockout macrophages was depressed, and their restricted ability to induce VS
56 timulation of L-type Ca(2+) channel currents was depressed approximately 75% in the G(alphaq) mice.
57 CD8(+)-T-cell response in the knockout mice was depressed, as determined by pCTL and IFN-gamma assay
59 4 %, Li 1.9 +/- 0.3 % in controls, P < 0.05) was depressed at 2 days by dorsal root section (Lc 4.8 +
60 xtracellular matrix immediately after injury was depressed at 20 and 50 mM peroxide, but by 12 h was
62 lthough the chemoreceptor reflex bradycardia was depressed at a low dose of ANGII (5 fmol), all highe
63 rotein kinase A (PKA) activity in ob/ob mice was depressed at baseline and correctable towards the ac
64 001), as LVES stress/LVES volume index ratio was depressed at baseline and following MV repair versus
66 ntractility fell significantly over time and was depressed at last follow-up in patients receiving mo
67 at frequencies below 10 kHz and above 20 kHz was depressed at low intensities, but was greatly enhanc
68 In both peas and soybeans, the oxygen uptake was depressed at low temperatures (<16 degrees C) and lo
72 idents with a GDS-15 score, 374 of 765 (49%) were depressed at baseline; 484 of 765 (63%) provided 12
73 d b-wave amplitudes of the electroretinogram were depressed at the first postoperative week testing b
74 ording to the calculations, the pK(a) values are depressed because of unfavorable self-energies and r
76 (P < 0.05), and blood glucose concentrations were depressed below the control during recovery (P < 0.
77 mong the major side effects of this strategy are depressed beneficial immunity and increased rates of
79 , and physical impairment are more likely to be depressed but may be less able to seek appropriate ca
80 onosynaptic potentials in Renshaw cells that are depressed, but not abolished, by cholinergic antagon
82 cardiomyocyte mechanics and calcium kinetics were depressed, but isoproterenol reversed the inhibitor
83 time, constrictions to angiotensin I and II were depressed, but there was no loss of NTG vasodilator
84 tes of POPC; lipids in the surrounding shell are depressed by 0.5-0.8 A, but otherwise the phosphoino
91 vesicular refilling with acetylcholine (ACh) is depressed by inhibitors, two sizes of quanta should b
93 that of pathway refinement and LTD magnitude is depressed by nitrendipine, an L-type Ca(2+) channel b
94 oprotein P in human plasma has shown that it is depressed by selenium deficiency and by cirrhosis.
95 ndependent recombination in Escherichia coli is depressed by the redundant action of single-strand ex
96 promise: left ventricular developed pressure was depressed by 20%, and cardiac phosphocreatine was de
97 sarcomere-length shortening (nanometers, nm) was depressed by 42% (151.0+/-10.7 [control] versus 87.0
99 state level of TPP+ accumulation in the lens was depressed by 90% when the lenses were cultured in a
107 vator and plasminogen activator inhibitor-1, was depressed by exposure to high glucose, with the redu
108 ly when the excitatory synaptic transmission was depressed by further lowering [Ca(2+)](o) with EGTA.
109 r for PGI(2), but not for other prostanoids, was depressed by HSD in WT and even more so in both muta
111 f the GHR, JAK2, and STAT5 in response to GH was depressed by nearly half in CRF (P < 0.05), and nucl
114 naptic input from the somatosensory thalamus was depressed by sensory stimulation, and this depressio
116 response evoked by chemoreceptor stimulation was depressed by suramin, but convergent excitatory inpu
120 measure of excitation-contraction coupling, were depressed by 50% in both the inflow and outflow por
121 (P < 0.05), and nuclear phospho-STAT5 levels were depressed by approximately one third (P < 0.01).
122 ) and pentobarbitone sodium (41 %, 1:1), and were depressed by bicuculline (-41 +/- 5.7 %, 5:8) and p
125 atory synapses onto hippocampal interneurons were depressed by either capsaicin, a potent TRPV1 chann
128 mission of MF origin, MF GABAergic responses were depressed by the activation of metabotropic glutama
129 rminals by endogenously released substances, were depressed by the GABAA receptor antagonist SR95531
130 erpolarizations evoked by cyclopiazonic acid were depressed by the gap junction inhibitor 2-aminoetho
131 tom third at age 9 years were more likely to be depressed (CIS-R) at age 18 years (adjusted odds rati
132 de evidence that net community calcification is depressed compared with values expected for pre-indus
133 ral blood mononuclear cells from TB patients was depressed, compared with that of healthy control sub
139 ediated by endothelium-derived nitric oxide) is depressed during the early, hyperdynamic stage of sep
140 mains controversial whether adrenal function is depressed during the late stage of polymicrobial seps
142 rising from a single cholinergic interneuron was depressed during repetitive 2 Hz firing, intrastriat
144 ison group was all mothers or only those who were depressed during pregnancy but not exposed to antid
148 epression, more frequently agreed that "if I were depressed, fellow medical students would respect my
149 VSCCs in spines of rat CA1 pyramidal neurons are depressed for at least 30 min after brief trains of
150 m flashes showed directly that synaptic gain was depressed for 100-200 ms following transmission of a
151 sured by the end-systolic stiffness constant was depressed from 3.66 +/- 0.20 to 2.65 +/- 0.12 (p < 0
152 actin sliding velocity of the mutant myosin were depressed >80% compared with wild-type myosin.
153 p had evidence of IHD; 10 of 15 patients who were depressed had 1 or more traditional IHD risk factor
154 dry conditions, at least when the population is depressed, has probably contributed to the persistenc
157 pid species containing hexadecatrienoic acid are depressed in all of the sfd ssi2 npr1 plants, we pro
158 undance and sarcoplasmic reticulum Ca uptake are depressed in both sarcomere mutation-positive and -n
161 the resultant neutrophil trafficking in vivo are depressed in G6PT-/- ascites during an inflammatory
163 ally deficient in the PGI2 receptor (IP) but are depressed in mice genetically deficient in the TxA2
165 ex of a mouse model of Alzheimer disease and are depressed in mouse brain cortex by deletion of p47(p
166 tuberculosis (MTB)-induced T cell responses are depressed in peripheral blood mononuclear cells of p
168 studies that, although calcification may not be depressed in all species, acidification will produce
169 escent conduct disorder, were more likely to be depressed in young adulthood compared to their counte
171 e hypotheses that striatal dopamine activity is depressed in ADHD and that this contributes to sympto
172 like growth factor 1 receptor (IGF1R), which is depressed in atrial tissue samples from patients with
173 ic neurons from Nckx4 knock-out animals, and is depressed in cells in which NCKX4 is overexpressed.
178 that are not affected by hypertrophy, and it is depressed in patients who have a mutation for hypertr
185 a(2+)-ATPase to phospholamban protein levels was depressed in 7-week AS compared with controls but no
192 splenic macrophage IL-6 productive capacity was depressed in cells from animals subjected to CL/ETX
194 Bradykinin, but not ACH-mediated vasomotion, was depressed in epicardial segments that constricted wi
197 ase/succinate dehydrogenase [COX/SDH]-ratio) was depressed in ILD (median = 0.10,) compared with cont
198 In contrast, in vivo LV systolic pressure was depressed in L-arginine treated versus untreated rat
199 percent decrease in flow induced by L-NMMA, was depressed in male compared with female patients (-17
200 or-mediated inhibitory postsynaptic currents was depressed in medium spiny neurons of ethanol drinkin
201 Insulin receptor tyrosine kinase activity was depressed in muscle from obese insulin-resistant pat
202 The response to increased pacing frequency was depressed in NOS1(-/-) compared with wild type myocy
203 the muscle protein fractional synthetic rate was depressed in patients on day 1 (0.035%/hour; 95% CI,
204 e intact experiments, the slope of the PRSWR was depressed in the cardioplegia group compared with ba
205 esults indicate that hepatocellular function was depressed in the early stage of sepsis (i.e., 5 hrs
206 on of vascular cell adhesion molecule-1 mRNA was depressed in the IL-1R1 null mice, and corresponding
213 ols; however, erbB2 and erbB4 protein levels were depressed in 22-week aortic stenosis animals (48% d
214 in contrast, erbB2 and erbB4 message levels were depressed in 22-week aortic stenosis animals at the
216 mmunoglobulin G against C. difficile toxin A were depressed in aged mice, and vancomycin treatment re
217 ker for the high-affinity choline transport, were depressed in aged rats and GM1 corrected the defici
218 of this synchronously regulated gene cluster were depressed in both ventral and dorsal horn neurons w
219 ron, IL-12, and tumor necrosis factor alpha) were depressed in cells from MAM-injected wild-type C3H/
225 rcoplasmic reticulum Ca2+-ATPase mRNA levels were depressed in male LVH but not in female LVH compare
226 viral and intracellular bacterial pathogens were depressed in mice in vivo and in humans to phorbol
232 ctional shortening and peak systolic [Ca2+]i were depressed in the MI compared with the control group
233 as by the ecto-ATPase inhibitor ARL67156 and were depressed in the presence of P2 receptor antagonist
235 diated slow inhibitory postsynaptic currents were depressed in wt but not betaarr-2 ko LC neurons aft
236 tension is pronounced during shortening, but is depressed/inhibited during lengthening, is consistent
237 dparent caregivers are more likely to report being depressed, it is crucial to identify whether poor
238 temperature of the chain melting transition is depressed less for t18:1-18:0PC (31.5 degrees C) than
239 asal excitatory synaptic transmission in CA1 was depressed, long-term potentiation (LTP) was enhanced
242 tocol that can be effective for patients who are depressed, markedly suppresses synaptic drive onto V
243 ich apolipoprotein B-containing lipoproteins were depressed may be via alterations in LDL receptor qu
245 In CLL specimens, E-cadherin expression was depressed more than 50% in 62% cases (relative to no
246 at 12 hours reperfusion wall thickening (WT) was depressed more, P<0.05, in CD (-46+/-5%) as compared
250 vivors who were disabled were more likely to be depressed (odds ratio 1.35 per activities of daily li
253 ilure (CHF) doubles either when CHF patients are depressed or when their plasma norepinephrine (NE) l
256 d in C3H/HeJ cell supernatants, whereas they were depressed or remained low in BALB/c cell supernatan
260 rall response rates in core-lesioned animals were depressed relative to that in the shell- or sham-le
261 tation in response to repetitive activation, are depressed significantly by baclofen, and do not expr
263 taining synthetic peptide, contractile force was depressed significantly by, 28% at 4 Hz, 37.7% at 5
265 rs (ie, V(max), CT, DTE, and S(1)-S(1)) also were depressed significantly by vernakalant and ranolazi
267 Populations of channels in single spines are depressed stochastically and synchronously, independ
269 authors studied 49 young people who had not been depressed themselves but who had a parent with a hi
271 vels of PIF1- and PIF2-deficient ODV binding were depressed threefold compared to control levels.
273 greatly impaired in factor VIII deficiency, being depressed to less than 1/29 (<1.9 nmol TAT/L/min)
275 eart allografts 5 days after transplantation is depressed to 40% of that of syngenic controls, and th
281 nucleotide exchange and the level of Ras-GTP were depressed to near basal values on the time scale of
282 eric myosin exhibited ATPase activities that were depressed to the levels observed in hearts expressi
284 n the floccular complex of the cerebellum to be depressed transiently at the time of the instruction
288 1992, however, the odds that a smoker would be depressed were three times the odds that a nonsmoker
289 nts joining after randomisation, the odds of being depressed were 0.76 (95% CI 0.53 to 1.09) for the
290 hic measures whereas their systolic function is depressed when measured by the two-dimensional echoca
292 , and metal assimilation efficiency (for Zn) was depressed, while the metal efflux rate (for Zn) was
293 nses to beta-adrenergic receptor stimulation are depressed with pressure overload-induced cardiac hyp
294 tractile reserve in response to rapid pacing was depressed with impaired augmentation of both peak-sy
296 direction (GFAP and NF-kappaB), while others were depressed with age (PDI and intensity of Nissl stai
297 s: when one input was potentiated, the other was depressed, with the direction and magnitude of chang