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1 specially children whose mothers continue to be depressed.
2 d less belonging support were more likely to be depressed.
3  three times the odds that a nonsmoker would be depressed.
4  are, if anything, more likely than girls to be depressed.
5 puts with high release probability tended to be depressed.
6 GAP knockout mice, whereas P38 MAPK function is depressed.
7 F cells discharge and sensory responsiveness is depressed.
8 s the input to the reward-related structures is depressed.
9 c firing rate information before the synapse is depressed.
10 eart failure, especially when renal function is depressed.
11 to the high-copper-mobility superionic phase is depressed.
12 of interventions for youth with diabetes who are depressed.
13 terviews would help to identify patients who are depressed.
14 by NMDA receptors that include NR2A subunits are depressed.
15 tured RPE, the mitotic activity of the cells was depressed.
16 and brachial artery flow-mediated dilatation was depressed.
17 ncreased with Ni exposure and calcium influx was depressed.
18 or shock whether or not ventricular function was depressed.
19 ence of calsyntenin-1, synaptic transmission was depressed.
20 nts were anxious and stressed than when they were depressed.
21 ion, sarcoplasmic reticulum (SR) Ca2+ stores were depressed.
22  intracellularly, inspiratory drive currents were depressed.
23  low: 11.9% had heightened anxiety, and 9.8% were depressed.
24 led receptors into the postsynaptic membrane were depressed.
25 was accelerated, whereas HF ventricular CaTs were depressed.
26 f sickness per depressed worker per month of being depressed.
27  did not see the infant were least likely to be depressed (13 of 33, 39%, vs three of 14, 21%, vs one
28 e that senescent cardiac angiogenic activity was depressed (2 of 17 allografts were viable in 18-mont
29            Contractile reserve to dobutamine was depressed (62.3 +/- 0.9 FA versus 49.2 +/- 1.5 PM, F
30 ,S)-SAM to ACC and methylthioadenosine (MTA) are depressed 630- and 25-fold for the E47Q and E47D enz
31 , especially at night when net calcification was depressed ~78% compared to ambient pCO2 .
32 lated by exogenous application of 5-HT: they are depressed after 5-HT exposure.
33           LV diastolic and systolic function are depressed after TOF repair.
34 AM), a newly discovered vasodilator peptide, is depressed after hemorrhage and resuscitation.
35 by activation of CRF1 receptors with CRF but was depressed after activation of CRF2 receptors with Uc
36 e excitatory neurotransmission in LA neurons was depressed after ischemia via presynaptic mechanisms.
37 s not involved, because AMPAR-mediated EPSCs were depressed after a train.
38  both resting-state field potentials and CBF were depressed after cocaine administration (19.8+/-4.7%
39               At rest, the second H-reflexes were depressed an average of 73% relative to the first.
40 ll unclear because some DA neurons appear to be depressed and others excited by aversive stimuli.
41 fferentiation, because synaptic transmission is depressed and fails entirely, in a cyclical manner, a
42 at the contractility of failing human hearts is depressed and that abnormalities of basal Ca2+ regula
43    Basal left ventricular (LV) contractility is depressed and the force-frequency relationship is blu
44 , synaptic transmission in autonomic ganglia is depressed and the periphery becomes deregulated.
45  were placed under anoxia, protein synthesis was depressed and no "new" proteins were detected.
46    Contractile function of isolated myocytes was depressed and preceded left ventricular dilation and
47 ion closure, peak oxygen consumption (VO(2)) was depressed and there was systemic desaturation at res
48 cyclooxygenase-2 (Cox-2) and IL-6 expression were depressed and delayed.
49 ts remained significant both in patients who were depressed and in those who were nondepressed at enr
50 nd motor output curves in small hand muscles were depressed and motor thresholds were elevated compar
51                 Calcium transient amplitudes were depressed and the decay was slowed in DHF, especial
52 e work performance among service workers who were depressed and those who were not depressed.
53 ines in response to an inflammatory stimulus is depressed, and this may contribute to delayed recover
54 okine release from FLAP knockout macrophages was depressed, and their restricted ability to induce VS
55                    The steady active tension was depressed approximately 45 % with 25 mM added P(i).
56 timulation of L-type Ca(2+) channel currents was depressed approximately 75% in the G(alphaq) mice.
57  CD8(+)-T-cell response in the knockout mice was depressed, as determined by pCTL and IFN-gamma assay
58 ress disorder were 3.93 times more likely to be depressed at 7 months.
59 4 %, Li 1.9 +/- 0.3 % in controls, P < 0.05) was depressed at 2 days by dorsal root section (Lc 4.8 +
60 xtracellular matrix immediately after injury was depressed at 20 and 50 mM peroxide, but by 12 h was
61 y was elevated and the L-type Ca(2+) channel was depressed at 8 months only.
62 lthough the chemoreceptor reflex bradycardia was depressed at a low dose of ANGII (5 fmol), all highe
63 rotein kinase A (PKA) activity in ob/ob mice was depressed at baseline and correctable towards the ac
64 001), as LVES stress/LVES volume index ratio was depressed at baseline and following MV repair versus
65 ncy excitation of BA interneurons (INs) that was depressed at higher frequencies.
66 ntractility fell significantly over time and was depressed at last follow-up in patients receiving mo
67 at frequencies below 10 kHz and above 20 kHz was depressed at low intensities, but was greatly enhanc
68 In both peas and soybeans, the oxygen uptake was depressed at low temperatures (<16 degrees C) and lo
69                                    Those who were depressed at 13 months were 7.96 times more likely
70                         Similarly, those who were depressed at 7 months were 9.5 times more likely to
71                       Among 354 patients, 68 were depressed at baseline.
72 idents with a GDS-15 score, 374 of 765 (49%) were depressed at baseline; 484 of 765 (63%) provided 12
73 d b-wave amplitudes of the electroretinogram were depressed at the first postoperative week testing b
74 ording to the calculations, the pK(a) values are depressed because of unfavorable self-energies and r
75                            The cardiac index was depressed below 3.0 L/min/m2 in 65 of 98 patients.
76 (P < 0.05), and blood glucose concentrations were depressed below the control during recovery (P < 0.
77 mong the major side effects of this strategy are depressed beneficial immunity and increased rates of
78              After CFPI alone CA1 ATP levels were depressed bilaterally only at 2 h (p < 0.05).
79 , and physical impairment are more likely to be depressed but may be less able to seek appropriate ca
80 onosynaptic potentials in Renshaw cells that are depressed, but not abolished, by cholinergic antagon
81                 Resting ventricular function was depressed, but responsiveness to beta-agonist was no
82 cardiomyocyte mechanics and calcium kinetics were depressed, but isoproterenol reversed the inhibitor
83  time, constrictions to angiotensin I and II were depressed, but there was no loss of NTG vasodilator
84 tes of POPC; lipids in the surrounding shell are depressed by 0.5-0.8 A, but otherwise the phosphoino
85                       Acetylcholine synapses are depressed by high-frequency stimulation, in part due
86             We found that hypocretin neurons are depressed by opiates, drugs of abuse that reduce cog
87 er serious adverse events, and tended to not be depressed by the end of treatment.
88 atalytic activity of K165M and K165C mutants is depressed by >10(3)-fold.
89 he Curie temperature, based on Arrott plots, is depressed by 1-2 K from the bulk value.
90                             Hypothalamic TRH is depressed by at least 75% compared with wild-type con
91 vesicular refilling with acetylcholine (ACh) is depressed by inhibitors, two sizes of quanta should b
92         High-frequency synaptic transmission is depressed by moderate rises in the extracellular pota
93 that of pathway refinement and LTD magnitude is depressed by nitrendipine, an L-type Ca(2+) channel b
94 oprotein P in human plasma has shown that it is depressed by selenium deficiency and by cirrhosis.
95 ndependent recombination in Escherichia coli is depressed by the redundant action of single-strand ex
96 promise: left ventricular developed pressure was depressed by 20%, and cardiac phosphocreatine was de
97 sarcomere-length shortening (nanometers, nm) was depressed by 42% (151.0+/-10.7 [control] versus 87.0
98             The amplitude of the second EPSC was depressed by 60% at 10 msec and recovered with tau a
99 state level of TPP+ accumulation in the lens was depressed by 90% when the lenses were cultured in a
100             The SR Ca(2+) fractional release was depressed by Ac-10C at relatively low SR Ca(2+) cont
101                                In SC-CA1, it was depressed by agonists to levels below control, where
102 r than those for wild-type, whereas the kcat was depressed by an order of magnitude.
103 ia except in the absence of glucose, when it was depressed by approximately 50 %.
104 terleukin-6 by Mphi from ovariectomized mice was depressed by approximately 50%.
105  failed to bind FGF-2 unless heparan sulfate was depressed by chlorate or heparinase treatment.
106           It was not affected by hypoxia but was depressed by cobalt.
107 vator and plasminogen activator inhibitor-1, was depressed by exposure to high glucose, with the redu
108 ly when the excitatory synaptic transmission was depressed by further lowering [Ca(2+)](o) with EGTA.
109 r for PGI(2), but not for other prostanoids, was depressed by HSD in WT and even more so in both muta
110 ,5-tetranor-prostane-1,20-dioic acid (PGE-M) was depressed by mPGES-1 deletion.
111 f the GHR, JAK2, and STAT5 in response to GH was depressed by nearly half in CRF (P < 0.05), and nucl
112                                     Since it was depressed by prior incorporation of the Ca(2+) buffe
113          Depolarisation-induced constriction was depressed by rotenone during hypoxia by 23 +/- 4 %;
114 naptic input from the somatosensory thalamus was depressed by sensory stimulation, and this depressio
115           Left ventricular ejection fraction was depressed by septic shock (p = .01) and further decr
116 response evoked by chemoreceptor stimulation was depressed by suramin, but convergent excitatory inpu
117                   PKCdelta specific activity was depressed by the addition of VEGF (by 41+/-8% [P<0.0
118 dinor 6-keto PGF(1alpha) (PGI-M), only PGI-M was depressed by the COX-2 inhibitor.
119           FS to pyramidal cell unitary IPSCs were depressed by (RS)-S-amino-3-(3-hydroxy-5-tert-butyl
120  measure of excitation-contraction coupling, were depressed by 50% in both the inflow and outflow por
121 (P < 0.05), and nuclear phospho-STAT5 levels were depressed by approximately one third (P < 0.01).
122 ) and pentobarbitone sodium (41 %, 1:1), and were depressed by bicuculline (-41 +/- 5.7 %, 5:8) and p
123  elevation of Ca(2+) and release of proteins were depressed by cytokines.
124 d by D1LRs whereas pallidal inputs to the EP were depressed by D2LRs.
125 atory synapses onto hippocampal interneurons were depressed by either capsaicin, a potent TRPV1 chann
126 ither release of Ca(2+) from stores nor SOCI were depressed by high glucose levels.
127                          When the discharges were depressed by severe hypoxia, exposure to light exci
128 mission of MF origin, MF GABAergic responses were depressed by the activation of metabotropic glutama
129 rminals by endogenously released substances, were depressed by the GABAA receptor antagonist SR95531
130 erpolarizations evoked by cyclopiazonic acid were depressed by the gap junction inhibitor 2-aminoetho
131 tom third at age 9 years were more likely to be depressed (CIS-R) at age 18 years (adjusted odds rati
132 de evidence that net community calcification is depressed compared with values expected for pre-indus
133 ral blood mononuclear cells from TB patients was depressed, compared with that of healthy control sub
134                        Synaptic transmission is depressed due to a reduction of cytoplasmic and readi
135 rictor effects of sympathetic nerve activity are depressed during hypoxia.
136                                    Women who are depressed during pregnancy have been found to have a
137 es and 13% of elderly beneficiaries reported being depressed during the previous year.
138 egulated via an AKT-dependent pathway, which is depressed during prolonged hypoxia.
139 ediated by endothelium-derived nitric oxide) is depressed during the early, hyperdynamic stage of sep
140 mains controversial whether adrenal function is depressed during the late stage of polymicrobial seps
141  torpid animals, indicating that translation is depressed during torpor.
142 rising from a single cholinergic interneuron was depressed during repetitive 2 Hz firing, intrastriat
143                           Twenty-one percent were depressed during at least 80% of the follow-up peri
144 ison group was all mothers or only those who were depressed during pregnancy but not exposed to antid
145 diabetes and 8.5% (n = 886) without diabetes were depressed during pregnancy or postpartum.
146 the AMR dynamics only when bacterial numbers were depressed during therapy.
147 rricanes from 1500 to 1670 CE, but frequency was depressed elsewhere in the Atlantic Ocean.
148 epression, more frequently agreed that "if I were depressed, fellow medical students would respect my
149 VSCCs in spines of rat CA1 pyramidal neurons are depressed for at least 30 min after brief trains of
150 m flashes showed directly that synaptic gain was depressed for 100-200 ms following transmission of a
151 sured by the end-systolic stiffness constant was depressed from 3.66 +/- 0.20 to 2.65 +/- 0.12 (p < 0
152  actin sliding velocity of the mutant myosin were depressed &gt;80% compared with wild-type myosin.
153 p had evidence of IHD; 10 of 15 patients who were depressed had 1 or more traditional IHD risk factor
154 dry conditions, at least when the population is depressed, has probably contributed to the persistenc
155                  Spouses were more likely to be depressed if they were older, less well educated, mor
156 oteins to maintain health, but its functions are depressed in aging and obesity.
157 pid species containing hexadecatrienoic acid are depressed in all of the sfd ssi2 npr1 plants, we pro
158 undance and sarcoplasmic reticulum Ca uptake are depressed in both sarcomere mutation-positive and -n
159                           Cells lacking Coa4 are depressed in CcO activity but show no impairment in
160           Although endothelial NOS responses are depressed in congestive heart failure (CHF), inducib
161 the resultant neutrophil trafficking in vivo are depressed in G6PT-/- ascites during an inflammatory
162                         Bradykinin responses are depressed in hypertension, and endothelial dysfuncti
163 ally deficient in the PGI2 receptor (IP) but are depressed in mice genetically deficient in the TxA2
164 s in the bone marrow, but not in the spleen, are depressed in mice treated with BQR.
165 ex of a mouse model of Alzheimer disease and are depressed in mouse brain cortex by deletion of p47(p
166  tuberculosis (MTB)-induced T cell responses are depressed in peripheral blood mononuclear cells of p
167         Resting energy expenditure (REE) can be depressed in affected individuals for undefined reaso
168 studies that, although calcification may not be depressed in all species, acidification will produce
169 escent conduct disorder, were more likely to be depressed in young adulthood compared to their counte
170                            Exercise capacity is depressed in ACHD patients (even in allegedly asympto
171 e hypotheses that striatal dopamine activity is depressed in ADHD and that this contributes to sympto
172 like growth factor 1 receptor (IGF1R), which is depressed in atrial tissue samples from patients with
173 ic neurons from Nckx4 knock-out animals, and is depressed in cells in which NCKX4 is overexpressed.
174               Because mitochondrial function is depressed in failing hearts and iron accumulation can
175 monstrate, for the first time, that RCAN1-1L is depressed in Huntington disease.
176           Because cytosolic Ca(2+) signaling is depressed in injured dorsal root ganglia (DRG) neuron
177        Calcium-dependent contractile reserve is depressed in myocytes from rats with postinfarction h
178 that are not affected by hypertrophy, and it is depressed in patients who have a mutation for hypertr
179 ity between a predictive stimulus and reward is depressed in proportion to predicted reward.
180 ed the hypothesis that RV functional reserve is depressed in SSc-PAH patients.
181                       RV contractile reserve is depressed in SSc-PAH versus IPAH subjects, associated
182 n 64.7 percent and left ventricular function was depressed in 26.0 percent.
183           Left ventricular systolic function was depressed in 30 patients (83%), with a median ejecti
184 ts (74.1%), while right ventricular function was depressed in 32 (59.3%).
185 a(2+)-ATPase to phospholamban protein levels was depressed in 7-week AS compared with controls but no
186 (i) increased in controls, but this response was depressed in 7-week AS.
187                                   Peak power was depressed in all fibre types with the greatest loss
188            CD4(+) T-cell expression of CD40L was depressed in B7KO mice in vivo and in vitro.
189                             LV contractility was depressed in both groups (for bolus patients, median
190                                        RV SE was depressed in both groups to a similar degree to that
191         Phosphorylation of the MPM-2 epitope was depressed in both X-ray and etoposide-treated cells,
192  splenic macrophage IL-6 productive capacity was depressed in cells from animals subjected to CL/ETX
193          We found that synaptic transmission was depressed in corticostriatal slices after perfusion
194 Bradykinin, but not ACH-mediated vasomotion, was depressed in epicardial segments that constricted wi
195                                IRES activity was depressed in H ferritin-deficient mouse embryonic fi
196                                At week 1, %S was depressed in HYPER, COMB, and HYPO (9+/-8%, 7+/-6%,
197 ase/succinate dehydrogenase [COX/SDH]-ratio) was depressed in ILD (median = 0.10,) compared with cont
198    In contrast, in vivo LV systolic pressure was depressed in L-arginine treated versus untreated rat
199  percent decrease in flow induced by L-NMMA, was depressed in male compared with female patients (-17
200 or-mediated inhibitory postsynaptic currents was depressed in medium spiny neurons of ethanol drinkin
201    Insulin receptor tyrosine kinase activity was depressed in muscle from obese insulin-resistant pat
202   The response to increased pacing frequency was depressed in NOS1(-/-) compared with wild type myocy
203 the muscle protein fractional synthetic rate was depressed in patients on day 1 (0.035%/hour; 95% CI,
204 e intact experiments, the slope of the PRSWR was depressed in the cardioplegia group compared with ba
205 esults indicate that hepatocellular function was depressed in the early stage of sepsis (i.e., 5 hrs
206 on of vascular cell adhesion molecule-1 mRNA was depressed in the IL-1R1 null mice, and corresponding
207 ereas the SITS-sensitive component of phi(E) was depressed in the low pH(i) range.
208                           Epimerase activity was depressed in the mutant, but increased upon restorat
209  role in the differentiation of osteoclasts, was depressed in these cells.
210 vation of B cell-specific gene transcription was depressed in these cells.
211 d myofilament-Ca(2)(+) responsiveness, which was depressed in untreated Tm-E54K mice.
212 -old mice although the responses to both Ags were depressed in 2-year-old mice.
213 ols; however, erbB2 and erbB4 protein levels were depressed in 22-week aortic stenosis animals (48% d
214  in contrast, erbB2 and erbB4 message levels were depressed in 22-week aortic stenosis animals at the
215           GABA concentration-response curves were depressed in a mixed/non-competitive fashion.
216 mmunoglobulin G against C. difficile toxin A were depressed in aged mice, and vancomycin treatment re
217 ker for the high-affinity choline transport, were depressed in aged rats and GM1 corrected the defici
218 of this synchronously regulated gene cluster were depressed in both ventral and dorsal horn neurons w
219 ron, IL-12, and tumor necrosis factor alpha) were depressed in cells from MAM-injected wild-type C3H/
220 aspase-3 activation and TUNEL-positive cells were depressed in COX-2(-/-) livers.
221 f breathing at rest and during gas exposures were depressed in EH but not HH.
222 ssure and left ventricular ejection fraction were depressed in GhOEs.
223                       Levels of TRH-[Gly(4)] were depressed in homozygotes.
224 sensitivity, and dynamic range of V2 neurons were depressed in infants compared with adults.
225 rcoplasmic reticulum Ca2+-ATPase mRNA levels were depressed in male LVH but not in female LVH compare
226  viral and intracellular bacterial pathogens were depressed in mice in vivo and in humans to phorbol
227             Finally, epicardial BK responses were depressed in patients with high ACE levels and in t
228       Mechanical bone properties of strength were depressed in Pb-exposed bones, but HFD had no signi
229 ed with increased expression of SERCA2a, and were depressed in Pim-1-deficient hearts.
230 ation trials revealed B. barbus growth rates were depressed in sympatry with L. idus.
231 hough transitional 2 (T2) B cell populations were depressed in the absence of Zfp318.
232 ctional shortening and peak systolic [Ca2+]i were depressed in the MI compared with the control group
233 as by the ecto-ATPase inhibitor ARL67156 and were depressed in the presence of P2 receptor antagonist
234                       SERCA2a protein levels were depressed in WT 7-week AS mice, but were preserved
235 diated slow inhibitory postsynaptic currents were depressed in wt but not betaarr-2 ko LC neurons aft
236 tension is pronounced during shortening, but is depressed/inhibited during lengthening, is consistent
237 dparent caregivers are more likely to report being depressed, it is crucial to identify whether poor
238  temperature of the chain melting transition is depressed less for t18:1-18:0PC (31.5 degrees C) than
239 asal excitatory synaptic transmission in CA1 was depressed, long-term potentiation (LTP) was enhanced
240 rastriatally evoked fast EPSCs in LA neurons were depressed &lt; or =24 hr after ischemia.
241  to model the probability that an individual was depressed, manic, or euthymic.
242 tocol that can be effective for patients who are depressed, markedly suppresses synaptic drive onto V
243 ich apolipoprotein B-containing lipoproteins were depressed may be via alterations in LDL receptor qu
244                            The positive-well-being, depressed-mood, and composite scores of the Psych
245      In CLL specimens, E-cadherin expression was depressed more than 50% in 62% cases (relative to no
246 at 12 hours reperfusion wall thickening (WT) was depressed more, P<0.05, in CD (-46+/-5%) as compared
247                                   CD4+ cells were depressed more than CD8+ cells.
248                         The 3 outcome states were depressed, nondepressed, and death, with scores of
249                          Cholinergic effects were depressed (not obliterated) by D-tubocurarine (D-TC
250 vivors who were disabled were more likely to be depressed (odds ratio 1.35 per activities of daily li
251 ely with iNOS inhibitors, pacemaker activity was depressed only at the anastomosis site.
252 lly predicted youth depression if the mother was depressed only once.
253 ilure (CHF) doubles either when CHF patients are depressed or when their plasma norepinephrine (NE) l
254 e victims of violence or victimization or to be depressed or suicidal.
255 sk of corneal graft rejection when IFN-gamma is depressed or absent.
256 d in C3H/HeJ cell supernatants, whereas they were depressed or remained low in BALB/c cell supernatan
257            In HF, dobutamine +dP/dt response was depressed (P<0.001 versus control), and increased co
258  patients undergoing major abdominal surgery are depressed preoperatively.
259           One defect of CD21/35(-/-) animals is depressed production of Ag-specific IgG3 which we sho
260 rall response rates in core-lesioned animals were depressed relative to that in the shell- or sham-le
261 tation in response to repetitive activation, are depressed significantly by baclofen, and do not expr
262          The baroreceptor reflex bradycardia was depressed significantly by NTS microinjections of AN
263 taining synthetic peptide, contractile force was depressed significantly by, 28% at 4 Hz, 37.7% at 5
264             Furthermore, cerebral metabolism was depressed significantly in a time-dependent manner w
265 rs (ie, V(max), CT, DTE, and S(1)-S(1)) also were depressed significantly by vernakalant and ranolazi
266             Calcineurin phosphatase activity was depressed similarly in both cyclosporine-treated gro
267     Populations of channels in single spines are depressed stochastically and synchronously, independ
268              Women were more likely (64%) to be depressed than men (44%).
269  authors studied 49 young people who had not been depressed themselves but who had a parent with a hi
270                                     When men are depressed they may experience symptoms that are diff
271 vels of PIF1- and PIF2-deficient ODV binding were depressed threefold compared to control levels.
272 ine-treated animals, putative BLA-mPFC EPSCs were depressed through a presynaptic mechanism.
273  greatly impaired in factor VIII deficiency, being depressed to less than 1/29 (<1.9 nmol TAT/L/min)
274 xidation of exogenous long-chain fatty acids is depressed to 30% wild-type levels.
275 eart allografts 5 days after transplantation is depressed to 40% of that of syngenic controls, and th
276                        The pKa value of K312 is depressed to 9.58, a value indicating that at physiol
277                              Testis selenium was depressed to 19% in mice fed an 0.1 mg selenium/kg d
278                               Brain selenium was depressed to 43%, but feeding 2 mg selenium/kg diet
279                                       Kidney was depressed to 76% and reached Sepp(+/+) levels on an
280                       Uptake of both tracers was depressed to a greater extent in areas of severe isc
281 nucleotide exchange and the level of Ras-GTP were depressed to near basal values on the time scale of
282 eric myosin exhibited ATPase activities that were depressed to the levels observed in hearts expressi
283 erminating a line of first-order transitions is depressed toward zero temperature.
284 n the floccular complex of the cerebellum to be depressed transiently at the time of the instruction
285                      EEG background activity is depressed transiently after moderate hypoxia-ischemia
286 the gene for inducible nitric oxide synthase was depressed transiently.
287                 Regional LAD wall thickening was depressed under baseline conditions (LAD 3.7+/-0.3 v
288  1992, however, the odds that a smoker would be depressed were three times the odds that a nonsmoker
289 nts joining after randomisation, the odds of being depressed were 0.76 (95% CI 0.53 to 1.09) for the
290 hic measures whereas their systolic function is depressed when measured by the two-dimensional echoca
291                                Urinary PGE-M was depressed, whereas urinary 2,3-dinor 6-keto PGF(1alp
292 , and metal assimilation efficiency (for Zn) was depressed, while the metal efflux rate (for Zn) was
293 nses to beta-adrenergic receptor stimulation are depressed with pressure overload-induced cardiac hyp
294 tractile reserve in response to rapid pacing was depressed with impaired augmentation of both peak-sy
295                                  The tension was depressed with increased [P(i)] in a concentration-d
296 direction (GFAP and NF-kappaB), while others were depressed with age (PDI and intensity of Nissl stai
297 s: when one input was potentiated, the other was depressed, with the direction and magnitude of chang
298                          Since recombination is depressed within genes and between distal regulatory
299                            Genetic diversity was depressed within esterase 5 within Sex-ratio chromos
300 and K(+)-induced increases in IPSC frequency were depressed without a change in IPSC amplitude.

 
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