ライフサイエンスコーパス: be explained by

1 The data are explained by a model in which the chromosomal struct

2 ing a simulation, we show that these results are explained by a simple model that combines among-indi

3 ized that these effects are linked and might be explained by a cisternal-specific delay in cargo tran

4 the concentration of damage near Compton can be explained by a combination of local site amplificatio

5 redundancy and herbivore specialization can be explained by a conflicting information transfer.

6 bon ages in Mediterranean-Anatolian wood can be explained by a divergence between high-resolution rad

7 istence of both in the same population might be explained by a dynamic evolutionary equilibrium that

8 Thus, KIF1A's activity can be explained by a fast rear-head detachment rate, a rate

9 We found that the dynamics of eS6-P can be explained by a feedforward circuit with inputs from b

10 ariance in cortex-wide activity (~75%) could be explained by a limited set of ~14 "motifs" of neural

11 Results are unlikely to be explained by a low quality of data arising from repea

12 ate to an active extended structure that can be explained by a modified Monod-Wyman-Changeux model.

13 t the representation of multiple stimuli can be explained by a normalization mechanism.

14 stant across chondrite groups, this can only be explained by a preferential sequestration of Nb relat

15 gh the large measured ejecta mass(3,4) could be explained by a progenitor system that is asymmetric i

16 at the evolution of many artefact traits can be explained by a shifting-optimum model of cultural sel

17 alytic behavior of both proteins that cannot be explained by a simple competition for POR.

18 ranscript levels in response to N-dose could be explained by a simple kinetic principle: the Michaeli

19 ing and memory; however, this process cannot be explained by a simple linear trajectory of transcript

20 Despite their diversity, these changes could be explained by a simple model where microstimulation ha

21 unfolding of each immunoglobulin domain can be explained by a simple two-state unfolding process, wi

22 asic dependence of Ca(2+) release on [P(i) ] is explained by a direct effect of P(i) acting on the SR

23 between DNA-binding proteins (DBPs) and DNA is explained by a facilitated diffusion mechanism, in wh

24 ta and suggested that podokinetic adaptation is explained by a short (141 s) and a long (27 min) time

25 dependent on membrane voltage and Ca(2+) and is explained by a stabilization of the ANO1 Ca(2+)-bound

26 ctacular example of 'epigenetic' inheritance is explained by a super-efficient maintenance enzyme plu

27 morphogenetic gradient" of molar proportions was explained by a balance between inhibiting/activating

28 ips (odds ratio = 0.8, 95% CI = 0.5-1.2) and was explained by a combination of differential fertility

29 that 45% of the variance in alpha-diversity was explained by a subset of 40 plasma metabolites (13 o

30 The enhanced stimulus specificity was explained by a surprising diversity.

31 fferences in functional responses to VNS can be explained by ABL of A- and B-fiber activation.

32 variability in posterior emissions estimates is explained by accounting for the sampling in time and

33 These disorders have generally been explained by accounts that focus on their behaviora

34 Progressive neurocognitive decline and death were explained by active granulomatous encephalitis, wit

35 heterogeneity of oil concentration, 89% can be explained by additional linked mean-effects genetic v

36 While some discordances can be explained by additional records of deportations withi

37 We demonstrate this trend cannot be explained by African admixture nor Neanderthal introg

38 d severe outcome among IMD-W cases could not be explained by age, gender, and comorbidities.

39 We reasoned that this could be explained by alterations in the consequences of chole

40 blood flow during hypoxia or hyperoxia could be explained by altered NO degradation in the parenchyma

41 served in several FLE conditions that cannot be explained by alternative (temporal) models of the FLE

42 red that the longest codon dwell times could be explained by aminoacylation levels or high codon usag

43 nput and recurrent activity, the results can be explained by an architecture in which neural populati

44 putational modeling revealed that this could be explained by an increased sensitivity to subjective v

45 cancer and higher all-cause mortality could be explained by an indirect pathway through EGFR mutatio

46 ission Overall, 15.6% of heterogeneity could be explained by an interplay of known behavioural, socio

47 that cytosolic CAs do stimulate NBCe1-A can be explained by an unanticipated stimulatory effect of t

48 ikely that the observed risk estimates could be explained by an unmeasured confounder.

49 This is explained by an early influence of NR4A3 deficiency o

50 Mechanistically, this is explained by an increased association between RLP44 a

51 Furthermore, the super-repressor phenotype is explained by an increased pool of non-nucleoid bound

52 e higher for two H3N2 strains, findings that were explained by analysis of primer/probe homology.

53 (hereafter "beta" for short) is unlikely to be explained by any single monolithic description, we he

54 k of IS the first 2 years after IE could not be explained by atrial fibrillation (AF) or inserted mec

55 ded-spectrum beta lactamase (ESBL) incidence were explained by background community-level trends, whi

56 by visual contrast with ambient light, which is explained by backscattering calculations for the comp

57 yet multi-year pair bonds are common, could be explained by benefits afforded by mate fidelity to ad

58 These characteristics can be explained by binary interaction.

59 binding patterns, which in some cases could be explained by binding to lipopolysaccharides or capsul

60 observed increases in gonorrhea rates could be explained by both increases in screening and the prev

61 ile the mechanism of CRY-mediated repression was explained by both in vitro and in vivo experiments,

62 enology trait, while genetic differentiation was explained by both intrinsic traits (wingspan and deg

63 anodiamonds in a highly shocked ureilite can be explained by catalyzed transformation from graphite d

64 This dichotomy in burst behavior is explained by cell-group-specific DAP dynamics.

65 st vulnerability when sea ice is absent, can be explained by changes in both heat and moisture transp

66 out of these loci, association with CAD can be explained by changes in gene expression in one or mor

67 he increase seen across birth cohorts cannot be explained by changes in occupation or levels of obesi

68 ce in calcification rates between corals can be explained by changes in the skeletal total amino acid

69 odine emissions from natural seawater cannot be explained by chemical losses of I(2) or hypoiodous ac

70 Age disparities in observed cases could be explained by children having lower susceptibility to

71 owever, it is unclear which fraction of risk is explained by cholesterol and triglycerides, respectiv

72 higher HCC-R compared with NoLRT-NoDS cannot be explained by clinicopathologic differences, suggestin

73 This pattern is explained by color variation within families: Chick c

74 The correlation was explained by common genes influencing both the level

75 y, we show the haplotype allele fraction can be explained by complex concatenated mtDNA-derived seque

76 m of T(1) excitons in this system, which can be explained by considering the exchange interaction in

77 rved inter-tissue expression differences can be explained by corresponding interspecies methylation c

78 of the variability in among-county mortality was explained by county-level race/ethnicity, poverty, u

79 ch in sociology, we show instead that it can be explained by cross-country differences in essentialis

80 he genome segments and propose that this can be explained by cryptic reassortment.

81 predicted visual acuity changes beyond what was explained by CST.

82 trophils and CD4 positive T cells, which can be explained by decreased levels of complement factor D

83 Our results suggest that this decline may be explained by decreased real or perceived visit needs,

84 The loss of QGIT positivity during pregnancy was explained by decreased IFNgamma production in respon

85 These associations may be explained by defective clearance of intraorgan microt

86 These changes are explained by degradation of main secoiridoids during

87 Sex-related differences in angle width are explained by differences among biometric measurement

88 nces in the levels of genetic variation that are explained by differences in the genetic distance fro

89 at differences between cell types can likely be explained by differences in backpropagation efficienc

90 , the differences in RF properties could not be explained by differences in convergence of V1 inputs

91 how variation in patient responsiveness can be explained by differences in peripheral immune cell si

92 in the two most common MS immunopatterns may be explained by different macrophage polarization, origi

93 ncreasingly contradictory results, which may be explained by differential effects of hormones under d

94 se differences in TERRA expression could not be explained by differential methylation of CpG islands

95 tributed to PTSD symptom severity, which may be explained by differential recruitment of prefrontolim

96 Dichromatism in mosaic canaries is explained by differential carotenoid degradation in t

97 These differences in genomic binding were explained by differing abilities to bind to previou

98 place), the performance of pain attenuation was explained by diffusion tensor imaging metrics of inc

99 Poor photoproduct yields are explained by donor-independent, fast charge recombin

100 nding on the adsorbate, a result that cannot be explained by effective medium approximations.

101 The strong anchoring is explained by electrostatic interaction of an ideally

102 reviously attributed to MYBPC3(Delta25), can be explained by enrichment of a derived haplotype, MYBPC

103 In oaks, NSC was explained by environment - values increasing for eve

104 Conifer RPF variability was explained by environment, increasing predominantly t

105 These findings are explained by environmental heterogeneity at coarser

106 to variation through time, and much of this is explained by environmental factors, particularly the

107 s in allergic disease can, at least in part, be explained by epigenetic suppression of human mast cel

108 Over 68% of plumage variation was explained by epistasis between the gene NDP and a ~2

109 t display conspicuous coloration that cannot be explained by existing theory.

110 The elevated risks could not be explained by exposure to chemotherapy, stem-cell tran

111 tment for about half of all couples, and may be explained by factors that are common across models.

112 nset and 20% (12-30) of that of age at death was explained by family membership.

113 These differences were explained by faster cortical thinning linearly thro

114 d that most apparent deviations from HWE can be explained by female hemizygosity rather than low hete

115 4) most variability in EM fungal communities is explained by fine-scale changes in edaphic properties

116 This heterogeneity may be explained by genetic variation within the fatty acid

117 We show that this can be explained by genuine Neanderthal ancestry due to migr

118 leaf T where R reached its maximum (T(max) ) were explained by growth T (mean air-T of 30-d before me

119 Most variability was explained by guidance and needle caliper (P = .15).

120 12.5%, respectively, of phenotypic variance is explained by GxE interactions and that low-frequency

121 % of the variance in language lateralization were explained by handedness.

122 The disparities were explained by health status before MI and characteri

123 nts with neither diabetes nor CKD, which may be explained by high AGE formation in diabetes and decre

124 The slower Tc uptake was explained by higher pyrite solubility under acidic c

125 t species in their endophyte communities may be explained by host specificity, leaf phenology, or mic

126 mol(-1) between diastereoisomeric complexes, is explained by hydrogen- and halogen-bonding, as well a

127 ith the oxidation state of the metal and can be explained by hyperconjugative interactions between en

128 preferences in (macaque) MT neurons that can be explained by image autocorrelation.

129 peculated that such paradoxical findings may be explained by impaired hypoxic pulmonary vasoconstrict

130 er genotype-derived heritability that cannot be explained by inclusion of milder cases and a higher p

131 ases in leaf nitrogen content with elevation were explained by increasing V(cmax) and leaf mass-per-a

132 re variance in cognitive phenotypes than can be explained by individual diagnoses, can be accurately

133 tatory effects, which our modeling shows can be explained by intracellular chloride processing.

134 cht7 colony formation following N refeeding is explained by its compromised viability during N depri

135 We show that the decreased growth rate can be explained by laggard replication fork progression nea

136 hermal stability within lakes, and only 8.4% was explained by lake thermal region or local lake chara

137 ated temperature and moisture regulation can be explained by "law of minimum," i.e., as temperature l

138 occurs in all individuals with SCA6, and can be explained by lesser temporal variability.

139 This effect could not be explained by lexical-semantic processing of the verbs

140 s that the English Industrial Revolution can be explained by Life History Theory's predictions for ps

141 The discrepancies between studies could be explained by limitations of the methods used to quant

142 er in the population of induced cells cannot be explained by limited duplication of centrioles, insta

143 uring the different phases of the task could be explained by linear dynamics for maintaining a distri

144 This is explained by macromolecular rate theory: A negative a

145 1% of the annual variation in wood-NPP could be explained by mean air temperature in May, precipitati

146 test if the difference in sensitivity could be explained by measured morphological and physiological

147 out half of the variation in PicoP abundance was explained by measured environmental variables.

148 shortly after stimulus onset, and could not be explained by mechanisms of response selection or moto

149 suggested that this tuning difference might be explained by mechanoreceptors' differential sensitivi

150 CVE percentages across metropolitan counties is explained by median income, the proportion of the pop

151 n of suicide attempt, which might not solely be explained by mental disorders.

152 ; 95%CI 1.16-3.53) at Tanner B4, which could be explained by metabolic and hormonal exposure in utero

153 Most deaths from cancer are explained by metastasis, and yet large-scale metasta

154 addition, GCF impacts on soil functionality are explained by microbial community structure and bioma

155 prevalent hypothesis on disease pathogenesis is explained by misexpression of a germ line, primate-sp

156 Our empirical data are explained by modeling, demonstrating that resistance

157 nature of chemical bonding in the molybdates is explained by molecular orbital theory and electronic

158 The observed directional CSF flow can be explained by naturally occurring and/or experimenter-

159 s of flowering strips on colony reproduction were explained by nectar availability, but effects of fl

160 ween spatially separated stimuli can largely be explained by normalization within MT.

161 NOTCH1 amplification and overexpression can be explained by NOTCH1 ability to block the DNA damage r

162 . liver, pancreas) in type 2 patients cannot be explained by obesity alone.

163 These relationships could not be explained by other functional annotations known to be

164 ghly variable among patients which could not be explained by other known variables such as hypertensi

165 es in social-distancing compliance could not be explained by other psychological and socioeconomic fa

166 al than ventricular myocytes, and this could be explained by our results showing that MDIMP preferent

167 der mass) increased with the oil load, which was explained by oxygen diffusion.

168 This could partly be explained by parental legacy, rewiring of divergent r

169 s irrigated maize fields in the US Corn Belt was explained by persistent factors and identified the u

170 us traits across multiple environments could be explained by pleiotropic QTL or multiple tightly link

171 conditions; 81.7% of edaphic variation could be explained by polyploidisation.

172 This discrepancy may be explained by posterior shadowing artifact and lack of

173 the association was not as strong, which may be explained by potential selection bias, sample size is

174 nge between two tumor samples can frequently be explained by pre-treatment heterogeneity, such that a

175 cted between the three studied periods, that were explained by precipitation, winter cold and terrain

176 These seemingly contradictory findings are explained by previously underappreciated myofiber lo

177 rol arm in the ACCORD trial is not likely to be explained by QT prolongation leading to lethal ventri

178 This relationship may be explained by race-related differences in central adip

179 large proportion of heritability that might be explained by rare but functionally important variants

180 es in fish abundance occurred too rapidly to be explained by recruitment or mortality, and must there

181 y on BMAL1/CLOCK driven transcription, which is explained by reduced affinity to BMAL1/CLOCK in the a

182 ost pronounced in an appendicular muscle and was explained by reduced myosin activity and fiber degen

183 All of these behavioral effects were explained by reduced evidence accumulation biases.

184 vary widely across nursing homes and cannot be explained by resident characteristics.

185 limitation on perceptual awareness could not be explained by retinal neuroanatomy or previous studies

186 d with beta-adrenoceptor antagonists use can be explained by reverse causation because prodromal Park

187 ether seasonal variation in resistance could be explained by seasonal variation in community antimicr

188 t behavioral and neural response selectivity is explained by sensitivity to spectral contrast rather

189 This neural response selectivity is explained by sensitivity to spectral contrast rather

190 and self-reported depression in adolescence was explained by serial mediation through testosterone t

191 hile the most of aberrant 3'-splice patterns were explained by SF3B1 mutations, we also detected nine

192 absence of seismicity in the flat-lying slab is explained by significantly lower stresses and higher

193 cesses occur on subsecond scales that cannot be explained by simple diffusion models.

194 esting that bacterial community assembly may be explained by simple ecological mechanisms.

195 less likely to co-express, co-localize, and be explained by simple mass action kinetics, and more li

196 fested many genomic anomalies that could not be explained by simple nondisjunction.

197 imized reward and information, but could not be explained by simple stimulus-response rules.

198 vestigated whether habitat distributions can be explained by species hydraulic strategies, and if hab

199 lization of spatial integration in PM cannot be explained by specific projections from V1 to the HVAs

200 arose with static and dynamic cues, couldn't be explained by strategic responding or unfamiliarity, g

201 mputational models suggest that paranoia may be explained by stronger higher-order beliefs about othe

202 gue that the historical NAWH can potentially be explained by such atmospheric mechanisms reliant on s

203 ent choices, little of the overall variation is explained by supply-side variables, and at least half

204 BCR might be explained by target miss.

205 nce severity, and the minority that have can be explained by task disengagement producing impaired ex

206 resent inconsistencies in the literature may be explained by temporal transients in neural signals dr

207 esis suggests that these conflicting results are explained by the background level of rain: Rainfall

208 he high delta(37)Cl values in this lithology are explained by the incorporation of a >30 per mille HC

209 The phenotype could be explained by the ability of the mutated proteins to p

210 proportion of head motion variance that can be explained by the average head trajectory across the l

211 served behavior of climatic complexity could be explained by the changes in cloud amount, and we rese

212 ecular scale, this surprising phenomenon can be explained by the combination of unfolding of disorder

213 ory is imprecise, and these imprecisions can be explained by the combined influences of random diffus

214 This parasite attenuation could not simply be explained by the decreased MIC2 level and strongly su

215 omposition of many empirical networks cannot be explained by the degree of each node alone, or equiva

216 These divergent stabilities could be explained by the different structures formed in solut

217 RNA and protein in developing thymocytes can be explained by the differential translatability of Satb

218 Our results could be explained by the early adoption of widespread testing

219 ed whether capacity limits in perception can be explained by the effects of attention on the allocati

220 The AC cannot be explained by the established mechanisms for conventio

221 We show that this can be explained by the fact that environmental conditions a

222 plastic released into the ocean but can also be explained by the fact that it is not well-known what

223 re we show that this change in mechanism can be explained by the formation of transient ternary compl

224 een hypothesized that the missing loss could be explained by the fragmentation of large aggregates in

225 n breast cancer and schizophrenia may partly be explained by the genetic overlap between the two phen

226 he variation in microbiota composition could be explained by the GFD.

227 take of anthocyanin-rich foods and VAT could be explained by the gut microbiome.

228 ppreciable increase in pore radii, which can be explained by the impact of charge exclusion on ion re

229 dapagliflozin-induced increase in EGP cannot be explained by the increase in plasma glucagon or decre

230 this lack of clinical success can, in part, be explained by the insufficiently stringent clinical sc

231 Properties of social systems can be explained by the interplay and weaving of individual

232 tensibility in roots, a phenotype that could be explained by the involvement of galactosidases in AGP

233 This can be explained by the low neutrophil surface expression of

234 d 18F-FDG uptake in non-metastatic nodes can be explained by the MDSC-mediated premetastatic niche fo

235 We further demonstrated that this could be explained by the mitigation of increased neutrophil e

236 This observation may be explained by the multidimensional response properties

237 F-induced heating of single-domain SPION can be explained by the Neel relaxation (reorientation of th

238 This can be explained by the observation that, on the Au electrod

239 These differential effects can be explained by the order of the strengths of pairwise a

240 The array data may be explained by the possible bivalent binding mode of a

241 Impaired antibody binding could be explained by the presence of insertion sequences or m

242 The mechanism underlying these symptoms can be explained by the presence of nonfiring pacemaker cell

243 milarity in the flanking regions could often be explained by the presence of similar long terminal re

244 rations of AAs (6.5-55 ug mL(-1)), which can be explained by the protein cleavage.

245 of the total variance of daily F(CH4) could be explained by the random forest machine learning algor

246 The recognition by ABCC1 could be explained by the reduction kinetics of a ternary Cu-C

247 ity (i.e., the number of cusps, crests), can be explained by the same developmental model.

248 se patterns of restricted connectivity could be explained by the short pelagic larval duration of S.

249 especially for Eastern countries), which may be explained by the signaling value of this rejection.

250 Such deviation from hard sphere behavior may be explained by the soft and deformable layer of ligands

251 This effect can most likely be explained by the stimulation of endogenous KA-recepto

252 the mechanochemical properties of KIF3AC can be explained by the strain-dependent kinetics of KIF3A a

253 The differences in connectivity could be explained by the strength of recurrent dynamics in at

254 ution of a trait in a clade of organisms can be explained by the sustained action of natural selectio

255 , and in 44% of pixels, this variation could be explained by the temperature.

256 cy tuning, of aged auditory nerve fibers can be explained by the well known reduction of endocochlear

257 The smoker's paradox may be explained by the younger age and fewer cardiovascular

258 portion of the selection exerted on microbes is explained by the environment and is associated with n

259 ients treated with belatacept and whether it is explained by the higher risk of CMV reactivation and

260 Approximately 60% of this variation is explained by the home environment of a clone and the

261 The mechanism inducing the enhancement is explained by the initially reparable pores generated

262 The elongation defect is explained by the loss of the elongation factors LEO1

263 This is explained by the massive reduction of the mass transf

264 This discrepancy is explained by the observation that N-helix binding is

265 This phenomenon is explained by the PPP2R1A inhibition impacting primari

266 This is explained by the prolonged time to response seen in m

267 Sixty-six percent of that variation is explained by the proportion of the population that ho

268 verall quantum efficiency of photoactivation is explained by the requirement of correct occupancy of

269 yst stability of the 5Ni/3Mg-ZrO(2) catalyst was explained by the ability of CO(2) to partially oxidi

270 d 11% of the excess hazard among black women was explained by the higher prevalence of malignant LVH

271 Having SAM among under-five children was explained by the individual-, household- and neighbo

272 e 2 diabetes, respectively (P < 0.05), which was explained by the physiologic age-dependent decline.

273 onset and 9% (3-21) of that of age of death was explained by the specific mutation, whereas 14% (9-2

274 8% of the observed oxidative stress response were explained by the detected chemicals, less than 10%

275 The adsorption kinetics were explained by the pseudo-second-order model, and the

276 These results were explained by the relative concentration and affinit

277 its inhibitory properties compared with dUMP were explained by the stronger acidity of the uracil N3

278 rse, reproducible expression levels that can be explained by their chance inclusion of functional TF

279 with and without protective symbionts cannot be explained by their difference in fitness alone.

280 tinct behaviors of the three HULIS types can be explained by their different chemical compositions, f

281 ive diffusion properties of lipid probes can be explained by their dynamic partitioning into Lo-like

282 transitions in the y = 1 to 7 materials can be explained by their structural properties.

283 mass correlation was high (r = 0.67) and may be explained by these formation routes, which, in part,

284 Importantly, the latter cannot be explained by theta power changes, head movement, anti

285 population of wild-type and mutant cells can be explained by this mechanism, coupled with a heterogen

286 al images, and that motion misperception can be explained by this speed-contrast association not a "s

287 ese metabolic effects of oncogenic KRAS have been explained by transcriptional upregulation of glucos

288 We show that this deviation can be explained by transmission of long wavelengths through

289 eely moving mice and found both observations are explained by two eye-head coupling types, associated

290 RI and electrophysiological measurements can be explained by two (non-mutually exclusive) characteris

291 Using an enhanced vertex model, we show this is explained by two effects: thresholded tension remodel

292 We argue that these phenomena can be explained by uncertainty about where events occurred.

293 Alternatively, they may be explained by unmeasured differences in early life exp

294 nsitivity to cholesterol depletion could not be explained by variation in Env components known to int

295 s in rhizosphere community composition could be explained by variation in plant growth dynamics.

296 w-stability layer in the polar region cannot be explained by vertical convection in the middle/lower

297 v(max) differences were explained by vertical ground reaction forces (vGRFs

298 tching success in response to noise exposure were explained by vocalization frequency, nesting locati

299 of the ambient burden of organic UV-filters are explained by volatilization from terrestrial and aqu

300 etails of this extremely distant interaction are explained by water waves quantified by thermodynamic