コーパス検索結果 (１語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 ction (H-function) in potential heart donors is ill defined.
2 cular basis for AC-induced inhibition of DCs is ill defined.
3 NA interaction by the acidic C-terminal tail is ill defined.
4 DNA methyltransferase (DNMT) gene expression is ill defined.
5 nomenclature for minimally invasive surgery is ill defined.
6 the virulence of gastrointestinal pathogens is ill defined.
7 functions, its role in cell differentiation is ill defined.
8 pores: how these pores are sensed and sealed is ill defined.
9 cardiometabolic disease in affected persons is ill defined.
10 but the molecular basis for Sen1 activities is ill defined.
11 nment to regulate circulating glucose levels is ill defined.
12 prostate cancer dissemination and lethality is ill defined.
13 including high-mobility group box 1 (HMGB1), is ill defined.
14 l activity leads to transporter inactivation is ill defined.
15 rial dysfunction and inflammasome activation is ill defined.
16 lar function of these receptors in cognition is ill defined.
17 ell's proliferative and apoptotic mechanisms is ill defined.
18 polarization in skeletal muscle regeneration is ill defined.
19 phocyte-predominant Hodgkin lymphoma (NLPHL) is ill defined.
20 e activating complexes of the lectin pathway is ill defined.
21 from traditional measures to novel therapies is ill defined.
22 machinery regulating exit from pluripotency is ill defined.
23 rticipate in innate defense against bacteria is ill defined.
24 The underlying molecular mechanism, however, is ill defined.
25 iac death for asymptomatic mutation carriers is ill defined.
26 poietic lineage differentiation in the adult is ill defined.
27 20% of bacterial genomes; however, its role is ill defined.
28 responses, the benefit for T cell responses is ill defined.
29 various nutrients, but their clinical impact is ill defined.
30 e appropriate amount of biventricular pacing is ill-defined.
31 mitochondria, but regulation of this process is ill-defined.
32 conformations, particularly if the receptor is ill-defined.
33 onse, however, the mechanism for this uptake is ill-defined.
34 cardiac imaging in ethnic minority patients is ill-defined.
35 d, its role in determining RVLM excitability is ill-defined.
36 although their role in Fas-induced apoptosis is ill-defined.
37 l to clinical and stress testing evaluation, is ill-defined.
38 The genetic basis of Gilbert's syndrome is ill-defined.
39 ered, aggregation-prone proteins are handled is ill-defined.
40 How non-M HIV-1s interact with huTRIM5alpha is ill-defined.
41 he role of pS-STAT3 among epithelial cancers is ill-defined.
42 ole of S100A8 in lung antibacterial immunity is ill-defined.
43 induced by Dox in the heart, its role in AIC is ill-defined.
44 tribution of these variants to tumorigenesis is ill-defined.
45 impact of its absence on biological systems is ill-defined.
46 as octamers and the cause of their pathology is ill-defined.
47 length of stay (LOS), but their performance is ill-defined.
48 lated by external signals, but the mechanism is ill-defined.
49 ounds such as perfluorooctanoic acid (PFOA), is ill-defined.
50 rgy expenditure, but exactly how this occurs is ill-defined.
51 M (IgM) in the first trimester of pregnancy is ill-defined.
52 reticulum (ER) stress leads to cytotoxicity is ill-defined.
53 ct of inferred size on attentional selection is ill-defined.
54 eting to either mitochondria or chloroplasts is ill-defined.
55 the relative importance of each interaction is ill-defined.
56 s that drive neurogenesis within these zones are ill defined.
57 immune protection in this patient population are ill defined.
58 fector activities in vivo at infection sites are ill defined.
59 nd 2 (M2; crisis) of human replicative aging are ill defined.
60 usceptibility to contact allergic dermatitis are ill defined.
61 rder, brain regions affected in the disorder are ill defined.
62 r complications, and associated risk factors are ill defined.
63 athways controlled by the actin cytoskeleton are ill defined.
64 ricuspid valve replacement in young children are ill defined.
65 s associated with renal and patient survival are ill defined.
66 or perturb the cooperative activation events are ill defined.
67 rapy (ALA-PDT), but its nature and mediation are ill defined.
68 the limitations of RNAi antiviral strategies are ill defined.
69 ors that promote tumor growth, their origins are ill defined.
70 Mechanisms mediating this enhanced risk are ill defined.
71 smission of pathogens during epidemic spread are ill defined.
72 ges associated with cellular differentiation are ill defined.
73 ate graft failure in acute cardiac rejection are ill defined.
74 atrix-degrading proteases during lung injury are ill defined.
75 on has been reported, but operative outcomes are ill defined.
76 or endoscopic predictors of their diagnosis are ill defined.
77 e transition, but the mechanisms responsible are ill defined.
78 rix (ECM), but their molecular underpinnings are ill defined.
79 ention strategies in immunocompromised hosts are ill defined.
80 HIV) and cryptococcal meningitis coinfection are ill defined.
81 ing pathways that regulate their development are ill defined.
82 development and progression, but mechanisms are ill defined.
83 nhibition play in the pathophysiology of FXS are ill defined.
84 statin and immune cells from the bovine host are ill defined.
85 s in this ecosystem and to clinical outcomes are ill defined.
86 nociceptors, but the details of this process are ill defined.
87 infection, but the cellular sources of iNOS are ill defined.
88 al mechanisms responsible for this, however, are ill defined.
89 tures justifying longer or shorter durations are ill defined.
90 mation, but the cellular mechanisms involved are ill defined.
91 induce hepatic character within the endoderm are ill defined.
92 eurons and oligodendrocytes after CNS injury are ill defined.
93 ive but histological features indicating UOC are ill defined.
94 ion in shear-resistant cell arrest on VCAM-1 are ill defined.
95 t govern neutrophil migration across the BBB are ill defined.
96 y Ranbp2 in tissue homeostasis, such as RPE, are ill defined.
97 ise mechanisms governing this susceptibility are ill defined.
98 he underlying causative molecular mechanisms are ill defined.
99 , although the precise contributions of each are ill defined.
100 in PDAC pathogenesis, epigenetic alterations are ill defined.
101 terms of molecular processes and cell fates are ill defined.
102 ms that regulate obesity-driven inflammation are ill defined.
103 ated, but the cell-specific roles in the StV are ill defined.
104 ers where the functions of pSTAT1 and uSTAT1 are ill defined.
105 llular mechanisms responsible for this event are ill defined.
106 fferent IFN-I sources and signaling pathways are ill defined.
107 isms by which CENP-Bs effect their functions are ill defined.
108 r mechanisms of action in pulmonary diseases are ill defined.
109 n recognized in the adult and its management are ill defined.
110 ar mechanisms that underlie these properties are ill defined.
111 isms which render a patient LC amyloidogenic are ill-defined.
112 related disorders, but the neural substrates are ill-defined.
113 rs; however, the mechanisms for this process are ill-defined.
114 any of the parameters, such as force fields, are ill-defined.
115 d with seasonal influenza virus NA proteins, are ill-defined.
116 rgent evolution of "new" enzymatic functions are ill-defined.
117 et the roles for the toxins during infection are ill-defined.
118 mechanistic links between Idic15 and autism are ill-defined.
119 oglial cells in vivo during CNS autoimmunity are ill-defined.
120 ated kinase pathway, although the mechanisms are ill-defined.
121 after, yet the underlying neural mechanisms are ill-defined.
122 d, the molecular functions of the polyamines are ill-defined.
123 However, the underlying molecular mechanisms are ill-defined.
124 underlie the immunosuppressive effects of uG are ill-defined.
125 al impacts of interferon hyperactivity in DS are ill-defined.
126 iteria for rational design of such compounds are ill-defined.
127 the mechanisms underlying this pathobiology are ill-defined.
128 ors underlying this exacerbated pathobiology are ill-defined.
129 rs of innate immunity that promote emphysema are ill-defined.
130 esponse to oxidative stress in the adult fly are ill-defined.
131 nfections, yet specific immune perturbations are ill-defined.
132 that govern IL-10(+) B cell differentiation are ill-defined.
133 nd mechanistic drivers following third doses are ill-defined.
134 s involved in the regulation of this process are ill-defined.
135 ilization aside from vitamin A status itself are ill-defined.
136 onal relevance and substrate choice of MGAT5 are ill-defined.
137 g SAg-associated immunosuppression in humans are ill-defined.
138 , the involved interhemispheric interactions are ill-defined.
139 g cellular and molecular mechanisms of which are ill-defined.
140 The benefits of prehabilitation are ill-defined.
141 The mechanisms allowing CPPs to enter cells are ill-defined.
142 opulation, though the "exposure-lag" effects are ill-defined.
143 m cell maintenance, but the ligands involved are ill-defined.
144 the determinants of LIG1 rNMP discrimination are ill-defined.
145 OVID-19 patients, but underlying aetiologies are ill-defined.
146 isruption (ECD) such as shiftwork and jetlag are ill-defined.
147 -OPA1 in mitochondrial fusion and energetics are ill-defined.
148 nal consequences of takotsubo cardiomyopathy are ill-defined.
149 However, the substrates for these enzymes are ill-defined.
150 s underlying enhanced somatic susceptibility are ill-defined.
151 ry mechanisms and neuronal circuit functions are ill-defined.
152 that contribute to a protumorigenic ecology are ill-defined.
153 P dynamics in these subcellular compartments are ill-defined.
154 r, biological predictors of response to CIRT are ill-defined.
155 mechanisms underlying such ultra-sensitivity are ill-defined.
156 ma, but its pathogenesis and natural history are ill-defined.
157 as being important, but their specific roles are ill-defined.
158 d mechanisms of response and resistance have been ill defined.
159 er their roles in astrocytic physiology have been ill defined.
160 y broad range of unique structures with many being ill-defined.
161 ronic infections such as active tuberculosis is ill defined, although several case reports and small
162 anisms of the anti-Bartonella immune defense are ill-defined and bacterial immune evasion strategies
166 ar and genetic bases of such sex differences are ill-defined and presumptively ascribed to X-chromoso
167 tion to RSV genetic and antigenic diversity, is ill defined and has implications for persistence and
168 inflammation, such as rheumatoid arthritis, is ill defined and probably requires tight regulation.
169 olved in cell cycle regulation, but its role is ill defined and the mechanisms have not been explored
172 at, in some literature, the term "attention" is ill-defined and the constraints of peripheral vision
175 in B cell and T cell maturation or function are ill defined, and few transcriptional targets are kno
176 x differences in peripheral vascular disease are ill defined, and there is a need to report and under
177 ltransferase in human T-cell differentiation is ill defined, and manipulation of G9a function for the
178 sence of periodicity, the structure of glass is ill-defined, and a large number of structural states
179 oint of the 'new' eve TAD on the nhomie side is ill-defined, and eve stripe enhancers activate a near
180 intains its plasmid-encoded resistance genes is ill-defined, and novel targets for the treatment of V
182 mes of carriers destined for many organelles are ill-defined because the vesicular intermediates are
183 role of human CAFs in cancer progression has been ill-defined because human CAFs lack a unique marker
185 ical consequences of this replicative stress are ill defined, but any "ageing" effect would carry the
187 2 in the pathogenesis of human breast cancer are ill-defined, but our analysis of publically availabl
189 sms of bladder granulomogenesis and fibrosis are ill defined due to the prior lack of tractable anima
193 after left ventricular assist device (LVAD) are ill-defined in elderly patients, and with new-genera
195 ingly, the transgenic cartilaginous elements were ill defined, intermingled with surrounding connecti
196 anisms underlying antigen cross-presentation are ill-defined, most notably in human dendritic cells (
199 Without smoothing, imputation is likely to be ill-defined or jump erratically from one strain to an
200 the precise mechanisms of MeHg neurotoxicity are ill-defined, oxidative stress and altered mitochondr
201 in sub-Saharan Africa but their epidemiology is ill-defined, particularly in highly malaria-endemic r
202 h amniotic fluid regulates fetal development is ill-defined partly due to an incomplete understanding
203 ution of the East Antarctic Ice Sheet (EAIS) is ill defined, restricting our appreciation of potentia
207 by which these improvements occur, however, are ill-defined; thus, the therapeutic potential of exer
208 rbed on GC, for which the voltammetric peaks were ill-defined, too small for coulometric analyses to