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1 r, to date, their function in fruit ripening is largely unknown.
2 ral formation of G4 structures in the genome is largely unknown.
3 ferentiation of other hematopoietic lineages is largely unknown.
4 tain in vivo models through a mechanism that is largely unknown.
5 scriptional identity and L4 cytoarchitecture is largely unknown.
6 on with high cardiometabolic disease burden, is largely unknown.
7 interkingdom QS and antimicrobial resistance is largely unknown.
8 ues in repair of the established vasculature is largely unknown.
9 k from brief oral steroid bursts (<=14 days) is largely unknown.
10 facilitating or preventing range expansions is largely unknown.
11 muli, though its role during virus infection is largely unknown.
12 modulated upon exposure to microbial agents is largely unknown.
13 ects compare to other anthropogenic forcings is largely unknown.
14 f RyR in physiologically relevant conditions is largely unknown.
15 to therapeutics targeting effects of gliosis is largely unknown.
16 ulture media their effect on differentiation is largely unknown.
17 transplantation on Epstein-Barr virus (EBV) is largely unknown.
18 systemic (versus specific) ecosystem changes is largely unknown.
19 ement of leukocytes in the peritoneal cavity is largely unknown.
20 d with the emergence of novel brain pathways is largely unknown.
21 nism by which DRP1 is regulated in the ovary is largely unknown.
22 matin remodeling contributes to this process is largely unknown.
23 olecular basis that underlies this mechanism is largely unknown.
24 role of ferroptosis in placental dysfunction is largely unknown.
25 ered in response to acute and chronic stress is largely unknown.
26 , but the plasticity of hydraulic strategies is largely unknown.
27 wever, how soil pathogens sense host signals is largely unknown.
28 Scrib and Lgl1 interaction on cell polarity is largely unknown.
29 this output signal within the mEC, however, is largely unknown.
30 onal risk factor exposure) of FMT protection is largely unknown.
31 ut the molecular basis for their involvement is largely unknown.
32 uses severer dental caries than other sugars is largely unknown.
33 ower, but the underlying molecular mechanism is largely unknown.
34 ortex (M1), which directs skilled movements, is largely unknown.
35 in secondary site for full-blown metastasis is largely unknown.
36 ication or influence therapy decision-making is largely unknown.
37 virus Receptor 1 (XPR1), regulation of which is largely unknown.
38 the cellular requirement for energy and heat is largely unknown.
39 biogeographies exhibit divergent behaviours is largely unknown.
40 ironment, their occurrence in surface waters is largely unknown.
41 er, how these events are controlled by light is largely unknown.
42 where the thermochemistry of PCET substrates is largely unknown.
43 bution of different cancer deposits to ctDNA is largely unknown.
44 tic regulation of NF-kappaB in tumor context is largely unknown.
45 s-induced changes in IPP on OvCa progression is largely unknown.
46 ng environmental parameters and nanoplastics is largely unknown.
47 How cells fine tune cohesin deposition is largely unknown.
48 R, whether it regulates presynaptic function is largely unknown.
49 l variants are and how they contribute to BD is largely unknown.
50 F) duration on outcomes and treatment effect is largely unknown.
51 breached; however, when and how this occurs is largely unknown.
52 on of upper thermal tolerance in vertebrates is largely unknown.
53 sector-specific guidelines on child outcomes is largely unknown.
54 ental proteins to provoke allergic responses is largely unknown.
55 stics of the nanocomposites composed thereof is largely unknown.
56 the role of RasGEF in plant pathogenic fungi is largely unknown.
57 different phases across the human life span is largely unknown.
58 n mitigate climatic risks via these pathways is largely unknown.
59 How FTD-causing disease genes interact is largely unknown.
60 variants (SVs) on gene expression in cancer is largely unknown.
61 s feature is linked to hippocampal functions is largely unknown.
62 le progression, but its molecular regulation is largely unknown.
63 ffect of this difference on CWD pathogenesis is largely unknown.
64 (TCR) signaling, but their role in lymphoma is largely unknown.
65 the function of colors in the fungal kingdom is largely unknown.
66 However, the mechanism of modulation is largely unknown.
67 poral processing in high-level visual cortex is largely unknown.
68 ion of SPL10 in regulating floral transition is largely unknown.
69 ch are primarily histo-blood group antigens, is largely unknown.
70 although the mechanism underlying rejection is largely unknown.
71 impact of this warming on the MJO life cycle is largely unknown.
72 which genetic factors increase risk for PAD is largely unknown.
73 ts of this key posttranscriptional regulator is largely unknown.
74 nnectivity (synchrony) between brain regions is largely unknown.
75 vascular endothelial identity and integrity is largely unknown.
76 ct, but the viral fraction of this community is largely unknown.
77 ot fully understood because their regulation is largely unknown.
78 l chemistry of this system at high pressures is largely unknown.
79 ology of its various forms and comorbidities is largely unknown.
80 How DISC1 regulates gene expression is largely unknown.
81 tion and overall cellular Ca(2+) homeostasis is largely unknown.
82 s transmembrane-conductance regulator (CFTR) is largely unknown.
83 ultimately achieve this aberrant production is largely unknown.
84 likelihood of GBS interspecies transmission is largely unknown.
85 onal diversity of valve cells in development is largely unknown.
86 The impact of surgery on patient survival is largely unknown.
87 nt CRC not recognized to have these features is largely unknown.
88 ctivity regulates synaptic vesicle recycling is largely unknown.
89 t its specific role in pancreatic beta-cells is largely unknown.
90 he function of ZO-1 in cardiac myocytes (CM) is largely unknown.
91 neous group of conditions the cause of which is largely unknown.
92 nism of bone formation during this procedure is largely unknown.
93 h proteoglycans regulate signal transduction is largely unknown.
94 their direct influence on the MIA phenotype is largely unknown.
95 valve endocarditis (PVE), long-term outcome is largely unknown.
96 end against plant root-knot nematodes (RKNs) is largely unknown.
97 shes, maintains and reactivates from latency is largely unknown.
98 ffect of such interventions on mental health is largely unknown.
99 el architecture within the 3D genome context is largely unknown.
100 ential growth pattern between the 2 chambers is largely unknown.
101 cular clock is involved in this coordination is largely unknown.
102 r, the mechanism by which KRAS regulates p53 is largely unknown.
103 r, the genetic architecture underlying NSCPO is largely unknown.
104 ever, how TET1 interacts with other proteins is largely unknown.
105 al regulation of transferrin receptor (TfR1) is largely unknown.
106 promising category of host plant resistance, is largely unknown.
107 s function during the progression of obesity is largely unknown.
108 How diapause protects living organisms is largely unknown.
109 sensitive versus tolerant to extreme drying are largely unknown.
110 arities and differences among these proteins are largely unknown.
111 y mechanisms for Cdc42 activity in podocytes are largely unknown.
112 ls, the mechanisms involved in such coupling are largely unknown.
113 AD, single cell-based molecular alterations are largely unknown.
114 ene repression and commitment in lymphocytes are largely unknown.
115 significance of nuclear Ca(2+)-changes in AF are largely unknown.
116 ir input signals and how these are processed are largely unknown.
117 ut human disorders related to KCNN2 variants are largely unknown.
118 function of temperate microbial communities are largely unknown.
119 species; however, the underlying mechanisms are largely unknown.
120 t progression, metastasis, and poor survival are largely unknown.
121 ) development, maintenance, and degeneration are largely unknown.
122 n the maternal skeleton and skeletal muscles are largely unknown.
123 the upper respiratory tract (URT) microbiome are largely unknown.
124 tacts control synapse formation and function are largely unknown.
125 lopment, the regulators of these transitions are largely unknown.
126 protein crystallization in vivo or in vitro are largely unknown.
127 t, but the mechanisms underlying this effect are largely unknown.
128 The mechanisms by which this happens are largely unknown.
129 mechanism of action and its activity on DNA are largely unknown.
130 s regulating their formation and arrangement are largely unknown.
131 underpinnings of this nutrient dose-response are largely unknown.
132 g mechanisms of antibody binding to bacteria are largely unknown.
133 at initiate the biogenesis of this structure are largely unknown.
134 rlying specific counter-regulatory responses are largely unknown.
135 echanisms of their role(s) in plant immunity are largely unknown.
136 s mediating lipid removal (i.e. deacylation) are largely unknown.
137 happens at a cellular and subcellular level are largely unknown.
138 s underlying breast cancer risk associations are largely unknown.
139 tionally important in these progenitor cells are largely unknown.
140 sease (NAFLD), but the underlying mechanisms are largely unknown.
141 cells evade the deleterious effects of CDCs are largely unknown.
142 munodeficiency virus (SHIV) in these tissues are largely unknown.
143 h neutrophils are recruited to these tissues are largely unknown.
144 ting to the strains' abilities to reactivate are largely unknown.
145 individual patients respond to kidney injury are largely unknown.
146 diomyocyte proliferation, but the mechanisms are largely unknown.
147 hat drive tissue-specific physiology in vivo are largely unknown.
148 eukaryotes, where their biological functions are largely unknown.
149 f the channel and their functions in mammals are largely unknown.
150 The cellular mechanisms are largely unknown.
151 for profiling rhesus Ig and TCR repertoires are largely unknown.
152 ive rise to specific patterns of MN activity are largely unknown.
153 nsformations enabling noise robust abilities are largely unknown.
154 amilial risk for developing mental illnesses are largely unknown.
155 e subcellular organization of the centrosome are largely unknown.
156 center (TC) versus the invasive margin (IM) are largely unknown.
157 luence on the subsequent development of PTSD are largely unknown.
158 ms, but the mechanisms enabling this process are largely unknown.
159 ciations and their impacts on specific genes are largely unknown.
160 olecular mechanisms underlying this function are largely unknown.
161 ; however, their roles in Drosophila ovaries are largely unknown.
162 patients respond, and resistance mechanisms are largely unknown.
163 ses, but the roles of mitochondrial circRNAs are largely unknown.
164 The biocidal mechanisms of FNA are largely unknown.
165 polarity of growth to generate novel shapes are largely unknown.
166 interactive effects and effect size (eta(2)) are largely unknown.
167 ysiology and molecular targets for treatment are largely unknown.
168 r mechanisms of its action in such processes are largely unknown.
169 capacity to intentionally control our memory are largely unknown.
170 underlying cellular and molecular mechanisms are largely unknown.
171 n, but their variabilities and consistencies are largely unknown.
172 sistance to therapy and duration of dormancy are largely unknown.
173 pathogenesis of prenatally triggered asthma are largely unknown.
174 tion of pores and their biological functions are largely unknown.
175 ors guiding this complex to specific targets are largely unknown.
176 their functions and role in AD pathogenesis are largely unknown.
177 dominant CA-MRSA clone in the United States, are largely unknown.
178 the mechanisms of aberrant SOX11 expression are largely unknown.
179 form of invasive pneumococcal disease (IPD), are largely unknown.
180 ry networks (TRNs) that control ILC function are largely unknown.
181 position and functions of the CA projections are largely unknown.
182 n and chromatin metabolism by mTOR signaling are largely unknown.
183 derpinning host defence and parasite offence are largely unknown.
184 ndent breast cancer and endocrine resistance are largely unknown.
185 cer, but the underlying molecular mechanisms are largely unknown.
186 regression, or probability for embolization are largely unknown.
187 The genetic foundations of this complex form are largely unknown.
188 herapeutically targeting mutant cancer cells are largely unknown.
189 nisms responsible for establishing normal OD are largely unknown.
190 nisms regulating aging at the cellular level are largely unknown.
191 pathogen derived, that mediate these changes are largely unknown.
192 and higher order genomic structural features are largely unknown.
193 anscription factor IIH (TFIIH), is recruited are largely unknown.
194 driving prophage induction in the microbiota are largely unknown.
195 the non-vascular plant Physcomitrella patens are largely unknown.
196 ic stem and progenitor cell (HSPC) expansion are largely unknown.
197 ctor T cells that contribute to this process are largely unknown.
198 l types responsible for these manifestations are largely unknown.
199 ferentiation and instruct its graded pattern are largely unknown.
200 these synapses, but these molecular players are largely unknown.
201 progression of chronic inflammatory diseases are largely unknown.
202 sms underlying different infiltration levels are largely unknown.
203 ve and evolutionary roles of leaf senescence are largely unknown.
204 the factors driving long-term changes in GPP are largely unknown.
205 in different cell types in mammalian tissue are largely unknown.
206 face sensitivity in visuo-social processing are largely unknown.
207 The mechanisms by which such elements work are largely unknown.
208 bly and functioning of reef fish communities are largely unknown.
209 anisms that regulate expression of SMN genes are largely unknown.
210 ulation of T95 in these specific areas of SR are largely unknown.
211 but associated mechanisms and links to IL-4 are largely unknown.
212 spermathecae, and their response to mating, are largely unknown.
213 style changes, but the underlying mechanisms are largely unknown.
214 ase, and vaccine and therapeutic development are largely unknown.
215 GLP-1R agonists on cocaine-seeking behavior are largely unknown.
216 asticity lies at the highest trophic levels, are largely unknown.
217 tuations and their putative role in function are largely unknown.
218 r the context of a rapidly changing climate, are largely unknown.
219 n identified, but their molecular mechanisms are largely unknown.
220 of sporadic benign pituitary adenoma growth are largely unknown.
221 nisms by which this heritable priming occurs are largely unknown.
222 e Anthropocene yet their interactive effects are largely unknown.
223 AHs and OPAHs in the atmospheric environment are largely unknown.
224 nisms of CD4(+) T cell-mediated pathogenesis are largely unknown.
225 f KLHL14 in DLBCL and its molecular function are largely unknown.
226 , but the causal variants and effector genes are largely unknown.
227 -dependent regulation of pathogen resistance are largely unknown.
228 abolic connections and requirements involved are largely unknown.
229 echanisms that support such memory retrieval are largely unknown.
230 aping life history divergence within species are largely unknown.
231 arly in the hippocampus, where its functions are largely unknown.
232 itive and neural bases of code comprehension are largely unknown.
233 e and mechanisms of action for human lncRNAs are largely unknown.
234 n signalling and its physiological relevance are largely unknown.
235 These interactions are largely unknown.
236 vents have been identified, yet the pathways are largely unknown.
237 ar mechanisms that regulate CPSF6 expression are largely unknown.
238 scles and their impact on muscle performance are largely unknown.
239 naling to induce opposing cellular functions are largely unknown.
240 ications for peptide biogenesis and activity are largely unknown.
241 d genes in the somatic tissues of Drosophila are largely unknown.
242 d mechanisms of reactivity with DOM moieties are largely unknown.
243 , the molecules causing poultry meat allergy are largely unknown.
244 but widespread regulators of chromatin state are largely unknown.
245 mechanism of Abi1 in smooth muscle migration are largely unknown.
246 iptional level, but the proteins responsible are largely unknown.
247 e to stroke and dementia, but the mechanisms are largely unknown.
248 which the mechanism of substrate recognition was largely unknown.
249 genesis, the downstream program it activates was largely unknown.
250 nderlie such uncertainty-guided control have been largely unknown.
252 The functioning of the holobiont as a whole is largely unknown, although bacteria and viruses are pr
254 intestinal (GI) tract and respiratory system are largely unknown and remained illusive for several de
255 transduce acutely painful mechanical forces is largely unknown and remains one of the 'final frontie
256 the normal function of TDP-43 in astrocytes are largely unknown, and the role of astroglial TDP-43 l
258 volvement in disorders with complex genetics is largely unknown, as is the extent of their heterogene
259 g effects of biodiversity loss on other taxa are largely unknown because baseline data are often unav
260 l potency and selectivity of BRD9 inhibitors is largely unknown because of the lack of structural inf
261 hose providing essential ecosystem services, are largely unknown, but directly impact our health and
262 ow PCBs affect paternal reproductive success is largely unknown, but could ultimately affect populati
263 The long-term effect of diet on skin aging is largely unknown, but evidence suggests that the antio
264 gulatory mechanism of this signaling pathway is largely unknown despite its importance in stem cell m
265 sound is generated in the hummingbird syrinx is largely unknown despite their complex vocal behavior.
266 rategies are shaped by winter habitat choice is largely unknown due to the general inability to track
267 ith L and how that affects the activity of L is largely unknown due to the lack of high-resolution st
268 ns, as those encountered in the high Arctic, is largely unknown, especially for species where the ges
269 genetic variation and breast cancer survival is largely unknown, especially in understudied minority
275 arkers that predict cognitive deficits in BD are largely unknown, however recent evidence suggests th
285 nce the system has dozens of parameters that are largely unknown, it remains a challenging problem to
286 pheral mechanisms underpinning this recovery are largely unknown, limiting development of new clinica
287 The geographic variation of human movement is largely unknown, mainly due to a lack of accurate and
288 mechanisms that trigger acute chest syndrome are largely unknown.Objectives: To identify the innate i
289 eness of disinfection processes on norovirus is largely unknown owing to the lack of a readily availa
290 f these and other functionally related genes are largely unknown, particularly in cancers not classic
291 public health implications of these actions are largely unknown, particularly with respect to mosqui
292 fied, but the causatives genes at these loci are largely unknown, so that functional investigation an
294 s and enzymes responsible for biodegradation are largely unknown, the relative roles of abiotic proce
296 ng PKA activation and aquaporin-2 regulation were largely unknown until the advent of modern protein
298 increases the risk of neurodegeneration, it is largely unknown whether HFE mutation modifies GABAerg
299 ng in all physiological systems, although it is largely unknown whether this response differs with in
300 controlling these transcriptional mediators are largely unknown, which limits therapeutic strategies