ライフサイエンスコーパス: be protected from

1 pts associated with arrested RNA polymerases are protected from 3'-5' degradation and thus, unstable

2 ed body weight and fat mass, LFABP(-/-) mice are protected from a high-fat feeding-induced decline in

3 eceptor (TLR) adaptor Mal (encoded by TIRAP) are protected from a number of infectious diseases, incl

4 because we show that vaccinated animals can be protected from a mucosal challenge with a heterologou

5 ina using a prime-boost vaccination strategy were protected from a subsequent lung challenge with P.

6 t manner while the conjugated-ubiquitin (Ub) is protected from active deubiquitination.

7 mice carrying the PTPN22 variant (619W mice) were protected from acute dextran sulfate sodium (DSS) c

8 These mice were protected from acute podocyte foot process effaceme

9 wild-type littermates, hCD39 transgenic mice were protected from acute renal injury at 24 hours, but

10 Hippocampal slices lacking Pyk2 are protected from AD-related Abeta oligomer suppression

11 PNLIP-knockout mice, unlike ATGL knockouts, were protected from adipocyte-induced pancreatic acinar

12 male) mice deficient in Arg-II (Arg-II(-/-)) are protected from age-associated glucose intolerance an

13 necrosis factor (TNF)-deficient mice, which are protected from age-associated inflammation, age-rela

14 ar fat mass and energy balance, M(IL10) mice were protected from aging-associated insulin resistance

15 WT but not Ido1(-/-) mice were protected from AIA by IFN-alpha, and Kyn, the main

16 ted that integrin alphavbeta6-deficient mice are protected from airway hyperresponsiveness, due in pa

17 Consistent with this, miR-155-deficient mice were protected from alcohol-induced disruption of autoph

18 inflammasome components NLRP3 and caspase-1 were protected from aldosterone-induced vascular damage.

19 In contrast, NT4(-/-) mice were protected from allergen-induced mucus overproductio

20 nce for AD pathophysiology, Pyk2(-/-) slices are protected from amyloid-beta-oligomer (Abetao)-induce

21 All animals were protected from an intrarectal SIVmac239 challenge,

22 s transferred from mice treated with OVA-Sp) were protected from anti-MPO autoimmunity and GN, confir

23 Within DCs A. terreus conidia were protected from antifungals, whereas A. fumigatus co

24 This hidden provirus is protected from antiviral drugs until it eventually re

25 that 13-lined ground squirrel tubular cells are protected from apoptotic cell death during IBA.

26 f IP-mediated relaxation, iSM-Gprc5b-KO mice were protected from arterial hypertension, and this prot

27 k-out mice that lacked PEMT showed that they were protected from atherosclerosis, diet-induced obesit

28 Such critical functions need to be protected from attack by pests and pathogens or from

29 NOD mice treated with AZD1480 were protected from autoimmune diabetes, and diabetes wa

30 l edge states within the inverted phase that are protected from backscattering by an emergent spin sy

31 ZDF rats with global deletion of CB1R are protected from beta-cell loss, hyperglycemia, and ne

32 ges are held away from the interface and can be protected from bimolecular recombination.

33 tic Ppp1r3b upon long-term fasting (12-36 h) were protected from blood ketone-body accumulation, unli

34 e offspring of exercised C57BL/6J-mouse dams were protected from body weight gain and NAFLD in adulth

35 cient in the double-stranded DNA sensor AIM2 are protected from both subtotal body irradiation-induce

36 engineered to express kinase-deficient IRAK4 were protected from both chemical (pristane-induced) and

37 uprooting, hurricane-induced mortality, and were protected from breakage during Hugo but not Maria.

38 Hereby, Asm(-/-) animals are protected from bronchial asthma, which possibly offe

39 ly and provides insight into how human cells are protected from bystander damage by the cell surface

40 absence of target, aptamer coated particles are protected from capture on the test line and are inst

41 MuRF1(-/-) mice were protected from cardiac atrophy and exhibited no red

42 v integrin is depleted in PDGFRbeta(+) cells are protected from cardiotoxin and laceration-induced sk

43 us for the ancestral C allele (EE genotype), are protected from cardiovascular disease (CVD), showing

44 Germ-free mice are protected from CCM formation, and a single course of

45 isplay elongated mitochondria in neurons and are protected from cerebral ischemic injury.

46 ntial sH1N1 influenza virus-infected ferrets were protected from challenge with a novel H1N1 influenz

47 Immune-competent recipients were protected from challenge with leukemia if they were

48 operties, are strikingly enriched in AT, and are protected from chemotherapy by the GAT microenvironm

49 Mice lacking IL-33 were protected from chronic ACD and its skin cancer sequ

50 e we demonstrate that mice deficient in Irp2 were protected from cigarette smoke (CS)-induced experim

51 ed cattle), despite vaccinated cattle having been protected from clinical disease.

52 TTPDeltaARE mice were protected from collagen antibody-induced arthritis,

53 Neonates are protected from colonizing bacteria by antibodies sec

54 The host is protected from complement-mediated damage by several

55 eficient mice, but not RIPK3-deficient mice, were protected from ConA-induced liver injury.

56 operties rivalling suspended graphene, while being protected from contamination and mechanical damage

57 transparent triage system, and 3) providers are protected from contracting the disease.

58 ssemia trait carriers displayed lower TC and were protected from coronary artery disease (CAD); (ii)

59 ionic doping, and the LLMO cathode materials are protected from corrosion induced by organic electrol

60 During interphase, BUB3 is protected from CRL4-mediated degradation by associati

61 al iron chelator or mice fed a low-iron diet were protected from CS-induced COPD.

62 ytochrome c oxidase, which have reduced COX, were protected from CS-induced pulmonary inflammation an

63 ype and Pkd2/Ift88 double-knockout mice that are protected from cyst growth served as controls.

64 Endothelial cells and isolated lungs were protected from cytotoxin-induced death by stimulati

65 del, we report here that TRPV4-knockout mice were protected from D. farinae-induced airway remodeling

66 dling development, the shoot apical meristem is protected from damage as the seedling emerges from so

67 letion had lower cardiac KLF5 expression and were protected from DbCM.

68 type in the heart during EAM, IL-4(-/-) mice were protected from DCMi like DeltadblGATA1 mice, and eo

69 effect of IL-5, as IL-5TgDeltadblGATA1 mice were protected from DCMi, whereas IL-5(-/-) mice exhibit

70 MGL(-/-) mice were protected from DDC-induced biliary fibrosis and inf

71 The mechanisms by which CGIs are protected from de novo methylation remain elusive.

72 ly assumed that mRNAs undergoing translation are protected from decay.

73 Other FNWs were protected from decomposition with an amorphous carb

74 or studies have assumed that decoy-bound TFs are protected from degradation, and in this case decoys

75 e plant cell wall and they are acetylated to be protected from degradation by glycoside hydrolases.

76 ergen extracts: The protein/DNA molecule can be protected from degradation, higher local concentratio

77 In LC, ZO-2 is protected from degradation by association to 14-3-3 p

78 ied C19ORF66 and reveal that this transcript is protected from degradation by its 3' untranslated reg

79 bstrate of the Lon-type protease and that it is protected from degradation by Nfs1, the sulfur donor

80 DNA are captured, and the extracellular DNA is protected from degradation.

81 terious interaction with the Bam complex but was protected from degradation and eventually inserted i

82 subunit, the palmitoylated CaV2.2 I-II loop was protected from degradation, although oligoubiquitina

83 blueberry polyphenols complexed with protein were protected from degradation during 16weeks at 4 degr

84 Growing microtubules are protected from depolymerization by the presence of a

85 ry diseases, ICER/CREM-deficient B6.lpr mice are protected from developing autoimmunity.

86 As a result, mice lacking ATDC are protected from developing PDA.

87 Ormdl3 knockout mice were found to be protected from developing allergic airways disease an

88 nfected with this genetically modified virus were protected from developing ASF after challenge with

89 nsferred with HDAC10(-/-) but not wild Treg, were protected from developing colitis.

90 rexpressing Sirt6 transgenic (Tg.Sirt6) mice were protected from developing obesity and insulin resis

91 Functionally, TICAM2 deficient mice were protected from developing severe systemic inflammat

92 r, mice with liver-specific deletion of MPC2 were protected from development of NASH on this diet.

93 red with WT, we demonstrated that Parp1(-/-) were protected from dextran-sulfate sodium-induced colit

94 lar to those of WT STZ mice, TLR4KO STZ mice were protected from diabetes-induced bladder hypertrophy

95 mice and mice treated with a P2X7 inhibitor were protected from diabetes-induced TNF-alpha, IL-1beta

96 DBim(-/-)) mice developed less insulitis and were protected from diabetes.

97 p66Shc that is not acetylatable on lysine 81 are protected from diabetic oxidative stress and vascula

98 more, adipose-specific Dnmt3a knock-out mice are protected from diet-induced insulin resistance and g

99 ice show an augmented energy expenditure and are protected from diet-induced obesity and insulin resi

100 Mice lacking LR11 are protected from diet-induced obesity associated with

101 llenged with high-fat diet (HFD), IRMOE mice are protected from diet-induced obesity.

102 Stk25(-/-) mice were protected from diet-induced liver steatosis accompa

103 , we found that mice lacking hepatic ZFP36L1 were protected from diet-induced obesity and steatosis.

104 heir severe fatty liver, the transgenic mice were protected from diet-induced obesity and type 2 diab

105 te-specific TFEB overexpression (Adipo-TFEB) were protected from diet-induced obesity, insulin resist

106 Notably, Il5 (Tg) /CD300f (-/-) were protected from diet-induced weight gain and glucose

107 vial fluids after injury and Cxcr3(-/-) mice being protected from disease development.

108 ted after vaccination, all immunized animals were protected from disease and death following lethal c

109 with a high-dose, oral-nasal virus challenge were protected from disease, whereas all controls died.

110 the pericentromeric heterochromatin regions are protected from DNA demethylation independently of EH

111 Moreover, DRP1-deficient mice were protected from Dox-induced cardiac damage, strongly

112 tatic and dynamic disorder, but how carriers are protected from efficient scattering with charged def

113 By 6 months, Mmp28(-/-) mice were protected from emphysema.

114 Cells are protected from endoplasmic reticulum stress through

115 n of IL-37 (IL-37tg mice) with intact IL-1R8 were protected from endotoxemia, IL-1R8-deficient IL-37t

116 docannabinoid 2-arachidonoyl glycerol (2-AG) are protected from enteric infection by Enterobacteriace

117 ting becomes viable when a quantum state can be protected from environment-induced error.

118 lergic airways disease in which treated mice were protected from eosinophilia, goblet cell hyperplasi

119 c epithelium of trpml1-null mice, where they are protected from eradication therapy.

120 Eventually, ERK inhibitor treated cells are protected from ETO-induced nuclear envelope (NE) rup

121 ic core of cyclodextrins and therefore, they are protected from exogenous stress.

122 Furthermore, CD43(-/-) mice were protected from experimental autoimmune encephalomye

123 n accordance, mice lacking mEF-G1 in T cells were protected from experimental autoimmune encephalomye

124 The fetus is thought to be protected from exposure to foreign antigens, yet CD45

125 ng facilities with large elderly populations are protected from extreme heat (for example through bac

126 genetic or pharmacological inhibition of BID are protected from Fas-mediated impairment of mitochondr

127 ogen synthesis, and as a result, CD36Tg mice were protected from fasting hypoglycemia.

128 tion-induced fibrosis whereas MDR2(-/-) mice are protected from fibrosis by the proteasome inhibitor

129 ule cell-specific amphiregulin knockout mice were protected from fibrosis after ischemia-reperfusion

130 Mice deficient in IRAK-M were protected from fibrosis and displayed a diminished

131 less nitrogen after 64 years than plots that were protected from fire.

132 e find that this smaller stem cell reservoir is protected from full depletion by an increase in quies

133 tive Staphylococcus aureus cells appeared to be protected from GA by an increased formation of nm-sca

134 mice, the tumor suppressor isoform of CUGBP1 is protected from Gank-mediated degradation.

135 proliferating cells in plant meristems must be protected from genome damage.

136 Stem cells need to be protected from genotoxic and proteotoxic stress to ma

137 to wild-type mice, Cd74-deficient mice also were protected from glomerular injury and ensuing activa

138 restoration, and that regeneration needs to be protected from grazing.

139 female neuron-specific SOCS3 knock-out mice were protected from HCD-induced obesity.

140 stimulation, whereas Bex1 gene-deleted mice are protected from heart failure-promoting insults.

141 ho resolved clinically significant PH (CSPH) were protected from hepatic decompensation.

142 Finally, HFD-fed AdSod2 KO mice were protected from hepatic steatosis, adipose tissue in

143 pe littermate control (M-JAK2(+/+)) mice and were protected from HFD-induced systemic insulin resista

144 le of binding leukocyte alphaMbeta2-integrin were protected from HFD-induced weight gain and elevated

145 expression of thermogenic genes in BAT, and are protected from high-fat diet-induced obesity and dev

146 the ovalbumin asthma model, Asm(-/-) animals were protected from high disease activity and showed bet

147 homologous residue Thr1150 (InsrT1150A mice) were protected from high fat diet-induced hepatic insuli

148 Furthermore we found that male AhRR Tg mice were protected from high-dose TCDD-induced lethality ass

149 pontaneous NASH whereas AEG-1(DeltaHEP) mice were protected from high-fat diet (HFD)-induced NASH.

150 show that GPR30 knockout (GPRKO) female mice were protected from high-fat diet (HFD)-induced obesity,

151 lacking both LDL receptor (LDLR) and Arhgef1 were protected from high-fat diet-induced atherosclerosi

152 note, adipocyte-specific gp130 knockout mice were protected from high-fat diet-induced hepatic steato

153 CreERT mice on different genetic backgrounds were protected from high-fat/ streptozotocin (STZ)-induc

154 Moreover, when Dual-CAR T cells were protected from HIV infection through expression of

155 nside a vacuole in the host cytosol where it is protected from host cytoplasmic innate immune respons

156 cative DNA damage following NS depletion and are protected from hydroxyurea-induced damage by NS over

157 The data show that telomeres are protected from hyper-resection through the repressio

158 Full-body Galpha(z)-null mice are protected from hyperglycemia and glucose intolerance

159 Panx1 knockout mice (Panx1(-/-)) were protected from hypersensitivity in two sciatic nerv

160 ike-overexpressing transgenic (Sike-TG) mice are protected from hypertrophic stimuli.

161 mmune privilege, in which immunogenic tissue is protected from immune attack.

162 Ptpn22(-/-) mice were protected from immune complex-mediated arthritis, i

163 challenged with enteropathogenic E coli, and were protected from immune infiltration, crypt dropout,

164 Chimeric FABP4(-/-) mice with WT bone marrow were protected from increased mortality seen in chimeric

165 over, retrogenic mice expressing these Tregs were protected from induction of EAE by the appropriate

166 These findings help explain why CF mice are protected from infection and nominate ATP12A as a po

167 carrying neutralizing antibodies against MuV are protected from infection by batMuV.

168 l mice that lack the capacity to produce IgG are protected from infection with the enteric pathogen e

169 ve neutralizing antibodies against MuV might be protected from infection by BMV.

170 e infected, while the 6 PrEP-treated animals were protected from infection.

171 ically reduced, and the periodontal ligament was protected from inflammation-induced destruction.

172 igh-dose UV B radiation, IR/IGF-1R(MKO) mice were protected from inflammation, whereas controls devel

173 As a consequence, lungs were protected from inflammatory injury at times of day

174 nism by which hosts with allergic asthma may be protected from influenza morbidity.

175 ned less body weight than wild-type mice and were protected from insulin resistance and hepatic steat

176 aRIIB globally or selectively in endothelium were protected from insulin resistance as a result of th

177 We found that the newly formed memory was protected from interference when it shared a common

178 A primary means by which the intestine is protected from its microbiota is via multi-layered mu

179 S1pr3(-/-)mice are protected from kidney IRI, because DCs do not mature

180 However, S1pr3(-/-) BMDC-pretreated mice were protected from kidney IRI.

181 r expressed on myeloid cells 2 (Trem2 (-/-)) were protected from LCMV-induced hepatitis and showed im

182 with genetic mitochondrial CaMKII inhibition are protected from left ventricular dilation and dysfunc

183 Vaccinated mice are protected from lethal challenge with diverse influen

184 ater column, seabirds, reptiles, and mammals are protected from lethal oiling at the surface, and mic

185 the CDV fusion and attachment glycoproteins were protected from lethal CDV challenge, whereas all an

186 usion protein-expressing recombinant viruses were protected from lethal CDV challenge.

187 Ns in response to B. pertussis infection and were protected from lethal infection by increased type I

188 Nlrp12(-/-) mice are protected from lethality during IAV infection and sh

189 Fetal/neonatal progenitors may therefore be protected from leukemic transformation because they a

190 role for SARM in pyroptosis, Sarm1(-/-) mice were protected from lipopolysaccharide (LPS)-stimulated

191 Plcbeta2-, Plcbeta3-, or Rac1-deficient mice were protected from lipopolysaccharide-induced lung inju

192 Lcn2(-/-) mice were protected from liver fibrosis caused by either etha

193 Furthermore, IRAK1 knockout mice were protected from LPS-induced PTB, which was seen in w

194 s erythematosus (SLE), and mice lacking Irf5 are protected from lupus onset and severity, but how IRF

195 We show that aging mice lacking FGFR4 are protected from LVH.

196 etection (SEC/MS) reveals the oligomers that are protected from lysis due to their tight association

197 malaria pathogenesis because EphB2(-/-) mice were protected from malaria-induced liver fibrosis despi

198 metabolic diseases, some people with obesity are protected from many of the adverse metabolic effects

199 Target genes are protected from MazF activity by recoding the gene se

200 ommunities accumulate on teeth in areas that are protected from mechanical abrasion forces.

201 ad a high conventional T/Treg cell ratio and were protected from melanoma challenge.

202 AC6 and have potent suppressive T(reg) cells are protected from microbiota-induced accelerated weight

203 Accordingly, Aag(-/-) cells are protected from MMS-induced NAD(+) depletion and glyc

204 ce vaccinated with the group 1 HA mini-stems are protected from morbidity and mortality against letha

205 (H5N2), A(H5N8), A(H6N1), or A(H7N9) viruses were protected from mortality and showed drastically red

206 Later-occurring recombination events are protected from Mph1-mediated dissociation by synapsi

207 The EFVs are stable, the cells inside them are protected from multiple stresses, and large numbers

208 n activating gene 1-deficient (Rag1-/-) mice were protected from NEC and transfer of intestinal lymph

209 ent in the adhesion molecule integrin alpha4 were protected from necrotic core expansion.

210 SCFA-treated diabetic mice were protected from nephropathy, but not in the absence

211 iable region 1 yielded vector particles that were protected from neutralization by natural antibodies

212 cine candidate to demonstrate that offspring are protected from nHSV following maternal immunization.

213 he system was used to demonstrate that dsRNA is protected from nuclease digestion by virus-induced me

214 nt biological catalysts of H2 oxidation, can be protected from O2 damage upon integration into a film

215 cantly reduced intestinal fat absorption and are protected from obesity, hepatic steatosis and insuli

216 immunoglobulin G (IgG) receptor FcgammaRIIB are protected from obesity-induced hypertension.

217 support the hypothesis that HFD female mice are protected from obesity-induced insulin resistance du

218 sue inflammation, cadherin-11-deficient mice were protected from obesity-induced glucose intolerance

219 Mice deficient in B cells were protected from obesity-induced hypertension.

220 Mice deficient in FcgammaRIIB in endothelium were protected from obesity-induced hypertension.

221 Rad51ap1-deficient mice were protected from oncogene-driven spontaneous mouse ma

222 phene channels prior to PE-ALD, the graphene is protected from oxidation enabling BN/Al(2)O(3) layers

223 U L3 XAS indicates that this U(V) species is protected from oxidation likely incorporated into oct

224 embly, and we also explain how these enzymes are protected from oxidative inactivation.

225 is Mast cell-deficient mice (Kit(W-sh/W-sh)) were protected from P. gingivalis-induced alveolar bone

226 Constitutive Lpar1 null mutant mice are protected from partial sciatic nerve ligation (PSNL)

227 ec9a(-/-) mice deficient in DC cross-priming are protected from persistent immune-mediated myocardial

228 istological indices, knockout-knockout pairs were protected from PH, whereas knockout mice in WT-knoc

229 Within these autophagosomes, the bacteria are protected from phagocytic killing, thus providing an

230 cient mice made diabetic with streptozotocin were protected from physiological and structural indices

231 Moreover, TRPV4 gene-KO mice were protected from Piezo1 agonist- and pressure-induced

232 Conversely, areas exposed to high SS are protected from plaque development, but the mechanism

233 ked as the most upregulated in vaccinees who were protected from Plasmodium falciparum infection.

234 KEX1-immunized macaques were protected from Pneumocystis pneumonia, compared wit

235 ice on a beta3 integrin-deficient background were protected from podocyte injury.

236 Most circulating SAA is protected from proteolysis and misfolding by binding

237 y degraded in vivo, antitoxin bound to toxin is protected from proteolysis, preventing release of act

238 low amplitudes of physiological force, PAK2 is protected from proteolysis, thereby ensuring cell sur

239 importance, mice that were deficient in MCU were protected from pulmonary fibrosis.

240 mice had increased macrophage apoptosis and were protected from pulmonary fibrosis.

241 n contrast, SMPDL3b overexpressing podocytes were protected from radiation-induced cytoskeletal remod

242 d CCHFV-specific B- and T-cell responses and were protected from reinfection.

243 SIRP-alpha(mut) mice were protected from renal IR compared with WT animals, d

244 Ksp-CreERT2(+) mice crossed to p62(-/-) mice were protected from renal tumor development.

245 develop high intraocular pressure (IOP) but are protected from retinal ganglion cell (RGC) dysfuncti

246 rains, we have determined whether cattle can be protected from rinderpest by inoculation with vaccine

247 Dogs vaccinated with WCA were protected from RMSF, whereas those receiving RCA de

248 wever, only the animals given wild-type PPRV were protected from RPV challenge.

249 Further, irradiated C1q-Flox mice were protected from RT-induced microglial activation and

250 NSAID users were protected from SAB (OR = 0.78, 95% CI 0.56-1.10), w

251 (sg/sg) bone marrow chimeric mice (C57BL/6J) were protected from Salmonella-induced intestinal fibros

252 ingle knockout IL-4(-/-) or IL-13(-/-) mice, were protected from Schistosoma-induced PH, with decreas

253 g for more than 40 weeks, and these macaques were protected from several infectious challenges with S

254 he baboons in the P. knowlesi-only group and were protected from severe anemia.

255 an inoculum containing Nod1 x 2(-/-) T cells were protected from severe graft versus host disease.

256 le stress fibers, whereas contractile fibers are protected from severing.

257 le explores the question of how the active X is protected from silencing by its own Xist locus, and t

258 d that mice lacking ephrin-B2 in fibroblasts are protected from skin and lung fibrosis and that a dis

259 n of Cosmc in 50% of crypts (IEC-Cosmc(+/-)) were protected from spontaneous inflammation and partial

260 hich also shows reduced muscle strength, but is protected from stretch-induced eccentric damage with

261 ence of anti-HSV-1 antibody, vaccinated mice are protected from subsequent challenge with wild-type H

262 Vmac239 challenge, whereas only four animals were protected from subsequent intravenous SIVmac239 cha

263 In addition, mice treated with EPIT were protected from subsequent sensitization and maintai

264 ns were significantly higher in children who were protected from symptomatic malaria compared with th

265 Factor X-deficient mice were protected from systemic Acinetobacter baumannii inf

266 Five weeks after TAC, C-dnO1 mice were protected from systolic dysfunction (assessed by pr

267 Moreover, these mice were protected from systolic dysfunction, hypertrophy, l

268 ll-dependent antiviral immune responses, and are protected from T cell-mediated autoimmunity and allo

269 graphite die and the graphite punches, which are protected from the alumina fiber film by a graphite

270 caspases promote proliferation and how cells are protected from the potentially harmful action of apo

271 cetylated STAT3 emerged upon HDAC inhibition was protected from the proteasome-mediated degradation o

272 e KDKE(4A)-SYA- and KDKE(4A)-inoculated pigs were protected from the challenge, because no KDKE(4A)-S

273 Intriguingly, Spns2(-/-) mice were protected from the development of experimental auto

274 e, OVA323-339, mice that received MPO409-428 were protected from the development of humoral and cell-

275 that lack HMGB1 in the intestinal epithelium were protected from the development of lung injury, conf

276 receptor Fcgamma receptor IIB (FcgammaRIIB) were protected from the disorder.

277 As expected, HCR rats were protected from the HFD.

278 Mice lacking RANKL in osteocytes were protected from the increase in osteoclast number an

279 ed a high level of inhibitory antibodies and were protected from the infection.

280 Similarly, Foxp3creInsrfl/fl mice were protected from the metabolic effects of aging, but

281 nd mice lacking the endogenous receptor CD36 were protected from the neuroinflammatory and BBB permea

282 epleted mice in contrast to nondepleted mice were protected from the stress effects measured by light

283 ose tissue metabolism in Asxl2DeltaLysM mice were protected from the suppressive effects of HFD, a ph

284 r niche (PVN) of distant tissues, where they are protected from therapy by vascular endothelium.

285 e and C5aR were pharmacologically inhibited, were protected from these adverse effects and consequent

286 The offspring of mothers with T1D are protected from this process.

287 predicted that platelet-COX-1-ko mice would be protected from thrombosis, forming less pro-thromboti

288 Instead, the HPRT locus appeared to be protected from transposon integration.

289 dramatically altered: encapsulated peptides are protected from trypsin hydrolysis, whereas physicoch

290 nfection, genitally infected IL-10(-/-) mice were protected from tubal pathologies and infertility, w

291 Most normal and tumor cells are protected from tumor necrosis factor alpha (TNFalpha

292 ice lacking nCDase treated with azoxymethane were protected from tumor formation.

293 Surprisingly, DNase-accessible euchromatin is protected from UV, while lamina-associated heterochro

294 tion is MALT1-independent, MALT1(PD/PD) mice are protected from vascular edema induced by either pass

295 Cancer cells are protected from VC-mediated cell death when co-cultur

296 VSIV causes disease and how healthy tissues are protected from VSIV-based therapies, it is crucial t

297 Medullary hyperosmolarity is protected from washout by countercurrent exchange and

298 ate the mechanism(s) by which Pemt(-/-) mice are protected from weight gain and insulin resistance.

299 carrying the gene for the human PV receptor are protected from wild-type PV when immunized with the

300 deficiency in complement C3 or C3a receptor were protected from WNV-induced synaptic terminal loss.