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1  plasma, whereas kallikrein inhibition alone was sufficient to abolish TG in FXII- or FXI-deficient p
2  cells via the canonical PD-L1-PD-1 axis and were sufficient to accelerate tumorigenesis, even in the
3  magnetic field gradient within a space that is sufficient to accommodate an organ-stage mouse embryo
4 f the disadvantages conferred by other fates is sufficient to account for the observed rate of return
5 r dynamics and coupled cluster calculations, is sufficient to accurately reproduce experimental guest
6                                  Sensitivity was sufficient to accurately analyze enrichments.
7  knockdown and MSI1 and PCBP2 overexpression are sufficient to activate many photoreceptor-specific e
8 an enzymes show that PCNA and its loader RFC are sufficient to activate the MutLgamma endonuclease.
9                                    IL-1alpha is sufficient to activate cultured fibroblasts and prima
10 overexpression of ST6Gal-I in the Suit2 line was sufficient to activate EMT pathways.
11                                           OI was sufficient to activate NRF2 in macrophages; however,
12                         A few lymphoma cells were sufficient to activate the angiogenic switch in lym
13 ow that altering foraging strategies may not be sufficient to adapt to a changing ocean.
14  cured a reasonable fraction of adults would be sufficient to advance to the clinic.
15 he missense variant identified in this study is sufficient to affect amelogenesis causing AI, but not
16            Whether oligodendrocyte pathology is sufficient to affect brain-wide structure and account
17 g enzymes in CD16a-expressing effector cells is sufficient to affect N-glycan composition.
18               Further, mistargeting cen mRNA is sufficient to alter cognate protein localization to c
19 at shifted neuronal post-mitotic development were sufficient to alter glial layering, indicating an i
20 h rhbeta-Gal, followed by enzyme withdrawal, is sufficient to augment beta-Gal activity levels in GM1
21 roteasome-nondegradable c-Myc protein mutant was sufficient to avoid glutathione depletion and rescue
22                          We found that Spire is sufficient to bind barbed ends and retain pointed end
23 c activity of Vav2 below specific thresholds is sufficient to block skin tumor initiation, promotion,
24         Strikingly, increasing CFTR activity was sufficient to block EMT.
25 educed levels of the encoded proteins, which is sufficient to bring about disorganized mitochondrial
26         Preventing Pli1-mediated SUMO chains was sufficient to bypass the need for anchorage to NPCs
27 umption of linear combination, which may not be sufficient to capture the coregulation patterns.
28 his is interesting and important, it may not be sufficient to carry us to a place of truly new insigh
29 , which is richly expressed in type II UBCs, is sufficient to cause ataxia.
30 suggesting that aberrant spontaneous release is sufficient to cause disease in humans.
31 e show that targeting signaling in myofibers is sufficient to cause significant increases in muscle m
32 cal substitutions into the ancestral protein is sufficient to cause strong tetramerization by creatin
33 wn cells, and modulation of their regulation is sufficient to cause the biofilm hyperinfectivity phen
34 specific activation of Tak1 in the brainstem was sufficient to cause an impairment in BA function in
35                   Chronic gestational IL-17A was sufficient to cause ASD-like phenotypes early and pe
36       Poly(I:C) or type I IFN administration was sufficient to cause breakthrough alloimmunization, w
37 ly knowing that one could alleviate scarcity is sufficient to change behavior.
38 induction of LTD at the BLA-GC input in vivo was sufficient to change the hedonic value of a taste st
39 ges generates a diversity of structures that are sufficient to characterize a minor conformational st
40 izen movements alone-absent external support-are sufficient to check newly emboldened autocrats.
41 on of a testis-specific transcription factor is sufficient to co-opt somatic transcriptional machiner
42 strate that hydrostatic pressures of 450 MPa are sufficient to completely dissociate the Bfr 24-mer i
43 g, disruption of a single transporter, CycA, was sufficient to completely overcome the repression of
44                    Genetic knockout of Sting was sufficient to completely prevent neurodegeneration a
45 monstrated that vHipp input photostimulation is sufficient to condition a flavor preference, while it
46 omain of PDE3A in cancer cells lacking PDE3A is sufficient to confer sensitivity to DNMDP, and substi
47 ance studies revealed that while this moiety was sufficient to confer binding to FGF14, altering the
48          Transformation of Fielder with WTK2 was sufficient to confer Pgt resistance.
49  that are linked to atrazine degradation and were sufficient to confer resistance in experimental was
50 , Mg) and micronutrients (Fe, Zn, Co, Mn, I) were sufficient to contribute to daily dietary mineral i
51 ndependent/IL-5- and CCR3-dependent pathways are sufficient to control filarial adult infection via a
52 cellular vesicles (EVs) from young astrocyte were sufficient to convey support for oligodendrocyte di
53 sing oncogenic RasV12 in untransformed cells is sufficient to decrease pHlys.
54           Here we show that MEIS1 expression is sufficient to decrease proliferation and metastasis o
55                  A single 30 ms step to 0 mV was sufficient to deplete the membrane-proximal vesicle
56 th an uncertainty smaller than 0.5 nm, which is sufficient to detect sub-monolayer coverage by bound
57 ormats of medial temporal lobe and neocortex are sufficient to determine both, memory contents and su
58  The inhibition of mitochondrial respiration was sufficient to diminish galactose-1-phosphate levels
59 ut not basophil-depleted mice, and basophils were sufficient to directly enhance NMB receptor express
60 nclude that a data-driven discovery approach is sufficient to discover groups of disease-related gene
61 e that guidance of inherent migratory forces is sufficient to disperse cell clusters under physiologi
62 cell cycle, suggesting that cell cycle delay is sufficient to disrupt Ace2 asymmetry.
63               Expressing the Epe1 C-terminus is sufficient to disrupt heterochromatin by outcompeting
64 cific genetic manipulation of eya expression is sufficient to disrupt the ability of flies to sense s
65 aling for two days during kidney development is sufficient to disrupt tubule formation, and significa
66 t stem cell marker genes (GSE70696_QNPbyTAP) was sufficient to distinguish between control and GBM sa
67 e show that these high levels of amino acids are sufficient to drive HLC and HepG2 drug biotransforma
68 tiple cancer subtypes, including breast, and are sufficient to drive oncogenic transformation in vitr
69 ate that selective inhibition of BD1 domains is sufficient to drive anti-inflammatory and antiprolife
70 ogenic ectodomain to branched actin assembly is sufficient to drive cell-cell fusion.
71 nosine triphosphate (GTP) signal [12], which is sufficient to drive continuous monopolar spindle moti
72 s during stress and its synthetic expression is sufficient to drive differentiation.
73       In C2A, a divalent metal ion in site 1 is sufficient to drive its weak association with phospha
74 show that tethering DCP1A to one Tsix allele is sufficient to drive monoallelic Xist expression.
75  and PDCD1LG2 lead us to identify that GATA2 is sufficient to drive PD-L1 and PD-L2 expression and is
76 hat overexpression of a forced E2A homodimer is sufficient to drive robust neural commitment in pluri
77  that the hyper-induction of Rap1 in the PFC is sufficient to drive stress-relevant cognitive and syn
78 r flavin reduction or the binding of NADP(H) is sufficient to drive the FAD to the in conformation.
79                        Elevation of CSF-CCL2 was sufficient to drive ChP immune cell recruitment, act
80          Furthermore, overexpression of Dlx5 was sufficient to drive T-ALL in mice by directly activa
81 p-regulated, and ectopic Igf2 overexpression was sufficient to drive tumorigenesis in Ptch1 (+/-) GNP
82              We propose that uncertainty may be sufficient to elicit learning differences in paranoid
83 ted that expression of the mutant DMPK 3'UTR is sufficient to elicit these features of DM1.
84 r endothelial injury, Wnt activation in MVPC was sufficient to elicit an emphysema-like phenotype cha
85 ctivation of VLM(CA) projections to the pPVT was sufficient to elicit robust feeding behavior in well
86                          Two administrations were sufficient to elicit dose-dependent gB-binding and
87 en is the cytosolic Malic Enzyme (ME1), that is sufficient to enable survival and proliferation of CI
88 mount of endogenous ET that they did produce was sufficient to enable ripening initiation and this co
89 gues demonstrates that mutant YAP expression is sufficient to enhance tumor cell dissemination in zeb
90 t cells, rescuing fork degradation might not be sufficient to ensure fork repair.
91 roved more than twofold and this improvement is sufficient to establish the dynamics of cell growth a
92                                         LuxR is sufficient to evict H-NS from promoter DNA in vitro,
93          Notably, Tlr9 deficiency in B cells was sufficient to exacerbate nephritis while extinguishi
94                            These differences are sufficient to explain differential recruitment of di
95 raction) parameters (dynamic dual box model) is sufficient to explain in-situ spectroscopic ellipsome
96 bsence of saccade planning or a spatial cue, is sufficient to explain the direction bias observed in
97            We show that this null hypothesis is sufficient to explain the lack of detected homologs o
98 lations predicted that a geometry-based rule is sufficient to explain the microtubule (MT) organizati
99 echanism that involves differential motility is sufficient to explain the spontaneous patterning of t
100 del demonstrated the separation of TF inputs is sufficient to explain these findings.
101 bution of I (leak), I (ir), I(m) , and I (h) was sufficient to explain the electrotonic dendritic str
102 unclear whether these known biological rules were sufficient to explain pattern formation.
103 ibition dominance nor broad inhibition alone were sufficient to explain the experimental findings; in
104     We demonstrate that <25% PrP suppression is sufficient to extend survival and delay symptoms in a
105  We show that although somatic disinhibition is sufficient to form place fields, dendritic inhibition
106  that activity-independent size fluctuations are sufficient to fully diversify glutamatergic synaptic
107                      A single dose of rFVIIa was sufficient to fully prevent the development of milde
108  a propensity for a neutral binding surface, are sufficient to generate a classifier able to identify
109 ctivity-independent size fluctuations, which are sufficient to generate a full repertoire of synaptic
110 ty and whether its local excitatory circuits are sufficient to generate epileptiform activity.
111 ated that antigen-experienced CD4(+) T cells are sufficient to generate protection.
112 tors combining bacterial GLIC and alpha3GlyR was sufficient to generate receptors with reduced conduc
113  model demonstrating growth factor signaling is sufficient to guarantee this robustness and which ant
114 ed in a reported kinase-dead form, LKB1K78I, was sufficient to hamper tumor aggressiveness and metast
115 on, we show that reducing LSD1 in these mice is sufficient to highly exacerbate tau-mediated neurodeg
116  findings suggest that a single biopsy would be sufficient to identify the important genetic drivers
117                                     Two SNPs were sufficient to identify 'Nebbiolo' from 1157 genotyp
118 antly, ectopic expression of this subcircuit was sufficient to imbue trunk crest with the ability to
119 y within host tissues and whether expression is sufficient to impact the growth kinetics of virulence
120 CD79B ubiquitination or Cbls ligase activity is sufficient to impede BCR-mediated antigen processing
121 re we show that membrane-reconstituted IrtAB is sufficient to import mycobactins, which are then redu
122 s of LPI, and the in vivo knockdown of GPR55 was sufficient to improve liver damage in mice fed a hig
123 Isolated exosomes from heat-shocked utricles were sufficient to improve survival of hair cells expose
124 ractions alone, in absence of DG activation, are sufficient to increase adult hippocampal astroglioge
125            Furthermore, G1 cell cycle delays are sufficient to increase chemotherapeutic resistance i
126 RH in primary human biliary epithelial cells is sufficient to increase cell proliferation and produce
127 overexpression of dcap-1 in neurons of worms is sufficient to increase lifespan through the function
128 amate release onto magnocellular neurons and was sufficient to increase blood pressure.
129 ory cortex to parietal cortex and found this was sufficient to increase minimum behavioral integratio
130 By contrast, enhanced muscle IL-13 signaling was sufficient to increase running distance, glucose tol
131 ally, we showed that interleukin-4 treatment was sufficient to increase the number of MGCs in AT, whe
132 onate pathway enzyme GcpE, and these changes were sufficient to increase the growth rate of 10403S to
133 of function mutations in Kras (G12D or G12V) are sufficient to induce brain arteriovenous malformatio
134                    Furthermore, Sspo defects are sufficient to induce oxidative stress and neuroinfla
135 ation of endogenous MERVL or miR-344-2 alone is sufficient to induce 2CLCs with activation of 2C gene
136 ual proximity of molecular or atomic species is sufficient to induce a finite scalar nuclear spin-spi
137                     Nevertheless, DNA damage is sufficient to induce activation of canonical p53 targ
138 ne whether the presence of a social stimulus is sufficient to induce activation of PVH-OT neurons, we
139 atory stimulus, mechanical compression alone is sufficient to induce an asthma-like molecular signatu
140 e with the PHOSPHO1 substrate phosphocholine is sufficient to induce cold tolerance, thermogenic gene
141 plasma membrane and synthetic membranes, and is sufficient to induce extensive membrane tubulations.
142                                   EpiGo-KRAB is sufficient to induce genomic clustering and de novo h
143 gs reveal that chronic p16(INK4a) expression is sufficient to induce hyperplasia through Wnt-mediated
144 , muscle fiber-specific activation of mTORC1 is sufficient to induce molecular signatures of sarcopen
145 ion-competent Orsay virus RNA1 segment alone is sufficient to induce most of the IPR genes in a manne
146 hat the loading capacity of the nanoclusters is sufficient to induce neuronal activation.
147 Ectopic activation of SHR and SCR in legumes is sufficient to induce root cortical cell division.
148 acological activation of m1 and m3 signaling is sufficient to induce the castration-resistant growth
149 )(y)Co(1-y)O(2) suggest that 5% substitution is sufficient to induce the kinetic limitation and that
150                   Moreover, type I IFN alone was sufficient to induce cytokine and chemokine producti
151  non-asthmatic donors, compression by itself was sufficient to induce inflammatory, late repair, and
152 l 50- and 385-Hz oscillating magnetic fields was sufficient to induce intracellular protease release,
153 ted from late-stage wounds into healthy skin was sufficient to induce itching in a manner dependent o
154   Selective stimulation of cholinergic axons was sufficient to induce LTP, which was prevented by che
155                           Loss of Myoc alone was sufficient to induce phenotypes identified in mouse
156         We found that overexpressing fcrX(+) was sufficient to induce swarming, luminescence and iron
157 ion of mitochondrial reactive oxygen species were sufficient to induce necroptosis, a caspase-indepen
158 n of the nucleotides (p)ppGpp, which we show are sufficient to inhibit protein synthesis in vivo.
159 ystemic or neutrophil-specific Akt1 deletion is sufficient to inhibit metastasis of Akt-proficient tu
160 yaluronic acid precursor UDP-glucuronic acid is sufficient to inhibit several mesenchymal-like proper
161 e activated during a previous bout of torpor is sufficient to initiate the key features of torpor, ev
162  while the C-terminal coiled-coil CC2 domain is sufficient to interact with RACB and to fulfill a fun
163 nerate fluences exceeding 10 mJ/cm(2), which is sufficient to irreversibly inactivate most common pat
164  the total edge length and number of loops - are sufficient to largely determine network exploration
165                   While depleting CDK4 and 6 was sufficient to limit proliferation in specific resist
166     Inhibition of VASH1 or activation of TTL is sufficient to lower stiffness and speed relaxation in
167                                 This pathway is sufficient to maintain continuous rod function and su
168                    Ingestion of 30 g protein is sufficient to maximize MyoPS rates during recovery fr
169  MyoPS rates (%/h) compared with 0 g PRO and was sufficient to maximize MyoPS rates after endurance e
170 t force-independent relief of autoinhibition is sufficient to mediate their tight interaction.
171 on of human respiratory epithelial cells and was sufficient to mediate the entry of HCoV-HKU1 into no
172 ntractile activity such that oxygen delivery is sufficient to meet metabolic demand.
173 ng infection and when expressed ectopically, was sufficient to mimic both the reorganization and bleb
174  connexin 43 or by disrupting lactate efflux was sufficient to mimic the effects of stress on long-te
175 ate that this level of cross-linking density is sufficient to model the architecture of the 19-subuni
176 microbiomes into genetically identical hosts was sufficient to modulate EAE susceptibility and system
177 ality of only two PS linkages in the DNA gap was sufficient to modulate RNase H1 cleavage patterns an
178                                 Shifts in pH were sufficient to modulate L. plantarum tolerance to ri
179 hibition in the ventral tegmental area (VTA) is sufficient to normalize behavior.
180 kness; a slip length of a few nanometers may be sufficient to observe alignment.
181 e amino acid to experimentally infected mice was sufficient to overcome the metabolic constraints imp
182 hT in the dorsomedial striatum of adult mice was sufficient to phenocopy maladaptive eating behaviors
183 e DNA polymerase to variously structured DNA is sufficient to predict the complex genomic behaviour o
184 ms associated with chromophore conformation, was sufficient to predict the lambdamax of Sws2 photopig
185 cient mice, physiologic levels of murine Epo were sufficient to preferentially expand EpoRm-CAR T cel
186 ch capacities for colonization and gene flow were sufficient to preserve genetic diversity despite re
187 onstitutively active version of Yap, Yap5SA, is sufficient to prevent cell cycle exit and to prolong
188 ease of acetylcholine (ACh) from motoneurons is sufficient to prevent changes induced by denervation.
189 MON-DNJ) capable of inhibiting Glu I in vivo is sufficient to prevent death in mice infected with let
190 of lysosomal two-pore channels by PKARIalpha was sufficient to prevent global calcium release from th
191 cells, we show how four rounds of sequencing are sufficient to produce 3D maps of 36 genomic targets
192 f GABA interneurons in the mPFC of male mice is sufficient to produce antidepressant actions, and con
193       Finally, we show that LC-NE activation is sufficient to produce extinction deficits, and this i
194 ations and show that mutual inhibition alone is sufficient to produce stable domains of gene expressi
195 d that stimulus-evoked short-term depression is sufficient to produce synchronized monotonic positive
196 neurons during the critical postnatal window is sufficient to program altered mood-related behavior,
197 differences and their trade-offs with growth are sufficient to promote diversity, and suggest previou
198 ansient cooperative interclonal interactions are sufficient to promote metastasis of CL31.
199 However, prolonged low blue light treatments are sufficient to promote phototropism through reduced c
200 y, conditioned media from Grn(-/-) microglia are sufficient to promote TDP-43 granule formation, nucl
201 gy, whereby CTIP2 or SATB2 of either species is sufficient to promote a subcerebral or commissural fa
202 sociated macrophages through STING signaling is sufficient to promote antitumor immune responses in t
203 c remodeling in the absence of heart failure is sufficient to promote cancer is unknown.
204 neurons during immediate extinction training is sufficient to promote enhanced extinction memories, a
205 se of endogenous BAs in the intestinal lumen is sufficient to promote ISC renewal and drives regenera
206 n of the normal bone marrow microenvironment is sufficient to promote leukemic transformation and sur
207  engagement and activation of FZD7 signaling is sufficient to promote mesendodermal differentiation o
208 ults suggest that glutamate release from VTA is sufficient to promote reinforcement independent of co
209 n both males and females, and that estradiol is sufficient to promote risk aversion in both sexes.
210 doptive transfer into B cell-deficient mice, was sufficient to promote EAE recovery.
211  further demonstrate that inhibition of mTOR was sufficient to promote Mefv expression and pyrin infl
212 ificantly inhibited LUSC metastases and CDR1 was sufficient to promote migration and metastases.
213 ence by directly delivering IL-17a into S1DZ was sufficient to promote sociability in monogenic mutan
214 onstrated that UBASH3A deficiency in T cells was sufficient to promote T1D development.
215 lial cell (IEC)-specific NAIP-NLRC4 activity is sufficient to protect mice from shigellosis.
216 brogation of EGFR signaling in myeloid cells was sufficient to protect against nephritis.
217 icantly, the light-induced fat thermogenesis was sufficient to protect mice from diet-induced body-we
218 hat become active following HBB construction is sufficient to provide a counting mechanism for contro
219         Although exhaust fan intake sampling is sufficient to provide critical information to assess
220 ion of EDTA at concentrations of >=30 mug/ml was sufficient to provide a zinc-limited medium, resulti
221 expensive and vaginally nontoxic antibiotic, is sufficient to pull CD8 T cells to the vaginal mucosa
222                               An 80-min scan was sufficient to quantify V (T) and BP (ND) The test-re
223 s, both hematopoietic and non-hematopoietic, were sufficient to reactivate lung CD8+ TRM cells, but t
224 y 20-40 kBq in the animal, the image quality was sufficient to readily identify activity in the liver
225 ther spatially patterned Rho1 activity alone is sufficient to recapitulate all aspects of this morpho
226  reduction of progranulin in microglia alone is sufficient to recapitulate inflammation, lysosomal dy
227 e separation, together with surface effects, is sufficient to recapitulate much of the large-scale mo
228 f this domain on the microscopic scale alone is sufficient to recapitulate previously observed behavi
229 hat loss of C9orf72 from myeloid cells alone is sufficient to recapitulate the age-dependent lymphoid
230 type HSPCs to an mTOR(ECKO) microenvironment was sufficient to recapitulate aging-associated phenotyp
231 e acids activated pregnane X receptor, which was sufficient to recapitulate the phenotype observed in
232 omyosin, capping protein, cofilin, and AIP1) are sufficient to reconstitute the formation of cables t
233                  Unexpectedly, the PIC alone was sufficient to reconstitute pausing, suggesting RNAPI
234 ic oxide (NO) in the absence of immune cells was sufficient to reconstruct microbial social behavior,
235 -MS experiments and by the model of exchange are sufficient to recover correctly weighted structural
236 ent Hedgehog signaling by Gli1 gene transfer is sufficient to recover salivary function impaired by i
237 r study focuses on CENP-T, which we found to be sufficient to recruit the Mis12 and Ndc80 outer kinet
238 lly altering the timing of Hgf/Met signaling is sufficient to redirect axon targeting and disrupt the
239 er variations, not including the Pmp22 gene, are sufficient to reduce gene expression and phenocopy a
240  25% reduction of adult PV levels in the PFC was sufficient to reduce local GABAergic transmission on
241 gainst only the AipA or Asp14 binding domain was sufficient to reduce the bacterial peripheral blood
242 storation of IL-1R1 on glutamatergic neurons was sufficient to reestablish the impairments of social
243 iling reveals that precocious E93 expression is sufficient to regulate chromatin accessibility at a s
244  neurons following delay extinction training is sufficient to reinstate a previously extinguished fea
245 dministration of IL-5, a major ILC2 product, was sufficient to repress aging-associated neuroinflamma
246 ets of reactions in a signaling network that are sufficient to reproduce experimentally observed data
247 2 dysfunction and increased SR Ca(2+)-uptake are sufficient to reproduce the Ca(2+)-handling phenotyp
248 hanges in voltage sensor activation kinetics are sufficient to reproduce the dose and frequency depen
249 ting the chemical potential of lipid species is sufficient to reproduce the experimentally observed i
250 ing puberty, allopregnanolone administration was sufficient to reproduce the decreased corticosterone
251 REDD1, a stress-induced metabolic regulator, is sufficient to reprogram lipid metabolism and drive pr
252 t synthetic repeat-derived endo-siRNA mimics are sufficient to rescue Dcr-2-deficiency-induced defect
253 omains of CD2AP or p110gamma to the membrane is sufficient to rescue CD2AP-knockdown phenotypes.
254 acellular integrin-binding domain of Basigin is sufficient to rescue peripheral glial compression.
255  question of how much dystrophin restoration is sufficient to rescue the heart from DMD-related patho
256 lastine prevented microtubule overgrowth and was sufficient to rescue centrosome polarization in KIF2
257 mination at the embryonic termini for 90 min was sufficient to rescue normal development, generating
258  Expression of either apoE3 or apoE4 in VMCs was sufficient to rescue the hypercholesterolemia and at
259 xpressed CX3CL1 in both male and female mice was sufficient to rescue the neurodegeneration, increase
260 rexpression of the transcription factor MITF was sufficient to rescue the small eye and adult lethal
261 nd inhibition of the steroidogenesis pathway is sufficient to restore anti-tumor immunity.
262 lly, expression of artificial PM-PER tethers is sufficient to restore retention in inp1Delta cells.
263 ring infection abrogated Ido1 expression and was sufficient to restore IgE production and worm expuls
264            Overexpression of IFI44 or IFI44L was sufficient to restrict RSV infection at an early tim
265 m, suggesting that latent epitope expression is sufficient to retain gB-CD8s.
266 Finally, restoration of myoferlin expression was sufficient to reverse PINCH-1-deficiency induced inh
267       Furthermore, aspartate supplementation was sufficient to reverse the antihypertrophic effect of
268 n, etomoxir at low micromolar concentrations was sufficient to saturate its target proteins and to bl
269  a subset panel of the 12 most powerful SNPs is sufficient to separate the two pure species, even whe
270 at muscle contractions, as part of exercise, are sufficient to shift the muscle circadian clock phase
271 of SERCA, and binding of a single Ca(2+) ion is sufficient to shift the protein population toward a s
272 pathy mice, even after pathology has formed, is sufficient to significantly delay neurodegeneration a
273 r simulations suggest that this gene network is sufficient to spontaneously polarise the developing r
274 -DNA complexes are force-sensitive and TFIIB is sufficient to stabilise TBP on a strained promoter.
275 e" in which BBB opening in the resting state is sufficient to stimulate a protective barrier at the G
276 cifically to the major PG synthase PBP1b and is sufficient to stimulate its biosynthetic glycosyltran
277 e, constitutively active forms of Yap or Taz were sufficient to stimulate macropinocytosis of extrace
278 rnally inherited copy of Dot1l in the embryo is sufficient to support development.
279                                   The repair is sufficient to support fertility and maintain health a
280 et a single wild-type allele of TIR1 or AFB2 is sufficient to support growth throughout development.
281 , we aimed to determine whether the evidence was sufficient to support recommendations in these areas
282 utamate cell bodies or axon terminals in NAc was sufficient to support reinforcement.
283 idative phosphorylation in activated T cells was sufficient to suppress proliferation and upregulate
284 rrent densities as low as 5 x 10(5) A cm(-2) are sufficient to switch the out-of-plane magnetization
285    Although such data is rather sparse, they are sufficient to train supervised models that capture t
286 ortex during the process of mitotic rounding is sufficient to translate interphase shape into a corti
287         We argue that no single therapy will be sufficient to treat SCI effectively and that a combin
288 at AGO mutations disrupting miRNA 3' binding are sufficient to trigger extensive miRNA 3' modificatio
289  cytokinin regulator in the Yh haplotype may be sufficient to trigger reversal to hermaphroditism.
290 charide transfection into the host cytoplasm is sufficient to trigger active cathepsin accumulation i
291  an allergen was administered a week earlier is sufficient to trigger an allergic response-but only i
292 und that decaging active MKK6 in fibroblasts is sufficient to trigger apoptosis in a p38-dependent ma
293 an formation of a specific protein assembly, is sufficient to trigger autophosphorylation.
294 0 dB SPL, and that this acoustic sensitivity is sufficient to trigger backward strike behavior.
295    Although a partial loss of BCCIP function is sufficient to trigger genomic instability and tumorig
296 ns associated with Synaptotagmin-1 by Ca(2+) is sufficient to trigger rapid (<100 msec) and synchrono
297 n of total RNA that phase-separates in vitro is sufficient to trigger SG formation.
298             A partial loss of BCCIP function was sufficient to trigger genomic instability and tumori
299              In this study, we show that TNF is sufficient to upregulate MINCLE, MCL, and DECTIN-2 in
300 ealed a high percentage of catenation, which is sufficient to yield 'nanolympiadane'(10), a nanoscale

 
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