戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 0% (during the first min after the treadmill was turned off).
2  magnetization, which relaxes when the field is turned off.
3  the future active X and persists until Xist is turned off.
4  and in their absence stop codon suppression is turned off.
5  maintains its decision even after the input is turned off.
6 emporal sequence even when light stimulation is turned off.
7 which revert to aggregates when illumination is turned off.
8 e this process is reversible when blue light is turned off.
9 osure to light that persists after the light is turned off.
10 ies from solution and the conjugate addition is turned OFF.
11 lodes' as a travelling wave when the tweezer is turned off.
12 rospray emitters when the ionization voltage is turned off.
13 the upstream DNA damage checkpoint signaling is turned off.
14 t permanently in G2 phase when Ku expression is turned off.
15 h apart the spindle poles when poleward flux is turned off.
16 nt mechanism that engages when poleward flux is turned off.
17  prevents expansion of slp stripes after Eve is turned off.
18 rotein is expressed and the wild-type Calpha is turned off.
19 his process is reversed, and KLF2 expression is turned off.
20 he magnetic relaxation signal when the field is turned off.
21  weeks of daily pacing at a time when pacing was turned off.
22 ion when DNA-templated primary transcription was turned off.
23 d in 13 of 14 APs after radiofrequency power was turned off.
24 ctional recovery that persisted when DBS(LH) was turned off.
25  health benchmarks for hours after the stove was turned off.
26        These changes persisted even when SCS was turned OFF.
27 rsening persisted even after the stimulation was turned off.
28 the expression of the Nrg1-ntfbeta transgene was turned off.
29 nents that was reversed after the high light was turned off.
30 ed moving through the x-ray beam or the beam was turned off.
31  diluted by growth after dnaA expression had been turned off.
32  strategy, all clinically nonrelevant alerts were turned off.
33 ted from the place cells after the landmarks were turned off.
34 d in the dark cycle), 1900 (before the light was turned off), 2200 (3 hours into the dark cycle), and
35 n and mRNA levels is transient and begins to be turned off 3-6 h after growth stimulation of cultured
36 h gene silencing remains stable even if Xist is turned off(8).
37  When the electrostatic protein-water forces are turned off, a dewetting transition occurs in the int
38       If as few as three inappropriate genes are turned off, a normal cell can be converted into a ca
39 hannel where the voltage across the nanopore is turned off after a user-defined number of single mole
40               In contrast, how transcription is turned off after activation is unclear.
41 ion occurs only in subconfluent cultures and is turned-off after a polarized epithelium has formed.
42 ssary for tumor progression, ATF3 expression was turned off after cardiac hypertrophy development fol
43 ylation; the silencing persisted after p15AS was turned off, although methylation and heterochromatin
44  For each day the engineered aeration system was turned off, an additional 5.1 mumol P m(-2) h(-1) wa
45                                 When STN DBS was turned off, an initial fast but partial loss of bene
46 the amphiphilicity of the copolymer that can be turned off and on by varying the polarity of the solv
47 all kinetochores come under tension, the SAC is turned off and APC/C(Cdc20) degrades cyclin B and sec
48 idic supramolecular receptors for fullerenes is turned off and on by changing the solvent, whereas th
49  the alphaHL channel, the DC driving voltage is turned off and the conductance of the channel is moni
50 However, the mechanism of how the pEMT phase is turned off and the redifferentiation phase is initiat
51                          Then, the metronome is turned off and the subject continues at the establish
52                         Second, the pressure is turned off and the voltage is applied to reverse the
53 mpairment was ameliorated when the transgene was turned off and D2R levels were normalized.
54 rovement was maintained when the stimulation was turned off and was accompanied by a higher number of
55 ields of individual neurons, then the lights were turned off and the object was silently removed.
56 sis, when chromatin condenses, transcription is turned off, and almost all double-stranded DNA-bindin
57 roms, can be stable for hours after the beam is turned off, and ions that screen charges on the filam
58 rsed spontaneously when calcineurin activity was turned off, and expression of fetal genes reverted t
59 matrix and cornea-transparency-related genes were turned off, and a repertoire of macrophage genes we
60 eas those associated with body column tissue are turned off as the tentacle zone expands.
61         When attractive van der Waals forces are turned off as well, the dewetting in the interdomain
62 in undifferentiated embryonic stem cells but was turned off as these cells differentiate in vitro and
63            Furthermore, when P3H2 expression was turned off, as seen naturally in cultured SAOS-2 ost
64                         How stress responses are turned off at the right time and place remains poorl
65  driven by the distal enhancer can no longer be turned off at late stages in lin-1 and lin-31 mutants
66 (1)F(o) complex explains how ATPase activity is turned off at night by a redox switch.
67 is specifically seen in progenitor cells and is turned off at the onset of neuronal differentiation.
68 etermining gene Sox9 in mutant Sertoli cells was turned off at embryonic day 14.5 after Wt1 ablation,
69        When transgenic human RTN3 expression was turned off at young age, formation of RIDNs was esse
70 rned on at 00:00 CT and increased after they were turned off at 12:00 CT.
71  it was found that reserpine oxidation could be "turned off" at flow rates as low as 2.5 microL/min a
72 RIDNs was dependent on whether the transgene was turned off before or after the formation of RTN3 agg
73  early stages of differentiation (when Vsx-2 is turned off), before decreasing to an intermediate lev
74                 This sheath or coaxial fluid was turned off between analysis of cells and on during c
75 remained stable even when the electric field was turned off but readily redissolved when counterions
76                              Galpha subunits are turned off by GTP hydrolysis.
77 ic system and allows the dynamic exchange to be turned off by addition of 18-crown-6, which binds the
78 pose that the Cyp6a8 gene in 91-C strain may be turned off by an active repressor which might be inhi
79  dATP but, unlike the E. coli enzyme, cannot be turned off by combinations of ATP and dGTP/dTTP.
80                                The surge can be turned off by fixing the rate of fluid flow through t
81              Moreover, gene expression could be turned off by removal of the inducing stimulus.
82 ransporters require PIP2 to function and can be turned off by signaling pathways that deplete PIP2.
83           We find that NR2F2-Iso2 expression is turned off by DNA methylation when NCCs differentiate
84 n healthy cells, the SR-BI apoptotic pathway is turned off by eNOS and HDL which prevents inappropria
85                    Crucially, this mechanism is turned off by host receptor interactions at the cell
86 hanism by which NF-kappaB-mediated signaling is turned off by its removal from the DNA in the presenc
87                                          Rh* is turned off by phosphorylation by rhodopsin kinase [G-
88  synthesis in both asexual and sexual spores is turned off by the NF-kB like fungal regulators VosA a
89 o distinct points in the cycle, one of which is turned off by the presence of P(i).
90                         When hetN expression was turned off by transferring cells to media lacking co
91 h in PDL pancreas were impaired when ERalpha was turned off chemically or genetically (ERalpha(-/-)),
92  presence of doxycycline, and the gene could be turned off completely by removal of doxycycline.
93       Transgene expression in these mice can be turned off completely with low doses of doxycycline (
94 cterial hosts against phage predation and to be turned off during prophage induction, balancing immun
95                             When the current is turned off during phases of the domain wall motion wh
96 esent study was to investigate how autophagy is turned off during prolonged oxidative stress, to avoi
97 a naive host, the activity of the M promoter is turned off early, preventing excessive synthesis of m
98                     After the electric field is turned off, electrical current is measured and observ
99              Accordingly, miR-150 repression was turned off following treatment with the DNMT inhibit
100 ve from this "memory" after the illumination is turned off for an extended period of time.
101  clinical status when deep brain stimulation was turned off for 2 days, suggesting that deep brain st
102                     When the smoke evacuator was turned off for 30 seconds, there was a more than 26-
103                           When the transgene was turned off from conception to 3 months of age, no ph
104 tablished in the host, these virulence genes are turned off, fully attenuating the vaccine strain.
105                             When these cells were turned off, generalization increased.
106                                  When spikes were turned off GOM recoveries returned to 60 +/- 19% of
107 scribed, but the pathway whereby the levator is turned off have not.
108                            When the stimulus was turned off, his body partially tilted back at an int
109 onditions, allowing the migration process to be turned off in the absence of an acetate additive and
110  Our findings indicate that the RASSF1A gene is turned off in a significant number of melanomas and t
111      In adults, Brg1 (also known as Smarca4) is turned off in cardiomyocytes.
112 ormation: miR-211 is a molecular switch that is turned off in melanoma cells, raising the hope that i
113 e demonstrate that (independent of EBV) TET2 is turned off in normal and malignant germinal center (G
114  downstream target of the GTPase Rheb, which is turned off in response to AMPK via the tuberous scler
115                             Optx2 expression is turned off in the future pigment epithelium but remai
116 e expression of episome maintenance proteins is turned off in the predominant long-term latency reser
117 at of 738-lob2A1::Km, suggesting that lob-2A was turned off in this phase.
118 phloem fibers undergoing tension stress, but was turned off in tissues undergoing compression on the
119  of MCC that takes place when the checkpoint is turned off involves the conversion of C-Mad2 back to
120 eversed, if expression of the mutant protein is turned off, is unknown.
121  the LTi developmental pathway, but needs to be turned off later to avoid diversion to the T cell fat
122  from a regulated galactose-induced promoter was turned off, leading to recapitulation of the iron re
123 eory in which all interspecific interactions are turned off leads to analytical results that are in a
124 rium on ultrafast timescales after the light is turned off, limiting their practical applications.
125                   When the checkpoint signal is turned off, MCC is disassembled and the checkpoint is
126                   When the checkpoint signal is turned off, MCC is disassembled, a process required f
127                               When the light was turned off motoneuron firing and locomotor frequency
128                         The XIST allele must be turned off on one X chromosome to maintain its activi
129 arge/polarity of an atom or functional group was turned off/on, and the resulting effects on binding
130                           How the SAC signal is turned off once all the chromosomes have successfully
131                        When PcG target genes are turned OFF, PcG proteins bind to PREs or CGIs, and P
132 within cellular grafts after NT-3 expression was turned off possibly via reduction in L1 and laminin
133 isible after-images of the stimuli when they were turned off ('prime-sight').
134 y phase, when an active p53 program needs to be turned off quickly.
135                     When the electrorotation was turned off, so that the load was again high, stator
136 sient behavior that occurs after the forcing is turned off suggests that the double knocks of Campeph
137                          When the plasma jet is turned off, the continuous surface charge decreased t
138 s to turn on anion transport, which can then be turned off through addition of the blocker dodecyl su
139 roteins is known, still obscure is how ORAI1 is turned off through Ca(2+)-dependent inactivation (CDI
140 interaction vanishes if the charges on water are turned off, thus establishing that electrostatic int
141                        The equilibration can be turned off to yield stable products or reactivated ag
142                              The ISC process is turned off to deactivate PDT ability, when the PS is
143 it is unclear how the Fe-deficiency response is turned off to prevent toxicity when Fe is sufficient.
144 ize in heterochromatic regions of genome and are 'turned off' under laboratory condition.
145                  We show that their emission is turned off upon breaking the conjugation of the cyclo
146 ing mRNA (RNA replicon) whose expression can be turned off using a genetic switch that responds to or
147  mechanism for receptor diversification that is turned off when antigen receptors are cross-linked by
148                    However, how this pathway is turned off when hepatocyte proliferation is no longer
149 tified by the integrated fluorophores, which are "turned off" when the particles become destructed an
150  significantly declined when NT-3 expression was turned off, whereas continued NT-3 expression sustai
151 r, and both galactose catabolic pathways can be turned off with a galactose analogue in a substrate-s
152 oved subsequent peeling performance when AFF was turned off, with reductions in average force, SD of

 
Page Top