ライフサイエンスコーパス: be turned on

1 nd see the moment when electrical conduction is "turned on".

2 d in T(2E) when the Josephson-based isolator is turned on.

3 zone (> 25 ppb) forms near the lamp when it is turned on.

4 the EBV latent-to-lytic switch protein ZEBRA is turned on.

5 phosphorescence of the hexathiobenzene core is turned on.

6 ing the dark and dephosphorylated when light is turned on.

7 s activating chromatin marks only when xol-1 is turned on.

8 re than an order of magnitude when the cloak is turned on.

9 the number of seeds produced once the switch is turned on.

10 adation were delayed when expression of US11 was turned on.

11 itive progeny even before the zygotic genome was turned on.

12 day period, during which time the NIr device was turned on.

13 ersive enclosed arm, a light and white noise were turned on.

14 le active stress in a nematic liquid crystal is turned on, +1/2 defects can self-propel, in analogy t

15 imes (24-hour clock): 0700 (before the light was turned on), 1000 (3 hours into the light cycle), 100

16 is not normally expressed in the OSE, but it is turned on after malignant transformation.

17 ng to fluorescent products, the fluorescence is turned on after the photoligation.

18 increased further and Stat6 gene expression was turned on after P. gingivalis infection in BALB/cByJ

19 In the PP + GES-ON group, the GES was turned on after surgery.

20 st tau pathology once soluble tau expression is turned on again.

21 g cycle, only a fraction of the fluorophores were turned on, allowing their positions to be determine

22 , such as cannibalism and biofilm formation, are turned on and a later phase in which genes that play

23 ally patterned by extracellular signals that are turned on and off at specific times.

24 f sRNAs with their target mRNAs so that they are turned on and off together to maximize duplex format

25 ecially those such as phototransduction that are turned on and off with great rapidity.

26 mers, revealing strong interactions that can be turned on and off as a function of the electrochemica

27 ich expression of the mutant transgene could be turned on and off as needed.

28 e incandescent and discharge lamps, LEDs can be turned on and off at high speeds.

29 strength in vivo, homosexual behavior could be turned on and off by altering glutamatergic transmiss

30 n scattering beacons (SERS beacons) that can be turned on and off by long-range plasmonic coupling, i

31 te a reversibly operating nanovalve that can be turned on and off by redox chemistry.

32 n generate self-sustaining activity that can be turned on and off by synaptic inputs.

33 ion in vivo and to enable such expression to be turned on and off effectively.

34 ansgenic animals, enabling reporter genes to be turned on and off rapidly.

35 Proliferation can be turned on and off repeatedly simply by shifting the e

36 hanism also allows the catalytic reaction to be turned on and off reversibly by specific molecular si

37 s reported in which excitonic coupling could be turned on and off simply by changing the solvent.

38 of the yeast PI-SceI homing endonuclease can be turned on and off using a redox switch.

39 at cell-signaling pathways can theoretically be turned on and off, both locally and globally, in resp

40 lecular and cell type specificity but cannot be turned on and off.

41 el that allowed Zap70-dependent signaling to be turned on and then off again.

42 of gene silencing antisense agents that can be turned on and turned off in mammalian tissue culture.

43 f odontoblasts requires unique sets of genes being turned on and off in a growth- and differentiation

44 re simultaneously present, the photoactivity is turned on and evidence is given for an electron trans

45 closed-loop method, where high-frequency DBS is turned on and off according to a feedback signal, whe

46 Signaling is turned on and off by regulating stability of the effe

47 antibody affinity in an ongoing response; it is turned on and off during the differentiation of B lym

48 f the cAMP/PKA pathway and that PKA activity is turned on and off on the timescale of tens of seconds

49 Each switch is turned on and off sequentially to distribute and rout

50 ed feedback stimulation (apLDF), where cpLDF is turned on and off using pre-defined amplitude thresho

51 ies have shown how this locus-control region is turned on and off.

52 ing the ORR, particularly when the fuel cell is turned on and off.

53 For only one group, the device was turned on and delivered a slow heartbeat-like vibrat

54 The ion flux was turned on and off, apparently because the water wett

55 and Perelson suggested that if hypermutation were turned on and off in a phasic manner, affinity matu

56 orientation, and conformation of the ligand are turned "on" and "off." Second, sampling of the ligan

57 exhibited fast response times (<1 s) and can be turned "on" and "off" using a single light source at

58 evelop a photoswitchable derivative that can be turned "on" and "off" with low-intensity light to spa

59 which a GTPase acts as a bimodal switch that is turned "on" and "off" by external regulatory factors,

60 a protein switch, whereby protein production is turned "on" and "off" by using short noise pulses.

61 mation about when, where, or how much a gene is turned on, and by dissecting the modules driving a gi

62 s the clusters rotate when the energy source is turned on, and fall apart when the energy input is of

63 s of the assembly of MCC when the checkpoint is turned on, and of its disassembly when the checkpoint

64 Remarkably, these degeneracies can be turned on-and-off on demand, as well as their number

65 the component parts of the 'acetate switch' are turned-on as a consequence of direct competition, on

66 face of adversity (stress), death mechanisms are turned on at a cellular (apoptosis) and organismal l

67 Transcription of P(QA) is turned on at the end of the DNA replication period, c

68 controlled by the Cdc25C phosphatase, which is turned on at the G(2)/M transition to catalyze Cdk1 a

69 gelB expression is turned on at the migrating epithelial front in the co

70 Promoter Px is turned on at the same time as promoter P3 during earl

71 of DnaA in Bacillus subtilis whose synthesis is turned on at the start of sporulation.

72 The tDCS-stimulator was turned on at 3 minutes after the scanning started.

73 Transgene expression was turned on at different time points: in utero, at bir

74 dian rhythm: IOP decreased after room lights were turned on at 00:00 CT and increased after they were

75 ned off at 48 h, while "cell division" genes were turned on at 48 h.

76 Some genes that were turned on at a low dose of Spo0A either had a high

77 enes, including a number of protein kinases, were turned on at this stage.

78 ther improvement when finally the stimulator is turned on, but in the case of saccadic latency the pa

79 ins that conduct ions across gated pores and are turned on by electrical or mechanical stimuli, respe

80 en disengaged interactions within a receptor are turned on by its guest, these intrahost interactions

81 t the specific endogenous apoptotic programs are turned on by loss of Dicer.

82 rials demonstrating phosphorescence that can be turned on by incorporating halogen bonding into their

83 ide the intact endocytic organelles, but can be turned on by redox activation after endolysosomal dis

84 g polymerization of epsilon-caprolactone can be turned on by the in situ opening of the triple-layer

85 1 cases) the expression of 20 genes that had been turned on by sigma(G) and represses the expression

86 erpolarizing light-gated channel, HyLighter, is turned on by a brief light pulse at one wavelength an

87 iana that gates with a unique modality as it is turned on by an electrical stimulus only after exposu

88 This immune circuit is turned on by an miR2977-SAMT (encoding a citrus Salic

89 e the XBP-1 axis normally, as follows: XBP-1 is turned on by cues that trigger differentiation and no

90 been described as a biochemical switch that is turned on by increases in intracellular calcium to me

91 chanism in which an inactive degradation tag is turned on by induced conformational changes.

92 Fluorescence of the reporter dye is turned on by rapid removal of the quinone quencher, a

93 Posterior neural markers were turned on by bFGF in AC expressing DN-BR or chordin

94 Here, we show that striatal circuits can be "turned on" by acute Gq-protein signaling or "turned

95 ) agents has been developed where the signal is "turned on" by altering the longitudinal relaxation t

96 When NT-3 gene expression was turned on, cholera toxin beta-subunit-labeled sensor

97 reactive alkynes and whether reactivity can be turned "on demand." When stereoelectronic amplificati

98 ions were detected only when the UV detector was turned on, demonstrating that UV light is necessary

99 e thus provided evidence that specific genes are turned on during catagen, which is therefore not sim

100 on of the mucoid exopolysaccharide alginate, are turned on during conversion to mucoidy.

101 sed genes found during this effort appear to be turned on during organogenesis, however, some genes w

102 No cues were turned on during testing.

103 hern blot analysis showed that AC expression was turned on early in development, by E7.0, and continu

104 erentiate into myofibroblasts when RhoA/ROCK is turned on, endothelial cells when turned off.

105 When expression of barnase is turned on, failure to form a complex between the muta

106 a completely quenched (19)F NMR signal that is "turned on" following reduction to Cu(I).

107 he device was kept off for 3 months and then was turned on for the following 3 months.

108 ch balance; thus, the angiogenic switch must be turned "on" for cancer progression.

109 We propose that when the checkpoint is turned on, free MCC is the precursor of APC/C-bound M

110 When they were turned on, generalization decreased.

111 y complex is key for understanding how genes are turned on in response to a Notch signal.

112 ding sine oculis (so) and eyes absent (eya), are turned on in their respective domains by Dpp.

113 es, and we show that sarcomere branching can be turned on in IF muscles by salm-mediated conversion t

114 29) Xe hyperCEST NMR contrast agent that can be turned on in response to H(2) O(2) , which is upregul

115 uminescence of the AuNCs@GSH-Fe(3+) can also be turned on in the presence of phosphate-containing met

116 ng in mice indicates that gastrin expression is turned on in a subset of differentiated beta-cells af

117 Despite the fact that MET3pr is turned on in all the cells, its activity still shows

118 tivated and the maintenance promoter, SxlPm, is turned on in both sexes; however, only in females is

119 ay be part of a differentiation program that is turned on in senescent cells.

120 number of existing probes whose fluorescence is turned on in the presence of the class of biological

121 first salivary gland genes, fork head (fkh), is turned on in the primordium of this organ, the saliva

122 pression of the bHLH gene, Thylacine1, which is turned on in the PSM as cells mature and segment, in

123 r1 regulon responds to copper limitation and is turned on in zinc deficiency, and Crr1 is required fo

124 ormation of PSC when expression of DeltaFosB was turned on in adult animals by removing Dox.

125 When CRT was turned on initially, immediate changes occurred in L

126 Which switching mode is turned on is controlled by the molecular environment.

127 When the checkpoint is turned on, it promotes the formation of the mitotic c

128 Underscoring how classic doctrine can be turned on its head, the peptidoglycan layer of polar-

129 This appealingly simplistic notion has been turned on its head in recent years with the discove

130 The usual approach is turned on its side by using the mathematical descript

131 se growth in an El Tor strain, transcription is turned on later in growth to levels in excess of thos

132 nential-phase growth, while the ctxAB operon was turned on later, after the rise in toxT expression.

133 Adhesion/invasion genes were turned on later in the maturation process.

134 in the first 10 min after an electric field is turned on, leading to more abundant and wider protrus

135 ystem, organic light-emitting diodes (OLEDs) are turned on locally where the surface is touched, and

136 vicious cycle such that the Hh pathway might be turned on nonclassically by stimuli from the tumor mi

137 n single cell clones, hMLH1 expression could be turned on, off, and on again by 5-azacytidine exposur

138 nce of UV light via diffusiophoresis and can be turned "on/off" in a controlled manner.

139 eterogeneous photoredox catalyst which could be turned on/off by changing a simple stimulus (O(2)) fo

140 e local plasmonic density of states that can be turned on/off or gradually tuned by the applied gate

141 The NIR signal is turned on only in the presence of SA-B-Gal, and the f

142 ty, in which a nanoassembly-cell interaction is turned on, only in the concurrent presence of two dif

143 More than 8,000 genes are turned on or off as progenitor cells produce the 7 c

144 Galpha(q) protein, the activity of which can be turned on or off at will in cardiac myocytes, develop

145 Crucially, this spin-flop can be turned on or off by applying an electric field across

146 oil field(4) suggested that seismicity might be turned on or off by cycling subsurface fluid pressure

147 rmation pathways activated by Galpha(12) can be turned on or off by the addition or removal of serum.

148 transgenic system in which Akt signaling can be turned on or off in the adult heart, we previously sh

149 r size allow gradient-controlled pathways to be turned on or off simply by altering the distance betw

150 nal split inducible Cas13 effectors that can be turned ON or OFF via small molecules in multiple cell

151 that the ATPase activation switch appears to be turned on or off when L175C/N820 are clamped at dista

152 Light can be turned on or off, and adjusting its intensity and dur

153 pecific signals, allowing kinase activity to be turned on or off.

154 uba7 electrodes with the deterrents randomly being turned on or kept off.

155 san nanofibres, the flow of protonic current is turned on or off by an electrostatic potential applie

156 ptional regulation, the expression of a gene is turned on or off by changes in the concentration of a

157 Ion flow through PAL-modified channels is turned on or off by photoisomerizing PAL with differe

158 The neurostimulator was turned ON or OFF for the first one month and then to

159 In BRCA1-deficient cells Hippo pathway is "turned On." Phosphorylation of YAP1 is crucial for t

160 ight, the switch of age-dependent cell death was turned on prematurely, as indicated by the accumulat

161 mitochondria, the nutrient sensing machinery is turned on remodeling transcription to block protein s

162 esorbable packaging and electronics that can be turned on remotely, after implantation, to provide th

163 opment in transgenic mice even when the gene is turned on shortly before the time of tumor developmen

164 in which all terms in the interaction energy are turned on simultaneously using 1/kT as the coupling

165 The flow was turned on suddenly so that the entire evolution of m

166 reased when the p53 transactivation activity was turned on, suggesting that PRC1 transcription could

167 The H-function can be turned on/switched off by the concentration of comple

168 For the LCD to be turned on, the cell voltage is boosted by additional

169 When mismatching of atoms or bonds is turned on, the compute times increase as expected, an

170 When the checkpoint is turned on, the mitotic checkpoint complex (MCC) inhib

171 dian time and begins at 00:00 CT when lights are turned on.) The maximum difference in IOP between li

172 when upper room germicidal air disinfection was turned on throughout the ward.

173 If the angiogenic switch needs to be turned on to facilitate the tumor growth, the questio

174 the late part of the assigned period, lights were turned on twice for a 1-hour interval.

175 design, an autoinhibited coiled-coil switch is turned on upon proteolytic cleavage, which results in

176 These results illuminate how GIV-GEF is turned on upon receptor activation, adds GIV to the r

177 pletely CEST-silent in the oxidized form but was "turned on" upon reduction with beta-NADH.

178 The device is turned on when the temporary occupation of the core s

179 artificial radiation was designed; the lamp was turned on when solar UV-A irradiance was below 20 W

180 the overall MS signal increased when the UV was turned on, which can help with the detection of low-

181 The bistable photoswitch can be turned on with millisecond-long pulses at one wavelen

182 The Scl gene is turned on within FLK1(+)CD4(-) cells to give rise to