ライフサイエンスコーパス: be unaltered

1 Sc) formation is altered yet disease outcome is unaltered.

2 e endothelial capacity to engage leukocytes) is unaltered.

3 ontext, GR function in COPD lung macrophages is unaltered.

4 to UV radiation or topoisomerase inhibitors is unaltered.

5 development of B lymphocytes in bone marrow is unaltered.

6 nd non-mGluR-mediated long-term potentiation is unaltered.

7 lthough both positive and negative selection are unaltered.

8 and the presynaptic residual calcium signals are unaltered.

9 lve endocytosis, and surface receptor levels are unaltered.

10 -1 levels, whereas other coagulation factors are unaltered.

11 tes in mice harboring Il10-deficient B cells are unaltered.

12 insulinotropic polypeptide (GIP) secretions are unaltered.

13 Decay kinetics of I(K) at negative voltages are unaltered.

14 n noradrenergic inputs to the olfactory bulb are unaltered.

15 tion even though vorinostat pharmacokinetics are unaltered.

16 ent cells, those of both AP- and betaE-sites are unaltered.

17 ude of mEPSCs and amplitudes of evoked EPSCs are unaltered.

18 specific protein, proteolipid protein (PLP), was unaltered.

19 e total number of membrane-proximal vesicles was unaltered.

20 on of endothelial aquaporin-1 water channels was unaltered.

21 is, fatty acid oxidation, and VLDL secretion was unaltered.

22 cytosolic PKC increased, PKC activity itself was unaltered.

23 but the essential character of the response was unaltered.

24 velopment of vestibular efferent innervation was unaltered.

25 es; however, TRPC1 and TRPC5 mRNA expression was unaltered.

26 amma3(R225Q) mice, whereas glucose tolerance was unaltered.

27 mbrane attack complex, the tumor progression was unaltered.

28 ever, the strength of the remaining synapses was unaltered.

29 mised, whereas their multi-lineage potential was unaltered.

30 For all procedures, the FERG was unaltered.

31 Postsynaptic targeting of recombinant Shank3 was unaltered.

32 fast-, slow- and non-deactivating fractions was unaltered.

33 channel protein levels, whereas excitability was unaltered.

34 red with healthy skin, but RelA distribution was unaltered.

35 cohol drinking; however, sucrose consumption was unaltered.

36 rosine kinase (Tyk) 2, and Jak1 by IFN-alpha was unaltered.

37 he beneficial effect of increased plasma HDL was unaltered.

38 to decline (P=0.096), whereas cardiac output was unaltered.

39 while AM expression in alveolar macrophages was unaltered.

40 lity sharply increased, whereas quantal size was unaltered.

41 ptor protein expression in the vagal complex was unaltered.

42 an controls, while malaria-associated anemia was unaltered.

43 blocked in BCATm(-/-) islets, KIC oxidation was unaltered.

44 hile mRNA expression of venular NCX isoforms was unaltered.

45 d the production of several innate cytokines was unaltered.

46 sed in a cohesin mutant, whereas SIR binding was unaltered.

47 human collecting duct cell line, while SPAK was unaltered.

48 olic content, whereas the carotenoid content was unaltered.

49 Alveolar macrophage TNFalpha production was unaltered.

50 psaicin-induced mEPSCs from C-fiber synapses was unaltered.

51 vel, whereas in H1299 cells the level of ATP was unaltered.

52 esulfonate (PFOS) found that linear (L)-PFOS was unaltered.

53 ation, whereas FRET with other tested SNAREs was unaltered.

54 the ability of insulin to suppress lipolysis was unaltered.

55 K cells, but the surface expression of Ly49A was unaltered.

56 yramidal cells, whereas D2 receptor function was unaltered.

57 ncentrations, cardiovascular risk biomarkers were unaltered.

58 t differentiation into cDC1 and cDC2 subsets were unaltered.

59 iatal levels of dopamine and its metabolites were unaltered.

60 endogenous regulatory elements of mouse Nod2 were unaltered.

61 and membrane association of the two variants were unaltered.

62 CB1 activation, sEPSCs in these same neurons were unaltered.

63 ing VIT (P < .001) although tryptase and IgE were unaltered.

64 asement membrane and systemic blood pressure were unaltered.

65 threshold (>/= 3.5 x 10(9) photons/cm(2)/s) were unaltered.

66 arget of rapamycin, and Forkhead box protein were unaltered.

67 biotin-based tracer in the stria vascularis were unaltered.

68 e high but embryo and endosperm ABA contents were unaltered.

69 Total beta-catenin levels were unaltered.

70 ximity of the single nucleotide polymorphism were unaltered.

71 in these animals, while analgesic responses were unaltered.

72 gnitude/properties, and mRNA levels of Scn5a were unaltered.

73 other immature and mature T cell populations were unaltered.

74 nants, transposons, and chromosome structure were unaltered.

75 Conversely, spleen DC were unaltered.

76 expression and long-term potentiation (LTP) were unaltered.

77 mEPSCs, decay time of mIPSCs, and spine size were unaltered.

78 acid (JA), but levels of salicylic acid (SA) were unaltered.

79 sion and NMDA receptor (NMDAR)-dependent LTD were unaltered.

80 yer IV and layer V, although EPSC amplitudes were unaltered.

81 rphyrin and the proteins affinity for oxygen were unaltered.

82 otal numbers of parvalbumin-positive neurons were unaltered.

83 e content and agonist-induced IP3 production were unaltered.

84 ration, tip trajectory, and variance of AFCL were unaltered.

85 methylated, whereas other chromatin proteins were unaltered.

86 n the lung, even though serum concentrations were unaltered.

87 rly steps in the internalization of the drug were unaltered.

88 al weight, litter size and crown rump length were unaltered.

89 ive stress while FoxP3(+) T-regulatory cells were unaltered.

90 evoked via the nucleus of the solitary tract were unaltered.

91 s from healthy individuals, and the increase was unaltered after 12 weeks of etanercept therapy.

92 Fractional excretion of Na+ was unaltered after HMP and HR but significantly increas

93 These findings were unaltered after the application of different statis

94 CD79B expression levels in stable patients were unaltered after transplantation in PBMC but showed

95 Although antagonist peptide binding was unaltered, almost all mutations affected GLP-1 pepti

96 e and a familiar face (control stimuli) that were unaltered, altered to include only high spatial fre

97 Cell rolling on E-selectin was unaltered although the number of adherent cells was

98 aturated fatty acids in the pah1Delta mutant were unaltered, although the ratio of palmitoleic acid t

99 Surface MART-1/HLA-A*0201 in these clones was unaltered and F5 CTLs recognized and interacted with

100 ect of vasopressin on mean arterial pressure was unaltered and that on renal vascular perfusion press

101 as pancreatic lymph node T cell populations were unaltered and T cell proliferation was unaffected b

102 sms were explored; adenylate kinase activity was unaltered, and although GAMT(-/-) hearts accumulated

103 percentage of C-terminally cleaved alpha2AP was unaltered, and that Arg407Lys did not influence alph

104 on site, the phosphorylation state of RNAPII was unaltered, and the transcription bubbles remained op

105 l muscle contraction in these mice, however, was unaltered, and there was no evidence for reduced pH-

106 rillary acidic protein (Gfap) message levels were unaltered, and other astrocytic markers were signif

107 hort-term plasticity at mossy fiber synapses are unaltered at 6 months in APP/PS1 mice.

108 Hearing acuity in treated animals is unaltered at postnatal day 30.

109 X-ray absorptiometry scan, body composition was unaltered at 3 or 6-7 months of age.

110 Constriction of retinal arterioles to ET-1 was unaltered at all time periods of hyperglycemia.

111 P = .0005), while total white matter volume was unaltered (beta = -10.10; 95% CI, -20.73 to 0.53; P

112 The FSHD Lymphoblast score is unaltered between FSHD myoblasts/myotubes and their c

113 glycoprotein 130, as well as IL-6 secretion, was unaltered between cultured myotubes from normal gluc

114 Circulating serum IL-6 concentration was unaltered between normal glucose tolerant and type 2

115 The frequency and spectra of hypermutation was unaltered between Polzeta(+/-) Poleta(-/-) and Polze

116 several beta-adrenergic signaling components were unaltered between control and DCM iPSC-CMs, we foun

117 MV neurones excited by cholecystokinin (CCK) was unaltered but the proportion of neurones in which CC

118 female d62MI-TP offspring insulin signalling was unaltered but there was a pancreatic phenotype with

119 etic regulatory genes including lmo2 and scl are unaltered, but levels of gata1 transcripts, encoding

120 nflammasome activation in mature macrophages is unaltered, but IL-18 production from monocytes is gre

121 id (atRA), the balance of daughter cell fate is unaltered, but the rate of cell division increases.

122 from parvalbumin-positive (PV) interneurons was unaltered, but PV cells of DS mice lost their classi

123 Endothelial cell production was unaltered, but there were defects in formation of a

124 xhibit constitutive [(32)P]GTP charging that is unaltered by amino acid withdrawal.

125 Dopamine synthesis capacity is unaltered by antipsychotic treatment, and therapeutic

126 killing activity by infecting cells) of rNDV is unaltered by changes in salt concentration despite mo

127 ugh the singing-related output of HVCX cells is unaltered by distorted auditory feedback (DAF), deafe

128 particle content, whereas the melt viscosity is unaltered by the presence of nanoparticles.

129 The number of infiltrating leukocytes was unaltered by abrogation of gal-3, but reduced expres

130 The homodimer configuration was unaltered by agonist treatment and was stable over a

131 he lung following N. brasiliensis infection, was unaltered by depletion of CD11c-expressing DCs and a

132 While cell recruitment was unaltered by diazepam, the cytokine response to infe

133 , beta-ENaC expression at the apical surface was unaltered by either of these manipulations.

134 t, for E960A-NKA the apparent Na(+)-affinity was unaltered by either PLM or forskolin-induced PLM pho

135 ably, water, food, and saccharin consumption was unaltered by either treatment.

136 ion of CpG islands in Nrf2 or NQO1 promoters was unaltered by GDM, decreased DJ-1 and increased phosp

137 tion and aggregation in response to agonists was unaltered by JAK2V617F expression.

138 FVEP was unaltered by lidocaine.

139 t and blood at the end of the imaging period was unaltered by lung inflammation.

140 In contrast, habitual responding for sucrose was unaltered by mTORC1 inhibition, suggesting that mTOR

141 P(Cl) /P(Na) of airway epithelia was unaltered by pH 7.4 vs.

142 hosphorylation on both phosphorylation sites was unaltered by PP2B or PP2C inhibitors.

143 ) penetration and nonspecific binding, which was unaltered by preadministration of the unlabeled agon

144 Whereas glucose incorporation to glycogen was unaltered by small interfering RNA against DAPK3, pa

145 ed HEK cells revealed that Nav1.7 activation was unaltered by the A1632T mutation but that steady-sta

146 behaviour of non-declining C. septempunctata was unaltered by the presence of H. axyridis.

147 T(H)17-induced disease, the amount of IL-10 was unaltered by treatment, although, unexpectedly, IFN-

148 Locomotor activity was unaltered by vector administration to either region.

149 ia-induced antibody production and pathology were unaltered by 11beta-HSD1 deficiency though plasma l

150 evels in the VTA of ClockDelta19 and WT mice were unaltered by acute AUT1 treatment.

151 Girk2 and GABA(B)R1 mRNA and protein levels were unaltered by cocaine exposure in VTA DA neurons, th

152 In V1, FF excitatory inputs were unaltered by DE, whereas lateral intracortical conn

153 Dilations induced by acidic pH were unaltered by inhibitors of K(ATP) channels or nitri

154 Endocrine cell mass and proliferation rates were unaltered by liraglutide treatment.

155 Nob1 are distinct from its cleavage site and were unaltered by mutation of the catalytic PIN domain.

156 regulatory partner, phospholamban (PLB) and were unaltered by PLB phosphorylation or changes in calc

157 The positive effects of F19A on NSC growth were unaltered by the addition of a functional blocking

158 These results were unaltered by the exclusion of all 178 genes that we

159 r fluxes through separate intrasubunit pores were unaltered by the furan compounds (at concentrations

160 and TGBp2 (which reside in the ER) proteins were unaltered by the presence of TGBp3, suggesting that

161 CXCR5(+)Th17 cell frequencies were unaltered by TNF blockade and in fact remained rema

162 -regulated (exercise training) or when these were unaltered (CD36 overexpression).

163 the initiation and termination phases (which are unaltered compared to the uncorrelated simulations).

164 ning an AT spacer between the two half-sites is unaltered compared with that containing a TA spacer,

165 arrow15%) in healthy rats such that PO(2)mv was unaltered (Control: 19.8 +/- 1.7, SMTC: 20.7 +/- 1.8

166 Although activation of STAT1 was unaltered, deletion of Socs1 relieved inhibition of

167 te, which is a membrane-selective ER ligand, was unaltered, demonstrating integrity of MISS actions.

168 ession of other Ca(2)(+) regulatory proteins are unaltered, dnOrai1 mice exhibit reduced body weight,

169 tly, a previous study showed that the SCN CC is unaltered during RF, which creates a misalignment bet

170 a within cells was reduced; this redox poise was unaltered during the anaerobic isolation of the orga

171 ion, GSK3beta expression and phosphorylation were unaltered during hyperammonemia, and depletion of G

172 ds and their theta-phase precession profiles were unaltered during the task, indicating that trial-re

173 Conversely, at high altitude, FMD was unaltered following moderate-intensity exercise, and

174 tured chromaffin cells; total protein levels were unaltered for any enzyme.

175 IF show a pattern of NF-kappaB dynamics that is unaltered from wild-type cells, but activation of the

176 tetramer formation by the mutant P proteins was unaltered from that for wild-type P.

177 SGLT-1 protein expression was unaltered; however, HS increased ileal GLUT-2 protei

178 s Ca(2+) release events and the rate of DADs were unaltered; however, DADs had lower amplitude in het

179 reduced if mice expressed reduced A1Rs, and were unaltered if mice lacked A1Rs.

180 Because Ees is unaltered, improved ventricular-arterial coupling is

181 neous and psychostimulant-induced locomotion are unaltered in miR-133b null mice, suggesting that miR

182 cyte development and naive T cell activation are unaltered in the absence of WASH.

183 nsistent with findings that vGAT mRNA levels are unaltered in the illness and confirming that the num

184 es, and abscisic acid levels and sensitivity are unaltered in the rdo5 mutant.

185 e, we reveal that two small molecule ligands are unaltered in their ability to allosterically modulat

186 form of balanced binocular suppression that is unaltered in amblyopia.

187 Here, we show that resistance to GPA is unaltered in an eds1 salicylic acid induction deficie

188 Given that beta2AR expression is unaltered in CHF, a beta-arrestin-biased agonist that

189 ocalization of other transmembrane receptors is unaltered in chp-1(lf) mutants, and the inhibition of

190 proteins with known roles in CMA production is unaltered in cmaL mutants.

191 RFP-Ldgp63 in GFP-LdRab1:WT-expressing cells is unaltered in comparison with control cells.

192 loss of miR-29a, because miR-29b expression is unaltered in miR-29a/b-1-null HSCs, and only ectopic

193 We found that Akt activation is unaltered in Mtmr13(-/-) and Mtmr2(-/-) mice.

194 e orientation of cortical microtubule arrays is unaltered in rhm1 mutants.

195 he glutamatergic astrocyte-neuron signalling is unaltered in the GAERS thalamus, the changes in some

196 ally bears no resemblance to RS1 and RS2 and is unaltered in the pf17 mutant.

197 Although apoptosis is unaltered in these mice, they recover more rapidly fr

198 tamate receptors in the hippocampal CA1 area is unaltered in TRPC5 KO mice, but is abolished in TRPC1

199 essing of APP and other neuronal substrates, was unaltered in 4CA mice despite the lack of BACE1 S-pa

200 sting GABA concentration decreased in V1 but was unaltered in a control parietal area.

201 HSV-1 entry was unaltered in both cells treated with small interferi

202 P than littermate controls, whereas basal BP was unaltered in Cdh5-CreERT2 Nox2KO mice (in which Nox2

203 dritic markers, including synaptic vesicles, was unaltered in cdk-5 mutant DB neurons.

204 Slc6a15 protein was unaltered in ChAT interneurons.

205 Although the total AMPK activity was unaltered in cKO hearts because of upregulation of g

206 or load-bearing protein within muscle cells, was unaltered in CP.

207 d integrin alphaIIbbeta3 affinity regulation was unaltered in Dok-2(-/-) platelets, Dok-2 deficiency

208 psy-induced mutation ct(6) and found that it was unaltered in dtopors homozygotes.

209 w from the Wolffian duct; however, apoptosis was unaltered in E12.5 mutant CNDs.

210 abolished in Epac2-KO and double-KO mice but was unaltered in Epac1-KO mice.

211 rs was reduced in WT mice with arthritis and was unaltered in Fas(-/-) mice.

212 ogalacturonide (OG)-induced immune signaling was unaltered in gae1 gae6 mutant plants, immune signali

213 Additionally, SphK2 expression was unaltered in lupus patients.

214 2(+) neurons, although D(2) receptor density was unaltered in Mecp2(+/-) mice.

215 Because lipid production was unaltered in meibomian glands from Dsg3-deficient mi

216 The genomic H3K27me3 landscape was unaltered in mutant tissue, and support for a demeth

217 yanodine receptor fractional phosphorylation was unaltered in pAF, whereas ryanodine receptor express

218 d by glgA, was greatly reduced in CTD153, it was unaltered in plasmid-deficient C. muridarum strains.

219 However, SIG6 expression was unaltered in pp7l mutants.

220 ssion onto parvalbumin-positive interneurons was unaltered in PSD-95 KO mice.

221 ted protein of mammalian target of rapamycin was unaltered in response to both resistance exercise an

222 phology of the cells in the epithelial layer was unaltered in the absence of Cadm1.

223 ice, whereas the GI eosinophil turnover rate was unaltered in the absence of CD22.

224 uridarum infection, C. trachomatis infection was unaltered in the absence of CD4(+) T cells.

225 Though the embryonic eye morphogenesis was unaltered in the Klf5CN mice, postnatal maturation w

226 Neuromuscular junction number was unaltered in the medial rectus muscles, but were sig

227 Even though basal synaptic transmission was unaltered in the neuromuscular synapses in IM-AA mic

228 This dopamine effect was unaltered in the presence of L-, R- and T-type calci

229 a-adrenergic regulation of the cardiac I(Ca) was unaltered in these mouse lines.

230 nt CD4(+) T cell response to M. tuberculosis were unaltered in all genotypes tested.

231 Protein and ash proportions were unaltered in any of the treatments.

232 In contrast, GAD(67) mRNA levels were unaltered in CB1R(+/-) andCB1R(-/-) mice.

233 we found that the cytokine and Ab responses were unaltered in CDG/Ag-immunized IFNAR(-/-) mice.

234 oreactivity ex vivo and hemodynamics in vivo were unaltered in DDAH1(En-/-) mice.

235 nd its inhibitory effect on IL-12 production were unaltered in Dusp16tp/tp macrophages.

236 fold, respectively, in ANDV-infected ECs but were unaltered in ECs infected by the nonpathogenic Tula

237 , PNN densities in the primary visual cortex were unaltered in either condition.

238 roducts of these enzymes (ie, RvD1 and RvD2) were unaltered in freshly isolated SMG cells suggesting

239 sed, and diacylglycerol and ceramide content were unaltered in gastrocnemius muscle from ob/ob-gamma3

240 s, T regulatory cells, and T-helper 17 cells were unaltered in HIV+ men, CD8 T-cell production of tum

241 Whereas BK currents and channel localization were unaltered in IHCs from Lrrc26 knockout (KO) mice, B

242 ltase, two major hepatic alpha-glucosidases, were unaltered in L-G6pc(-/-) mice, pharmacological inhi

243 E1 as well as the antiapoptotic gene bcl-xl were unaltered in LRH-1 expressing islets.

244 expression compared with limb muscles, which were unaltered in microgravity.

245 a2+ current, and Na+ /Ca2+ -exchange current were unaltered in pAF, indicating the absence of AF-indu

246 whole-body knockouts, brain manganese levels were unaltered in pan-neuronal/glial Slc30a10 knockouts

247 ced vaso-obliteration and neovascularization were unaltered in Par2 knockout mice, suggesting compens

248 oxygen consumption and fatty-acid oxidation were unaltered in primary HNKO hepatocytes.

249 Other biophysical channel properties were unaltered in R1389H channels including ion selectiv

250 Aldehyde dehydrogenase-2 levels were unaltered in response to alcohol, suggesting that t

251 Conversely, RGC survival and axon growth were unaltered in RGCs from AC1/AC8 double knock-out mic

252 While whole tissue respiratory rates were unaltered in roots and shoots, TCA cycle intermedia

253 ion and stimulation of glucose disappearance were unaltered in RYGB subjects.

254 n low-density lipoprotein receptor knockouts were unaltered in TC knockouts.

255 ross-bridge turnover and detachment kinetics were unaltered in TG fibers.

256 ure development and rate of relaxation (tau) were unaltered in TG(S282A) mice.

257 yte selection and peripheral TCR sensitivity were unaltered in the absence of Shp1.

258 Plasminogen mRNA levels were unaltered in the AD or VaD groups compared to the c

259 phorylated tau levels and ubiquitin staining were unaltered in the IFN-gamma cohorts.

260 ity during inflammation of the skin and lung were unaltered in the mutant mice.

261 charide (LPS) surface charge and aggregation were unaltered in the presence of OligoG CF-5/20.

262 r, SCA feeding-induced salicylic acid levels were unaltered in the sorghum tolerant genotype.

263 RNA replication and viral titre were unaltered in viruses possessing only one mutated st

264 The propagation velocity of the CSD waves was unaltered indicating stable neuronal excitability.

265 The levels of other lipid classes were unaltered indicating that AIG1 and ADTRP specifical

266 However, PLANT DEFENSIN1.2 expression was unaltered, indicating a repressor role for LBD20 in

267 e expression of Sost encoding for sclerostin was unaltered, indicating that osteoblastic Lrp4 retains

268 eurons and GODZ or SERZ-beta KO brain slices were unaltered, indicating that GODZ-mediated palmitoyla

269 Although total JPH2 levels were unaltered, JPH2 phosphorylation levels were found t

270 rotated in the opposite direction (FAST) or was unaltered (NATURAL).

271 We show that glutamate transmission is unaltered onto striatal projection neurons (SPNs) of

272 In contrast, these transcripts were unaltered, or only modestly changed, in parvalbumin

273 es of Schwann cell-specific Pex5 mutant mice were unaltered regarding axon numbers, axonal calibers,

274 In these mice, basal DA release or uptake was unaltered relative to controls, but attenuation of m

275 Although early phases of neural development were unaltered, Slc7a5 knockdown effected late phases of

276 Although fetal weight is unaltered, specific Rtl1 transcripts and protein leve

277 he decay of caffeine-induced Ca2+ transients was unaltered, suggesting increased SERCA2a function.

278 sion of T-reg/Th2 markers after GC treatment was unaltered, suggesting that T-cell-driving NP inflamm

279 However, IL-17A and IL-17C expression was unaltered, suggesting that the increased inflammatio

280 (ie, REG3G, DEFB4A, S100A9, MUC1, and MUC13) were unaltered, suggesting an adequate production of ant

281 of the P/Q channel depleted layer VI neurons was unaltered, T-type calcium currents in the postsynapt

282 While KLF4 and PRDM1 levels were unaltered, the expression levels of KLF4 transcript

283 ly releasable SV pool size, and quantal size were unaltered, the reduced synaptic strength in the abs

284 ency in the peripheral blood of T1D patients is unaltered, their suppressive abilities are diminished

285 se curvature and tortuosity of the bile duct are unaltered, this enlargement of the biliary tree is c

286 Although overall cardiomyocyte dimensions were unaltered, total surface area was increased.

287 The tyrosine and phenylalanine pathways were unaltered under both conditions.

288 mitotic checkpoint because k-MT attachments are unaltered upon Mad1 depletion and Mad2 overexpressio

289 splays a unique sedimentation profile, which is unaltered upon starvation-induced autophagy.

290 Connectivity is unaltered when blue cone transmission is suppressed.

291 However, DNA polymerase fidelity is unaltered when these modifications are in the templat

292 However, the VDAC level was unaltered when the phosphatase-inactive mutant of Tb

293 ose together or touching, whereas judgements were unaltered when adjacent fingers were stimulated.

294 eron, the MeC status of bulk chromosomal DNA is unaltered, whereas both MeC and C nucleobases in tran

295 -induced activation of sympathetic signaling was unaltered, whereas AMPK was enhanced, in AdKO IWAT.

296 acemaker, the suprachiasmatic nucleus (SCN), is unaltered while the molecular clock in the hippocampu

297 In this unpaired setting, ApoE levels were unaltered, while ApoA1 was reduced in the anaphylac

298 Unexpectedly, arogenate levels were unaltered, while shikimate and Trp levels were decr

299 NKA-PLM FRET was unaltered with F956A or F967A, reduced with L964A, a

300 resting membrane potential, basic properties were unaltered with age, including neuronal Cl(-) homeos