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1 premature menopause is associated with CHIP is unknown.
2 n in tissue versus infiltrating immune cells is unknown.
3 a neuron-activating, cough-inducing molecule is unknown.
4 spatio-temporal dynamics of auxin gradients is unknown.
5 piratory syndrome coronavirus 2 (SARS-CoV-2) is unknown.
6 ion of this mark within the infectious cycle is unknown.
7 Acomys response to spinal cord injury (SCI) is unknown.
8 mors, such as small-cell lung cancer (SCLC), is unknown.
9 us has been measured, but vertical structure is unknown.
10 The function of Nur77 in iNKT cells is unknown.
11 The enzyme introducing the Delta14Z DB is unknown.
12 the authors the association with tau burden is unknown.
13 tributes to this neurodegenerative condition is unknown.
14 oron on cardiac contractility and remodeling is unknown.
15 sion of atherosclerosis in patients with CKD is unknown.
16 ophy and failure in congenital heart disease is unknown.
17 to enhancers, though the mechanism involved is unknown.
18 s megakaryocyte-specific PLAU overexpression is unknown.
19 s dominates during aging of individual cells is unknown.
20 tations of these stationary sounds, however, is unknown.
21 condensation and nuclear envelope breakdown is unknown.
22 eral in vivo requirement for these functions is unknown.
23 als supporting contemporary anticancer drugs is unknown.
24 h these fibroblasts differentiate and expand is unknown.
25 impact on cardiovascular health and survival is unknown.
26 confers on endogenous cholinergic signaling is unknown.
27 ell as the biological role of m3C in mammals is unknown.
28 However, its role in AKI is unknown.
29 he timing and cellular location of uncoating is unknown.
30 l-expressed by myeloid cells, where its role is unknown.
31 ation posed by structured substrate proteins is unknown.
32 o features of atopy or mild asthma in adults is unknown.
33 eceptors have uniquely obtained this ability is unknown.
34 EHS are mediated by epigenetic reprogramming is unknown.
35 consequence of LEC priming of CD8(+) T cells is unknown.
36 ow the receptor could escape desensitization is unknown.
37 Yet the mechanism of glial tau transmission is unknown.
38 evelopmental, genetic and phylogenetic level is unknown.
39 in reptiles but the physiological mechanism is unknown.
40 , the optimal level of pharmacologic support is unknown.
41 named EACBE) in dynamic chromatin regulation is unknown.
42 ink between COPA mutations and IFN signaling is unknown.
43 cardiometabolic risk among African Americans is unknown.
44 ing or regulating active avoidance responses is unknown.
45 loss (HL) in highly seropositive populations is unknown.
46 its impact on pancreatic beta-cell function is unknown.
47 lerance-inducing effect of alcohol, however, is unknown.
48 onal significance of such orderly patterning is unknown.
49 anslation coupling mitigates these conflicts is unknown.
50 t of 10 cases, the molecular cause, however, is unknown.
51 omerizes to mediate the assembly of virions, is unknown.
52 role of Lgmn in PAR(2)-dependent cancer pain is unknown.
53 ructure and account for NF1 imaging findings is unknown.
54 m elevation necessary to induce pancreatitis is unknown.
55 onse to physiological or pathological stress is unknown.
56 iretrovirals to chronic kidney disease (CKD) is unknown.
57 alyzing this post-translational modification is unknown.
58 phocyte navigation, the nature of this pilot is unknown.
59 but how expression variations are generated is unknown.
60 r, how cyclin L2 is regulated in macrophages is unknown.
61 ukaryotes, the molecular function of the RSs is unknown.
62 ver, the mechanism underlying DR5 activation is unknown.
63 ischemia-reperfusion injury and regeneration is unknown.
64 How the brain motivates this behavior is unknown.
65 poxia on negative regulators of angiogenesis is unknown.
66 avage of RIPK1 on RIPK1 activation in humans is unknown.
67 ned; however, the efficacy of h5B3.1 in vivo is unknown.
68 is endemic in Nepal and its economic burden is unknown.
69 ng exercise heart rate are impaired in HFpEF is unknown.
70 Whether these associations persist over time is unknown.
71 contributes to TSC-associated tumorigenesis is unknown.
72 this signal is received by epithelial cells is unknown.
73 orientation signals for updating grasp plans is unknown.
74 lecular machinery that links these processes is unknown.
75 w these variants might contribute to disease is unknown.
76 mportance of the diencephalon in TCA mapping is unknown.
77 hese peripheral alterations influence eating is unknown.
78 The pathogenesis of idiopathic CMV disease is unknown.
79 tion associated with intravitreal injections is unknown.
80 l solution when data are noisy and the model is unknown.
81 the mechanistic basis for TASK2 gating by pH is unknown.
82 anism behind the migraine-stroke association is unknown.
83 cardiac arrest (RSCA) on a population basis is unknown.
84 ut how SIRT6 is able to accomplish this feat is unknown.
85 modulates locomotion through motor circuits is unknown.
86 n (AF) in reference to the time of diagnosis is unknown.
87 , the contribution of lymphatics to fibrosis is unknown.
88 however, the durability of immunity to ZIKV is unknown.
89 ne triphosphate (3TC-TP) exposure in the FGT is unknown.
90 id use in patients undergoing lung resection is unknown.
91 ween mitochondrial function and angiogenesis is unknown.
92 host-derived H(2)S in microbial pathogenesis is unknown.
93 ity are a higher risk for transplant failure is unknown.
94 s represent genuine psychobiological effects is unknown.
95 s of plant species composition for infection is unknown.
96 nd therefore compromise prevention efficacy, is unknown.
97 ry phenotype associated with CAD progression is unknown.
98 en-specific T cell receptor (TCR) repertoire is unknown.
99 The role of TET2 in PAH is unknown.
100 Hitherto, their role in cancer is unknown.
101 rejection assessment for patient management is unknown.
102 bacteria-specific or tumor-specific immunity is unknown.
103 d into the brain to drive migratory behavior is unknown.
104 the functional relevance of this interaction is unknown.
105 published and the true PeV infection burden is unknown.
106 the mechanism underlying these associations is unknown.
107 to transcription regulation in Ewing sarcoma is unknown.
108 uding increased exacerbation susceptibility, is unknown.
109 d eligibility criteria and bridging therapy, is unknown.
110 nic liver injury sequelae, such as fibrosis, is unknown.
111 in regions is established during development is unknown.
112 ing the need for red blood cell transfusions-is unknown.
113 Its molecular basis is unknown.
114 y factors that distinguish human populations is unknown.
115 tial role in Pel-dependent biofilm formation is unknown.
116 y-onset calcium oxalate kidney stone disease is unknown.
117 -mediated bacterial-plant/algal interactions is unknown.
118 chanistic role in ANCA-associated vasculitis is unknown.
119 me and sudden unexplained death in the young is unknown.
120 tribution to the clinical course and outcome is unknown.
121 te-to-large primary spontaneous pneumothorax is unknown.
122 V lineages but not others evolve in this way is unknown.
123 illimeter distributed representations in VTC is unknown.
124 y among children initially surviving sepsis, is unknown.
125 x differences observed in the clinical arena is unknown.
126 ing and receptor recycling are more complex, is unknown.
127 is pathway is active in the developing brain is unknown.
128 y congestion carries prognostic implications is unknown.
129 eservoir of HIV-bound immune complexes (ICs) is unknown.
130 but the structure of the central core module is unknown.
131 mbination with intensive chemotherapy in AML is unknown.
132 illness severity correlates with infectivity is unknown.
133 nt and infectiousness in contact individuals are unknown.
134 r mechanisms that precipitate plaque rupture are unknown.
135 but other cytokines involved in pathogenesis are unknown.
136 of El Nino cycles on this key source region are unknown.
137 senolytic drugs on vision in human patients are unknown.
138 ch the cerebellum impacts seizures, however, are unknown.
139 The determinants of this process are unknown.
140 foods on glutamatergic plasticity in females are unknown.
141 aryotes; however, their molecular activities are unknown.
142 impairments in males, but effects on females are unknown.
143 tterns in NSTIs and their impact on outcomes are unknown.
144 h which these biological effects are exerted are unknown.
145 reversible thiolate/disulfide transformation are unknown.
146 the mechanisms by which Abeta affects sleep are unknown.
147 ate models; however, the mechanisms involved are unknown.
148 the factors contributing to this observation are unknown.
149 e neuronal milieu towards increased activity are unknown.
150 r uric acid (UA) crystals in CKD progression are unknown.
151 (mCD40L) on its immune-stimulatory function are unknown.
152 nonadherence in the etiology of elevated VL are unknown.
153 The pathogenesis and molecular features are unknown.
154 of HIV and tuberculosis on COVID-19 outcomes are unknown.
155 isease but the precise pathogenic mechanisms are unknown.
156 nts differentially contribute to weight gain are unknown.
157 emory recall, and subsequent food motivation are unknown.
158 ins vital functions prior to tissue recovery are unknown.
159 on of hyperglycemia with depressive symptoms are unknown.
160 gical mediators of this adverse relationship are unknown.
161 that allow preservation of the chloroplasts are unknown.
162 n eosinophils, particularly upon activation, are unknown.
163 horus control in patients receiving dialysis are unknown.
164 cells and specifically in neurite outgrowth are unknown.
165 vity to GLP-1, yet the underlying mechanisms are unknown.
166 ence, the validity of decision-making models are unknown.
167 persons with substance use disorders (SUDs) are unknown.
168 anisms underlying the observed comorbidities are unknown.
169 T1D inflammatory milieu on these mechanisms are unknown.
170 groups of resistant and susceptible strains are unknown.
171 the mechanisms underlying these aberrancies are unknown.
172 ian species, but their ancestral function(s) are unknown.
173 and the yield of these lesions after recall are unknown.
174 phatic pumping activity in MetSyn conditions are unknown.
175 cognitive enhancement but precise mechanisms are unknown.
176 experienced fluctuations in SAM availability are unknown.
177 ory failure requiring mechanical ventilation are unknown.
178 recognize and process these atypical chains are unknown.
179 and the molecular mechanism of potentiation are unknown.
180 for the elevated expression of NKG2D ligands are unknown.
181 expressed only after several decades of life are unknown.
182 enting with an acute coronary syndrome (ACS) are unknown.
183 nd T cell receptor signaling to this process are unknown.
184 nd pathways of transfer in fluvial sediments are unknown.
185 nd functions of PROM1(+) cells in AH and HCC are unknown.
186 through this long presynaptic nerve terminal are unknown.
187 nics governing TRM cell reactivation in situ are unknown.
188 gh Kidney Donor Profile Index (KDPI) kidneys are unknown.
189 onsible for mediating this protective effect are unknown.
190 the triggers initiating aberrant methylation are unknown.
191 ent cycling, however, related silicon fluxes are unknown.
192 basophils play in regulating these responses are unknown.
193 llular mechanisms of carotid body activation are unknown.
194 s diagnostic or prognostic molecular markers are unknown.
195 tive immune responses with neurodegeneration are unknown.
196 d the response of PDAC cells to radiotherapy are unknown.
197 neurocognitive mechanisms of this phenomenon are unknown.
198 icacy and safety of selective RET inhibition are unknown.
199 RyR2 inactivation leads to Ca(2+) alternans are unknown.
200 dication costs across the health care system are unknown.
201 osition, yet its effects on disease outcomes are unknown.
202 disease (PD), but its molecular determinants are unknown.
203 Demographic and genetic consequences are unknown.
204 itive allosteric modulation of the receptor, are unknown.
205 specify the mammalian trachea and esophagus are unknown.
206 ontext of Candida and Aspergillus infections are unknown.
207 However, the additional risks of its use are unknown.
208 potential influence on long-term durability are unknown.
209 nd factors associated with this variability, are unknown.
210 regimes on TE dynamics in a specific genome are unknown.
211 ade transport on the organelles and the axon was unknown.
212 flicker on neuroimmune biochemical signaling was unknown.
213 , although where it intersects GLUT4 traffic was unknown.
214 l now, however, the protective mechanism has been unknown.
215 olecular mechanisms involved in this process were unknown.
218 pendent responses, and underlying mechanisms are unknown and its role in human conditions has not bee
219 tuximab can clear MRD, but long-term results are unknown and optimal timing of rituximab undefined.
221 mmune cells contributing to oxidative stress is unknown, and therapies to target their neurotoxic fun
223 However, the sources of this LHb activation are unknown, as the LHb receives many excitatory inputs
224 e is a molecular basis for these disparities is unknown, as very few Hispanic/Latino patients have be
228 tolerates these multi-stressor environments is unknown Core cooling by 1.0 degrees C reduced cerebra
229 the effect of gut inflammation on this axis is unknown despite reports of taste changes in gastroint
230 e potential of adipocyte progenitors in vivo is unknown (Faust et al., 1976; Faust et al., 1977; Hirs
232 of protection conferred by natural immunity is unknown for many enteropathogens, but it is important
234 ular antigens has been studied in detail, it is unknown how a lifetime of exposures to many antigens
239 nd cDC2s, respectively) is well accepted; it is unknown how robust this dichotomy is under inflammato
242 read in solid surrounding healthy tissue, it is unknown how softer tumors can grow within a more rigi
247 ey transplants or play any role in rejection is unknown, however, in part because of limited techniqu
249 ess FA in cells is known to be cytotoxic, it is unknown if an increase in FA level might be associate
250 However, this is not well defined and it is unknown if exercise timing induces directional shifts
253 eral and bone disorder have been defined, it is unknown if persistent phosphate wasting in nephropath
254 lular carcinoma (HCC) as compared to men; it is unknown if there are sex-based differences in HCC rec
255 though the pathogenesis of bipolar disorders is unknown, implicated processes include disturbances in
261 tent of this poorly ionizable pool of carbon is unknown, is presumably variable between samples, and
263 ients with premorbid low/high blood pressure are unknown.Objectives: To investigate the relationship
264 hether IGF-1 treatment can prevent BPD or PH is unknown.Objectives: To evaluate whether postnatal tre
265 biological significance of these infections is unknown.Objectives: We aimed to evaluate longitudinal
268 ne protein, but its topological organization is unknown, raising questions about the subcellular loca
272 nce on empirical data where the ground truth is unknown, we introduce a phylogeny-based measure for i
273 l of these processes consume energy(4,5), it is unknown whether and how cells adapt their metabolic a
275 remain sensitive to touch for weeks, but it is unknown whether calves experience ongoing, non-evoked
278 tical neurons with shared sensory inputs, it is unknown whether correlated noise limits coding fideli
280 nflammation could impair immune response, it is unknown whether inflammatory markers are associated w
283 croenvironment (TME) remains limited, and it is unknown whether it is sculpted differentially by prim
285 diseases and modulated by microRNAs, but it is unknown whether microglial microRNAs have sex-specifi
287 s facilitate chromatin transcription, but it is unknown whether Pol I has an intrinsic capacity to tr
288 infections of the respiratory tract, but it is unknown whether sphingosine can be also employed to p
290 ssociated with rheumatoid arthritis (RA), it is unknown whether tenosynovitis of the forefoot is asso
293 tid genome-wide level and for many genes, it is unknown whether their expression responds to altered
298 vivo While VZV is known to grow in skin, it was unknown whether skin could support an HCMV infection