ライフサイエンスコーパス: beat-to-beat

1 ivation and amplitude of release varied from beat to beat.

2 Ca(2+) release from these sites varied from beat to beat.

3 ts indicating a change in contractility from beat to beat.

4 s occur with durations varying randomly from beat to beat.

5 Despite the key role of beat-to-beat action potential (AP) variability in the on

6 , increasing refractoriness, and diminishing beat-to-beat action potential variability.

7 cium entry from extracellular spaces for its beat-to-beat activation.

8 rm for addressing heart disease and enabling beat-to-beat adaptation of cardiac pacing in response to

9 ow over several minutes) and dynamic (rapid, beat-to-beat adjustments) autoregulation.

10 ng, and were not merely passive responses to beat-to-beat alterations in AP; 3) the complex Ca(2+) dy

11 t modulation of stroke volume (SV) caused by beat-to-beat alterations in left ventricular filling, wh

12 is study appears to be largely the result of beat-to-beat alternans of [Ca2+]i.

13 idents such as early afterdepolarization and beat-to-beat alternans.

14 own that small depolarizing pulses produce a beat to beat alternation in the amplitude of the systoli

15 Beat-to-beat alternation (alternans) of the cardiac acti

16 Cardiac alternans refers to a beat-to-beat alternation in contraction, action potentia

17 tive trigger event for cardiac reentry, is a beat-to-beat alternation in membrane potential and calci

18 A beat-to-beat alternation in the action potential duratio

19 heart, in which rapid stimulation elicits a beat-to-beat alternation in the action potential duratio

20 Ca(2+) alternans, a beat-to-beat alternation in the amplitude of the [Ca(2+)

21 aim of this work was to investigate whether beat-to-beat alternation in the amplitude of the systoli

22 Beat-to-beat alternation in the cardiac intracellular Ca

23 Alternans, a condition in which there is a beat-to-beat alternation in the electromechanical respon

24 henomenon of T-wave alternans (TWA) (i.e., a beat-to-beat alternation in the morphology and amplitude

25 complementary mechanism, CTA is caused by a beat-to-beat alternation in the number of refractory RyR

26 Alternans, the beat-to-beat alternation in the shape of cardiac electri

27 n the surface ECG was explained primarily by beat-to-beat alternation in the time course of cellular

28 CLs of 300 to 250 ms caused more pronounced beat-to-beat alternation of action potential duration (A

29 Although this alternation results from beat-to-beat alternation of intracellular Ca(2+) wave pr

30 uced mild to moderate TWA principally due to beat-to-beat alternation of repolarization of cells in t

31 +) alternans is a potentially arrhythmogenic beat-to-beat alternation of the amplitude of the action

32 ns (TWA), an ECG phenomenon characterized by beat-to-beat alternation of the morphology, amplitude, a

33 a period doubling bifurcation, manifest as a beat-to-beat alternation, or alternans, of cardiac actio

34 Increased AP duration (APD) and beat-to-beat alternations in AP morphology lowered the p

35 ICaCC played a decisive role in shaping the beat-to-beat alternations in AP morphology observed duri

36 ular level, cardiac alternans is observed as beat-to-beat alternations in contraction strength, actio

37 ular level, cardiac alternans is observed as beat-to-beat alternations in contraction strength, actio

38 the cellular level alternans is observed as beat-to-beat alternations in contraction, action potenti

39 Cardiac alternans, described as periodic beat-to-beat alternations in contraction, action potenti

40 Cardiac alternans--periodic beat-to-beat alternations in contraction, action potenti

41 At the cellular level alternans manifests as beat-to-beat alternations in contraction, action potenti

42 ever, it remains an unresolved issue whether beat-to-beat alternations in intracellular Ca(2+) ([Ca(2

43 Beat-to-beat alternations of AP morphology and CaT ampli

44 Beat-to-beat analysis of up to 4 full days of electrocar

45 propagation became completely variable from beat to beat and thus transformed into fibrillatory cond

46 Cardiac myocyte intracellular calcium varies beat-to-beat and calmodulin (CaM) transduces Ca2+ signal

47 I(NaL) inhibition significantly reduced the beat-to-beat and short-term variabilities of APD.

48 of Ca(2+) -activated Cl(-) channels reduced beat-to-beat AP alternations, but prolonged APD and fail

49 The beat-to-beat association between HR and MCA-PI increased

50 as characterized by significant increases in beat-to-beat atrial CL, MAPD, and diastolic interval var

51 Furthermore, the beat-to-beat autonomic reflex control of HR was found to

52 ction to increases in myocardial demand on a beat-to-beat basis and mitochondrial calcium release dep

53 eleration of conduction was compensated on a beat-to-beat basis by an equal degree of slowing in the

54 Atrial flutter is modulated on a beat-to-beat basis by an interplay between the autonomic

55 gesting that NCX is regulated by Ca(2+) on a beat-to-beat basis during excitation-contraction couplin

56 tivity to baroreflex (CSB) were derived on a beat-to-beat basis from these data.

57 d mechanisms for the occurrence of EADs on a beat-to-beat basis have been proposed.

58 low probe placed around the aortic root on a beat-to-beat basis in seven anesthetized open-chested ca

59 egulate cardiac pacemaker cell function on a beat-to-beat basis remains unknown.

60 oop that modulates the heart's function on a beat-to-beat basis to control arterial pressure.

61 regulates CL in cardiac pacemaker cells on a beat-to-beat basis, and suggest a more realistic numeric

62 ther mitochondria take up Ca2+ rapidly, on a beat-to-beat basis, or slowly, by temporally integrating

63 daily living, especially when measured on a beat-to-beat basis.

64 f cardiac muscle contraction is altered on a beat-to-beat basis.

65 adjust cardiac performance or perfusion on a beat-to-beat basis.

66 ructed aortic waveforms were correlated on a beat-to-beat basis.

67 ventricular filling with cardiac output on a beat-to-beat basis.

68 Measured beat-to-beat BF time history derived from real-time phas

69 Beat-to-beat blood pressure (BP) measurements were colle

70 al Doppler ultrasound along with noninvasive beat-to-beat blood pressure (BP), heart rate, and transc

71 reflex sensitivity (BRS) with ambulatory and beat-to-beat blood pressure (BP).

72 microneurography, 12 paired recordings), and beat-to-beat blood pressure (BP; photoplethysmography) d

73 Beat-to-beat blood pressure (finger photoplethysmography

74 ontinuous heart rate (electrocardiogram) and beat-to-beat blood pressure (finger plethysmography) wer

75 temporal sequence over 5 years by recording beat-to-beat blood pressure and R-R intervals over 10 mi

76 ension, and orthostatic hypotension based on beat-to-beat blood pressure methods in a population-repr

77 nt study derived BPV metrics from continuous beat-to-beat blood pressure monitoring data.

78 urements of heart rate (ECG) and noninvasive beat-to-beat blood pressure recording (Finapres), with 5

79 Beat-to-beat blood pressure responses to the Valsalva ma

80 bility may be the most reliable and specific beat-to-beat blood pressure variability metric due to it

81 Heart rate, noninvasive beat-to-beat blood pressure, and muscle sympathetic nerv

82 Heart rate, beat-to-beat blood pressure, and Vanderbilt Orthostatic

83 Beat-to-beat blood pressure, carotid ultrasonography at

84 od leading to vasovagal syncope we monitored beat-to-beat blood pressure, heart rate (HR) and forearm

85 s were instrumented for electrocardiography, beat-to-beat blood pressure, respiratory rate, CO-Modelf

86 tic nerve activity (MSNA; microneurography), beat-to-beat BP (photoplethysmography) and heart rate (e

87 Ambulatory beat-to-beat BP was recorded using a portable device for

88 Resting MSNA, respiration and beat-to-beat BP were recorded in 20 T2D (49.1 +/- 7.4 ye

89 nd frequency domain HRV indices, BRS, office beat-to-beat BP, and heart rate (HR) were measured for 1

90 Beat-to-beat BP, R-R interval and respiratory excursions

91 ons among HRV, HR, BRS and ambulatory/office beat-to-beat BP.

92 paucity of existing literature investigating beat-to-beat BPV in clinically stable post-stroke patien

93 activity is essential in the maintenance of beat-to-beat Ca(2+) homeostasis in cardiac myocytes.

94 ive stress, also have deleterious effects on beat-to-beat [Ca(2+)](c) handling and excitation-contrac

95 We propose that beat-to-beat [Ca(2+)](i) transient alternans during isch

96 Compared with BsCaM-2, BsCaM-45 tracks the beat-to-beat Ca2+-CaM alterations more closely following

97 ulse contour analysis can be used to provide beat-to-beat cardiac output (CO) measurement.

98 c resonance imaging of brain and noninvasive beat-to-beat cardiovascular monitoring, we show that sti

99 Our newly developed assessment of beat-to-beat carotid diameters during baroreflex engagem

100 lays a significant role in the regulation of beat-to-beat CBF in humans.

101 ially mediating the association with greater beat-to-beat cerebral pulsatility (average DeltaMCA-PI v

102 early afterdepolarizations (EADs) result in beat to beat changes in the origin and direction of the

103 phic ventricular arrhythmias may result from beat to beat changes in wave propagation patterns initia

104 ith AF, no current models can both replicate beat-to-beat changes during AF and be fitted to individu

105 CaT alternans leads to complex beat-to-beat changes in Ca(2+)-regulated ion currents th

106 lts in acceleration and amplification of the beat-to-beat changes in cytosolic Ca(2+) in cardiomyocyt

107 Beat-to-beat changes in HR and MAP were recorded through

108 Spectral transfer function gain between beat-to-beat changes in left ventricular end-diastolic p

109 2 (n = 2 out of 5) showed persistent complex beat-to-beat changes in nodal line formation of DA assoc

110 Beat-to-beat changes in renal blood flow velocity (RBV;

111 hroughout the heart at high speeds producing beat-to-beat changes in the activation sequence.

112 total cellular fluorescence failed to track beat-to-beat changes of mitochondrial fluorescence.

113 orresponding to progressively higher rate of beat-to-beat CL changes.

114 nsidered an accurate technique for measuring beat-to-beat CO with limited risk to the patient.

115 a highly specialized role in regulating the beat-to-beat contraction of the heart.

116 and exchangers, but are actively involved in beat to beat control of cardiac function by neural and h

117 We investigated the beat-to-beat control of atrial flutter cycle length usin

118 t fluctuations in heart rate responsible for beat-to-beat control of heart activity, both at rest and

119 In conclusion, beat-to-beat dispersion and sequence-based metrics of BP

120 e oscillations are irregular and change from beat to beat due to the sensitivity of voltage repolariz

121 Analysis of multiscale complexity of beat-to-beat dynamics at high temporal resolution has po

122 or blood pressure, quantification of complex beat-to-beat dynamics using multiscale entropy was able

123 frequency, kurtosis, and higher degree of a beat-to-beat electrogram similarity than areas without o

124 I sequences (RT-PC) can provide a continuous beat-to-beat flow signal that makes it possible to quant

125 DCM is associated with beat-to-beat fluctuations in QT interval that are larger

126 T-wave alternans (TWA) reflects beat-to-beat fluctuations in the electrocardiographic T-

127 veloped a computational model that simulates beat-to-beat haemodynamic changes resulting from the unc

128 e lacking the capabilities to both replicate beat-to-beat haemodynamic variations during AF at the sa

129 Beat-to-beat haemodynamics (Modelflow), muscle sympathet

130 Beat-to-beat heart period, systolic blood pressure, and

131 Beat-to-beat heart rate and blood pressure responses to

132 To investigate the effects of DR on beat-to-beat heart rate and diastolic blood pressure var

133 Supine, resting, 2-min beat-to-beat heart rate data were collected at the basel

134 Baseline, supine, resting beat-to-beat heart rate data were collected.

135 tion of the instantaneous variability of the beat-to-beat heart rate): spontaneous swallowing 12.02 +

136 Beat-to-beat hemodynamic functions were determined nonin

137 , arrhythmia detection, calcium handling and beat-to-beat heterogeneity.

138 metoprolol or propranalol) conditions, while beat-to-beat HR and BP were continuously measured.

139 Beat-to-beat HR was strongly associated with concurrent

140 al contractility is constantly changing from beat to beat in atrial fibrillation because of the influ

141 At 13 minutes, hemodynamics was analyzed beat-to-beat in the end-inspiratory and end-expiratory c

142 In these cardiomyocytes, which produce NO beat-to-beat, inhibition of mtNOS increased myocyte shor

143 decreased with decreases in the steady-state beat-to-beat interval (P = .0008).

144 The short-term variations of beat-to-beat interval exhibited strongly and consistentl

145 multiscale complexity dynamics) measures of beat-to-beat interval variability were analyzed in two m

146 including standard deviation of the average beat to beat intervals over a 5-minute period, percentag

147 he NOX4 embryos displayed much more variable beat-to-beat intervals (mean S.D. of beat-to-beat interv

148 , the distributions of the variations in the beat-to-beat intervals for all healthy subjects are desc

149 However, sequences of beat-to-beat intervals obtained from these recordings ar

150 nces and sample entropy, calculated from the beat-to-beat intervals of the PPG signal, to distinguish

151 ariable beat-to-beat intervals (mean S.D. of beat-to-beat intervals was 0.027 s/beat in control embry

152 fraction </= 45%) and sleep apnea underwent beat-to-beat measurement of SV by digital photoplethysmo

153 based techniques are less invasive and offer beat-to-beat measurements and excellent trending ability

154 ed neck chamber that was developed to enable beat-to-beat measurements of stroke volume using pulse-d

155 4% for daytime, sleeping, 24-hour and office beat-to-beat measurements.

156 namics of the Starling mechanism, namely the beat-to-beat modulation of stroke volume (SV) caused by

157 a short, regular cycle length with identical beat-to-beat morphology, and the rest of the atria were

158 Beat-to-beat NO production and altered shortening by NOS

159 A by microneurography, and blood pressure by beat-to-beat noninvasive technique.

160 val and nonlinear analyses (newly developed, beat-to-beat nonlinear measurement of the repetitiveness

161 Dynamic, beat-to-beat or electrocardiogram-to-electrocardiogram,

162 We hypothesized that beat-to-beat oscillations in APD may explain AF substrat

163 arization instability, manifested by TWA and beat-to-beat oscillations of T-wave amplitudes at other

164 In this study, beat-to-beat pulse rate variability (PRV) and BPV were m

165 sing a semiautomated algorithm that measured beat-to-beat QT duration in 817 MADIT II patients.

166 Increased beat-to-beat QT interval variability (QTV) on the electr

167 We sought to test the hypothesis that beat-to-beat QT interval variability is increased in DCM

168 Beat-to-beat QT interval variability was measured by aut

169 ibit labile ventricular repolarization using beat-to-beat QT variability analysis.

170 udy was to determine the predictive value of beat-to-beat QT variability in heart failure patients ac

171 Beat-to-beat QT variability index (QTVI), log-transforme

172 n the heart and the brain is pivotal for the beat-to-beat regulation of cardiac function and the clos

173 noatrial node and atria play a major role in beat-to-beat regulation of the heart rate.

174 Beat-to-beat relationships between HR, blood pressure an

175 AP sequences were compared in terms of beat-to-beat restitution (btb-ER) and of the collections

176 s undertaken to produce fetal heart rate and beat-to-beat rhythm strips.

177 As expected, cardiac pacing eliminated beat-to-beat RR interval variability.

178 ctuations is reduced, and the time series of beat-to-beat RR intervals (RRIs) become highly non-stati

179 the fibrillatory cycle lengths with varying beat-to-beat sequences suggestive of unstable trajectori

180 Alternans is the periodic beat-to-beat short-long alternation in action potential

181 rnative mechanisms, we measured simultaneous beat-to-beat stroke volume (flow) using Doppler echocard

182 However, beat-to-beat stroke volume (SV) has not been assessed in

183 Accurate beat-to-beat stroke volume and BF were estimated using V

184 Beat-to-beat systolic BP, diastolic BP, and heart rate r

185 similar baseline characteristics but greater beat-to-beat TCL variability.

186 2+) uniporter catalyzes Ca(2+) uptake during beat-to-beat transients of mitochondrial free Ca(2+), wh

187 Thus, beat-to-beat V(m) fluctuations during late DD phase refl

188 phase to the DD later part, which exhibited beat-to-beat V(m) fluctuations with an amplitude of appr

189 Beat-to-beat validation of VMHDVCG-derived BF was perfor

190 ticity and intranodal conduction, leading to beat-to-beat variability and reentry.

191 mal culture conditions, MFS CMs show a lower beat-to-beat variability compared to corrected CMs using

192 Beat-to-beat variability in atrial cycle length (baselin

193 surface lead electrocardiograms by analyzing beat-to-beat variability in ECG morphology using a smart

194 istent Na current, a factor that may promote beat-to-beat variability in late Na current.

195 HIV+ men have greater beat-to-beat variability in QT interval (QTVI) than HIV-

196 horter effective refractory periods, greater beat-to-beat variability of action potential durations,

197 horter effective refractory periods, greater beat-to-beat variability of action potential durations,

198 max boundaries was associated with increased beat-to-beat variability of conduction velocity and dire

199 Beat-to-beat variability of left-ventricular monophasic-

200 d afterdepolarizations (DADs) and has a high beat-to-beat variability of repolarization (BVR) during

201 ease the action potential duration (APD) and beat-to-beat variability of repolarization (BVR) of APD

202 e of delayed afterdepolarizations (DADs) and beat-to-beat variability of repolarization (BVR) was hig

203 Excessive beat-to-beat variability of repolarization duration (BVR

204 assessed by spectral analysis of spontaneous beat-to-beat variability of RR and QT intervals from sta

205 fidence interval, 1.7 to 9.3), and decreased beat-to-beat variability of the heart rate (odds ratio,

206 heir effect on the beating frequency and the beat-to-beat variability seemed largely independent of t

207 ery near the pulmonary vein ostia, and their beat-to-beat variability was greater than control (1.93+

208 Models showed severe beat-to-beat variability with decreased overall autonomi

209 28S abbreviated refractoriness and increased beat-to-beat variability, leading to early afterdepolari

210 ice insertion, transmitral flow showed rapid beat to beat variation in each patient, from abnormal re

211 nchronous LV assistance produced significant beat to beat variation in filling indexes, but overall a

212 The cardiac beat-to-beat variation evoked at the moment of lung infl

213 Moreover, the beat-to-beat variation in arterial and throbbing events

214 Beat-to-beat variation in heart rate (f H ) has been use

215 Notably, in this study, beat-to-beat variation in left ventricular end-diastolic

216 A beat-to-beat variation in the cardiac action potential d

217 is crucial for stable heart rate and regular beat-to-beat variation.

218 Our data reveal rapid beat-to-beat variations in [ATP](i).

219 The mechanism of the small beat-to-beat variations in cycle length of atrial flutte

220 whereas ERP restitution underlies temporal, beat-to-beat variations in refractoriness during rapid p

221 In these examples, we observed large beat-to-beat variations in the cell activation times, de

222 The maps showed incessantly changing beat-to-beat wave fronts and varying spatiotemporal beha

223 Ca(2+) transients from beat to beat were comparable in amplitude with identical

224 ibility study by 2 leadless pacemakers using beat-to-beat, wireless communication, achieving a succes

225 Forearm blood flow (FBF) was measured beat-to-beat with Doppler ultrasound, while saline or dr

226 hat varied its location and orientation from beat to beat, with the majority of ventricular myocardiu