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1 equired for the development or expression of behavioral sensitization.
2  after chronic cocaine exposure that induced behavioral sensitization.
3 rug experience-dependent plasticity, such as behavioral sensitization.
4  function contributing to the development of behavioral sensitization.
5 e following repeated exposure, indicative of behavioral sensitization.
6 -nitroindazole [7-NI]) block cocaine-induced behavioral sensitization.
7 files of PTT and cocaine, both drugs produce behavioral sensitization.
8  changes in specific binding were related to behavioral sensitization.
9 nt manner that paralleled the development of behavioral sensitization.
10 ical areas are related to the development of behavioral sensitization.
11 n may occur only when drug treatments induce behavioral sensitization.
12 en reported to block psychostimulant-induced behavioral sensitization.
13 ronic treatment, indicating the emergence of behavioral sensitization.
14 ne (5 mg/kg) for 5 consecutive days produced behavioral sensitization.
15 excitatory amino acids in the development of behavioral sensitization.
16 hanges in behavior ranging from addiction to behavioral sensitization.
17  nicotine-induced physiological, neural, and behavioral sensitization.
18 tic spine formation and for cocaine-mediated behavioral sensitization.
19 aine-induced locomotor activity and occluded behavioral sensitization.
20 xpression of peripheral inflammation-induced behavioral sensitization.
21 d its extent strongly predicts the degree of behavioral sensitization.
22 efrontal cortex (PFC) prevented MPD elicited behavioral sensitization.
23 escending glutamate from PFC in MPD elicited behavioral sensitization.
24 ayed an enhanced response to cocaine-induced behavioral sensitization.
25 played a clear resistance to cocaine-induced behavioral sensitization.
26  that repeated administration of MPD induced behavioral sensitization.
27 RK) as a molecular substrate underlying this behavioral sensitization.
28 five days with daily 2.5 mg/kg MPD to elicit behavioral sensitization.
29 for plasticity underlying the development of behavioral sensitization.
30  the formation of long-term facilitation and behavioral sensitization.
31 to which repeated cocaine injections produce behavioral sensitization.
32  positively correlated with the magnitude of behavioral sensitization.
33 erations depends critically on the extent of behavioral sensitization.
34 re was no correlation between S/I ratios and behavioral sensitization.
35   Daily injections of cocaine in rats caused behavioral sensitization.
36 of experience-dependent plasticity including behavioral sensitization.
37  locomotor activity and also cocaine-induced behavioral sensitization.
38 teins, in a drug-induced phenomenon known as behavioral sensitization.
39 input, (2) chronic treatment of MDMA elicits behavioral sensitization, (3) chronic administration of
40 ibute to the neural adaptations that mediate behavioral sensitization, a model for core aspects of ad
41 roadaptation occurs during the expression of behavioral sensitization, a model of psychostimulant-ind
42  treatment regimen led to the development of behavioral sensitization, a separate set of animals (n=8
43 y developed strikingly different patterns of behavioral sensitization after daily treatment with low
44           The mutant mice exhibit attenuated behavioral sensitization after repeated exposure to coca
45 ons from animals that did or did not exhibit behavioral sensitization after repeated methamphetamine
46          Eticlopride and nafadotride blocked behavioral sensitization, although nafadotride was more
47 allenge, normal subjects showed a pattern of behavioral sensitization (an increase in dyskinetic-like
48 or trafficking to plasticity associated with behavioral sensitization, an animal model of drug addict
49 epetitive daily amphetamine injections cause behavioral sensitization and a significant change of cir
50  in the NAc, a change that may contribute to behavioral sensitization and addiction.
51 ral tegmental area, enhances cocaine-induced behavioral sensitization and cocaine-seeking.
52 e of nNOS by 7-NI-attenuated ethanol-induced behavioral sensitization and completely blocked the rewa
53 city during short-term and intermediate-term behavioral sensitization and dishabituation in a semi-in
54 anges could be linked to phenomena including behavioral sensitization and drug self-administration in
55 medial prefrontal cortex (mPFC) that promote behavioral sensitization and drug-seeking behavior.
56 mbens (NAc) synapses, a phenomenon linked to behavioral sensitization and drug-seeking.
57 nt with neuropathic sensitization, including behavioral sensitization and neuronal hyperexcitability,
58 ed orexin's contribution to morphine-induced behavioral sensitization and place preference.
59 s been shown to effectively induce long-term behavioral sensitization and synaptic facilitation, is n
60 cocaine including escalation of drug intake, behavioral sensitization, and cocaine-primed reinstateme
61 ulation of GIR expression may be involved in behavioral sensitization, and GIR may play a role at the
62  psychostimulant administrations is known as behavioral sensitization, and is an indicator of a drug'
63 ffects of amphetamine, its ability to induce behavioral sensitization, and its ability to induce c-fo
64 is, in cocaine conditioned place preference, behavioral sensitization, and motor stereotypy.
65 ion to cocaine, there are sex differences in behavioral sensitization, and sensitization that develop
66 uisition and expression of context-dependent behavioral sensitization, as this behavior is blocked by
67 ing ketamine-evoked postsynaptic density and behavioral sensitization, as well as in opioid blockade-
68 l addiction-related brain region, to mediate behavioral sensitization but not cocaine reward.
69 ergolide and ondansetron normalized not only behavioral sensitization, but also the increases in thes
70 pamine also failed to exhibit l-DOPA-induced behavioral sensitization, but this may be caused, at lea
71 A) receptor may contribute to l-DOPA-induced behavioral sensitization by facilitating adaptations wit
72 lessness and painlessness, may attenuate the behavioral sensitization caused by Amphetamine.
73 ittent treatment with amphetamine leads to a behavioral sensitization characterized in rats by an inc
74 evaluated for its effects on cocaine-induced behavioral sensitization, conditioned place preference (
75  morphine and cocaine on locomotor activity, behavioral sensitization, conditioned place preference,
76  OX(1), have demonstrated its involvement in behavioral sensitization, conditioned place-preference,
77 t to determine whether the expression of TDF behavioral sensitization could be prevented by the DA D1
78  RO5263397 reduced the expression of cocaine behavioral sensitization, cue- and cocaine prime-induced
79 urons or their terminals in the DS disrupted behavioral sensitization, demonstrating the utility of t
80 esioning of PFC eliminated the expression of behavioral sensitization elicited by chronic MPD adminis
81 pamine receptor stimulation, we compared the behavioral sensitization elicited by repeated l-DOPA tre
82 d using Western blots in rats that developed behavioral sensitization following repeated amphetamine
83 hanced hyperlocomotor activity and increased behavioral sensitization following treatment with psycho
84                                         This behavioral sensitization has been implicated in maintena
85                  In contrast, any rotational behavioral sensitization in A(2A) KO mice was transient
86                  Amphetamine produced robust behavioral sensitization in all environments, but when a
87 an endogenous neurotransmitter that mediates behavioral sensitization in Aplysia[1-3], induces long-t
88 Daily amphetamine administration resulted in behavioral sensitization in both Dbh+/- and Dbh-/- mice,
89 refore, tolerance can mask the expression of behavioral sensitization in rats.
90  shown that the fruitfly Drosophila exhibits behavioral sensitization in response to repeated exposur
91 est the hypothesis that ERK1 is required for behavioral sensitization in rodent pain models.
92  to the development and robust expression of behavioral sensitization in terms of vertical activity.
93                     MA treatment resulted in behavioral sensitization in the open field, consistent w
94 tic ionotropic glutamate receptors underlies behavioral sensitization in this snail.
95 contingent cocaine injections, which lead to behavioral sensitization, increase AMPA receptor (AMPAR)
96  but not for conditioned place preference or behavioral sensitization induced by morphine.
97 e network-level adaptations suggest that the behavioral sensitization induced by repeated psychomotor
98 hibitors and suggest that the development of behavioral sensitization is a uniform characteristic of
99                            Context-dependent behavioral sensitization is a well-documented phenomenon
100                                              Behavioral sensitization is a well-studied model of beha
101 udies have demonstrated that cocaine-induced behavioral sensitization is associated with persistent f
102 he finding that cocaine- and ethanol-induced behavioral sensitization is associated with upregulation
103                                         This behavioral sensitization is proposed to model the increa
104                                              Behavioral sensitization is the progressive augmentation
105 ocaine administration to wild types produced behavioral sensitization, knock-outs exhibited no additi
106 duce forms of cellular plasticity related to behavioral sensitization may also contribute to addictio
107                          A failure of normal behavioral sensitization mechanisms after dextroamphetam
108 tations compared with controls and exhibited behavioral sensitization of contralateral rotations indu
109                                              Behavioral sensitization of psychostimulant-induced loco
110 eated ethanol (EtOH) injections that induced behavioral sensitization on subsequent acquisition of Et
111 on cellular function that may be relevant in behavioral sensitization or cocaine self-administration.
112 or PLX3397 (40 mg/kg, p.o.) and submitted to behavioral sensitization or conditioned place preference
113  propensity of a given rat to exhibit either behavioral sensitization or conditioned place preference
114 lter cocaine-induced psychomotor activation, behavioral sensitization, or conditioned place preferenc
115 l test environment, a protocol that produces behavioral sensitization, or in the home cage, a protoco
116 dbrain DA neurons, although not required for behavioral sensitization per se, may contribute to the a
117                          This effect, called behavioral sensitization, persists months after the last
118 ent long-term potentiation as central to the behavioral sensitization phenomenon of LIDs.
119 ed the development but not the expression of behavioral sensitization produced by repeated administra
120                  CSF1R inhibition attenuated behavioral sensitization, reduced the number of Iba-1(+)
121   The data add support to the inference that behavioral sensitization represents not only a quantitat
122  are involved in induction and expression of behavioral sensitization respectively and glutamate from
123 opa-induced dyskinesia (LID) is a persistent behavioral sensitization that develops after repeated le
124  is the enhanced locomotor response known as behavioral sensitization that occurs with prolonged expo
125 ections of 2.5 mg/kg methamphetamine induced behavioral sensitization that was demonstrated (expresse
126 bens (NAc) neurons have been correlated with behavioral sensitization, the molecular pathways governi
127          The acquisition of context-specific behavioral sensitization to AMPH and extinction of condi
128 ling in dopamine neurons is not required for behavioral sensitization to AMPH.
129 duced and restoration of NR2B loss prevented behavioral sensitization to AMPH.
130             Last, we show that CNO prevented behavioral sensitization to amphetamine and increased AM
131                       It has been shown that behavioral sensitization to amphetamine develops paralle
132 cross-reactivity with chronic FRAs, and that behavioral sensitization to amphetamine is not accompani
133 n-like immunoreactivity (FRALI) accompanying behavioral sensitization to amphetamine was assessed in
134 nvestigate neural mechanisms associated with behavioral sensitization to amphetamine, we studied the
135  a variety of neuroeffectors are involved in behavioral sensitization to amphetamine-induced stereoty
136 cal studies suggested that the initiation of behavioral sensitization to cocaine and amphetamine invo
137         One episode of social stress induced behavioral sensitization to cocaine as indicated by an a
138 t that NT-3 contributes to the initiation of behavioral sensitization to cocaine by activating the Ra
139 pisode of social defeat stress is related to behavioral sensitization to cocaine challenge.
140  signal transduction pathway in long-lasting behavioral sensitization to cocaine in rats, an animal m
141                  It is well established that behavioral sensitization to cocaine is accompanied by in
142                  It has been postulated that behavioral sensitization to cocaine is associated with a
143                  These findings suggest that behavioral sensitization to cocaine is associated with a
144             In contrast, both the neural and behavioral sensitization to cocaine was diminished in Co
145                                Surprisingly, behavioral sensitization to cocaine was elicited in GluR
146                           In this study, the behavioral sensitization to cocaine was established in S
147                                              Behavioral sensitization to cocaine was tested for in ra
148                                        After behavioral sensitization to cocaine, the depolarization
149 or the expression, but not the induction, of behavioral sensitization to cocaine.
150  kinase (MAP) signal transduction cascade in behavioral sensitization to cocaine.
151 al transduction cascade on the initiation of behavioral sensitization to cocaine.
152 ted to the ability to express stress-induced behavioral sensitization to cocaine.
153  nucleus accumbens only in rats that develop behavioral sensitization to cocaine.
154 aluated the effects of NMU microinjection on behavioral sensitization to cocaine.
155 inhibitor deprenyl completely alleviated the behavioral sensitization to cocaine.
156 en free access to high-fat diets demonstrate behavioral sensitization to D2/3R agonists but not to an
157           Furthermore, Drosophila develops a behavioral sensitization to intermittent doses of cocain
158                                 The enhanced behavioral sensitization to l-DOPA in TAAR1 KO mice was
159 rtant role in the neuroplasticity underlying behavioral sensitization to L-DOPA, supporting considera
160 t only when mice developed context-dependent behavioral sensitization to morphine in which morphine t
161                            Context-dependent behavioral sensitization to morphine was also associated
162 , synaptic plasticity, and context-dependent behavioral sensitization to morphine.
163 ment of the dopaminergic afferents of PFC in behavioral sensitization to MPD is discussed.
164 PD as well as in its chronic effects such as behavioral sensitization to MPD.
165 at the 6-OHDA lesion group failed to exhibit behavioral sensitization to MPD.
166 ceptors is sufficient for the development of behavioral sensitization to NMDA.
167            Taken as a whole, we propose that behavioral sensitization to psychostimulant drugs is att
168                                              Behavioral sensitization to psychostimulants involves ne
169 (NMDARs) is essential for the development of behavioral sensitization to psychostimulants such as amp
170 stigated the role of PFC DA in mediating the behavioral sensitization to repeated administration of M
171 nd serotonin neurons of living flies blocked behavioral sensitization to repeated cocaine exposures.
172 elopment and particularly the persistence of behavioral sensitization to repeated l-DOPA treatment.
173 c system in amphetamine- and cocaine-induced behavioral sensitization to stereotypy in mice.
174 lphenidate (MPD) has been reported to elicit behavioral sensitization to their locomotor and stereoty
175                                              Behavioral sensitization was facilitated by alpha2A-AR a
176 ay 61, expression of methamphetamine-induced behavioral sensitization was obtained with an acute meth
177                                          The behavioral sensitization we measured was primarily 'drug
178 ocedures, limbic brain regions implicated in behavioral sensitization were assayed for extracellular
179 ting effects of cocaine and the magnitude of behavioral sensitization were greater in HRs.
180 nucleus accumbens abolish the development of behavioral sensitization when they are administrated dur
181 triatopallidal neuronal activity facilitated behavioral sensitization, whereas decreasing excitabilit
182 ed behaviors known as 'reverse tolerance' or behavioral sensitization, which may model the behavioral
183 ue-induced reinstatement and cocaine-induced behavioral sensitization without altering locomotor acti

 
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