コーパス検索結果 (1語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 hydrochloride, 2-N-methylellipticinium, and berberine).
2 translation via utilizing a plant compound, berberine.
3 ill-in dGMP is covered and well-protected by berberine.
4 ex I-inhibiting interventions: metformin and berberine.
5 thelial cells line in vitro (Calu3 cells) by berberine.
6 ically important alkaloids: magnoflorine and berberine.
7 o oxidized forms, including (RS)-canadine to berberine.
8 translation via utilizing a plant compound, berberine.
9 iquid-to-solid ratio- 30 mL/g], the yield of berberine (11.91 +/- 0.12 mg/g DW), berbamine (11.85 +/-
11 of PCSK9 can be blocked by the treatment of berberine, a natural cholesterol-lowering compound which
16 The first total synthesis of the dimeric berberine alkaloid ilicifoline (ilicifoline B) is report
24 e NMR structure can guide rational design of berberine analogues that target the PDGFR-B vG4 or dGMP-
25 four-electron oxidations of (S)-canadine to berberine and (S)-tetrahydropapaverine to papaverine.
26 alone but strongly potentiated the action of berberine and other NorA substrates against S. aureus.
27 with small molecules, such as the alkaloids berberine and palmatine and the DNA intercalator ethidiu
28 the relative intensities of the bands due to berberine and palmatine suggest that the ancient paper i
30 resin as most effective for the recovery of berberine and palmatine, while, XAD-7HP resin is best su
31 berberine ursodeoxycholate, an ionic salt of berberine and ursodeoxycholic acid), versus placebo that
35 ution, the binding mode and stoichiometry of berberine are substantially different from the crystal s
37 current study may lead to the development of berberine as an orally, active therapeutic against COVID
43 e plant-derived hypocholesterolemic compound berberine (BBR) up-regulates LDLR expression while down-
44 microbiota like oral intake of probiotics or berberine (BBR), a bacteriostatic agent, merit metabolic
45 nt in the suppression of PCSK9 expression by berberine (BBR), a natural cholesterol-lowering compound
46 r cells, we observed that the plant compound berberine (BBR), a potent activator of AMP-activated pro
47 r cells, we observed that the plant compound berberine (BBR), a potent activator of AMP-activated pro
52 Gelatin (CA/Gel) electrospun mat loaded with berberine (Beri) as the DFU-specific wound dressing.
54 sed on a previous crystal structure in which berberine binds as a dimer to a parallel G-quadruplex.
55 tially different from the crystal structure: berberine binds as a monomer to MycG4 using a base-recru
56 (S)-canadine synthase enzyme involved in the berberine biosynthesis pathway may play critical roles i
57 lution NMR structure of a ternary complex of berberine bound to the dGMP-fill-in PDGFR-B vG4 in potas
58 The effects of the well-tolerated alkaloid Berberine (BRB), used for treating metabolic disorders,
60 l alcohol oxidase as reported previously for berberine bridge enzyme and other FAD-dependent oxidored
61 ium poppy stem transcriptome databases using berberine bridge enzyme yielded several candidate genes,
63 g of the pyrazinone ring by an FAD-dependent berberine bridge enzyme-like oxidoreductase has been pro
66 characterisation of a group of P. infestans berberine bridge enzyme-like proteins (BBEs) and their r
69 fies RXRalpha as a direct protein target for berberine but also dissects their binding mode and valid
70 associated with the antimetastatic effect of berberine by showing a nearly complete inhibition on inv
72 Of the non-flavonoid phytochemicals tested, berberine, celastrol, ellagic acid, limonin, oleanolic a
74 retinoid X receptor alpha (RXRalpha), where berberine concomitantly binding to and synergistically a
75 y determination of a 2 ng/microL solution of berberine contained as a dry residue in the bottom of a
76 aken together, these findings suggested that berberine could reduce metastasis and angiogenesis of ce
79 ructure-based rational design of G4-targeted berberine derivatives, and this study demonstrates that
80 e findings implied that the incorporation of berberine did not compromise the physical properties of
84 toxic, 5 compounds (all-trans retinoic acid, berberine, fisetin, propranolol, and ritonavir) extended
87 MDR-dependent efflux of ethidium bromide and berberine from S. aureus cells was completely inhibited
91 ence with a small molecule GQ-binding agent, berberine hydrochloride, further increased GQ stability.
92 The most potent apoptosis was induced by berberine in ALL cells with both MDM2 overexpression and
94 us DUX4 confirmed the therapeutic effects of berberine in downregulating DUX4 protein expression, inh
95 ates the potential therapeutic mechanisms of berberine in preventing and treating COVID-19 and SARS u
97 nificantly increased oral bioavailability of berberine, increased IFN-y production by CD8+ T cells, a
98 ssects their binding mode and validates that berberine indeed suppresses beta-catenin signaling and c
103 of exonuclease I, leading to the release of berberine into solution and consequently, quenched berbe
105 sm of CAR metabolic pathway, suggesting that berberine is a promising candidate in anticancer adjuvan
106 tosis; and protecting against tissue damage, berberine is a promising candidate in preventing and tre
107 COVID-19 to be combined with other therapy, berberine is a promising orally administered therapeutic
113 ynthesis of the biologically active alkaloid berberine is reported, and a versatile palladium-catalyz
114 aptamer (ATP-aptamer) and a DNA binding dye, berberine, is digested upon the addition of exonuclease
116 nd anti-inflammatory activities displayed by berberine may be mediated in part through the suppressio
117 ults indicated that the antiproliferation of berberine might be mediated by the unique epigenetic mod
118 lex has a 2:1:1 binding stoichiometry with a berberine molecule bound at each the 5'- and 3'-end of t
120 the high oral bioavailability and safety of berberine nanomedicine, the current study may lead to th
121 that downregulation of MDM2 in ALL cells by berberine occurred at a posttranslational level through
124 cholesterol was significantly decreased with berberine (P<0.05) and HDL was significantly improved (P
126 tion curves (2.5-100 pmol) for the standards berberine, palmatine, and hydrastinine spotted as a mixt
132 ciency-aggravated periodontal bone loss, and berberine represents a promising adjuvant therapeutic by
138 was complexed with berberine to form a nano-berberine-starch complex and cast into active films.
139 ation and a subsequent upregulation of MDM2, berberine strongly induced persistent downregulation of
141 fat proportion also decreased (P<0.05) with berberine supplementation and the abdominal fat thicknes
146 beta-carboline harmine and the isoquinoline berberine, that ameliorated certain aspects of the DM1 p
148 ovide the molecular basis for the ability of berberine to act as an anticancer and anti-inflammatory
150 rformance, cassava starch was complexed with berberine to form a nano-berberine-starch complex and ca
151 binding mechanism of the anticancer alkaloid berberine to the human telomeric G4 (d[AG3(T2AG3)3]), co
152 naturally occurring GQ stabilizing compound, berberine, to these non-canonical genetic structures usi
155 -related immune responses were attenuated in berberine-treated OVX-periodontitis rats with a lower se
156 udy in FSHD patient myoblasts indicated that berberine treatment reduced DUX4 expression and also exp
157 el and tail vein injection model showed that berberine treatment reduced tumor growth and lung metast
158 mography and histologic analyses showed that berberine treatment significantly reduced alveolar bone
159 s, subjects receiving 1000 mg twice a day of berberine ursodeoxycholate had significantly greater red
161 bo-controlled trial of two doses of HTD1801 (berberine ursodeoxycholate, an ionic salt of berberine a
163 Several Berberis medicinal plants producing berberine were found also to synthesize an inhibitor of
166 d by a SEM experiment showing the binding of berberine with one of the nucleoside derivatives, which