ライフサイエンスコーパス: beta-actin

1 tion of endothelial nitric oxide (eNOS) with beta-actin.

2 a and is rescued by reintroducing NLS-tagged beta-actin.

3 llular scaffolding proteins beta-catenin and beta-actin.

4 alization against housekeeping genes such as beta-actin.

5 clear membrane and interacts with emerin and beta-actin.

6 l DNA: non-muscle myosin heavy chain IIA and beta-actin.

7 mma-actin depolymerizes half as fast as does beta-actin.

8 , and a somewhat slower elongation rate than beta-actin.

9 c61 translocon, and nonmuscle myosin IIA and beta-actin.

10 , consistent with known roles of cytoplasmic beta-actin.

11 n and one of its major structural components beta-actin.

12 sed amounts of nuclear, but not cytoplasmic, beta-actin.

13 of the actin-related protein Arp1 and one of beta-actin.

14 Peripheral axotomy triggers transport of the beta-actin 3'-UTR containing transgene mRNA into axons.

15 These data show for the first time that the beta-actin 3'-UTR is sufficient for axonal localization

16 les and stimulates translation driven by the beta-actin 3'UTR in neurites.

17 erence genes, adenosylhomocysteinase (AHC1), beta actin (ACTB), eukaryotic translation elongation fac

18 Sam68 binds to the mRNA of beta-actin (actb), an integral cytoskeletal component of

19 raldehyde-3-phosphate dehydrogenase (Gapdh), beta-actin (Actb), or beta2-microglobulin (beta2m) showe

20 , myelin-oligodendrocyte glycoprotein [MOG], beta-actin [ACTB], thymosin beta-10 [TB10], and superior

21 howardi (Barber), housekeeping genes (HKG), beta-actin (Actin), beta-tubulin (Tubulin), elongation f

22 ion status of the CpG island of the modified beta-actin allele in differentiated but not in undiffere

23 The modified beta-actin allele was expressed in undifferentiated embr

24 ession in mice heterozygous for the modified beta-actin allele.

25 multaneously followed transcription from the beta-actin alleles in real time and observed transcripti

26 os expressing fluorescent fusion proteins of beta-Actin, alpha-Catenin, and the ERM family member Moe

27 TDC WB can also clearly probe the intrinsic beta-actin, alpha-tubulin, and glyceraldehyde 3-phosphat

28 Gene silencing of beta-actin also affected mitochondrial DNA copy number a

29 Of great interest, ablation of beta-actin altered the ratio of globular actin (G-actin)

30 ACTB encodes beta-actin, an abundant cytoskeletal housekeeping protei

31 Gs glyceraldehyde-3-phosphate dehydrogenase, beta actin and adenosine triphosphate synthase subunit t

32 wed that Stain-Free staining was superior to beta-actin and as good as or better than Ponceau S stain

33 (+/-5000 bps) or at negative control genes (beta-actin and beta-globin).

34 Phosphorylated HtrA2/Omi therefore cleaves beta-actin and decreases the amount of filamentous actin

35 Nevertheless, double-mutant mice lacking beta-actin and expressing fascin-2 p.R109H have a more s

36 This process requires both beta-actin and gamma-actin as knock-out mice lacking eit

37 Fascin-2 binds beta-actin and gamma-actin filaments with similar affini

38 satory increase in expression of cytoplasmic beta-actin and gamma-actin isoforms in Acta2(-/-) HSCs,

39 Ubiquitously expressed beta-actin and gamma-actin isoforms play critical roles

40 ich in turn display altered fractionation of beta-actin and gamma-tubulin.

41 beta-actin mRNA depletes axons of endogenous beta-actin and GAP-43 mRNAs and attenuates both in vitro

42 re haploinsufficient for axonal transport of beta-actin and GAP-43 mRNAs and for regeneration of peri

43 ta-actin mRNA 3'UTR competes with endogenous beta-actin and GAP-43 mRNAs for binding to ZBP1 and axon

44 ddress the functions of axonally synthesized beta-actin and GAP-43 proteins.

45 aracterized axonal mRNAs in model organisms, beta-actin and GAP43, within hESC-neuron projections usi

46 sence of serum, and the expression levels of beta-actin and human antigen R (HuR) were analyzed by us

47 and iodixanol gradient analysis suggest some beta-actin and non-muscle myosin heavy chain IIA reside

48 ase, leading to excessive S-nitrosylation of beta-actin and possibly profilin.

49 of the putative ABSs, specifically bound to beta-actin and prevented eNOS association with beta-acti

50 previously determined structures of profilin-beta-actin and profilin-alpha-actin.

51 Here, we establish that beta-actin and the actin crosslinking protein fascin-2 c

52 s and the revised Figures containing matched beta-actin and their respective quantification data are

53 icient production of cystoskeletal proteins (beta-actin and vimentin) containing bicyclo[6.1.0]nonyne

54 toplasm, reduced interaction with emerin and beta-actin, and activation of glycogen synthase kinase-3

55 ctin, transforming growth factor (TGF) beta1/beta-actin, and FOXP3/beta-actin than did IgG4-negative

56 poral dynamics for three human genes: KDM5B, beta-actin, and H2B.

57 o transfer include those encoding GFP, mouse beta-actin, and human Cyclin D1, BRCA1, MT2A, and HER2.

58 gamma-actin was translated more slowly than beta-actin, and this slower processing resulted in the e

59 Although ubiquitous beta-actins are essential for cell survival during early

60 We show that myosin II and alpha- and beta-actins are present in the nuclei of colonic smooth

61 ctrometry and immunoprecipitation identified beta-actin as a protein that is specifically deglutathio

62 acellular signal-regulated kinase (ERK 1/2), beta-actin as well as p21/WAF/Cip1 levels were determine

63 ctors, we identified more than 60 additional beta-actin-associated RBPs by RNA-BioID.

64 o significantly reduced the increase in eNOS-beta-actin association and protein tyrosine nitration.

65 To study whether eNOS-beta-actin association contributes to increased peroxyni

66 d an eNOS mutant that exhibited reduced eNOS-beta-actin association, decreased NO production, and inc

67 ia resulted in significant increases in eNOS-beta-actin association, eNOS activity, and protein tyros

68 revented hyperoxia-induced increases in eNOS-beta-actin association, eNOS activity, NO and peroxynitr

69 ng endothelial monolayer and attenuates eNOS-beta-actin association, peroxynitrite formation, endothe

70 refore, Shh induces the local translation of beta-actin at the growth cone.

71 actin interaction were inhibited by reducing beta-actin availability or by using a synthetic peptide

72 th real-time RT-PCR for expression levels of beta-actin, Bax, Bcl-xl, Bcl-2 and TNFR1 genes with mean

73 y the cytomegalovirus early enhancer/chicken beta-actin/beta-globin promoter into the Rosa26 locus, r

74 al alanine was unable to interfere with eNOS-beta-actin binding and to prevent beta-actin-induced cha

75 together, this novel finding indicates that beta-actin binding to eNOS through residues 326-333 in t

76 ybrid cytomegalovirus (CMV) enhancer/chicken-beta-actin (CBA) transduced both Muller and ganglion cel

77 orff-perfused mouse heart preparations, Cx43/beta-actin complexing is disrupted during acute ischemia

78 ubunit and beta-tubulin, but very little for beta-actin, consistent with the morphology of their tips

79 beta-Actin contains an Asp(1)-Asp(2)-Asp(3) and Val(10)

80 nregulation of lamin-A,C and upregulation of beta-actin, corroborating the predictions of the model.

81 Colocalization of PDI and beta-actin could be abolished by the membrane-permeable

82 , we provided evidence that PDI binds to the beta-actin Cys(374) thiol.

83 ixed-cell microscopy of wild-type and mutant beta-actin demonstrates that incorporation of actin mono

84 We found that beta-actin depletion affects induction of several adipog

85 The tagged proteins include alpha tubulin, beta actin, desmoplakin, fibrillarin, nuclear lamin B1,

86 41297 promoter than strong promoters such as beta-actin, ef-1beta, and many other promoters.

87 In wild-type mouse embryos, beta-actin expression was prominent in the kidney, heart

88 nslation system, we found that inhibition of beta-actin folding by PDCL3 can be relieved by exchangin

89 ity in vitro and fascin-2 does not depend on beta-actin for localization in vivo.

90 Arp7A, which has 43% sequence identity with beta-actin, forms a complex with the cytoskeletal protei

91 PAmKate-transferrin receptor, and PAmCherry1-beta-actin fusion constructs, revealing correlations bet

92 mmunogold labeling of tropomyosin, spectrin, beta-actin, gamma-actin, espin, and prestin.

93 that regenerating spinal cord axons contain beta-actin, GAP-43, Neuritin, Reg3a, Hamp, and Importin

94 evious studies have shown that disruption of beta-actin gene critically impacts cell migration and mo

95 The beta-actin gene expression was analyzed at the transcrip

96 cassette into the 3'-untranslated region of beta-actin gene led to widespread but not ubiquitous lac

97 mouse lacking beta-actin protein by editing beta-actin gene to encode gamma-actin protein, and vice

98 e amount of genomic DNA quantitated with the beta-actin gene was almost 20 times lower in dry samples

99 The expression of the beta-actin gene was downregulated in the stroma of the s

100 the 3' untranslated region of the essential beta-actin gene.

101 For endogenous beta-actin genes in mouse embryonic fibroblasts, we obse

102 FP) was primarily in neurons, whereas scAAV9/beta-actin-GFP drove transgene expression in astrocytes.

103 detected in NeuN(+) neurons, whereas scAAV9/beta-actin-GFP drove transgene expression in GFAP(+) ast

104 ng neonatal rat hair cells in culture with a beta-actin-GFP fusion, and evidence was found that actin

105 itoring incorporation of inducibly expressed beta-actin-GFP in adult mouse hair cells in vivo and by

106 hair cells in vivo and by directly measuring beta-actin-GFP turnover in explants.

107 ods confirmed this: in hair cells expressing beta-actin-GFP we bleached fiducial lines across hair bu

108 t 1, DEAD (Asp-Glu-Ala-Asp) box polypeptide, beta-actin, glyceraldehyde 3-phosphate dehydrogenase (G3

109 ld in Cln3(Deltaex7/8) mice receiving scAAV9/beta-actin-hCLN3 versus scAAV9/MeCP2-hCLN3 after a singl

110 ld in Cln3(Deltaex7/8) mice receiving scAAV9/beta-actin-hCLN3 versus scAAV9/MeCP2-hCLN3.

111 s) injection of scAAV9/MeCP2-hCLN3 or scAAV9/beta-actin-hCLN3, with green fluorescent protein (GFP)-e

112 ation reporter analysis reveals that de novo beta-actin hotspots colocalize with docked RNA granules

113 lammatory response in DSS-induced colitis in beta-actin-hPepT1 and villin-hPepT1 transgenic mice, ind

114 nduced more severe levels of inflammation in beta-actin-hPepT1 transgenic mice than wild-type litterm

115 Nod2-/-, beta-actin-hPepT1 transgenic/Nod2-/-, and villin-hPepT1

116 gher expression ratios of interleukin (IL)-4/beta-actin, IL-10/beta-actin, IL-13/beta-actin, transfor

117 tios of interleukin (IL)-4/beta-actin, IL-10/beta-actin, IL-13/beta-actin, transforming growth factor

118 presented the same samples and therefore the beta-actin immunoblot was reused.

119 ry Figs 6a, 8a, 8d, 9, and in some cases the beta-actin immunoblots were erroneously described in the

120 At 4 h after estradiol treatment, both beta-actin immunofluorescence and filopodial spines were

121 was used to visualize spine shape, and then beta-actin immunoreactivity was used as a semiquantitati

122 In the present study, we used both beta-actin immunostaining and Golgi staining to visualiz

123 To study the role of beta-actin in adipocyte differentiation, we conducted RN

124 expression pattern of LIMK-1, cofilin-1, and beta-actin in all the experimental groups by semiquantit

125 gration defects upon conditional ablation of beta-actin in highly motile T cells.

126 cate that increased association of eNOS with beta-actin in PAEC contributes to hyperoxia-induced incr

127 These data support an essential role for beta-actin in regulating cell migration and gene express

128 that NKCC1 interacted with beta-tubulin and beta-actin in spinal cords.

129 We propose that loss of beta-actin in the corneal stroma might be a triggering f

130 mine the expression levels and regulation of beta-actin in the stroma of keratoconus (KC) and normal

131 of LIMK-1, cofilin-1, phospho-cofilin-1, and beta-actin in the whole brain lysates as well as formati

132 gtin, and refold the physiological substrate beta-actin in vitro.

133 ta-actin and prevented eNOS association with beta-actin in vitro.

134 ase domain and that association of eNOS with beta-actin increases eNOS activity and nitric oxide (NO)

135 ously reported that association of eNOS with beta-actin increases eNOS activity and NO production in

136 with eNOS-beta-actin binding and to prevent beta-actin-induced changes in NO and superoxide formatio

137 Peptide 326 also prevented beta-actin-induced decrease in superoxide formation and

138 In the present study, we found that beta-actin-induced increase in NO production was accompa

139 Disruption of eNOS-beta-actin interaction in endothelial cells using ABS pe

140 We found that disruption of eNOS-beta-actin interaction prevented hyperoxia-induced incre

141 to increased peroxynitrite production, eNOS-beta-actin interaction were inhibited by reducing beta-a

142 rporation of (15)N-labelled protein and EGFP-beta-actin into nascent stereocilia.

143 gamma-actin are highly homologous, but only beta-actin is amino-terminally arginylated in vivo, whic

144 We have previously reported that beta-actin is associated with eNOS oxygenase domain and

145 inked to the 3' untranslated region (UTR) of beta-actin is delivered into cells fated to become the r

146 Our results show that beta-actin is downregulated in the corneal stroma of pat

147 ggest that the essential in vivo function of beta-actin is provided by the gene sequence independent

148 ckout (Actb(-/-)) mice and demonstrated that beta-actin is required for early embryonic development.

149 alysis and in vitro validation revealed that beta-actin is the receptor of C7H2 in the tumor cells.

150 organization and induction of the nonmuscle, beta actin isoform.

151 we conducted RNA sequencing on wild-type and beta-actin knockout mouse embryonic fibroblasts (MEFs) a

152 We generated whole-body beta-actin-knockout (Actb(-/-)) mice and demonstrated th

153 e growth impairment and migration defects in beta-actin-knockout primary mouse embryonic fibroblasts

154 mice at P4, 5 months, and 12 months, whereas beta-actin levels were significantly decreased at P4.

155 in fluorescence signal intensity between the beta-actin LNA probe and a biotinylated, nonspecific con

156 The same beta-actin loading control image thus necessarily applie

157 and protein synthesis-dependent increase in beta-actin localization in growth cones.

158 As beta-actin-MBS was ubiquitously expressed, we could uniq

159 rons were produced by tamoxifen treatment of beta-actin-MerCreMer/LpL(flox/flox) mice.

160 hybridization to demonstrate that dendritic beta-actin messenger RNA (mRNA) and ribosomes are in a m

161 ipt in vitro and in vivo SHAPE-MaP for human beta-actin messenger RNA that revealed similar global fo

162 for the thiol-disulfide rearrangement in the beta-actin molecule of adhering cells.

163 a reporter mRNA with the axonally localizing beta-actin mRNA 3'UTR competes with endogenous beta-acti

164 This GFP-3'-beta-actin mRNA accumulates in injured PNS axons before

165 We found that FUBP3 binds to the 3' UTR of beta-actin mRNA and is essential for beta-actin mRNA loc

166 for assessing the functional consequences of beta-actin mRNA and protein localization by tethering th

167 P1), an mRNA-binding protein that transports beta-actin mRNA and releases it for local translation up

168 Further, our work demonstrates that beta-actin mRNA and ribosomes are in a masked state that

169 not Cal PA-XFS, induced degradation of host beta-actin mRNA and suppressed host protein synthesis, s

170 cin D (actD) on the interactions of HuR with beta-actin mRNA and with poly(A)+ mRNA at both native an

171 ve ZBP1 construct diminished localization of beta-actin mRNA but not of Ca(2+)/calmodulin-dependent p

172 e of these transcripts exceeded the level of beta-actin mRNA by more than 150-fold.

173 duction of a reporter mRNA with the 3'UTR of beta-actin mRNA competes with endogenous mRNAs for bindi

174 mRNA, and introducing GFP with the 3'UTR of beta-actin mRNA depletes axons of endogenous beta-actin

175 length of axons, while overall depletion of beta-actin mRNA from DRGs decreased the number of axon b

176 oted that ZBP1 and PAT1 co-locate along with beta-actin mRNA in actively transported granules in livi

177 for netrin-1 stimulated local translation of beta-actin mRNA in axonal growth cones.

178 s been implicated in the axonal transport of beta-actin mRNA in both primary and transformed neuronal

179 expression level and spatial distribution of beta-actin mRNA in HeLa cells and to observe the real-ti

180 otein at the 3' UTR of endogenous MS2-tagged beta-actin mRNA in mouse embryonic fibroblasts.

181 ls and to observe the real-time transport of beta-actin mRNA in mouse embryonic fibroblasts.

182 We found that beta-actin mRNA in primary fibroblasts localizes predomi

183 or the transport and/or local translation of beta-actin mRNA in the growth cones of motor neurons.

184 mRNA increased length and axonally targeted beta-actin mRNA increased branching of sensory axons gro

185 We demonstrate the dynamics of the beta-actin mRNA interactome by characterizing its change

186 ve developed a transgenic mouse in which all beta-actin mRNA is fluorescently labeled.

187 have provided the first direct evidence that beta-actin mRNA is present in SMN cytoplasmic complexes

188 hydrogenase, wild-type HSV infection reduced beta-actin mRNA levels to between 20 and 30% of those in

189 transfected with any of three eIF4H siRNAs, beta-actin mRNA levels were indistinguishable in infecte

190 The aberrant beta-actin mRNA localization resulted in abnormal dendri

191 Furthermore, BDNF-stimulated beta-actin mRNA localization was attenuated in Zbp1-defi

192 UTR of beta-actin mRNA and is essential for beta-actin mRNA localization, but does not interact with

193 BP3/MARTA2 has been shown to be required for beta-actin mRNA localization.

194 llowing detection and localization of single beta-actin mRNA molecules in various mouse tissues.

195 Ribosomes and single beta-actin mRNA motility increase after stimulation, ind

196 Importantly, beta-actin mRNA near focal adhesions exhibited sub-diffu

197 studies indicate that axonal translation of beta-actin mRNA supports axon branching and axonal trans

198 " in the 3' untranslated region (UTR) of the beta-actin mRNA that is important for its localization a

199 hat the zipcode-binding protein 1 transports beta-actin mRNA to the focal adhesion compartment, where

200 Inhibition of axonal beta-actin mRNA translation disrupts arbor dynamics prim

201 n 1 (ZBP1) is required for the regulation of beta-actin mRNA transport and local translation underlyi

202 est a critical role for PAT1 in BDNF-induced beta-actin mRNA transport during postnatal development a

203 ZBP1 interaction with beta-actin mRNA was enhanced perinuclearly in neurons co

204 beta-Actin mRNA was targeted in whole A549 epithelial ce

205 de binding protein 1 (ZBP1) and ribosomes to beta-actin mRNA within subcellular compartments of prima

206 als the Src-dependent phosphorylation of the beta-actin mRNA zipcode binding protein 1 (ZBP1), which

207 ZBP1 and HuD bind the zipcode element in rat beta-actin mRNA's 3' UTR.

208 Using both coding (beta-actin mRNA) and long non-coding (NEAT1) RNAs, we sh

209 ZBP1 is needed for axonal localization of beta-actin mRNA, and introducing GFP with the 3'UTR of b

210 required ZBP1-mediated local translation of beta-actin mRNA, and therefore ZBP1 regulates protein sy

211 used these improvements to study endogenous beta-actin mRNA, which has 24xMS2 binding sites inserted

212 GAP-43 mRNA can deplete axons of endogenous beta-actin mRNA.

213 ced levels of a stabilizing factor (HuR) for beta-actin mRNA.

214 ucleoprotein-associated assembly factors and beta-actin mRNA.

215 own for its role in the local translation of beta-actin mRNA.

216 d acute brain slices, we found that multiple beta-actin mRNAs can assemble together, travel by active

217 We show that beta-actin mRNAs freely access the entire nucleus and fe

218 Therefore, we imaged single beta-actin mRNAs tagged with MS2 stem loops colocalizing

219 limited spot, and the localization of single beta-actin mRNAs was measured in space and time.

220 me occurs naturally in the 3'-UTR of Vg1 and beta-actin mRNAs.

221 tropomyosin complexes containing filamentous beta-actin, nonmuscle myosin-2B (NM-2B) constructs, and

222 Ca-form, phosphate release from polymerizing beta-actin occurs much more rapidly and extensively than

223 Live-imaging EGFP-beta-actin or dendra2-beta-actin reveal stable F-actin c

224 ing with siRNA-resistant constructs encoding beta-actin or GAP-43 proteins, but only if the mRNAs wer

225 eptides, for example, from the C terminus of beta-actin or NLSIII of the p53 protein, slide along DNA

226 of Stain-Free staining as an alternative to beta-actin or the protein stain Ponceau S showed that St

227 which hPepT1 expression was regulated by the beta-actin or villin promoters; colitis was induced usin

228 , moesin, plastin 1, lamin B1, vimentin, and beta-actin) or in protein biosynthesis (glutamyl-prolyl-

229 ding RhoA, dynamin-1, kinesin, beta-tubulin, beta-actin, oxysterol-binding protein (OSBP)-related pro

230 Formation of the beta-actin-PDI complex was mediated by integrin-dependen

231 of NO and superoxide formation from eNOS by beta-actin plays an important role in endothelial functi

232 ns, aberrant NDRG4 and BMP3 methylation, and beta-actin, plus a hemoglobin immunoassay.

233 or knockdown leads to a marked reduction in beta-actin polymerization and cytoskeleton formation.

234 C7H2 induces beta-actin polymerization and F-actin stabilization, lin

235 Disruption of estradiol-induced beta-actin polymerization with cytochalasin D attenuated

236 We propose a role for the nuclear beta-actin pool in maintaining open chromatin for transc

237 ing the human BBS4 gene under control of the beta actin promoter.

238 sitional independence is linked to the human beta-actin promoter and is most likely a result of its t

239 ssion of the fragments driven by the chicken beta-actin promoter at nearly identical levels.

240 , T2/Onc3, in which the CMV enhancer/chicken beta-actin promoter drives oncogene expression.

241 ere isolated from transgenic FVB mice with a beta-actin promoter driving firefly luciferase and green

242 TG mice that overexpressed GLUT12 under a beta-actin promoter were generated.

243 zebrafish ubiquitin promoter and common carp beta-actin promoter, harboring a 250-bp homologous regio

244 Tomato-ttk) driven by a constitutive chicken beta-actin promoter.

245 orescent protein (GFP) using a small chicken beta-actin promoter.

246 biquitously express human PEDF driven by the beta-actin promoter.

247 vector with a modified CMV enhancer/chicken beta-actin promoter.

248 ansgenic mice were produced with the chicken beta-actin promoter.

249 omposite cytomegalovirus-IE enhancer/chicken beta-actin promoter.

250 Overexpressing Sox2 from a constitutive (beta-actin) promoter induces the expression of the prone

251 the methyl-CpG-binding protein 2 (MeCP2) and beta-actin promoters to drive low versus high transgene

252 the methyl-CpG-binding protein 2 (MeCP2) and beta-actin promoters.

253 LOD of 40 pM) as compared to the ubiquitous beta-actin protein (ca. 64 million molecules or 4.5 pg p

254 In particular, Shh stimulation increases beta-actin protein at the growth cone even when the cell

255 rate here that generation of a mouse lacking beta-actin protein by editing beta-actin gene to encode

256 icient neurons, which impaired enrichment of beta-actin protein in the growth cone.

257 Most notably, this treatment also restores beta-actin protein levels in axonal growth cones of SMN-

258 changes in newly synthesized HIF-1alpha and beta-actin protein levels mirror alterations in correspo

259 rat neurons, increasing axonal synthesis of beta-actin protein while decreasing axonal synthesis of

260 Decreasing axonal synthesis of beta-actin protein while increasing axonal synthesis of

261 ls can afford washing-free imaging of single beta-actin proteins.

262 We also compared NPM-ALK/ beta-actin ratios determined by ELISA to those independe

263 r between zipcode-binding protein 1 (ZBP1, a beta-actin RBP) and the kinesin-I motor complex.

264 Live-imaging EGFP-beta-actin or dendra2-beta-actin reveal stable F-actin cores with turnover and

265 We show that beta-actin's 3'-UTR can drive axonal localization of GFP

266 ng mutant fascin-2 (p.R109H) or mice lacking beta-actin share a common phenotype including progressiv

267 Immunofluorescence detection of beta-actin showed that it was absent in the KC fibroblas

268 y distinguish between photoluminescence from beta-actin-specific RCA and DNA probes freely diffusing

269 de (Ln) complexes and dyes that hybridize to beta-actin-specific RCA products in HaCaT cells can affo

270 ing protein 1 (ZBP1), which is necessary for beta-actin synthesis and growth cone turning.

271 We report that Netrin-1 triggers a burst of beta-actin synthesis at multiple non-repetitive sites, p

272 Local beta-actin synthesis in growth cones of developing axons

273 f plasticity and may be facilitated by local beta-actin synthesis, but dynamic information is lacking

274 wth factor (TGF) beta1/beta-actin, and FOXP3/beta-actin than did IgG4-negative MZL (p < 0.05).

275 cause a selective energy barrier relative to beta-actin that retards the equilibration between G- and

276 isolated mRNA was used for amplification of beta-actin to confirm the compatibility.

277 slices revealed immediate early induction of beta-actin transcription after depolarization.

278 r-identical performance in identifying mouse beta-actin transcripts, in comparison with the conventio

279 n (IL)-4/beta-actin, IL-10/beta-actin, IL-13/beta-actin, transforming growth factor (TGF) beta1/beta-

280 SDF-1, VEGF, and beta-actin up-regulation was detected at early time poin

281 on of stromal cell-derived factor-1 (SDF-1), beta-actin, vascular endothelial growth factor (VEGF), g

282 anization by the thiol-disulfide exchange in beta-actin via a redox-dependent mechanism.

283 reporter with the 3' untranslated region of beta-actin was attenuated with the Src family kinase-spe

284 gures 2e, 2f-g, 4a, 4j, 5a and 6b, unmatched beta-actin was inadvertently used as loading control for

285 The LNA bound to beta-actin was then stained using phycoerythrin-conjugat

286 The expression levels of HuD, ATG5, LC3, and beta-actin were determined by Western blot and quantitat

287 and integrin subunits alphav and beta5) and beta-actin were quantified by Western blotting at 0.5 an

288 ctions among FAK, VE-cadherin, vinculin, and beta-actin were simultaneously decreased, resulting in a

289 e cell, where it forms a disulfide bond with beta-actin when MEG-01 cells adhere via the alphaIIbbeta

290 e mRNA localization and local translation of beta-actin where the new actin participates in stabilizi

291 active species leading to S-nitrosylation of beta-actin, which causes temporary inhibition of beta(2)

292 posed to hyperoxia causes S-nitrosylation of beta-actin, which increases formation of short actin fil

293 One gene with a 5'-CpG island is cytoplasmic beta-actin, which is an abundantly expressed protein and

294 at PDI forms a disulfide-bonded complex with beta-actin with a molecular mass of 110 kDa.

295 tent to which the association of filamentous beta-actin with these different tropomyosin cofilaments

296 but does not interact with the characterized beta-actin zipcode element.

297 ding elements (PBEs) into mRNAs containing a beta-actin zipcode prevented axonal localization and tra

298 BP1 and HuD bind to overlapping sites in the beta-actin zipcode, but they recognize different feature

299 binding protein 1 (ZBP1/IMP1/IGF2BP1) to the beta-actin zipcode.

300 terized the interaction between ZBP1 and the beta-actin zipcode.