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1 f nicotine was blocked by prior injection of beta-adrenergic antagonists.
2 is effect was canceled by prior injection of beta-adrenergic antagonists.
3      Freshly isolated strips were exposed to beta-adrenergic antagonists and agonists and assayed for
4 nd decrements are reversed by propranolol, a beta-adrenergic antagonist, and abolished by selective b
5 microl) of either propranolol (a nonspecific beta-adrenergic antagonist), atenolol (a beta1-adrenergi
6              Consistent with these findings, beta-adrenergic antagonists blocked both the late and th
7                                              Beta-adrenergic antagonists completely blocked the relea
8                       Propranolol, a generic beta-adrenergic antagonist, decreased proliferation of K
9  D(2) receptor antagonist) or propranolol (a beta-adrenergic antagonist) did not alter DA-stimulated
10 de) systemic co-administration of alpha- and beta-adrenergic antagonists in young (n = 9; 26 +/- 1 ye
11 erging evidence suggests that beta-blockers (beta-adrenergic antagonists) increase brain clearance of
12  leptin-deficient and wild-type mice while a beta-adrenergic antagonist increases bone mass in wild-t
13 was completely blocked by prior injection of beta-adrenergic antagonists, indicating that beta-adrene
14                           Infusions of these beta-adrenergic antagonists into the central nucleus of
15 beneficial heart failure treatments, such as beta-adrenergic antagonists, involve improved contractil
16                  These findings suggest that beta adrenergic antagonists may have a therapeutic role
17 earance was blocked by pretreatment with the beta-adrenergic antagonist, nadolol (0.5 mg/kg), but not
18                      Nonselective alpha- and beta-adrenergic antagonists or saline were infused in th
19                               Treatment with beta-adrenergic antagonists or with S107, a small molecu
20 e with the nonselective alpha-adrenergic and beta-adrenergic antagonists phentolamine and propranolol
21                                 Although the beta-adrenergic antagonist propranolol (1) binds at rode
22                        Administration of the beta-adrenergic antagonist propranolol at encoding aboli
23    This effect was completely blocked by the beta-adrenergic antagonist propranolol but was not affec
24                                     When the beta-adrenergic antagonist propranolol was added to the
25 bits (saline, 1, 3, 5 or 10 mg kg(-1) of the beta-adrenergic antagonist propranolol) were trained wit
26 abbits (saline, 1, 3, 5 or 10 mg kg-1 of the beta-adrenergic antagonist propranolol) were trained wit
27 ing in the NPE but not PE was blocked by the beta-adrenergic antagonist propranolol, the gap junction
28 els, and these increases were blocked by the beta-adrenergic antagonist propranolol.
29          Moreover, a single injection of the beta-adrenergic antagonist, propranolol, prevented the n
30                                          The beta-adrenergic antagonist sotalol (10-4 M) reduced LL a
31 Although it is the most important target for beta-adrenergic antagonists, such as beta-blockers, rela
32 d infusion of dobutamine or treatment with a beta-adrenergic antagonist, suggesting that RLC is const
33                                          The beta-adrenergic antagonists timolol and propranolol did
34 E's effect could be reversed by propranolol (beta-adrenergic antagonist) treatment.
35 pineal melatonin formation and is induced by beta-adrenergic antagonists, which block melatonin produ
36 taAR in the heart and is the main target for beta-adrenergic antagonists, widely used in the treatmen