ライフサイエンスコーパス: beta-glucuronidase

1 an Escherichia coli gene encoding the enzyme beta-glucuronidase).

2 cally modified enzyme was superior to native beta-glucuronidase.

3 vity and could label bystander proteins near beta-glucuronidase.

4 8 of uidA, an E. coli-specific gene encoding beta-glucuronidase.

5 s activated by the oncologically significant beta-glucuronidase.

6 ORF was replaced with that of marker protein beta-glucuronidase.

7 nd after treatment of the urine samples with beta-glucuronidase.

8 alcohol drug surrogates under the action of beta-glucuronidase.

9 ith the lysosomal hydrolases cathepsin D and beta-glucuronidase.

10 vity was based on a loop unique to bacterial beta-glucuronidases.

11 op" present in microbial, but not mammalian, beta-glucuronidases.

12 A mixed enzymatic treatment containing beta-glucuronidase (100 U mL(-1)) and sulfatase (2.5 U m

13 beta-glucuronidase-activated FITC-TrapG did not interfer

14 uction by strain B301D and reduced levels of beta-glucuronidase activities of the sypA::uidA and syrB

15 eIMP-2 promoter as measured by a decrease in beta-glucuronidase activity after treatment.

16 ne, gus and BG, have been reported to encode beta-glucuronidase activity among human colonic bacteria

17 -activated FITC-TrapG did not interfere with beta-glucuronidase activity and could label bystander pr

18 tissue-specific expression when analyzed by beta-glucuronidase activity assays, differences in gusA

19 as non-sorbitol fermenting and negative for beta-glucuronidase activity but serotyped O nontypeable:

20 ssing the BOS1-beta-glucuronidase transgene, beta-glucuronidase activity could be detected only after

21 of transgenic Arabidopsis, with the highest beta-glucuronidase activity detected in pollen.

22 Here we detected an increase in beta-glucuronidase activity in faecal samples from obese

23 w that the upstream sequence of POTH1 drives beta-glucuronidase activity in response to light and in

24 (Logan) of (18)F-FEAnGA also correlated with beta-glucuronidase activity in the same brain regions.

25 glycyrrhiza (TFG), can significantly enhance beta-glucuronidase activity in vitro.

26 may provide a valuable tool for visualizing beta-glucuronidase activity in vivo.

27 ted versions of the AtSUS2:promoter fused to Beta-glucuronidase activity revealed an internal 421 bp

28 carried only the BG gene gave relatively low beta-glucuronidase activity that was not induced by 4-ni

29 e isolates that possessed only the gus gene, beta-glucuronidase activity was induced.

30 pment of near-IR (NIR) probes for imaging of beta-glucuronidase activity would be ideal to allow esti

31 ibition of root elongation, promotion of DR5-beta-glucuronidase activity, and reduction of Aux/IAA pr

32 oped two fluorescent probes for detection of beta-glucuronidase activity, one for the NIR range (cont

33 nsgenic plants were functionally assayed for beta-glucuronidase activity.

34 is probably the result of reduced bacterial beta-glucuronidase activity.

35 enetically disparate populations can provide beta-glucuronidase activity.

36 on of tumor growth in tumor models with high beta-glucuronidase activity.

37 might be associated with elevated levels of beta-glucuronidase, an enzyme previously associated with

38 Our studies show elevated levels of beta-glucuronidase, an enzyme that hydrolyzes the glycos

39 gene expression using northern and promoter-beta-glucuronidase analyses and found overlapping but di

40 criptome analysis are supported by promoter::beta-glucuronidase analyses of CHX genes and by other me

41 Promoter-beta-glucuronidase analyses revealed that SHM1 is predom

42 RT-PCR and beta-glucuronidase analyses showed that LTP5 is present

43 AtHAK5 promoter-beta-glucuronidase and -green fluorescent protein fusion

44 as the influence of carbohydrate quality on beta-glucuronidase and cancer activity, deserve further

45 ecimens were enzymatically deconjugated with beta-glucuronidase and extracted by a solid-phase extrac

46 he expression pattern of recombinant ProBTS::beta-GLUCURONIDASE and found that it is expressed in dev

47 tail by expressing in soybean roots promoter beta-glucuronidase and green fluorescent protein fusions

48 n promoter, when fused to the reporter genes beta-glucuronidase and green fluorescent protein, direct

49 is study sheds new light on the mechanism of beta-glucuronidase and helps to make industrial producti

50 rt inhibition can be mimicked by recombinant beta-glucuronidase and is associated with proteolytic de

51 In addition, the use of mixtures of beta-glucuronidase and sulfatase enzymes from different

52 ed by promoter and protein fusions using the beta-glucuronidase and the green fluorescent protein, re

53 curonidase, the auxin-sensitive reporter DR5:beta-glucuronidase, and auxin-dependent growth defects.

54 and several digestive enzymes (acid lipase, beta-glucuronidase, and cathepsins B and D).

55 nidase without inhibiting purified mammalian beta-glucuronidase, and they do not impact the survival

56 Thus, by probing the actions of microbial beta-glucuronidases, and by understanding which substrat

57 were additionally hydrolysed enzymatically (beta-glucuronidase/arylsulphatase, cellulase), the compo

58 ve mutant (fls2) as the scion and ALMT1(pro):beta-glucuronidase as the rootstock revealed that both C

59 2AF35b in roots were revealed by a promoter::beta-glucuronidase assay, with atU2AF35b expressed stron

60 d varied activities in FGF23 co-receptor and beta-glucuronidase assays in vitro and distinct properti

61 is analyses of the syrB1::uidA reporter with beta-glucuronidase assays.

62 ntitative real time-PCR, GGPPS promoter-GUS (beta-glucuronidase) assays and publicly available microa

63 ydrolytic kinetics catalyzed by bovine liver beta-glucuronidase at interstitial pH = 7.4 fit the Mich

64 Using a whole-gene translational fusion to beta-glucuronidase, AtSUC9 expression was found in sink

65 MPS VII is due to deficiency in beta-glucuronidase (beta-glu) enzymatic activity, which

66 beta-galactosidase (beta-gal) and beta-glucuronidase (beta-glucur) are both produced by E.

67 The direct inhibition of bacterial beta-glucuronidase (betaG) activity is expected to reduc

68 ated that there is an increase in release of beta-glucuronidase by activated microglia into the extra

69 rthermore, selective disruption of bacterial beta-glucuronidases by small molecule inhibitors allevia

70 to ethanol while incubating our samples with beta-glucuronidase, confirming that the methyl protons o

71 lysis, expression analysis of a PLP promoter-beta-glucuronidase construct in transgenic plants and in

72 repress ABA induction of the HVA22 promoter-beta-glucuronidase construct, while OsWRKY72 and -77 syn

73 Furthermore, AtLETM2 promoter beta-glucuronidase constructs displayed exclusive matern

74 atography (HPLC) showed that the activity of beta-glucuronidase could be increased by 2.66-fold via t

75 lysaccharidosis type VII (MPS VII) caused by beta-glucuronidase deficiency.

76 hensive analysis of AAV1-treated brains from beta-glucuronidase-deficient mice (mucopolysaccharidosis

77 We used the beta-glucuronidase-deficient nonobese diabetic/severe co

78 structure of one inhibitor bound to E. coli beta-glucuronidase demonstrates that it contacts and ord

79 validation study, coadministration with oral beta-glucuronidase derived from Escherichia coli and pre

80 zation of auxin redistribution using the DR5:beta-glucuronidase (DR5:GUS) auxin-responsive reporter s

81 A was able to detect an increased release of beta-glucuronidase during neuroinflammation.

82 Urine samples are incubated with beta-glucuronidase (E. coli K12) and then analyzed by li

83 six mutant alleles in the same codon of the beta-glucuronidase-encoding GUS transgene.

84 glycosidase enzyme (beta-gly) and W492G in a beta-glucuronidase enzyme (beta-gluc), in which we engin

85 In the GI tract, the microbiota express beta-glucuronidase enzymes that remove the glucuronic ac

86 es have been conventionally studied by using beta-glucuronidase enzymes to release the phase I metabo

87 Bacterial beta-glucuronidases expressed by the symbiotic intestina

88 ly trapped on purified beta-glucuronidase or beta-glucuronidase-expressing CT26 cells (CT26/mbetaG) b

89 etaG) but not on bovine serum albumin or non-beta-glucuronidase-expressing CT26 cells used as control

90 chrome to nucleophilic moieties located near beta-glucuronidase-expressing sites.

91 ute vacuum treatment resulted in the highest beta-glucuronidase expression in the leaf, male and fema

92 y reverse transcription PCR, promoter-driven beta-glucuronidase expression in transgenic plants, and

93 Promoter-beta-glucuronidase expression of one transcription facto

94 Promoter::beta-glucuronidase expression studies show that individu

95 beta-Glucuronidase expression was detected in mature pol

96 and a delay in the asymmetric auxin-induced beta-glucuronidase expression with gravistimulation as c

97 ement of hydrogen peroxide-responsive AoPR10-beta-glucuronidase expression, suppression of plant stre

98 a heterologous in vivo Pv-ALF/phas-GUS (for beta-glucuronidase) expression system in transgenic Arab

99 (luciferase, green fluorescent protein, and beta-glucuronidase) facilitated in vivo profiling at the

100 Beta-glucuronidase from both Escherichia coli and bovine

101 preparations from different sources, such as beta-glucuronidase from Escherichia coli, were found to

102 Therefore, the use of beta-glucuronidase from H. pomatia combined with an enzy

103 trices (urine and plasma) were studied using beta-glucuronidase from Helix pomatia.

104 t and under different light treatments using beta-glucuronidase fusion constructs with the promoters

105 by using both reverse transcription-PCR and beta-glucuronidase fusion constructs.

106 Promoter-beta-glucuronidase fusion experiments showed that MEDIAT

107 of Arabidopsis plants containing the AtHD2C:beta-glucuronidase fusion gene revealed that AtHD2C was

108 s of Arabidopsis plants containing the HDA19:beta-glucuronidase fusion gene revealed that HDA19 was e

109 que NF-Y complexes, we have created promoter:beta-glucuronidase fusion lines for all 36 Arabidopsis g

110 wild-type and ABA response mutants, an ABI8-beta-glucuronidase fusion protein is localized primarily

111 ots by a promoter::green fluorescent protein-beta-glucuronidase fusion revealed strong gene expressio

112 sent at similar levels, and the two promoter-beta-glucuronidase fusion transgenes show very similar e

113 The promoter:beta-glucuronidase fusions also demonstrated that RAP2.6

114 Expression patterns of SWEET2-beta-glucuronidase fusions confirmed that SWEET2 accumul

115 RAP2.6L promoter:beta-glucuronidase fusions demonstrated that the up-regu

116 R1-GFP (green fluorescent protein) and NaKR1-beta-glucuronidase fusions driven by the native promoter

117 work, transgenic plants expressing ProRPL10:beta-glucuronidase fusions show that, while AtRPL10A and

118 Arabidopsis lines carrying AtWRKY30 promoter-beta-glucuronidase fusions showed transcriptional activi

119 resulting in increased activity of secreted beta-glucuronidase fusions that result from gene trap in

120 were independently validated using promoter:beta-glucuronidase fusions with the MtCRE1 CK receptor g

121 ression of each GGT in plants containing GGT:beta-glucuronidase fusions, the temporal and spatial pat

122 enes using data mining and promoter-reporter beta-glucuronidase fusions.

123 beta-Glucuronidase-fusions to full-length ARR2, ARR12, a

124 erobacteriaceae) and approximately 9% higher beta-glucuronidase gene abundance compared with nonrespo

125 e expression of two reporter constructs: the beta-glucuronidase gene driven by the GA-inducible Amy32

126 A-stimulated lateral root production and DR5-beta-glucuronidase gene expression.

127 el signature sequencing, and promoter-driven beta-glucuronidase gene expression.

128 equired for the transgenic expression of the beta-glucuronidase gene fused to a synthetic auxin-induc

129 ta-Synthase-like1 (MtCBS1), using a promoter-beta-glucuronidase gene fusion, which revealed expressio

130 ox genes in Arabidopsis thaliana by promoter-beta-glucuronidase gene fusions and by quantitative RT-P

131 The Lactobacillus gasseri ADH beta-glucuronidase gene, gusA, was cloned previously and

132 cally processed as predicted by bovine liver beta-glucuronidase, generating 2-aminoethylGdDO3A, 2.

133 on analyses using a complete set of promoter-beta-glucuronidase/green fluorescent protein reporter li

134 Here, RNA analysis and SWEET17-beta-glucuronidase/-GREEN FLUORESCENT PROTEIN fusions ex

135 oral expression patterns as shown by DAO1pro:beta-glucuronidase (GUS) activity and DAO1pro:YFP-DAO1 s

136 sion of VHA-c1, monitored by promoter-driven beta-glucuronidase (GUS) activity was responsive to ligh

137 e gene and protein levels was analyzed using beta-glucuronidase (GUS) activity, quantitative reverse

138 dopsis lines were constructed expressing the beta-glucuronidase (GUS) and green fluorescence protein

139 e RT-PCR and transcriptional fusions to both beta-glucuronidase (GUS) and green fluorescent protein (

140 germinating seeds in qRT-PCR analysis, while beta-glucuronidase (GUS) assays on OsACBP2pro::GUS rice

141 upstream of these two genes was confirmed by beta-glucuronidase (GUS) assays.

142 e kinetics of wild-type and in vitro evolved beta-glucuronidase (GUS) at the single molecule level.

143 An inherited deficiency of beta-glucuronidase (GUS) causes mucopolysaccharidosis ty

144 e recently reported that PerT-GUS, a form of beta-glucuronidase (GUS) chemically modified to eliminat

145 a common manifestation of MPS VII because of beta-glucuronidase (GUS) deficiency.

146 For Arabidopsis research, beta-glucuronidase (GUS) enhancer-trap lines have been c

147 Bacterial beta-glucuronidase (GUS) enzymes cause drug toxicity by

148 Gut microbial beta-glucuronidase (GUS) enzymes have been suggested to

149 The gut microbiota harbor diverse beta-glucuronidase (GUS) enzymes that liberate glucuroni

150 (GI) tract toxicity caused by gut bacterial beta-glucuronidase (GUS) enzymes.

151 In proof-of-principle experiments, a 35S::beta-glucuronidase (GUS) expression cassette was introdu

152 Here we show that beta-glucuronidase (GUS) expression from sense T-strands

153 ion identified multiple lines that exhibited beta-glucuronidase (GUS) expression in the micropylar en

154 xpression patterns, as monitored by promoter-beta-glucuronidase (GUS) fusion and RT-PCR experiments.

155 Promoter-beta-glucuronidase (GUS) fusion experiments and seed mRN

156 At5g23960 and At5g44630 promoter-beta-glucuronidase (GUS) fusion experiments demonstrated

157 Furthermore, promoter-beta-glucuronidase (GUS) fusion transgenics were generat

158 gene fusion assay of RIE1 promoter with the beta-glucuronidase (GUS) gene.

159 Inheritance analyses using beta-glucuronidase (GUS) histochemical staining revealed

160 a transgene with three direct repeats of the beta-glucuronidase (GUS) open reading frame (ORF) is ass

161 tive promoter and a coding region for either beta-glucuronidase (Gus) or glyphosate acetyltransferase

162 slated region (UTR) was used to drive either beta-glucuronidase (GUS) or green fluorescent protein (G

163 cyp71av1 promoter sequence was fused to the beta-glucuronidase (GUS) reporter gene and two varieties

164 lant system, we analyzed the activation of a beta-glucuronidase (GUS) reporter gene by enhancers cont

165 terium tumefaciens strain AGL1 harboring the beta-glucuronidase (GUS) reporter gene driven by the cau

166 Transcriptional regulatory region-beta-glucuronidase (GUS) reporter gene fusions introduce

167 ncrease in the transcription of a downstream beta-glucuronidase (GUS) reporter gene in tobacco leaves

168 nsformed with the RTE1 promoter fused to the beta-glucuronidase (GUS) reporter gene revealed that RTE

169 Gene and enhancer trap vectors carrying the beta-glucuronidase (GUS) reporter gene were inserted int

170 se regulator (ARR)5 gene promoter fused to a beta-glucuronidase (GUS) reporter gene, and cytokinin ox

171 , TAPNAC promoter elements were fused to the beta-glucuronidase (GUS) reporter gene, and spatial and

172 At3g25830 promoter activity, measured with a beta-glucuronidase (GUS) reporter gene, was primarily fo

173 ybean containing a PG11a promoter fused to a beta-glucuronidase (GUS) reporter gene.

174 s promoter mutants driving expression of the beta-glucuronidase (gus) reporter gene.

175 s containing an INPACT cassette encoding the beta-glucuronidase (GUS) reporter had negligible backgro

176 -responsive Em promoter from wheat linked to beta-glucuronidase (GUS) to determine whether ABI3/VP1,

177 ansgenic Arabidopsis lines bearing promoter::Beta-glucuronidase (GUS) transcriptional fusions as well

178 Seven of 10 single-crossover beta-glucuronidase (GUS) transcriptional reporters in ge

179 have examined the differential expression of beta-glucuronidase (GUS) transgenes under the control of

180 of transgenic plants harboring an SOB5:SOB5-beta-glucuronidase (GUS) translational fusion under the

181 to direct the evolution of Escherichia coli beta-glucuronidase (GUS) variants with increased beta-ga

182 Expression of the reporter construct EBS: beta-glucuronidase (GUS) was detected in Arabidopsis roo

183 ncoded the minor activity, and ARGAH1-driven beta-glucuronidase (GUS) was expressed throughout the se

184 Treatment of human beta-glucuronidase (GUS) with sodium metaperiodate follo

185 despite the fact that the uidA gene product, beta-glucuronidase (GUS), was produced only when the cel

186 or-activated variants of the reporter enzyme beta-glucuronidase (GUS).

187 P) and nonphosphorylated (NP) forms of human beta-glucuronidase (GUS).

188 order resulting from inherited deficiency of beta-glucuronidase (GUS).

189 r to infection with Agrobacterium carrying a beta-glucuronidase (GUS, uidA) gene with an artificial i

190 cco to regulate expression of uidA (encoding beta-glucuronidase; GUS) and the cytokinin-biosnythetic

191 ysosomal storage disease caused by deficient beta-glucuronidase (GUSB) activity resulting in defectiv

192 k cancer and Transferrin receptor (TFRC) and beta-Glucuronidase (GUSB) in pancreatic cancer were iden

193 The lysosomal enzyme beta-glucuronidase (Gusb) is a key regulator of Lyme-ass

194 evention of severe cardiac manifestations in beta-glucuronidase (GUSB) null mice BM-transplanted i.v.

195 approach, we identified the lysosomal enzyme beta-glucuronidase (GUSB), a member of a large family of

196 Selective inhibitors of gut bacterial beta-glucuronidases (GUSs) are of particular interest in

197 The human gut microbiota encodes beta-glucuronidases (GUSs) that play key roles in health

198 in's ability to ferment sorbitol and express beta-glucuronidase have complicated its detection and id

199 asured the distribution of recombinant human beta-glucuronidase (hGUS) and reduction in storage by we

200 vels were unchanged compared to control (DR5:beta-glucuronidase), however, in the seedlings expressin

201 auxin resistance, ectopically expressed DR5:beta-glucuronidase in developing embryos, and defective

202 a higher activity of the auxin reporter DR5-beta-glucuronidase in lateral root apices.

203 Quin-C1 induces chemotaxis and secretion of beta-glucuronidase in peripheral blood neutrophils with

204 expected, incubation of these prodrugs with beta-glucuronidase in the culture medium led to much mor

205 Bacterial beta-glucuronidase in the human colon plays an important

206 nsin] and Thi1.2 [thionin]) or SA (PR1 [PR1a-beta-glucuronidase in tobacco]) signaling when both sign

207 y to express the reporter gusA gene encoding beta-glucuronidase in transgenic tobacco seeds relative

208 ase inhibitors that inhibit Escherichia coli beta-glucuronidase in vitro with Ki values between 180 n

209 moieties from drug metabolites by bacterial beta-glucuronidases in the GI lumen can significantly da

210 and structural basis of selective microbial beta-glucuronidase inhibition, which may improve human d

211 y effect of Klotho on NaPi-2a was blocked by beta-glucuronidase inhibitor but not by protease inhibit

212 Here we characterize novel microbial beta-glucuronidase inhibitors that inhibit Escherichia c

213 Potent bacterial beta-glucuronidase inhibitors were identified by high-th

214 cent protein fusion, beta-galactosidase, and beta-glucuronidase) into the F14.5L, J2R (encoding thymi

215 ossing of the abscission marker, Pro(PGAZAT):beta-glucuronidase, into the mutant reveals that while f

216 ction with A. tumefaciens cells carrying the beta-glucuronidase intron reporter gene.

217 beta-glucuronidase is an attractive reporter and prodrug

218 beta-glucuronidase is involved in the hydrolysis of glyc

219 beta-Glucuronidase is the key enzyme in the biotransform

220 xpression of the auxin-induced reporter (DR5-beta-glucuronidase) is reduced in initiating lateral roo

221 ession patterns inferred from these promoter:beta-glucuronidase lines for roots, light- versus dark-g

222 GPA1 promoter::beta-glucuronidase lines indicate that the GPA1 promoter

223 vector-adapted vectors with three reporters, beta-glucuronidase, luciferase, and green fluorescent pr

224 n of the untranslated regions of StBEL5 to a beta-glucuronidase marker, translation in tobacco (Nicot

225 beta-glucuronidase-mediated hydrolysis of the glucuronyl

226 Therefore, beta-glucuronidase might be a biomarker for ongoing neur

227 7 that are non-sorbitol fermenting (NSF) and beta-glucuronidase negative (GUD(-)) carry a large virul

228 t recent common ancestor of the contemporary beta-glucuronidase-negative, non-sorbitolfermenting STEC

229 TC-TrapG was selectively trapped on purified beta-glucuronidase or beta-glucuronidase-expressing CT26

230 monotherapy of necrotic tumors that liberate beta-glucuronidase or for antibody-directed enzyme prodr

231 The Vp1 promoter fused to beta-glucuronidase or green fluorescent protein reproduc

232 nd temporal characterization, using Pro(HWS):beta-glucuronidase or Pro(HWS):green fluorescent protein

233 enerating stable transgenic lines expressing beta-glucuronidase plus (GUSplus), green fluorescent pro

234 EC) O55:H7 (sorbitol fermenting [SOR(+)] and beta-glucuronidase positive [GUD(+)]), through sequentia

235 The beta-glucuronidase-positive O157 variants, although phyl

236 It is well-known that hydrolysis with beta-glucuronidase presents some limitations that may re

237 RT-PCR and beta-glucuronidase-promoter fusion analyses demonstrated

238 and brain microvasculature, indicating that beta-glucuronidase reached brain parenchyma via the peri

239 or because ISL could reduce the Km and Ea of beta-glucuronidase reacting with GL.

240 e PET tracer (18)F-FEAnGA was able to detect beta-glucuronidase release during neuroinflammation in a

241 Green fluorescent protein and beta-glucuronidase reporter analyses indicated that NRT1

242 enic studies with an ABA-INSENSITIVE2 (ABI4)-beta-glucuronidase reporter construct revealed that in r

243 ProOskn2:beta-glucuronidase reporter expression was down-regulate

244 Beta-glucuronidase reporter expression, driven by YSL1 a

245 AGP31 promoter-beta-glucuronidase reporter gene analysis showed express

246 In an Arabidopsis-protoplast beta-glucuronidase reporter gene assay, as well as in a

247 eaction analyses and promoter fusions to the beta-glucuronidase reporter gene confirmed the expressio

248 CYP72B1 translational fusions with the beta-glucuronidase reporter gene demonstrated that prote

249 e ipx genes were variably induced in planta; beta-glucuronidase reporter gene expression analysis of

250 1 expression pattern, determined by promoter-beta-glucuronidase reporter gene expression, is associat

251 Expression analysis of the beta-glucuronidase reporter gene fused to the NtSCP1 tra

252 We linked this to the beta-glucuronidase reporter gene gusA.

253 nslation start site direct expression of the beta-glucuronidase reporter gene primarily in the vascul

254 , expression of the RPT2 promoter fused to a beta-glucuronidase reporter gene shows differential expr

255 were established to be root-specific using a beta-glucuronidase reporter gene strategy.

256 eased expression of the auxin-responsive DR5:beta-glucuronidase reporter gene, suggesting a perturbat

257 ying four copies of the GCC-box fused to the beta-glucuronidase reporter gene, we showed that the GCC

258 tested in C. reinhardtii chloroplasts using beta-glucuronidase reporter genes, and the nearly identi

259 -head oriented green fluorescent protein and beta-glucuronidase reporter genes, both transcripts and

260 OCALIZATION SIGNAL-GREEN FLUORESCENT PROTEIN/beta-GLUCURONIDASE reporter lines throughout the life cy

261 Through microarray analysis and beta-glucuronidase reporter lines, we showed that the ge

262 of the gene expression patterns in promoter:beta-glucuronidase reporter lines.

263 is of auxin influx facilitator expression in beta-glucuronidase reporter plants revealed that AUXIN R

264 In addition, primer extension analyses and a beta-glucuronidase reporter system were used to quantita

265 KN infection using both quantitative PCR and beta-glucuronidase reporter transgenic lines.

266 otein and tags mutant pollen grains with the beta-glucuronidase reporter.

267 n pollen, as indicated from a promoter::GUS (beta-glucuronidase) reporter analysis and expression pro

268 that OsGZF1 can down-regulate a GluB-1-GUS (beta-glucuronidase) reporter and OsGZF1 was also able to

269 upts the expression pattern of the GL2::GUS (beta-glucuronidase) reporter gene.

270 of POTH1 when fused to an expression marker beta-glucuronidase, repressed its translation in tobacco

271 hesis and biological evaluation of the first beta-glucuronidase-responsive albumin-binding prodrug de

272 We developed a platform employing beta-glucuronidase selective activity-based probes to de

273 Promoter fusions to beta-glucuronidase showed strong expression in the stele

274 ed by 2.66-fold via the addition of ISL to a beta-glucuronidase solution that contained GL at a 3:10

275 As evaluated by beta-glucuronidase staining and independently confirmed

276 In the flower, beta-glucuronidase staining occurred throughout the pist

277 ptase-polymerase chain reaction and promoter:beta-glucuronidase studies indicate that all AtGT genes

278 Promoter:beta-glucuronidase studies show that ECA3 is expressed i

279 evere diarrhea caused by symbiotic bacterial beta-glucuronidases that reactivate the drug in the gut.

280 localization of the provascular marker Athb8:beta-glucuronidase, the auxin-sensitive reporter DR5:bet

281 rug therapy with antibodies that can deliver beta-glucuronidase to target tumor cells.

282 In transgenic plants expressing the BOS1-beta-glucuronidase transgene, beta-glucuronidase activit

283 ntity markers such as a viviparous1 promoter-beta-glucuronidase transgene.

284 We used both promoter-beta-glucuronidase transgenic plants and immunolocalizat

285 Tissue-specific accumulation of an OBP3:OBP3-beta-glucuronidase translational fusion is regulated by

286 reversed 6 weeks after gene transfer in AAV4 beta-glucuronidase-treated MPS VII mice.

287 atly reduced by either chondroitinase ABC or beta-glucuronidase treatment.

288 ' deletions, were fused to the reporter gene beta-glucuronidase (uidA) and analyzed in transgenic Nic

289 A herpes simplex virus type 2 (HSV-2) UL24 beta-glucuronidase (UL24-betagluc) insertion mutant was

290 in vivo functional assay using the reporter beta-glucuronidase under the auxin-inducible DR5 promote

291 ion, which can be rescued by expressing SUF4-beta-glucuronidase under the control of the SUF4 promote

292 -2 protoplasts inhibited nuclear import of a beta-glucuronidase-VirD2 nuclear localization signal fus

293 nteraction affinity for the lysosomal enzyme beta-glucuronidase was also much lower (K(d) = 54 microm

294 Thus, an enzymatic deconjugation with beta-glucuronidase was optimized.

295 uman IGF-II fused to the C terminus of human beta-glucuronidase was taken up by MPS VII fibroblasts i

296 beta-Glucuronidase was used to remove nonreducing-termin

297 Recombinant AAV4 vectors encoding beta-glucuronidase were injected unilaterally into the l

298 ociated with BC share an enzymatic activity, Beta-Glucuronidase, which may promote breast cancer.

299 usA2 and gusA3, were recovered that produced beta-glucuronidase with increased activity in neutral pH

300 All compounds are selective for E. coli beta-glucuronidase without inhibiting purified mammalian