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1  cells to decrease CXCL12 levels through the beta3-adrenergic receptor.
2 ment-binding protein 1, glycerol kinase, and beta3-adrenergic receptor.
3 n WAT and could be reversed by antagonism of beta3 adrenergic receptors.
4 nd by genetic deletion of stromal beta2- and beta3-adrenergic receptors.
5  of adipocyte lipolysis by fasting (24 h) or beta3-adrenergic receptor activation by CL316, 243 (CL)
6 olerant, expended less energy in response to beta3-adrenergic receptor activation, and were more insu
7  Ppp1r3c are rapidly induced following acute beta3-adrenergic receptor activation.
8 e, the liver fatty acid-binding protein, the beta3-adrenergic receptor, adipsin and the peroxisome pr
9                                    Sustained beta3 adrenergic receptor (ADRB3) activation simultaneou
10 y to a twofold increase in the expression of beta3 adrenergic receptor (Adrb3) at both the mRNA and p
11 er a beiging response: cold temperatures and beta3-adrenergic receptor (Adrb3) agonists.
12 ccompanied by sustained sympathetic tone and beta3-adrenergic receptor (ADRB3) signaling in BAT.
13 ptin (OB), the leptin receptor (OBR/DB), the beta3-adrenergic receptor (ADRB3), lipoprotein lipase (L
14                          Acute activation of beta3-adrenergic receptors (ADRB3) triggers lipolysis an
15                                          The beta3-adrenergic receptor (ADRbeta3) is a seven-membrane
16 moneutrality were treated with the selective beta3 adrenergic receptor agonist CL 316, 243 and underw
17                        We used CL-316,243, a beta3 adrenergic receptor agonist, to acutely elevate bl
18 made mice cold intolerant and insensitive to beta3 adrenergic receptor agonist-induced increase in wh
19 m-adapted rats by infusions of the selective beta3-adrenergic receptor agonist CL 316,243.
20 ital, and given intraperitoneally either the beta3-adrenergic receptor agonist CL-316,243, 1 mg/kg (n
21                           Treatment with the beta3-adrenergic receptor agonist CL316,243 increased Cx
22 Here, we report a role of Tregs in enhancing beta3-adrenergic receptor agonist CL316243 (CL)-stimulat
23 ghly enantioselective total synthesis of the beta3-adrenergic receptor agonist Solabegron.
24 of these receptors with BRL37344, a specific beta3-adrenergic receptor agonist, promoted migration th
25 mors were treated with CL-316243, a specific beta3-adrenergic receptor agonist, which sensitizes insu
26 -regulation of UCP1 after stimulation with a beta3-adrenergic receptor agonist.
27 e consume less oxygen after treatment with a beta3-adrenergic-receptor agonist and that they are sens
28 etanilides were synthesized and evaluated as beta3-adrenergic receptor agonists (beta3-AR) for the tr
29 ermogenic fat cells that can be activated by beta3-adrenergic receptor agonists.
30 resistance to therapeutic interventions with beta3-adrenergic receptor agonists.
31 c signaling molecules, i.e., norepinephrine, beta3-adrenergic receptor, and cAMP; the transcriptional
32 adipogenic genes (LpL, adipsin, GLUT-4, aP2, beta3-adrenergic receptor, and peroxisome proliferator-a
33 ed the effects of treatment with a selective beta3 adrenergic receptor (AR) agonist (CL 316,243 [1 mg
34                           Epinephrine or the beta3-adrenergic receptor (AR) agonist CL 316,243 (CL) s
35 nergic receptor agonist isoproterenol or the beta3-adrenergic receptor (AR)-specific agonist CL 31624
36                                   beta1- and beta3-adrenergic receptors (AR) are the predominant beta
37                                      Cardiac beta3-adrenergic receptors (ARs) are upregulated in dise
38                    BACKGROUNDMirabegron is a beta3-adrenergic receptor (beta3-AR) agonist approved on
39                                          The beta3-adrenergic receptor (beta3-AR) has important biolo
40          A robust change in abundance of the beta3-adrenergic receptor (beta3-AR) is observed in brow
41 whether ROS or antioxidant treatment affects beta3-adrenergic receptor (beta3-AR) stimulation-induced
42 ulated by the sympathetic nervous system via beta3-adrenergic receptors (beta3-AR).
43                                              beta3-Adrenergic receptors (beta3-ARs) are expressed pre
44                      Adipose tissue contains beta3-adrenergic receptors (beta3-ARs), and this study w
45  due to enhanced signaling through adipocyte beta3-adrenergic receptors (beta3-ARs), indicating that
46 uences of dual Gs/Gi protein coupling of the beta3-adrenergic receptor (beta3AR) in 3T3-F442A adipocy
47                                          The beta3-adrenergic receptors (beta3ARs) may represent a ne
48 s on lipolysis are driven by lower levels of beta3-adrenergic receptor, decreased cAMP and PKA signal
49                                              beta3-Adrenergic receptor downregulation also occurs aft
50 ogenesis is transduced by the beta2, but not beta3, adrenergic receptor expressed on stromal cells of
51                                       Murine beta3-adrenergic receptor expression in adipocytes is or
52  women with the Trp64Arg polymorphism of the beta3-adrenergic receptor gene have lower daily energy e
53       In mice with genetic deficiency of the beta3 adrenergic receptor, hematopoietic stem cells did
54                               Stimulation of beta3-adrenergic receptors increases metabolic rate via
55 ces adipogenesis and adult deletion enhances beta3-adrenergic-receptor-induced beige adipocyte format
56 hese data indicate that neural activation of beta3-adrenergic receptors is an important determinant o
57                                          The beta3 adrenergic receptor, located on chromosome 8, is a
58              These data show that alpha1 and beta3-adrenergic receptors may contribute to the mediati
59                                Activation of beta3 adrenergic receptors on the surface of adipocytes
60 s, whereas pharmacological activation of the beta3-adrenergic receptor on BAs was sufficient, suggest
61   We show, for the first time, expression of beta3-adrenergic receptors on cultured retinal endotheli
62 nt study, we hypothesized that activation of beta3-adrenergic receptors on retinal endothelial cells
63 (2+) cycling by activation of alpha1- and/or beta3-adrenergic receptors or the SERCA2b-RyR2 pathway s
64  to be regulated by insulin (e.g. Glut-1 and beta3-adrenergic receptor), other novel insulin-sensitiv
65    These results support the hypothesis that beta3-adrenergic receptors play a role in proliferation
66 ived brown adipocytes, displayed an impaired beta3-adrenergic receptor response that was characterize
67                  Moreover, cold exposure and beta3-adrenergic receptor signaling, conditions that ind
68 on dioxide production which was dependent on beta3-adrenergic receptor signaling, thereby favoring ne
69 romoted regeneration by activating beta2 and beta3 adrenergic receptor signalling in LepR(+) cells, a
70          Here we show that BAT activation by beta3-adrenergic receptor stimulation protects from athe
71 ion about which signals activate BA, besides beta3-adrenergic receptor stimulation, is limited.
72                                    beta3-AR (beta3-adrenergic receptor) stimulation improved systolic
73            NE enhances thermogenesis through beta3-adrenergic receptors to activate brown adipose tis
74 r comparison, polymorphisms in the beta2 and beta3 adrenergic receptors were also evaluated.
75  receptor nor polymorphisms in the beta2 and beta3 adrenergic receptors were associated with resting
76                                              Beta3-adrenergic receptors were not detected.