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1 s the localized interaction of PD-L1 and the beta4 integrin.
2 ls to resist ferroptosis in concert with the beta4 integrin.
3 ntial for its binding with the cytodomain of beta4 integrin.
4 th the upregulation of MMP-9 and cleavage of beta4 integrin.
5 atic MNNG-HOS cells have increased levels of beta4 integrin.
6  p63 was rescued by signalling downstream of beta4 integrin.
7 s, epitope-tagged PMP22 forms a complex with beta4 integrin.
8 e.g. hCLCA2, mCLCA5, mCLCA1, and bCLCA2) and beta4 integrin.
9 if within the extracellular I-like domain of beta4 integrin.
10 essed in endothelia, failed to interact with beta4 integrin.
11 elial cells, which express little endogenous beta4 integrin.
12  autoantibodies in OCP sera specific for the beta4 integrin.
13 y up-regulated genes, including survivin and beta4-integrin.
14 was positive, yet attenuated, for alpha6 and beta4 integrins.
15 uced the expression of hemidesmosomal alpha6/beta4-integrins.
16                       Phosphorylation of the beta4 integrin, a hemidesmosome component, induces disas
17                                The levels of beta4 integrin, a molecule involved in the linkage betwe
18  epithelial cells upregulate and internalize beta4-integrin along with its matrix substrate, laminin.
19 bnormalities in the expression of the alpha6 beta4 integrin, an integral component of hemidesmosomes.
20 dc)-1 engaging the cytoplasmic domain of the beta4 integrin and coupling of the integrin to human epi
21 eviously uncharacterized interaction between beta4 integrin and ezrin, a membrane-cytoskeletal linker
22 portantly, the concomitant overexpression of beta4 integrin and FAK significantly correlates with mal
23 physical and functional interactions between beta4 integrin and FAK that influence breast cancer mali
24                                              beta4 integrin and focal adhesion kinase (FAK) are often
25 a4 integrin, which, in turn, recruits FAK to beta4 integrin and leads to FAK activation and signaling
26  inflammation was delayed, and expression of beta4 integrin and p21 was reduced in lysates of constit
27 cular arrest of cancer cells via adhesion to beta4 integrin and promote early, intravascular, metasta
28 However, the amount of Rac1 associating with beta4 integrin and the activity of both Rac1 and cofilin
29  transfected constructs encoding the mutated beta4 integrins and a GFP-conjugated wild type beta4 int
30 e following levels: p < 0.001 for alpha6 and beta4 integrins and the beta3 chain of laminin 5; p < 0.
31   In these same cells, BPAG1e, the truncated beta4 integrin, and type XVII collagen (Col XVII), a tra
32  three signaling pathways (NF-kappaB, alpha6-beta4-integrin, and IL-1) as differentially upregulated
33                                         Anti-beta4 integrin antibody attenuated rLN-alpha2G-mediated
34                             Circulating anti-beta4 integrin antibody may have an important role in th
35          Treatment of K8(-/-) mice with anti-beta4-integrin antibody up-regulated survivin, and induc
36 ta-catenin was markedly elevated in PBK, and beta4 integrin appeared to be reduced in PBK.
37 uring early stages of myelination, PMP22 and beta4 integrin are coexpressed at the cell surface and c
38 also a functional connection between CD9 and beta4 integrins, as evidenced by anti-CD9 antibody effec
39 es and JEB keratinocytes, induced to express beta4 integrin, assemble laminin-332 in linear tracks ov
40      Here, we tested the hypothesis that the beta4 integrin-associated plakin protein, bullous pemphi
41 keratinocytes also display reduced levels of beta4 integrins at the cell surface but increased total
42 tinct sites at the extreme C terminus of the beta4 integrin cytoplasmic domain.
43                                              beta4 integrin-deficient (JEB) keratinocytes display abe
44 odia, and directed migration are restored in beta4 integrin-deficient cells by inducing expression of
45  typical of confluent, stationary cells, and beta4 integrin dynamics are reduced in knockdown cells c
46 on was accompanied by increases in beta1 and beta4 integrin epidermal progenitor markers.
47 r hemidesmosomal proteins, nor the amount of beta4 integrin expressed at the cell surface.
48   Our analysis revealed that manipulation of beta4 integrin expression and signaling impacted SPARC e
49                               Suppression of beta4 integrin expression by shRNA and disruption of bet
50            These findings suggest a role for beta4 integrin expression in the metastatic phenotype in
51      These data begin to integrate ezrin and beta4 integrin expression into a model of action for the
52 on appears to be critical for maintenance of beta4 integrin expression.
53 helial cells contingent upon epithelial cell beta4-integrin expression.
54                                          The beta4 integrin-ezrin interaction appears to be critical
55 escribes a pro-metastatic EGFR/Src-dependent beta4 integrin/FAK complex that is involved in breast ca
56                       Upon disruption of the beta4 integrin/FAK complex, tumorigenesis and metastasis
57 tegrin expression by shRNA and disruption of beta4 integrin function by transfection of dominant-nega
58 ta4 integrins and a GFP-conjugated wild type beta4 integrin (GFP-beta4WT) into 804G cells, which asse
59                   Although the expression of beta4 integrin had no effect on the rate of cell movemen
60 the 90- and 35-kDa CLCA subunits bind to the beta4 integrin in a metal ion-dependent manner.
61 re rescued following expression of wild-type beta4 integrin in JEB cells.
62                                   A chimeric beta4 integrin in which the indicated SDL sequence had b
63                     Expression of the alpha6 beta4 integrin is altered at the dermal-epidermal baseme
64                  In this study, we show that beta4 integrin is highly expressed in human osteosarcoma
65 The corresponding CLCA-binding domain of the beta4 integrin is localized to the specific determining
66             Instead, the cytoplasmic tail of beta4 integrin is necessary for basal and epidermal grow
67 , suggesting that signaling through beta1 or beta4 integrins is sufficient for survival.
68    The alpha6beta4 integrin (referred to as "beta4" integrin) is a receptor for laminins that promote
69 e gene encoding the hemidesmosome-associated beta4 integrin (ITGB4), and in the gene for the hemidesm
70                   Hemidesmosomal alpha3- and beta4-integrin levels were not affected even though ther
71                         In summary, both the beta4 integrin ligand-binding and cytoplasmic domains to
72 d a mutation within the I-like domain of the beta4 integrin, lying outside the SDL region (GFP-beta4V
73 ncreased proliferation (Ki-67) and adhesion (beta4 integrin) markers, and induced inflammatory cell i
74 ense mutation in the extracellular domain of beta4 integrin may affect ligand binding or dimerization
75  We report here that alphaE-catenin inhibits beta4 integrin-mediated activation of SRC tyrosine kinas
76  alpha6 integrin knockdown cells, alpha3 and beta4 integrin mRNAs levels are unaffected.
77 of either primary human keratinocytes (NHK), beta4 integrin-null human keratinocytes (beta4-), or tho
78 ve in keratinocytes exhibiting deficiency in beta4 integrin or knockdown of the plakin protein Bullou
79 to migrate and can be induced by EGF through beta4 integrin phosphorylation.
80  and ITGB4 genes which encode the alpha6 and beta4 integrin polypeptides, respectively.
81 ITGA6 and ITGB4, which encode the alpha6 and beta4 integrin polypeptides, respectively.
82             These findings indicate that the beta4 integrin promotes prostate tumorigenesis by amplif
83 , re-expression of alpha6 integrin increases beta4 integrin protein at the cell surface, which result
84 es expressing both full-length and truncated beta4 integrin proteins.
85  stiffness and composition is sensed through beta4 integrin, Rac1, and the PI3K pathway, and suggest
86 entified a novel phosphorylation site on the beta4 integrin: S(1424).
87 ic studies revealed the balance of beta1 and beta4 integrin signaling, hemidesmosome formation, and l
88 ng that the clustering of alpha6beta4 with a beta4 integrin-specific antibody promotes p53-dependent
89 -catenin involves negative regulation of the beta4 integrin-SRC signaling pathway and that SRC-mediat
90 h Sdc1 engages the cytoplasmic domain of the beta4 integrin subunit allowing HER2-dependent motility
91           Expression of either the alpha6 or beta4 integrin subunit also restored some aspects of a l
92 provide evidence that the SDL segment of the beta4 integrin subunit is required for ligand binding an
93 s associated with the phosphorylation of the beta4 integrin subunit on serine residues.
94 n of the alpha2beta1 integrin, the alpha6 or beta4 integrin subunit was expressed in our Mm5MT model.
95  their ability to increase expression of the beta4 integrin subunit, a component of the alpha6beta4 i
96 as well as reduced labeling for plectin, the beta4 integrin subunit, and for type XVII collagen.
97  of patients with MMP and OCP recognize only beta4 integrin subunit, and sera of OP patients recogniz
98  cicatricial pemphigoid (OCP) sera recognize beta4 integrin subunit, oral pemphigoid sera recognize a
99 er adhesion-independent cross-linking of the beta4 integrin subunit.
100 he notion of close association of the alpha6 beta4 integrin subunits and further attest to the critic
101 ults in up-regulation of both the alpha6 and beta4 integrin subunits but no change in the alpha1, alp
102 rther investigate the role of the alpha6 and beta4 integrin subunits in mediating the phenotypic chan
103 ligand binding or dimerization of alpha6 and beta4 integrin subunits.
104 urface expression of the alpha2, alpha3, and beta4 integrin subunits.
105                   Here, we observed that the beta4 integrin sustains the expression of miR-200s that
106  generated point mutations within the SDL of beta4 integrin tagged with green fluorescent protein (GF
107  mammary tumors lacking functional beta1 and beta4 integrin through activation of NFkappaB, and overe
108 was also shown to colocalize with Rab27B and beta4 integrin to early adhesion initiation sites in spr
109                 This interaction enables the beta4 integrin to engage the actin cytoskeleton and prom
110 in ECS cells, where it interacts with alpha6/beta4 integrin to stimulate FAK and Src signaling, leadi
111 ction by interacting with and localizing the beta4 integrin to the rear enabling this integrin to sti
112 ative genes, 14-3-3sigma, S100P, S100A6, and beta4 integrin, to neoplastic cells in pancreatic tumors
113 M. leprae interacts with cells by binding to beta4 integrin via an LN-alpha2G bridge.
114 an keratinocytes (beta4-), or those in which beta4 integrin was reexpressed (beta4+), were tracked du
115 unction by transfection of dominant-negative beta4 integrin was sufficient to revert this highly meta
116 istant metastases express high levels of the beta4 integrin, which binds to laminin-5.
117 y inducing expression of a truncated form of beta4 integrin, which lacks binding sites for BPAG1e and
118 ing triggers the tyrosine phosphorylation of beta4 integrin, which, in turn, recruits FAK to beta4 in
119 otility depends on cytoplasmic engagement of beta4 integrin with Sdc4.

 
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