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1 eduction but different protein distributions between meals.
2  agents by lowering the set-point for hunger between meals.
3  hunger compared with BS with no differences between meals.
4 ucose appearance or in glucose disappearance between meals.
5 ly, at 2 h) were not significantly different between meals.
6 ntestinal epithelium and for taurine capture between meals.
7 6Pase-alpha) to maintain glucose homeostasis between meals.
8  concentrations did not differ significantly between meals.
9 se is critical for blood glucose homeostasis between meals.
10 ls during which animals drink; (3) intervals between meals.
11 rts recommend that iron supplements be taken between meals.
12 t to a prolonged self-imposed fasting period between meals.
13  intake driven by shorter temporal intervals between meals.
14  RS1-Reg-blocked exocytotic pathway of SGLT1 between meals.
15 thing other than water, 2) eat sugary snacks between meals, 3) consume sugary drinks between meals, 4
16 acks between meals, 3) consume sugary drinks between meals, 4) receive topical fluoride from a health
17 fferent distribution of daily protein intake between meals and snacks does not result in significant
18 her the different daily balances of proteins between meals and snacks in a low-calorie diet may influ
19        A direct association between snacking between meals and T2D risk was mediated by BMI.
20 - 8.9% after meal 2 (P = 0.01 for difference between meals), and then decreased toward baseline (P <
21 cholecystokinin concentration did not differ between meals, but the 2 low-fat meals elicited a greate
22                    This glucose is generated between meals by the hydrolysis of glucose-6-phosphate (
23 other, have bleeding gums, eat sugary snacks between meals, consume sugary drinks between meals, eat
24 e of an aqueous multivitamin (MV) supplement between meals (control group), 2) the same porridge and
25  snacks between meals, consume sugary drinks between meals, eat or drink something other than water b
26 AAs (n = 16) or placebo (n = 12) twice daily between meals for 1 week before and 2 weeks after TKA.
27 nsulin total AUC and iAUC were not different between meals (iAUC: 159.65 +/- 20.0 mU/L . 3 h for red
28 s no statistical difference (P = 0.34) in SI between meals in type 1 diabetic subjects, the diurnal p
29 derly, administration of dietary supplements between meals instead of with meals may be more effectiv
30                    However, a prolonged fast between meals is necessary for many of the benefits of c
31 und suggesting that extended periods of time between meals may not be beneficial for older adults.
32  1.46,3.01) and consumption of citrus fruits between meals (OR = 1.69, 95%CI = 1.04, 2.73) were assoc
33 1.18, 3.01) and consumption of citrus fruits between meals (OR = 2.22, 95%CI = 1.30, 3.81) were posit
34 ver, the glucose total AUC was not different between meals (P = NS).
35 hough meal glucose appearance did not differ between meals, suppression of endogenous glucose product
36                                              Between meals, the primary source of blood glucose is gl
37  natural satiation and the period of satiety between meals through coupling of ERK activation to both
38  uptake into the three depots did not differ between meals; visceral fat accounted for only approxima
39                          The only difference between meals was higher postprandial glucose following
40                                  Differences between meals were tested with the use of a repeated-mea
41 e lasting worries about maintaining fullness between meals, which may motivate opportunistic eating i