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1 ive to HFDs with a reproducible depletion of bifidobacteria.
2 s (P = 0.069) and proportions (P = 0.029) of bifidobacteria.
3  into the governance of carbon metabolism in bifidobacteria.
4 rides promote the colonization of beneficial bifidobacteria.
5 otype-age interaction were Lactobacillus and Bifidobacteria.
6 c potential by crude enzyme preparation from bifidobacteria.
7 with IBS or other FBDs, but they do increase bifidobacteria.
8 fied essential genes that appear specific to bifidobacteria.
9 sential functions not previously examined in bifidobacteria.
10 lose relationship between HMO and infant-gut bifidobacteria.
11  microorganisms for prebiotic approaches are bifidobacteria.
12 ey in order to ensure effective detection of bifidobacteria.
13 ment of potential pathogens and depletion of bifidobacteria.
14 carbohydrates by comparison with adult-borne bifidobacteria.
15 by a microbial consortium often dominated by bifidobacteria.
16  Lab4P probiotic comprising lactobacilli and bifidobacteria (50 billion cfu/day).
17 h a specific "healthy" microbiota containing bifidobacteria, a genus commonly observed in the feces o
18 han (Trp) metabolic pathways and reduces gut Bifidobacteria, a known beneficial bacterial genus that
19                                     Relative bifidobacteria abundance was higher in EG compared with
20 lted in a significant increase in numbers of bifidobacteria after a 24h fermentation compared to a ne
21 ifferences in mean +/- SD abundance of fecal bifidobacteria after consumption of whole almonds (8.7%
22    BlG16BP homologues occur predominantly in bifidobacteria and a few Firmicutes but lack in other HG
23 biotic formulation led to an increase in gut Bifidobacteria and attenuated changes in locomotor activ
24 e are warranted because the low abundance of bifidobacteria and butyrate-producing species could adve
25 hypothesis, we determined salivary levels of Bifidobacteria and caries-associated organisms for 156 o
26 l molecular tool for scientific discovery of bifidobacteria and identifies targets for further studie
27 d with reduced numbers of beneficial colonic bifidobacteria and impaired immunity.
28 testinal microbiota, including reductions in bifidobacteria and key butyrate producers.
29 robial dysbiosis with selective depletion in Bifidobacteria and Lactobacillaeae.
30                                              Bifidobacteria and lactobacilli are purportedly benefici
31 vely, are preferentially fermented by mainly bifidobacteria and lactobacilli in the human gut.
32 d glucagon like peptide-1 content as well as Bifidobacteria and Lactobacilli populations in the caecu
33 ficial barrier commensal gut bacteria (e.g., bifidobacteria and lactobacilli) and increase the abunda
34 s, in addition to their selective effects on bifidobacteria and lactobacilli, influence many aspects
35 iet rich in yacon FOS promoted the growth of bifidobacteria and lactobacilli, resulting in high level
36 ministered with prebiotics, or by endogenous bifidobacteria and lactobacilli, whose metabolic activit
37 sity gut microbiome, and dysbiotic levels of Bifidobacteria and Lactobacillus (most robust features o
38           We also found associations between bifidobacteria and prenatal maternal pro-inflammatory cy
39 cterial diversity and relative abundances of Bifidobacteria and Prevotella.
40 ulose have increased densities of intestinal bifidobacteria and reduced incidence of systemic infecti
41 sugar to enumerate total anaerobes, aerobes, bifidobacteria, and enterobacteria, and to assay for bet
42                  Here we show that a lack of bifidobacteria, and in particular depletion of genes req
43 ally pathogenic bacteria and the increase of bifidobacteria, and possible beneficial commensals, conf
44 ely correlated with Sutterella, Akkermansia, Bifidobacteria, and Roseburia abundance, and positively
45                Combinations of Lactobacilli, Bifidobacteria, and Streptococcus salivarius prevent rel
46 e storage period, while the viability of the bifidobacteria ( approximately 10(7)cfu/g) also remained
47               In the gastrointestinal tract, bifidobacteria are a key genus, but are often under-repr
48                                              Bifidobacteria are aciduric bacteria that might play a r
49 her levels of beneficial gut bacteria called Bifidobacteria are associated with the human lactase non
50            The glycan-degrading abilities of bifidobacteria are believed to reflect available carbon
51                                              Bifidobacteria are beneficial saccharolytic microbes tha
52                                              Bifidobacteria are common and frequently dominant member
53                                              Bifidobacteria are common gut commensals with purported
54  that when commercially available strains of bifidobacteria are cultured in milk, spiked with perchlo
55                                              Bifidobacteria are dominant members of the microbial com
56                                              Bifidobacteria are early colonizers of the human gut and
57                                              Bifidobacteria are exposed to substantial amounts of die
58                                              Bifidobacteria are frequently proposed to be associated
59                                              Bifidobacteria are important members of human gut microb
60                                              Bifidobacteria are important members of the human gut fl
61                                              Bifidobacteria are the dominant intestinal bacteria in b
62                                      Because bifidobacteria are thought to reduce the risk for intest
63                          Despite interest in bifidobacteria as a live biotic therapy, our understandi
64    Our findings solidify the crucial role of Bifidobacteria as primary colonizers in shaping the micr
65 e numbers of beneficial bacteria, especially bifidobacteria, at the expense of less beneficial groups
66 and subjected to quantitative PCRs to detect bifidobacteria, bacteroides, lactobacilli, Escherichia c
67                  To test the hypothesis that Bifidobacteria behave as caries-associated organisms, as
68 Glc-alpha1,3-l-Ara) significantly stimulated bifidobacteria but was suppressed with trehalose, ribose
69  diet consistently led to lower abundance of Bifidobacteria, but there were no clear effects on diver
70 culum in kojibiose medium profoundly induced bifidobacteria by 44 % and 55 % upon reinoculation into
71 mula containing GOS sustained high levels of bifidobacteria compared with formula containing B. longu
72                                              Bifidobacteria comprise a significant proportion of the
73 ined.We sought to determine the effects of a bifidobacteria-containing formula on the healthy human i
74                                              Bifidobacteria counts were lower in cases at all time po
75 genetic attenuation that may be occurring in bifidobacteria cultures, we obtained the complete genome
76                                              Bifidobacteria demonstrate an antagonistic correlation w
77 isation (FISH) shows that the proportions of bifidobacteria detected in faecal samples were in agreem
78 roidetes), Clostridium leptum, C. coccoides, bifidobacteria, Escherichia coli and Archaea in stool.
79 n of orthologous genes differed between both bifidobacteria even when grown on identical substrates.
80  samples, thereby supporting the notion that bifidobacteria expand the human glycobiome.
81 ost protection during DR is compromised when Bifidobacteria expansion is prevented, indicating that m
82                        Infant gut-associated bifidobacteria has a metabolic pathway that specifically
83 many substances that stimulate the growth of bifidobacteria in vitro and also in the small intestine
84 gosaccharides (GOSs) stimulate the growth of bifidobacteria in younger adults, but little is known ab
85                        Albeit insignificant, bifidobacteria increased with 4'-epikojibiose(Glc-alpha1
86               Consumer interest in probiotic bifidobacteria is increasing, but industry efforts to se
87 actions, and the health-promoting effects of bifidobacteria is limited.
88 f fatty acid metabolism to administration of bifidobacteria is strain-dependent, and strain-strain di
89  growth of health-promoting lactobacilli and bifidobacteria is supported by FOS, giving it the classi
90 ization of glycolipids from the cell wall of bifidobacteria is the first step in correlating glycolip
91                                              Bifidobacteria levels at 6 months of age, and relative a
92                                     Salivary Bifidobacteria levels were positively associated with th
93 involved in probiotic conditioned media from Bifidobacteria longum subsp infantis.
94 iarrhea had differences in the proportion of bifidobacteria (median: 0.4% and 3.7%; interquartile ran
95                                              Bifidobacteria metabolize lactose without gas production
96                                              Bifidobacteria metabolize lactulose, produce high concen
97                                     Specific bifidobacteria might have cytoprotective activities, but
98 n early gut microbiota including E. coli and bifidobacteria might promote this maturation.
99                           Salivary levels of Bifidobacteria, mutans streptococci, lactobacilli, and y
100 concentrations were linked to changes in the bifidobacteria number.
101 indeed show selective growth of infant-borne bifidobacteria on milk oligosaccharides or core componen
102 raction namely OS4 was utilized by probiotic bifidobacteria only.
103                  We have designed modified, 'bifidobacteria-optimised' universal primers, which we ha
104        Infant formulas are supplemented with bifidobacteria or complex oligosaccharides, notably gala
105 05), Streptococcus sobrinus (p < 0.005), and Bifidobacteria (p < 0.0001) were associated with S-ECC,
106 sed fecal pH (P < 0.001) and increased fecal bifidobacteria (P < 0.001) and fecal lactate (P < 0.001)
107  significantly associated with only salivary Bifidobacteria (p < 0.001) and yeast (p < 0.001) levels
108 gsiae (p = 0.003), Streptococcus mutans with bifidobacteria (p < 0.001), and S. mutans with S. wiggsi
109 wing that there is a significant decrease of Bifidobacteria (p .047) and Lactobacillus (p .038) in ob
110 nclusions from this study further reveal how bifidobacteria participate in establishing functional ne
111 tervention), or to a control formula without bifidobacteria (placebo).
112                                              Bifidobacteria represent a dominant constituent of human
113                                              Bifidobacteria represent one of the dominant groups of m
114                                              Bifidobacteria resident in the gastrointestinal tract (G
115       Our studies suggest that lactulose and bifidobacteria serve as a synbiotic to reduce rates of i
116                                              Bifidobacteria should be regarded as caries-associated o
117 ver, clinical feeding studies with exogenous bifidobacteria show they don't remain in the intestine,
118                              Finally, use of bifidobacteria slightly increased CLA relative content i
119  abundances found known associations such as Bifidobacteria species, as well as novel associations.
120                                 For example, Bifidobacteria, staphylococci, and streptococci signific
121 x viable microorganisms of Lactobacillus and Bifidobacteria strains are safe to be consumed at four w
122 ganisms of 30 x 10(10) cfu Lactobacillus and Bifidobacteria strains for six months on clinical outcom
123 ented by 11 lactic acid bacteria (LAB) and 9 bifidobacteria strains.
124                In contrast, adult-associated bifidobacteria such as the closely related B. longum sub
125                                              Bifidobacteria-supplemented animals had a significant re
126                           Infants exposed to bifidobacteria-supplemented formula showed decreased occ
127 gastrointestinal tract is often dominated by bifidobacteria that flourish on milk glycans.
128 cantly less typical infant bacteria, such as Bifidobacteria, that have potential health benefits.
129 et caused an increase in total anaerobes and bifidobacteria, the highest densities occurred during su
130 This has encouraged scientific research into bifidobacteria, though recalcitrance to genetic manipula
131 thesis of saccharidic resource sharing among bifidobacteria through species-specific metabolic specia
132 often facilitated by mobile elements, allows bifidobacteria to adapt to fermentation environments in
133  variables (P > 0.05).The supplementation of bifidobacteria to infant diet can modulate the occurrenc
134 e, at least partially, due to the ability of bifidobacteria to metabolize Human Milk Oligosaccharides
135 a and transcend the lack of genetic tools in bifidobacteria to study the basis for their health-promo
136 ed light on key molecular mechanisms used by bifidobacteria to use high-mannose N-glycans, a perennia
137  the influence of inulin on iron absorption, bifidobacteria, total bacteria, short-chain fatty acids
138  phospholipase A(2) expression were lower in bifidobacteria-treated pups than in controls, supporting
139  the relative abundances of Lactobacilli and Bifidobacteria, two taxa predominant in breastfed human
140                                  Infant-type bifidobacteria utilize these soluble carbohydrate oligom
141       To understand the relationship between bifidobacteria utilizing HMOs and how the metabolites th
142 c combination (oral lactic acid bacteria and bifidobacteria + vaginal lactobacilli), for 4 months.
143 probiotic (G2, oral lactic acid bacteria and bifidobacteria + vaginal placebo), vaginal probiotic (G3
144  multistrain preparation of lactobacilli and bifidobacteria was effective in prevention of AAD or CDD
145                    The relative abundance of bifidobacteria was higher, while environmentally transmi
146 t increase on the growth of lactobacilli and bifidobacteria was observed after exposition to the bark
147 nd facilitate functional genomic analyses in bifidobacteria, we created a large Tn5 transposon mutant
148 early colonization with Escherichia coli and bifidobacteria were associated with higher numbers of CD
149                                        While bifidobacteria were the dominant genus in the infant gut
150 fants from these countries were dominated by bifidobacteria, were different from each other, and were
151 onal CLNA isomers, never reported before for bifidobacteria, were observed.
152  colonic microbiome appear to be specific to Bifidobacteria with no consistent impacts on other micro
153  multistrain preparation of lactobacilli and bifidobacteria, with a total of 6 x 10(10) organisms, on
154 ntly, DR promotes the expansion of commensal Bifidobacteria within the large intestine, which produce
155   Prebiotics increased absolute abundance of bifidobacteria (WMD: 1.16 log10 copies of the 16S riboso

 
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