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1 brosis, and ultrastructural abnormalities of bile canaliculi.
2 e increased secretion of fluorescent PC into bile canaliculi.
3  the mutant protein to reach the membrane of bile canaliculi.
4 ericellular fibrosis, and dilated, misshapen bile canaliculi.
5 croscopy also revealed structures resembling bile canaliculi.
6  the lumina, reminiscent of those seen about bile canaliculi.
7 e apical membrane of the hepatocyte into the bile canaliculi.
8 ence of damage to transgenic hepatocytes and bile canaliculi.
9 d excrete fluorescein diacetate into visible bile canaliculi.
10 hepaticus was noted infrequently and only in bile canaliculi.
11 es and translocates phosphatidylcholine into bile canaliculi.
12 rferes with the bilirubin excretion into the bile canaliculi.
13  transports bile salts from hepatocytes into bile canaliculi.
14 cantly higher accumulation of CDF in cells + bile canaliculi (32.3 +/- 2.5 versus 24.4 +/- 4.3 pmol/m
15 hese cells as shown by impaired formation of bile canaliculi, absence of polarity, and reduced secret
16 a organize their apical membrane domain into bile canaliculi and ducts, which are not accessible to c
17 onally, these mutants develop defects in the bile canaliculi and have marked biliary paucity, suggest
18 advection (flow) and diffusion in individual bile canaliculi and in interlobular bile ducts of intact
19 rve-shaped bacteria predominantly located in bile canaliculi and occasionally in bile ducts.
20 ts provided a trans-epithelial shunt between bile canaliculi and sinusoids by which bile constituents
21  generate lumina at cell-cell contact sites (bile canaliculi) and orient MTs horizontally.
22 ) cultures had epithelial morphology, showed bile canaliculi, and expressed albumin messenger RNA (mR
23 he liver, secreted from hepatocytes into the bile canaliculi, and subsequently stored in the gall bla
24                  In WIF-B9 cells, which lack bile canaliculi, apical ABC transporters colocalized wit
25        Electron microscopy showed developing bile canaliculi at 1 day.
26  anisotropically to generate a 3D network of bile canaliculi (BC).
27  anisotropically, generating a 3D network of bile canaliculi (BC).
28 ntrast, organize their luminal surfaces (the bile canaliculi; BC) between their lateral membranes, an
29 ecreted as biliary glycoprotein 1 (BGP1) via bile canaliculi (BCs).
30 were normally processed and expressed at the bile canaliculi, but their stability was reduced.
31 ttle or no effect on ABCB4 expression at the bile canaliculi caused a decrease (F357L, T775M, and G95
32 wing to tissue localization in the placenta, bile canaliculi, colon, small bowel, and brain microvess
33 re rapidly in CP-treated animals, and hybrid bile canaliculi developed even 1 day after cell transpla
34  localization of polarity proteins, impaired bile canaliculi formation, and loss of microvilli.
35 epresent a suitable in vitro system to study bile canaliculi formation, bile drainage and how differe
36 ane regenesis, with hybrid gap junctions and bile canaliculi forming over 3 to 7 days after cell tran
37  was manifested by sequential progression of bile canaliculi from small structures to a fully branche
38 ron microscopy revealed dilated and tortuous bile canaliculi in KO livers along with decreased canali
39  whereas it rapidly appeared in the abnormal bile canaliculi in LKB1(-/-) mice, confirming that junct
40 ile flow, which is normally expressed in the bile canaliculi in the liver.
41  functional, consistently draining bile from bile canaliculi into the bile duct.
42 oholic fatty liver disease, such as decay of bile canaliculi network and ductular reactions.
43                              We compared the bile canaliculi network in the organoids incubated with
44 cible hepatocyte organoids with a functional bile canaliculi network that retain morphological featur
45 pheroids spontaneously assemble a functional bile canaliculi network, extending from the surface to t
46                            In the liver, the bile canaliculi of hepatocytes are connected to intrahep
47                                          The bile canaliculi of hepatocytes contract spontaneously, a
48 cobacter hepaticus, infects the intrahepatic bile canaliculi of mice, causing a severe chronic hepati
49  the distribution of the ecto-ATPDase in the bile canaliculi of the chicken liver.
50 tion of filamentous actin in hepatocytes and bile canaliculi of the ischemic lobes compared with the
51 r that extrudes phosphatidylcholine into the bile canaliculi of the liver.
52 , present focally, was manifested as dilated bile canaliculi, partial loss of microvilli, and retenti
53 e ultrastructural characteristics, including bile canaliculi, peroxisomes, and glycogen granules, whe
54 hanced cell differentiation and formation of bile canaliculi, probably through an effect on hepatocyt
55         The organoids organized a functional bile canaliculi system, which was disrupted by cholestas
56        The hepatocyte's apical surface forms bile canaliculi that transport bile to the hepatic ducts
57 hepatic organoids; development of functional bile canaliculi was imaged live.
58 ct, i.e., increase in the mature form at the bile canaliculi, was obtained by cell treatments with cy
59 ng revealed impaired bile secretion into the bile canaliculi, which was secondary to loss of canalicu
60 s possess structural polarity and functional bile canaliculi with normal differentiated function.