ライフサイエンスコーパス: bile duct injury

1 ene attenuated BEC proliferation after acute bile duct injury.

2 The primary outcome was bile duct injury.

3 -17A production and ameliorated intrahepatic bile duct injury.

4 e phenotypes, and proliferate in response to bile duct injury.

5 tivity, potentially allowing for exaggerated bile duct injury.

6 have been linked to pathogenic mechanisms of bile duct injury.

7 on in the portal region, without evidence of bile duct injury.

8 gen(s) responsible for T-cell activation and bile duct injury.

9 ced, subsequent autoreactive T cell-mediated bile duct injury.

10 ntigen-specific animal model of inflammatory bile duct injury.

11 e might be able to decrease the incidence of bile duct injury.

12 the normal biliary proliferative response to bile duct injury.

13 ,411 for all care related to repair of their bile duct injury.

14 ures with a recognized morbidity relating to bile duct injuries.

15 of laparoscopic cholecystectomy (LC)-related bile duct injuries.

16 tectomy was associated with a higher rate of bile duct injury (0.4% vs 0.2%; RR, 1.88 [95% CI, 1.14-2

17 my was associated with a lower risk of major bile duct injury [0.28% vs 0.53%, relative risk (RR)=0.5

18 orrhage, 0.3%; subhepatic collections, 2.9%; bile duct injury, 0.08%; and retained stones, 3.1%); the

19 gnant biliary obstruction (1.8%), history of bile duct injury (2.2%), or complications of liver trans

20 The authors analyzed 252 laparoscopic bile duct injuries according to the principles of the co

21 s treated at a single institution with major bile duct injuries after laparoscopic cholecystectomy ma

22 of laparoscopic techniques, the frequency of bile duct injury after operation has increased.

23 Benign biliary stricture occurs secondary to bile duct injury, anastomotic narrowing, or chronic infl

24 Major bile duct injuries and postoperative bile duct stricture

25 PTC and PTBD performed for management of bile duct injury and complications of liver transplantat

26 ency of serious rare complications including bile duct injury and death.

27 hat autoimmunity plays a role in the ongoing bile duct injury and progression of disease.

28 ell Polarity signalling components following bile duct injury and promote the formation of ductular s

29 Outcomes of bile duct injuries are best with surgical management and

30 helialitis disproportionate to the degree of bile duct injury are features that appear unique to pedi

31 Bile duct injury as a background histologic finding shou

32 rimental BA, CD25+ cell depletion aggravated bile duct injury at 12 dpi after RRV inoculation, as pla

33 Iatrogenic bile duct injuries (BDI) are a devastating complication.

34 A majority of bile duct injuries (BDI) sustained during laparoscopic c

35 has led to a rise in the incidence of major bile duct injuries (BDI).

36 y of bile biochemistry for the assessment of bile duct injury (BDI).

37 holecystectomy, operative complications (eg, bile duct injury, bleeding), and mortality.

38 On multivariable analysis, bile duct injury, bowel perforation, and high clinical s

39 his analysis were to compare the outcomes of bile duct injuries by specialist over time and the role

40 Major bile duct injuries can be managed successfully by combin

41 Almost all bile leaks and many bile duct injuries can be managed successfully by endosc

42 Repair of cholecystectomy-related bile duct injuries can run 4.5 to 26.0 times the cost of

43 February 1, 2000, and November 23, 2011 for bile duct injury, cholangiocarcinoma, choledochal cysts,

44 With the exception of bile duct injuries discovered and repaired during surger

45 e incidence and mechanism of injury of major bile duct injuries during laparoscopic cholecystectomy h

46 iologic management of 89 patients with major bile duct injuries during laparoscopic cholecystectomy.

47 There seems to be an increase in the rate of bile duct injuries during SILC when compared with histor

48 Bile duct injury during cholecystectomy is a serious com

49 imary endpoint was the occurrence of a major bile duct injury during laparoscopic cholecystectomy (bi

50 lso be critical in the early phases of small bile duct injury found in primary biliary cirrhosis.

51 valuating QOL after surgical repair of major bile duct injuries from LC.

52 after successful surgical repair of a major bile duct injury from a LC treated at the Johns Hopkins

53 Biliary complications consisted of five bile duct injuries (group A = 4, group B = 1) and two cu

54 The incidence of bile duct injuries has increased dramatically since the

55 Bile duct injuries have been reported in several series

56 ly indicated in the management of iatrogenic bile duct injuries (IBDI), but occasionally, it becomes

57 Male surgeons had more bile duct injuries in elective surgery (OR, 1.69; 95% CI

58 Thirty percent of bile duct injuries in group A and 32.9% of all injuries

59 on of CVS contributes to the stable rates of bile duct injuries in LC.

60 helialitis disproportionate to the degree of bile duct injury in all 7 patients; periportal/perivenul

61 ological, and molecular markers of liver and bile duct injury in Mdr2-/- mice and also had direct pro

62 in the development of cholestatic liver and bile duct injury in mouse models of sclerosing cholangit

63 ategies to block progression of intrahepatic bile duct injury in patients with BA.

64 ed in vivo in multiple models of hepatic and bile duct injury, including bile duct ligation and CCl(4

65 ry epithelium associated with p53 mutations, bile duct injury, inflammation, and fibrosis.

66 murine biliary atresia, and the progressive bile duct injury is due in part to a bile duct epithelia

67 pathogenesis of biliary atresia (BA) is that bile duct injury is initiated by a virus infection, foll

68 RF-induced bile duct injury may be decreased significantly with an

69 ost common associated complications included bile duct injury (n = 397), bowel perforation (n = 96),

70 tectomy was associated with a higher rate of bile duct injury necessitating a definitive operative re

71 intermittent toxin exposure, which provokes bile duct injury/necrosis and proliferation, fibroblast

72 It has been suggested that the majority of bile duct injuries occur as a result of operator disorie

73 Hepatocellular, but not bile duct, injury occurs during experimental pancreatiti

74 ted at the Johns Hopkins Hospital with major bile duct injuries or postoperative bile duct strictures

75 nfidence interval [CI]: 0.31-0.90], of major bile duct injury or death (1.36% vs 1.88%, RR=0.72, 95%

76 No bile duct injury or death occurred.

77 Secondary outcomes included major bile duct injury or death, 30-day postcholecystectomy mo

78 Overall QOL scores for bile duct injury patients in the three domains (physical

79 Thirty-one percent of responding bile duct injury patients reported having sought legal r

80 as evaluated from a psychological dimension, bile duct injury patients reported QOL scores in the phy

81 Thus, p53 deficiency, chronic bile duct injury/proliferation, and the fibrotic matrix

82 lecystectomy appears to have a higher common bile duct injury rate and a lower mortality rate.

83 juries were identified for a SILC-associated bile duct injury rate of 0.72%.

84 Except for a higher common bile duct injury rate, laparoscopic cholecystectomy appe

85 pic cholecystectomy (n = 35 037) had similar bile duct injury rates (0.37% [128 of 35 037] vs 0.39% [

86 ction may not be the cause of differences in bile duct injury rates among patients undergoing robotic

87 Recent evidence suggests higher bile duct injury rates for patients undergoing robotic-a

88 this cohort study of Medicare beneficiaries, bile duct injury rates were higher among low-, medium-,

89 and laparoscopic cholecystectomy had similar bile duct injury rates, but robotic-assisted cholecystec

90 s sufficient data to determine mortality and bile duct injury rates.

91 postoperative bile duct strictures and major bile duct injuries remains a challenge for even the most

92 Postoperative bile duct injuries require multidisciplinary management.

93 Sixty-four percent of all major bile duct injuries required biliary reconstruction, and

94 The primary outcome was rate of bile duct injury requiring definitive surgical reconstru

95 Secondary outcomes were composite outcome of bile duct injury requiring less-invasive postoperative s

96 The primary outcome of interest was bile duct injury requiring operative intervention after

97 The primary outcome was major bile duct injury requiring operative repair within 6 mon

98 ey bias underestimates the true frequency of bile duct injuries, residency training decreases the lik

99 Bile duct injury seems to be a multistep process.

100 ata show that errors leading to laparoscopic bile duct injuries stem principally from misperception,

101 e reported in 278 (1.3%) patients and severe bile duct injuries (Strasberg grades B-E) were reported

102 o determine the optimal timing for repair of bile duct injuries sustained during cholecystectomy.

103 Patients with bile duct injuries that were recognized immediately at t

104 The bile duct injury that occurs in patients with BA might r

105 sults concerning complications, particularly bile duct injury, to those reported in open cholecystect

106 having bile leaks (type A, n = 239, 45%) or bile duct injuries (types B-E, n = 289, 55%).

107 spective analysis of 200 patients with major bile duct injuries was completed.

108 Common bile duct injury was defined by a second surgical proced

109 Common bile duct injury was found in 2380 (0.39%) of 613 706 pa

110 Bile duct injury was higher among patients undergoing ro

111 Bile duct injury was induced by the administration of 3,

112 Patients with LC-related bile duct injuries were billed a mean of $51,411 for all

113 ent for laparoscopic cholecystectomy-related bile duct injuries were divided into 8 categories.

114 Nineteen bile duct injuries were identified for a SILC-associated

115 Patients with bile duct injuries were managed most often by endoscopis

116 Bile duct injuries were more likely to be discovered dur

117 Bile duct injuries were recognized intraoperatively only

118 cholecystectomy (bile leaks without a major bile duct injury were not tabulated).

119 sis in a cohort of patients with Strasberg E bile duct injuries who underwent HJ after a minimum foll

120 y's finding of significantly higher rates of bile duct injury with robotic-assisted cholecystectomy c