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1 ed gastric emptying, pancreatic fistula, and biliary strictures).
2 ts after bilioenteric anastomoses for benign biliary stricture.
3 curacy than a standard ERCP in indeterminate biliary stricture.
4 oscopic methods to pass a guidewire across a biliary stricture.
5 e incidence of postoperative cholangitis and biliary stricture.
6                  Nine patients had developed biliary strictures.
7 lammatory response of NP creates challenging biliary strictures.
8  stent placement for nonresectable malignant biliary strictures.
9 ted with an increased risk of nonanastomotic biliary strictures.
10 onfunction, early allograft dysfunction, and biliary strictures.
11 ining acceptance for the treatment of benign biliary strictures.
12  included 19 (3.3%) bile leaks and 27 (4.6%) biliary strictures.
13 n the diagnostic algorithm for indeterminate biliary strictures.
14 described in the evaluation of indeterminate biliary strictures.
15 anastomoses in the evaluation and therapy of biliary strictures.
16          There have been no reoperations for biliary strictures.
17 s were performed during 112 treatments of 84 biliary strictures.
18 BD) was performed in 85 patients with benign biliary strictures.
19 olangiopancreatography for the evaluation of biliary strictures.
20 se of metallic endobiliary stents for benign biliary strictures.
21  the differentiation of benign and malignant biliary strictures.
22 initial modality for treating posttransplant biliary strictures.
23 ients had 14 (23.7%) bile leaks and 4 (6.8%) biliary strictures.
24 dren had three bile leaks (6%) and six (12%) biliary strictures.
25                       Patients with complete biliary stricture (10/12) required hepaticojejunostomy.
26                                   Of the 125 biliary strictures, 86 (68.8%) could be passed via endos
27 12 men, aged 51+/-11 years) with anastomotic biliary stricture after LDLT.
28  was performed to determine the incidence of biliary stricture after PD.
29 le and safe for the treatment of anastomotic biliary strictures after LDLT.
30 f a protocol of initial balloon dilation for biliary strictures after liver transplantation.
31 prerequisite for the endoscopic treatment of biliary strictures after living donor liver transplantat
32  There was no difference in the incidence of biliary strictures after resection for benign (n = 10, 2
33 e protection against radiological changes of biliary stricture, although possibly to a lesser degree
34 a lower incidence of primary nonfunction and biliary strictures, although this difference did not rea
35                   Among patients with benign biliary strictures and a bile duct diameter 6 mm or more
36          Revision of the HA may help prevent biliary strictures and allow for good long-term graft fu
37 tic endotherapy by ERCP for the treatment of biliary strictures and chronic pain in chronic pancreati
38 also assessed the incidence of ischemic type biliary strictures and hepatic artery thrombosis, and ev
39              The incidence of nonanastomotic biliary strictures and hepatic histologic findings sugge
40 ansplant (LT) with duct-to-duct anastomosis, biliary strictures and leaks are typically managed with
41          Many patients with IAC present with biliary strictures and obstructive jaundice, making chol
42  its use in the evaluation and management of biliary strictures and stones.
43 al or radiographic evidence of postoperative biliary stricture, and all patients underwent successful
44 ary complications: 21 had bile leaks, 22 had biliary strictures, and 4 had both complications.
45 rmance of selective guidewire passage across biliary strictures, and diagnosis and treatment of bilia
46 of Mdm2 in cholangiocytes causes senescence, biliary strictures, and fibrosis.
47 urvival, method of HA revision, incidence of biliary strictures, and long-term HA patency.
48 ences in IC among centers, the importance of biliary strictures as a risk factor for graft failure, a
49 percent of the procedures were performed for biliary stricture assessment, and using visual signs or
50       IAC should be suspected in unexplained biliary strictures associated with increased serum IgG4
51 nically significant restenosis after PBBD of biliary strictures at anastomotic and nonanastomotic sit
52 in pancreatic duct, with associated multiple biliary strictures at ERCP.
53                        It mimics both benign biliary strictures (BBS) and malignant biliary stricture
54                                       Benign biliary strictures (BBS) are complications of chronic pa
55                                       Benign biliary strictures (BBS) respond to placement of multipl
56 al vein thrombosis, primary nonfunction, and biliary stricture between the two groups.
57                                    Regarding biliary stricture, bile leakage was the only significant
58                                              Biliary strictures, both anastomotic and nonanastomotic,
59                Diagnosing and characterizing biliary strictures (BS) remains challenging.
60 ed diagnostic features including presence of biliary stricture by using a five-point confidence scale
61  define the incidence and natural history of biliary stricture caused by NP.
62 mary outcome was development of intrahepatic biliary strictures consistent with IC.
63                                              Biliary stricture developed in 108 (16%) patients.
64                 Median time from NP onset to biliary stricture diagnosis was 4.2 months (IQR, 1.8-10.
65 ertion is the preferred treatment for benign biliary stricture due to chronic pancreatitis.
66 biliary-enteric anastomoses in 79 (20%), and biliary strictures following liver transplantation in 88
67 fusion led to a lower risk of nonanastomotic biliary strictures following the transplantation of live
68 institution review examined the incidence of biliary stricture formation after pancreaticoduodenectom
69             The pathogenesis of intrahepatic biliary stricture formation in patients with primary scl
70                                              Biliary stricture formation is an infrequent complicatio
71 e only significant univariate predictors for biliary stricture formation were preoperative and postop
72 reoperative jaundice did not protect against biliary stricture formation.
73 en the year of transplant and nonanastomotic biliary strictures frequency, primary graft dysfunction
74                                Indeterminate biliary strictures frequently present as a diagnostic co
75 in situ hybridization in brushing samples of biliary strictures had a sensitivity of 50%, 57%, and 86
76 ndardized classification of pCLE findings of biliary strictures has been proposed.
77                                              Biliary strictures have a negative effect on HCV fibrosi
78  years was associated with increased rate of biliary strictures (hazard ratio [HR], 1.67; 95% confide
79                                              Biliary strictures (HR = 2.25, P = 0.0006), creatinine a
80 ] = 1.38 for doubling of AST, P = 0.005) and biliary strictures (HR = 2.68, P = 0.0001) were associat
81 low less than 400 mL/min was associated with biliary strictures (HR, 1.53; 95% CI, 1.04-2.24; P = 0.0
82                                 Intrahepatic biliary strictures (IHBS) without hepatic artery thrombo
83                               All cases with biliary stricture in DCD group finally led to graft loss
84                                              Biliary stricture in necrotizing pancreatitis (NP) has n
85                         The development of a biliary stricture in patients who have undergone PD for
86 most frequent indications were postoperative biliary strictures in 123 (31%), stenosed biliary-enteri
87 n with advances, such as stent placement for biliary strictures in a patient after liver transplantat
88 hould exert a therapeutic role in preventing biliary strictures in liver allografts.
89  it does not seem to prevent non-anastomotic biliary strictures in livers donated after circulatory d
90  incidence and later onset of nonanastomotic biliary strictures in patients with PSC compared with a
91                            The evaluation of biliary strictures in patients with PSC is especially ch
92 crease the intraoperative HA flow to prevent biliary strictures in such patients.
93 L/min were associated with increased rate of biliary strictures in younger donors (<50 years old), an
94 00 mL/min was associated with higher rate of biliary strictures in younger donors with duct-to-duct r
95  of stay at LDLT, hepatic artery thrombosis, biliary stricture, infection, and disease recurrence wer
96  is the first-line treatment for most benign biliary strictures; it is possible that fully covered, s
97 rtal vein stenosis/thrombosis, ischemic-type biliary stricture (ITBS), and primary nonfunction were s
98 enign biliary strictures (BBS) and malignant biliary strictures (MBS).
99            Early identification of malignant biliary strictures (MBSs) is challenging, with up to 20%
100 luding chronic pancreatitis (n = 10), benign biliary stricture (n = 1), pancreatic complex cyst (n =
101 AC (n = 1) and a benign chronic inflammatory biliary stricture (n = 1), respectively.
102  detection of bile duct dilatation (n = 27), biliary strictures (n = 10), and intraductal abnormaliti
103 olving patients with treatment-naive, benign biliary strictures (N = 112) due to orthotopic liver tra
104 al study in patients with symptomatic benign biliary strictures (N = 187) due to various etiologies r
105  in bile was associated with non-anastomotic biliary strictures (NAS) and acute cellular rejection (A
106                               Nonanastomotic biliary strictures (NAS) are a major cause of morbidity
107 lantation time on the risk of nonanastomotic biliary strictures (NAS) occurring within 1 year and of
108 ted with an increased risk of nonanastomotic biliary strictures (NAS).
109 emains the high incidence of non-anastomotic biliary strictures (NAS).
110     Portal vein stricture, bile leakage, and biliary stricture occurred in 0.6% (n=4), 3.3% (n=21), a
111                               Nonanastomotic biliary strictures occurred in 11% of transplants, predo
112                                              Biliary strictures occurred in 16.4% SCS (n=12) and 6.3%
113                               Nonanastomotic biliary strictures occurred in 6% of the patients in the
114                               Nonanastomotic biliary strictures occurred only in SCS (n=4).
115                                              Biliary stricture occurs frequently after necrotizing pa
116              SUMMARY/BACKGROUND DATA: Benign biliary stricture occurs secondary to bile duct injury,
117 transplant, 162 (21.8%) patients developed a biliary stricture, of which 88 (11.8%) exhibited intrahe
118 ng was favored with pretest probabilities of biliary stricture or malignancy 0%-73% for patients aged
119 t seen in patients with either postoperative biliary strictures or recurrence of PSC.
120 of hepatic artery thrombosis, nonanastomotic biliary strictures, or 4-month allograft survival.
121 d 25 consecutive patients with indeterminate biliary stricture over 3 years.
122 prolonged transplantation time (P=0.02), and biliary stricturing (P=0.016) were identified as signifi
123 tion of varying etiologies (including benign biliary stricture, papillary stenosis, choledocholithias
124 Endoscopic treatment of post-cholecystectomy biliary strictures (PCBS) with multiple plastic biliary
125         Endpoints included the assessment of biliary strictures, performance of selective guidewire p
126 osis of cholangiocarcinoma, and treatment of biliary strictures poses a similarly significant clinica
127 c pancreatitis and pancreatic cancer-induced biliary strictures, postoperative bile leaks, the use of
128 percentile (P=0.035), reoperation (P=0.005), biliary stricturing postoperatively (P=0.046), and gastr
129            No differences in non-anastomotic biliary strictures, primary nonfunction and hepatic arte
130 rs in 11 countries, 187 patients with benign biliary strictures received FCSEMS.
131                       No patient developed a biliary stricture, required re-exploration for intra-abd
132 w up of 542 days, 4 (18%) patients developed biliary strictures requiring re-transplantation.
133                            Steroid-sensitive biliary strictures resembling primary sclerosing cholang
134                                              Biliary stricture risk factors and outcomes were evaluat
135 idered essential in diagnosing indeterminate biliary strictures, since the procedure is associated wi
136 mptomatic and are still alive, 1 developed a biliary stricture that was stented and is alive 105 mont
137 clinical dilemma because they harbor chronic biliary strictures that are difficult to distinguish fro
138 ch in the endoscopic management of malignant biliary strictures, the broadening therapeutic indicatio
139                The baseline characteristics, biliary stricture treatment, and stent indwelling time w
140 rimary graft dysfunction, and nonanastomotic biliary strictures (univariate logistic regression).
141 cted against the formation of nonanastomotic biliary strictures versus CC/CT patients (12.6%; P = 0.0
142                                              Biliary stricture was identified on cholangiography as n
143                       Operative treatment of biliary stricture was more likely in patients with infec
144                          The overall rate of biliary strictures was greater in the DCD group at 1 yea
145         One-year incidence of nonanastomotic biliary strictures was lower after DHOPE-COR-NMP (3%, P
146 tive number of treatments for nonanastomotic biliary strictures was lower by a factor of almost 4 aft
147 wed, and factors related to bile leakage and biliary stricture were analyzed using the multivariable
148                 Incidences of bile leaks and biliary strictures were 11.4% and 20.6%, respectively.
149                      By univariate analysis, biliary strictures were associated with preoperative per
150                             At presentation, biliary strictures were confined to the intrapancreatic
151 nown causes of posttransplant nonanastomotic biliary strictures were excluded leaving 120 patients fo
152 ospital stays, rejection, and nonanastomotic biliary strictures were highest in group A with lowest s
153 ospital stays, rejection, and nonanastomotic biliary strictures were highest in group A with lowest s
154       Complete records from 96 patients with biliary strictures were retrospectively reviewed.
155                Postoperatively, incidence of biliary strictures were significantly greater in high-gr
156 hybridization (FISH) can be used to evaluate biliary strictures with greater accuracy than convention
157         Distinguishing benign from malignant biliary strictures with routine biliary cytology in this
158 ic cholestasis, but many develop progressive biliary strictures with time, leading to recurrent chola
159 nd point was the incidence of nonanastomotic biliary strictures within 6 months after transplantation

 
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