ライフサイエンスコーパス: bind

1 ns in the PDE3A active site abolish compound binding.

2 ely small conformational changes upon target binding.

3 mediate the efficiency of reovirus host cell binding.

4 al understanding of membrane-mediated ligand binding.

5 sition states and favors the enthalpy-driven binding.

6 hus explaining why H2 is necessary for tight binding.

7 insertions that are not correlated with H-NS binding.

8 showed weakening of actin-mutant tropomyosin binding.

9 e to MEK inhibition by enhancing interfacial binding.

10 ngs, along with histological analyses of IAV binding.

11 in BBSome conformation induced by ARL6(GTP) binding.

12 he inclusion of an auxiliary site capable of binding a Lewis acid (LA(II)); we used this unique featu

13 TTR binds Abeta, alters its aggregation, and inhibits its to

14 (7)S-) were found to be critical to the PI5P-binding ability.

15 ize immature Zika virus via an antibody that binds across the E and prM proteins, resulting in a subn

16 localizes to the sarcomere and can directly bind actin.

17 and anti-obesity drugs by inhibiting its DNA-binding activities.

18 ding region for EphA2, we compared the EphA2 binding activity of EBV gH/gL and the EBV gH/gL-N(69)L/S

19 We demonstrate a strategy to improve the binding affinity of aptamers by modifying their sequence

20 of A9 unspecific binding and to increase the binding affinity to the receptor.

21 tween disorder in H2 and its contribution to binding affinity, and that sequence variations in H2 tha

22 the wild type allele by measuring its ligand binding affinity, CCL2 scavenging efficiency, and cell a

23 cterization, we present simple peptides that bind alpha-cobratoxin (alpha-Cbtx) and prevent its inhib

24 lternative to the native fold for RNase P to bind and mature SRP RNA co-transcriptionally.

25 The 2'-methylation uniquely enhanced binding and agonist potency at alpha7 receptors.

26 molecular determinants critical for agonist binding and biased signaling through PAR4.

27 ninflammatory because of reduced FcepsilonRI binding and enhanced CD23-dependent serum clearance.

28 couple the energy of nucleoside triphosphate binding and hydrolysis to mechanical movement along a po

29 the tendency of UPF1 to release RNA upon ATP binding and hydrolysis.

30 olutionarily more recent site diminishes p38 binding and made the phosphoswitch differently sensitive

31 to minimize the percentage of A9 unspecific binding and to increase the binding affinity to the rece

32 creased histone acetylation, increased c-Jun binding, and upregulation of nearby genes implicated in

33 cids) affording high-affinity lanthanide ion binding, and X-ray fluorescence microscopy (XFM).

34 The C-terminal BON domain binds anionic phospholipids through an extensive membran

35 l uses bursts at theta frequency to flexibly bind appropriate task modules by synchrony.

36 ct, changes of NH protection during antibody binding are measured.

37 rve separable effects of Na(+) and succinate binding at several positions suggesting distinct effects

38 results suggest that interfering with ZNF274 binding at the maternal SNORD116 locus is a potential th

39 duces two-fragment holoreceptors that remain bound at the cell surface.

40 ether the succinyl CoA (SCoA) cosubstrate is bound at the MmOGOR active site.

41 demonstrate that both substrates have to be bound before transpeptidation occurs.

42 ot change the level of disorder show similar binding behavior.

43 ABM300 was characterized in vitro (receptor binding, beta-arrestin2 recruitment, ERK1/2 phosphorylat

44 esigned two cyclic peptide mimics of the TAR-binding beta2-beta3 loop sequences present in two high-a

45 IP) and biochemical analyses revealed direct binding between endogenous TruB1 and the stem-loop struc

46 new sequencing chemistry removes limitations bound by sequence-by-ligation chemistry of ISS.

47 The resultant radiance bound by the T(4) law limits the ability to regulate rad

48 On the other hand, CpGs at sites not bound by transcription factors during the global re-meth

49 ved through a hydrophobic trimethyl-L-lysine-binding 'cage' formed by BAHCC1(BAH), mediating colocali

50 Moreover, other Sirtuins share some DSB-binding capacity and DDR activation.

51 lular levels of anticancer drugs through ATP-binding cassette (ABC) pumps.

52 es, peroxisomal acyl-activating enzymes, ATP binding cassette (ABC) transporters, and central carbon

53 gest how changes in the association of lipid-binding caveolar proteins upon flattening of caveolae co

54 My model assumes that an Hsp90-bound client can transition between a deactivating confo

55 An isolated, bound cofilin compromises longitudinal filament contacts

56 Fittings of NMR and ITC binding curves to the Hill model yielded n(Hill) ~2.9, n

57 ns, we demonstrate that cbEGF domain calcium binding decreases under mechanical stress (i.e. cbEGF do

58 w helical propensity and establishes that H2 binds directly to the coiled-coil 1B (CC1B) domain of p1

59 is not mutually exclusive, and the proteins bind distinct regions of SidI.

60 an extended loop in the C-terminal receptor-binding domain (HC) of BoNT/B (HC/B) has been proposed t

61 The receptor-binding domain (RBD) is immunodominant and the target of

62 gM, and IgA antibodies to the spike receptor binding domain (RBD), S1+S2, nucleocapsid, and ORF6 to O

63 re, we report a crystal structure of the DNA-binding domain of a model ASO-binding protein PC4, in co

64 f the ankyrin repeat motifs in the substrate binding domain of cpSRP43 drives its activation.

65 n July, 2020, using a spike protein receptor binding domain total antibody chemiluminescence assay (1

66 rsenic-coordinating cysteines within the DNA-binding domain, distal to the zinc-binding site.

67 The binding domains of A. phagocytophilum adhesins A. phagoc

68 -binding surface exhibiting a preference for binding double-strand RNA (dsRNA) over single-strand RNA

69 as activity-based probes and as irreversibly binding drugs.

70 Newer formulations with carbohydrate cores binding elemental iron more tightly allow complete iron

71 te expressions for the prediction of optimal binding energies of important surface intermediates and

72 r the loss or overexpression of the membrane-bound ephrin-B1 in astrocytes during postnatal day (P) 1

73 In vitro DNA-binding experiments and structural prediction show that

74 nes) and antioxidant capacity, especially in bound extracts (1.3 g GAE.100 g(-1); 0.6 g catechin eq.1

75 ntaining polyglutamine tracts including core-binding factor alpha1, mediator subunit 12, transcriptio

76 e compared to that observed when organically bound Fe dominates with this effect because of the stron

77 rol S assay indicated that these metabolites bind ferric iron, which suppresses their production when

78 RA method is able to detect small changes in binding free energy with a sensitivity comparable to in

79 Our findings differentiate effector binding from biological function, which has ramification

80 (cryoEM) structure of PDE6 complexed to GTP-bound Galpha(T).

81 inhibitors bearing two pharmacophores-a zinc binding group and a Cys-reactive warhead-were designed t

82 ly by facilitating the linking between PRDM9-bound hotspots and the nascent chromosome axis through i

83 nists and heteromeric receptors that contain binding-impaired subunits, as we show for both kainate a

84 ultiple regulatory factors that specifically bind in an allele-imbalanced manner to the fSNPs on the

85 s show how the donor and acceptor substrates bind in the active site and how ethambutol inhibits arab

86 hanced phosphorylation (Ser73), and AP-1/DNA-binding in response to S. mansoni infection.

87 both a stabilized HA and alpha-2,6 receptor binding in tandem pose greater pandemic risk.

88 catechin gallate (ECGC) exhibited very rapid binding (in the order of seconds) with Human Salivary al

89 Loss of barbed-end binding increases nucleation by Spire and synergy with C

90 Biophysical insight into the binding interaction between the major whey protein, beta

91 ds high-affinity bonds by using several weak binding interactions simultaneously.

92 ing regions of myosin assures a proper actin-binding interface and active site have formed before pro

93 dditional hIL-23p19 substitutions within its binding interface to hIL-23R and found that the combined

94 spectroscopy experiments indicated that YecA binds iron via its MBD.

95 ases, our work demonstrates that drug-target binding is a major predictor of bacterial responses to a

96 Multivalent binding is essential to many biological processes becaus

97 concentration-dependent manner and that the binding is facilitated by the presence of phosphatidylse

98 The Helstrom-Holevo lower bound is used to derive a proper time-energy/mass uncert

99 g a protein binding motif will alter protein binding, it has been shown that single nucleotide polymo

100 C) and epigallocatechin (EGC) exhibited slow binding kinetics (in the order of minutes), epicatechin

101 Conversely, poly(A) binding KPAF4 shields the nascent A-tail from uridylatio

102 d mutations that diminish PCSK9's ability to bind LDL reported here supports the notion that PCSK9-LD

103 Additionally, high-mannose-binding lectins possess a broad capacity to neutralize a

104 eir ability to internalize together with the bound ligand.

105 stem-loop structure of pri-let-7, which also binds Lin28A/B.

106 teracting with lipid membranes (termed lipid-binding loop [LBL]).

107 Cryo-electron tomography of liposomes with bound MACA showed an amorphous protein layer on the memb

108 Molecules that covalently bind macromolecular targets have found widespread applic

109 exhibit irreversible or reversibly covalent binding mechanisms towards cysteine thiols and other ami

110 that the fluorescence properties of amyloid-bound MHB can be correlated to the change of binding sit

111 c the CXCR4-binding site to MIF, selectively bind MIF with nanomolar affinity and block MIF/CXCR4 wit

112 Based on the binding mode of adenosine 5'-(alpha,beta-methylene)dipho

113 iting in vitro, suggesting that PPR65 cannot bind modified bases due to differences in the structure

114 While it is clear that changing a protein binding motif will alter protein binding, it has been sh

115 TAC") does so by rediscovering ab initio the binding motifs for known regulators and some unknown one

116 affect RNA-protein interactions from outside binding motifs through altered RNA secondary structure.

117 tion initiation factor eIF4E and by ribosome-bound nascent chain ribopuromycylation.

118 ce data indicate three cholesterol molecules bound near F673 in each trimer.

119 -pH resting closed state and a low-pH proton-bound non-conducting state.

120 out evidence of donor and acceptor substrate binding obtained using a crystal engineering approach.

121 s by the KDEL receptor is pH dependent, with binding occurring under acidic conditions in the Golgi a

122 the agonist, ATR, as well as the noncovalent binding of beta-ionone, an antagonist for G protein acti

123 The binding of E. coli to the M13 phage on the cytosensor su

124 Binding of FH increases meningococcal resistance to comp

125 The binding of Fpr to various DNA motifs are mediated by its

126 ted STAT3 or GLI1 knockdown reduced promoter binding of GLI1 and STAT3, respectively.

127 ome buried at the complex interface, disrupt binding of hIL-23p19 to hIL-23R.

128 This process was likely accomplished by the binding of Mt2 ectodomain to Hjv and Hfe.

129 However, how the binding of multiple factors at any given locus is coordi

130 singly charged metals differ profoundly from binding of multiply charged ions, often leading to overa

131 Binding of NucC trimers to a cyclic tri-adenylate second

132 ations involving the strong and/or selective binding of O(2).

133 iochemical, and biologic work has shown that binding of PIP(3) to the pleckstrin homology (PH) domain

134 ke only protein sequence as input to predict binding of protein to DNA, RNA, and other proteins.

135 To detect any NR2B-specific binding of radioligand in brain, various preblocking or

136 resistance, probably owing to high-affinity binding of SAP2 to pyrethroid insecticides.

137 tion of the T cell receptor (TCR) results in binding of the adapter protein Nck (noncatalytic region

138 iosensors can be used to monitor equilibrium binding of the agonist, ATR, as well as the noncovalent

139 ctures in complex with HIV-1 RT/dsDNA showed binding of the conjugates at the polymerase active site,

140 by their typically low affinity and variable binding of the SIMs in parallel and antiparallel orienta

141 ERV into an A-repeat deficient Xist rescues binding of Xist RNA to Spen and results in strictly loca

142 , with non-inferiority declared if the lower bound of the 95% confidence interval was greater than -1

143 Lck is present either in coreceptor-bound or coreceptor-unbound (free) forms, and we here pr

144 e self-organisation and dynamics of membrane-bound organelles such as the Golgi apparatus, remain elu

145 n protein-protein interactions to favor Cas2 binding over tetramerization; this in turn led to prefer

146 ygen transport and exchange by cooperatively binding oxygen with moderate affinity.

147 Crystal structures of arsenic-bound p53 mutants reveal a cryptic allosteric site invol

148 This spurs interest in simulations of (un)binding pathways of TSPO ligands, which could reveal the

149 ay-based platform yields high-quality MHC-II-binding peptide datasets that can be used to improve the

150 In this study, free and bound phenolic acids (PAs) profile, betaine and choline

151 Extraction of free and bound phenols from millet in acidic and basic hydrolytic

152 ealed a large, membrane-accessible substrate-binding pocket that alternately faced the ER lumen and c

153 metabolites within the ATP and D-cycloserine binding pockets of Ddl.

154 polymerized microtubule ends, to which KIF5B binds poorly, likely because its cofactors, MAP7-family

155 an be used to improve the accuracy of MHC-II binding prediction algorithms, and potentially enhance o

156 However, current MHC-binding prediction methods lack an analysis of the major

157 We found that folding-upon-binding primarily occurred through induced-folding pathw

158 We further show that those different binding properties directly translate to distinct biolog

159 ently suggests that the knowledge of the DNA-binding properties of the proteins is in itself not suff

160 e transcription factor cAMP response element-binding protein (CREB) to enhance the expression of prot

161 strained, and enriched for cis-eQTLs and RNA-binding protein (RBP) interactions.

162 rotein (GFP), siderocalin (Scn), and retinol-binding protein 4 (RBP4) as model proteins and screened

163 TAR DNA-binding protein 43 (TDP-43) has emerged as a key player

164 tent with adipogenesis, and the phospholipid-binding protein annexin A3 (AnxA3), a negative regulator

165 ogin (SCGN) is a recently discovered calcium-binding protein belonging to the group of EF-hand calciu

166 show that the phosphorylation of the RNA-DNA binding protein fused in sarcoma (FUS) is higher in Itga

167 Insoluble, hyperubiquitylated TAR DNA-binding protein of 43 kDa (TDP-43) in the central nervou

168 ure of the DNA-binding domain of a model ASO-binding protein PC4, in complex with a full PS 2'-OMe DN

169 teracts with the TRFH domain of the telomere binding protein TRF2.

170 t the promoter, recruits the C/EBPbeta (CREB-binding protein) and CBP transcription factors and activ

171 excitement around ProQ as a novel global RNA-binding protein, and its potential to serve as a matchma

172 ned to the filament domain containing myosin binding protein-C, the "C-zone." Myosin motors in domain

173 transcriptional coactivator CBP and TATA-box binding protein.

174 nce has indicated that circRNAs can directly bind proteins and participate in a myriad of different b

175 rands and crosslink them: class A penicillin-binding proteins (aPBPs) and complexes of SEDS proteins

176 he endoplasmic reticulum (ER) immunoglobulin binding proteins (BiPs) are molecular chaperones involve

177 ition for binding sites among protective RNA-binding proteins and decay factors, PTBP1 promotes displ

178 To identify RNA binding proteins potentially driving these patterns, we

179 for calcium management (calmodulin, calcium-binding proteins), pH regulation (V-type proton ATPase),

180 Here we have developed monobodies, synthetic binding proteins, that bind the N-terminal four-helix bu

181 mine the binding selectivities of several HS-binding proteins.

182 in belonging to the group of EF-hand calcium-binding proteins.

183 tified two distal quinone-binding sites with bound quinones.

184 nks GRAF1b/2 to Rab8a/b and Rab10, and WDR44 binds Rab11.

185 y correlates with EGFR numbers available for binding, rather than specific signalling events.

186 defined as less than 65% of putamen striatal binding ratio expected for the individual's age.

187 e whether this glycosylation site may be the binding region for EphA2, we compared the EphA2 binding

188 ormed, using CRISPR-Cas9 to delete MafK-int6 binding region in IRF8 expression-restrictive cells.

189 binding restricts the dynamics in the Ca(2+)-binding region.

190 ent at the central transcription factor (TF) binding regions and at the flanking eRNA initiation regi

191 mall changes outside of highly conserved DNA-binding regions can lead to profound changes in protein

192 munication between the actin- and nucleotide-binding regions of myosin assures a proper actin-binding

193 that increase the surface area of the actin-binding regions promoting myosin interaction with actin,

194 a-interface, mapped to Switch I and effector-binding regions, (ii) alpha-interface at the allosteric

195 ion, some fundamental aspects of protein-DNA binding remain poorly understood(1,2).

196 Knowledge of protein-binding residues (PBRs) improves our understanding of pr

197 Ca(2+) binding restricts the dynamics in the Ca(2+)-binding reg

198 n the microtubule-bound state by slowing ATP-binding, resulting in high-force production at both homo

199 rinted as a glycan microarray to examine the binding selectivities of several HS-binding proteins.

200 Activated purified CaMKIIalpha also directly binds Shank3 between residues 829 and 1130.

201 The structure reveals a composite binding site bridging over three domains of one capsid p

202 d or in chromosomal DNA, containing the same binding site but with a different reporter.

203 nal repression of Wapl through a single Pax5-binding site by recruiting the polycomb repressive compl

204 tyrosine to phenylalanine substitution at a binding site could be detected.

205 To date, a binding site for P on L had not been described.

206 ctivity when targeting the peptide substrate binding site of NTMT1/2.

207 hylated context, either deletion of the CTCF binding site or depletion of RAD21 cohesin complex prote

208 Disruption of the NAD(+)-binding site or the ARM-TIR interaction caused constitut

209 bound MHB can be correlated to the change of binding site polarity and that a tyrosine to phenylalani

210 tides ('msR4Ms') designed to mimic the CXCR4-binding site to MIF, selectively bind MIF with nanomolar

211 n the DNA-binding domain, distal to the zinc-binding site.

212 n happens at a position far from the antigen binding site.

213 e MITF as a model, we show that low-affinity binding sites act as a competitive reservoir in vivo fro

214 zation that depend on direct competition for binding sites among protective RNA-binding proteins and

215 ture.ORG for further investigation of glycan-binding sites and glycan structures.

216 lycomb repressive complex occupancy and CTCF binding sites are associated with cancer-specific gene d

217 nalysis of the data allowed pinpointing CodY-binding sites at close to single-nucleotide resolution.

218 ally identify ZMAT3-regulated RNAs and their binding sites at nucleotide resolution in intact colorec

219 Modeling studies unveil the specific binding sites for acetylene capture as well as the inter

220 during infection without the need to evolve binding sites for antisilencing proteins at each foreign

221 reporter assay (MPRA) libraries composed of binding sites for SOX2, POU5F1 (OCT4), KLF4, and ESRRB.

222 We identify binding sites for substrate K(+) and Cl(-) ions, demonst

223 of both the allosteric ligands and receptor binding sites important for these allosteric activities.

224 PSF30-hFip1 complex in vitro, and both hFip1 binding sites in CPSF30 can support polyadenylation.

225 ed in this way often contain multiple ligand binding sites or modification sites, which can operate t

226 We also identified two distal quinone-binding sites with bound quinones.

227 8 forms a dimeric structure with four Zn(2+) binding sites within each subunit: a highly conserved pr

228 Using TR ChIP-seq peaks or imputed TR binding sites, Lisa probes the chromatin models using in

229 on of the protein depending on the preferred binding sites.

230 been proposed to be mediated by their lysine-binding sites.

231 OTAIR-sbid impaired the ability of HOTAIR to bind Snail and, in turn, trigger H3K27me3/EZH2-mediated

232 ides were printed as a microarray to examine binding specificities of lectins, anti-ganglioside antib

233 s stabilize motor domains in the microtubule-bound state by slowing ATP-binding, resulting in high-fo

234 Our recent work demonstrated that B2 RNA binds stress genes to retard transcription elongation.

235 through remodeling and activation of an ATF4-bound, stress-responsive enhancer.

236 ffold with basic patches constituting an RNA-binding surface exhibiting a preference for binding doub

237 ious DNA motifs are mediated by its flat DNA-binding surface, which is centered on a short loop spann

238 nding to the same consensus motif, their DNA-binding syntax is different, suggesting discriminatory f

239 SN7-13 does not inhibit coagulation, binds Tau with low nanomolar affinity, and inhibits cell

240 monobodies, synthetic binding proteins, that bind the N-terminal four-helix bundle (4HB) "killer" dom

241 anized VHH phage libraries that specifically bind the S1 SARS-CoV-2 spike protein, and block the inte

242 and HPV-31 Y102E are similar in that neither binds the C terminus of Brd4, but in all other aspects t

243 ivision machinery by hijacking NuMA from LGN binding, thereby favoring TIC self-renewal.

244 uclear receptors (NRs), NR2F2 and NR2C2, can bind to (TCAGGG)(n) variant repeats within telomeres and

245 nto living cells and recover conjugates that bind to an intracellular target.

246 f p44/p62 complexes without XPD reveals they bind to and randomly diffuse on DNA, however, in the pre

247 well-established that transcription factors bind to specific DNA sequences using a combination of ba

248 structure reveals that two Rab33B molecules bind to the diverging alpha-helices of the dimeric Atg16

249 ation during reductive ER stress but did not bind to this protein during nonreductive ER stress.

250 SidI binding to eEF1A and Lpg2505 is not mutually exclusive,

251 , multivalent interactions couple productive binding to efficient deacetylation of histones on endoge

252 We compared plasma antibody binding to HIV antigens between 51 nontransmitting mothe

253 changes in PCSK9 required for high-affinity binding to LDL particles.

254 as been proposed to also contribute to toxin binding to neurons by interacting with lipid membranes (

255 blast HS genetically to make it incapable of binding to OPG.

256 t is involved in DNA replication by directly binding to specific motifs within their promoters.

257 enza IgG monoclonal antibodies for selective binding to the activating Fcgamma receptor FcgammaRIIa r

258 ently by promoting oligomerization involving binding to the C-terminal region.

259 otes immunostimulatory secondary necrosis by binding to the phagocytic marker phosphatidylserine on d

260 While binding to the same consensus motif, their DNA-binding s

261 thambutol inhibits arabinosyltransferases by binding to the same site as both substrates in EmbB and

262 Results: LW223 binding to TSPO was not susceptible to the rs6971 geneti

263 ulin); 5) is competitive with paclitaxel for binding to tubulin but not with vinblastine, crocin, or

264 and pro-metastatic effects through directly binding to Vimentin and competitively abrogating Trim16-

265 Besides binding to well-established receptors, an extended loop

266 NSP1 binds to 18S ribosomal RNA in the mRNA entry channel of

267 These studies show that PGC-1alpha binds to intronic RNA sequences, some of them controllin

268 osomes revealed that the Aster-B GRAM domain binds to membranes in a cholesterol concentration-depend

269 , and WDR41 is a beta-propeller protein that binds to SMCR8 such that the whole structure resembles a

270 ion is controlled by the MatP protein, which binds to specific sites (matS) within ter, and interacts

271 ased the IC(50) value, suggesting that MDIMP binds to the extracellular side of the pore.

272 HDL NPs are a cholesterol-poor ligand that binds to the receptor for cholesterol-rich HDLs, scaveng

273 Because STING directly binds to TRIF, we identified the STING-interacting domai

274 s even after the removal of the compound; 3) binds to tubulin [dissociation constant (K(d)) 0.4 +/- 0

275 crystal structure of the LSD1/CoREST complex bound to a 191-bp nucleosome.

276 Moreover, in cells, MANF bound to a model ER protein exhibiting improper disulfid

277 was able to detect C. albicans cells, and it bound to Adh1 in yeast and Adh2 in hyphae among the cell

278 ASL(Arg1)(ICG) and ASL(Arg2)(ICG) constructs bound to cognate and wobble codons on the ribosome revea

279 ring human memory B cells are constitutively bound to IgA.

280 Crm proteins from different orthopoxviruses bound to membrane-associated TNF and dampened inflammato

281 different miRNAs or when the two sites were bound to miRNAs loaded into two different AGO paralogs,

282 ing reactivator, monoxime RS194B, reversibly bound to native and venomous agent X (VX)-inhibited huma

283 PTBP1 promotes displacement of UPF1 already bound to potential substrates.

284 is constitutively present in healthy tissues bound to resident cells via its high-affinity receptor,

285 frared light sensitivity using gold nanorods bound to temperature-sensitive engineered transient rece

286 f pore formation for the CDC intermedilysin, bound to the human immune receptor CD59 in a nanodisc mo

287 ed by the nuclear receptor NR4A1/Sp1 complex bound to the proximal germinal center (GC)-rich region o

288 her report the cryoEM structure of human CMG bound to the replisome hub AND-1 (CMGA).

289 corporation, and additionally, we show that, bound to UvrC, the [4Fe4S] cofactor is susceptible to ox

290 experiments, and inhibition analysis of GD3-binding toward Siglec-7 using synthetic sialoglycoconjug

291 of four highly conserved Trp residues in the binding tunnel had been replaced with Ala.

292 How integrin binding under shear stress mechanosignals a functional s

293 New pS22-Lamin A/C binding was accompanied by increased histone acetylation

294 Cooperative binding was retained when the two sites were for two dif

295 ation between raphe and hippocampal 5-HT(1A) binding which was more pronounced in HV.

296 latory constraints to which this industry is bound will be outlined in brief, as well as an introduct

297 We measured eosinophil binding with a Sykes-Moore adhesion chamber.

298 moniae is a dimeric protein that potentially binds with single-stranded DNA (ssDNA) in a manner simil

299 nform ongoing controversy about where XPA is bound within the NER bubble, provide structural insights

300 inst non-specific, adventitious nanoparticle binding, without the need for complex surface chemistrie