ライフサイエンスコーパス: binding factor

1 that has high-affinity binding to the CCCTC-binding factor.

2 i allele extensively binds the protein CCCTC-binding factor.

3 king site for TFIID, the major core promoter-binding factor.

4 rred in the putative A protein, an important binding factor.

5 is a gamma-tubulin small complex (gammaTuSC) binding factor.

6 cribed regions via association with upstream binding factor.

7 the mitochondrial protein LRPPRC as an NS5A binding factor.

8 ing sites where nucleosomes compete with DNA-binding factors.

9 lastic moduli of hydrogels, with and without binding factors.

10 s HO activation to one of two paralogous DNA-binding factors.

11 by moderating the activities of E2 promoter-binding factors.

12 interaction with regulatory elements and DNA-binding factors.

13 nucleosome-induced cooperativity between DNA-binding factors.

14 ell-type-specific regulation directed by DNA binding factors.

15 ting elements, alternative splicing, and RNA-binding factors.

16 uding interaction with sequence-specific DNA-binding factors.

17 RBP AU-binding factor 1 (AUF1) has four isoforms resulting from

18 The RBP AU-rich-binding factor 1 (AUF1) isoform p37 was found to have hi

19 onal coactivation complex through C promoter-binding factor 1 (CBF1; RBPJ) and strengthens the associ

20 gamma (PPARgamma), leptin, osteonectin, core binding factor 1 (CBFA1), and FBJ murine osteosarcoma vi

21 racts with the distal appendage proteins Fas-binding factor 1 (FBF1) and centrosomal protein 83 (CEP8

22 ctroscopy to study the human telomere repeat binding factor 1 (hTRF1) in complex with Escherichia col

23 o suppressed beta-catenin, lymphoid enhancer-binding factor 1 (LEF-1), cyclin D1, N-Myc, and INSM1 le

24 The transcription factor lymphoid enhancer-binding factor 1 (LEF-1), which plays a definitive role

25 n via the nuclear protein, lymphoid enhancer-binding factor 1 (LEF-1).

26 odermin (EOMES; mesoderm), lymphoid enhancer-binding factor 1 (LEF1) and mesoderm posterior BHLH tran

27 that NRARP interacts with lymphoid enhancer-binding factor 1 (LEF1) and potentiates Wnt signaling in

28 contain normal amounts of lymphoid enhancer-binding factor 1 (Lef1) and transcription factor 1 (Tcf1

29 ers a binding site for the lymphoid enhancer-binding factor 1 (LEF1) transcription factor, reducing L

30 nd increased expression of lymphoid enhancer-binding factor 1 (LEF1), a downstream effector in the ca

31 nuclear proteins, i.e. the lymphoid enhancer-binding factor 1 (Lef1), histone H3, and Brahma-related

32 argets by interacting with lymphoid enhancer binding factor 1 (LEF1).

33 ners: the PARylated partner telomeric repeat-binding factor 1 (TRF1) and the non-PARylated partner GD

34 Telomeric repeat binding factor 1 (TRF1) is essential to the maintenance

35 Here, we show that Telomere Repeat Binding Factor 1 (TRF1), a component of the shelterin co

36 Here we establish that telomeric repeat-binding factor 1 (TRF1), a core component of the telomer

37 the nucleolar transcription factor upstream binding factor 1 (UBF1) on rDNA promoters and recruiting

38 e it binds the transcription factor upstream binding factor 1 (UBF1) on the promoter regions of ribos

39 CSNK2A2 phosphorylates telomeric repeat binding factor 1 and plays an important role for regulat

40 y the transcription factor lymphoid enhancer-binding factor 1 downstream of the Wnt signaling pathway

41 A-binding protein GRSF1 (G-rich RNA sequence-binding factor 1) critically maintains mitochondrial hom

42 the collection of RNAs regulated by AUF1 (AU-binding factor 1), an RBP linked to cancer, inflammation

43 an antigen R) and GRSF1 (G-rich RNA sequence-binding factor 1), that associated with the nuclear DNA-

44 The mammalian RNA-binding protein AUF1 (AU-binding factor 1, also known as heterogeneous nuclear ri

45 roles of RBPs HuR, CUG-binding protein 1, AU-binding factor 1, and several GI epithelial-specific miR

46 llerRed chromophore with the telomere repeat binding factor 1, TRF1, we developed a novel approach to

47 f the canonical Wnt target lymphoid enhancer binding factor 1.

48 f the transcription factor lymphoid enhancer-binding factor-1 (LEF-1).

49 ing putative T-cell factor/lymphoid enhancer binding factor-1 (TCF/LEF) motifs, consistent with canon

50 We identified nuclear receptor binding factor 2 (Nrbf2) as a Beclin 1-interacting prote

51 t a putative fifth subunit, nuclear receptor binding factor 2 (NRBF2), is a tightly bound component o

52 nthesis, including sterol regulatory element binding factor 2 (Srebf2) and 3-hydroxy-3-methylglutaryl

53 ivator protein 1 (RAP1) and telomeric repeat-binding factor 2 (TRF2) are two subunits in shelterin th

54 To examine the role of telomeric repeat-binding factor 2 (TRF2) in epithelial tumorigenesis, we

55 The role of the telomere repeat-binding factor 2 (TRF2) in telomere maintenance is well-

56 e 3' overhang facilitated by telomere repeat-binding factor 2 (TRF2) into telomeric DNA, forming t-lo

57 he telomere-binding protein telomeric repeat-binding factor 2 (TRF2).

58 t a non-telomeric isoform of telomere repeat-binding factor 2 (TRF2-S) is a novel RBP that regulates

59 functionally interacts with telomere repeat-binding factor 2 that in turn regulates its helicase act

60 of the shelterin component telomeric repeat-binding factor 2, cells survived but remained dormant an

61 nother shelterin component, telomeric repeat binding factor 2, interacting protein (p.Ala104Pro and p

62 enging RNA-binding proteins, including Fem-3-binding-factor 2, Argonaute 2 and Ribonuclease III, Nucl

63 ion and telomere binding of telomeric repeat-binding factor-2 (TRF2), associated with increased DNA d

64 ER, forkhead box protein A1 (FOXA1) and GATA-binding factor 3 (GATA3).

65 (Zea mays), several genes, including a G-BOX BINDING FACTOR 3 (GBF3) were identified as candidate dro

66 yeloma tumor protein 2, interleukin enhancer binding factor 3 (IEBP3), guanine nucleotide binding pro

67 Interleukin enhancer-binding factor 3 (ILF3), an RNA-binding protein, is best

68 vealed a greater increase in binding of GATA-binding factor 6 (GATA-6) and NF-kappaB to their cognate

69 GATA-binding factor 6 (GATA6) is a zinc-finger transcription

70 itoneal cavity and immediately infected GATA-binding factor 6-positive (GATA6+) peritoneal cavity mac

71 homolog of the bacterial RbfA (for ribosome-binding factor A) protein that was identified as a cold-

72 Cocaine exposure reduces binding of CCCTC-binding factor, a chromosomal scaffolding protein, and i

73 se 1 via mTOR (P < 0.02), and total upstream binding factor abundance were significantly greater in E

74 d-Sequential Induction in Patients With Core Binding Factor Acute Myeloid Leukemia and Treating Patie

75 Although core-binding factor-acute myeloid leukemia (CBF-AML) (t[8;21]

76 ntaining polyglutamine tracts including core-binding factor alpha1, mediator subunit 12, transcriptio

77 ML and are usually reported together as core binding factor AML (CBF-AML).

78 o 60 years of age, excluding those with core binding factor AML and acute promyelocytic leukemia, wer

79 able-risk cytogenetics (eg, t[15;17] or core-binding factor AML) or who had received previous BCL2-in

80 ontain an insulator element that binds CCCTC-binding factor and reduces its insertional genotoxicity.

81 ions of CDKs and cyclins, and of E2 promoter-binding factors and dimerization partners confirmed inte

82 example of ATG transcript regulation via 3' binding factors and exosomal degradation.

83 tation experiments for tens of different DNA-binding factors and from ribosome profiling experiments

84 e coordinated integration and release of ES6 binding factors and likely plays a pivotal role in remod

85 probably enhances Ana2 avidity for centriole-binding factors and may bridge multiple factors as requi

86 gh a competitive interplay between chromatin-binding factors and nucleosomes.

87 tors such as corepressors, coactivators, DNA-binding factors and PTM modifying enzymes.

88 tions for how chromatin accessibility to DNA-binding factors and the RNA transcription machinery is r

89 areas: the extra-telomeric role of telomere-binding factors and the role of telomeres in pluripotenc

90 E2 Y102F protein interacted with cellular E2-binding factors and the viral helicase E1; however, in c

91 o chromatin structural proteins, Ctcf (CCCTC-binding factor) and Hmgb2 (high mobility group protein B

92 the transcriptional insulator protein CCCTC-binding factor, and interacted with each other, creating

93 cluding histone modifications and associated binding factors, and their functional contribution to tr

94 Other identified HVD-binding factors are also involved in EEEV replication, b

95 atin domains, whereas weak or non-nucleosome binding factors are excluded from the domains and enrich

96 Cohesin and CTCF (CCCTC-binding factor) are key regulators; perturbing the level

97 nd functioned as ethylene-responsive element binding factor-associated amphiphilic repression (EAR) d

98 coding gene with ethylene-responsive element binding factor-associated amphiphilic repression (EAR) m

99 members, and the ethylene-responsive element binding factor-associated amphiphilic repression (EAR-li

100 presence of the ethylene-responsive element-binding factor-associated amphiphilic repression motif.

101 PF74's potency depended on these CA-binding factors at low doses.

102 plex is composed of the unique cofactor core binding factor beta (CBF-beta) and canonical ligase comp

103 ith HIV-1 Vif and the cellular cofactor core-binding factor beta (CBFbeta) at 3.9- angstrom resolutio

104 and analyze the possible involvement of core binding factor beta (CBFbeta) in this process.

105 ort that the transcriptional controller core binding factor beta (CBFbeta) is required for ILC2 activ

106 ariants to the transcriptional cofactor core binding factor beta (CBFbeta), which is expressed in cel

107 Vif critically depends on the cellular core binding factor beta (CBFbeta).

108 nscription factor fusion CBFbeta-SMMHC (core binding factor beta and the smooth-muscle myosin heavy c

109 on factor 1 (RUNX1) and its associating core binding factor-beta (CBFB) play pivotal roles in leukemo

110 Here, we show that deletion of CTCF (CCCTC-binding factor)-binding sites at TAD and sub-TAD topolog

111 ding of HvGAMYB and the Barley Prolamine-Box binding Factor (BPBF) to their target sequences.

112 se, and TAF12, an RNA polymerase II TATA-box binding factor, cause CIN when overexpressed in human ce

113 rd genetic screen for regulators of C-REPEAT BINDING FACTOR (CBF) gene expression (RCFs), we identifi

114 ns of gene expression in the poplar C-repeat binding factor (CBF) gene family.

115 e map to a region that includes the C-repeat binding factor (CBF) locus.

116 The C-REPEAT-BINDING FACTOR (CBF) pathway has important roles in plan

117 lerance that involves action of the C-repeat binding factor (CBF) regulatory pathway.

118 Expression of the C-repeat-binding factor (CBF) transcription factors is induced by

119 thaliana, acting downstream of the C-repeat binding factor (CBF) transcription factors to recruit th

120 including rearrangements involving the core-binding factor (CBF) transcriptional complex.

121 the C-REPEAT/DEHYDRATION-RESPONSIVE ELEMENT BINDING FACTOR (CBF) transcriptional regulators that con

122 ved specific adaptation in the C-repeat/DREB binding factor (CBF)-dependent and CBF-independent cold

123 ene expression under cold stress by C-REPEAT BINDING FACTOR (CBF)-dependent and CBF-independent pathw

124 The C-repeat binding factor (CBF)/dehydration-responsive element bind

125 ble in 165/1026 (16.1%) of aberrant non-core-binding-factor (CBF) karyotype patients.

126 RUNX1/CBFbeta (core binding factor [CBF]) is a heterodimeric transcription f

127 ric transcription is repressed by centromere-binding factor Cbf1 and histone H2A variant H2A.Z(Htz1)

128 epeat (CRT)/drought-responsive element (DRE) binding factor CBF1/DREB1b is a transcriptional regulato

129 The beta-subunit of core binding factor (CBFbeta), that heterodimerizes with RUNX

130 Core Binding Factors (CBFs) are a small group of heterodimeri

131 C-Repeat Binding Factors (CBFs) are DNA-binding transcriptional a

132 factors mediating cold acclimation, C-REPEAT BINDING FACTORs (CBFs), interact with PHYTOCHROME-INTERA

133 cleus-encoded transcription factors C-REPEAT BINDING FACTORS (CBFs).

134 he viral envelope contains the mammalian LPS-binding factors CD14, TLR4, and MD-2, which, in conjunct

135 epair factor), with nuclear entry of the DNA-binding factor cGAS.

136 pproach uncovered the chromatin helicase DNA-binding factor CHD1 as a putative synthetic-essential ge

137 activator Sub1, recently identified as a G4-binding factor, contributes to genome maintenance at G4-

138 hancers, promoters, insulators and chromatin-binding factors cooperatively regulate gene expression.

139 in an inter-chromosomal Sox-2 enhancer/CCCTC-binding factor CTCF/cohesin loop) to an active (within a

140 that could be rescued by deleting the CCCTC-binding factor (CTCF) anchor of the stripe.

141 ropic "tension globule." In the other, CCCTC-binding factor (CTCF) and cohesin act together to extrud

142 erved chromosome loop anchors bound by CCCTC-binding factor (CTCF) and cohesin are vulnerable to DNA

143 CCCTC-binding factor (CTCF) and cohesin play critical roles in

144 ence specificity in vitro and in vivo, CCCTC-binding factor (CTCF) and its paralog brother of the reg

145 set of DNA-binding proteins, including CCCTC-binding factor (CTCF) and pluripotency factors.

146 fic chromatin interactions mediated by CCCTC-binding factor (CTCF) and RNA polymerase II (RNAPII) wit

147 ink pairing to allelic partitioning of CCCTC-binding factor (CTCF) and show that tethering DCP1A to o

148 mains are enriched for binding sites of CTCC-binding factor (CTCF) and the cohesin complex, implicati

149 in part by the architectural proteins CCCTC binding factor (CTCF) and the cohesin complex, which co-

150 Sequence variants in CCCTC-binding factor (CTCF) and transcription factor binding s

151 inding sites for the insulator protein CCCTC-binding factor (CTCF) are constitutively occupied, a sub

152 tin fiber and the proteins cohesin and CCCTC-binding factor (CTCF) as key players anchoring such chro

153 ant interactions that are enriched for CCCTC-binding factor (CTCF) bidirectional motifs and interacti

154 g transcription start sites (TSSs) and CCCTC-binding factor (CTCF) binding sites, and uncovered an in

155 elements, many of which coincide with CCCTC-binding factor (Ctcf) binding sites, for example, the cl

156 methylated regions to be enriched with CCCTC-binding factor (CTCF) binding sites.

157 ing but decreased transcription factor CCCTC-binding factor (CTCF) binding.

158 EMENT This work provides evidence that CCCTC-binding factor (CTCF) controls an early fate decision po

159 The host cell CCCTC-binding factor (CTCF) functions in genome-wide chromatin

160 Here we demonstrate that the CCCTC-binding factor (CTCF) functions to insulate these adjace

161 th the architectural/insulator protein CCCTC-binding factor (CTCF) genome-wide.

162 key regulator of genome organization, CCCTC-binding factor (CTCF) has been characterized as a DNA-bi

163 Insulators also gain H3K27ac and CCCTC-binding factor (CTCF) in anaphase/telophase.

164 The function of the CCCTC-binding factor (CTCF) in the organization of the genome

165 MIR insulators appear to be CCCTC-binding factor (CTCF) independent and show a distinct lo

166 The CCCTC-binding factor (CTCF) is a central regulator of chromati

167 ial dysfunction.SIGNIFICANCE STATEMENT CCCTC-binding factor (CTCF) is a DNA-binding protein that orga

168 CCTC-binding factor (CTCF) is a key regulator of gene express

169 CCCTC-binding factor (CTCF) is a key regulator of nuclear chro

170 CCCTC-binding factor (CTCF) is a multi-functional chromatin re

171 The CCCTC-binding factor (CTCF) is a zinc finger DNA binding prote

172 CCCTC-binding factor (CTCF) is an 11 zinc finger DNA-binding d

173 er determined that chromatin insulator CCCTC-binding factor (CTCF) is TGF-beta inducible and facilita

174 ss of architectural proteins, of which CCCTC-binding factor (CTCF) is the best characterized.

175 The CCCTC-binding factor (CTCF) organises the genome in 3D through

176 ed box 5 (PAX5), Yin Yang 1 (YY1), and CCCTC-binding factor (CTCF) play a role in regulating the acce

177 CCCTC-binding factor (CTCF) plays a key role in the formation

178 CCCTC-binding factor (CTCF) plays an essential role in regulat

179 DNA demethylation facilitated CCCTC-binding factor (CTCF) recruitment to the maternally expr

180 The highly conserved zinc finger CCCTC-binding factor (CTCF) regulates genomic imprinting and g

181 Chromatin looping mediated by the CCCTC binding factor (CTCF) regulates V(D)J recombination at A

182 s of which are enriched in cohesin and CCCTC-binding factor (CTCF) required for long-range interactio

183 lysine 4 trimethylation (H3K4me3) and CCCTC-binding factor (CTCF) signals.

184 f2 ICR [Delta2,3] removing two of four CCCTC-binding factor (CTCF) sites and the intervening sequence

185 ed TAD borders reveals the presence of CCCTC-binding factor (CTCF) sites with diverging orientations

186 polymerase III (Pol III) elements and CCCTC binding factor (CTCF) sites.

187 mediated by the architectural protein CCCTC-binding factor (CTCF) that binds to the boundaries of to

188 Binding of CCCTC-binding factor (CTCF) to the first exon of the hTERT gen

189 tment of the transcriptional repressor CCCTC-binding factor (CTCF) to the MSMP enhancer region was de

190 The transcriptional insulator CCCTC binding factor (CTCF) was shown previously to be critica

191 nt) database identified association of CCCTC-binding factor (CTCF) with Ahr promoter in mouse livers.

192 c changes regulated the association of CCCTC-binding factor (CTCF) with select genomic sites.

193 n this report, we demonstrate that the CCCTC-binding factor (CTCF), a crucial chromatin organizer, is

194 indicate that LINC00346 interacts with CCCTC-binding factor (CTCF), a known transcriptional repressor

195 eracts with androgen receptor (AR) and CCCTC-binding factor (CTCF), and modulates AR-dependent gene e

196 The multidomain CCCTC-binding factor (CTCF), containing a tandem array of 11 z

197 The CCCTC-binding factor (CTCF), which anchors DNA loops that orga

198 DNF chromatin by DNA insulator protein CCCTC-binding factor (CTCF), which is associated with suppress

199 alized by simultaneous mutation of two CCCTC-binding factor (CTCF)-binding elements (CBE1 and CBE2) w

200 We found weak CCCTC-binding factor (CTCF)-binding sites and reduced CTCF bin

201 gene desert form a 1 Mbp loop between CCCTC-binding factor (CTCF)-binding sites that is separated fr

202 xhibit hypermethylation at cohesin and CCCTC-binding factor (CTCF)-binding sites, compromising bindin

203 cterized by clusters of hypomethylated CCCTC-binding factor (CTCF)-bound elements, which were predomi

204 n start sites (TSS), and the number of CCCTC-binding factor (CTCF)-cohesin complexes between the inte

205 -H1 interactions, is propagated over a CCCTC-binding factor (CTCF)-demarcated region through a distin

206 on, long noncoding RNAs (lncRNAs), and CCCTC-binding factor (CTCF)-RNA interactions, but systematic a

207 domain (TAD) boundaries, enriched for CCCTC-binding factor (CTCF).

208 ovement by transcription and DNA-bound CCCTC-binding factor (CTCF).

209 tifs for the enhancer blocking protein CCCTC-binding factor (CTCF).

210 on, transcription factor [Abeta(42) or CCCTC-binding factor (CTCF)] binding, and Txnip promoter hypom

211 by the binding of hundreds of different DNA binding factors (DBFs) to the genome.

212 sis that apical HS in the airway serves as a binding factor during infection, and HS modulating compo

213 rotein that functions dually as a centromere binding factor during segrosome assembly and as a transc

214 ion by competing with eIF5 for the Met-tRNAi-binding factor eIF2.

215 itiate translation in the absence of the cap-binding factor eIF4E.

216 recruited to the miR-31 promoter by the DNA binding factor ETS-1, and it represses miR-31 transcript

217 gene pABRE:NCED enhanced NCED and ABF (ABRE-binding factor) expression in Arabidopsis Columbia-0 see

218 r of C-repeat/dehydration responsive element-binding factor expression1/scream2 that are required for

219 and ABA-INSENSITIVE5/ABA-responsive element binding factor family identified specific members that c

220 n the Caenorhabditis elegans germline, fem-3 Binding Factor (FBF) partners with LST-1 to maintain ste

221 milio (PUM) and Caenorhabditis elegans fem-3-binding factor (FBF), recognize specific sequences in th

222 s that were only occupied transiently by DNA-binding factors following passage of the replication for

223 Adding binding factors from collagen and RGD peptides increases

224 Zic family of proteins as enhancer-specific binding factors functioning in development.

225 dies on a beta-globin enhancer- and promoter-binding factor, GATA-1, the founding member of the GATA

226 Yet the DNA-binding factors guiding this complex to specific targets

227 ii evades complement-mediated destruction by binding factor H (FH), a host-derived negative regulator

228 hbB protein contributes to immune evasion by binding factor H (FH).

229 g competent substrate, human telomere repeat binding factor (hTRF1), which is bound to DnaK in a glob

230 The interleukin enhancer binding factors ILF2 (NF45) and ILF3 (NF90/NF110) have b

231 al deletion of the chromatin remodeler CCCTC-binding factor impaired nerve regeneration, implicating

232 Besides other mechanisms, GAGA-binding factors in animals can guide PRC members in a se

233 g similarities between animal and plant GAGA-binding factors in the recruitment of PRC1 and PRC2 comp

234 ETX) is one of the best characterised R-loop-binding factors in vivo.

235 ctions of RIOK3 with actin and several actin-binding factors including tropomyosins (TPM3 and TPM4) a

236 ble regions that were enriched for other DNA binding factors, including FOXA1, CTCF, and OCT1.

237 Ethylene Response Factor 1 (ERF1), a GCC-box binding factor is in synchrony with expression of AtNudt

238 CTCF (CCCTC-binding factor) is a highly conserved multifunctional DN

239 The host chromatin-binding factor LEDGF/p75 interacts with HIV-1 integrase

240 comprising a beta-catenin/lymphoid enhancer binding factor (LEF) site overlapping with an HNF-1beta

241 able rDNA are active, as defined by upstream binding factor loading.

242 cally interacts with the MluI cell cycle box-binding factor (MBF) transcription complex that regulate

243 Loss of STAG2 inhibited CCCTC-binding-factor-mediated expression of dual specificity p

244 n clinical specimens showed that the heparin-binding factor midkine is a systemic inducer of neo-lymp

245 In this study, we investigate the distinct binding factors needed on apposed membranes for Rab effe

246 ions for the transcriptional regulator CCCTC-binding factor of ESR1 and ESR2, consistent with sustain

247 on with pituitary tumour-transforming 1, the binding factor of PTTG1lP, and with vimentin and E-cadhe

248 and Ska complexes are the major microtubule-binding factors of the kinetochore responsible for maint

249 s VAL1/2 possibly in combination with GT-box binding factors, other AtBMI1 regulatory networks requir

250 tion of abscisic acid-dependent and C-REPEAT-BINDING FACTOR pathways.

251 lastoma tumor suppressor protein/E2 promoter binding factor (pRb/E2F1) pathway, which we have previou

252 rabidopsis (Arabidopsis thaliana) GAGA-motif binding factor protein basic pentacysteine6 (BPC6) inter

253 Pumilio/fem-3 mRNA binding factor (PUF) proteins bind RNA with sequence spe

254 minization of XX and XO animals (fem)-3 mRNA-binding factor (PUF) proteins bind sequence specifically

255 core subunit, Rb-binding protein 4 chromatin-binding factor (RBBP4), further enhances deacetylase act

256 a context-dependent manner, and cis-element-binding factors recruit chromatin regulators that mediat

257 Tau downregulation decreases upstream binding factor recruitment, ribosomal RNA synthesis, rib

258 The soil-binding factors released into rhizospheres to form rhizo

259 ified topoisomerase IIalpha (TOP2A) as a DNA-binding factor required for TCF-transcription.

260 nding protein hnRNPK as the principal XR-PID binding factor required to recruit PCGF3/5-PRC1.

261 s an imprint-specific, sequence-specific DNA binding factor responsible for maintaining methylation a

262 nuclear factor, erythroid 2 (Nfe2) and core-binding factor, runt domain, alpha subunit 2, translocat

263 ng development by the nuclear matrix and DNA binding factor Satb2 (ISR(Satb2)).

264 They assess how a DNA-binding factor Satb2 controls cell position, molecular i

265 or the SCB (Swi4/6-dependent cell cycle box) binding factor (SBF) complex (composed of Swi4 and Swi6)

266 cription factors CREB (cAMP response element binding factor), SRF (serum response factor), and MEF2 (

267 vascular endothelial growth factor and core-binding factor subunit alpha-1 were equivalent to conven

268 Because core-binding factor subunit beta and CypA are both highly con

269 HIV-1 utilize the transcription factor core-binding factor subunit beta as a noncanonical cofactor t

270 cell proteins Elongin-C, Elongin-B, and core-binding factor subunit beta, forming a four-protein comp

271 thought to be generally inaccessible to DNA-binding factors, such as micrococcal nuclease (MNase).

272 Many DNA-binding factors, such as transcription factors, form oli

273 f 53BP1 to methyl K810 occurs on E2 promoter binding factor target genes and allows pRb activity to b

274 egulation is mainly achieved through two DNA-binding factors, Tbf1 and Mcm1.

275 We now discover that the telomere duplex DNA-binding factor, TbTRF, also plays a critical role in VSG

276 nin with the T-cell factor/lymphoid enhancer-binding factor (TCF/LEF) transcription factors and subse

277 , as well as T cell factor/lymphoid enhancer-binding factor (TCF/LEF), causing subsequent repression

278 sent interactions with the site-specific DNA-binding factors that establish and maintain epigenomic m

279 or negative cooperativity by extrinsic G3BP1-binding factors that strengthen or weaken, respectively,

280 strong preferences toward proximal promoter binding, factors that target intergenic and intronic DNA

281 oneybees have evolved a secreted dietary RNA-binding factor to concentrate, stabilize, and share RNA

282 Transcription is regulated through binding factors to gene promoters to activate or repress

283 dging molecule that cooperates with other PS-binding factors to promote the phagocytosis of apoptotic

284 both with components of condensates and DNA-binding factors to selectively occupy super-enhancer-ass

285 Tau is recruited, along with upstream-binding factor, to ribosomal DNA loci.

286 mpling of TRFs, both DNA-binding and non-DNA-binding factors, to provide a framework for the subseque

287 complex, competing with the telomeric-repeat binding factors TRF1 and TRF2.

288 tra-telomeric binding of the telomere repeat binding factor TRF2 within the promoter of the cyclin-de

289 s and insufficient retention of the telomere-binding factor TRF2.

290 PPAN depletion induces NPM and upstream-binding factor (UBF) degradation, which is independent o

291 Herein, we found that upstream binding factor (UBF) interacts with ESET, a histone H3K9

292 ), RNA Polymerase I (Pol I) and the Upstream Binding Factor (UBF).

293 A polymerase I transcription factor upstream binding factor (UBF).

294 forms for the rational design of diverse DNA binding factors useful for synthetic biology and other p

295 Identification of differentially binding factors was performed using mass spectrometry (L

296 ins defined by constitutive binding of CCCTC binding factor, which appears to restrict the regulatory

297 tein-associated splicing factor (PSF) as OEE-binding factors, which was confirmed by ChIP assay from

298 shift analysis identified Pdx1 as the Site 4 binding factor, while an 80-88 kilodalton (kDa) beta-cel

299 at this molecule is a general immunoglobulin-binding factor with highest affinity for IgE.

300 study of nucleosome dynamics and nucleosome-binding factors, with a focus on the experimental advanc