ライフサイエンスコーパス: binge drinking

1 =5 (women) and >=7 (men) units per instance (binge drinking).

2 ening young adults for a repeated history of binge drinking.

3 n have been implicated in sex differences in binge drinking.

4 , after adjusting for age, sex, smoking, and binge drinking.

5 ncy, and quantity of alcohol consumption and binge drinking.

6 ore than 14% of men and 3% of women reported binge drinking.

7 ze injury to marginal fatty grafts caused by binge drinking.

8 y (less than once per month) had episodes of binge drinking.

9 s via hM4Di was sufficient to reduce alcohol binge drinking.

10 ts among subjects reporting higher levels of binge drinking.

11 students, and schools with medium levels of binge drinking.

12 ivity and adaptations to experiences such as binge drinking.

13 posure to abuse, substance use disorder, and binge drinking.

14 ocial defensiveness and abstinence-escalated binge drinking.

15 vioral maladaptations during withdrawal from binge drinking.

16 nd blocked the expression of withdrawal from binge drinking.

17 sodes of excessive alcohol drinking known as binge drinking.

18 t against severe intoxication in response to binge drinking.

19 inhibited target gene expression and blunted binge drinking.

20 and personality as important antecedents of binge drinking.

21 on targets, which plays an important role in binge drinking.

22 predicted all 3 levels of binge and extreme binge drinking.

23 hold (5 drinks) often used in definitions of binge drinking.

24 0.03; p < 0.001) and a higher probability of binge drinking (0.04 percentage points; 95% CI: 0.002, 0

25 -0.02, p = 0.02) and a higher probability of binge drinking (0.17 percentage points; 95% CI: 0.04, 0.

26 of 20.2% of high school seniors reported 5+ binge drinking, 10.5% reported 10+ extreme binge drinkin

27 l inactivity (31%), routine check-ups (14%), binge drinking (11%), lack of insurance (6%), and food i

28 ts; OR 1.75 [95% CI 1.39-2.20]; I(2)=91.0%), binge drinking (13 studies, 25 603 participants; 1.80 [1

29 rdous drinking (14.2 [5.91-34.0]), hazardous/binge drinking (18.9 [7.98-44.8]), and alcohol-related d

30 The authors studied the effect of binge drinking 3 months before pregnancy and during the

31 s of 2.00-59.99 g/day (n = 44,476), frequent binge drinking ( 5 units at least once per month) was no

32 ipants [44.4%] vs 136 participants [35.8%]), binge drinking (95 participants [17.9%] vs 46 participan

33 at least a 4.0-percentage-point increase in binge drinking (95% confidence interval: 0.9, 7.0) and a

34 n (ADE = -0.12; 95% CI, -0.23 to -0.02); not binge drinking (ADE = -0.15; 95% CI, -0.26 to -0.04); or

35 ve stress-induced social interactions and in binge drinking after forced abstinence in female mice.

36 How does binge drinking alcohol change synaptic function, and do

37 In male, but not female mice, we found that binge drinking alcohol produced glutamatergic synaptic a

38 We found that the first binge-drinking alcohol session produced enduring enhance

39 Even in the absence of binge drinking, alcohol consumption during pregnancy can

40 Self-reported monthly binge drinking, alcohol-related harms, and hazardous alc

41 Heavy/binge drinking among college students has become a major

42 mong those who never drank, (2) the onset of binge drinking among those who were never binge drinkers

43 By 2012, the prevalence of binge drinking among young men with children (38.5%) dec

44 lcohol use has been studied extensively, but binge drinking among youth in the United States is not y

45 and understanding predictors of such extreme binge drinking among youth will benefit public health ef

46 orrelates of at-risk alcohol use (especially binge drinking) among middle-aged and elderly persons in

47 Rates of 5+ binge drinking and 10+ extreme binge drinking have decli

48 ther saline (sham) or ethanol (as a model of binge drinking and acute ethanol intoxication).

49 documented clinical relevance to adolescent binge drinking and alcoholism in humans to test whether

50 Scheduled Access) to induce escalating heavy binge drinking and anxiety-like behavior in mice.

51 ddle-aged and older adults should screen for binge drinking and coexisting use of other substances.

52 al relevance of PKCepsilon translocation for binge drinking and determination of potential upstream s

53 h-risk alcohol consumption patterns, such as binge drinking and drinking before driving, and underage

54 eing, comparable to reports of the impact of binge drinking and exercise.

55 l study compares trends in the prevalence of binge drinking and heavy alcohol consumption among pregn

56 trate a pronounced interaction between heavy binge drinking and high PRS, resulting in a relative exc

57 he sera of healthy individuals after alcohol binge drinking and in mice after binge or chronic alcoho

58 ed TLR4 expression in the CeA contributes to binge drinking and may be a key early neuroadaptation in

59 ts govern behavioral sequences that maintain binge drinking and may serve as a circuit-based biomarke

60 tivity to U- and P-threat and probability of binge drinking and number of binging episodes.

61 ratio was related to lower probabilities of binge drinking and of any alcohol use, with a 10-unit in

62 le phases were significantly associated with binge drinking and progesterone-to-estradiol ratio.

63 s study provides a foundation that shows how binge drinking and the oral microbiome dysbiosis lead to

64 The association between binge drinking and total mortality tended to be similar

65 Both future binge-drinking and familial alcoholism were associated w

66 exceeded medical guidelines for acute harm (binge drinking) and chronic harm (heavy drinking).

67 ing patterns (ie, frequency of drinking and "binge" drinking) and consumption at different times of a

68 nts (387 were people with AUD, 38 engaged in binge drinking, and 359 were controls).

69 ded moderate drinking, 2812 (23.8%) reported binge drinking, and 4527 (38.3%) engaged in hazardous dr

70 + binge drinking, 10.5% reported 10+ extreme binge drinking, and 5.6% reported 15+ extreme binge drin

71 eded moderate drinking, 329 (23.4%) reported binge drinking, and 540 (38.4%) engaged in hazardous dri

72 aseline prevalence of heavy weekly drinking, binge drinking, and alcohol-related problems among Reser

73 sed risk of new-onset heavy weekly drinking, binge drinking, and alcohol-related problems.

74 ression, manic-depressive disorder, heavy or binge drinking, and alcohol-related problems.

75 t concentrations seen in blood alcohol after binge drinking, and diminished ALDH1a1 leads to enhanced

76 r three health behaviors: cigarette smoking, binge drinking, and heavy alcohol consumption.

77 xiety disorder), (2) substance use (smoking, binge drinking, and illicit drug use), and (3) domestic

78 , current health insurance coverage, asthma, binge drinking, and physical activity) was not substanti

79 Participants were assessed for drinking, binge drinking, and problem drinking before randomizatio

80 characterized plasma exosomal contents in a binge-drinking animal model and their effect on ALC/APAP

81 OR], 1.69 [95% CI, 1.17-2.44]), the onset of binge drinking (AOR, 1.38 [95% CI, 1.08-1.77]), and the

82 associated with a high-risk trajectory were binge drinking (aOR, 1.69; 95% CI, 1.13-2.54), cigarette

83 CI, 1.6-23.3), and maternal first-trimester binge drinking (aOR, 8.2; 95% CI, 1.9-35.3).

84 Heavy alcohol consumption, particularly binge drinking, appears to be an independent risk factor

85 Repeated bouts of binge drinking ( approximately 1.5 g/kg per 30 min) elev

86 At-risk and binge drinking are frequently reported by middle-aged an

87 Alcohol use disorder (AUD) and binge drinking are highly prevalent public health issues

88 on and increases in heavy episodic drinking (binge drinking) are a growing public concern, due to the

89 lcohol-induced steatosis in a mouse model of binge drinking as acute ethanol (EtOH) intoxication has

90 s caused profound and selective reduction of binge drinking associated with inhibition of alpha2 expr

91 rinking (OR = 1.92, 95% CI: 1.44, 2.56); the binge drinking association remained after adjustment for

92 sters, is superior to usual care in reducing binge drinking at 3 months post discharge.

93 creased MF behavioral score predicted higher binge drinking at age 21 but not its future development.

94 sive symptoms and initiation of drug use and binge drinking at least monthly.

95 nt segment of students also reported extreme binge drinking at levels 2 and 3 times higher.

96 Binge drinking at the traditionally defined 5+ drinking

97 of comorbidity between binge eating (BE) and binge drinking (BD) behaviors, suggesting a common neuro

98 Binge drinking (BD) contributes strongly to the harms of

99 to alcohol cues in individuals who engage in binge drinking (BD) may play a role in motivation to con

100 e importance of assessing multiple levels of binge drinking behavior and their predictors among youth

101 The reduction in binge drinking behavior at 3-month follow-up compared to

102 isodes was 6 +/- 1, and the mean duration of binge drinking behavior was 4 +/- 0.6 years.

103 in PV interneurons (PV:delta(-/-)) increased binge drinking behavior, reduced sensitivity to alcohol-

104 -0.259, SE = 0.132, P = .049) and growth of binge drinking (beta = -0.244, SE = 0.073, P = .001), du

105 = -0.400, SE = 0.179, P = .03) and growth in binge drinking (beta = -0.716, SE = 0.274, P = .009) and

106 tified between genetically predicted GCT and binge drinking (beta, -2.52; 95% CI, -4.13 to -0.91) and

107 Binge drinking (blood-alcohol levels >/= 0.08 g% in a 2-

108 ing after adjustment for weekly consumption, binge drinking, BMI, and smoking.

109 4 siRNA vector (pHSVsiLTLR4a) also inhibited binge drinking, but neither vector functioned when infus

110 Binge drinking, but not frequency of drinking, was assoc

111 rican and less educated were associated with binge drinking, but race/ethnicity and educational level

112 ally significant consequences of their heavy/binge drinking, but they do not appear to be at greater

113 t in the mOFC that serves to protect against binge drinking by reducing alcohol intake, which may off

114 % CI: 0.3, 12.2; p = 0.041), and past-30-day binge drinking, by 3.5 percentage points (95% CI: -0.1,

115 Alcohol use, binge drinking, cannabis use, and vaping nicotine or can

116 t of past 30-day substance use (alcohol use, binge drinking, cannabis use, vaping cannabis, and vapin

117 lf-reported use of any tobacco, any alcohol, binge drinking, cannabis, and any other illegal or misus

118 with one large dose of ethanol, which mimics binge drinking, causes marginal fatty liver and decrease

119 s had very large effect sizes in response to binge drinking (Cohen's d > 1).

120 e of prescription drugs were associated with binge drinking compared with no alcohol use among women.

121 Among male subjects, binge drinking compared with no alcohol use was associat

122 was inversely associated with mortality, but binge drinking completely attenuated this relation.

123 to ethanol at concentrations obtained during binge drinking constricts cerebral arteries in several s

124 Binge drinking (consuming > or =5 alcoholic drinks on 1

125 Binge drinking contributes to an increasing number of em

126 -reported data and measurement invariance in binge drinking cutoffs across study years.

127 showed a notable reduction in mean predicted binge drinking days by 1.2 days (95% CI: [-2.3, -0.3]; p

128 f the intervention in reducing the number of binge drinking days, the trial's primary outcome, in the

129 h a significantly reduced reported number of binge-drinking days (incidence rate ratio [IRR]=0.74, 95

130 nth (IRR=0.69, 95% CI=0.50, 0.97), number of binge-drinking days (IRR=0.67, 95% CI=0.47, 0.95), and a

131 ing (IRR=0.84, 95% CI=0.71, 0.99), number of binge-drinking days (IRR=0.67, 95% CI=0.47, 0.96), and P

132 otionality, which were associated with fewer binge-drinking days and fewer alcohol related problems.

133 inge drinking in the past week; 3) number of binge-drinking days in the past week; and 4) number of d

134 g men (ages 18-29) with children, among whom binge drinking declined.

135 iting AIC->DLS circuitry in male mice during binge drinking decreased alcohol, but not water consumpt

136 We examined national trends in binge drinking, defined as 5 or more drinks in a single

137 Binge drinking, defined as reaching a blood alcohol leve

138 al confounders (age, sex, smoking, heavy and binge drinking, diabetes, use of preventive dental servi

139 Alcohol binge drinking disinhibited pyramidal neurons by augment

140 In turn, greater frequency of binge drinking during follow-up was associated with grea

141 Binge drinking during Oktoberfest did not associate with

142 ants and 45.5% of control infants engaged in binge drinking during pregnancy.

143 19 eligible patients, 250 (94% men) reported binge drinking during the prior year, and a total of 318

144 period (aOR = 2.09, 95% CI: 1.08, 4.03), and binge drinking during the second month of pregnancy (aOR

145 To assess the short-time effects of a binge drinking episode on CB1R availability, 20 healthy

146 , episodes of binge drinking (mean number of binge drinking episodes during previous 30 days decrease

147 with an increased frequency of drinking and binge drinking episodes in adolescents.

148 had a significant reduction in the number of binge drinking episodes within the last 28 days at 12 mo

149 ths, the relative reduction in the number of binge drinking episodes within the last 28 days in the A

150 At 12 months, the number of binge drinking episodes within the last 28 days was sign

151 mary outcome was the self-reported number of binge drinking episodes within the last 28 days, assesse

152 Between 1993 and 2001, the total number of binge-drinking episodes among US adults increased from a

153 hest among those aged 18 to 25 years, 69% of binge-drinking episodes during the study period occurred

154 Per capita binge-drinking episodes have increased, particularly sin

155 Men accounted for 81% of binge-drinking episodes in the study years.

156 Overall, 47% of binge-drinking episodes occurred among otherwise moderat

157 ng drinking limit guidelines in adults or on binge-drinking episodes or social consequences of alcoho

158 .2 billion to 1.5 billion; during this time, binge-drinking episodes per person per year increased by

159 Between 1995 and 2001, binge-drinking episodes per person per year increased by

160 Although rates of binge-drinking episodes were highest among those aged 18

161 state and regional differences in per capita binge-drinking episodes.

162 reinforce evidence-based counseling against binge drinking even at the earliest stages of pregnancy.

163 ke has been associated with lower mortality, binge drinking, even among light drinkers, appears to be

164 Binge drinking, even among otherwise light drinkers, inc

165 development of compulsive drinking across a binge-drinking experience in male mice.

166 ess norms were more strongly associated with binge drinking for women than for men (p(interaction) =

167 -0.176, SE = 0.073, P = .02), and growth in binge drinking frequency (beta = -0.183, SE = 0.092, P =

168 fter alcohol consumption predicted increased binge drinking frequency during follow-up.

169 rinks per week [DPW] in 537 349 individuals, binge drinking frequency in 143 685 individuals, and alc

170 r symptoms over time associated with greater binge-drinking frequency.

171 o enhance physique were more likely to start binge drinking frequently (2.06; 1.58-2.69) and using dr

172 for alcohol consumption (units per week and binge drinking) from Scottish Health Surveys done in 199

173 health insurance, body mass index, smoking, binge drinking, general health status, eyesight worry, a

174 erall, we demonstrate synergistic effects of binge drinking, genetics, and diabetes on ARC, with pote

175 Surprisingly, the frequent binge drinking group had a lower risk of any mental or b

176 hey are associated with reduced frequency of binge drinking >=6 drinks (B(IVW) = -0.198, 95% CI, -0.2

177 the last 3 months, the number of episodes of binge drinking (> or = 6 drinks per occasion), and the C

178 n in young adults with histories of repeated binge drinking (>/= 5 standard drinks in 2 h in men, >/=

179 nking (>2 drinks on a typical drinking day), binge drinking (>=6 drinks on 1 occasion), and hazardous

180 Although binge drinking has been associated with a higher risk of

181 Frequent binge drinking has been linked to heart disease, high bl

182 oncerning for adult morbidity and mortality: binge drinking has increased among both sexes, and heavy

183 Rates of 5+ binge drinking and 10+ extreme binge drinking have declined since 2005, but rates of 15

184 eclined since 2005, but rates of 15+ extreme binge drinking have not significantly declined.

185 Familial alcoholism and adolescent binge-drinking have both been associated with altered wh

186 Despite widespread increases in binge drinking, heavy drinking declined or remained stab

187 onfidence interval (CI): 1.05, 1.55) but not binge drinking; however, social network and individual d

188 year), hazard ratios were increased for ever binge drinking (HR = 1.29, 95% CI: 1.15, 1.45) or blacki

189 inhibit TLR4 or MCP-1 expression nor reduce binge drinking, identifying a neuronal TLR4/MCP-1 signal

190 s 30-44 without children (from 21% reporting binge drinking in 2006 to 42% in 2018); the exception wa

191 suggest that the shell critically regulates binge drinking in both sexes, with shell CP-AMPARs suppo

192 might have more off-target effects) reduced binge drinking in both sexes.

193 The influence of ERs on binge drinking in female mice suggests that treatments f

194 Our data show that acute alcohol binge drinking in healthy volunteers results in increase

195 the escalation of alcohol intake, similar to binge drinking in humans.

196 at 6 g/kg, which has been shown to represent binge drinking in humans.

197 However, the role of binge drinking in increasing myocardial AEA levels, whic

198 silencing of PL SST neurons reduced alcohol binge drinking in males and females, with no effect on s

199 d educational level were not associated with binge drinking in men who reported using alcohol.

200 y be an important pharmacotherapy to address binge drinking in populations with mild to moderate alco

201 inge drinking, and 5.6% reported 15+ extreme binge drinking in the last 2 weeks.

202 d e-cigarette use (measured in days) and any binge drinking in the past 3 months.

203 days (IRR=0.67, 95% CI=0.47, 0.95), and any binge drinking in the past week (IRR=0.79, 95% CI=0.63,

204 number of drinks in the past 30 days; 2) any binge drinking in the past week; 3) number of binge-drin

205 The prevalence of binge drinking in the United States is rising.

206 ugs in the past year in both sexes, and with binge drinking in women.

207 ses than those traditionally associated with binge drinking in young adults.

208 Delineating various levels of binge drinking, including extreme levels, and understand

209 Binge drinking increased for both sexes in nearly all ag

210 These data indicate that binge drinking increases apoptosis and liver injury in o

211 th survey data were analyzed to determine if binge drinking increases the risk of incident major even

212 Adolescent binge drinking increases Toll-like receptor 4 (TLR4), re

213 The findings suggest that frequent binge drinking, independent of average alcohol intake, d

214 the amygdala to examine both idiopathic and binge drinking-induced changes in constitutive PKCepsilo

215 aches to examine the functional relevance of binge drinking-induced changes in glutamate receptors, t

216 The study's primary endpoints were binge-drinking intensity, defined as 1) number of drinks

217 ber needed to treat [NNT]=2), weeks with any binge drinking (IRR=0.83, 95% CI=0.72, 0.96; NNT=7.4), n

218 quency in the study), naltrexone reduced any binge drinking (IRR=0.84, 95% CI=0.71, 0.99), number of

219 Adolescent binge drinking is a major risk factor for psychiatric di

220 Binge drinking is a substantial and growing health probl

221 Binge drinking is associated with disease and death, and

222 Binge drinking is associated with increased risk for cer

223 Teen binge drinking is associated with low frontal white matt

224 Binge drinking is common among adolescents despite mount

225 Binge drinking is common among most strata of US adults,

226 identifying pharmaceutical targets to treat binge drinking is of paramount importance.

227 Binge drinking is rising among aged adults (>65 years of

228 Binge drinking is short-term drinking that achieves bloo

229 Binge drinking is strongly associated with alcohol-impai

230 differences in the estimated prevalences of binge drinking (mail = 20.3%, telephone = 13.1%) or beha

231 Binge drinking may be an early indicator of vulnerabilit

232 Reports indicate that binge drinking may be increasing in the United States.

233 the present findings suggest that adolescent binge drinking may produce sex-specific and long-lasting

234 Binge drinking may reduce telomere length.

235 s for controls; t=4.33; P<.001), episodes of binge drinking (mean number of binge drinking episodes d

236 D hippocampus and following a rat adolescent binge drinking model.

237 ibutes to excessive alcohol consumption in a binge-drinking model.

238 ion of the plasma exosomal CYP2E1 level in a binge drinking murine model.

239 There were no associations for binge drinking; not being in education, employment, or t

240 ake of alcohol, drinks per drinking episode, binge drinking odds, Alcohol Use Disorders Identificatio

241 -1.08]; P = .14) and effects on the risks of binge drinking (odds ratio [OR], 0.84 [99.17% CI, 0.67-1

242 R modulator, LM11A-31, significantly reduced binge drinking of alcohol.

243 Patients reported their usual frequency of binge drinking of beer, wine, and liquor, defined as int

244 Few studies have examined the effect of binge drinking on human fetal growth.

245 he effects of familial alcoholism and future binge-drinking on white matter microstructural developme

246 Adolescent binge drinking or adult dependence induction reduced the

247 ontrast, heavier alcohol consumption such as binge drinking or consuming on average >=3 drinks a day

248 .31 locus oppositely associated with GCT and binge drinking or DPW (FDR = 0.05).

249 4-hour, 2-bottle choice drinking in the dark binge drinking or limited access 2-bottle choice.

250 white matter regions identified where future binge-drinking or familial alcoholism were significantly

251 drinking (OR = 1.20, 95% CI: 1.03, 1.39) and binge drinking (OR = 1.92, 95% CI: 1.44, 2.56); the bing

252 e were no differences in the odds of monthly binge drinking (OR, 0.80 [95% CI, 0.56-1.13]) or hazardo

253 also associated with reduced odds of monthly binge drinking (OR, 0.87, [95% CI, 0.77-0.99]) and hazar

254 3; 95% confidence interval [CI], 1.36-1.96), binge drinking (OR, 1.46; 95% CI, 1.24-1.71), and alcoho

255 unmarried, working long hours, more frequent binge drinking) or longer (e.g., being younger, Mexican

256 assified as nonhazardous drinking, hazardous/binge drinking, or alcohol-related diagnosis.

257 rol group; measured past 30-day alcohol use, binge drinking, or susceptibility to use alcohol among n

258 For binge drinking outcomes only, we saw evidence of interac

259 le diseases); health risks (tobacco smoking, binge drinking, overweight, and anaemia); and social det

260 ed with other risk-taking behaviors, such as binge drinking (P < .001 and P = .006, respectively), un

261 Emerging adults with binge drinking patterns also might be aided by selective

262 were indicators of psychosocial functioning (binge drinking; perceived stress; not being in education

263 ly than alcoholics in the COGA to experience binge drinking, physical fighting, driving while intoxic

264 8]), suicide attempts (PR 1.99 [1.62-2.44]), binge drinking (PR 1.10 [1.01-1.20]), sleep problems (PR

265 l neurons projecting to the brainstem before binge drinking predicted the ultimate emergence of compu

266 use problems (frequent alcohol use, frequent binge drinking, prescription drug misuse, and over-the-c

267 menstrual, follicular, and ovulatory phases (binge drinking probability and odds ratios vs. late lute

268 teal phase, females showed a lower predicted binge drinking probability of 13% and a higher predicted

269 pproaches within the context of two distinct binge drinking procedures, drinking in the dark and sche

270 Lastly, repeated binge drinking produced a female-like phenotype in the m

271 may be useful for curbing and/or preventing binge drinking, protecting vulnerable individuals from p

272 tes (beta = -0.320, SE = 0.145, P = .03) and binge drinking rates (beta = -0.400, SE = 0.179, P = .03

273 Binge drinking rates are highest on college campuses and

274 In follow-up studies we found that binge drinking reduced myelin density in the mPFC in ado

275 While several binge-drinking-related effects persisted throughout adol

276 For all respondents, binge drinking relative to no alcohol use was associated

277 sessed every 6 months) or an interview-based binge drinking score (grams of alcohol/occasion; assesse

278 The best predictors for high binge drinking score were premature responding in the Sx

279 ively associated with the development of the binge drinking score; MF reward prediction error blood o

280 iables, alcohol use (including at-risk use), binge drinking, serious psychological distress, and self

281 the mortality and morbidity associated with binge drinking should be widely adopted, including scree

282 Binge and heavy binge drinking significantly increase the risk of alcoho

283 we found that EWcp(peptidergic) cells reduce binge drinking specifically in female mice.

284 Two fMRI-based models predicted binge drinking status better than chance, corresponding

285 , household income, smoking history, current binge drinking status, and state of residence.

286 ificant heterogeneity by race and ethnicity, binge drinking status, and state residence.

287 GHSR inverse agonist and antagonist reduced binge drinking, suggesting direct actions of ghrelin.

288 These data suggest binge drinking superimposed with advanced age promotes e

289 nalysis method to establish the link between binge drinking, the oral microbiome and AD.

290 crobiome in relation to alcohol misuse- from binge drinking to addiction.

291 Binge drinking unmasked individual differences, revealin

292 re, we found that repeated cycles of alcohol binge drinking via the Drinking-in-the-Dark (DID) model

293 Binge drinking was declared by 28% of drinkers.

294 Binge drinking was found to be the most predictable phen

295 Binge drinking was inhibited by a GABA(A) alpha1 siRNA v

296 For IV estimates, only binge drinking was robust to adjustments for multiple hy

297 The odds ratios associated with binge drinking were elevated but did not demonstrate sig

298 At-risk alcohol use and binge drinking were more frequent among respondents 50 t

299 eased MB behavioral score was protective for binge drinking, while an increased MF behavioral score p

300 of alcohol intake in rats with a history of binge drinking without dependence is currently unknown.