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1 own Obsessive Compulsive Scale (modified for binge eating).
2 ress-induced drug seeking, in stress-induced binge eating.
3 onal psychotherapy further reduced objective binge eating.
4 ing, unhealthy weight-control behaviors, and binge eating.
5 ction, may also be effective in ameliorating binge eating.
6 lthy weight-control behaviors, and engage in binge eating.
7 K1521498, in obese individuals with moderate binge eating.
8  receptors (Sig-1Rs) blocked compulsive-like binge eating.
9 also be applicable to adolescent bulimia and binge eating.
10 nd the propensity to purge in the absence of binge eating.
11 n more weight than do children not reporting binge eating.
12 nce that reward, over metabolic need, drives binge eating.
13  to feed is a key contributor to obesity and binge eating.
14 tify quantitative trait loci associated with binge eating.
15 el pharmacological treatment for compulsive, binge eating.
16 d disorders, eg, substance-use disorders and binge-eating.
17  loss-of-control eating [8.0% (5.1-11.0) for binge eating; 1.6 (-0.1 to 3.3) for loss of control, vs
18  and motivational processing of food, and in binge eating, a behaviour strongly linked to obesity.
19 apist-assisted (33.3%) conditions had higher binge eating abstinence rates than the self-help (17.9%)
20 ment for binge eating disorder led to higher binge eating abstinence rates, greater reductions in bin
21                                        PC1 ('binge-eating'), accounting for 38% of variation, capture
22                                 However, how binge eating affects children's food intake at meals is
23                                              Binge eating afflicts approximately 5% of US adults, tho
24 antly more effective than BWL in eliminating binge eating after 2 years.
25                          Obese subjects with binge eating also have impaired discrimination of subjec
26                               Assessments of binge eating and associated eating disorder psychopathol
27  in the Arab region; and increasing rates of binge eating and bulimia nervosa in Hispanic and Black A
28 treatment experienced greater improvement in binge eating and depression than did patients receiving
29                  NAS DBS was found to reduce binge eating and increased c-Fos levels in this region.
30 uses evolved protective mechanisms including binge eating and increased metabolic efficiency and fat
31 4.5)], eating continuously [1.6% (0.1-3.1)], binge eating and loss-of-control eating [8.0% (5.1-11.0)
32               Secondary endpoints related to binge eating and medical parameters, safety, and treatme
33  was to examine whether dieting would elicit binge eating and mood disturbance in individuals free of
34 petitive responses in psychopathology; e.g., binge eating and opiate or alcohol abuse, disorders in w
35 estigators fear that dieting may precipitate binge eating and other adverse behavioral consequences.
36                                              Binge eating and other forms of disordered eating behavi
37 ore, results suggested that higher levels of binge eating and overeating in males at age 13 y likely
38                Furthermore, higher levels of binge eating and overeating may cause higher BMI in late
39 MI at age 7 y likely causes higher levels of binge eating and overeating, weight and shape concerns,
40  major significant genetic factor underlying binge eating and provide a behavioral paradigm for futur
41 choanalytic psychotherapy (N=34) had stopped binge eating and purging (odds ratio=13.40, 95% confiden
42 ychoanalytic psychotherapy group had stopped binge eating and purging (odds ratio=4.34, 95% CI=1.33-1
43             Thirteen female adolescents with binge eating and purging behaviors (i.e., bulimia nervos
44 purging distinguishes among individuals with binge eating and purging behaviors.
45  who responded with complete abstinence from binge eating and purging to cognitive behavioral therapy
46 vioral therapy with complete abstinence from binge eating and purging.
47  eating pathology, most engaged in recurrent binge eating and purging.
48 sdexamfetamine, SGAs, and topiramate reduced binge eating and related psychopathology, and lisdexamfe
49                                              Binge eating and substance dependence are disorders char
50 cing behavioral symptoms of bulimia nervosa (binge eating and vomiting).
51 ian visits, exhibited a greater reduction in binge eating and vomiting, and had a greater improvement
52 ically significant effects in the context of binge eating and weight regain prevention requires furth
53 ent of binge eating disorder, impacting both binge eating and weight.
54                                Self-reported binge eating and/or purging were used to indicate presen
55 are mainly vagally mediated functions, since binge-eating and vomiting produce intense stimulation of
56                              The decrease in binge-eating and vomiting under ondansetron treatment wa
57 rimary outcome measures were abstinence from binge-eating and vomiting, as assessed by interview at e
58 isition in disorders involving both natural (binge eating) and artificial (methamphetamine) rewards,
59 tary intake, food tolerance, hedonic hunger, binge eating, and gastrointestinal symptoms assessed wit
60 ers (eg, pathological gambling and shopping, binge eating, and hypersexuality), punding (ie, abnormal
61 utative causal factor in chronic overeating, binge eating, and obesity.
62  contribute to the onset of extreme dieting, binge eating, and purging.
63 neural circuitry of restrictive food choice, binge eating, and the contribution of specific serotonin
64  body dissatisfaction, weight preoccupation, binge eating, and the use of compensatory behaviors) wer
65 amen/pallidal responses in obese people with binge eating are sensitive to altered mu-opioid function
66 ustify whether classification of obesity and binge eating as an addictive disorder is merited.
67                                              Binge eating as measured by the Eating Disorder Examinat
68                               Overeating and binge eating assessed via questionnaire every 12 to 24 m
69 led condition also showed more reductions in binge eating at end of treatment and follow-up assessmen
70  than health education at reducing objective binge eating at the 12-mo follow-up (P < 0.05).
71                                              Binge eating (BE) is common in overweight and obese indi
72   Identifying genetic variants that regulate binge eating (BE) is critical for understanding the fact
73                    We assessed the change in binge-eating (BE) behaviors measured as days per week (b
74 eus (PVN) is necessary for the expression of binge-eating behavior in female rats.
75 a primary focus on anorexia nervosa (AN) and binge-eating behavior, and encourages further study of a
76 ats, RXFP3 blockade within the PVN prevented binge-eating behavior.
77  hypothalamic RLN3/RXFP3 signaling regulates binge-eating behavior.SIGNIFICANCE STATEMENT Binge-eatin
78 rette's syndrome, the obsessions of OCD, the binge eating behaviors of bulimia, and the self-starvati
79 r improving glycemic control and suppressing binge eating behaviors.
80 n disordered eating and obesity, emphasizing binge eating, binge eating disorder and food addiction a
81 psychotherapy (IPT) has been shown to reduce binge eating but its long-term impact and time course on
82 order characterized by recurrent episodes of binge eating but without purging behaviors.
83                    In fully adjusted models, binge eating, but not overeating, was associated with in
84      d-Fenfluramine reduced the frequency of binge eating by obese women with binge eating disorder.
85            After the standardized breakfast, binge-eating children reported a shorter satiety duratio
86 y a role in the greater weight gain found in binge-eating children.
87 tional properties of stimuli that elicit the binge eating commonly associated with obesity.
88  8 weeks), lisdexamfetamine responders (</=1 binge eating day per week for 4 consecutive weeks and CG
89                      Change from baseline in binge eating days/week at weeks 11-12 (primary efficacy
90 eatment differences for change from baseline binge eating days/week at weeks 11-12 significantly favo
91 g/day) was superior to placebo in decreasing binge eating days/week from baseline and improving binge
92 reported significant reductions in objective binge-eating days (beta=-0.66, 95% CI=-1.06, -0.25; Cohe
93 derate to severe binge eating disorder (>/=3 binge-eating days per week for 14 days before open-label
94 mary outcome variable, time to relapse (>/=2 binge-eating days per week for 2 consecutive weeks and >
95 tions may promote hallmark bulimic behaviors-binge eating, dietary restriction, and purging.
96 disorder criteria and had moderate to severe binge eating disorder (>/=3 binge-eating days per week f
97  were depression (19% [95% CI, 14%-25%]) and binge eating disorder (17% [95% CI, 13%-21%]).
98 prevalence of eating disorders (EDs) such as binge eating disorder (BED) and bulimia nervosa (BN) amo
99 l survey, the average lifetime prevalence of binge eating disorder (BED) was 2%.
100 dexamfetamine dimesylate (LDX) vs placebo in binge eating disorder (BED) was evaluated in two multice
101 teen obese individuals seeking treatment for binge eating disorder (BED) were compared with 19 non-BE
102  subjects with (n = 30) and without (n = 30) binge eating disorder (BED) were compared with matched h
103 anorexia nervosa (AN), bulimia nervosa (BN), binge eating disorder (BED), and purging disorder (PD).
104 ents the criterion standard for treatment of binge eating disorder (BED), most individuals do not hav
105 ide the United States on the epidemiology of binge eating disorder (BED).
106 IPT) is an effective specialty treatment for binge eating disorder (BED).
107 has documented efficacy for the treatment of binge eating disorder (BED).
108 n phenotypes: pathological gambling (PG) and binge eating disorder (BED).
109                             The Internet and Binge Eating Disorder (INTERBED) study is a prospective,
110 s among relatives with lifetime diagnoses of binge eating disorder (N=131), bulimia nervosa (N=17), a
111 N=4, 0.4%) met criteria for bulimia nervosa; binge eating disorder also was more common among white w
112 f 300 overweight or obese probands (150 with binge eating disorder and 150 with no lifetime eating di
113         A community sample of 162 women with binge eating disorder and 251 healthy and 107 psychiatri
114 d bulimia nervosa and more likely to exhibit binge eating disorder and eating disorder not otherwise
115 ating and obesity, emphasizing binge eating, binge eating disorder and food addiction as useful conce
116 sa and bulimia nervosa; and the inclusion of binge eating disorder as a formal diagnosis.
117 y of research also supports the inclusion of binge eating disorder as a formal diagnosis.
118 port for conceptualizing bulimia nervosa and binge eating disorder as discrete syndromes.
119 s to evaluate topiramate in the treatment of binge eating disorder associated with obesity.
120 ell tolerated in the short-term treatment of binge eating disorder associated with obesity.
121 sorder and 2.9% had partial or full-criteria binge eating disorder but no association with the outcom
122         This study assessed the stability of binge eating disorder in a community sample.
123 ce of anorexia nervosa, bulimia nervosa, and binge eating disorder in a geographically and economical
124 s for anorexia nervosa, bulimia nervosa, and binge eating disorder in adult women are 1.42%, 0.46%, a
125 ciated with an increased risk for developing binge eating disorder in black women and in white women
126   Our findings support a distinct subtype of binge eating disorder in obesity with similarities in ri
127                                              Binge eating disorder is an addiction-like disorder char
128                                              Binge eating disorder is associated with obesity.
129                  These findings suggest that binge eating disorder is at least as chronic as the well
130 led group cognitive-behavioral treatment for binge eating disorder led to higher binge eating abstine
131 dy mass index >=18.5, met criteria for DSM-5 binge eating disorder or bulimia nervosa, had 12 months
132                          White subjects with binge eating disorder reported significantly higher rate
133                                              Binge eating disorder represents a public health problem
134 n the anticipation of rewards, subjects with binge eating disorder show greater risk-taking, similar
135 ere significantly higher in white women with binge eating disorder than in matched psychiatric compar
136 ere significantly higher in black women with binge eating disorder than in psychiatric comparison sub
137  was to compare three types of treatment for binge eating disorder to determine the relative efficacy
138                The mean lifetime duration of binge eating disorder was 14.4 years (SD=13.9), signific
139                       Neither depression nor binge eating disorder was consistently associated with d
140         A total of 259 adults diagnosed with binge eating disorder were randomly assigned to 20 weeks
141 icipants (N=304) who met DSM-IV criteria for binge eating disorder were randomly assigned to 24 weeks
142          Thirty-four outpatients with DSM-IV binge eating disorder were randomly assigned to receive
143                         Anorexia nervosa and binge eating disorder were relatively uncommon.
144 l, 61 outpatients (53 women, eight men) with binge eating disorder who were obese (body mass index >/
145 exia nervosa, 0.12%; bulimia nervosa, 0.08%; binge eating disorder, 0.42%).
146 ies reporting pharmacological treatments for binge eating disorder, advances in treatment for adults
147 ries are: anorexia nervosa, bulimia nervosa, binge eating disorder, and eating disorder not otherwise
148  systems: anorexia nervosa, bulimia nervosa, binge eating disorder, avoidant-restrictive food intake
149 or attention deficit hyperactivity disorder, binge eating disorder, cocaine addiction, obesity, and t
150 trol disorders, including gambling disorder, binge eating disorder, compulsive sexual behaviour, and
151 sibutramine is effective in the treatment of binge eating disorder, impacting both binge eating and w
152 gnosis of anorexia nervosa, bulimia nervosa, binge eating disorder, or eating disorder not otherwise
153                          In black women with binge eating disorder, rates of sexual abuse, physical a
154 pled with a lack of control over eating, and binge eating disorder, the Diagnostic and Statistical Ma
155 tablished treatments for bulimia nervosa and binge eating disorder, with stepped-care approaches show
156 stigation towards pharmacologically treating binge eating disorder.
157 trial to test the efficacy of sibutramine in binge eating disorder.
158 e in risk is specific for the development of binge eating disorder.
159  binge intake of high-fat food (HFF) seen in binge eating disorder.
160 al abuse and discrimination) in the risk for binge eating disorder.
161 ctive and well tolerated in the treatment of binge eating disorder.
162 e efficacy of sertraline in the treatment of binge eating disorder.
163 jor eating disorders and a related syndrome, Binge Eating Disorder.
164 se female patients meeting full criteria for binge eating disorder.
165 ppressant d-fenfluramine in the treatment of binge eating disorder.
166 requency of binge eating by obese women with binge eating disorder.
167 rgery patients-in particular, depression and binge eating disorder.
168                                              Binge-eating disorder (BED) is characterized by recurrin
169                                              Binge-eating disorder (BED), a public health problem ass
170 l or food, in alcohol use disorders (AUD) or binge-eating disorder (BED), suggest a disturbance in ex
171                                              Binge-eating disorder (BED)-a syndrome that only recentl
172                                   Women with binge-eating disorder (BED; n = 38) and age- and weight-
173 ade for atypical eating disorders except for binge-eating disorder (cognitive behavioural therapy was
174 rvosa (n = 13), bulimia nervosa (n = 6), and binge-eating disorder (n = 1), published between January
175                                              Binge-eating disorder aggregated strongly in families in
176 5 and 5 y of follow-up, 134 individuals with binge-eating disorder and 134 individuals with no histor
177 o assess longitudinally the relation between binge-eating disorder and components of the metabolic sy
178               The best treatment options for binge-eating disorder are unclear.
179                                   Women with binge-eating disorder before and during pregnancy had hi
180 a before and during pregnancy and those with binge-eating disorder before pregnancy exhibit dietary p
181                 Eligible adults met DSM-IV-R binge-eating disorder criteria and had moderate to sever
182 up intakes of women with bulimia nervosa and binge-eating disorder during pregnancy and compared thes
183                          Women with incident binge-eating disorder during pregnancy had higher intake
184 comparison of individuals with and without a binge-eating disorder in analyses adjusted for age, sex,
185                                              Binge-eating disorder is a familial disorder caused in p
186                                              Binge-eating disorder is a newly described eating disord
187                                              Binge-eating disorder is characterized by excessive, unc
188 binge-eating behavior.SIGNIFICANCE STATEMENT Binge-eating disorder is the most common eating disorder
189                                              Binge-eating disorder is the most common eating disorder
190                                              Binge-eating disorder may confer a risk of components of
191                                              Binge-eating disorder may represent a risk factor for th
192 n circuits and neurotransmitters involved in binge-eating disorder pathology and identify RXFP3 as a
193 on across the genome of anorexia nervosa and binge-eating disorder patients.
194 es of anorexia nervosa, bulimia nervosa, and binge-eating disorder were 0.3%, 0.9%, and 1.6%, respect
195 -five outpatients with a DSM-IV diagnosis of binge-eating disorder were randomly assigned to receive
196   Among a referred sample of 207 adults with binge-eating disorder, 187 participants (160 females [85
197 ther eating disorders (OED: bulimia nervosa, binge-eating disorder, and eating disorder not otherwise
198 ding eating disorders such as overeating and binge-eating disorder, but the brain structural mechanis
199 ic substrate for observed sex differences in binge-eating disorder.
200 o time did any participant meet criteria for binge-eating disorder.
201 ns of anorexia nervosa, bulimia nervosa, and binge-eating disorder.
202 ns of anorexia nervosa, bulimia nervosa, and binge-eating disorder.
203 t outcome measures in the acute treatment of binge-eating disorder.
204  efficacy of fluvoxamine in the treatment of binge-eating disorder.
205 f efficacy in adults with moderate to severe binge-eating disorder.
206 and topiramate reduced weight in adults with binge-eating disorder.
207 he characteristic symptomatology observed in binge-eating disorder.
208 as a potential pharmacological treatment for binge-eating disorder.
209 ncts to psychotherapy for bulimia nervosa or binge-eating disorder; in the case of anorexia nervosa,
210 derstanding of the etiology and treatment of binge eating disorders.
211 ty of children with and without a history of binge eating during buffet meals.
212  (eg, more sedentary time, eating fast food, binge eating, eating continuously, not weighing oneself
213 ference in the number of days with objective binge eating episodes (OBEs) during the previous 28 days
214   Within the patient group, the frequency of binge eating episodes during the 4 weeks prior to the st
215 frequency of vomiting episodes, frequency of binge eating episodes, Clinical Global Impression severi
216 y were categorized into those reporting past binge-eating episodes (n = 10) and those reporting no su
217 e primary outcome measures were frequency of binge eating, expressed as log ([binges/week]+1), and Cl
218 of the variance in eating disorder symptoms: binge eating, fear of fatness/compensatory behaviors, an
219 ting abstinence rates, greater reductions in binge eating frequency, and lower attrition compared to
220 ramate also increased abstinence and reduced binge-eating frequency and related psychopathology.
221 tion among patients sustaining recovery from binge eating from posttreatment to 1-year follow-up.
222              Children who report episodes of binge eating gain more weight than do children not repor
223 ic capacity, perhaps resulting from repeated binge eating, gives rise to delayed gastric emptying and
224    After the overnight fast, children in the binge-eating group consumed more energy [x (+/-SD): 1748
225  min; P = 0.03) than did children in the non-binge-eating group.
226  the anticipation of losses, obesity without binge eating had a similar pattern to other substance-us
227                                  We assessed binge eating in closely related C57BL/6 mouse substrains
228  Cyfip2 as a major genetic factor underlying binge eating in heterozygous knockout mice on a C57BL/6N
229 ve both resulted in short-term reductions in binge eating in obese patients with BED.
230 pin-releasing factor (CRF) in stress-induced binge eating in our model.
231 e of CRF receptors in BNST in stress-induced binge eating in our rat model.
232 n-active antidepressant medications decrease binge eating in patients with bulimia nervosa has fueled
233 (12-41) decreased frustration stress-induced binge eating in rats with a history of food restriction.
234                               No episodes of binge eating in the aspiration therapy group or serious
235  regression analysis showed that the rate of binge eating in the d-fenfluramine group fell three time
236                      The median frequency of binge eating in the previous 28 days declined from 22 to
237                       Finally, in a model of binge-eating in female rats, RXFP3 blockade within the P
238                                              Binge eating increased slightly through follow-up but re
239 t pills, laxatives, or diuretics, engaged in binge eating, induced vomiting, or exercised excessively
240                                              Binge eating is a highly heritable trait associated with
241                                              Binge eating is associated with obesity and has been con
242       The authors tested the hypothesis that binge eating is mediated by changes in opioid control of
243  marijuana and other drug use, we found that binge eating is uniquely predictive of incident overweig
244 nd although butorphanol did not trigger chow binge eating, it enhanced binge eating of palatable food
245 lso showed flexibility in foraging patterns, binge-eating less and using feeders more when they exper
246                                          The binge eating model was characterized by four temporally
247                      We developed recently a binge-eating model in which female rats with a history o
248 e opioid system and food-related behavior in binge-eating obese individuals, these results support a
249 antagonist GSK1521498 on eating behaviour in binge-eating obese subjects.
250 ricted mice showed a significant increase in binge eating of a palatable high-fat food during stress
251 testing the ability of butorphanol to elicit binge eating of chow when palatable food was absent.
252                                  Compulsive, binge eating of highly palatable food constitutes a core
253 d not trigger chow binge eating, it enhanced binge eating of palatable food.
254 %, however, continued to engage in recurrent binge eating or purging behaviors (incidence rate, 0.026
255                                   Infrequent binge eating or purging or overeating without a loss of
256 rom bulimia nervosa (they had no episodes of binge eating or purging, were at normal weight, and had
257 weight and regular menstrual cycles, without binge eating or purging.
258 ation of food intake without the presence of binge-eating or purging behavior.
259 rts to integrate these models by focusing on binge eating phenotypes as the subgroup of obese individ
260  drink frequently, while both overeating and binge eating predicted starting to use marijuana and oth
261 mpulsive behaviours included hypersexuality, binge eating, punding, compulsive use of dopamine replac
262 ould yield greater reductions in symptoms of binge eating, purging, and eating disorders compared wit
263 han half crossed between the restricting and binge eating/purging anorexia nervosa subtypes over time
264                                          The binge eating/purging group showed significantly greater
265  (i.e., bulimia nervosa or anorexia nervosa, binge eating/purging type);14 with anorexia nervosa, res
266                                              Binge-eating recovery rates were equivalent for CBT and
267                                              Binge eating, recurrent and persistent episodes of overe
268                                      Risk of binge-eating relapse over 6 months was lower in particip
269 eating days/week from baseline and improving binge eating-related key secondary endpoints.
270 GAs (MD, -3.84 [CI, -6.55 to -1.13]) reduced binge-eating-related obsessions and compulsions, and SGA
271 etrograde endocannabinoid signaling, whereas binge eating resulted in the downregulation of a gene se
272 lts with body mass index >/= 30 kg m(-2) and binge eating scale scores >/= 19 received 1-week single-
273 olerance questionnaire, Power of food scale, Binge eating scale, and Gastrointestinal symptom rating
274 estionnaires: Yale Food Addiction Scale 2.0, Binge Eating Scale, The PTSD Checklist for DSM-5, Life E
275 acebo in its effects on weight, fat mass and binge eating scores.
276 r monetary incentives linked alcohol use and binge eating severity.
277 x correlated negatively with alcohol use and binge eating severity.
278  including the percentage of abstinence from binge eating (sibutramine group: 58.7%; placebo group: 4
279 ed lower energy consumption at baseline, and binge-eating status was associated with greater energy c
280 ome to inform molecular mechanisms mediating binge eating susceptibility and establishment.
281 d obsessional processes that accompany human binge-eating syndromes is discussed.
282 nd CBTgsh resulted in greater remission from binge eating than BWL (P < .05; odds ratios: BWL vs CBTg
283 ic food cravings, compulsive overeating, and binge eating that may represent a phenotype of obesity.
284  in the R + S rats with naloxone suppressing binge eating to control levels, and although butorphanol
285  reward characterizes disorders ranging from binge eating to drug addiction.
286 on a C57BL/6N background that showed reduced binge eating toward a wild-type C57BL/6J-like level.
287                       Neither overeating nor binge eating was associated with starting to binge drink
288 arge cohort of adolescents and young adults, binge eating was more common among females than males.
289 (e.g., locating food) and maladaptive (e.g., binge eating) ways.
290                             Using a model of binge-eating, we demonstrated that relaxin-3/RXFP3 signa
291 ated the efficacy of sibutramine in reducing binge eating, weight, and associated psychopathology.
292  28, significantly more (P < 0.003) cases of binge eating were observed in MR participants than in th
293 tations, including starvation resistance and binge eating when food becomes available.
294     The authors developed an animal model of binge eating where history of caloric restriction with f
295                   The neuronal substrate for binge eating, which can at times lead to obesity, is not
296 licated in reward-seeking behaviors, such as binge eating, which contributes to treatment resistance
297      Furthermore, compulsive food intake and binge eating will be considered from an evolutionary per
298 ted with a smartphone app, Noom Monitor, for binge eating with or without purging.
299  of longer duration, or by not eating, or by binge-eating without vomiting.
300  a rodent model to test whether a history of binge eating would augment subsequent responding for coc
301         A non-treatment-seeking sample of 90 binge eating young adults were evenly randomised to unde

 
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