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1 fate, genistein 7-sulfate, or genistein from biochanin A.
2 cubated with [4-(14)C]genistein and [4-(14)C]biochanin A.
3 cell lines by the isoflavones genistein and biochanin A.
4 trations of the isoflavones formononetin and biochanin A.
6 orogenic acid (15.4 +/- 0.05 mug mg(-1)) and biochanin A (9.6 +/- 0.06 mug GAE mg(-1)), while minor c
7 ein, genistein, glycitein, formononetin, and biochanin-A and their mammalian metabolites equol and O-
8 istin, daidzein, genistein, formononetin and biochanin A) and 3 lignans (secoisolariciresinol, matair
9 flavonoids, isoflavonoids (formononetin and biochanin A) and flavones (7,4'-dihydroxyflavone), respe
10 flavones (genistein, genistin, daidzein, and biochanin A) and soy phytochemical concentrate exhibit d
11 flavonoids and isoflavones such as daidzein, biochanin A, and 7,3'-dimethoxy-5,6,4' trihydroxyisoflav
12 isoflavones (genistein, daidzein, glycitein, biochanin A, and formononetin), lignans (secoisolaricire
14 ch are aglycons, namely daidzein, genistein, biochanin A, and two of which, daidzin and genistin, are
15 oybean isoflavones (genistein, daidzein, and biochanin A) are ERbeta-selective agonists of transcript
16 From a panel of phytoestrogen isoflavones, biochanin A (BCA) was identified as the most potent indu
17 ERRalpha agonists and safe food supplements biochanin A, daidzein or genistein, each rescued the hyp
18 n (genistein 7-sulfate) and 2 metabolites of biochanin A (genistein and genistein 7-sulfate) were det
21 s of the isoflavones daidzein, formononetin, biochanin A, genistein, and equol were studied under sim
23 timulated release of PSA, presumably because biochanin A increased UDPGT and increased the intracellu
27 nap2 mutants and show that the phytoestrogen biochanin A specifically reverses the mutant behavioral
28 f genistein 7-sulfate from genistein or with biochanin A sulfate, genistein 7-sulfate, or genistein f
30 avonoid metabolites such as formononetin and biochanin-A that peaked at 12 to 18 h following elicitat