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1 fate, genistein 7-sulfate, or genistein from biochanin A.
2 cubated with [4-(14)C]genistein and [4-(14)C]biochanin A.
3  cell lines by the isoflavones genistein and biochanin A.
4 trations of the isoflavones formononetin and biochanin A.
5                                Glucosides of biochanin A (4'-O-methylgenistein) and pratensein (3'-hy
6 orogenic acid (15.4 +/- 0.05 mug mg(-1)) and biochanin A (9.6 +/- 0.06 mug GAE mg(-1)), while minor c
7 ein, genistein, glycitein, formononetin, and biochanin-A and their mammalian metabolites equol and O-
8 istin, daidzein, genistein, formononetin and biochanin A) and 3 lignans (secoisolariciresinol, matair
9  flavonoids, isoflavonoids (formononetin and biochanin A) and flavones (7,4'-dihydroxyflavone), respe
10 flavones (genistein, genistin, daidzein, and biochanin A) and soy phytochemical concentrate exhibit d
11 flavonoids and isoflavones such as daidzein, biochanin A, and 7,3'-dimethoxy-5,6,4' trihydroxyisoflav
12 isoflavones (genistein, daidzein, glycitein, biochanin A, and formononetin), lignans (secoisolaricire
13 ding the flavonol kaempferol, the isoflavone biochanin A, and the chalcone isoliquiritigenin.
14 ch are aglycons, namely daidzein, genistein, biochanin A, and two of which, daidzin and genistin, are
15 oybean isoflavones (genistein, daidzein, and biochanin A) are ERbeta-selective agonists of transcript
16   From a panel of phytoestrogen isoflavones, biochanin A (BCA) was identified as the most potent indu
17  ERRalpha agonists and safe food supplements biochanin A, daidzein or genistein, each rescued the hyp
18 n (genistein 7-sulfate) and 2 metabolites of biochanin A (genistein and genistein 7-sulfate) were det
19                 Environmental half-lives for biochanin A, genistein, and equol are expected to vary o
20                                              Biochanin A, genistein, and equol degraded relatively sl
21 s of the isoflavones daidzein, formononetin, biochanin A, genistein, and equol were studied under sim
22 n, isoformononetin glycoside and malonylated biochanin A glycoside the major compounds.
23 timulated release of PSA, presumably because biochanin A increased UDPGT and increased the intracellu
24                                Additionally, biochanin A markedly decreased prostate specific antigen
25                    Intact cells treated with biochanin A produced testosterone-glucuronide from testo
26                                              Biochanin A significantly decreased the testosterone-sti
27 nap2 mutants and show that the phytoestrogen biochanin A specifically reverses the mutant behavioral
28 f genistein 7-sulfate from genistein or with biochanin A sulfate, genistein 7-sulfate, or genistein f
29 ted methylated form of each isoflavone and a biochanin A sulfate.
30 avonoid metabolites such as formononetin and biochanin-A that peaked at 12 to 18 h following elicitat
31 expressed in LNCaP cells and was enhanced in biochanin A-treated LNCaP cells.
32                     Of the compounds tested, biochanin A was the most potent, with increased activity
33                              Neoponcirin and biochanin A were identified for the first time in the Me
34                                  Chrysin and biochanin A were the most potent BCRP inhibitors, produc