ライフサイエンスコーパス: bioenergetic

1 lities that could be attributed to astrocyte bioenergetics.

2 etabolic feedback circuits and mitochondrial bioenergetics.

3 additional processes in the brain, including bioenergetics.

4 st into oxidative phosphorylation and muscle bioenergetics.

5 activates autophagy, while the other targets bioenergetics.

6 omplicated, which is also reflected in their bioenergetics.

7 ytosolic signaling events with mitochondrial bioenergetics.

8 stress (mPOS) in the cytosol independent of bioenergetics.

9 ulting iPSCs and ESCs, suggesting comparable bioenergetics.

10 ramming of lipid metabolism and mitochondria bioenergetics.

11 checkpoint for mitochondrial biogenesis and bioenergetics.

12 utonomous clocks in the timing of organismal bioenergetics.

13 uated if 2-AI compounds affect mycobacterial bioenergetics.

14 h age-related differences in skeletal muscle bioenergetics.

15 standing and interpretation of intracellular bioenergetics.

16 es astrocytes and neurons and supports brain bioenergetics.

17 tive phosphorylation, known as mitochondrial bioenergetics.

18 es, and downregulation of genes important in bioenergetics.

19 cellular and organellar functions including bioenergetics.

20 L expression, which plays a role in cellular bioenergetics.

21 hronize organismal food intake with cellular bioenergetics.

22 lasmic reticulum and mitochondria to sustain bioenergetics.

23 ntial for functions apart from mitochondrial bioenergetics.

24 and plays an important role in mitochondrial bioenergetics.

25 t alter mitochondrial membrane potential and bioenergetics.

26 of disrupted mitochondrial architecture and bioenergetics.

27 ent kinase II activation and altered myocyte bioenergetics.

28 fusion and to maintain optimal mitochondrial bioenergetics.

29 Brain bioenergetic abnormalities have been observed frequently

30 Previous studies have shown muscle bioenergetic abnormalities in PBC, including increased m

31 esis that fatigue in PBC is driven by muscle bioenergetic abnormality related to AMA, and that AMA re

32 ER-mediated mitochondrial Ca(2+) signaling, bioenergetic activation, and mitochondrial density.

33 namics of chronological and endocrinological bioenergetic aging in female brain.

34 eurons, in addition to their primary role in bioenergetics, also contribute to specialized functions,

35 Bioenergetic analysis showed that ZIKV infection evokes

36 Using transcriptomics and bioenergetic analysis, we discovered that DF induces gly

37 The bioenergetics analysis identified a moderate, direct imp

38 r cells frequently utilize glutamine to meet bioenergetic and biosynthetic demands of rapid cell grow

39 e of responding to and meeting the increased bioenergetic and biosynthetic demands placed on it.

40 tissue-damaged environment have significant bioenergetic and biosynthetic needs.

41 ino acid transport is pivotal for subsequent bioenergetic and biosynthetic programs and licences T ce

42 manipulates the host mitochondria to support bioenergetic and biosynthetic requirements for replicati

43 ions in lipid metabolic pathways to meet the bioenergetic and biosynthetic requirements is a principa

44 een hyper- and normo-glycaemic conditions on bioenergetic and functional parameters.

45 mechanisms in B cells with implications for bioenergetic and metabolic pathways that control cellula

46 d translation led to the hypothesis that the bioenergetic and synthetic demands of cell growth were p

47 d amino acids to levels that exceed a cell's bioenergetic and synthetic needs has been documented in

48 omponent ion circuit for [Formula: see text] bioenergetics and a 2nd 2-component ion circuit for Na(+

49 in resistance causes alterations in cellular bioenergetics and activation of inflammatory signaling i

50 ction and has emerged as central hub between bioenergetics and all major cellular events.

51 st that PINK1 is critical for modulating the bioenergetics and antioxidant responses in PBMCs whereas

52 letal dysplasia by interfering with cellular bioenergetics and biosynthesis.

53 to researchers working in the field of brain bioenergetics and brain diseases.

54 electron- and proton-transport reactions in bioenergetics and catalysis in general.

55 Bioenergetics and cell survival were instead unaffected

56 ng a tool to better understand mycobacterial bioenergetics and develop compounds with improved anti-m

57 t mitochondrial energy metabolism, measuring bioenergetics and enzyme activities of the electron tran

58 glucose deprivation stimulate mitochondrial bioenergetics and formation of respiratory supercomplexe

59 We also assessed cellular bioenergetics and generation of reactive oxygen species

60 d complex V, resulting in impaired oxidative bioenergetics and heightened ROS production.

61 lecular oxygen (O(2)) sustains intracellular bioenergetics and is consumed by numerous biochemical re

62 )S pathway sustain endothelial mitochondrial bioenergetics and loss of CSE increases the production o

63 tabolic reprograming in order to provide the bioenergetics and macromolecular precursors needed to su

64 plexes in mitochondrial membranes to support bioenergetics and maintain mitochondrial architecture.

65 cular oxygen (O(2)) plays important roles in bioenergetics and metabolism and is implicated in bioche

66 A comparison of bioenergetics and mitochondrial function between isogeni

67 linked to dysregulation of the mitochondrial bioenergetics and oxidative status.

68 ed Mn(2+) ions may have played a key role in bioenergetics and possibly facilitated early geological

69 lore more effective therapies, mitochondrial bioenergetics and redox homeostasis were assessed in VLC

70 l electron transport chain, which compromise bioenergetics and suggest a mechanism by which ET-1 prom

71 l, our results provide insights into mitotic bioenergetics and suggest that cell division is not a hi

72 itical role of TRPM2 in gastric cancer cells bioenergetics and survival; however, its role in gastric

73 hat couple mitochondrial fusion/fission with bioenergetics and their impacts on neurodegeneration how

74 herefore, IDH2 is a dual regulator of cancer bioenergetics and tumor cell motility.

75 e for quantitative proteomics, mitochondrial bioenergetics and tumor growth in mice were conducted.

76 ual roles of mitochondria in ATP production (bioenergetics) and apoptosis (cell life/death decision)

77 expressing several naive-like developmental, bioenergetic, and epigenomic features despite providing

78 yntrophic conversions by connecting kinetic, bioenergetic, and physiological effects.

79 drial matrix uptake of Ca(2+), mitochondrial bioenergetics, and autophagic flux.

80 ake, a process crucial for Ca(2+) signaling, bioenergetics, and cell death.

81 etabolomics and focused metabolite analyses, bioenergetics, and cell viability assays, we show that t

82 ondrial morphology, attenuates mitochondrial bioenergetics, and induces mitochondrial DNA oxidative i

83 is work resolves a long-standing question in bioenergetics, and renders a chemical-biological basis f

84 Ca(2+) uptake by mitochondria regulates bioenergetics, apoptosis, and Ca(2+) signaling.

85 g the integrity of the genome and sustaining bioenergetics are both fundamental functions of the cell

86 rgeting mitochondria protection and cellular bioenergetics are presented, with emphasis on those that

87 ondria, long viewed solely in the context of bioenergetics, are increasingly emerging as critical hub

88 The enhancement of mitochondrial bioenergetics as well as the increase in mitochondrial p

89 y the sub pathways involved in mitochondrial bioenergetics, as observed in other neurodegenerative di

90 Bioenergetics aspects of the 3B model are further evalua

91 Using a combination of real-time bioenergetics assays and metabolomics approaches, we inv

92 fibroblasts showed overactive mitochondrial bioenergetics associated with atypical morphology and al

93 ctral exponent of local field potentials and bioenergetics based on the activity of mitochondrial Cyt

94 n fatigability during dynamic exercise has a bioenergetic basis and is explained by an increased accu

95 gene expression signature, the mitochondrial bioenergetics, biogenesis and fuel catabolic functions a

96 cellular plasticity by sustaining oxidative bioenergetics, buffering ROS production, and supporting

97 ed to occur independently of follicular bulb bioenergetics by a tractor mechanism involving the inner

98 llenges oxidative stress imposes on membrane bioenergetics by shifting redox balance to glycolysis an

99 AMs) are central microdomains that fine-tune bioenergetics by the local transfer of calcium from the

100 ier remains a key uncertainty underlying the bioenergetics calculations.

101 tilization, and alterations in mitochondrial bioenergetics can additionally propel stem cell deficits

102 ment, including local synaptic E-I ratio and bioenergetics, can be modeled by cerebral organoids (CO)

103 s overall flexibility of ATP supply; and the bioenergetic capacity quantifies the maximum rate of tot

104 reater rates of oxidative metabolism, higher bioenergetic capacity, differential use of pyruvate, and

105 ar proliferation as well as increased global bioenergetic capacity.

106 of all mammalian tissues, where it regulates bioenergetics, cell death, and Ca(2+) signal transductio

107 st that lifestyles that include intermittent bioenergetic challenges, most notably exercise and dieta

108 ry profiles, which were also associated with bioenergetic changes and induction of a senescent-like p

109 in suPAR-overexpressing mice and normalized bioenergetic changes in HK-2 cells.

110 to acute bacterial infection and results in bioenergetic changes which underpin emergency granulopoi

111 e changes in extracellular glucose than were bioenergetic changes.

112 15i triggers oxidative stress which induces bioenergetic collapse and apoptosis of the parasite by d

113 Initiating bioenergetic collapse and cell death, it has been implic

114 ds to stabilization of PINK1, in response to bioenergetic collapse.

115 The resultant bioenergetic compromise blocked proliferation by limitin

116 These changes released eukaryotes from the bioenergetic constraints on prokaryotes, facilitating th

117 emodelling of eukaryotic proteomes, and that bioenergetic constraints selected for temporal organisat

118 to stress, yet whether changes to astrocyte bioenergetic control of synapses contributes to stress-i

119 In pancreatic beta-cells, mitochondrial bioenergetics control glucose-stimulated insulin secreti

120 How TGFbeta supports the bioenergetic cost of matrix protein synthesis is not ful

121 timately induced mitochondrial metabolic and bioenergetic crises in the rat stomach, indicated by com

122 homeostatic circuit that protects cells from bioenergetic crisis and mitochondrial Ca(2+) overload du

123 os) have been presumed to trigger an overall bioenergetic crisis in the neuron, resulting in cell dea

124 rgistically with vemurafenib in effectuating bioenergetic crisis, DNA damage and cell death selective

125 oma cells through induction of mitochondrial bioenergetic crisis.

126 otein kinase (AMPK) is a master regulator of bioenergetics crucial for glucose metabolism in acute my

127 PERK activation is sufficient to rescue bioenergetic defects caused by complex I missense mutati

128 omplexes, thereby likely contributing to the bioenergetic defects characteristic of AD.

129 Converging evidence suggests bioenergetic defects contribute to the pathophysiology o

130 The retention of bioenergetic defects in cultured cells indicates that th

131 sosomal acidity is sufficient to restore the bioenergetic defects.

132 fragmentation and mtDNA lesion, and reduces bioenergetic deficits and cell death in HD mouse- and pa

133 perfusion, termed extreme fusion, leading to bioenergetic deficits.

134 glucose and glutamine carbons to support the bioenergetic demand of translation.

135 ing, structural remodeling, and considerable bioenergetic demand.

136 rough the intermembrane space to support the bioenergetic demands of phasic DA release.

137 eir "neighbors" to maintain biosynthetic and bioenergetic demands while escaping immunosurveillance o

138 ssue of the JCI, Mandarano et al. illuminate bioenergetic derangements of ME/CFS T cell subsets.

139 ient-reported outcomes and immunological and bioenergetics disease parameters.

140 Despite evidence of bioenergetic disruption within heart mitochondria, there

141 is known about the metabolic requirement and bioenergetics during osteoclastogenesis.

142 cterize cellular derangements, mitochondrial bioenergetics, dynamics, endoplasmic reticulum (ER)-mito

143 This study provides in vivo evidence for bioenergetic dysfunction in ALS in brain and skeletal mu

144 The consistency of cellular bioenergetic dysfunction in different cell types support

145 s build upon the body of evidence supporting bioenergetic dysfunction in schizophrenia, and suggests

146 t transcriptional signature of mitochondrial bioenergetic dysfunction in skeletal muscle, with low PG

147 isease models and conclusive data to support bioenergetic dysfunction in vivo in patients is currentl

148 nd fatty acids normally, indicating that the bioenergetic dysfunction lies upstream of the TCA cycle.

149 al tool for future clinical trials targeting bioenergetic dysfunction.

150 esults in a gene signature characteristic of bioenergetic dysfunction.

151 as seen, potentially linking AMA with muscle bioenergetics dysfunction; however, this was not related

152 c systems of biology that were paralleled by bioenergetic dysregulation in midlife aging female brain

153 ss involved in different aspects of cellular bioenergetics; dysregulation of lipid oxidation is often

154 his process is called excitation-contraction-bioenergetics (ECB) coupling.

155 to use kynurenine metabolism to increase the bioenergetic efficiency of glucose oxidation.

156 in prostate cancer cells impaired oxidative bioenergetics, elevated reactive oxygen species (ROS) pr

157 Finally, AP39, a modulator of mitochondrial bioenergetics enhanced cytochrome c oxidase activity, re

158 es indicate that insulin influences cerebral bioenergetics, enhances synaptic viability and dendritic

159 ally fluctuate in response to changes in the bioenergetic environment, regulation of mRNA structure b

160 Mechanistically, bioenergetics experiments revealed that PF reduces mitoc

161 This bioenergetic failure is thought to underlie pathologies

162 Mitochondrial fragmentation and bioenergetic failure manifest in Huntington's disease (H

163 immune response, metabolic derangements and bioenergetic failure.

164 Live-cell bioenergetic flux analysis confirmed that mensacarcin di

165 criptomic and metabolomic systems underlying bioenergetic function in brain and its relationship to p

166 pport a model that includes changes in brain bioenergetic function in subjects with major depression.

167 he absence of obesity, it is unclear whether bioenergetic function in the subpopulations of mitochond

168 nts and healthy controls to look at cellular bioenergetic function in whole cells.

169 These results suggest abnormal bioenergetic function, as well as a neuron-specific defe

170 her we identify a critical role for BUD23 in bioenergetics gene expression, by promoting efficient tr

171 rylation are rheostats in immune cells whose bioenergetics have functional outputs in terms of their

172 lize nutrients to produce the ATP needed for bioenergetic homeostasis has been well-characterized.

173 ardiomyocyte-specific gene program governing bioenergetic homeostasis in the adult heart.

174 ndria play a fundamental role in maintaining bioenergetic homeostasis of both OXPHOS-competent and OX

175 ays a critical role in energy metabolism and bioenergetic homeostasis.

176 Mitochondria are well established as bioenergetic hubs for generating ATP but have also been

177 Body-wide changes in bioenergetics, i.e., energy metabolism, occur in normal

178 novel mutation in SPART leads to a profound bioenergetic imbalance.

179 rather than respiratory chain defects in the bioenergetic impacts of tafazzin deficiency.

180 A distinct bioenergetic impairment of heart mitochondrial subpopula

181 and a 2nd 2-component ion circuit for Na(+) bioenergetics in a strictly anaerobic rumen bacterium.

182 Here, we studied bioenergetics in an induced pluripotent stem cell (iPSC)

183 tworks indicates a broader role of astrocyte bioenergetics in determining how experience-dependent in

184 enic response and restored the mitochondrial bioenergetics in endothelial cells.

185 tive phosphorylation, inhibits mitochondrial bioenergetics in lung cancer cells and mitigates lung ca

186 es in autophagy, mitochondrial dynamics, and bioenergetics in mouse models of acute and chronic Dox-c

187 of evidence suggests abnormalities in brain bioenergetics in psychiatric disorders, including both b

188 al carbon metabolism fluxes and central cell bioenergetics in response to ammonium availability and n

189 The capacity of cells to alter bioenergetics in response to the demands of various biol

190 Restoring mitochondrial bioenergetics in the colonic epithelium with 5-amino sal

191 esembling pre-IBD and impaired mitochondrial bioenergetics in the colonic epithelium, which triggered

192 henotypes, normal performance, and preserved bioenergetics in the heart at baseline.

193 ology and deficits in metabolic and cellular bioenergetics in the pathology of PD.

194 nfluencing gluconeogenesis and mitochondrial bioenergetics in the UCD-T2DM rat model of diabetes.

195 of mitochondrial Ca(2+) uptake in regulating bioenergetics in these cells, we used OXPHOS-competent a

196 analysis, we investigated adult muscle fiber bioenergetics in this mouse model.

197 en species production, and enabled oxidative bioenergetics in tumor cells.

198 -glutamate pathways disrupting mitochondrial bioenergetics, increased polyamine biosynthesis and brea

199 ons, we use the cell membrane potential as a bioenergetic indicator of EET by S. oneidensis MR-1 cell

200 We report findings on the bioenergetic interplay of astrocytes and neurons and dis

201 We propose that aberrant neural bioenergetics is a common feature between CDD and RTT di

202 Mitochondrial bioenergetics is dynamically coupled with neuronal activ

203 Reactivation of mitochondrial bioenergetics is followed by dramatic reorganization of

204 Mitochondrial bioenergetics is regulated by calcium uptake through the

205 The significance of the dual fuel bioenergetics is unclear and may be related to an interm

206 ributions from oxidative and substrate-level bioenergetics is unknown.

207 f DISC1 in astrocytes could impair astrocyte bioenergetics, leading to abnormalities in synaptic neur

208 he first time, been elucidated at a protonic bioenergetics level: 1) The formation of cristae creates

209 rdial substrate metabolism and mitochondrial bioenergetics, lipotoxicity, and altered signal transduc

210 drawn with the more universal mitochondrial bioenergetic machinery, from whence molecular-scale insi

211 abolism, occur in normal aging and disturbed bioenergetics may be an important contributing mechanism

212 Mutations affecting mitochondrial bioenergetics may lead to isolated vision loss or life-t

213 ated glutamatergic signalling and changes in bioenergetics may mediate the behavioural phenotype indu

214 lic reprogramming assessed through real-time bioenergetic measurement and metabolomics upregulated gl

215 Here high-resolution quantitative imaging, bioenergetics measurements and mitochondrial membrane po

216 Mitochondria internalised not only the bioenergetic membranes but also the genetic machinery ne

217 altered the topology of genes in relation to bioenergetic membranes.

218 utophagy, cellular senescence, inflammation, bioenergetic metabolism, Ca(2+) fluxes, and redox homeos

219 Mitochondria are responsible for bioenergetics, metabolism and apoptosis signals in healt

220 ations between maternal CM and mitochondrial bioenergetics (mitochondrial respiration and intracellul

221 Further, we used a mechanistic bioenergetics model (Niche Mapper), to compare the energ

222 ms a previously published "species-specific" bioenergetics model.

223 To develop bioenergetics models for shark and ray megafauna, increm

224 trient uptake to levels that exceed a cell's bioenergetic needs, adaptive changes in intermediate met

225 This study reports data on mitochondrial bioenergetics of healthy mother-newborn dyads with varyi

226 a distinct dichotomy in the polarization and bioenergetics of in vitro models, with M2 macrophages ut

227 We surmised that PINK1 modulates the bioenergetics of peripheral blood mononuclear cells (PBM

228 In this study, we defined the bioenergetics of Th17 effector cells generated in vivo.

229 organelles occupy a critical position in the bioenergetics of the cardiovascular system, mitophagy is

230 Here we assess the impact of mitochondrial bioenergetics on neovascularisation, by deleting cox10 g

231 cs aimed at improving vascular mitochondrial bioenergetics or reducing inflammation before hyperlipid

232 Bioenergetic parameter estimates were derived including:

233 cells were analyzed by Western blot and cell bioenergetic parameters by extracellular flux analysis.

234 microscopy was used to study functional and bioenergetic parameters in cardiomyocytes isolated after

235 Proliferating HepaRG bioenergetic parameters were severely impaired by day 8,

236 rely on oxidative phosphorylation for their bioenergetics, particularly during the activation proces

237 Fatty acid beta-oxidation (FAO) is the main bioenergetic pathway in human prostate cancer (PCa) and

238 Fatty acid oxidation (FAO) is a key bioenergetic pathway often dysregulated in diseases.

239 phosphorylation (OXPHOS) is the predominant bioenergetic pathway to support osteoclast differentiati

240 transcriptomics data, suggest that multiple bioenergetic pathways are differentially regulated by AP

241 We examined bioenergetic pathways in the dorsolateral prefrontal cor

242 el implications for the interconnectivity of bioenergetic pathways, and suggest a provocative strateg

243 tricarboxylic acid (TCA) cycle, two central bioenergetic pathways.

244 and phosphoproteome and reveal signaling and bioenergetics pathways that mediate lymphocyte exit from

245 d primarily involved protein translation and bioenergetics pathways.

246 dynamics of these organelles are central to bioenergetic performance and plant physiology, this chal

247 While bioenergetics performance is acceptable, the 3B model se

248 zoan complexity and life-style depend on the bioenergetic potential of mitochondria.

249 sh that H(2)S stimulates Mtb respiration and bioenergetics predominantly via cytochrome bd oxidase, a

250 sory perception, but also the most important bioenergetic process on earth: photosynthesis.

251 rt across cellular membranes is required for bioenergetic processes and metabolic signaling.

252 Here we investigate the bioenergetic processes which facilitate the HSC expansio

253 ing cell signalling, molecular transport and bioenergetic processes.

254 e used to allocate energy flow into specific bioenergetic products.

255 rial function revealed a surprisingly normal bioenergetics profile.

256 We investigated the bioenergetic profiles of fibroblasts from LOAD patients

257 Here, we combined bioenergetic profiling and (13)C-glucose infusion techni

258 Cellular bioenergetic profiling following treatment with P-AscH(-

259 ogenesis and glycolysis-/glutamine-dependent bioenergetics provide insight into the cellular environm

260 CL plays an important role in mitochondrial bioenergetics, recent evidence in the yeast model indica

261 , may help to explain the paradox of lacking bioenergetic recovery despite enhanced TFAM expression.

262 n that the EC is susceptible to differential bioenergetic regulation in response to a metabolic stres

263 g immunometabolism now allow the analysis of bioenergetic regulation with cellular resolution and, as

264 n, thus allowing for intrinsic mitochondrial bioenergetics, relative to the underlying proteome, to b

265 However, brain lipid bioenergetics remain largely uncharacterized.

266 and spare respiratory capacity (an index of bioenergetic reserve: 6.2 +/- 4.3 vs 9.6 +/- 3.1; p = 0.

267 the central nervous system, glycogen-derived bioenergetic resources in astrocytes help promote tissue

268 prion infection have been reported, yet the bioenergetic respiratory status of mitochondria from pri

269 chronic proteotoxic stress disrupts retinal bioenergetics resulting in mitochondrial dysfunction, an

270 r comprehensive analysis of pancreatic islet bioenergetics reveals that Drp1 does not control insulin

271 In sum, we define a bioenergetic sensor in muscle that utilizes intracellula

272 brain metastasis by inhibiting mitochondrial bioenergetics, stimulating the formation of reactive oxy

273 as an endogenous mechanism used to mitigate bioenergetic stress and distribute the impact of neurode

274 hilin D-knockout littermates did not develop bioenergetic stress in response to IH challenge and full

275 Thus "bioenergetic stress" drives systemic inflammation-induce

276 xibility is important in the CNS in times of bioenergetic stress.

277 ochondrial respiration, leading to transient bioenergetic stress.

278 t renders the donating tissue susceptible to bioenergetic, structural, and physiological degradation.

279 re, mass spectrometry-based metabolomics and bioenergetics studies identify defects in fatty acid uti

280 hat limit metabolic disturbances and provide bioenergetic support.

281 ive effects on tumor cells, altered cellular bioenergetics, suppressed matrix metalloproteinases and

282 emonstrate that heme loading drives a unique bioenergetic switch in macrophages, which involves a met

283 c analysis revealed stage-specific shifts in bioenergetic systems of biology that were paralleled by

284 A key bioenergetic target for the Krebs cycle, the electron tr

285 y age-related impairments in skeletal muscle bioenergetics that result in a greater accumulation of m

286 challenge may weaken the local mitochondrial bioenergetics that the fuel postsynaptic activities of t

287 cooperate to impair epithelial mitochondrial bioenergetics, thereby triggering microbiota disruptions

288 arance, the IECs respond by rapidly shifting bioenergetics to aerobic glycolysis, which leads to oxyg

289 activity for the regulation of mitochondrial bioenergetics to meet fluctuating neuronal energy demand

290 imary fibroblasts by measuring mitochondrial bioenergetics, ultrastructural and dynamic parameters to

291 scue of the metabolic impairment of neuronal bioenergetics underlying neurodegeneration in multiple s

292 the same mitochondrion behave as independent bioenergetic units, preventing the failure of specific c

293 argeting mitochondrial function and cellular bioenergetics upstream of cellular damage may offer adva

294 bility, mtDNA copy number, and mitochondrial bioenergetics utilizing trypan blue, Southern blotting,

295 The resulting impact on mitochondrial bioenergetics was evaluated using a respiratory diagnost

296 Mitochondrial bioenergetics were measured in neonatal (P14) and young

297 Mitochondrial bioenergetics were modulated sequentially using respirat

298 Although maternal and neonatal mitochondrial bioenergetics were positively correlated, maternal CM on

299 ionnaire Maternal and neonatal mitochondrial bioenergetics were quantitatively comparable and positiv

300 ntial for growth but is required to maintain bioenergetics when the function of the cytochrome bc(1):