ライフサイエンスコーパス: bioenergetics

1 lities that could be attributed to astrocyte bioenergetics.

2 standing and interpretation of intracellular bioenergetics.

3 es astrocytes and neurons and supports brain bioenergetics.

4 tive phosphorylation, known as mitochondrial bioenergetics.

5 es, and downregulation of genes important in bioenergetics.

6 cellular and organellar functions including bioenergetics.

7 L expression, which plays a role in cellular bioenergetics.

8 hronize organismal food intake with cellular bioenergetics.

9 lasmic reticulum and mitochondria to sustain bioenergetics.

10 ntial for functions apart from mitochondrial bioenergetics.

11 ereas ncOGT predominantly regulates cellular bioenergetics.

12 luteotropin, and estrogen, on corneal stroma bioenergetics.

13 e regulation of mitochondrial biogenesis and bioenergetics.

14 lect intracellular ATP turnover and cellular bioenergetics.

15 the circadian clock governs skeletal muscle bioenergetics.

16 mportant role in mitochondrial processes and bioenergetics.

17 ndrial membrane potential, and mitochondrial bioenergetics.

18 se in FECD indicated deficient mitochondrial bioenergetics.

19 ty control, maintaining the functionality of bioenergetics.

20 pression of genes important in mitochondrial bioenergetics.

21 at ncOGT is a negative regulator of cellular bioenergetics.

22 nd enables reuse of organelle components for bioenergetics.

23 adaptive response to stimulate mitochondrial bioenergetics.

24 s by connecting ROS partitioning to cellular bioenergetics.

25 s, problems that are at the core of cellular bioenergetics.

26 increased mitochondrial biogenesis and tumor bioenergetics.

27 chondrial calcium transfer and mitochondrial bioenergetics.

28 ll invasion without changes in mitochondrial bioenergetics.

29 and plays an important role in mitochondrial bioenergetics.

30 microaerobic conditions to maintain membrane bioenergetics.

31 thermodynamic reference for calibrating PSII bioenergetics.

32 er doxorubicin, confirming impaired cellular bioenergetics.

33 es mitochondrial RNA (mtRNA) homeostasis and bioenergetics.

34 in many genes associated with mitochondrial bioenergetics.

35 tochondrial dysfunction and inhibiting tumor bioenergetics.

36 of disrupted mitochondrial architecture and bioenergetics.

37 t alter mitochondrial membrane potential and bioenergetics.

38 ent kinase II activation and altered myocyte bioenergetics.

39 fusion and to maintain optimal mitochondrial bioenergetics.

40 etabolic feedback circuits and mitochondrial bioenergetics.

41 additional processes in the brain, including bioenergetics.

42 st into oxidative phosphorylation and muscle bioenergetics.

43 activates autophagy, while the other targets bioenergetics.

44 omplicated, which is also reflected in their bioenergetics.

45 ytosolic signaling events with mitochondrial bioenergetics.

46 stress (mPOS) in the cytosol independent of bioenergetics.

47 ulting iPSCs and ESCs, suggesting comparable bioenergetics.

48 ramming of lipid metabolism and mitochondria bioenergetics.

49 checkpoint for mitochondrial biogenesis and bioenergetics.

50 utonomous clocks in the timing of organismal bioenergetics.

51 uated if 2-AI compounds affect mycobacterial bioenergetics.

52 h age-related differences in skeletal muscle bioenergetics.

53 eurons, in addition to their primary role in bioenergetics, also contribute to specialized functions,

54 The bioenergetics analysis identified a moderate, direct imp

55 omponent ion circuit for [Formula: see text] bioenergetics and a 2nd 2-component ion circuit for Na(+

56 in resistance causes alterations in cellular bioenergetics and activation of inflammatory signaling i

57 Our methodology combines optical bioenergetics and advanced signal processing and allows

58 ction and has emerged as central hub between bioenergetics and all major cellular events.

59 st that PINK1 is critical for modulating the bioenergetics and antioxidant responses in PBMCs whereas

60 chondrial biogenesis, improved mitochondrial bioenergetics and attenuated mitochondria-regulated apop

61 letal dysplasia by interfering with cellular bioenergetics and biosynthesis.

62 to researchers working in the field of brain bioenergetics and brain diseases.

63 genation by maintaining better mitochondrial bioenergetics and by decreasing ROS.

64 ondrial Ca(2+) uptake, a process crucial for bioenergetics and Ca(2+) signaling, is catalyzed by the

65 vels, and deregulation of both mitochondrial bioenergetics and Ca(2+)homeostasis was rescued by Mcl-1

66 electron- and proton-transport reactions in bioenergetics and catalysis in general.

67 are fundamental mechanisms for mitochondrial bioenergetics and cell function.

68 Bioenergetics and cell survival were instead unaffected

69 cell cycle progression, repair/maintenance, bioenergetics and cell-cell signaling - whose disrupted

70 ng a tool to better understand mycobacterial bioenergetics and develop compounds with improved anti-m

71 e investigated reactivation of mitochondrial bioenergetics and dynamics using Arabidopsis thaliana as

72 t mitochondrial energy metabolism, measuring bioenergetics and enzyme activities of the electron tran

73 Virtual fish were realistic both in terms of bioenergetics and feeding.

74 glucose deprivation stimulate mitochondrial bioenergetics and formation of respiratory supercomplexe

75 These changes impact T cell bioenergetics and function.

76 e Mfn2 overexpression enhances mitochondrial bioenergetics and functions, and promotes the differenti

77 We also assessed cellular bioenergetics and generation of reactive oxygen species

78 of Bnip3 knockdown on adipose mitochondrial bioenergetics and glucose disposal.

79 d complex V, resulting in impaired oxidative bioenergetics and heightened ROS production.

80 of mitochondrial CypD results in a shift in bioenergetics and in activation of glucose-metabolism re

81 atic airway epithelium with consequences for bioenergetics and inflammation.

82 lecular oxygen (O(2)) sustains intracellular bioenergetics and is consumed by numerous biochemical re

83 )S pathway sustain endothelial mitochondrial bioenergetics and loss of CSE increases the production o

84 tabolic reprograming in order to provide the bioenergetics and macromolecular precursors needed to su

85 rely on multiple nutrients to meet cellular bioenergetics and macromolecular synthesis demands of ra

86 larly dependent on glucose and glutamine for bioenergetics and macromolecule biosynthesis.

87 plexes in mitochondrial membranes to support bioenergetics and maintain mitochondrial architecture.

88 oxidative phosphorylation complexes, altered bioenergetics and metabolic shift are often seen in canc

89 cular oxygen (O(2)) plays important roles in bioenergetics and metabolism and is implicated in bioche

90 Bioenergetics and mitochondrial DNA (mtDNA) damage were

91 A comparison of bioenergetics and mitochondrial function between isogeni

92 nteract HD-related deficits in mitochondrial bioenergetics and motor function.SIGNIFICANCE STATEMENT

93 Differences in mitochondrial bioenergetics and mtDNA damage associated with maternal

94 pt and whether SIRT5 regulates mitochondrial bioenergetics and neuroprotection against cerebral ische

95 of these methodologies can help tease apart bioenergetics and other biological complexities in C. el

96 linked to dysregulation of the mitochondrial bioenergetics and oxidative status.

97 ed Mn(2+) ions may have played a key role in bioenergetics and possibly facilitated early geological

98 nk TDP-43 toxicity directly to mitochondrial bioenergetics and propose the targeting of TDP-43 mitoch

99 rough TRPM2 is required to maintain cellular bioenergetics and protect against hypoxia-reoxygenation

100 fies HSF1 as a central regulator of cellular bioenergetics and protein homeostasis that benefits mali

101 lore more effective therapies, mitochondrial bioenergetics and redox homeostasis were assessed in VLC

102 ic approach in form of altered mitochondrial bioenergetics and redox status of cancer cells with unde

103 n lymphatic muscle cells (LMCs) affects cell bioenergetics and signaling pathways that consequently a

104 holipid with critical roles in mitochondrial bioenergetics and signaling.

105 ajor ion reservoir that can be mobilized for bioenergetics and signaling.

106 l electron transport chain, which compromise bioenergetics and suggest a mechanism by which ET-1 prom

107 l, our results provide insights into mitotic bioenergetics and suggest that cell division is not a hi

108 itical role of TRPM2 in gastric cancer cells bioenergetics and survival; however, its role in gastric

109 hat couple mitochondrial fusion/fission with bioenergetics and their impacts on neurodegeneration how

110 herefore, IDH2 is a dual regulator of cancer bioenergetics and tumor cell motility.

111 e for quantitative proteomics, mitochondrial bioenergetics and tumor growth in mice were conducted.

112 Bok controls neuronal Ca(2+)homeostasis and bioenergetics and, contrary to previous assumptions, exe

113 ual roles of mitochondria in ATP production (bioenergetics) and apoptosis (cell life/death decision)

114 drial matrix uptake of Ca(2+), mitochondrial bioenergetics, and autophagic flux.

115 ake, a process crucial for Ca(2+) signaling, bioenergetics, and cell death.

116 etabolomics and focused metabolite analyses, bioenergetics, and cell viability assays, we show that t

117 l functions, mitochondrial functions such as bioenergetics, and functions related to transcription su

118 ondrial morphology, attenuates mitochondrial bioenergetics, and induces mitochondrial DNA oxidative i

119 omena that are also at play in photobiology, bioenergetics, and information processing.

120 is work resolves a long-standing question in bioenergetics, and renders a chemical-biological basis f

121 Ca(2+) uptake by mitochondria regulates bioenergetics, apoptosis, and Ca(2+) signaling.

122 g the integrity of the genome and sustaining bioenergetics are both fundamental functions of the cell

123 Mitochondrial bioenergetics are critical for cellular homeostasis and

124 rgeting mitochondria protection and cellular bioenergetics are presented, with emphasis on those that

125 ondria, long viewed solely in the context of bioenergetics, are increasingly emerging as critical hub

126 The enhancement of mitochondrial bioenergetics as well as the increase in mitochondrial p

127 rial biogenesis, coupled with aberrant tumor bioenergetics, as a potential therapy escape mechanism a

128 y the sub pathways involved in mitochondrial bioenergetics, as observed in other neurodegenerative di

129 secting fields of mitochondrial dynamics and bioenergetics, as treatment of defective dynamics in mit

130 Bioenergetics aspects of the 3B model are further evalua

131 Using a combination of real-time bioenergetics assays and metabolomics approaches, we inv

132 fibroblasts showed overactive mitochondrial bioenergetics associated with atypical morphology and al

133 TMX1 reduce ER-mitochondria contacts, shift bioenergetics away from mitochondria, and accelerate tum

134 ctral exponent of local field potentials and bioenergetics based on the activity of mitochondrial Cyt

135 gene expression signature, the mitochondrial bioenergetics, biogenesis and fuel catabolic functions a

136 by a prolonged deregulation of mitochondrial bioenergetics.bok deficiency led to a specific reduction

137 ected proteostasis to maintain mitochondrial bioenergetics, buffer oxidative stress, and enable metas

138 cellular plasticity by sustaining oxidative bioenergetics, buffering ROS production, and supporting

139 ative phosphorylation is central to cellular bioenergetics but cumbersome to measure.

140 e of BMI1 in the regulation of mitochondrial bioenergetics, but also provide new mechanistic insights

141 r the risk of proteotoxic stress to preserve bioenergetics, but the role of these mechanisms in disea

142 ed to occur independently of follicular bulb bioenergetics by a tractor mechanism involving the inner

143 pocytes were additionally examined for their bioenergetics by extracellular flux analysis as well as

144 Improving bioenergetics by overexpression of PGC-1alpha enhanced f

145 llenges oxidative stress imposes on membrane bioenergetics by shifting redox balance to glycolysis an

146 AMs) are central microdomains that fine-tune bioenergetics by the local transfer of calcium from the

147 ier remains a key uncertainty underlying the bioenergetics calculations.

148 tilization, and alterations in mitochondrial bioenergetics can additionally propel stem cell deficits

149 Real-time monitoring of changes to cellular bioenergetics can provide new insights into mechanisms o

150 ment, including local synaptic E-I ratio and bioenergetics, can be modeled by cerebral organoids (CO)

151 ORP4L knockdown results in suboptimal bioenergetics, cell death and abrogation of T-ALL engraf

152 of all mammalian tissues, where it regulates bioenergetics, cell death, and Ca(2+) signal transductio

153 broblasts displayed suppressed mitochondrial bioenergetics consistent with a lower substrate availabi

154 In pancreatic beta-cells, mitochondrial bioenergetics control glucose-stimulated insulin secreti

155 otein kinase (AMPK) is a master regulator of bioenergetics crucial for glucose metabolism in acute my

156 a, acutely lowered SNPH levels, resulting in bioenergetics defects and increased superoxide productio

157 ient-reported outcomes and immunological and bioenergetics disease parameters.

158 arian cancer cell lines revealed significant bioenergetics diversity.

159 of mitochondrial mass and abrogates cellular bioenergetics during degeneration of post-mitotic cells

160 Deficits in bioenergetics during early postnatal brain development c

161 mming of hepatocellular lipid metabolism and bioenergetics during HCV infection, which are predicted

162 is known about the metabolic requirement and bioenergetics during osteoclastogenesis.

163 econd-messenger production and mitochondrial bioenergetics during oxidative stress.

164 toration of N source preference and cellular bioenergetics during the early stage of recovery; (2) fl

165 cterize cellular derangements, mitochondrial bioenergetics, dynamics, endoplasmic reticulum (ER)-mito

166 as seen, potentially linking AMA with muscle bioenergetics dysfunction; however, this was not related

167 ss involved in different aspects of cellular bioenergetics; dysregulation of lipid oxidation is often

168 his process is called excitation-contraction-bioenergetics (ECB) coupling.

169 in prostate cancer cells impaired oxidative bioenergetics, elevated reactive oxygen species (ROS) pr

170 Finally, AP39, a modulator of mitochondrial bioenergetics enhanced cytochrome c oxidase activity, re

171 es indicate that insulin influences cerebral bioenergetics, enhances synaptic viability and dendritic

172 Mechanistically, bioenergetics experiments revealed that PF reduces mitoc

173 her we identify a critical role for BUD23 in bioenergetics gene expression, by promoting efficient tr

174 Mitochondrial bioenergetics has been implicated in a number of vital c

175 l (Deltapsi), which is central to organismal bioenergetics, has been successfully measured via flow c

176 rylation are rheostats in immune cells whose bioenergetics have functional outputs in terms of their

177 Body-wide changes in bioenergetics, i.e., energy metabolism, occur in normal

178 We evaluated mitochondrial bioenergetics in 10 sets of LCLs from children with ASD,

179 and a 2nd 2-component ion circuit for Na(+) bioenergetics in a strictly anaerobic rumen bacterium.

180 Here, we studied bioenergetics in an induced pluripotent stem cell (iPSC)

181 tworks indicates a broader role of astrocyte bioenergetics in determining how experience-dependent in

182 enic response and restored the mitochondrial bioenergetics in endothelial cells.

183 HCFs revealing a novel role for hormones on bioenergetics in KC.

184 onfirmed that miR-29a inhibits mitochondrial bioenergetics in LCC9 cells.

185 tive phosphorylation, inhibits mitochondrial bioenergetics in lung cancer cells and mitigates lung ca

186 es in autophagy, mitochondrial dynamics, and bioenergetics in mouse models of acute and chronic Dox-c

187 PGC-1alpha and Tug1 modulates mitochondrial bioenergetics in podocytes in the diabetic milieu.

188 of evidence suggests abnormalities in brain bioenergetics in psychiatric disorders, including both b

189 al carbon metabolism fluxes and central cell bioenergetics in response to ammonium availability and n

190 The capacity of cells to alter bioenergetics in response to the demands of various biol

191 Examination of mitochondrial bioenergetics in stable cell lines overexpressing GFP-ta

192 Restoring mitochondrial bioenergetics in the colonic epithelium with 5-amino sal

193 esembling pre-IBD and impaired mitochondrial bioenergetics in the colonic epithelium, which triggered

194 Assessments of mitochondrial bioenergetics in the cortex of wild type (WT) and SIRT5-

195 t obese women exhibit impaired mitochondrial bioenergetics in the form of decreased efficiency and im

196 henotypes, normal performance, and preserved bioenergetics in the heart at baseline.

197 haracterized the effects of CypD ablation on bioenergetics in the kidney.

198 ology and deficits in metabolic and cellular bioenergetics in the pathology of PD.

199 ssociated with improvements in mitochondrial bioenergetics in the podocytes of diabetic mice.

200 nfluencing gluconeogenesis and mitochondrial bioenergetics in the UCD-T2DM rat model of diabetes.

201 of mitochondrial Ca(2+) uptake in regulating bioenergetics in these cells, we used OXPHOS-competent a

202 analysis, we investigated adult muscle fiber bioenergetics in this mouse model.

203 en species production, and enabled oxidative bioenergetics in tumor cells.

204 haviors and motor function, as well as brain bioenergetics, in a mouse model (luc) carrying a spontan

205 al deregulation and changes in mitochondrial bioenergetics, including pyruvate dehydrogenase (PDH) dy

206 -glutamate pathways disrupting mitochondrial bioenergetics, increased polyamine biosynthesis and brea

207 Suppression of T cell bioenergetics involved restricted glucose uptake and use

208 We propose that aberrant neural bioenergetics is a common feature between CDD and RTT di

209 Mitochondrial bioenergetics is dynamically coupled with neuronal activ

210 Reactivation of mitochondrial bioenergetics is followed by dramatic reorganization of

211 Mitochondrial bioenergetics is regulated by calcium uptake through the

212 The significance of the dual fuel bioenergetics is unclear and may be related to an interm

213 ributions from oxidative and substrate-level bioenergetics is unknown.

214 ssociation is reflected in the intramuscular bioenergetics is unknown.

215 sphate carrier (PiC), encoded by SLC25A3, in bioenergetics is well accepted.

216 f DISC1 in astrocytes could impair astrocyte bioenergetics, leading to abnormalities in synaptic neur

217 t role for 14-3-3zeta in regulating platelet bioenergetics, leading to decreased platelet PS exposure

218 he first time, been elucidated at a protonic bioenergetics level: 1) The formation of cristae creates

219 rdial substrate metabolism and mitochondrial bioenergetics, lipotoxicity, and altered signal transduc

220 abolism, occur in normal aging and disturbed bioenergetics may be an important contributing mechanism

221 Mutations affecting mitochondrial bioenergetics may lead to isolated vision loss or life-t

222 ated glutamatergic signalling and changes in bioenergetics may mediate the behavioural phenotype indu

223 Here high-resolution quantitative imaging, bioenergetics measurements and mitochondrial membrane po

224 Mitochondria are responsible for bioenergetics, metabolism and apoptosis signals in healt

225 des are redox coenzymes that are critical in bioenergetics, metabolism, and neurodegeneration.

226 ations between maternal CM and mitochondrial bioenergetics (mitochondrial respiration and intracellul

227 Further, we used a mechanistic bioenergetics model (Niche Mapper), to compare the energ

228 We used a spatio-temporal bioenergetics model of the Northeast Pacific Ocean to qu

229 ics data from the literature, to construct a bioenergetics model to quantify predation rates on key f

230 ms a previously published "species-specific" bioenergetics model.

231 and abiotic parameters can be obtained, then bioenergetics modelling offers an alternative approach t

232 To develop bioenergetics models for shark and ray megafauna, increm

233 Bioenergetics models indicate that the sharks require ap

234 cy in the pass and feeding behavior and used bioenergetics models to understand energy flow.

235 (+) T cells were already unable to match the bioenergetics of effector T cells generated during acute

236 This study reports data on mitochondrial bioenergetics of healthy mother-newborn dyads with varyi

237 The bioenergetics of IF1 transiently silenced cancer cells h

238 a distinct dichotomy in the polarization and bioenergetics of in vitro models, with M2 macrophages ut

239 We surmised that PINK1 modulates the bioenergetics of peripheral blood mononuclear cells (PBM

240 In this study, we defined the bioenergetics of Th17 effector cells generated in vivo.

241 organelles occupy a critical position in the bioenergetics of the cardiovascular system, mitophagy is

242 ought to have been needed to account for the bioenergetics of the first single-celled organisms.

243 rentiation has been extensively studied, the bioenergetics of Treg cell trafficking remains undefined

244 Here we assess the impact of mitochondrial bioenergetics on neovascularisation, by deleting cox10 g

245 cs aimed at improving vascular mitochondrial bioenergetics or reducing inflammation before hyperlipid

246 of H2S-producing enzymes suppresses critical bioenergetics parameters in lung adenocarcinoma cells.

247 rely on oxidative phosphorylation for their bioenergetics, particularly during the activation proces

248 and phosphoproteome and reveal signaling and bioenergetics pathways that mediate lymphocyte exit from

249 d primarily involved protein translation and bioenergetics pathways.

250 While bioenergetics performance is acceptable, the 3B model se

251 The bioenergetics phenotype of ovarian cancer cell lines cor

252 istory, there is no reason to think membrane bioenergetics played a direct, causal role in the transi

253 sh that H(2)S stimulates Mtb respiration and bioenergetics predominantly via cytochrome bd oxidase, a

254 xysmal manifestations and a normalised brain bioenergetics profile in patients with GLUT1-DS.

255 rial function revealed a surprisingly normal bioenergetics profile.

256 sm was associated with increased NCM356 cell bioenergetics, proliferation, invasion through Matrigel,

257 ogenesis and glycolysis-/glutamine-dependent bioenergetics provide insight into the cellular environm

258 such, assessment of skeletal muscle cellular bioenergetics provides a powerful means to understand th

259 CL plays an important role in mitochondrial bioenergetics, recent evidence in the yeast model indica

260 t surprising that mitochondrial dynamics and bioenergetics reciprocally influence each other.

261 is an important process regulating cellular bioenergetics, redox responses, and apoptosis.

262 Lignin biosynthetic genes and bioenergetics-related genes were up-regulated in the hig

263 n, thus allowing for intrinsic mitochondrial bioenergetics, relative to the underlying proteome, to b

264 However, brain lipid bioenergetics remain largely uncharacterized.

265 Abeta levels and compromise in mitochondrial bioenergetics result in dysfunctional synaptic plasticit

266 chronic proteotoxic stress disrupts retinal bioenergetics resulting in mitochondrial dysfunction, an

267 Analysis of bioenergetics revealed thatNrf2(-/-)white adipose tissue

268 r comprehensive analysis of pancreatic islet bioenergetics reveals that Drp1 does not control insulin

269 Lower bioenergetics segregated with increased incidence of low

270 brain metastasis by inhibiting mitochondrial bioenergetics, stimulating the formation of reactive oxy

271 lucose, and had greater glycolytic flux in a bioenergetics stress test.

272 re, mass spectrometry-based metabolomics and bioenergetics studies identify defects in fatty acid uti

273 iple aspects of mitochondrial biology beyond bioenergetics support transformation, including mitochon

274 ive effects on tumor cells, altered cellular bioenergetics, suppressed matrix metalloproteinases and

275 ve stress and maintains complex II-dependent bioenergetics, sustaining local tumor growth while restr

276 tively greater fluctuations in intramuscular bioenergetics than in VO2 compared to longer intervals.

277 ata identify Arg1 as a key regulator of ILC2 bioenergetics that controls proliferative capacity and p

278 y age-related impairments in skeletal muscle bioenergetics that result in a greater accumulation of m

279 challenge may weaken the local mitochondrial bioenergetics that the fuel postsynaptic activities of t

280 cooperate to impair epithelial mitochondrial bioenergetics, thereby triggering microbiota disruptions

281 n demonstrated by comparison of mitochondria bioenergetics through extracellular flux analyses.

282 zed physiological regulator of mitochondrial bioenergetics through its ability to interact with ATP s

283 arance, the IECs respond by rapidly shifting bioenergetics to aerobic glycolysis, which leads to oxyg

284 activity for the regulation of mitochondrial bioenergetics to meet fluctuating neuronal energy demand

285 We examined mitochondrial bioenergetics, transcript and protein levels of oxidativ

286 as a therapeutic approach to reduce cellular bioenergetics, tumor growth, and enhance susceptibility

287 imary fibroblasts by measuring mitochondrial bioenergetics, ultrastructural and dynamic parameters to

288 re was no marked alteration in mitochondrial bioenergetics under basal conditions, culture of patient

289 scue of the metabolic impairment of neuronal bioenergetics underlying neurodegeneration in multiple s

290 argeting mitochondrial function and cellular bioenergetics upstream of cellular damage may offer adva

291 bility, mtDNA copy number, and mitochondrial bioenergetics utilizing trypan blue, Southern blotting,

292 The resulting impact on mitochondrial bioenergetics was evaluated using a respiratory diagnost

293 Improved bioenergetics were confirmed in vivo after dosing with A

294 To test this hypothesis, mitochondrial bioenergetics were determined in endothelial cells from

295 Mitochondrial bioenergetics were measured in neonatal (P14) and young

296 Mitochondrial bioenergetics were modulated sequentially using respirat

297 Although maternal and neonatal mitochondrial bioenergetics were positively correlated, maternal CM on

298 ionnaire Maternal and neonatal mitochondrial bioenergetics were quantitatively comparable and positiv

299 ntial for growth but is required to maintain bioenergetics when the function of the cytochrome bc(1):

300 ly activates genes involved in mitochondrial bioenergetics, whereas it normally down-regulates genes