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1                   Topical application of the bioflavonoid 4',5,7-trihydroxyflavone (apigenin) to mous
2                   However, the activities of bioflavonoids against the individual isoforms of human t
3                                              Bioflavonoids are agents with potential immunosuppressiv
4                                              Bioflavonoids are human dietary components that have bee
5 ssays, we demonstrated that ferulic acid and bioflavonoids are indeed highly bactericidal to CLas, wi
6                                              Bioflavonoids are plant compounds touted for their poten
7 experiments demonstrated a religation of the bioflavonoid as well as the VP16-induced MLL cleavage si
8 motivate the use of this and similar dietary bioflavonoids as relatively nontoxic inhibitors of AhR a
9                                      Because bioflavonoids block PAH-induced cell transformation and
10       Proanthocyanidins (PAs) are a group of bioflavonoids consisting of oligomers based on catechin
11                                              Bioflavonoids do not require redox cycling for activity
12 successfully demonstrated the utility of the bioflavonoid (-)-EGCG, a natural product as a chiral sol
13 echanism of action of three major classes of bioflavonoids, flavones, flavonols, and isoflavones, aga
14 le the 4'-OH moiety contributes primarily to bioflavonoid function.
15       These data support the hypothesis that bioflavonoids function as topoisomerase II poisons in hu
16                   Urinary HO-1 was higher in bioflavonoid groups.
17                                          The bioflavonoids hold promise as agents that can reduce imm
18                                        These bioflavonoids improve early outcomes in cadaveric renal
19          PBE (a concentrate of water-soluble bioflavonoids, mainly including phenolic compounds) has
20 e results suggest that maternal ingestion of bioflavonoids may induce MLL breaks and potentially tran
21 jects tasted and rated five solutions of the bioflavonoid naringin in 4% sucrose.
22 ents inducing infant leukemia, we identified bioflavonoids, natural substances in food as well as in
23 ulating effects of several tea catechins and bioflavonoids on DNA methylation catalyzed by prokaryoti
24 d the effects of quercetin and curcumin, two bioflavonoids, on ischemia-reperfusion in the rat.
25 use inhibition of beta-catenin with shRNA or bioflavonoid quercetin prevents calcification in primary
26                                          The bioflavonoids quercetin and curcumin are renoprotective
27 (-)-epigallocatechin-3-O-gallate (EGCG)] and bioflavonoids (quercetin, fisetin, and myricetin) inhibi
28  out both in the presence and absence of the bioflavonoid, quercetin.
29                                     Of seven bioflavonoids screened, galangin (3,5,7-trihydroxyflavon
30  topoisomerase II (topo II) as the target of bioflavonoids similar to VP16 and Dox.
31                Genistein was the most active bioflavonoid tested and stimulated enzyme-mediated DNA c
32                                  Based on 20 bioflavonoids tested, we identified a common structure e
33 on was inhibited by apigenin, a nonmutagenic bioflavonoid that has been shown to prevent mouse skin c
34                                              Bioflavonoid therapy improved early graft function.
35                                 Quercetin, a bioflavonoid which inhibits the synthesis of hsp90, hsp7
36                 Genistein, a widely consumed bioflavonoid with chemopreventative properties in adults