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1 sms are with bioavailable concentrations and biological half-life.
2 etary manganese affected 54Mn absorption and biological half-life.
3 finity for its receptor and by extending its biological half-life.
4 be used to predict PFAS binding strength and biological half-life.
5 fit (94% sensitivity and 53% specificity for biological half-life; 94% sensitivity and 58% specificit
6  healing, but its use is hampered by a short biological half-life and lack of tissue selectivity.
7 ral sclerosis (ALS), is limited by its short biological half-life and poor water solubility necessita
8 c polypeptide (GIP) is hindered by its short biological half-life and rapid renal clearance.
9  a cell-permeable mimetic that increases the biological half-life and therefore enhances the biologic
10    [Carbonyl-11C]4a cleared the brain with a biological half-life averaging 41 min.
11  radioactivity was eliminated via the urine (biological half-life ca. 4 h), the urinary bladder wall
12  addiction treatment is limited by its short biological half-life (e.g., 8 h or shorter in rats).
13  like an antibody, has a considerably longer biological half-life (e.g., approximately 107 h in rats)
14 old increase in clotting activity, and has a biological half-life equivalent to recombinant wild-type
15                                          The biological half-life for aqueous (226)Ra ranged from 8.9
16 activity against (-)-cocaine and the longest biological half-life in rats.
17 s pervasiveness in the environment, its long biological half-life in tissues, and its association wit
18  poor ribonuclease (RNase) resistance, short biological half-life, lack of tissue targeting, ineffici
19 ing from multivalency along with an improved biological half-life makes the tetravalent construct an
20 affinity are reflected by differences in the biological half-life, not the absolute uptake.
21 se holoenzyme activity was eliminated with a biological half-life of 1.3 hours, and the MEGC-PEG-rMET
22 pounds is highly metabolically stable with a biological half-life of 10.5 h, suggesting a once-daily
23 PLGA based optimized formulation exhibited a biological half-life of 18.64 h, which was ~4.5 times lo
24 efflux from the whole brain, clearing with a biological half-life of 35 min.
25 25-30% of the total 123I administered with a biological half-life of 45 h.
26 -PEG-rMETase apoenzyme was eliminated with a biological half-life of 90 hours, an approximately 36-fo
27       Given the general observation that the biological half-life of a protein drug is significantly
28 inding protein, which naturally prolongs the biological half-life of growth hormone.
29 its physical half-life (78.41 h) matches the biological half-life of IgG antibodies.
30 ide a mechanistic explanation for the longer biological half-life of PFOS in humans, compared to PFOA
31                                          The biological half-life of scleral proteoglycans was signif
32 iation dose to the tumor (r = 0.9), with the biological half-life of the antibody in the tumor being
33 f disaccharides is unfavorable, the extended biological half-life of the GB1107 surrogate indicated t
34 rganism OBT is a slow process with a tritium biological half-life on the order of months.
35  including controlled drug release, improved biological half-life, reduced toxicity and targeted deli
36 vironmental toxicant exhibiting a years-long biological half-life (t(1/2)) in humans and is linked wi
37                                              Biological half-life was longest when subjects with high