ライフサイエンスコーパス: bipolar

1 sed their usual TURP procedure (monopolar or bipolar).

2 fered between schizophrenia (0.54-0.95 AUC), bipolar (0.48-0.65 AUC), autism (0.52-0.81 AUC) and anor

3 ificant progression of voltage was observed (bipolar: 38 cm(2) [interquartile range (IQR), 25-54] ver

4 um disorder and 49.7% (95% CI 48.1-51.3) for bipolar affective disorder.

5 us KA-receptors described in horizontal, OFF-bipolar, amacrine or ganglion cells, which could not be

6 within 6 classes: photoreceptor, horizontal, bipolar, amacrine, retinal ganglion and non-neuronal cel

7 ubsequently transmit signals to second-order bipolar and horizontal cells.

8 We found that the density of OFF-midget bipolar and OFF-midget ganglion cells can support one-to

9 Participants recruited by the Bipolar and Schizophrenia Network for Intermediate Pheno

10 ht that only unfolded monomers assemble into bipolar and side-polar (smooth muscle myosin) filaments.

11 es of PV-immunoreactive neurons: multipolar, bipolar and spherical-shaped neurons, based on the shape

12 After 4 h, the myosins form thick bipolar and, for smooth muscle myosin, side-polar filame

13 ology into four types: multipolar, bitufted, bipolar, and irregular.

14 based on bionanoparticle interaction with a bipolar atmosphere induced, e.g., by a radioactive alpha

15 re extracted for 77 models of schizophrenia, bipolar, autism or anorexia across 13 studies.

16 tal disorder, including depression, anxiety, bipolar, borderline personality disorder, schizophrenia,

17 cell (AII AC) provides inhibition onto cone bipolar cell (BC) axons and retinal ganglion cell (RGC)

18 ere we map the distribution of all known OFF bipolar cell (BC) populations and horizontal cells.

19 mouse retina, the cone photoreceptor type 4 bipolar cell (BC4) synapse, and show that its developmen

20 nctions within the Aii amacrine cell-ON cone bipolar cell (CBC) network are essential for night visio

21 Despite bipolar cell dendritic retraction and moderate loss of h

22 t PRDM1 promotes photoreceptor fate and VSX2 bipolar cell fate.

23 actor OTX2 is required for photoreceptor and bipolar cell formation in the retina.

24 present in a narrow temporal window to drive bipolar cell formation.

25 we further demonstrate that Bax acts via the bipolar cell population, rather than cell-intrinsically,

26 egeneration with precise control and analyze bipolar cell responses and their effects on vision throu

27 pmental state, and potentiation of rod - rod bipolar cell signaling following rod photoreceptor degen

28 monstrate that Sfxn3 is expressed in several bipolar cell subtypes, retinal ganglion cells, and some

29 rod pathway(s) (e.g., direct rod to OFF cone bipolar cell synapses and/or glycinergic synapses from A

30 he development and maintenance of functional bipolar cell synapses, and TPBG may play a similar role

31 In young retinas, we find that three bipolar cell types precisely restore input synapse numbe

32 In mature retinas, only one of four bipolar cell types rewires homeostatically.

33 Of the three bipolar cell types that rewire, two contact new cones wi

34 ontacting a single cone by mid-gestation and bipolar cell-ganglion cell connectivity undergoing a mor

35 novel metabotropic glutamate receptors of ON bipolar-cell dendrites, are both present in lamprey.

36 naptic connections with different classes of bipolar cells (BCs) to propagate light signals.

37 were previously shown to be enriched in rod bipolar cells (BCs), secondary neurons of the retina tha

38 roretinogram recordings suggest that retinal bipolar cells (BCs), which filter and transmit photorece

39 Cone photoreceptors, OFF-midget bipolar cells (P pathway), OFF-diffuse bipolar (DB) type

40 lpha-dependent phosphoprotein in retinal rod bipolar cells (RBCs).

41 GABAergic synapses on axon terminals of rod bipolar cells (RBCs).

42 Moreover, S-cone bipolar cells (SCBCs) are also skewed towards ventral re

43 is accompanied by reduced light responses of bipolar cells and deficits in visual behaviors.

44 that cone photoreceptors and P-type pathway bipolar cells are tightly connected throughout the retin

45 omplete picture of the response diversity of bipolar cells at a "single glance".

46 d that P-type bipolar cells outnumber M-type bipolar cells at all eccentricities.

47 g beginning early in fetal life, with midget bipolar cells contacting a single cone by mid-gestation

48 We conclude that parasol and midget pathway bipolar cells deliver high-acuity spatial signals to the

49 or ablation have shown adaptive responses in bipolar cells expected to support normal vision.

50 generate the negative-going a-wave, while ON-bipolar cells generate positive-going b-waves.

51 We find that P-type bipolar cells outnumber M-type bipolar cells at all ecce

52 Here, different types of bipolar cells stratifying at distinct depths relay the e

53 normal positioning of Muller cell bodies and bipolar cells was evident throughout the inner neuroblas

54 hich rewiring of second-order neurons (i.e., bipolar cells) could preserve function.

55 ement and release at ribbon sites in retinal bipolar cells, and find that, although ribbon synapses d

56 Ellis et al. show that retinal ON and OFF bipolar cells, and the novel metabotropic glutamate rece

57 erage, they received 21% of their input from bipolar cells, excitatory local circuit neurons receivin

58 ry glutamatergic input exclusively from S-ON bipolar cells.

59 uller glia, rod and cone photoreceptors, and bipolar cells.

60 ions of neurons including photoreceptors and bipolar cells.

61 iency of synaptic transmission from cones to bipolar cells.

62 LRRTM4 is expressed specifically by rod bipolar cells; eliminating it in mouse retina perturbs t

63 ome sample, when compared to a (210)Po based bipolar charge equilibration device.

64 ng spindle bipolarization without predefined bipolar cues.

65 idget bipolar cells (P pathway), OFF-diffuse bipolar (DB) types DB3a and DB3b (M pathway), and gangli

66 This newly recognized bipolar deposition episode has consequently been deemed

67 a, developing effective treatments for acute bipolar depression remain inadequate.

68 gine is effective at treating and preventing bipolar depression.

69 ntipsychotics have proven to be effective in bipolar depression.

70 ve low resistance films which can be used in bipolar devices.

71 es the charge carrier density and suppresses bipolar diffusion, resulting in superior power factors t

72 so make the alloys more prone to detrimental bipolar diffusion.

73 utations in sorCS2 have been associated with bipolar disease, schizophrenia, and attention deficient-

74 andard deviation increase; p = 8 x 10(-13)), bipolar disorder (1.53[1.44; 1.63]; p = 1 x 10(-43)), sc

75 or bipolar disorder predicted progression to bipolar disorder (adjusted hazard ratio for a one-standa

76 tory still strongly predicted progression to bipolar disorder (adjusted hazard ratio=5.02, 95% CI=3.5

77 Schizophrenia (SCZ) and bipolar disorder (BD) are severe mental disorders associ

78 s granule neurons derived from patients with bipolar disorder (BD) as well as a hyperexcitability tha

79 Distinguishing bipolar disorder (BD) from major depressive disorder (MD

80 Bipolar disorder (BD) is a chronic affective disorder wi

81 Bipolar disorder (BD) is a highly heritable psychiatric

82 Bipolar disorder (BD) is one of the most heritable menta

83 within frontal lobe gray and white matter of bipolar disorder (BD) patients have been consistently re

84 affective disorder (SAD), 132 with psychotic bipolar disorder (BD)), 315 of their nonpsychotic first-

85 depressive disorder (MDD), 29 subjects with bipolar disorder (BD), and 33 healthy controls who perfo

86 Schizophrenia (SCZ), bipolar disorder (BD), and major depressive disorder (MD

87 isorders (MPDs), such as schizophrenia (SZ), bipolar disorder (BD), and major depressive disorder (MD

88 Lithium is a first-line medication for bipolar disorder (BD), but only one in three patients re

89 disorders: autism spectrum condition (ASC), bipolar disorder (BD), major depressive disorder (MDD),

90 1 in every 50 to 100 people is affected with bipolar disorder (BD), making this disease a major econo

91 ations with Major Depressive Disorder (MDD), Bipolar Disorder (BD), Schizophrenia, anxiety, and Post

92 ion of the kynurenine pathway (KP) occurs in bipolar disorder (BD).

93 opathological findings have been reported in bipolar disorder (BD).

94 Lithium (Li) is a first-line treatment for bipolar disorder (BD).

95 their relevance for schizophrenia (SCZ) and bipolar disorder (BD).

96 s from patients with schizophrenia (SCZ) and bipolar disorder (BD).

97 irs affected with schizophrenia (SCZ) and/or bipolar disorder (BIP) (i.e., dual-affected pairs).

98 ric Genomics Consortium-schizophrenia (SCZ), bipolar disorder (BIP), major depression (MD), attention

99 ool to differentiate schizophrenia (SZ) from bipolar disorder (BP) even after balancing patients for

100 tal illnesses such as schizophrenia (SZ) and bipolar disorder (BP) frequently accompany metabolic con

101 elucidate the underlying pathophysiology of bipolar disorder (BP).

102 gamma-aminobutyric acid (GABA) mediated) and bipolar disorder (excitatory).

103 ) and patients with schizophrenia (N = 696), bipolar disorder (N = 211), autism spectrum disorder (N

104 wed significant evidence of association with bipolar disorder (n = 24) to map QTL influencing regiona

105 isk for schizophrenia (P <= 1 x 10(-10)) and bipolar disorder (P <= 0.005).

106 ed by more schooling years is an artefact of bipolar disorder - not education.

107 ychiatric phenotypes (e.g., r(g) = 0.36 with bipolar disorder and 0.34 with neuroticism) and negative

108 0% of the causal variants for schizophrenia, bipolar disorder and attention-deficit/hyperactivity dis

109 ite a lack of evidence for their efficacy in bipolar disorder and concerns about increasing the risk

110 eractivity disorder, but is much less so for bipolar disorder and depression.

111 al loci from existing GWAS data by analyzing bipolar disorder and epilepsy phenotypes available from

112 ancy should be planned during remission from bipolar disorder and lithium prescribed within the lowes

113 ignificant differences between schizophrenia/bipolar disorder and major depressive disorder were foun

114 The absolute risks of progression to bipolar disorder and psychotic disorders were 7.3% and 1

115 PRSs for bipolar disorder and schizophrenia are associated with r

116 d recurrent major depressive disorder (MDD), bipolar disorder and schizophrenia would enhance statist

117 MAD1L1 was previously identified in GWASs of bipolar disorder and schizophrenia.

118 ently detected in psychiatric disorders like bipolar disorder and schizophrenia.

119 ssion with self-reported MDD, recurrent MDD, bipolar disorder and schizophrenia.

120 netic basis of major depressive disorder and bipolar disorder and to highlight disorder-specific asso

121 w that differences between schizophrenia and bipolar disorder are concentrated in excitatory neurons

122 atric phenotypes including hyperactivity and bipolar disorder as well as epilepsy.

123 27) of extended pedigrees heavily loaded for bipolar disorder ascertained from genetically isolated p

124 tic studies of major depressive disorder and bipolar disorder can be combined effectively to enable t

125 Genetic correlations revealed that type 2 bipolar disorder correlates strongly with recurrent and

126 at least one diagnostic code reflective of a bipolar disorder diagnosis within 3 months of index anti

127 Schizophrenia and bipolar disorder do not appear to share similar mitochon

128 dy of the corpus callosum; schizophrenia and bipolar disorder featured comparable changes in the limb

129 DEGs in the bipolar disorder group were not enriched for functional

130 Bipolar disorder has a high heritability (approximately

131 Pharmacological options for treating bipolar disorder have increased over the past 20 years,

132 idepressants occurred in 47.0% of visits for bipolar disorder in the 1997-2000 period and 57.5% in th

133 abilizers decreased from 62.3% of visits for bipolar disorder in the 1997-2000 period to 26.4% in the

134 creasing from 12.4% of outpatient visits for bipolar disorder in the 1997-2000 period to 51.4% in the

135 ood stabilizers are better for patients with bipolar disorder in the maintenance phase.

136 s confer advantages, which serve to maintain bipolar disorder in the population.

137 Bipolar disorder is a chronic neuropsychiatric condition

138 Bipolar disorder is a chronic relapsing condition in whi

139 Bipolar disorder is a highly heritable illness, associat

140 Bipolar disorder is a lifelong mood disorder characteriz

141 Moreover, antidepressant prescription in bipolar disorder is associated, in many cases, with mood

142 he therapeutic window for lithium therapy of bipolar disorder is very narrow, and more frequent monit

143 s and clinical features in schizophrenia and bipolar disorder may be linked via mitochondrial dysfunc

144 While schizophrenia and bipolar disorder may have similar pathological character

145 Bipolar disorder may thus be better conceptualized as a

146 tween polygenic liability and progression to bipolar disorder or psychotic disorders among individual

147 are associated with risk for progression to bipolar disorder or psychotic disorders, respectively, a

148 al changes have occurred in the treatment of bipolar disorder over the past 20 years, with second-gen

149 = 46), schizophrenia patients (n = 25), and bipolar disorder patients with (n = 17) and without (n =

150 threshold compared with control subjects and bipolar disorder patients without psychotic features.

151 Comparing between schizophrenia spectrum and bipolar disorder patients, the models for positive sympt

152 riant data were available, schizophrenia and bipolar disorder polygenic risk were significantly overt

153 ustment for the other PRSs, only the PRS for bipolar disorder predicted progression to bipolar disord

154 ng subtypes of major depressive disorder and bipolar disorder provides evidence for a genetic mood di

155 ilar to the differences in schizophrenia and bipolar disorder relative to HCS.

156 ular mechanisms underlying schizophrenia and bipolar disorder remain poorly understood.

157 Currently, the pathophysiology of bipolar disorder remains elusive, but treatment with lit

158 , the effective treatment, and prevention of bipolar disorder represents an area of significant unmet

159 ndelian randomization of schooling years and bipolar disorder reveals that the increased risk of schi

160 oate (VPA) has been used in the treatment of bipolar disorder since the 1990s.

161 traits, ranging from 2.44% h(2) decrease for bipolar disorder to 14.2% h(2) decrease for Tourette syn

162 = 49), schizoaffective disorder (n = 37), or bipolar disorder with psychosis (n = 72), and identified

163 Patients with psychosis (schizophrenia and bipolar disorder with psychotic features) had an elevate

164 (1.36%) subsequently received a diagnosis of bipolar disorder within 3 months.

165 ers (including major depressive disorder and bipolar disorder) affect 10% to 20% of the population.

166 depression (including data from 23andMe) and bipolar disorder, and an additional major depressive dis

167 rison with HCS, we found that schizophrenia, bipolar disorder, and autism spectrum disorder share sim

168 pathways that have been implicated in human bipolar disorder, and changes in intestinal microbiota.

169 was most severe in schizophrenia, milder in bipolar disorder, and indistinguishable from healthy ind

170 polygenic nature of the risk architecture of bipolar disorder, and its overlap with other major neuro

171 e networks have been observed in depression, bipolar disorder, and post-traumatic stress disorder.

172 demonstrate MixEHR on MIMIC-III, Mayo Clinic Bipolar Disorder, and Quebec Congenital Heart Disease EH

173 nic risk scores (PRSs) for major depression, bipolar disorder, and schizophrenia were generated using

174 ces toward the goal of better treatments for bipolar disorder, and we outline major challenges for th

175 In patients with schizophrenia and bipolar disorder, both alterations in cerebral connectiv

176 ACKGROUNDAlthough mania is characteristic of bipolar disorder, it can also occur following focal brai

177 ychosis and participants with schizophrenia, bipolar disorder, major depressive disorder, and obsessi

178 sorders, including autism spectrum disorder, bipolar disorder, major depressive disorder, and schizop

179 y disorder (ADHD), autism spectrum disorder, bipolar disorder, major depressive disorder, and schizop

180 for 6 psychiatric disorders (schizophrenia, bipolar disorder, major depressive disorder, cross disor

181 rential volume alterations in schizophrenia, bipolar disorder, multiple sclerosis, mild cognitive imp

182 s (Schizophrenia spectrum, n = 79; Psychotic Bipolar disorder, n = 61) and matched healthy controls (

183 anxiety, psychosis, schizophrenia, mania, or bipolar disorder, personality disorder, substance use, a

184 nxiety (HR = 6.87, 95%CI 3.97-11.90); mania, bipolar disorder, psychosis, or schizophrenia (HR = 2.70

185 ntion deficit/hyperactivity disorder [ADHD], bipolar disorder, schizophrenia, alcohol dependence diso

186 nked to neuropsychiatric diseases, including bipolar disorder, schizophrenia, and autism.

187 ic overlaps with psychiatric conditions like bipolar disorder, schizophrenia, and cross-disorder susc

188 abnormalities in major depressive disorder, bipolar disorder, schizophrenia, and obsessive-compulsiv

189 sorders including major depressive disorder, bipolar disorder, schizophrenia, obsessive-compulsive di

190 s (ICD9/10CM) codes for anxiety, depression, bipolar disorder, schizophrenia, or other psychotic diso

191 ied the ANK3 W1989R variant in a family with bipolar disorder, suggesting a potential role of this va

192 GWAS statistics from genetically correlated bipolar disorder, the effect size of SNP genotypes on ge

193 direct comparison between schizophrenia and bipolar disorder, there were no significant differences.

194 l and genetic overlap between depression and bipolar disorder, we investigated whether a polygenic su

195 therwise specified and no prior diagnosis of bipolar disorder, who received at least one of the nine

196 atric disorders-autism spectrum disorder and bipolar disorder-to show that it is applicable to develo

197 ared with parental history, particularly for bipolar disorder.

198 rivalry, the other on the pathophysiology of bipolar disorder.

199 al neurons of subjects with schizophrenia or bipolar disorder.

200 gical agents in the outpatient management of bipolar disorder.

201 a diagnosis of depressive disorder to one of bipolar disorder.

202 e present novel evidence of association with bipolar disorder.

203 mer's disease, autism spectrum disorder, and bipolar disorder.

204 chiatric traits, including schizophrenia and bipolar disorder.

205 st effective mood-stabilizing medications in bipolar disorder.

206 atric conditions including schizophrenia and bipolar disorder.

207 transferable models to predict transition to bipolar disorder.

208 f lithium use during pregnancy in women with bipolar disorder.

209 updating could facilitate risk prediction in bipolar disorder.

210 a viable approach for novel interventions in bipolar disorder.

211 a/hypomania and depression that characterize bipolar disorder.

212 schizophrenia, major depressive disorder and bipolar disorder.

213 Bipolar disorders (BDs) are among the leading causes of

214 The mortality gap between populations with bipolar disorders and the general population is principa

215 Bipolar disorders are a complex group of severe and chro

216 ly, effective pharmacological treatments for bipolar disorders are not universally available, particu

217 Antidepressants are widely prescribed for bipolar disorders despite a paucity of compelling eviden

218 Although the pathogenesis of bipolar disorders is unknown, implicated processes inclu

219 Bipolar disorders share genetic risk alleles with other

220 Bipolar disorders substantially reduce psychosocial func

221 ising agent for the treatment of people with bipolar disorders, and has antimanic, antidepressant, an

222 n to improve health outcomes for people with bipolar disorders.

223 tely account for morbidity and mortality in, bipolar disorders.

224 athogenetic, and treatment considerations in bipolar disorders.

225 ogy, mechanisms, screening, and treatment of bipolar disorders.

226 This work identifies inhibitors of bipolar division in polyploid cells and provides a ratio

227 ls that inherit a single centrosome during a bipolar division with asymmetric centrosome clustering a

228 However, bipolar doping (n-type and p-type doping) and realizing

229 In the combined bipolar electrode cell, the overall potential is kept co

230 (PB) films in multiple closed ion-selective bipolar electrodes (BPEs), which gives a physical separa

231 Filtered unipolar and bipolar electrograms of continuous 2-minute AF recording

232 ex fractionated electrograms were defined as bipolar electrograms with >=5 directional changes occupy

233 Saline electrodes recorded unipolar and bipolar electrograms; microwave ablation caused reductio

234 diation, generated by a directly accelerated bipolar electron distribution, and the light transmitted

235 esultant pro-amniotic cavity, formation of a bipolar embryonic sac, and specification of primordial g

236 However, double enhancer targeting favored bipolar fate outcomes, whereas double gene targeting fav

237 tromal-derived factor 1, NMIIA activity, and bipolar filament assembly.

238 MK migration in the BM without compromising bipolar filament formation but led to divergent adhesion

239 on than in the absence of ATP and forms only bipolar filaments with bare zones.

240 ce of extrinsic finger muscle activity using bipolar fine-wire electrodes inserted into the extrinsic

241 RS was based on the latest schizophrenia and bipolar genome-wide association studies.

242 rmal radiation into electrical power using a bipolar grating-coupled complementary metal-oxide-silico

243 reas 8% were differentially expressed in the bipolar group.

244 f severe and chronic disorders that includes bipolar I disorder, defined by the presence of a syndrom

245 Although mania defines bipolar I disorder, depressive episodes and symptoms dom

246 pment for the treatment of schizophrenia and bipolar I disorder.

247 presence of a syndromal, manic episode, and bipolar II disorder, defined by the presence of a syndro

248 rkable non-divergent circuit: cone -> midget bipolar interneuron -> midget ganglion cell (the "privat

249 or the development of novel therapeutics for bipolar mania.

250 ) with a cation exchange membrane (CEM) or a bipolar membrane (BPM)], no chemical input was required

251 f-concept electrochemical system that uses a bipolar membrane electrodialysis (BPMED) cell and a vapo

252 oxide ions is important both for fabricating bipolar membranes (BPMs) that can couple different pH en

253 ric motor and tail domains at both ends of a bipolar minifilament.

254 keleton is extensively reorganized so that a bipolar mitotic spindle can be correctly assembled.

255 oublecortin-positive (DCX(+)) multipolar and bipolar neurons in the hippocampal formation than focal

256 In cephalochordates, Retzius bipolar neurons with intramedullary perikarya likely cor

257 signaling by triggering GABA-release onto ON-bipolar neurons.

258 chiatric disorders (including schizophrenia, bipolar or unipolar depression, anxiety, and substance u

259 nebula has two opposing fronts, suggesting a bipolar outflow of material from TYC 2597-735-1.

260 differentially impacts circadian rhythms in bipolar patient cell lines and crucially if lithium's ef

261 e and negative information was quantified in bipolar patients during euthymia, who were then monitore

262 pluripotent stem cells (iPSCs) derived from bipolar patients to further identify important molecular

263 sholds using genetic data obtained from 2586 bipolar patients who received lithium treatment and took

264 Bipolar patients with a low polygenic load for MD were m

265 e is a single-arm study evaluating biphasic, bipolar PFA using a multispline catheter for PVI and LAP

266 trols and individuals with major depression, bipolar psychosis and schizophrenia were evaluated.

267 e mechanism not previously observed in other bipolar pulse cancellation studies.

268 electric field reversal, a phenomenon called bipolar pulse cancellation.

269 romising for the potential use of nanosecond bipolar pulse technologies for remote electrostimulation

270 se) when the delay between each phase of the bipolar pulse was 30 ns.

271 litude of the second (negative) phase of the bipolar pulse was half that of the first (positive) phas

272 The response was eliminated by a 2-ns bipolar pulse with positive and negative phases of equal

273 In this review, we present some of what Bipolar-Schizophrenia Network for Intermediate Phenotype

274 massive Heinrich event, and the onset of the Bipolar Seesaw Mechanism (BSM) that eventually permitted

275 Two modes, related to global climate and the bipolar seesaw, have been proposed previously.

276 ed with a significant increase in RV volume (bipolar: Spearman rho, 0.6965, P=0.006; unipolar: Spearm

277 We find that bipolar-specific accessible regions are more frequently

278 .3% Other ethnicity) at high or low risk for bipolar spectrum disorder (BSD).

279 ng to help focus spindle poles for efficient bipolar spindle assembly.

280 is, Kif11, a kinesin motor protein, promotes bipolar spindle formation and chromosome movement, and d

281 ions have to be met to robustly assemble the bipolar spindle in a multicentrosomal cell: 1) the stren

282 ation favors the establishment of an initial bipolar spindle scaffold, facilitating chromosome captur

283 te chromosomes and simultaneously assemble a bipolar spindle, we developed a computational model of f

284 ary for the formation and maintenance of the bipolar spindle.

285 pha-tubulin or developed an abnormally small bipolar spindle.

286 von Economo neurons (VENs) are bipolar, spindle-shaped neurons restricted to layer 5 of

287 ticentrosomal, yet they are able to assemble bipolar spindles by clustering centrosomes into two spin

288 motors pulling on astral microtubules align bipolar spindles with the interphase long cell axis, wit

289 entrioles/centrosomes and the maintenance of bipolar spindles.

290 Bipolar surface electromyogram (EMG) was recorded from s

291 Together with the demonstration of bipolar synchrony in atmospheric response, this provides

292 that it can spontaneously self-assemble into bipolar synthetic thick filaments (STFs) in low-ionic-st

293 tail-folded antiparallel tetramers, unfolded bipolar tetramers, and small filaments.

294 nce of NaSn(m)BiTe(m+2) is limited by strong bipolar transport and only exhibits modest maximum ZTs ~

295 zation, and use of a massive array of closed bipolar ultramicroelectrodes (UMEs) in electrochemical i

296 The bipolar UME array is 1 cm(2) in size and contains >146 0

297 The bipolar UME array was used in electrochemical imaging to

298 results suggest that microfabricated closed bipolar UME arrays can be useful for imaging fast and tr

299 Bipolar VNS trains of both polarities elicited mixed Del

300 % increase in RV endocardial surface area of bipolar voltage consistent with scar is uncommon during