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1 pment for the treatment of schizophrenia and bipolar I disorder.
2 ecurrence of any mood episode in adults with bipolar I disorder.
3 in recently manic or hypomanic patients with bipolar I disorder.
4 campal region in male patients with familial bipolar I disorder.
5 oliferation in this brain region in familial bipolar I disorder.
6 uronal dysfunction, or neuropil reduction in bipolar I disorder.
7 ing symptoms of acute mania in patients with bipolar I disorder.
8 suicidal behavior in high-risk patients with bipolar I disorder.
9 lated with a poorer outcome in patients with bipolar I disorder.
10 ) than normal comparison subjects (none) had bipolar I disorder.
11 psychotic and 18 nonpsychotic probands with bipolar I disorder.
12 malities were confirmed in the subgroup with bipolar I disorder.
13 t of 124 consecutively treated patients with bipolar I disorder.
14 te of poor outcome in the acute treatment of bipolar I disorder.
15 he acute treatment response of patients with bipolar I disorder.
16 n affectively ill relatives of probands with bipolar I disorder.
17 he psychosocial functioning of patients with bipolar I disorder.
18 he treatment of adults with schizophrenia or bipolar I disorder.
19 th genetic vulnerability to schizophrenia or bipolar I disorder.
20 renia and schizophrenia-related disorders or bipolar I disorder.
21 s role in the treatment of schizophrenia and bipolar I disorder.
22 th major depressive episodes associated with bipolar I disorder.
23 Only 3% met DSM-IV criteria for bipolar I disorder.
24 alysis to family genetic studies of ADHD and bipolar I disorder.
25 reviewing family genetic studies of ADHD and bipolar I disorder.
26 either drug alone for relapse prevention in bipolar I disorder.
27 ed patients with acute mania associated with bipolar I disorder.
28 for 12.2% to bipolar II disorder and 7.5% to bipolar I disorder.
29 rent mood events in patients with stabilized bipolar I disorder.
30 ophrenia and to some extent among those with bipolar I disorder.
31 showed individual genotypic association with bipolar I disorder.
32 pine as monotherapy in relapse prevention in bipolar I disorder.
33 th pharmacotherapy alone in the treatment of bipolar I disorder.
34 enia, schizoaffective disorder, or psychotic bipolar I disorder, 1,055 of their first-degree relative
36 as higher in the 169 trials in patients with bipolar I disorder (30.8%) than the 59 trials in patient
37 zoaffective disorder, and 115 with psychotic bipolar I disorder), 369 of their first-degree relatives
38 f 1,162 women with clinically treated DSM-IV bipolar I disorder (479 pregnancies/283 women), bipolar
39 es were 58 currently depressed patients with bipolar I disorder, 58 age- and sex-matched unipolar dep
40 6%), obsessive-compulsive disorder (9%), and bipolar I disorder (6%) were more common among patients
41 mpared with 4.0% of the asenapine group) and bipolar I disorder (6.3% compared with 1.6%) in the doub
44 nds and a significantly higher prevalence of bipolar I disorder among relatives of ADHD probands.
46 d in 15 euthymic male patients with familial bipolar I disorder and 20 healthy male comparison subjec
47 average density of 1 SNP per 11.9 kb in 323 bipolar I disorder and 274 schizophrenia or schizoaffect
48 rty-four lithium-free euthymic patients with bipolar I disorder and 31 healthy comparison subjects un
49 youths with acute manic or mixed episodes of bipolar I disorder and 60 group-matched healthy controls
50 (18-65 years old) who met DSM-5 criteria for bipolar I disorder and a current depressive episode were
51 Patients with a primary DSM-IV diagnosis of bipolar I disorder and a current manic or mixed episode
53 ood disorder episodes, 12 were patients with bipolar I disorder and a positive history of psychotic s
55 line reduces cocaine use in outpatients with bipolar I disorder and current cocaine dependence and ac
56 postpartum mood disorder are more common in bipolar I disorder and manic and psychotic presentations
58 a trait-related abnormality in patients with bipolar I disorder and that male and female patients sho
59 ing recurrence of mood events in adults with bipolar I disorder and was generally well-tolerated.
60 order, and 129 of individuals with psychotic bipolar I disorder), and 200 healthy comparison subjects
61 s; 39.3% female; 45% with schizophrenia, 36% bipolar I disorder, and 19% schizoaffective disorder), 1
62 e posed: Does stress precipitate episodes of bipolar I disorder, and does sensitivity to stress diffe
64 ubstance use disorders, major depressive and bipolar I disorders, and antisocial and borderline perso
65 se disorder (aOR, 6.60; 95% CI, 2.01-21.67), bipolar I disorder (aOR, 2.39; 95% CI, 1.19-4.80), postt
66 schizoaffective disorder bipolar type (SAB), bipolar I disorder (BD I), and bipolar II disorder (BD I
71 mately 2 years apart for 35 adolescents with bipolar I disorder (BDI) and 37 healthy adolescents.
73 0.17; 95% CI, 0.02 to 0.32, respectively) or bipolar I disorder (beta, 0.20; 95% CI, 0.04 to 0.36 and
74 predicted lower lifetime prevalence rates of bipolar I disorder, bipolar II disorder, and bipolar spe
75 ing of perinatal mood episodes in women with bipolar I disorder, bipolar II disorder, and recurrent m
76 me prevalence rates in various countries for bipolar I disorder, bipolar II disorder, bipolar spectru
77 as affected if they had been diagnosed with bipolar I disorder; bipolar II disorder; or schizoaffect
78 ty in bipolar disorder is based primarily on bipolar I disorder (BP-I) and does not relate disability
79 f weekly symptomatic status of patients with bipolar I disorder (BP-I) during long-term follow-up.
82 enazi pedigrees with a proband affected with bipolar I disorder (BPI) and at least one other member a
83 ion downregulation in euthymic subjects with bipolar I disorder (BPI) and healthy control subjects.
84 ersons diagnosed with schizophrenia (SCZ) or bipolar I disorder (BPI) are at high risk for self-injur
85 ainment of families, most linkage studies of Bipolar I disorder (BPI) have used relatively small samp
86 ien, and judgment filings) for patients with bipolar I disorder (BPI) or schizophrenia, which has lim
87 atives ascertained through a proband who had bipolar I disorder (BPI) were interviewed by a psychiatr
89 schizoaffective disorder, bipolar type, and bipolar I disorder, but the generalizability of these fi
90 to determine whether comorbidity of ADHD and bipolar I disorder constitutes a familial subtype distin
91 f severe and chronic disorders that includes bipolar I disorder, defined by the presence of a syndrom
92 e to the comparison group, the patients with bipolar I disorder demonstrated significantly lower conc
96 rticipants with nondeficit schizophrenia and bipolar I disorder did not show significant differences
97 to cortical disconnectivity in patients with bipolar I disorder (diffusion-weighted magnetic resonanc
98 s, family history, and treatment patterns to bipolar I disorder document the validity of the bipolar
99 renia and related psychotic disorders and/or bipolar I disorder during the study period were included
100 oup, fixed-dose study in adult patients with bipolar I disorder experiencing a current major depressi
103 330 patients aged 16 years and older with bipolar I disorder from 41 sites in the UK, France, USA,
104 on deficit hyperactivity disorder (ADHD) and bipolar I disorder has been documented in clinical and e
105 s provided 12 estimates of the prevalence of bipolar I disorder in 1,877 relatives of ADHD probands a
107 esponse to lithium maintenance treatment for bipolar I disorder in patients of Han Chinese descent.
108 Study participants included 15 patients with bipolar I disorder in the euthymic state and 25 normal c
114 rder phenotype was defined as current DSM-IV bipolar I disorder (manic or mixed phase) with at least
116 of 116 outpatients 7 to 18 years of age with bipolar I disorder, manic or mixed, were recruited at 20
117 ded individuals with schizophrenia (n = 36), bipolar I disorder (n = 29), and healthy controls (n = 3
118 from two academic centers and patients with bipolar I disorder (n = 39) and matched healthy controls
120 st-degree relatives (N=966) of probands with bipolar I disorder (N=192) and schizoaffective disorder,
122 t total of 413 youths (ages 7-17 years) with bipolar I disorder (N=244), bipolar II disorder (N=28),
124 studies that included both individuals with bipolar I disorder (n=4270) and those with bipolar II di
126 2B, IL2RB, and TUBA8) met this criterion for bipolar I disorder; only DAO has been previously associa
128 rence was examined among 1,177 patients with bipolar I disorder or bipolar II disorder, including 458
130 jects were excluded if they met criteria for bipolar I disorder or major depressive disorder with psy
131 lts with a DSM-5 diagnosis of schizophrenia, bipolar I disorder, or schizoaffective disorder who had
133 clinical armamentarium for the management of bipolar I disorder, particularly with respect to prophyl
134 ays relapse into subsequent mood episodes in bipolar I disorder patients who responded to open-label
136 hizophrenia patients with hallucinations, 74 bipolar-I disorder patients with hallucinations, 42 bipo
137 ent dysconnectivity pattern was observed for bipolar-I disorder patients with hallucinations, which i
138 connectivity alterations were found between bipolar-I disorder patients without hallucinations and c
139 -I disorder patients with hallucinations, 42 bipolar-I disorder patients without hallucinations, and
140 s whether a prepubertal and early-adolescent bipolar I disorder phenotype (PEA-BP-I) is the same illn
144 ed magnetic resonance imaging on 25 euthymic bipolar I disorder subjects and 24 gender- and age-equiv
146 marker, was lower in subjects with familial bipolar I disorder than in healthy comparison subjects,
147 ng chart review of 184 adult inpatients with bipolar I disorder, the authors assessed patients' past
148 ve been implicated in the pathophysiology of bipolar I disorder, the neural mechanisms underlying bip
149 We compared this prevalence in people with bipolar I disorder versus those with bipolar II disorder
150 ing of psychosis was also apparent when only bipolar I disorder was considered the affected phenotype
151 follow-up, from adolescence into adulthood, bipolar I disorder was rare among bipolar offspring.
152 ates of suicide attempts among patients with bipolar I disorder were compared to rates during a 2-yea
157 Subjects with Research Diagnostic Criteria bipolar I disorder were prospectively followed up for as
160 Families ascertained through probands with bipolar I disorder were stratified into three groups bas
161 redicted mood-incongruence in relatives with bipolar I disorder when compared with all other subjects
163 recurrence of mood episodes in patients with bipolar I disorder who had recently experienced a manic
164 atients and outpatients age 60 or older with bipolar I disorder who presented with a manic, hypomanic
166 s were 65 patients in the depressed phase of bipolar I disorder who were enrolled in a larger ongoing
167 mples from one subgroup of 294 patients with bipolar I disorder who were receiving lithium treatment.
168 of 1761 patients of Han Chinese descent with bipolar I disorder who were recruited by the Taiwan Bipo
169 ment outcome in a group of 175 patients with bipolar I disorder who were treated for an acute affecti
170 4), schizoaffective disorder (z = -1.2), and bipolar I disorder with psychosis (z = -0.5) all had sig
171 = -0.29, schizoaffective disorder z = -0.15, bipolar I disorder with psychosis z = -0.13), and antips
174 schizophrenia, schizoaffective disorder, and bipolar I disorder with psychosis) is as important for d
175 541 with schizoaffective disorder, 457 with bipolar I disorder with psychosis, and 823 relatives of
176 schizophrenia, schizoaffective disorder, or bipolar I disorder with psychosis; and relatives of pati
179 schizoaffective disorder [SAD; N = 129] and bipolar I disorder with psychotic features [BPD+; N = 26
180 iduals with schizophrenia and 41 people with bipolar I disorder with psychotic features in order to:
181 c remission from a manic or mixed episode of bipolar I disorder (Young Mania Rating Scale [YMRS] tota