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1 um disorder and 49.7% (95% CI 48.1-51.3) for bipolar affective disorder.
2          Lithium is a first-line therapy for bipolar affective disorder.
3  (95% CI, 1.6-4.5) times more likely to have bipolar affective disorder.
4 (95% CI, 2.7-20.6) times more likely to have bipolar affective disorder.
5 ived neurotrophic factor in the aetiology of bipolar affective disorder.
6 s being overrepresented in the patients with bipolar affective disorder.
7 may help define more homogeneous subtypes of bipolar affective disorder.
8 ctive disorder (N=26 380), of which 1928 had bipolar affective disorder.
9 everity and course, is a familial feature of bipolar affective disorder.
10  some neuropsychiatric conditions, including bipolar affective disorder.
11 ociated gene expression in schizophrenia and bipolar affective disorder.
12 re effective mood-stabilizing treatments for bipolar affective disorder.
13 es with exclusively maternal transmission of bipolar affective disorder.
14 ts and lymphocytes of euthymic patients with bipolar affective disorder.
15 ion of lithium and in the pathophysiology of bipolar affective disorder.
16 ng are associated with functional decline in bipolar affective disorder.
17 cludes schizophrenia as well as unipolar and bipolar affective disorders.
18 is relation is present for both unipolar and bipolar affective disorders.
19 rated for men and women and for unipolar and bipolar affective disorders.
20  of foreign residence had increased IRRs for bipolar affective disorder, affective disorders, persona
21 typing was carried out in 1099 patients with bipolar affective disorder and 1152 healthy comparator i
22 and lymphocytes of 44 euthymic patients with bipolar affective disorder and 27 matched comparison sub
23 f the standard antimanic treatments used for bipolar affective disorder and 38% of the treatments use
24 nd D21S171, a region which includes loci for bipolar affective disorder and a recessive form of deafn
25  set including 67 males and 113 females with bipolar affective disorder and a similar number of match
26 ty at illness onset is a familial feature of bipolar affective disorder and is associated with import
27 e results suggest a molecular basis for both bipolar affective disorder and its treatment.
28 alence of a diagnosis of SMI (schizophrenia, bipolar affective disorder and other psychoses, and othe
29                      A possible link between bipolar affective disorder and schizophrenia remains arg
30 nosis, implicating dopamine dysregulation in bipolar affective disorder and schizophrenia, in line wi
31                              In the study of bipolar affective disorder and schizophrenia, there is s
32  familial spastic paraplegia, schizophrenia, bipolar affective disorder and spinocerebellar ataxia ty
33  gene for schizophrenia, was associated with bipolar affective disorder and tested this hypothesis us
34  previous reports of high Gs alpha levels in bipolar affective disorder and, furthermore, suggest tha
35 llection, consisting of 15 schizophrenia, 15 bipolar affective disorder, and 15 control brains.
36  in three or more generations, an absence of bipolar affective disorder, and a single progenitor sour
37 chiatric disorders, including schizophrenia, bipolar affective disorder, and borderline personality d
38 ng schizophrenia, major depressive disorder, bipolar affective disorder, and obsessive compulsive dis
39 ss schizophrenia, major depressive disorder, bipolar affective disorder, and OCD.
40  are likely to contain genes contributing to bipolar affective disorder are also relevant to schizoph
41                 Molecular genetic studies of bipolar affective disorder are beginning to show some po
42                            Schizophrenia and bipolar affective disorder are chronic, disabling illnes
43 thesis that lithium's therapeutic effects in bipolar affective disorder are mediated by alterations i
44                                              Bipolar affective disorder (BD) is a severe psychiatric
45  An increased rate has also been reported in bipolar affective disorder (BD).
46 usly, we demonstrated evidence of linkage to bipolar affective disorder (BP) in a single large, multi
47                                              Bipolar affective disorder (BP) is a common, highly heri
48                                              Bipolar affective disorder (BP) is a major neuropsychiat
49 ), have been implicated in susceptibility to bipolar affective disorder (BP) through genome-wide asso
50  in 110 parent-offspring trios affected with bipolar affective disorder (BP).
51 clarify the issue of genetic linkage between bipolar affective disorder (BPAD) and chromosome 18q, co
52                                              Bipolar affective disorder (BPAD) and schizophrenia (SZ)
53                                              Bipolar affective disorder (BPAD) is a complex disease w
54          Decades of longitudinal research on bipolar affective disorder (BPAD) revealed cosegregation
55 have reported evidence suggesting linkage of bipolar affective disorder (BPAD) to chromosome 18.
56 tics, and to have comorbid depression, mania/bipolar affective disorder (BPAD), hypertension, central
57 s in recurrent depressive disorder (RDD) and bipolar affective disorder (BPAD).
58                                              Bipolar affective disorder (BPAD; manic-depressive illne
59                              Twin studies of bipolar affective disorder (BPD) have either been small
60 pletion may be the way that lithium works in bipolar affective disorder, but others have suggested th
61 and RFLP at the monoamine oxidase A locus in bipolar affective disorder cases and controls in the UK
62 hospitalization with nonaffective psychosis, bipolar affective disorder, depressive disorder, eating
63                                Patients with bipolar affective disorder differed from those with unip
64  in participants with BD, measured using the Bipolar Affective Disorder Dimensional Scale.
65           There is also linkage evidence for bipolar affective disorder, epilepsy and autism in this
66 s, a significant proportion of patients with bipolar affective disorder experience frequent relapses.
67 rate of lithium monotherapy for treatment of bipolar affective disorder (from 84% to 43%) and schizoa
68  the actual treatment of schizoaffective and bipolar affective disorders had changed in light of rece
69 , and SAP102 in subjects with schizophrenia, bipolar affective disorder I, and a comparison group.
70 olecules in hippocampus in schizophrenia and bipolar affective disorder I.
71 earby locus contributes to susceptibility to bipolar affective disorder in some families.
72  bipolar affective disorder, suggesting that bipolar affective disorder in the Old Order Amish is inh
73                 The main clinical feature of bipolar affective disorder is a change of mood to depres
74                                              Bipolar affective disorder is a common neuropsychiatric
75 cifically designed for relapse prevention in bipolar affective disorder is a useful tool in conjuncti
76                                              Bipolar affective disorder is clinically heterogeneous,
77                         Their involvement in bipolar affective disorder is much less prominent.
78            Rapid cycling among patients with bipolar affective disorders is important because of its
79 peutic efficacy in manic-depressive illness (bipolar affective disorder) is the inositol depletion hy
80 chizophrenia, schizoaffective disorders, and bipolar affective disorders) is well described, but litt
81          The most characteristic features of bipolar affective disorder (manic-depressive illness) ar
82                                              Bipolar affective disorder (manic-depressive illness) is
83                Molecular genetic research in bipolar affective disorder may lead to the development o
84 polygenic diseases, including schizophrenia, bipolar affective disorder, non-insulin-dependent diabet
85 the apparent excess maternal transmission of bipolar affective disorder observed in some families.
86                 The results demonstrate that bipolar affective disorder occurs across all of the majo
87                         Reported linkages of bipolar affective disorder on chromosomes 11, 18, 21 and
88 unilineal multiplex families segregating the bipolar affective disorder phenotype.
89 f dementia, hereditary ataxia, Parkinsonism, bipolar affective disorder, schizophrenia and autism.
90 pecifically designed to prevent relapses for bipolar affective disorder showed encouraging results wh
91 onsidered a good candidate to investigate in bipolar affective disorder since this enzyme plays an im
92 rs and impulsivity), DRD3 (schizophrenia and bipolar affective disorder), SLC6A3 (susceptibility to c
93 6, 13 and 15 harbour susceptibility loci for bipolar affective disorder, suggesting that bipolar affe
94 confirm or refute previous reports that link bipolar affective disorder to polymorphic DNA markers at
95 s (average age = 34 +/- 16.5) diagnosed with Bipolar Affective Disorder to three patient groups all d
96 cation of candidate genes that predispose to bipolar affective disorder, to the completion of the seq
97 idual with an atypical movement disorder and bipolar affective disorder type II contains 46 triplets,
98                                   Those with bipolar affective disorder were more likely to receive s
99 ene may play a role in the susceptibility to bipolar affective disorder, which underscores a potentia