ライフサイエンスコーパス: bisphenol A

1 micals such as phthalates and phenols (e.g., bisphenol A).

2 ahistidine peptide for the quantification of bisphenol A.

3 ates, diethylhexyl adipate, alkylphenols and bisphenol A.

4 her than the corresponding concentrations of bisphenol A.

5 co-crystallized with an alkyl derivative of bisphenol A.

6 tly with K-Ras and that Rheb weakly binds to bisphenol A (10) and 4,4'-biphenol derivatives.

7 We show for the first time that bisphenol A (10) has the capacity to interact directly w

8 Our results propose a new mode of action for bisphenol A (10) that advocates a reduced exposure to th

9 Bisphenol A (2,2-bis[4-hydroxyphenyl]propane, BPA), the

10 trations of BADGE and its three derivatives, bisphenol A (2,3-dihydroxypropyl) glycidyl ether [BADGE.

11 water consumption was associated with higher bisphenol A, 2,4-dichlorophenol, and 2,5-dichlorophenol

12 Ethyl-paraben, bisphenol A, 2,5-dichlorophenol, and DIDP tended to be a

13 e concentrations of 8 phthalate metabolites, bisphenol A, 25 polychlorinated biphenyls (PCBs), 6 orga

14 hydroxypropyl) glycidyl ether [BADGE.H(2)O], bisphenol A (3-chloro-2-hydroxypropyl) (2,3-dihydroxypro

15 the approach allows the direct detection of bisphenol A, a low molecular weight (228 Da) target usua

16 an environmentally relevant concentration of bisphenol A, a nearly ubiquitous environmental contamina

17 We investigated whether bisphenol-A, a prominent contaminant with endocrine-disr

18 Upon bisphenol A addition, the competitive dissociation of th

19 midispersive solid phase extraction of eight bisphenols (A, AF, AP, C, BP, G, M, and Z) from river wa

20 By combination with an antibody directed to bisphenol A (alphaBPA), this conducting polymer-based bi

21 Bisphenol A, an endocrine disrupting compound, is widely

22 ite dental restoration materials may release bisphenol-A, an endocrine-disrupting chemical.

23 PA), 2,4-di-tert-butylphenol, and a possible bisphenol A analog.

24 n sample age groups for case study chemicals bisphenol A and 2,5-dichlorophenol.

25 Bisphenol A and adiposity in an inner-city birth cohort.

26 not identified for the nonantimicrobial EDCs bisphenol A and benzophenone-3 (P > .2).

27 ime polymerase chain reaction indicated that bisphenol A and bisphenol AF consistently activated endo

28 e strain CBDB1 completely converted TBBPA to bisphenol A and BPB to phenol red with a stepwise remova

29 With the exceptions of bisphenol A and DEET, all TOrCs that were detected in th

30 ansgenic reporter demonstrated that the EEDs bisphenol A and genistein preferentially activate estrog

31 Combined effects of high-dose bisphenol A and oxidizing agent (KBrO3) on cellular micr

32 Human exposure to phenols, including bisphenol A and parabens, is widespread.

33 cals as EDCs, and we use diethylstilbestrol, bisphenol A and perchlorate as examples to illustrate th

34 Urinary concentrations of bisphenol A and phthalate metabolites measured during pr

35 Recent fast food consumption and bisphenol A and phthalates exposures among the U.S. popu

36 minent plastic-associated chemicals, such as bisphenol A and phthalates, have been thoroughly studied

37 g and toxic pollutants, many of which (e.g., bisphenol A and substituted phenols) are known to be ins

38 ants of emerging concern were performed with bisphenol A and sulfamethoxazole.

39 dichlorophenols, triclosan, benzophenone-3, bisphenol A) and 11 phthalate metabolites were measured

40 b copper, 5 ppb cadmium, 10 ppb lead, 10 ppb bisphenol A, and 1 ppb paraoxon, tested as safety limits

41 environmental contaminants (benzo[a]pyrene, bisphenol A, and acrolein) in human serum via a competit

42 to exhibit the highest sorption capacity for bisphenol A, and it was almost irreversible, probably du

43 al smoking/nicotine, organotins, phthalates, bisphenol A, and pesticides.

44 hyl paraben, propyl paraben, benzophenone-3, bisphenol A, and triclosan in urine collected during 199

45 unds that contain a phenolic ring (parabens, bisphenol A, and triclosan) were completely transformed

46 lphenol, octylphenol, ibuprofen, diclofenac, bisphenol A, and triclosan.

47 l molecules, as we demonstrated with a novel bisphenol A aptamer.

48 We demonstrate that bisphenol A as the template in combination with phthaloy

49 hemical detection of the endocrine disruptor bisphenol A, as well as the capture of living nonadheren

50 ained from horse radish peroxidase (HRP) and bisphenol A assays were 12.5 ng/ml (2.84x10(-10) M ) and

51 ere used to examine the sorption affinity of bisphenol A, atrazine, and diuron to five soils of varyi

52 ata values, with diethyl hexyl phthalate and bisphenol A being the most ubiquitous compounds detected

53 Exposure to environmental phenols (e.g., bisphenol A, benzophenone-3, and triclosan) and parabens

54 erum testosterone concentrations and urinary bisphenol A, benzophenone-3, triclosan, and paraben leve

55 ihydroxypropyl) ether [BADGE.HCl.H(2)O], and bisphenol A bis (2,3-dihydroxypropyl) ether [BADGE.2H(2)

56 esorcinol bis(diphenylphosphate) (RBDPP) and bisphenol A bis(diphenylphosphate) (BPA-BDPP) are two ha

57 Bisphenol A, bisphenol AF, and 2-2-bis(p-hydroxyphenyl)-

58 Bisphenol A, bisphenol S, and 4-hydroxyphenyl 4-isoproox

59 Environmental exposure to bisphenol A (BPA) affects mammary gland development in r

60 hinylestradiol (EE2)] and two xenoestrogens [bisphenol A (BPA) and 4-n-nonyl-phenol (NP)] in surface

61 Bisphenol A (BPA) and bisphenol A diglycidyl ether (BADG

62 The thermal degradation of bisphenol A (BPA) and bisphenol S (BPS) was investigated

63 al was to provide a definitive evaluation of bisphenol A (BPA) and explain disparities between tradit

64 Bisphenol A (BPA) and its brominated derivative tetrabro

65 The health effects related to bisphenol A (BPA) and its exposure sources have undergon

66 cerns exist regarding children's exposure to bisphenol A (BPA) and other phenols because of the highe

67 the impact of serially assessed exposure to bisphenol A (BPA) and phthalates on measures of kidney f

68 ence regarding associations for exposures to bisphenol A (BPA) and phthalates with type 2 diabetes (T

69 is growing concern that prenatal exposure to bisphenol A (BPA) and phthalates, which are widely used

70 The endocrine disruptor bisphenol A (BPA) and the pharmaceutical 17alpha-ethinyl

71 Phthalates and bisphenol A (BPA) are endocrine disruptors, and previous

72 Phthalates and bisphenol A (BPA) are used in some personal care product

73 Phthalates and bisphenol A (BPA) are widely used industrial chemicals t

74 Increasing concern over bisphenol A (BPA) as an endocrine-disrupting chemical an

75 cale sequencing batch reactors (SBRs), using bisphenol A (BPA) as the TOrC.

76 erns, variability, and predictors of urinary bisphenol A (BPA) concentrations in 337 children from th

77 ective optical sensor to detect and quantify Bisphenol A (BPA) contamination.

78 ivity, bisphenol S (BPS) as a substitute for bisphenol A (BPA) could actually increase the risk of en

79 ry method for the determination of different bisphenol A (BPA) derivatives such as bisphenol A diglyc

80 ent study revealed that prenatal exposure to bisphenol A (BPA) disrupted the transcriptome profiles o

81 A positive association between Bisphenol A (BPA) exposure and coronary heart disease ha

82 n expert panel reviewed associations between bisphenol A (BPA) exposure and reproductive health outco

83 We used in utero bisphenol A (BPA) exposure as a model environmental expo

84 Bisphenol A (BPA) exposure early in life results in orga

85 Prenatal bisphenol A (BPA) exposure has been associated with adve

86 Gestational phthalate and bisphenol A (BPA) exposure may increase the risk of adve

87 ticipants previously randomly selected for a bisphenol A (BPA) exposure study who had unusually high

88 Human health risks due to bisphenol A (BPA) exposure through canned food consumpti

89 As concern regarding the toxic effects of bisphenol A (BPA) grows, BPA in many consumer products i

90 As the evidence of the toxic effects of bisphenol A (BPA) grows, its application in commercial p

91 Early-life exposure to bisphenol A (BPA) has been implicated to play a role in

92 Bisphenol A (BPA) has been linked with pediatric asthma

93 Exposure to bisphenol A (BPA) has been reported to alter global gene

94 Prenatal exposure to nonylphenol (NP) and/or bisphenol A (BPA) has been reported to be associated wit

95 Widespread environmental contamination by bisphenol A (BPA) has created the need to fully define i

96 widespread environmental disrupting chemical bisphenol A (BPA) has estrogenic activity, but its impli

97 Pressures to ban bisphenol A (BPA) has led to the use of alternate chemic

98 Although prenatal exposure to bisphenol A (BPA) has recently been associated with the

99 We report on an innovative heterogeneous bisphenol A (BPA) immunoassay based on an electrolyte-ga

100 try, has been developed for determination of bisphenol A (BPA) in canned tomatoes.

101 ead use of the endocrine disrupting chemical bisphenol A (BPA) in consumer products has resulted in n

102 The determination of bisphenol A (BPA) in foods and beverages sold in Turkish

103 orted the occurrence of compounds other than bisphenol A (BPA) in sediment.

104 s increasingly been used as a substitute for bisphenol A (BPA) in some "BPA-free" consumer goods and

105 ectrochemical biosensor for the detection of Bisphenol A (BPA) in water has been developed by immobil

106 y bottles, little attention has been paid to bisphenol A (BPA) intake from packaged water consumption

107 Bisphenol A (BPA) is a base chemical used extensively in

108 Bisphenol A (BPA) is a common chemical used in the manuf

109 Bisphenol A (BPA) is a high production volume chemical u

110 Bisphenol A (BPA) is a high-production-volume chemical a

111 Bisphenol A (BPA) is a synthetic chemical widely used in

112 Bisphenol A (BPA) is a ubiquitous compound that is emerg

113 Bisphenol A (BPA) is a well-known endocrine disruptor th

114 Bisphenol A (BPA) is a widely used chemical that has bee

115 Bisphenol A (BPA) is an endocrine disrupting compound th

116 Bisphenol A (BPA) is an endocrine disruptor and potentia

117 Bisphenol A (BPA) is an endocrine disruptor frequently d

118 Bisphenol A (BPA) is an endocrine disruptor that affects

119 Bisphenol A (BPA) is an endocrine-disrupting compound wi

120 Bisphenol A (BPA) is an environmental endocrine disrupto

121 Bisphenol A (BPA) is an estrogenic endocrine disruptor w

122 Bisphenol A (BPA) is an industrial compound and a well k

123 The plastic additive bisphenol A (BPA) is commonly found in landfill leachate

124 Bisphenol A (BPA) is found in polycarbonate plastic and

125 Bisphenol A (BPA) is known to be biologically active in

126 The plastic monomer bisphenol A (BPA) is one of the highest production volum

127 ure to the environmental endocrine disruptor bisphenol A (BPA) is ubiquitous and associated with the

128 Bisphenol A (BPA) is used widely to manufacture food con

129 Bisphenol A (BPA) is widely recognized being an endocrin

130 The endocrine-disrupting chemical bisphenol A (BPA) is widely used in food and beverage pa

131 Bisphenol A (BPA) is widely used in the manufacture of p

132 Bisphenol A (BPA) is widely used in the manufacture of p

133 Laboratory studies show that bisphenol A (BPA) leaches from bisphenol A-glycidyl meth

134 products from store shelves after reports of bisphenol A (BPA) leaching from baby bottles, reusable d

135 oprobe is designed for the quantification of bisphenol A (BPA) levels in the blood after human exposu

136 rly-life exposure to the endocrine disruptor bisphenol A (BPA) may contribute to the development of o

137 tory actions and public concerns, the use of bisphenol A (BPA) may decrease, while the use of BPA alt

138 Early-life exposure to bisphenol A (BPA) may increase childhood obesity risk, b

139 1.0 microM) and a response time of 250 s for bisphenol A (BPA) monitoring.

140 Effects of bisphenol A (BPA) on ovarian transcript profiles as well

141 Certain phthalates and bisphenol A (BPA) show reproductive effects in animal st

142 The lowest concentration of bisphenol A (BPA) that induces a response was ~0.1 mg/L

143 Humans are exposed to low-dose bisphenol A (BPA) through plastic consumer products and

144 ion on the homogeneous Fenton degradation of bisphenol A (BPA) used as a model pollutant, was investi

145 The xenoestrogen bisphenol A (BPA) used in the manufacturing of various p

146 However, bisphenol A (BPA) was identified by chemical:gene covari

147 In this study, bisphenol A (BPA) was selected as a model compound.

148 The electrochemistry of bisphenol A (BPA) was studied by voltammetric methods at

149 to replace the endocrine disrupting chemical bisphenol A (BPA) while exerting estrogenic effects of c

150 scharge of the endocrine disrupting compound bisphenol A (BPA) with wastewater treatment plant (WWTP)

151 sed on the MS results these were assigned to bisphenol A (BPA), 2,4-di-tert-butylphenol, and a possib

152 Bisphenol A (BPA), a chemical incorporated into plastics

153 Studies have implicated exposure to bisphenol A (BPA), a commonly used chemical, in the deve

154 t chronic exposure of synthetic xenoestrogen bisphenol A (BPA), a component of consumer plastic produ

155 Bisphenol A (BPA), a component of some dialysis membrane

156 Bisphenol A (BPA), a high volume production chemical com

157 Bisphenol A (BPA), a manufactured chemical, is found in

158 Bisphenol A (BPA), a plastics component, has been descri

159 Bisphenol A (BPA), a prevalent endocrine-disrupting chem

160 Bisphenol A (BPA), a ubiquitous endocrine disruptor that

161 al models suggests that prenatal exposure to bisphenol A (BPA), a ubiquitous endocrine-disrupting che

162 Bisphenol A (BPA), a widely used endocrine-disrupting ch

163 Exposure of humans to bisphenol A (BPA), a widely used industrial chemical, is

164 S (BPS) are increasingly used to substitute bisphenol A (BPA), a widespread environmental endocrine

165 Bisphenol A (BPA), a xenoestrogenic endocrine-disrupting

166 perimental laboratory evidence suggests that bisphenol A (BPA), an endocrine disruptor, is a neurodev

167 Bisphenol A (BPA), an endocrine-disrupting chemical that

168 Bisphenol A (BPA), an endocrine-disrupting compound with

169 Bisphenol A (BPA), an environmental contaminant with wea

170 to investigate the carcinogenic property of Bisphenol A (BPA), an environmental estrogen, by long-te

171 tions have been reported between phthalates, bisphenol A (BPA), and child behavior.

172 Phthalate esters (PEs), bisphenol A (BPA), and parabens (PBs), which are used in

173 crine-disrupting chemicals (EDCs), including bisphenol A (BPA), are environmental ubiquitous pollutan

174 We examined the association of bisphenol A (BPA), benzophenone-3 (BP-3), triclosan (TCS

175 phenoxyethanol (2-PE), 4-nonylphenol (4-NP), bisphenol A (BPA), benzylbuthyl phthalate (BBP) and dime

176 al samples were analyzed for the presence of bisphenol A (BPA), bis (2-ethylhexyl) phthalate (DEHP),

177 like activity is attributed to EDCs, such as bisphenol A (BPA), bisphenol AF (BPAF), and zearalenone

178 ion of bisphenol F diglycidyl ether (BFDGE), bisphenol A (BPA), bisphenol B (BPB), bisphenol F (BPF)

179 e promising lignin-derivable alternatives to bisphenol A (BPA), but limited bioassay data are availab

180 Three selected chemicals, bisphenol A (BPA), diethylhexyl phthalate (DEHP) and min

181 ironment of some of these chemicals, such as bisphenol A (BPA), external contamination during handlin

182 One such exposure, bisphenol A (BPA), has been associated with obesity and

183 e effect of one suspicious food contaminant, bisphenol A (BPA), in vivo.

184 of environmental contaminants, particularly bisphenol A (BPA), is understudied with regard to PTB.

185 docrine disrupting compounds (EDCs), such as bisphenol A (BPA), may cause adverse health effects in w

186 vestigated the direct effects of exposure to bisphenol A (BPA), mono-n-butyl phthalate (Pht), and pol

187 in (GEN), and in industrial chemicals, e.g., bisphenol A (BPA), present in plastics and other product

188 With the pressure to ban or limit the use of bisphenol A (BPA), production of alternatives such as bi

189 on ROS scavengers inhibited the oxidation of bisphenol A (BPA), suggesting the possible involvement o

190 hlorophenol (25-DCP), benzophenone-3 (BP-3), bisphenol A (BPA), triclosan (TCS), butyl paraben (B-PB)

191 Analytes included parabens, phthalates, bisphenol A (BPA), triclosan, ethanolamines, alkylphenol

192 s of a single endocrine disrupting compound, bisphenol A (BPA), was developed.

193 sh register receipts, contain high levels of bisphenol A (BPA).

194 e electrochemical biosensor for detection of bisphenol A (BPA).

195 ting endocrine-disrupting chemicals, such as bisphenol A (BPA).

196 hesis-based studies to assess the effects of bisphenol A (BPA).

197 oplasia from early-life exposure to low-dose bisphenol A (BPA).

198 te (ICC: 0.55), whereas the ICC was 0.39 for bisphenol A (BPA).

199 for label-free and single step detection of Bisphenol A (BPA).

200 like estrogen, the well-studied xenoestrogen bisphenol A (BPA, a plastics monomer), induces strong br

201 A-seq in endometrial cancer cells exposed to bisphenol A (BPA; found in plastics), genistein (GEN; fo

202 ndocrine disrupting chemicals (EDCs) such as bisphenol-A (BPA) and diethylstilbestrol (DES).

203 health hazards related to persisting use of bisphenol-A (BPA) are well documented.

204 In contrast, Bisphenol-A (BPA) elicited a non-monotonic response.

205 The endocrine disruptor Bisphenol-A (BPA) has been shown to modulate estrogenic,

206 Bisphenol-A (BPA) is an endocrine disrupting chemical us

207 Bisphenol-A (BPA) is an environmentally ubiquitous estro

208 Bisphenol-A (BPA) is widely used in the manufacture of p

209 Bisphenol-A (BPA), a potent endocrine disrupting compoun

210 Exposure to Bisphenol-A (BPA), an endocrine disruptor used in plasti

211 lastic derived endocrine disruptor compounds bisphenol-A (BPA), bis(2-ethylhexyl)phthalate (DEHP) and

212 ny endocrine disrupting chemicals, including bisphenol-A (BPA), induce non-monotonic dose response (N

213 t famous member of this family of compounds, bisphenol-A (BPA), when spiked (at 1 mug.g(-1) concentra

214 s had overall false positive rates of </=5%, bisphenol A, caffeine, NP, OP, and triclosan had false p

215 The adsorption and partition of bisphenol A can be quantified using a dual-mode model of

216 tion of phenolic compounds including phenol, bisphenol A, catechol and cresols is reported.

217 Linking Academic and Regulatory Insights on Bisphenol-A (CLARITY-BPA) is a rare collaboration of gui

218 the formation of chlorinated derivatives of Bisphenol A (ClxBPA), that have higher estrogenic activi

219 fication and temporal variability in urinary bisphenol A concentrations among couples in Utah-the HOP

220 in diastolic BP were observed with maternal bisphenol-A concentrations (0.7 mm Hg [95% CI: 0.1 to 1.

221 e patterns were observed on spin-coated poly(bisphenol A decane ether) (BA-C10) films prepared with c

222 e were no measured chlorinated byproducts of bisphenol A despite occasionally high concentrations of

223 We present here a proof-of-concept for bisphenol A detection; the device could be readily adapt

224 Despite reports of the occurrence of bisphenol A diglycidyl ether (BADGE) and its derivatives

225 Epoxy resins incorporating bisphenol A diglycidyl ether (BADGE) and novolac glycidy

226 p-Hydroxybenzoic acid esters (parabens) and bisphenol A diglycidyl ether (BADGE) are widely present

227 A), bisphenol B (BPB), bisphenol F (BPF) and bisphenol A diglycidyl ether (BADGE) down to 0.50ng/mL;

228 ferent bisphenol A (BPA) derivatives such as bisphenol A diglycidyl ether (BADGE), bisphenol F diglyc

229 Bisphenol A (BPA) and bisphenol A diglycidyl ether (BADGE), used in manufactur

230 Bisphenol A diglycidyl ether (BADGE)- and bisphenol F di

231 ly lacquered with epoxy resins (ER) based on bisphenol A diglycidyl ether (BADGE).

232 against similarly filled methacrylate-based bisphenol A diglycidyl ether dimethacrylate / triethylen

233 PPARgamma antagonists (bisphenol A diglycidyl ether or GW9662) blunted the effe

234 th were randomized to amalgam or composites (bisphenol-A-diglycidyl-dimethacrylate composite for perm

235 cerol dimethacrylate (PMGDM) and ethoxylated bisphenol A dimethacrylate (EBPADMA) at mass ratio of 1:

236 xanediol dimethacrylate (HDDMA), ethoxylated bisphenol A dimethacrylate (EBPADMA), and urethane dimet

237 lectivity towards closely related compounds (bisphenol A dimethacrylate, and dibutyl phthalate).

238 lutants) and nonpersistent (some phthalates, bisphenol A) environmental pollutants, and exposure to a

239 d respiratory health, with ethyl-paraben and bisphenol A exhibiting some consistency across respirato

240 Bisphenol A exposure alters placentation and causes pree

241 ne the multigenerational effects of maternal bisphenol A exposure on mouse pancreatic islets.

242 trogen response after in utero xenoestrogen (bisphenol-A) exposure.

243 gel-filled, and water-filled (most labeled "bisphenol A-free"), were collected from the U.S. market

244 d disinfection byproducts of salicylic acid, bisphenol A, gemfibrozil, naproxen, diclofenac, technica

245 which factor, the dimethacrylate component, bisphenol A glycerolate dimethacrylate (BisGMA) or photo

246 2-hydroxyethyl methacrylate (HEMA) and 5% of bisphenol A glycidyl dimethacrylate (BisGMA) (PEHB).

247 The monomer mixture consisted of bisphenol A glycidyl methacrylate (BisGMA), hexanediol d

248 als composed of a trimethacrylate monomer or bisphenol A glycidyl methacrylate, which is a monomer st

249 ies show that bisphenol A (BPA) leaches from bisphenol A-glycidyl methacrylate (bisGMA)-based dental

250 Bisphenol A has been reported to be a ubiquitous contami

251 for rapid detection of estrogenic substance (bisphenol A) has been proposed.

252 Several functional substitutes of bisphenol A have been proposed in the literature, produc

253 ens, including the environmental contaminant bisphenol A, have also been linked to reproductive probl

254 The change in the amount of bisphenol A in all of these food, based on expiration da

255 compounds 2,4-dichlorophenol, triclosan, and bisphenol A in chlorinated waters containing I(-).

256 In this study, the sorption and release of bisphenol A in marine water were investigated through la

257 osensor capable of detecting the presence of bisphenol A in ultra-low concentrations, yielding a wave

258 Gestational bisphenol-A in male mice primed macrophages in adulthood

259 that gestational, but not adult, exposure to bisphenol-A increased the development of experimental au

260 see that in these kind of food the amount of bisphenol A increases with an increase in the amount of

261 Finally, we observe that bisphenol A induces rapid and parallel modulation of 5S

262 visualization of dose-dependent induction of Bisphenol A inducible genes showed a weak gene activatio

263 e proved to be a persistent source releasing bisphenol A into aquatic environments.

264 Bisphenol A is an oil-derived, large market volume chemi

265 However, bisphenol A is not considered safe due to its endocrine

266 Bisphenol A is widely used in food and drinks packaging.

267 Bisphenol A led to a wide range of stress responses, and

268 Early life exposure to bisphenol-A may represent an environmental consideration

269 factor blocked susceptibility to disease in bisphenol-A mice.

270 method was applied for the determination of bisphenol A migrated from some polycarbonate plastic pro

271 ribe a mixture of phthalates, pesticides and bisphenol A (mixture N1) detected in pregnant women of t

272 The binding of bisphenol A onto the polymer film was successfully chara

273 ct on pbPPARG, while hexabromocyclododecane, bisphenol A, oxychlordane, and endosulfan were weak anta

274 hromic activation of spiropyran in high T(g) bisphenol A polycarbonate.

275 or by four major endocrine disruptor classes:bisphenol A, polychlorinated biphenyls, organophosphates

276 The trends between bisphenol A pregnancy level and increased asthma and bro

277 lently modified with an aptamer specific for bisphenol A recognition.

278 Bisphenol-A's rising concentrations in the environment r

279 , the UV filter benzophenone-2 (BP2) and the bisphenol A substitute bisphenol S (BPS) was deciphered

280 w and comparison of the hormonal activity of bisphenol A substitutes.

281 ed for phthalates and to a lesser extent for bisphenol A, synthetic musks, some pesticides, and PAHs.

282 n estrogen receptor alpha (hERalpha), called bisphenol A-targeted receptor (BPA-R).

283 intramolecular glycosylations using a rigid bisphenol A template supplemented with linkers of variou

284 ith FEV1% (beta=-0.59; 95% CI: -1.24, 0.05); bisphenol A tended to be associated with increased rates

285 f other xenoestrogens (e.g., alkylphenols or bisphenol A) that are sometimes found in surface water.

286 free chlorine alone, while for triclosan and bisphenol A the free iodine mediated transformations wer

287 actone, genistein, daidzein, benzophenone-3, bisphenol A, the sum of parabens (methyl, ethyl, and pro

288 compounds detected, one plastics component (bisphenol A), three pharmaceuticals (carbamazepine, sulf

289 rders of magnitude from a low of 0.6 ppm for bisphenol A to a high of > 180,000 ppm for methyl parabe

290 n significantly reduce the teratogenicity of bisphenol A, triclosan and 17alpha-ethinyl estradiol wit

291 pathways have been relatively well studied, bisphenol A, triclosan, and ibuprofen.

292 Other exposures (total parabens, bisphenol A, triclosan, benzophenone-3, total phthalates

293 oducts, namely methylparaben, propylparaben, bisphenol A, triclosan, galaxolide, and 4- methylbenzili

294 utrition Examination Survey in which urinary bisphenol A; triclosan; benzophenone-3; propyl, methyl,

295 dence of associations between raised urinary bisphenol A (uBPA) and increased incidence of reported c

296 at levels ranging from 0.6 to 4.6ngg(-1) and bisphenol A was detected in all the samples at concentra

297 e, phthalates, fatty acids, alkylphenols and bisphenol A, were quantified.

298 y diarylpropionitrile, genistein, equol, and bisphenol A, whereas its coactivation at the AP-1 site i

299 cles can serve as transportation vectors for bisphenol A, which may constitute an ecological risk.

300 mmonly targeted EDC and their isobars (e.g., bisphenol A, (Z)- and (E)-diethylstilbestrol, hexestrol,