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1                                        Seven bitemporal amnesic patients and 7 controls were randomly
2 al dementia is clinically heterogeneous with bitemporal and inferior frontal lobe dysfunction contrib
3 ment (N = 246 with right unilateral, 79 with bitemporal, and 61 with mixed) who underwent ECT.
4 tons 2800-4000 ms postlabeling in bifrontal, bitemporal, and biparietal regions within 7 days of mTBI
5 (aphasic, perceptual-motor, frontal lobe and bitemporal categories) encompass the majority of atypica
6 the electrode placement (right unilateral or bitemporal) determines the geometric shape of the electr
7 ive complications, hippocampal remnants, and bitemporal disease do not account for all failures; extr
8 ateral parahippocampal changes may suggest a bitemporal disorder in some patients.
9 f the eligible patients, 69 were assigned to bitemporal ECT and 69 to unilateral ECT.
10 hreshold) right unilateral ECT is similar to bitemporal ECT but may have fewer cognitive side effects
11  high-dose unilateral ECT is not inferior to bitemporal ECT for depression and may be preferable beca
12 patients given bifrontal ECT and those given bitemporal ECT in the number of treatments required to r
13  High-dose unilateral ECT was noninferior to bitemporal ECT regarding the 24-item HAM-D scores after
14                                              Bitemporal ECT was associated with a lower percent recal
15 eekly moderate-dose (1.5x seizure threshold) bitemporal ECT with high-dose unilateral ECT in real-wor
16 ni-Mental State scores of the patients given bitemporal ECT worsened more after treatment than did th
17 n were treated with a course of bifrontal or bitemporal ECT.
18  bifrontal electrode placement with standard bitemporal electrode placement in the treatment of patie
19 ssant outcomes at the mid-ECT assessment and bitemporal electrode placement switch.
20 retrospective cohort study of 15 adults with bitemporal epilepsy who had a device that provides chron
21 life, and a further affected child displayed bitemporal epileptogenic discharges on EEG without overt
22 cate that patients with infantile spasms and bitemporal glucose hypometabolism on PET comprise a rela
23 d to the emergency department with new-onset bitemporal headache, dizziness, and bilateral lower extr
24 nd 18 patients with a relative homonymous or bitemporal hemianopia were tested with both conventional
25 bination with subtle facial features such as bitemporal narrowing, broad nasal tip, thin upper lip, p
26 ants were independently randomly assigned to bitemporal or high-dose unilateral ECT.
27 raction to determine whether patients fit a "bitemporal" or "prefrontal" model of sensory dysfunction
28 al electrode placement was as efficacious as bitemporal placement and resulted in less cognitive impa
29 ich the characteristic diagnostic feature is bitemporal scar-like lesions that resemble forceps marks
30        Abnormal EEG findings (showing mainly bitemporal slow activity) were recorded in 37 of 57 pati