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1 mplantation development and rarely reach the blastocyst stage.
2 an be traced back to polarity present at the blastocyst stage.
3 ccurs during a 4- to 8-h period at the early blastocyst stage.
4 ks such as H3K27me3 is achieved by the early blastocyst stage.
5 ODC did not block normal development to the blastocyst stage.
6 e embryos lacking Eomesodermin arrest at the blastocyst stage.
7 sult induces a high rate of apoptosis at the blastocyst stage.
8 nd are expressed early in development at the blastocyst stage.
9 mplantation development was normal until the blastocyst stage.
10 the ability to regenerate TE up to the early blastocyst stage.
11 sis of ZO-1alpha+ was only apparent from the blastocyst stage.
12 tage and was zonular just prior to the early blastocyst stage.
13 h isoforms to be phosphorylated at the early blastocyst stage.
14 tus could remain coupled ionically up to the blastocyst stage.
15 tivate both X chromosomes and die before the blastocyst stage.
16 on and in vitro embryonic development to the blastocyst stage.
17 elay in the DNA demethylation process at the blastocyst stage.
18 maps of mouse embryos from the zygote to the blastocyst stage.
19 tively expressed in the trophectoderm at the blastocyst stage.
20 n living mouse oocytes and embryos up to the blastocyst stage.
21 by pacing down their development around the blastocyst stage.
22 loped slower and largely failed to reach the blastocyst stage.
23 early human embryonic development beyond the blastocyst stage.
24 d the percentage of embryos that reached the blastocyst stage.
25 o development and reduces progression to the blastocyst stage.
26 but only if the host embryos are at the pre-blastocyst stage.
27 NANOG and reduced expression of CDX2 at the blastocyst stage.
28 ylase inhibitors, promote development to the blastocyst stage.
29 es to be activated from the four-cell to the blastocyst stage.
30 tic null embryos that develop until the late blastocyst stage.
31 ge, and becomes spatially restricted by late blastocyst stage.
32 ts in defects in TE specification before the blastocyst stage.
33 3.3 KD embryos and allows development to the blastocyst stage.
34 os fail to correctly specify lineages at the blastocyst stage.
35 ergenic and become hypermethylated after the blastocyst stage.
36 y passive loss that reaches a minimum at the blastocyst stage.
37 o arrest of embryonic development around the blastocyst stage.
38 e on the expression of specific miRNA at the blastocyst stage.
39 the resultant triploid cells develop to the blastocyst stage.
40 mics of all ICM cells from the early to late blastocyst stage.
41 dependent in the majority of spindles by the blastocyst stage.
42 neration of a stable outer epithelium by the blastocyst stage.
43 ivisions of all cells from the 2- to 32-cell blastocyst stage.
44 cell adhesiveness and a rounded shape at the blastocyst stage.
45 tage embryos were thawed and cultured to the blastocyst stage.
46 en it increases, becoming predominant at the blastocyst stage.
47 and severe degeneration before reaching the blastocyst stage.
48 al and functional changes from the morula to blastocyst stage.
49 avage stage, progressing into the 8-cell and blastocyst stages.
50 dramatically increased during the morula and blastocyst stages.
51 uencing on 73 embryos spanning the zygote to blastocyst stages.
52 anscripts in the human TE from early to late blastocyst stages.
53 rom 74 human embryos, spanning the morula to blastocyst stages.
54 s at the four-cell, eight-cell, 16-cell, and blastocyst stages.
55 and CHOP at the 8-cell, 16-cell, morula, and blastocyst stages.
56 yos but restored again in compact morula and blastocyst stages.
57 onic diapause) occurs when the embryo at the blastocyst stage achieves a state of suspended animation
58 rapidly elevated in embryos at the 4-cell to blastocyst stages after exposure to 10 nM calcitonin.
59 er, these uniparental embryos develop to the blastocyst stage, allowing the derivation of ES cell lin
61 f developmental pluripotency, develop to the blastocyst stage and also die after implantation, becaus
62 normal, Klf5 mutant embryos arrested at the blastocyst stage and failed to hatch due to defective TE
63 ) were compared between embryos reaching the blastocyst stage and growth-arrested embryos (degenerate
64 dormancy is active in human cells around the blastocyst stage and is reversible at both functional an
65 d that zygotic Eset expression begins at the blastocyst stage and is ubiquitous during postimplantati
68 Following IVF, embryos are biopsied at the blastocyst stage and several trophectoderm cells removed
69 expressed in pre-implantation embryos at the blastocyst stage and show that NAT1 is also expressed in
70 he oocyte, these aneuploidies persist at the blastocyst stage and the reasons for the high incidence
71 ted in the oocyte compared to 1-cell through blastocyst stages and may reflect the oocyte's response
72 ted oocytes developed in vitro to the morula/blastocyst stage, and 8% of these embryos developed to l
73 stage, reached a maximal level at the early blastocyst stage, and decreased subsequent to blastocyst
74 d not reduce developmental efficiency to the blastocyst stage, and genome integrity was maintained pr
75 mpetent to become fertilized, advance to the blastocyst stage, and give rise to live young than their
76 VIgmyc allele is unmethylated at the day-3.5 blastocyst stage, and the final, adult methylation patte
77 of monkey (Macaca fascicularis) eggs to the blastocyst stage, and their use to create a pluripotent
78 ryos at the two-cell, eight-cell, morula and blastocyst stages, and revealed four modes of translatio
79 cell lines, whereas those that have achieved blastocyst stage are a robust source of normal hES cells
80 curs first in extraembryonic lineages at the blastocyst stage around the time of implantation before
81 ence of pyruvate and develop normally to the blastocyst stage as well as produce normal viable offspr
82 er insemination decreased development to the blastocyst stage at day 7 and reduced numbers of trophec
83 nt of mouse embryos from the morula to early blastocyst stage, based on 4D confocal image volumes.
84 yos failed to proceed from the morula to the blastocyst stage because of defects in the molecular arc
85 the hypothesis that increased apoptosis at a blastocyst stage because of maternal hyperglycemia may r
89 rt normal preimplantation development to the blastocyst stage but failed to produce embryonic stem (E
90 at the morula stage; BHMT is abundant at the blastocyst stage but not other preimplantation stages, a
91 embryonic cells that becomes apparent at the blastocyst stage, but it is not known if the heterozygou
92 s embryos lacking Lias appear healthy at the blastocyst stage, but their development is retarded glob
93 , we find that success in progression to the blastocyst stage can be predicted with >93% sensitivity
95 is increased in 16-cell embryos, and by the blastocyst stage cells fail to properly adopt a TE gene
96 inactive promoters after major ZGA until the blastocyst stage, coinciding with the loss of promoter e
97 the development of eight-cell embryos to the blastocyst stage compared to controls (no added immunogl
99 ction between the trophectoderm cells of the blastocyst stage embryo and the endometrial cells of the
100 mbryonic stem cells (ESCs) isolated from the blastocyst stage embryo do not respond to TNF-alpha and
101 In conclusion, DKK1 can act on the morula-to-blastocyst stage embryo to modify subsequent trophoblast
102 ithin the inner cell mass (ICM) of the mouse blastocyst stage embryo, are segregated into adjacent ti
105 of XEN cells, which are isolated from mouse blastocyst stage embryos and represent the PrE lineage.
107 t human embryonic stem cells and cleavage to blastocyst stage embryos, also identifies the adult mese
108 of embryonic and extra-embryonic lineages in blastocyst stage embryos, the formation of the three ger
110 Embryonic stem (ES) cells are derived from blastocyst-stage embryos and are thought to be functiona
112 embryonic stem cells (ESCs) are derived from blastocyst-stage embryos but have very different biologi
113 include the establishment of XEN cells from blastocyst-stage embryos in either standard embryonic or
114 from CTBs of the first-trimester placenta or blastocyst-stage embryos in trophoblast stem cell medium
115 It is critical that epiblast cells within blastocyst-stage embryos receive the necessary regulator
116 r transfer (SCNT) method was used to produce blastocyst-stage embryos that have similar developmental
117 aneuploidy occurred in at least 80% of human blastocyst-stage embryos, with often less than 20% of ce
121 ges, prior to functional inactivation at the blastocyst stage, exclusively from X(P) in female embryo
122 d in embryos that are chronologically at the blastocyst stage following culture of 8-cell embryos in
125 one significant nuclear reprogramming by the blastocyst stage; however, problems may occur during red
126 Plakoglobin was exclusively expressed at the blastocyst stage in human and mouse embryos - further st
127 bly the second, persists intact to the early blastocyst stage in nearly two-thirds of mouse conceptus
128 isation, and pre-implantation development to blastocyst stage in response to physiological demands in
130 x, a Bcl-2-like protein, is increased at the blastocyst stage in the presence of high concentrations
131 d in decreased development of embryos to the blastocyst stage in vitro and a reduction in inner cell
132 Following IVF embryos were cultured to the blastocyst stage in vitro or transferred to diabetic and
136 nockout mice embryos are not viable past the blastocyst stage, indicating an essential role of Vav in
138 opment of zygotes of domestic species to the blastocyst stage is facilitated by culture in groups, su
139 , telomere elongation between the 8-cell and blastocyst stage is impaired, with shorter telomeres app
140 the application of aneuploidy testing at the blastocyst stage is quickly growing, and the latest repo
141 erge at the transition from the B2 to the B3 blastocyst stage, just before blastocyst expansion.
142 tion of Cdx2 develop normally until the late blastocyst stage leading to the conclusion that Cdx2 is
143 ble cleavage-stage embryos) to strategies of blastocyst-stage (n = 497) or cleavage-stage (n = 495) s
144 We show that inhibition of aPKC from the mid blastocyst stage not only prevents sorting of PrE precur
145 a single-celled fertilized egg, through the blastocyst stage of a ball of cells and the embryonic st
146 evoid of Ron (Ron-/-) are viable through the blastocyst stage of development but fail to survive past
147 immature oocyte, mature egg, and through the blastocyst stage of embryonic development, where express
148 kt are expressed from the 1-cell through the blastocyst stage of murine preimplantation embryo develo
151 ploidies may arrest between the cleavage and blastocyst stages of embryogenesis(5,6), a high number o
153 expressed in the early mouse embryo from the blastocyst stage onwards and mediate Foxh1-dependent act
156 cant and large effects on development to the blastocyst stage, pregnancy rate at day 30, calving rate
158 ch synchronized embryonic development to the blastocyst stage, preparation of the uterus for the rece
160 ertilization (IVF) favors embryo transfer at blastocyst stage, several centres worldwide employ cleav
161 and fertilized in vitro can progress to the blastocyst stage, the developmental potential of blastoc
163 become committed to the trophectoderm at the blastocyst stage through Cdx2 activity, but here we show
164 on of trophoblast development from the early blastocyst stage through the onset of implantation appea
165 were able to progress from the morula/early blastocyst stage to more advanced stages of development.
166 hole-chromosome aneuploidies detected at the blastocyst stage to obtain evidence-based conclusions on
167 ogmas: that a single trophectoderm biopsy at blastocyst stage to perform prenatal genetic testing can
168 g day 5 to 7.5 of development (the morula to blastocyst stage) to promote embryonic elongation on day
169 opment of these fertilized oocytes up to the blastocyst stage was also similar to that registered in
170 The development of individual zygotes to the blastocyst stage was optimal when they were cultured 81-
173 D) is a temporary arrest of an embryo at the blastocyst stage when it waits for the uterine receptivi
174 Src-family PTK activity continued until the blastocyst stage when strong cortical activity became ev
175 sands of maternally biased domains until the blastocyst stage, whereas maternally biased H2AK119ub1 d
176 ic lethality, null embryos survive until the blastocyst stage, which may be explained by the presence
177 l fate decision in development occurs at the blastocyst stage with establishment of the trophoblast a
178 This promotes efficient development to the blastocyst stage with no detectable effect on aneuploidy
179 a-A deficiency causes embryonic death at the blastocyst stage with pronounced cell proliferation fail