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1 49% to 0.38%, EVST from 50% to 41%, variceal bleeding from 0.15% to 0.11%, and hospitalization from 9
2 ged from 0.3% to 1.1% among groups for major bleeding, from 0.2% to 0.9% for pulmonary embolism, from
3 anged from 0.9% to 2.03% (P<0.001); of major bleeding, from 1.22% to 4.53% (P<0.001); and of mortalit
4  Comparative Audit of Lower Gastrointestinal Bleeding from 143 hospitals in the UK in 2015.
5 3%] to 1.39% [95% CI, 1.30%-1.48%) and major bleeding (from 2.50% [95% CI, 2.37%-2.63%] to 2.07% [95%
6 MINSA mortality records for gastrointestinal bleeding from 2003 to 2022.
7 try, we examined temporal trends in post-PCI bleeding from 2005 to 2009 among patients with elective
8 uded 20,127 trauma patients with significant bleeding from 274 hospitals in 40 countries.
9 yocardial infarction, revascularization, and bleeding from a 12-month landmark after stenting that ex
10 er and chills in six patients (24%) and mild bleeding from a bile duct wall during removal of an adhe
11  with untreated CCMs who had had symptomatic bleeding from a CCM lesion within the previous year were
12     There was an increased risk of recurrent bleeding from a coiled aneurysm compared with a clipped
13  describes a very rare case of recurrent UGI bleeding from a splenic artery pseudoaneurysm caused by
14 ermore, there may be net harm in the form of bleeding from aggressive antithrombotic treatment in pat
15           Nine patients had gastrointestinal bleeding from an indeterminate source and had negative r
16 ts the oldest documented first occurrence of bleeding from an iris vascular tuft.
17 ated to hypertension, thromboembolic events, bleeding from anticoagulation, or some combination of th
18               A patient's individual risk of bleeding from antithrombotic therapy should be assessed,
19             Yet outcome analyses showed that bleeding from any reviewed site was associated with redu
20                          The total amount of bleeding from chest tube were independently associated w
21 abnormal mucocutaneous, surgical, and dental bleeding from childhood, requiring >/=1 blood or platele
22 ation and quantify the risk of perioperative bleeding from chronic kidney disease.
23 sma coagulation is the favored treatment for bleeding from chronic radiation proctopathy.
24 ment is generally ineffective in controlling bleeding from chronic radiation proctopathy.
25 erts in the field on endoscopic treatment of bleeding from chronic radiation proctopathy.
26                                 Uncontrolled bleeding from coagulopathy signals imminent death in sev
27 t arteriography demonstrated active arterial bleeding from colic branches of the superior or inferior
28 rtal hypertension, porto-pulmonary shunting, bleeding from collateral bypass vessels, and eventual de
29                   In an unusual situation of bleeding from collateral circulation near the pseudocyst
30 th ACP and PI eyes with PI also causing mild bleeding from damaged iris vessels.
31 lignant, causes of SVC obstruction result in bleeding from downhill varices at such a high rate, desp
32 obstruction are the most common cause actual bleeding from downhill varices.
33 leed from esophageal or gastric varices, but bleeding from ectopic varices or portal hypertensive gas
34  with intraoperatively in all but 1 patient (bleeding from enlarged left liver lobe).
35 ing the portal venous system and controlling bleeding from esophageal and gastric varices.
36 and often results in portal hypertension and bleeding from esophageal varices.
37 y with ligation alone in patients with major bleeding from esophageal varices.
38 stroke due to inadequate anticoagulation and bleeding from excessive anticoagulation during this crit
39                       Direct effects include bleeding from garlic, ginkgo, and ginseng; cardiovascula
40                                              Bleeding from gastric varices is treated by injection wi
41 s, management of refractory gastrointestinal bleeding from gastric varix or vasculature by fine-needl
42 essions were needed to eradicate varices; no bleeding from gastroesophageal varices was observed afte
43 gradation of von Willebrand factor (vWF) and bleeding from gastrointestinal angiodysplasia at an alar
44 owing to recurrent epistaxis (nosebleeds) or bleeding from gastrointestinal telangiectases.
45 y, of lichenoid esophagitis in which massive bleeding from generalized epithelial sloughing and a lar
46 , causes less trauma and less false-positive bleeding from healthy tissues.
47 ed venous thromboembolism without increasing bleeding from injury sites.
48 lena or hematochezia and absence of upper GI bleeding) from January 1, 2000 through December 31, 2007
49 tations have a dynamic phenotypical range of bleeding from lack of platelet adhesion to severe thromb
50  intestinal symptoms, including intermittent bleeding from large intestine.
51 zation, for salvage therapy, and for diffuse bleeding from malignancy.
52                     For patients with active bleeding from malignant tumors, the panel suggested topi
53 y or variant that could increase the risk of bleeding from microelectrode mapping.
54 this condition include nosebleeds, bruising, bleeding from minor wounds, menorrhagia or postpartum bl
55 at can result in epistaxis, gastrointestinal bleeding from mucocutaneous telangiectasias, and arterio
56 ay be considered in patients with persistent bleeding from mucosal oozing or puncture wound bleeding
57 ered porous hemostat effectively stopped the bleeding from murine liver wounds, swine liver and carot
58                      In patients with active bleeding from MWTs (oozing and spurting), the panel sugg
59          There was a high incidence (75%) of bleeding from nonbronchial systemic collateral vessels a
60 e 5: Patients at high risk for ulcer-related bleeding from NSAIDs should take a PPI if they continue
61                                       Severe bleeding from other sites can occur but is rare.
62                             Gastric variceal bleeding from pancreatitis-induced splenic vein thrombos
63                       Upper gastrointestinal bleeding from peptic ulcer disease is not a new clinical
64                       Upper gastrointestinal bleeding from peptic ulcers or other nonvariceal causes
65 ND & AIMS: Liver cirrhosis is complicated by bleeding from portal hypertension but also by portal vei
66                                              Bleeding from portal hypertensive gastropathy or ectopic
67 ase warranting DES but have a higher risk of bleeding from prolonged dual antiplatelet therapy.
68 ive disorder, severe ACR, fungal sepsis, and bleeding from pseudoaneurysm, respectively, at a mean ti
69                Of the various symptoms, only bleeding from radiation-induced telangiectasias is amena
70 tegrity of inflamed blood vessels to prevent bleeding from sites of leukocyte infiltration.
71                       Clinically significant bleeding from SRMD is relatively uncommon with modern in
72    Recent observational studies suggest that bleeding from stress ulceration is extremely uncommon in
73 ncompass control of epistaxis and intestinal bleeding from telangiectases, screening for and treatmen
74 arction, urgent revascularization, and major bleeding from the 4 large-scale, placebo-controlled, ran
75 e pancreatitis and one case of postoperative bleeding from the anastomotic suture line were reported.
76 procedure in three (6%) (n = 1 each: massive bleeding from the catheter site; sepsis; and acute myoca
77     Twenty-six percent had at least 1 day of bleeding from the chest tube greater than 100 mL/kg/d.
78  B or C) presenting with UGIB, by preventing bleeding from the EVL site.
79 ducing AWP is also effective for controlling bleeding from the hepatic vein and safer than increasing
80     By increasing pneumoperitoneum pressure, bleeding from the hepatic vein cannot be controlled unde
81        High PPP is often employed to control bleeding from the hepatic vein during pure laparoscopic
82        Progression to renal carcinoma, fatal bleeding from the liver hemangiomas, and extremity angio
83 the most common complication being excessive bleeding from the palate after harvesting tissue.
84   Although sometimes successful, stimulating bleeding from the periapical area of the tooth can be ch
85 linical study was to evaluate whether evoked bleeding from the periapical tissues elicits the influx
86 wing chemomechanical debridement, intracanal bleeding from the periapical tissues was achieved, and i
87                                              Bleeding from the pterygoid venous plexus was profound w
88 omography angiography, which revealed active bleeding from the ruptured aneurysm with haematoma sprea
89  suggest that the long-term risks of further bleeding from the treated aneurysm are low with either t
90  clinically important stress-related mucosal bleeding from the upper gastrointestinal tract.
91 mulate postsurgical hemostasis did not cause bleeding from the wound, whereas soluble tPA caused prof
92 : Endoscopists should understand the risk of bleeding from therapeutic endoscopic interventions (eg,
93 s thromboembolism or death, along with major bleeding, from time of hospital discharge to 12 weeks af
94 of caplacizumab to SOC also led to increased bleeding from transient reductions in von Willebrand fac
95 -) VKDP or prothrombin significantly reduced bleeding from TT or LL.
96                      Four patients developed bleeding from ulcerations in the small or large intestin
97                 Acute upper gastrointestinal bleedings from ulcers or esophago-gastric varices are li
98 ia Type 1 (HHT1), a disease characterised by bleeding from vascular malformations.