ライフサイエンスコーパス: blood cell

1 atlases (such as conserved specification of blood cells).

2 stance and enhanced cytotoxicity towards red blood cells.

3 wn to modulate immune response in peripheral blood cells.

4 enced loss of leukocytes, platelets, and red blood cells.

5 s sphincters and causes vascular trapping of blood cells.

6 in the tails of a quantitative trait for red blood cells.

7 ential for egress of parasites from host red blood cells.

8 elial, endothelial, mesenchymal, neural, and blood cells.

9 h indicated a low binding of [(3)H]23 to red blood cells.

10 disability and high levels of infected white blood cells.

11 cur as Plasmodium parasites replicate within blood cells.

12 to the bone marrow for the production of red blood cells.

13 n of these organelles across a wide range of blood cells.

14 al protein level, white blood cells, and red blood cells.

15 THC acutely alters gene expression in 15,973 blood cells.

16 ch as glucose while blocking the much larger blood cells.

17 s and differentiate to generate all types of blood cells.

18 the haemoglobin profile of the patient's red blood cells.

19 by rounds of asexual replication within red blood cells.

20 2 protein expression in brain and peripheral blood cells(4-6).

21 ion, respectively were (median +/- IQR): Red blood cell: 6.0 +/- 0.5 (10(6)/muL) and 6.5 +/- 0.4 (10(

22 Among the new candidates identified in blood cells, 655 CpG sites, located in 99 genes, were di

23 invasion efficiency while accounting for red blood cell accessibility to parasites.

24 r release of fibrinogen correlating with red blood cell aggregation and microvascular plugging.

25 d blood cell structure, for example, and red blood cells also undergo dramatic changes in morphology

26 (2) enhanced phagocytic activity toward red blood cells (an in vitro model of hematoma clearance aft

27 blood cell differential, 2) quantitative red blood cell and hemoglobin characterization, 3) clear ide

28 flux occurs predominantly via elevating red blood cell and plasma flux in already flowing capillarie

29 lly influenced telomere length increased red blood cell and white blood cell counts, decreased mean c

30 236 genes with an altered DNA methylation in blood cells and 201 genes in diabetic islets.

31 ), a disease resulting in failure to produce blood cells and a predisposition to cancer(3,4).

32 L env sequences were recovered in antemortem blood cells and across 28 tissues (IQR: 5-9).

33 clearing hemoglobin that has leaked from red blood cells and also restricts the availability of extra

34 e of Plasmodium falciparum-infected host red blood cells and binds to specific chondroitin-4-sulfate

35 es of patient blood samples containing white blood cells and CTCs.

36 Analyses of engineered human blood cells and expression quantitative trait loci verif

37 tor preference as measured with modified red blood cells and glycan arrays.

38 3/+) mice, accompanied by an increase in red blood cells and hemoglobin and a decrease in reticulocyt

39 ciated with sequestration of parasitized red blood cells and increased gastrointestinal permeability.

40 affect global protein expression in primary blood cells and may modulate cellular pathways linked to

41 understanding cytoadherence of infected red blood cells and potentially provides a starting point fo

42 ficantly impaired in its ability to lyse red blood cells and survive in defibrinated blood.

43 ted proviral DNA from the genome of infected blood cells and tissues known to be viral reservoirs inc

44 accumulation progressively decreased in red blood cells and urine, and skin photosensitivity in CEP

45 e enables lineages to be tracked by sampling blood cells and using DNA sequencing to identify the vec

46 ida species and major blood cells (i.e., red blood cells and white blood cells) have a size distribut

47 sitemias in Plasmodium-infected cultured red blood cells, and discrimination between healthy individu

48 iglyceride level, total protein level, white blood cells, and red blood cells.

49 -MSCs reduced serum levels of anti-sheep red blood cell antibody and have limited effects on neutroph

50 ontributes to survival on retail meats where blood cells are abundant.

51 Moreover, while changes in peripheral blood cells are becoming increasingly understood, the ho

52 urthermore, tracking fluorescent-labeled red blood cells at the endocrine-exocrine interface revealed

53 ncer and gastric cancer, as well as multiple blood cell-based malignant tumors, such as lymphoma.

54 Red blood cell-based solutions, artificial hemoglobin soluti

55 verage a reduced number of circulating white blood cells, because of the Duffy-null (CC) genotype at

56 rombi and emboli contained few biconcave red blood cells but many polyhedrocytes or related forms of

57 subvert the natural short life-span of these blood cells by inducing a distinct activation of Akt, a

58 sphingolipid de novo synthesis in peripheral blood cells by measuring the incorporation of stable iso

59 (CH), a state in which somatic mutations in blood cells cause an expanded population of mutant hemat

60 a cancer derived from the myeloid lineage of blood cells, characterized by overproduction of leukemic

61 licated in antiphospholipid syndrome, or red blood cells coated with anti-(alpha)-Rh(D) antibodies th

62 d to controls after adjusting for gender and blood cell composition (p = 1.85 x 10(-5)).

63 In PHEO patients (n=10), the peripheral blood cell composition showed a myeloid bias and an incr

64 temperature >=39 degrees C, peripheral white blood cell count >=20 000/mm3, C-reactive protein >=70.0

65 ea (12 [5%] vs 17 [7%]), and decreased white blood cell count (18 [7%] vs nine [4%]).

66 skin infection (aOR 1.14), recent high white blood cell count (aOR 1.08), and genitourinary disorder

67 ody mass index (BMI; P = .003), higher white blood cell count (P = .005), and higher D-dimer levels (

68 on dysfunction, and various biochemistry and blood cell count abnormalities.

69 White blood cell count and C-reactive protein level were eleva

70 ECOG) performance status of 3 or less, white blood cell count less than 10 x 10(9) per L, and adequat

71 ng features (age >= 1 and < 10 years), white blood cell count of <50 x109/L, lack of extramedullary l

72 studies revealed leukocytosis, with a white blood cell count of 15.1 x 10(3)/uL (15.1 x 10(9)/L) (no

73 studies revealed leukocytosis, with a white blood cell count of 15.1 x 103/muL (15.1 x 109/L) (norma

74 thin normal limits, including a normal white blood cell count of 6400/muL.

75 White blood cell count was higher in the n-3 PUFA group at the

76 The median initial white blood cell count was significantly higher in patients wh

77 is easily calculated from a routine complete blood cell count with differentials.

78 Lower levels of C-reactive protein, white blood cell count, absolute neutrophil count, and procalc

79 des, waist circumference), and immune (white blood cell count, C-reactive protein) markers.

80 hematological cancer requires complete white blood cell count, followed by flow cytometry with multip

81 AKI included initial respiratory rate, white blood cell count, neutrophil/lymphocyte ratio, and lacta

82 60 years and for high vs lower initial white blood cell count, no significant differences between TDT

83 erum levels of albumin and sodium, and white blood cell count, to identify metabolites that differed

84 gene fusions, immunophenotypic groups, white blood cells count, gender or age.

85 se, we propose a unique low cost device as a blood cell counting platform.

86 s with severe COVID-19 had higher peak white blood cell counts (15.8 vs 7 x 10(3) /uL, P = .019), C-r

87 gmentation raises inflammatory-related white blood cell counts (neutrophils and monocytes), thereby i

88 from oncology to psychiatry, can lower white blood cell counts and thus access to these treatments ca

89 In this study, we investigate blood cell counts as a potential mechanism linking mLOY

90 d evidence for associations between mLOY and blood cell counts that should stimulate investigation of

91 een mLOY, detected by genotyping arrays, and blood cell counts were assessed by multivariable linear

92 ted mouse serum and significantly increasing blood cell counts, BM hematopoietic cellularity and stem

93 ls showed accelerated recovery in peripheral blood cell counts, bone marrow colony forming units, ste

94 re length increased red blood cell and white blood cell counts, decreased mean corpuscular hemoglobin

95 y disease and had significantly higher white blood cell counts, lower lymphocyte counts, and increase

96 increasing platelet counts, and lower white blood cell counts.

97 thetic paper") by the local agglutination of blood cells coupled with the capillary separation of the

98 White blood cell cystine concentration is the current gold sta

99 significant independent predictors for white blood cell cystine levels in patients of all ages with c

100 enal complications and was superior to white blood cell cystine levels in predicting the presence of

101 r therapeutic control (on the basis of white blood cell cystine levels of <2 nmol 1/2 cystine/mg prot

102 ition, transcriptomic analysis of peripheral blood cells demonstrated a marked change in transcript l

103 edrocytes or related forms of compressed red blood cells, demonstrating that these structures are a s

104 ssessment of red blood cell integrity, white blood cell differential counts, and plasma biochemical a

105 ach yields 1) a quantitative five-part white blood cell differential, 2) quantitative red blood cell

106 progenitor self-renewal and defective white blood cell differentiation.

107 imaging showed reduced (129)Xe uptake by red blood cells early in the progression of the disease, and

108 l hyperelastic constitutive model of the red blood cell (erythrocyte) membrane based on recently impr

109 ly sampled intravenous glucose test, and red blood cell fatty acid profiles were measured by gas chro

110 a species and remove most of the interfering blood cells for accurate molecular analysis, inertial so

111 receiving at least 150 mL/kg per year of red blood cells for the past 2 years at the time of enrolmen

112 s implications for understanding its role in blood cell formation and for therapeutically targeting t

113 C ratios, a decrease was observed in the red blood cell-free layer and platelet margination due to an

114 an important cyclic nucleotide exporter, red blood cells from ABCC4null/PEL-negative individuals exhi

115 similar to what has previously been shown in blood cells from CFS patients.

116 s from small molecules and surface bound red blood cells from dimethyl sulfoxide for antigen typing.

117 Cord blood cells from females of HIV-uninfected sex-discordan

118 AC-seq) to over 8,000 human immunophenotypic blood cells from fetal liver and bone marrow.

119 ized NOD/SCID mouse model engrafted with red blood cells from G6PD deficient donors.

120 ymphocyte, macrophage, karyorrhexis, and red blood cells from standard hematoxylin and eosin-stained

121 reases mobilization and trafficking of white blood cells from the bone marrow.

122 quencing analyses of matched cfDNA and white blood cells from the same patient.

123 Packed blood cell (granulocyte) TL from 621 children were compa

124 a simultaneous increase in circulating white blood cells, granulocytes, and interleukin 17A (IL-17A).

125 Telomere length (TL) in blood cells has been studied extensively as a biomarker

126 blood cells (i.e., red blood cells and white blood cells) have a size distribution of 3-5 and 6-30 mu

127 The mechanisms driving blood cell homing and their interactions with hematopoie

128 tivated macrophages that engulf healthy host blood cells (i.e., hemophagocytosis) and contribute to t

129 da-related sepsis, Candida species and major blood cells (i.e., red blood cells and white blood cells

130 cytometry to count 14 subsets of peripheral blood cells in 206 depression cases and 77 age- and sex-

131 ar lens and tubular remnant of HA containing blood cells in its lumen freely rotating in the anterior

132 theory to predict how peripheral mononuclear blood cells in mice transition from a healthy state to a

133 the asymptomatic clonal expansion of mutated blood cells in the elderly, and this phenomenon is conne

134 red the capacity of phagocytes to engulf red blood cells in the ICH brain and in primary cultures.

135 sion by increasing entrapment of sickled red blood cells in the microvasculature.

136 s molecule enhances clearance of CO from red blood cells in vitro and in vivo Herein, we tested wheth

137 SPM concentrations and reprograms peripheral blood cells, indicating a role for SPM in mediating the

138 easurements performed on intact infected red blood cells (intact infected RBC, 77.3% and 79.2%).

139 eters indicates that blood function and thus blood cell integrity were maintained throughout oxygenat

140 Assessment of red blood cell integrity, white blood cell differential coun

141 odel were: prothrombin activity, urea, white blood cell, interleukin-2 receptor, indirect bilirubin,

142 tential regulator of genes important for red blood cell invasion.

143 Plasmodium invasion of red blood cells involves malaria proteins, such as reticuloc

144 at transcript abundance in circulating white blood cells is associated with fertility in heifers.

145 Whether ZIKV infection of white blood cells is required for dissemination of the virus t

146 cers, HbF is present only in a subset of red blood cells known as F cells.

147 ing extracellular matrix components, the fly blood cells known as hemocytes can be relocated to tissu

148 g the retina act together to acidify the red blood cell leading to O(2) secretion.

149 e differentiation and maturation of multiple blood cell lineages over 4 weeks while improving CD34(+)

150 We identify associations with blood cell, lipid and inflammatory traits that are speci

151 mbination of substance P, periostin, and red blood cell lysate (representing hemosiderin).

152 he analysis of endogenous alpha-syn from red blood cells lysate of healthy controls and PD patients.

153 Thus, the ecology of red blood cells may play a key role in determining the rate

154 of polymeric cores wrapped with modified red blood cell membrane with two inserted key components: me

155 We used "red blood cell mimicry" to achieve long sensor circulation t

156 ytometry (CD45.1/45.2) demonstrated abundant blood cell mixing between parabionts after 2 wk.

157 reclinical experiments have shown that donor blood cells, modified in vitro by an alkylating agent (m

158 1), S-nitrosothiols (P = 0.03) and total red blood cell NO (P = 0.001) were collectively reduced by ~

159 put, blood vessel growth and circulating red blood cell numbers.

160 heteroplasmy and cell state in thousands of blood cells obtained from three unrelated patients who h

161 lying on distinct invasion pathways into red blood cells of different ages.

162 ria parasite replicates asexually in the red blood cells of its vertebrate host employing epigenetic

163 lecs) are expressed on the majority of white blood cells of the immune system and play critical roles

164 intracellular parasites that infect the red blood cells of their mammalian host, leading to severe o

165 n vitro reactivity against stimulatory donor blood cells on day 360, whereas reactivity against third

166 tion of an ESBL gene; (3) pyuria (>=10 white blood cells per high powered field in the urine); and (4

167 ic variants independently associated with 29 blood cell phenotypes covering a range of variation impa

168 sess the relevance of the omnigenic model to blood cell phenotypes, delineate relevant hematopoietic

169 In this study, we aimed to characterize blood-cell phenotypes and plasma biomarkers associated w

170 ecific antibody titres, plasma cytokines and blood-cell phenotyping in patients with moderate COVID-1

171 Blood cells play essential roles in human health, underp

172 throughout human history, selecting for red blood cell polymorphisms that confer innate protection a

173 Blood cell populations and ex vivo innate whole blood cy

174 Third-party packed red blood cells (pRBCs) are often used as an oxygen carrier

175 emia (AML) disrupts the generation of normal blood cells, predisposing patients to hemorrhage, anemia

176 d to individuals with normal levels of white blood cells, principally in order to minimize risk of se

177 The HEU peripheral blood cells produced less interleukin (IL)-2 (P = .004),

178 ts haematopoiesis depends on the analysis of blood cell production in unperturbed mice.

179 can lead to clonal expansion and imbalanced blood cell production.

180 FAO) is essential for the differentiation of blood cell progenitors.

181 attrition reducing proliferative reserve in blood-cell progenitors is causal has important public-he

182 counterparts, increasing polygenic drive for blood-cell proliferation traits.

183 n-coding genes expressed in peripheral white blood cells (PWBCs), and circulating micro RNAs in plasm

184 inhibitor dapagliflozin on haematocrit, red blood cell (RBC) counts and reticulocyte levels in high-

185 ese progenitors are unneeded to maintain red blood cell (RBC) counts.

186 The influence of red blood cell (RBC) deformability in whole blood on platele

187 Red blood cell (RBC) invasion by malaria merozoites involves

188 he biconcave disk shape of the mammalian red blood cell (RBC) is unique to the RBC and is vital for i

189 The mature red blood cell (RBC) lacks a nucleus and organelles characte

190 slational modifications (PTMs) of Hb and red blood cell (RBC) membrane proteins of transgenic SCD mic

191 rt of a 3-component complex that affects red blood cell (RBC) membranes.

192 re commonly used for the manipulation of red blood cell (RBC) suspensions and analyses of flow-mediat

193 t many enhancer variants associated with red blood cell (RBC) traits map to enhancers that are co-bou

194 There is no consensus on the benefit of red blood cell (RBC) transfusion after transcatheter aortic

195 Evidence regarding red blood cell (RBC) transfusion practices and their impact

196 flammatory complications associated with red blood cell (RBC) transfusions.

197 this paper, we investigate the role that red blood cell (RBC) transport plays in establishing oxygen

198 rameters such as hematocrit, hemoglobin, red blood cell (RBC), white blood cell (WBC), and platelet c

199 ed the most abundant EVs of human blood, red blood cell (RBC)- and platelet (PLT)-derived EVs and stu

200 lymerization of HbS leads to sickling of red blood cells (RBC).

201 transferrin receptor 2 [Tfr2] to control red blood cell [RBC] synthesis).

202 easurement of the Hb content in a single red blood cell, RBC, based on magnetophoretic mobility.

203 ed to the blood flow, clearing senescent red blood cells (RBCs) and recycling iron from hemoglobin.

204 Stored red blood cells (RBCs) are needed for life-saving blood tran

205 affect the shape, number, and content of red blood cells (RBCs) dramatically.

206 Malaria parasites invade healthy red blood cells (RBCs) during the blood stage of the disease

207 ), followed by the transfusion of murine red blood cells (RBCs) expressing the human KEL glycoprotein

208 ed human labor to distinguish the normal red blood cells (RBCs) from sickled cells.

209 g were tested for IgM and IgG binding to red blood cells (RBCs) from wild-type (WT), alpha1,3-galacto

210 cles (NPs) to glycophorin A receptors on red blood cells (RBCs) improved the blood half-life.

211 he phospholipids in the outer leaflet of red blood cells (RBCs) is reported.

212 nsequence of the synchronous bursting of red blood cells (RBCs) on completion of the malaria parasite

213 pable of accurately tracking the flow of red blood cells (RBCs) through a no-reaction lateral flow as

214 S polymerizes into rigid fibers, causing red blood cells (RBCs) to sickle; leading to numerous advers

215 6PD) deficiency decreases the ability of red blood cells (RBCs) to withstand oxidative stress.

216 Red blood cells (RBCs) transport oxygen to tissues and remov

217 nsient pore formation on the membrane of red blood cells (RBCs) under high mechanical tensions is of

218 so relate to inefficient gas exchange by red blood cells (RBCs), a process that is poorly characteriz

219 hensive biological investigations on the red blood cells (RBCs), advanced strategies of RBC-mediated

220 Transfusion of blood, or more commonly red blood cells (RBCs), is integral to health care systems w

221 Specifically, we discuss how red blood cells (RBCs), platelets, neutrophils, mesenchymal

222 each other within a group of individual red blood cells (RBCs), which is crucial for imaging cells i

223 nfrared (ATR-FTIR) to detect B. bovis in red blood cells (RBCs).

224 parasite Plasmodium falciparum to invade red blood cells (RBCs).

225 seases are associated with stiffening of red blood cells (RBCs; e.g., sickle cell anemia or malaria),

226 e has evolved from viewing erythrocytes (red blood cells [RBCs]) as passive carriers of oxygen to rec

227 eir ability to opsonize erythrocytes (or red blood cells, RBCs) and cause anemia.

228 ncer cells deform as they move through while blood cells remain intact, thus generating differential

229 rom the original sample while removing white blood cells, replacing the seminal plasma, and reducing

230 minally differentiated intestinal, skin, and blood cells require "professional" stem cells to produce

231 ation also led to a regulation of peripheral blood cell responses.

232 Platelets are blood cells responsible for vascular integrity preservat

233 ngle surgical unit preoperative differential blood cell results including neutrophil (N), lymphocyte

234 Flow cytometry and sorted-blood-cell RNA-seq in additional patients were used to v

235 As red blood cells rupture, the overall conductivity of the blo

236 ch as positron emission tomography and white blood cell scintigraphy have been shown to reduce the ra

237 nt bone marrow-derived neutrophils and white blood cells showed a severely impaired chemotactic respo

238 Mutant peripheral-blood cells showed decreased ubiquitylation and activate

239 Furthermore, white blood cells showed defective in vitro killing of Staphyl

240 Host genetic background affects red blood cell structure, for example, and red blood cells a

241 these DNA methylation changes happen in all blood cell subtypes or if they are cell-type specific.

242 Herein, we analyzed peripheral blood cell surface and intracellular cytokine phenotypin

243 it may also reflect the heterogeneity of red blood cell surfaces within and between hosts.

244 e protein families are expressed on infected blood cell surfaces.

245 The abundance of infected red blood cells that accumulate in the cerebral vasculature

246 Clonally expanded blood cells that contain somatic mutations (clonal haema

247 s may express proteins on the surface of red blood cells that elicit immune responses in naturally ex

248 Splenic sequestration of red blood cells, the appearance of circulating poikilocytes,

249 As representation of blood cells, the microsphere concentrations may provide

250 ter filtering alterations from matched white blood cells, the presence of ctDNA predicts recurrence w

251 ) were inoculated with P. vivax-infected red blood cells to initiate infection, and were treated with

252 thus they function as gates, allowing normal blood cells to pass by while forcing the interactions be

253 llary network and dynamically channeling red blood cells toward the initiating signal.

254 rm a network of blood vessels that regulates blood-cell traffic as well as the maintenance and functi

255 These results show the power of large-scale blood cell trait GWAS to interrogate clinically meaningf

256 ciation of genetically elevated levels of 36 blood cell traits (platelets, mature/immature red cells,

257 being applied to tackle the V2F problem for blood cell traits, illuminating how human genetic variat

258 same directions of associations with various blood cell traits, suggesting a shared mechanism linking

259 MEPE on bone density, and IQGAP2 and GMPR on blood cell traits.

260 ancestry (n = 9190) and from GWA studies of blood-cell traits, also in those of European ancestry (n

261 Further development of white blood cell transcriptional biomarkers for inflamed depre

262 l experiments showed that normal human CD34+ blood cells transduced with a lentiviral MYC vector and

263 o evidence of difference in incidence of red blood cell transfusion for a titration-dose strategy ver

264 beral (n = 492) or restrictive (n = 521) red blood cell transfusion thresholds based on infants' post

265 CD4(+) T cells in response to allogeneic red-blood-cell transfusion.

266 ns that are present at high frequency on red blood cells, transfusion incompatibility problems, due t

267 2 transmissions per million (106) packed red blood cell transfusions (95% CI, 0.29-0.65 transmissions

268 Red blood cell transfusions are commonly administered to inf

269 st transplant complications, with packed red blood cell transfusions being the most common interventi

270 KD and anemia, we evaluated incidence of red blood cell transfusions for participants randomized to r

271 renal replacement therapy, postoperative red blood cell transfusions, time to first extubation, time

272 ive exposure-while reducing the need for red blood cell transfusions-is unknown.

273 Hb toxicity resulting from physiological red blood cell turnover.

274 studied the relationship between major white blood cell types and blood pressure in the UK Biobank po

275 d gene expression and genotype data from six blood cell types from 226 to 710 individuals.

276 nal deconvolution of whole-blood miRNAs into blood cell types suggests that cell intrinsic gene expre

277 his work describes the AFM-IR spectra of red blood cells under polyvinyl alcohol hydrogels.

278 K562, JK1, and Jurkat), and 480 primary cord blood cells undergoing erythroblast differentiation.

279 t who received at least 1 uncrossmatched red blood cell unit in the trauma bay (2013-2016).

280 ser ophthalmoscope to measure changes in red blood cell velocities, vessel diameter, and flow in inte

281 of CMV and high-level EBV DNA in peripheral blood cells was associated with changes in HIV DNA molec

282 processing of a semen sample to remove white blood cells, wash away seminal plasma, and reduce sample

283 PCR rejection criteria, based on white blood cell (WBC) and platelet (PLT) counts, were develop

284 bnormal CSF diagnosis was defined as a white blood cell (WBC) count >20/uL, a CSF protein reading >50

285 tive predictive values of a normal CSF white blood cell (WBC) count for ME panel targets were 100% (1

286 duction cycles were ELN risk group and white blood cell (WBC) counts; treatment with midostaurin had

287 Pleocytosis with white blood cell (WBC) levels of >=5 cells/mm(3) and >=10 cell

288 ween periodontitis, hours of sleep and white blood cell (WBC) markers among a nationally representati

289 rit, hemoglobin, red blood cell (RBC), white blood cell (WBC), and platelet counts with an accuracy >

290 levels of SPMs-RvD3, RvD4 and PD1-and white blood cells (WBC) and platelets.

291 Proper circulation of white blood cells (WBCs) in the pulmonary vascular bed is cruc

292 ression of phosphorylated STAT3 (p-STAT3) in blood cells were evaluated and correlated with serum-der

293 Circulating red blood cells were found to retain transferrin receptor (C

294 ife cycle, Plasmodium falciparum invades red blood cells, where it catabolizes hemoglobin and sequest

295 arasites must have access to susceptible red blood cells, which they invade using pairs of parasite l

296 he synchronous release of parasites from red blood cells, which triggers 48-hour fever cycles in the

297 tion of indiscriminate host clearance of red blood cells while increasing the half-life of that respo

298 sorting for droplets containing a single red blood cell with 85% purity.

299 ogy of malaria is caused by infection of red blood cells with unicellular Plasmodium parasites.

300 nt work characterized the movement of single blood cells within the retinal vasculature (Joseph et al