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1 Va is central for amplified flux through the blood coagulation cascade.
2 re primarily restricted to components of the blood coagulation cascade.
3 tein complexes formed, and regulation of the blood coagulation cascade.
4 se inhibitors and the major inhibitor of the blood coagulation cascade.
5 th the cell membrane are critical within the blood coagulation cascade.
6 rokinase, which have opposing effects on the blood coagulation cascade.
7 d fibrin monomers in the final stages of the blood coagulation cascade.
8 al intravascular thrombus development in the blood coagulation cascade.
9 ated protein C in the down-regulation of the blood coagulation cascade.
10 VIIa complexes on cell surfaces triggers the blood coagulation cascade.
11  receptor tissue factor (TF) to initiate the blood coagulation cascade.
12 ieved to be the physiologic initiator of the blood coagulation cascade.
13 , without inhibiting serine proteases in the blood coagulation cascade.
14 R, maintain vitamin K levels and sustain the blood coagulation cascade.
15                     Fibrin, a product of the blood coagulation cascade, accompanies many type 1 immun
16              Thrombin, a key protease of the blood coagulation cascade and a potent inducer of angiog
17 ice partially restored the activation of the blood coagulation cascade and accumulation of platelets.
18 ein S, which is both an anticoagulant in the blood coagulation cascade and an activating ligand for t
19  as a key enzyme for the intervention of the blood coagulation cascade and for the development of new
20 he initiator of the extrinsic pathway of the blood coagulation cascade and normally released from dam
21 serine protease that plays a key role in the blood coagulation cascade and represents an attractive t
22 inhibition of factor Xa, a proteinase of the blood coagulation cascade, and is typically achieved wit
23 nds which are inhibitors of factor Xa in the blood coagulation cascade are described.
24 or VIIa is involved in the first step of the blood coagulation cascade, as a membrane-associated enzy
25 ntly showed that polyphosphate modulates the blood coagulation cascade at 3 steps: it triggers the co
26 by tissue factor-initiated activation of the blood coagulation cascade at the feto-maternal interface
27 r for factor VIIa (fVIIa) that initiates the blood coagulation cascade during vascular injury.
28 flammatory protease that is regulated by the blood coagulation cascade, exerts similar effects depend
29 (TF), the cellular initiator of the protease blood coagulation cascade, has been shown to be expresse
30 d serine proteases, which participate in the blood coagulation cascade, have focused attention on the
31                It utilizes components of the blood coagulation cascade in the presence of polystyrene
32                                          The blood coagulation cascade includes a step in which the s
33 elet-CTC interactions inherently rely on the blood coagulation cascade including platelet activation.
34  tissue factor gene F3, which is involved in blood coagulation cascade initiation, appears to be spec
35 rombin, and its activation at the end of the blood coagulation cascade results in the formation of th
36 y inhibitor (TFPI) inhibits proteases in the blood coagulation cascade that lead to the production of
37 tamic acid domain-containing proteins of the blood coagulation cascade, the two PRGPs are expressed i
38                                       In the blood coagulation cascade, thrombin cleaves fibrinopepti
39 al modification of several proenzymes of the blood coagulation cascade, using either collisions or in
40 hich participates in the middle stage of the blood coagulation cascade was cloned from a rat liver cD
41 cular understanding of a key reaction of the blood coagulation cascade where cofactor Va enhances act