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1 ll disease (SCD) is a common, severe genetic blood disorder.
2 is an inherited, progressively debilitating blood disorder.
3 tory of the world's most prevalent heritable blood disorder.
4 herapeutic strategy against this devastating blood disorder.
5 Myelofibrosis (MF) is a devastating blood disorder.
6 a mechanosensitive ion channel involved in a blood disorder.
7 in the diagnosis and management of this rare blood disorder.
8 s and their derivatives for the treatment of blood disorders.
9 ZOs cause somatosensory, proprioceptive, and blood disorders.
10 d clinical trials for treating SCD and other blood disorders.
11 y of these effects across different types of blood disorders.
12 ce and for therapeutic strategies in genetic blood disorders.
13 an have potential impact on the treatment of blood disorders.
14 dy of serological responses in patients with blood disorders.
15 ons, inflammation, blood loss, and inherited blood disorders.
16 ells (HSPCs) holds great potential to remedy blood disorders.
17 enue for assessing, treating, and monitoring blood disorders.
18 editing in HSCs to treat inherited monogenic blood disorders.
19 ation in cellular immunotherapy of malignant blood disorders.
20 r mutations are responsible for a variety of blood disorders.
21 at is used to treat preleukemic and leukemic blood disorders.
22 tributed between malignant and non-malignant blood disorders.
23 mpanion Article to obtain the total costs of blood disorders.
24 from individuals without evidence of clonal blood disorders.
25 ic understanding of normal hematopoiesis and blood disorders.
26 ietic grafts to treat inherited and acquired blood disorders.
27 lymphocytic leukemia (CLL) and other clonal blood disorders.
28 y atherosclerosis, but also septic emboli or blood disorders.
29 velopment and provide new insight into human blood disorders.
30 spectrin tetramer formation, and may lead to blood disorders.
31 o the pathophysiology of cardiopulmonary and blood disorders.
32 o generate transplantable cells for treating blood disorders.
33 apeutic potential of RNAi for treating these blood disorders.
34 mportant target for the treatment of various blood disorders.
35 ed levels of engraftment for gene therapy of blood disorders.
36 t of patients with nonmalignant or malignant blood disorders.
37 to treat hematologic malignancies and other blood disorders.
38 ic approach for treatment of inherited human blood disorders.
39 sed therapy for hematologic malignancies and blood disorders.
40 to age-related macular degeneration and rare blood disorders.
41 In one form of beta-thalassemia, a genetic blood disorder, a mutation in intron 2 of the beta-globi
42 d to assess the economic burden of malignant blood disorders across the 28 countries in the European
43 assess the economic burden of non-malignant blood disorders across the 28 countries of the European
46 s a promising approach to permanently remedy blood disorders, although its application in engrafting
47 globin gene transfer in treating a fulminant blood disorder and strongly support the efficacy of gene
49 cell distributions and vascular stresses in blood disorders and compare simulation results with expe
50 upregulated in a variety of human neoplastic blood disorders and constitutive upregulation of miR-125
52 udied exclusively in patients with inherited blood disorders and hepatitis C virus (HCV) infection.
60 ickle cell disease (SCD) is a common genetic blood disorder associated with acute and chronic pain, p
61 ed in hemoglobin (Hb) AS and HbAC, inherited blood disorders associated with protection against sever
64 poietic cells from unrelated donors can cure blood disorders but carries a significant risk of acute
65 s, including acute leukaemias and congenital blood disorders, but obtaining suitable numbers of cells
66 ng factors are recurrently mutated in clonal blood disorders, but the impact of dysregulated splicing
67 genital, disease-associated, or drug-induced blood disorders can cause significant morbidities and mo
71 a (beta-Thal) is a group of life-threatening blood disorders caused by either point mutations or dele
76 ickle cell disease (SCD) is a severe genetic blood disorder characterized by hemolytic anemia, episod
77 paroxysmal nocturnal hemoglobinuria, a rare blood disorder characterized by persistent complement-me
78 urpura (iTTP) is a rare and life-threatening blood disorder characterized by the formation of blood c
79 CSAs) are a heterogeneous group of inherited blood disorders characterized by pathological mitochondr
87 c cell transplant (HCT) for the treatment of blood disorders, even with optimal donor HLA matching an
88 dies of allelic variation underlying genetic blood disorders have provided important insights into hu
89 disease (SCD), the most prevalent inherited blood disorder, have a higher risk of developing certain
90 these costs we added those due to malignant blood disorders (ICD-10 C81-96 and D47) as estimated in
92 ll disease (SCD) is a highly complex genetic blood disorder in which red blood cells (RBC) exhibit he
98 the pathophysiology and treatment of several blood disorders, including the anaemia of renal disease.
100 ain impediments to effective gene therapy of blood disorders is the resistance of human hematopoietic
102 Sickle cell disease (SCD) is an inherited blood disorder marked by homozygosity of hemoglobin S, w
103 iesis, and its deregulation leads to various blood disorders, most importantly myeloproliferative neo
104 obesity, infectious disease susceptibility, blood disorders, neurosensory disorders, drug addiction
105 infection in immunocompromised patients with blood disorders or after bone marrow transplantation to
108 ights the economic burden that non-malignant blood disorders place on European health-care systems an
113 ovide a new candidate for the rare inherited blood disorder stomatocytosis with uncompensated anemia.
114 ovascular, cerebrovascular, renal, liver, or blood disorders; substance dependency; and epilepsy.
116 g of the erythrocyte mechanics in hereditary blood disorders such as spherocytosis, elliptocytosis, a
118 Sickle cell anemia (SCA) is an inherited blood disorder that causes morbidity and early mortality
119 Sickle cell disease (SCD) is a hereditary blood disorder that causes sickling of red blood cells u
121 turnal hemoglobinuria (PNH) is a rare clonal blood disorder that manifests with hemolytic anemia, bon
125 udies of various diseases, including cancer, blood disorders, thrombosis, inflammatory and autoimmune
127 utations in these genes are commonly seen in blood disorders underscores their critical roles and hig
131 ifying disease-causing variants in Mendelian blood disorders were identified and implemented as AbFab
132 t patients had primary immunodeficiencies or blood disorders, while 4 others had abnormal immune func
134 ll disease (SCD) is a debilitating monogenic blood disorder with a highly variable phenotype characte
135 ary platelet disorders (HPDs) are a group of blood disorders with variable severity and clinical impa