ライフサイエンスコーパス: blood flow

1 sions from bone marrow megakaryocytes in the blood flow.

2 nctions and thereby regulate branch-specific blood flow.

3 ical fluorescent dye used widely for imaging blood flow.

4 ression and induced HSPCs in embryos lacking blood flow.

5 on the vasculature to elicit an increase in blood flow.

6 ction due, in part, to lesser uterine artery blood flow.

7 pushed into the bleeding artery to stop the blood flow.

8 ved fetal growth and elevated uterine artery blood flow.

9 control of arterial blood pressure and brain blood flow.

10 xpansion, cortical myelination, and cerebral blood flow.

11 ormation about tumoral vessel structures and blood flow.

12 tent, inflammation, and edema), and cerebral blood flow.

13 peak, cardiac output, stroke volume and leg blood flow.

14 through intermittent occlusion of hind limb blood flow.

15 lar obstructions that impaired microvascular blood flow.

16 e pericytes and smooth muscle cells and thus blood flow.

17 tes arteries and may increase uterine artery blood flow.

18 platelet cytoskeletal changes that occur in blood flow.

19 e possibly preceded by undetected changes in blood flow.

20 ranial baroreceptors, tuned to monitor brain blood flow.

21 ole in coupling neural activity and cerebral blood flow.

22 hincters are bottlenecks for brain capillary blood flow.

23 butes to sound induced reduction of cochlear blood flow.

24 occurring, for example, during the onset of blood flow.

25 racts and a colocalized increase in cerebral blood flow.

26 pstream arterioles to rapidly increase local blood flow.

27 lls in response to cellular stress caused by blood flow.

28 ood pressure, body temperature, and cerebral blood flow.

29 induce arterial dilation and increase local blood flow(1).

30 (5 mg kg(-1) GLI superfusion) would decrease blood flow (15 um microspheres), interstitial space oxyg

31 Baseline skin blood flow (31 [17-113] vs 16 [9-32] arbitrary perfusion

32 Restoration of coronary blood flow after a heart attack can cause reperfusion in

33 gether with increased insulin-stimulated leg blood flow and a more oxidative muscle fiber type distri

34 rdiomyocytes, pericardial effusion, impaired blood flow and aberrant valvulogenesis.

35 g murine model enhanced the microcirculatory blood flow and accelerated the wound tissue regeneration

36 iovascular response that includes attenuated blood flow and an augmented pressor response.

37 s established and what we know about how the blood flow and arterial and venous pressures in giraffes

38 sticity is coupled to the reestablishment of blood flow and behavioral recovery.

39 relation was seen between pulmonary arterial blood flow and BPD outcomes.

40 tissues in vivo and permits visualization of blood flow and cellular recruitment in a system which is

41 Renal blood flow and cortical oxygen tension increased in both

42 Acoustic trauma disrupts cochlear blood flow and damages sensory hair cells.

43 xing the contractile cells to increase local blood flow and delivery of nutrients to the local cardia

44 At inclusion, skin blood flow and DeltaSBF/DeltaT were lower in patients th

45 This latching sheltered T cells from blood flow and enabled unidirectional migration to the b

46 nitrite concentrations, uterine artery (UtA) blood flow and endothelial function were assessed, and p

47 ith hypertension can exhibit impaired muscle blood flow and exaggerated increases in blood pressure d

48 nd venous anastomoses which rapidly restores blood flow and facilitates long-term survival of the gra

49 , HBOC-201 treated kidneys had similar renal blood flow and function compared with blood-treated kidn

50 s) control the immune response by regulating blood flow and immune cell recruitment in lymphoid tissu

51 SVD produces reductions in cerebral blood flow and impaired blood-brain barrier function, wh

52 ndlimb skeletal muscle) resolved a decreased blood flow and interstitial PO(2) during twitch contract

53 tors allows the quantification of myocardial blood flow and is now also suited to patients with a hig

54 lead to irreversible pathological changes in blood flow and kidney tissue, and provides retention of

55 rate were significantly greater, whereas leg blood flow and leg vascular conductance were significant

56 ffects of kidney transplantation on cerebral blood flow and magnetic resonance spectroscopic imaging

57 In this context, it is blood flow and not blood oxygen content that is the main

58 t rate with the purpose of maintaining brain blood flow and oxygen delivery.

59 of Willis are a central source of collateral blood flow and play an important role in pathologies suc

60 endothelin (ET)-1 plays a role in regulating blood flow and pressure during exercise in health, littl

61 otent vasoconstrictor, is a key modulator of blood flow and pressure during in health and has been im

62 During exercise, cardiac output, leg blood flow and radial artery and femoral venous blood ga

63 act primarily through regulating glomerular blood flow and reducing filtration pressure.

64 fluence of reproductive hormones on cerebral blood flow and sex differences in the ability of the cer

65 ion of recombinant EDN significantly reduced blood flow and subsequent gefitinib accumulation in xeno

66 V1 in arteriolar myocytes modulates regional blood flow and systemic blood pressure, and suggest that

67 very to the tumors by limiting drug-carrying blood flow and the drug concentration in tumors.

68 troke volume, blood pressure, and peripheral blood flow) and electrocardiogram findings during food c

69 ical relationship to the sleep-waking cycle, blood flow, and brain temperature in specific brain area

70 ls to generate perfusion maps in 3D of tumor blood flow, and identified repeatable quantitative featu

71 remodeling, preserved arteriovenous fistula blood flow, and prolonged primary arteriovenous fistula

72 hat comprise the vascular system; facilitate blood flow; and regulate permeability, angiogenesis, inf

73 The work unveils the structure of the liver blood flow architecture as a combination of superimposed

74 eter ratio, duct length ratio) as the entire blood flow architectures follow the principle of equipar

75 2+) channels, vasoconstriction and decreased blood flow are prevented in AKAP5 null arterial myocytes

76 alformations, but functional measurements of blood flow are scarcely used in fetal echocardiography d

77 e pancreas, not necessarily a unidirectional blood flow as in a so-called insuloacinar portal system.

78 requires maintaining normal RV intra-cavity blood flow as quantified using KE method.

79 d-brain barrier formation, and regulation of blood flow, as well as metabolic support of other brain

80 striction with pupil diameter and measure 3D blood flow at 99 volumes/second.

81 By controlling blood flow at junctions, contractile pericytes within a

82 BQ-123 increased blood flow at rest (79 +/- 87 ml/min; P = 0.03) and augm

83 features most muscle capillaries supporting blood flow at rest, and, rather than capillaries activel

84 Hb in the stereochemical model of Perutz, to blood flow autoregulation (hypoxic vasodilation governin

85 response of afferent arterioles and in renal blood flow autoregulation, which were rescued in Add3 tr

86 rrounding cells as well as the bidirectional blood flow between the endocrine and exocrine pancreas,

87 iable time-series data such as in vivo tumor blood flow (BF), blood volume (BV) and intravascular oxy

88 al arterial and venous blood samples and leg blood flow (by thermodilution) in eight patients with se

89 Alterations in fingertip skin blood flow can be evaluated using a laser Doppler therma

90 Resting coronary blood flow (CBF) (24.6 +/- 2.0 cm/s vs. 16.6 +/- 3.9 cm/

91 cting voxelwise comparisons between cerebral blood flow (CBF) and tau positron emission tomography (P

92 re cooling by 1.0 degrees C reduced cerebral blood flow (CBF) by 20-30% and cerebral oxygen delivery

93 disease, attenuates the increase in cerebral blood flow (CBF) evoked by neural activity (functional h

94 , nonresponders exhibited increased cerebral blood flow (CBF) in bilateral anterior hippocampus, whil

95 nstriction and marked reductions in cerebral blood flow (CBF) in the PFC, which were exacerbated with

96 Cerebral blood flow (CBF) reductions are an early feature of AD a

97 mean arterial blood pressure (MAP)-cerebral blood flow (CBF) relationship, with little attention giv

98 determinant of CVR, to explain the cerebral blood flow (CBF) response to a sudden change in mean art

99 ral vessels play a critical role in cerebral blood flow (CBF) restoration following ischemic stroke.

100 ivity leads to an increase in local cerebral blood flow (CBF) to allow adequate supply of oxygen and

101 robic exercise elicits increases in cerebral blood flow (CBF), as well as core body temperature; howe

102 ), white matter lesions (WMLs), and cerebral blood flow (CBF).

103 t conducts continuous monitoring of cerebral blood flow (CBF).

104 ); however, such exercise increases cerebral blood flow (CBF; +10-20%) mediated via small elevations

105 roimaging technique used to measure cerebral blood flow (CBF; perfusion) to understand brain function

106 is associated with abnormalities in cerebral blood flow, cerebral neurochemical concentrations, and w

107 of the cerebral vasculature to increase its blood flow (cerebrovascular reactivity) are relatively n

108 ophages (RPMs) are constantly exposed to the blood flow, clearing senescent red blood cells (RBCs) an

109 r remodelling by improving central and local blood flow control mechanisms.

110 ting modifications of both central and local blood flow control mechanisms.

111 Here, we reveal a mechanism for cerebral blood flow control, a precapillary sphincter at the tran

112 of such regulation on vascular function and blood flow control.

113 was to determine whether alteration in tumor blood flow could augment drug delivery and improve antit

114 immune cell profiling was performed by whole blood flow cytometry: CD4(+) T cells, Th2 cells (CD4(+)

115 used a forward signal model to simulate OCT blood flow data for training of a neural network (NN).

116 m partially abolished the observed wild-type blood flow decrease but not the salsa increase.

117 Blood flow did not change in the contralateral ear.

118 No cerebral blood flow differences between groups were found in any

119 = 8; Protocol 2, n = 8), we measured forearm blood flow (Doppler ultrasound) and calculated changes i

120 fferent cardiac cell types and is subject to blood flow-driven forces.

121 coupled with volumetric measures of cerebral blood flow (duplex ultrasound) to quantify resting cereb

122 We evaluated whether changes in skin blood flow during circulatory shock were related to surv

123 m could be a therapeutic approach to improve blood flow during exercise in hypertension.

124 l sympatholysis) is critical for maintaining blood flow during exercise-mediated sympathoexcitation.

125 Finally, we show that changes in blood flow during hypoxia or hyperoxia could be explaine

126 ple IHRs, and a limited increase in cerebral blood flow during SE with a high degree of moment-to-mom

127 ion in terms of effects on regional cerebral blood flow during two hours post-dosing.

128 blood pressure is consistent with choroidal blood flow dysregulation in patients with CSCR and indic

129 vascular system distributes exercise-induced blood flow elevations among and within those vascular be

130 found that Abeta attenuates the increases in blood flow evoked by neural activation through a reducti

131 Endothelial function was assessed by forearm blood flow (FBF) response to acetylcholine, and nitric o

132 pHLIP ICG is non-toxic, marks blood flow for hours after injection, and effectively de

133 Baseline tumor blood flow from DSC MRI was lower in patients who had po

134 Conclusion Low tumor blood flow from dynamic susceptibility contrast MRI was

135 voxel-based pathophysiology (VBP) studies of blood flow, glucose metabolism, regional homogeneity, an

136 Particularly, islet blood flow has been consistently illustrated as one-way

137 Phosphorescence lifetime and blood flow imaging were performed in both eyes to measur

138 n of apoE4, but not apoE3, reduced arteriole blood flow, impaired spatial learning, and increased anx

139 readily used for quantification of abnormal blood flow in adult hearts, however, existing in utero a

140 e IOP, has shown to also improve the retinal blood flow in animals.

141 arteriole and first order capillary, linking blood flow in capillaries to the arteriolar inflow.

142 ulation, we recorded concurrent BP and renal blood flow in conscious rats, comparing animals with ren

143 r the investigation of causality of abnormal blood flow in CVD.

144 ting a potentially systemic role for altered blood flow in driving pathologies, including endothelial

145 conveyed by CE-induced increases in cerebral blood flow in frontal brain regions and changes in white

146 lectro-metabolic microvascular regulation of blood flow in heart.

147 e signalling mechanisms that regulate muscle blood flow in humans.

148 phil aggregation, and restored microvascular blood flow in lung arterioles of SCD mice in vivo and SC

149 nly lowers IOP, but may also enhance retinal blood flow in POAG patients with a normal IOP.

150 ving fetal growth and raising uterine artery blood flow in pregnancy, which may be important in pregn

151 vivo imaging to track vascular structure and blood flow in residual peri-infarct cortex after ischemi

152 to evaluate longitudinal changes in retinal blood flow in response to flicker stimulation and system

153 Longitudinal assessments of retinal blood flow in response to flicker stimulation and system

154 nction (functional connectivity and cerebral blood flow in resting state).

155 therapeutics shown to improve microvascular blood flow in sickle transgenic mice undergoing I/R, and

156 ages to provide some local information about blood flow in the ascending aorta, based on maximum valu

157 s growth rate, vascularization, and regional blood flow in the FME and LOX tumor xenografts.

158 Local control of blood flow in the heart is important yet poorly understo

159 ctifier K(+) (Kir2.1) channels in regulating blood flow in the heart.

160 ensional visualization and quantification of blood flow in the human fetal heart and major vessels.

161 ed drug penetration into tumors and restored blood flow in tumor-associated vasculature.

162 The retinal blood flow in vascular area surrounding the optic nerve

163 2 and blood pressure External carotid artery blood flow increased by ~43% during both exercise and pa

164 lement markedly promotes vascular volume and blood flow, increased proliferation of PCs and ECs, and

165 gly the same sound protocol induced cochlear blood flow increases in salsa mice.

166 pression of neural activity-induced cerebral blood flow increases that precedes tau pathology and cog

167 provoked ~16% increases in vertebral artery blood flow, independent of changes in end-tidal PCO2 and

168 ias and variation of US-derived quantitative blood flow indicated its potential to become a clinical

169 or that transduces signals from collagen and blood flow-induced shear force to activate G protein 13

170 Blood flow is a fundamental determinant of tissue CO(2)/

171 ous studies, however, have shown that sickle blood flow is affected even at high oxygen tensions, sug

172 Our results suggest that sickle blood flow is altered systemically, from the arterial to

173 Overall, this study highlights that sickle blood flow is altered systemically, which can drive nume

174 Shear stress on arteries produced by blood flow is important for vascular development and hom

175 Skin blood flow is rapidly altered during circulatory shock a

176 ately matched increases in regional cerebral blood flow) is preserved during both exercise and temper

177 e, compared to a 13% decrease in parenchymal blood flow, itself a leading candidate biomarker of earl

178 flow derived tricuspid stroke volume and RV blood flow KE E/A ratio.

179 Additionally, the right ventricular blood flow KEi(EDV) E/A ratio demonstrated the strongest

180 nal flow (4D flow) derived right ventricular blood flow kinetic energy assessment could circumvent th

181 lar tone dysfunction impair microcirculatory blood flow, leading to organ injury and, potentially, li

182 m Early CT Score >= 6, CTP:regional cerebral blood flow (<30%) < 70ml with mismatch ratio >= 1.2 and

183 on-based U-Net in extracting both the inner (blood flow) lumen and the wall structure of the aortic a

184 e able to obtain distinct edges of tumor and blood flow mapping of the tumor microvascular with impro

185 loyable, and label-free quantitative retinal blood flow mapping technique.

186 Resting and adenosine stress myocardial blood flow (MBF) and myocardial flow reserve (MFR) were

187 prognostic significance of stress myocardial blood flow (MBF) and myocardial perfusion reserve (MPR,

188 Decreased microvascular blood flow (MBF) increases the likelihood of vaso-occlus

189 ement of rest and stress absolute myocardial blood flow (MBF) using a 2-injection single-scan imaging

190 Measurement of myocardial blood flow (MBF) with single photon emission computed to

191 and flow measures for mean stress myocardial blood flow (MBF; 2.25 mL/min/g +/- 0.59 vs 2.24 mL/min/g

192 % CI = 8.4-14.8; P < .0001]), and peripheral blood flow (mean increase 19.7% [95% CI = 10.8-28.6; P <

193 provides consistent and reproducible retinal blood flow measurement in adult mice.

194 is built on the basis of analyses of in vivo blood flow measurements in mice and an in situ three-dim

195 imultaneous bilateral EDL force and hindlimb blood flow measurements were made during electrical stim

196 XyCAM RI, therefore, by obtaining repeatable blood flow measurements with high temporal resolution, p

197 s, with no change in internal carotid artery blood flow Neurovascular coupling (i.e. the relationship

198 for at least 48 h which has consequences of blood flow not being restored and tissue damage being ob

199 Therapy to improve blood flow not only protects neurological function but a

200 volume (nrCBV), Gaussian-normalized relative blood flow (nrCBF), and tumor metabolic rate of oxygen (

201 PulseCam can also detect venous or partial blood flow occlusion that is difficult to identify using

202 Utilizing limb blood flow occlusion, we demonstrate that critical force

203 etal muscle are significantly altered during blood flow occlusion.

204 expected sound induced decrease in cochlear blood flow occurred in CBA/CaJ mice, but surprisingly th

205 graphy angiography, we measured the cochlear blood flow of salsa and wild-type mice in response to lo

206 the light-dark cycle, brain temperature, and blood flow on the function of the glymphatic system.

207 xia, or changes in fetal heart rate, carotid blood flow or carotid oxygen delivery.

208 numerous salivary factors that enhance host blood flow or suppress the host inflammatory response.

209 01), due in part to increased uterine artery blood flow (P < 0.0001).

210 ses in contractility (P < 0.05) and coronary blood flow (P < 0.05) were seen in vitro during variable

211 the multitude of co-existing conditions and blood flow parameters in real patient data.

212 fferential assessment of arterial and venous blood flow patterns in the retina that may facilitate re

213 and bends of arteries are exposed to complex blood flow patterns that exert low or low oscillatory sh

214 g-duration spaceflight may increase cerebral blood flow, possibly due to reduced haemoglobin concentr

215 Perfusion, a measure of microvascular blood flow, provides information on nutrient delivery.

216 Acute passive stretching (PS) effects on blood flow ( Q ), shear rate ( Y ), and vascular functio

217 levels under both conditions by pump-driven blood flow ( Q ).

218 Background Quantitative blood flow (QBF) measurements that use pulsed-wave US re

219 In this work, a robust blood flow quantification framework is presented based o

220 e effects of varying the degree of stenosis, blood flow rate, and viscosity on two diagnostic metrics

221 into the mice, which is consistent with the blood-flow rate.

222 tenance, and regulation of regional cerebral blood flow (rCBF).

223 knocked-down in vivo in ECs, reperfusion and blood flow recovery are markedly promoted.

224 ndlimb ischemia model, with accelerated limb blood flow recovery compared to controls.

225 -1alpha (HIF-CSC) significantly improved the blood flow recovery.

226 uce permanent hearing damage causes cochlear blood flow reduction, which may contribute to hearing lo

227 Sex and hormonal influences on integrative blood flow regulation have further implications during c

228 nsory epithelium involvement in the cochlear blood flow regulation pathway is not fully described.

229 Blood flow regulation requires adequate functioning of t

230 of distinct hemodynamic niches; (iii) tumor blood flow regulation via local vasomotion; (iv) the hem

231 utically targetable, must exist for cochlear blood flow regulation.

232 o insulin secretion, cardiac protection, and blood flow regulation.

233 the rate of motor unit deactivation, and (2) blood flow reperfusion (REP) would result in muscle reco

234 od with sub-capillary spatial resolution and blood flow-resolving speed is lacking.

235 re retinal oxygen contents and total retinal blood flow, respectively.

236 ermore, the finding that ET-1 constrains the blood flow response to exercise suggests that ET(A) rece

237 Differences in phase shifts between blood flow signals and respiratory and PB oscillations c

238 han en-face imaging in detecting neovascular blood flow signals under both rest (P = 0.125) and stres

239 Retinal blood flow significantly increased with flicker stimulat

240 We first validated blood flow simulations against in vitro measurements in

241 nts of flowing human blood and cell resolved blood flow simulations.

242 ry flow-mediated dilation, abnormal cerebral blood flow, skeletal myopathy, and intrinsic kidney dise

243 means and standard deviations of bone marrow blood flow, spatial SNR, and temporal SNR from the quant

244 This study used VISTA to analyze relative blood flow speeds in the microvascular changes associate

245 algorithm was applied to visualize relative blood flow speeds.

246 d to originate from areas of relatively slow blood flow speeds.

247 er mechanisms to restrict gravity effects on blood flow, suggesting that these species also experienc

248 corporeal respiratory support, including low blood flow systems providing mainly extracorporeal CO2 r

249 Calculation of tumoural blood flow (TBF) and apparent diffusion coefficient (ADC

250 SCD showed a greater progressive decrease in blood flow than did the controls, with poor recovery bet

251 h valvular dysfunction and severe retrograde blood flow that persist into adulthood.

252 TRPC3smcKO mice showed a greater increase in blood flow that was less variable and was positively cor

253 DOF measurements empirically correlate with blood flow, they remain far-removed from light scatterin

254 The increased blood flow through these peripheral AVMs explained the d

255 SCD mice and in vitro imaging of SCD patient blood flowing through a microfluidic system was performe

256 The deduced integrated blood flow throughout the entire pancreas suggests direc

257 , transient restriction and recirculation of blood flow to a limb after traumatic brain injury (TBI),

258 Blood flow to active skeletal muscle is augmented with g

259 low-flow states) that can result in reduced blood flow to bowel loops.

260 tentiates the pressor response and restricts blood flow to contracting muscles, this interaction enta

261 Both organs rely on fine tuning of local blood flow to match metabolic demand.

262 ing: neural activity rapidly increases local blood flow to meet moment-to-moment changes in regional

263 These results link disturbed blood flow to NF-kappaB-mediated inflammation, which pro

264 ehavioral recovery after stroke by restoring blood flow to peri-infarct cortex.

265 ehavioral recovery after stroke by restoring blood flow to peri-infarct regions.

266 been long known that these help to regulate blood flow to the brain.

267 e the size of its vascular foramina reflects blood flow to the brain.

268 nchoring the pregnancy and ensuring adequate blood flow to the fetus.

269 Blood flow to the inferior frontal lobe was evaluated as

270 gnant dams, which likely suppresses critical blood flow to the placenta and fetus.

271 lthy heart function, possibly due to reduced blood flow to the sweat gland resulting in a lack of tis

272 hesion to endothelial receptors, obstructing blood flow to vital organs.

273 During exercise, blood flow to working skeletal muscle increases in paral

274 was calculated from the product of cerebral blood flow (ultrasound) and the radial artery-jugular ve

275 The coefficient of variation of retinal blood flow under resting condition was analyzed every 2

276 edure with continuous monitoring of cerebral blood flow using laser Doppler, NIRS and ICP.

277 We assessed skin blood flow using skin laser Doppler on the fingertip for

278 ) receptor antagonism markedly increased leg blood flow, vascular conductance, oxygen delivery, and o

279 Cerebral blood flow velocities (transcranial Doppler) from middle

280 The mean blood flow velocity index (BFVi) in the optic disc and i

281 luorescence imaging has seen enduring use in blood flow visualization and is now finding a new range

282 usate at 37 degrees C for 75 min, mean renal blood flow was 110 ml/min/100 g and produced 85 ml of ur

283 On day 1 of the experiment, retinal blood flow was assessed every 20 s for 6 min during and

284 light (12 Hz) stimulation; on day 2, retinal blood flow was measured every minute for 20 min during a

285 ree of moment-to-moment variability in which blood flow was not correlated with neuronal activity.

286 This approach to altering tumor blood flow was termed "dynamic control." Dynamic control

287 In vivo BP and regional blood flow were assessed using Doppler flow and telemetr

288 A/AO) diameter ratio, and pulmonary arterial blood flow were determined.

289 Three models of islet blood flow were previously proposed, all based on the as

290 Baseline mean arterial pressure and renal blood flow were similar in both dietary groups.

291 Breathing, ECG and microvascular blood flow were simultaneously monitored for 30 min in 2

292 Cocaine-induced vasoconstriction reduces blood flow, which can jeopardize neuronal function and i

293 Cerebral blood flow, which was higher in patients pretransplant c

294 ty was strongly predictive of restoration of blood flow, which was in turn predictive of behavioral r

295 city that was associated with restoration of blood flow, which was in turn predictive of recovery of

296 ent to dural venous sinuses: regions of slow blood flow with fenestrations that can potentially permi

297 nd compare changes in vascular structure and blood flow with high spatiotemporal precision after phot

298 usses evolving methods to measure myocardial blood flow with positron emission tomography and single-

299 us appeared to correspond with the amount of blood flow within them.

300 ase and ageing create an O(2) delivery (i.e. blood flow x arterial [O(2) ], QO2 ) dependency that slo