ライフサイエンスコーパス: blood group

1 o receive standard-issue red cells (standard-blood group).

2 he highest rate (51%) among secretors with O blood group.

3 d, consequently, manifesting the rare In(Lu) blood group.

4 confirm SMIM1 as the gene underlying the Vel blood group.

5 tion occurred in secretors regardless of ABO blood group.

6 ule and stratified by centre and patient ABO blood group.

7 cur preferentially in individuals of certain blood groups.

8 ed ABH were expressed on erythrocytes of all blood groups.

9 pancreatic cancer include smoking and non-O blood groups.

10 Group 1: biomaterial hydrated in blood; Group 2: biomaterial hydrated in physiologic sali

11 ce treatment with MHA mixed with whole human blood; group 6 (G6) implant samples were treated in the

12 3 (7%), alpha thalassaemia 438 (28%), type O blood group 621 (40%), and G6PD deficiency 72 (9%) in 76

13 functionalized with immunoglobulin G against blood group A (anti-A IgG) by forming a self-assembled m

14 The accuracy for detecting blood group A (n=12), B (n=13), AB (n=9), O (n=14), and

15 We show that mAb F77 can also bind to blood group A and B analogs but with lower intensities.

16 rase (GTB) catalyze the final step in ABO(H) blood group A and B antigen synthesis through sugar tran

17 nal step of the biosynthesis of ABO(H) human blood group A and B antigens.

18 igands supported this preference over type 1 blood group A and B oligosaccharides.

19 cause in-hospital mortality in patients with blood group A and O who were transfused.

20 In analogy with histo-blood group A antigen, Forssman (Fs) antigen terminates

21 Acbeta3Galalpha4Galbeta4Glcbeta1Cer), i.e. a blood group A determinant on a type 4 core chain.

22 ulfated and methylated variants of the histo-blood group A epitope.

23 recombinant lectin, rCNL, agglutinates human blood group A erythrocytes and is specific for the uniqu

24 asite glycans structurally distinct from the blood group A oligosaccharides on the hemocyte surface m

25 that CvGal1 preferentially recognizes type 2 blood group A oligosaccharides.

26 Blood group A or B patients had lower antibody levels th

27 sitization, donor blood group O to recipient blood group A or B transfer, and ciclosporin treatment h

28 lood stored 7 days or less (in patients with blood group A or O: 0.92, 0.74-1.15, p=0.48; in all 0.90

29 blood product use history (in patients with blood group A or O: hazard ratio 0.94, 95% CI 0.73-1.20,

30 Blood group A subjects had higher levels of anti-pig IgM

31 up-specific analysis showed a higher risk in blood group A than in other blood groups (odds ratio, 1.

32 osylceramide) were identified as well as the blood group A type 1 hexaosylceramide.

33 B6 wild-type (WT) mice were sensitized with blood group A-human erythrocytes; others received passiv

34 d no longer than 7 days, both in patients of blood groups A and O and all patients.

35 Blood groups A, AB and B significantly reduced IE bindin

36 Blood group ABH(O) carbohydrate antigens are carried by

37 , cytomegalovirus serostatus, HLA match, and blood group ABO match) and survival after transplantatio

38 tors for cardioembolic stroke included histo-blood group ABO system transferase, coagulation factor X

39 (arylamine N-acetyltransferase) (NAT2), ABO blood group (ABO), and GRB2 associated binding protein 3

40 RBCs fixation on paper accurately detected blood groups (ABO and RhD) using ascending buffer for 10

41 ons target antibodies directed toward HLA or blood groups (ABO) to allow better allocation and posttr

42 Although blood group active O-glycans of the Lewis-type form the

43 y B-1b cell immune responses against the ABO-blood group Ags, while blocking both BCRs and TLR-MyD88

44 t 90 days, 448 patients (37.0%) in the fresh-blood group and 430 patients (35.3%) in the standard-blo

45 also saw some expected antibodies to ABO(H) blood group and alpha-Gal-type antigens, although these

46 ed summary odds ratio estimates according to blood group and by populations endemic versus nonendemic

47 Although non-O blood group and red blood cell (RBC) transfusion are kno

48 This includes the choice of ABO blood group and Rhesus D status, the need for special re

49 eported significant associations between ABO blood group and risk of cardiovascular disease.

50 ed the effects of host age, hemoglobin type, blood group and severe malaria on levels of IE adhesion

51 risks of death and relapse between the cord-blood group and the two other unrelated-donor groups app

52 are found as a defining feature of different blood group and tissue antigens, as well as the building

53 Studies over 5 decades have examined ABO blood groups and risk of pancreatic cancer in Western, A

54 s to investigate the association between ABO blood groups and the incidence of first and recurrent ve

55 e assigned to receive fresh red cells (fresh-blood group) and 1219 patients were assigned to receive

56 arison after adjustment for VWF:Ag, FVIII:C, blood group, and age.

57 en (VWF:Ag), factor VIII activity (FVIII:C), blood group, and age.

58 l trait (HbAS) sickle cell polymorphism, ABO blood group, and other hemoglobinopathies remain the few

59 haemoglobin C (HbC), alpha thalassaemia, ABO blood groups, and glucose-6-phosphate dehydrogenase (G6P

60 s, there were no changes in the viral or the blood group antibodies in these patients.

61 o P. marinus trophozoites, no binding of ABH blood group antibodies was observed.

62 nt non-donor specific HLA antibody, viral or blood group antibody rise was found.

63 onors for patients with low to moderate anti-blood group antibody significantly increases transplant

64 the first to examine the distribution of ABO blood group antibody titers in a population of pediatric

65 atible donors is allowed in cases where anti-blood group antibody titres are deemed amenable to remov

66 Techniques for quantifying blood group antibody-antigen interactions are also very

67 haran Africa due to the absence of the Duffy blood group antigen (Duffy antigen), the only known eryt

68 oV-like biological features, including histo-blood group antigen (HBGA) binding, rhesus enteric calic

69 nked immunosorbent assay (ELISA) and a histo-blood group antigen (HBGA) blocking assay using sera fro

70 s influenced by genetically determined histo-blood group antigen (HBGA) expression.

71 Histo-blood group antigen (HBGA) Lewis/secretor phenotypes pre

72 trains correlates with an individual's histo-blood group antigen (HBGA) profile, the biological basis

73 e change, Q396R, is able to modify the histo-blood group antigen (HBGA) recognition pattern.

74 a region topologically similar to the histo-blood group antigen (HBGA)-binding sites of the human no

75 d were compared with those measured in histo-blood group antigen (HBGA)-blocking assays.

76 on waveguides is applied to the detection of blood group antigen A on whole erythrocytes.

77 able to infect humans lacking the Duffy (Fy) blood group antigen because this receptor is critical fo

78 Expression of the Helicobacter pylori blood group antigen binding adhesin A (BabA) is more com

79 s in genogroups I and II interact with histo-blood group antigen carbohydrates, bovine noroviruses (g

80 Previous studies utilized SPR for blood group antigen detection, however, showed poor rege

81 an only be remedied when the identity of the blood group antigen is discovered.

82 The Vel blood group antigen is expressed on the red blood cells

83 The clinically important MAM blood group antigen is present on haematopoietic cells o

84 genes, which encode a serologically distinct blood group antigen known as Dantu.

85 is dependent on the expression of the Duffy blood group antigen on erythrocytes.

86 ysis suggested pathways related to the histo-blood group antigen production, and the regulation of io

87 inity to glycophorin A, a well-studied human blood group antigen that forms TM homodimers.

88 P. vivax binds the Duffy blood group antigen through its Duffy-binding protein 1

89 -immunoglobulin, immunoglobulin A, and histo-blood group antigen-blocking (HBGA) antibody levels amon

90 ination as panimmunoglobulin, IgA, and histo-blood group antigen-blocking antibodies against both vac

91 ty binding of Helicobacter pylori to the ABO blood group antigen-glycosylated gastric mucosa.

92 In comparison to blood group antigen-specific IA, nonantigen-specific IA

93 d cost of nonantigen-specific as compared to blood group antigen-specific IA.

94 ce that ABCC4 is the gene underlying the PEL blood group antigen.

95 -(1->3)-linked galactose units from a linear blood group antigen.

96 oth norovirus and rotavirus, involving histo-blood group antigenic associations, which may also prove

97 ibit both strains of VLPs' bindings to histo-blood group antigens (HBGA) receptors on human cells at

98 ns have shown the association of human histo-blood group antigens (HBGAs) and susceptibility to infec

99 Noroviruses are known to bind to histo-blood group antigens (HBGAs) and the specific binding pa

100 Histo-blood group antigens (HBGAs) are a source of antigenic v

101 Carbohydrate-dependent histo-blood group antigens (HBGAs) are known to be critical fo

102 taviruses are known to recognize human histo-blood group antigens (HBGAs) as a host ligand that is be

103 NoVs use histo-blood group antigens (HBGAs) as attachment factors.

104 oviruses (NoVs) are known to recognize histo-blood group antigens (HBGAs) as attachment factors.

105 demic acute gastroenteritis, recognize histo-blood group antigens (HBGAs) as host attachment factors

106 Human noroviruses (huNoVs) recognize histo-blood group antigens (HBGAs) as host susceptibility fact

107 ons of human noroviruses (huNoVs) with histo-blood group antigens (HBGAs) as receptors or attachment

108 viruses and differential expression of histo-blood group antigens (HBGAs) between populations may con

109 Histo-blood group antigens (HBGAs) expressed on enterocytes ar

110 ing of the RV surface protein (VP4) to histo-blood group antigens (HBGAs) in an RV genotype-dependent

111 Histo-blood group antigens (HBGAs) in saliva and gut recognize

112 rface carbohydrate structures known as histo-blood group antigens (HBGAs) prior to internalization, a

113 Histo-blood group antigens (HBGAs) such as fucosyltransferase

114 Noroviruses bind to gut histo-blood group antigens (HBGAs), but only 70%-80% of indivi

115 nsialylated glycoconjugates, including histo-blood group antigens (HBGAs), in the infectivity of huma

116 the genus Norovirus and recognizes the histo-blood group antigens (HBGAs), similarly to human norovir

117 had no binding affinity for synthetic histo-blood group antigens (HBGAs), suggesting that PSVs could

118 olled secretor-dependent expression of histo-blood group antigens (HBGAs), which are also critical fo

119 ytes in a genotype-specific manner via histo-blood group antigens (HBGAs), which are also detectable

120 n cores 2, 4, and 6, as well as type 1 histo-blood group antigens (HBGAs).

121 rocess have been identified, including human blood group antigens (HBGAs).

122 otype-dependent binding to nonsecretor histo-blood group antigens (HBGAs).

123 (P[4], P[6], and P[8]) recognize human histo-blood group antigens (HBGAs).

124 human RVs interacts with the secretor histo-blood group antigens (HBGAs).

125 The presence of histo-blood group antigens (secretor, ABO, and Lewis type) was

126 Histo-blood group antigens affect incidence of all-cause diarr

127 ions of genotyping for red cell and platelet blood group antigens affecting several areas of medicine

128 On the other hand, histo-blood group antigens also present in the cell surface ca

129 FUT2 participates in the production of histo-blood group antigens and has previously been implicated

130 ligosaccharides, on the basis of human histo-blood group antigens and milk oligosaccharides, against

131 e determines the secretor phenotype in which blood group antigens are found in non-blood body fluids.

132 FUT2 determines whether histo-blood group antigens are secreted at mucosal surfaces.

133 Analogous to human leukocyte antigens, blood group antigens are surface markers on the erythroc

134 y charged, but neutral glycans such as histo-blood group antigens can also function as receptors.

135 As expected, IgG and IgM antibody signals to blood group antigens correlated strongly with blood type

136 ting enzyme effects more complete removal of blood group antigens from cell surfaces, demonstrating t

137 digestive and respiratory epithelia of histo-blood group antigens involved in the attachment of patho

138 ess the BabA adhesin, which binds to ABO/Leb blood group antigens on gastric mucin and epithelial cel

139 It is not known whether the expression of blood group antigens on platelet proteins alters platele

140 pean descent affects the expression of histo-blood group antigens on the mucosal epithelia of human r

141 ress abnormally high or low levels of A or B blood group antigens on their platelet surfaces (high ex

142 in particular terminal fucosylated Lewis and blood group antigens present on N- and O-glycans.

143 These histo-blood group antigens serve as host receptor sites necess

144 cluding high mannose structures, fucosylated blood group antigens, and glycans with terminal 6-sulfat

145 CR1 contains the Knops blood group antigens, including the antithetical pairs S

146 cosylated glycans, which are primarily histo-blood group antigens, is largely unknown.

147 ty problems, due to the absence of undefined blood group antigens, may cause difficulty in finding ma

148 Both viruses recognize and bind to histo-blood group antigens, which are expressed by the fucosyl

149 ecificity of B cell tolerance to donor/graft blood group antigens.

150 ynthesis enzymes, cell-cycle regulators, and blood group antigens.

151 up IV and VI viruses can interact with histo-blood group antigens.

152 and binds glycans, including the Lewis histo-blood group antigens.

153 cid residues that are linked to diseases and blood group antigens.

154 Kidney recipients in the O blood group are at a disadvantage in kidney exchange pro

155 Blood groups are defined by membrane proteins that are e

156 mean (+/-SD) of 6.1+/-4.9 days in the fresh-blood group as compared with 22.0+/-8.4 days in the stan

157 sis, the hazard ratio for death in the fresh-blood group, as compared with the standard-blood group,

158 ations that allow accommodation of fucose in blood group B active GH110 enzymes or, in the case of Pd

159 The blood group B antigen and linear alpha-Gal epitope can b

160 yme was active on Galalpha1-3Gal but not the blood group B antigen.

161 nhibitor for the donor binding site of human blood group B galactosyltransferase (GTB).

162 e, particularly for patients with unknown or blood group B or AB types.

163 Those of blood group B or O may experience a significantly longer

164 explanation for contrasts in associations of blood groups B and AB between CagA-endemic and -nonendem

165 despite smaller body size and more uncommon blood groups (B, AB) among Indo-Asians.

166 orption and could be transplanted across the blood group barrier.

167 nstrate that the African-specific Duffy-null blood group-believed to confer resistance against Plasmo

168 pylori adhesin BabA binds mucosal ABO/Le(b) blood group (bg) carbohydrates.

169 nor substrate specificity and utilization in blood group biosynthesis, which can very likely be explo

170 ogical processes including cell adhesion and blood group biosynthesis.

171 er in the HLA-matched group than in the cord-blood group but not significantly so (hazard ratio, 1.69

172 The antigens defining the blood groups can be either the proteins themselves or th

173 We conclude that x2 fulfills blood group criteria and is synthesized by UDP-N-acetylg

174 ilized monolayer and allows for quantitative blood group detection.

175 a conformation similar to that of the Le(x) blood group determinant, bringing several sulfate groups

176 ABO(H) blood group determinants are expressed on different glyc

177 plasma-VWF, platelet-VWF does not express AB blood group determinants, although precursor H antigen e

178 also made based on subject age, gender, ABO blood group, diet, and history of vaccination.

179 cipient and donor characteristics, including blood group distribution, level of recipient's sensitiza

180 ecause they can only receive an organ from O blood group donors.

181 is healthy population of blood donors, non-O blood groups explain >30% of venous thromboembolic event

182 ion included terminal sialylation and ABO(H) blood group expression on VWF.

183 , and sensitive assay for rapid detection of blood group for point-of-care applications.

184 ological antibody-based methods to determine blood groups for matching donor to recipient.

185 was recently identified in an RNAi screen of blood group genes as a host factor for invasion, and we

186 w the bacteria discriminately bind different blood group glycans.

187 ral information of a dual specificity cis-AB blood group glycosyltransferase in complex with a synthe

188 zymological information for a representative blood group GT.

189 ostate cancers is a consequence of increased blood group H expression together with up-regulated bran

190 esults reveal F77 antigen to be expressed on blood group H on a 6-linked branch of a poly-N-acetyllac

191 oup and 430 patients (35.3%) in the standard-blood group had died (absolute risk difference, 1.7 perc

192 The whole blood group had faster time to resolution of base defici

193 ABO blood groups have been shown to be associated with incre

194 two unrelated-donor groups than in the cord-blood group (hazard ratio in the HLA-mismatched group, 3

195 in the HLA-mismatched group than in the cord-blood group (hazard ratio, 2.92; 95% confidence interval

196 iated glycan modifications, the emergence of blood group I antigens (or I-branched glycans) as key ce

197 nd cross-match tests were performed with all blood group identical kidney offers.

198 ot increase after transfusion of 2 fresh ABO blood group-identical platelet concentrates (therapeutic

199 MV, VZV and Anti-HBs IgG antibody levels and blood group IgG, IgM and IgA antibodies were quantified.

200 ificant comorbidities, including one case of blood group incompatibility.

201 Tailored desensitization in pediatric blood group incompatible kidney transplantation results

202 or 86% of the listed pairs and 52% were also blood group incompatible to their coregistered donor.

203 Blood group incompatible transplantation (ABOi) in child

204 Organ transplantation from ABO blood group-incompatible (ABOi) donors requires accurate

205 simulation modeling the effect of allocating blood group-incompatible deceased-donor kidneys to those

206 Acceptance of blood group-incompatible donors for patients with low to

207 Exclusion criteria were recipients were blood group-incompatible or crossmatch-positive or had C

208 Blood group-incompatible transplantation is one strategy

209 angeable platform capable of quantifying the blood group interactions between red blood cells (RBCs)

210 ABO blood group is associated with cardiovascular disease, w

211 The accurate and reliable typing of blood groups is essential prior to blood transfusion.

212 ctor (VWF) half-life, as modified by the ABO blood group, is a strong regulator of FVIII PK.

213 Here we show that Lewis and blood group isomers, which have identical fragmentation

214 We demonstrate that genotype at the ABO blood group locus plays a critical role in modulating th

215 ignal at locus 9q34.2 coincided with the ABO blood group locus; in this cohort, a blood-group-specifi

216 of 110 patients with low VWF compared with O blood group-matched healthy controls.

217 The O and Rh- blood groups may be associated with a slightly lower ris

218 The ABO and rhesus (Rh) blood groups may influence risk for severe acute respira

219 Although ABO blood groups may potentially be used with available pred

220 It occurs following ABO blood group mismatched solid organ and/or bone marrow tr

221 The occurrence of terminal blood group moieties on oyster dominin and on hemocyte s

222 s excellent reproducibility in that the same blood groups, namely A, AB, and O, were reported by usin

223 bited less human antibody binding than human blood group O allogeneic RBC in 22% of tested sera.

224 previously frozen plasma (four units each of blood group O and A), which can be issued immediately fo

225 P = 1.48x10(-4)) and a protective effect in blood group O as compared with other blood groups (odds

226 to the Globo H hexaglycosylceramide, i.e. a blood group O determinant on a type 4 core chain, the ge

227 abA-expressing H. pylori strains bind to the blood group O determinants on type 1 core chains, i.e. t

228 ar disease, with significantly lower risk in blood group O individuals.

229 e generated a series of HLA class I-negative blood group O induced pluripotent stem cell (iPSC) lines

230 mic equivalents [gEq]) for secretor-positive blood group O or A persons and 7.0 (approximately 2800 g

231 Twenty-one persons were infected (all blood group O or A), and 67% of those infected developed

232 In blood group O patients, levels of anti-A antibodies were

233 or B patients had lower antibody levels than blood group O patients.

234 were isolated from the small intestine of a blood group O pig and characterized by mass spectrometry

235 Blood group O piglets received kidney allografts from gr

236 Previous ABO sensitization, donor blood group O to recipient blood group A or B transfer,

237 In addition, blood group O VWF demonstrates enhanced susceptibility t

238 th VWF:RCo < 50 U/dL (< 40 for patients with blood group O) fulfilled the acquired von Willebrand syn

239 ible adult patients (aged >/= 18 years) with blood group O+, who required up to two whole blood unit

240 rmediate rate (30%) among secretors with non-blood group O, and the highest rate (51%) among secretor

241 Compared with blood group O, non-O blood groups were associated with h

242 el was associated with high SES, white race, blood group O, private insurance, and residence in regio

243 k is significantly reduced in individuals in blood group O.

244 fect in blood group O as compared with other blood groups (odds ratio, 0.65; 95% CI, 0.53 to 0.79; P

245 a higher risk in blood group A than in other blood groups (odds ratio, 1.45; 95% CI, 1.20 to 1.75; P

246 t cell wall oligosaccharides and human histo-blood group oligosaccharides H-type 2 and Lewis Y are bo

247 the simultaneous determination of ABO and Rh blood groups on the same device.

248 A rise is specific in contrast to the viral, blood group or third party antibodies post transplantati

249 cestry (OR = 1.8, 95% CI: 1.1, 3.1), and ABO blood group other than O (OR = 1.3, 95% CI: 1.0, 1.8).

250 ompared with 22.0+/-8.4 days in the standard-blood group (P<0.001).

251 , 0.63 [95% CI, 0.49-0.79; P < .001]) or the blood group (patient-level OR, 0.62 [95% CI, 0.46-0.83;

252 hes, reveal the molecular identity of a rare blood group (PEL) that was first reported almost 40 year

253 ed on cells of the rare P/P1/P(k)-negative p blood group phenotype.

254 astroenteritis associate with secretor histo-blood group phenotype.

255 erythrocytes from individuals of all common blood group phenotypes and elevated on cells of the rare

256 These studies explain the occurrence of rare blood group phenotypes with simultaneous altered express

257 his review describes the genetics of unusual blood group phenotypes, particularly those with altered

258 ill provide patients' comprehensive extended blood group profile as part of their medical record to b

259 The K blood group remains an important target in hemolytic dis

260 ed controls on panel reactive antibody, age, blood group, renal replacement time, prior kidney transp

261 Host ABO blood group, reticulocyte count, and parasitemia were no

262 the ABO blood group locus; in this cohort, a blood-group-specific analysis showed a higher risk in bl

263 rand factor antigen (VWF:Ag) levels, and ABO blood group status can influence FVIII PK, they do not a

264 age, we proposed that x2 joins P in the GLOB blood group system (ISBT 028) and is renamed PX2 (GLOB2)

265 Our data establish MAM as a new blood group system and demonstrate an interaction of EMP

266 Our results establish Augustine as a new blood group system and place SLC29A1 as a new candidate

267 PURPOSE OF REVIEW: The importance of ABO blood group system compatibility in platelet transfusion

268 Diets that are based on the ABO blood group system have been promoted over the past deca

269 opoietic tissue and the basis of a new histo-blood group system in man, FORS.

270 therapy-related drug resistance, and the new blood group system Langereis.

271 confirmed a potential involvement of the ABO blood-group system.

272 altered expression of antigens from several blood group systems and illuminate the role of KLF1 in g

273 te also has general information on the human blood group systems and the genes responsible for them.

274 her are known to affect antigens of 30 human blood group systems.

275 probability of relapse was lower in the cord-blood group than in either of the other groups.

276 The ABO histo-blood group, the critical determinant of transfusion inc

277 00, when Landsteiner first described the ABO blood groups, to the present, the methods used to charac

278 ection for O blood group versus A, AB, and B blood groups together was 0.88 (95% CI, 0.84 to 0.92; AR

279 The direction of blood group transfer, from donor to recipient, was O Rh

280 Three pairs of NGLs based on the blood group type A and B trisaccharides, with three diff

281 Attachment to carbohydrates of the histo-blood group type of several human Rotavirus strains (RVA

282 , multi-functional platform for quantitative blood group typing via SPR detection is achieved by immo

283 nd extend the set of markers for genetic Vel blood group typing.

284 an interesting alternative for quantitative blood grouping using SPR analysis.

285 The blood group variant Dantu provides 74% protection agains

286 The blood group Vel was discovered 60 years ago, but the und

287 The aRR of SARS-CoV-2 infection for O blood group versus A, AB, and B blood groups together wa

288 D-19 illness or death associated with type O blood group versus all others (aRR, 0.87 [CI, 0.78 to 0.

289 8); P = 0.03] were administered to the whole blood group versus component group.

290 ccounted for Organ Procurement Organization, blood group, wait time, DR antigens, and prior offer his

291 incidence of graft failure in the peripheral-blood group was 3% (95% CI, 1 to 5), versus 9% (95% CI,

292 l survival rate at 2 years in the peripheral-blood group was 51% (95% confidence interval [CI], 45 to

293 Donor blood group was insignificant on Cox regression for over

294 h-blood group, as compared with the standard-blood group, was 1.1 (95% CI, 0.9 to 1.2; P=0.38).

295 ' disease, non-Hodgkin lymphoma, and various blood groups were accurate, as individuals manifesting t

296 Compared with blood group O, non-O blood groups were associated with higher incidence of bo

297 the simultaneous determination of ABO and Rh blood groups, which is promising for use in developing c

298 through atherosclerosis, except for the ABO blood groups, which show that A and B are associated wit

299 , CAV1, atypical chemokine receptor 1 [duffy blood group]) whose mRNA transcript levels in plasma exo

300 re PEL-negative phenotype is one of the last blood groups with an unknown genetic basis.