ライフサイエンスコーパス: blood monocyte

1 CNS, microglia self-renew without input from blood monocytes.

2 yzed the putative functions of stabilin-1 in blood monocytes.

3 y to stimulate the release of TNF-alpha from blood monocytes.

4 abolished by deletion of IL-1beta in CCR2(+) blood monocytes.

5 situ, but require constant replenishment by blood monocytes.

6 d CD4-mediated macrophage differentiation of blood monocytes.

7 3 in cell-mediated immunity using peripheral blood monocytes.

8 intact in the absence of circulating LyC6(-) blood monocytes.

9 flammatory profile and expression of CD36 on blood monocytes.

10 of human monocytic cell lines and peripheral blood monocytes.

11 e both taken up by the same peripheral human blood monocytes.

12 through 7 days of infection in both AMs and blood monocytes.

13 K in aortic endothelial cells and peripheral blood monocytes.

14 nc transporter gene expression in peripheral blood monocytes.

15 te cytokine production from human peripheral blood monocytes.

16 lize on the cell surface of human peripheral blood monocytes.

17 in contrast to other tissue macrophages and blood monocytes.

18 nd represent a newly derived population from blood monocytes.

19 cytokine gene expression in human peripheral blood monocytes.

20 nd growth factors by normal human peripheral blood monocytes.

21 ome tissues under steady-state conditions by blood monocytes.

22 ent on human B cells, T cells and peripheral blood monocytes.

23 ion equal to pDC from blood and greater than blood monocytes.

24 subpopulation of AAMvarphis originates from blood monocytes.

25 r cells having the morphologic appearance of blood monocytes.

26 ished prenatally and can be replenished from blood monocytes.

27 ion of Tyr(402)) in primary human peripheral blood monocytes.

28 hibited during LC differentiation from human blood monocytes.

29 the functional effect of hyperlipidaemia on blood monocytes.

30 a rather than from recruitment of peripheral blood monocytes.

31 a wide range of antiviral responses in cord blood monocytes across the population.

32 myeloid cells derived from human peripheral blood monocytes activate IGF1R and directly support surv

33 significantly lower viraemia (p<0.0001) and blood monocyte-activation patterns (p=0.0233).

34 For instance, blood monocytes after stress had a transcriptional signa

35 microglia and/or macrophages, and CX3CR1 on blood monocytes) allowed us to distinguish animals with

36 tes the biology of target primary peripheral blood monocytes, allowing HCMV to use monocytes as "vehi

37 ine alveolar macrophages, but not peripheral blood monocytes, also formed METs in response to M. haem

38 ional macrophages are derived primarily from blood monocytes, although recent research has shown that

39 rom lipopolysaccharide-stimulated peripheral blood monocytes and 30% lower monocyte surface Toll-like

40 s on the adhesive interactions of peripheral blood monocytes and activated endothelial cell monolayer

41 Viral DNA isolated from blood monocytes and alveolar macrophages (but not T cell

42 We cultured cord blood monocytes and assessed IFNG and CCL5 mRNA producti

43 toring innate immune cells in BM, increasing blood monocytes and blood/liver bacteria clearance, and

44 multiple markers (CD44v6, CCR2, and CCR5 on blood monocytes and brain microglia and/or macrophages,

45 nature that is closer to nontumor "resident" blood monocytes and embryonic macrophages than TAMs, sug

46 PS-stimulated inflammatory response in human blood monocytes and explored potential mechanisms of vit

47 e control of processing and release in human blood monocytes and human monocyte-derived macrophages.

48 n-like protein 4 (Angptl4) in adherent human blood monocytes and in human lung microvascular endothel

49 DNA samples derived from peripheral blood monocytes and in vitro differentiated dendritic ce

50 re protein (HCVc) engages TLR2 on peripheral blood monocytes and induces production of multiple infla

51 e-4 trimethylation (H3K4Me3), and H3K9Me2 in blood monocytes and lymphocytes obtained from 30 DCCT co

52 for frequencies and functional parameters of blood monocytes and macrophages in duodenal mucosa.

53 Human peripheral blood monocytes and macrophages were incubated in the pr

54 iption stimulated by LPS in human peripheral blood monocytes and macrophages.

55 Neonatal cord and adult peripheral blood monocytes and MoDCs were cultured in autologous pl

56 y expressed in membranes of human peripheral blood monocytes and monocytoid cells.

57 4-stimulated normal and sepsis-adapted human blood monocytes and mouse splenocytes.

58 o-photon and epifluorescence microscopy that blood monocytes and neutrophils crawling along and adher

59 rs may alter epigenetic states of peripheral blood monocytes and resident tissue macrophages.

60 duce production of TNF from human peripheral blood monocytes and rheumatoid synovial membrane culture

61 been gained into how the properties of human blood monocytes and rodent macrophages are influenced by

62 toneally injected Abeta could be detected in blood monocytes and some peripheral tissues (liver, sple

63 els of both Gr-1(low) and Gr-1(intermediate) blood monocytes and splenic macrophages and, in a paracr

64 otein (IC(50) = 0.15 uM) in human peripheral blood monocytes and stimulates IL-10 production.

65 y dynamic interactions between intravascular blood monocytes and the endothelium.

66 ed the innate immune system; venom increased blood monocytes and the migration of macrophages to the

67 e expression of stabilin-1 was diminished on blood monocytes and tissue macrophages under proinflamma

68 ch committed marrow progenitors give rise to blood monocytes and tissue macrophages.

69 d immature DCs from healthy donor peripheral blood monocytes and treated them with plasma from uninfe

70 an cytomegalovirus (HCMV) infects peripheral blood monocytes and triggers biological changes that pro

71 we found that it alters PRMT1 expression in blood monocytes and tumor-associated macrophages in huma

72 rentiated from macaque and baboon peripheral blood monocytes and used to compare viral replication an

73 ated protein kinase phosphatase 1 (MKP-1) in blood monocytes, and hematopoietic MKP-1-deficiency in a

74 we found that FABP4 regulates SIRT3 in human blood monocytes, and inhibition of FABP4 in wound macrop

75 f transfected HEK293 cells, human peripheral blood monocytes, and mouse peritoneal exudate cells in a

76 etected in isolated brain mononuclear cells, blood monocytes, and peritoneal macrophages, suggesting

77 pression on adult monocytes compared to cord blood monocytes, and that CD300c and CD300e-mediated act

78 ated cells including bone marrow precursors, blood monocytes, and tissue macrophages.

79 pathways, and with gene expression in whole blood, monocytes, and myocardial tissue.

80 Two subsets of blood monocytes are commonly described in mice and human

81 Old reports suggest that blood monocytes are distributed into circulating and mar

82 Blood monocytes are heterogeneous effector cells of the

83 ial requirements for HIV-1 entry, peripheral blood monocytes are infrequently infected with HIV-1 in

84 associated macrophages (TAMs) recruited from blood monocytes are key in establishing an immunosuppres

85 Human peripheral blood monocytes are known to promote Ag presentation and

86 Blood monocytes are produced by bone marrow and splenic

87 Primary peripheral blood monocytes are responsible for the hematogenous dis

88 Circulating blood monocytes belong to the first line of defense agai

89 Deletion of IL-1beta in blood monocytes blunts the production of IL-22 by ILC3 a

90 on of human THP-1 macrophages and peripheral blood monocytes but does not occur during infection of m

91 d LN had similar gene expression profiles to blood monocytes but elevated transcripts of a limited nu

92 or (EGFR) on the surface of human peripheral blood monocytes but not on other blood leukocyte populat

93 alysis of fluorescence-activated cell-sorted blood monocytes (CD11b(+)/Ly6C(hi)) and brain monocytes

94 oduced are most closely similar to the major blood monocyte (CD14(+), CD16(low), CD163(+)).

95 In contrast to the peripheral blood monocytes, CD86 was still expressed by part of the

96 We show that CLA inhibits human peripheral blood monocyte cell adhesion to activated endothelial ce

97 mplete year of follow-up data for peripheral blood monocyte cell specimens were also analyzed.

98 metry and by Western blot in both peripheral blood monocyte cells and CD3+ T cells of renal transplan

99 We obtained peripheral blood monocyte cells from 29 living donor-recipient pair

100 ft and subcutaneous injections of peripheral blood monocyte cells from an antigen-positive donor.

101 ve animal after immunization with peripheral blood monocyte cells.

102 s were co-cultured with recipient peripheral blood monocyte cells.

103 After 6 weeks, we observed blood monocyte chimerism of 35.3+/-3.4%, whereas heart m

104 synovial fluid macrophages and RA peripheral blood monocytes compared with RA and normal peripheral b

105 Human blood monocytes comprise at least 3 subpopulations that

106 wild-type but not PDL-1-deficient peripheral blood monocytes containing apoptotic cell-associated OVA

107 Recruited blood monocytes contribute to the establishment, perpetu

108 conditioned media from Gd-exposed peripheral blood monocytes could induce a profibrotic phenotype in

109 mmatory response is rich in macrophages, and blood monocyte count is low in human NEC.

110 igh-density lipoprotein cholesterol suppress blood monocyte counts and atherosclerosis.

111 High blood monocyte counts may represent a previously unrecog

112 educed high-density lipoprotein levels, high blood monocyte counts, and accelerated atherosclerosis.

113 each subject were assessed using peripheral blood monocytes cultured with 10% autologous serum (n =

114 candidate adjuvants, but human neonatal cord blood monocytes demonstrate impaired T(H)1-polarizing re

115 s completely abrogated by in vivo peripheral blood monocyte depletion by gadolinium chloride (GdCl3)

116 igarette smoke on model THP-1 and peripheral blood monocyte derived macrophages, and discovered a mar

117 acellular HA was also observed in peripheral blood monocytes derived from three different hyperglycem

118 r activation by TLR agonists in inflammatory blood monocyte-derived DCs that express inducible NO syn

119 DCs) are the closest known equivalent of the blood monocyte-derived DCs that have been used for human

120 greatly downregulated in Mtb-infected human blood monocyte-derived DCs, indicating that reduction of

121 aB signaling pathway when porcine peripheral blood monocyte-derived dendritic cells (MoDCs) were trea

122 When human blood monocyte-derived dendritic cells were used, IL-38

123 apoptotic GECs to THP-1 cells or peripheral blood monocyte-derived macrophages as assayed by confoca

124 pha stabilization was detected in peripheral blood monocyte-derived macrophages at 2 to 24 h after in

125 deficiency virus type 1 (HIV-1) infection of blood monocyte-derived macrophages that expressed IFN-la

126 vitro migratory response of human peripheral blood monocyte-derived macrophages to oxLDL.

127 lammation is primarily based on the study of blood monocyte-derived macrophages, cells that have neve

128 olk sac-derived Kupffer cells and peripheral blood monocyte-derived macrophages.

129 21 was noted in postefferocytotic peripheral blood monocyte-derived macrophages.

130 pro-inflammatory cytokine profile similar to blood monocyte-derived macrophages.

131 effect of BBI on HIV infection of peripheral blood monocyte-derived macrophages.

132 d CD16/32, and become indistinguishable from blood monocyte-derived Mphis.

133 id (GC) treatment during maturation of human blood monocyte-derived or murine bone marrow-derived mac

134 ivo engulfment of EMPs or cMPs by peripheral blood monocytes-derived macrophages was associated with

135 genome in either liver tissue or peripheral blood monocytes, despite constant negative results from

136 st that NUR77, which is required for LyC6(-) blood monocyte development, is expressed but not critica

137 ndometrium, endometrial cells and peripheral blood monocytes did not generate inflammatory responses

138 In atherosclerotic arteries, blood monocytes differentiate to macrophages in the pres

139 orphyromonas gingivalis, donor-matched human blood monocytes, differentiated DCs, and macrophages wer

140 onic inflammatory systemic disorder, CD14(+) blood monocytes display reduced miR-125b expression as c

141 luated the diagnostic accuracy of peripheral blood monocyte distribution width alone and in combinati

142 CD11c, which increases on blood monocytes during hypercholesterolemia, plays an im

143 turnover rates that were similar to those of blood monocytes during steady-state homeostasis.

144 fferential contribution of Kupffer cells and blood monocytes during the development of NASH; Kupffer

145 Blood monocytes exist in two primary subpopulations, cha

146 mitted bone marrow macrophage precursors and blood monocytes express EGFP in these animals.

147 Blood monocytes expressed KUL01, class II MHC, and CSF1R

148 4 (M2), we cultured primary human peripheral blood monocytes for 6 d.

149 Compared with normal subjects, peripheral blood monocytes from acne patients expressed significant

150 n vivo, as CD138-bound APRIL is expressed on blood monocytes from active SLE patients.

151 specific to all U12-type introns detected in blood monocytes from affected individuals.

152 Lung tissues and blood monocytes from asthma patients expressed significa

153 on was significantly decreased in peripheral blood monocytes from asthma patients.

154 septic lethality was diminished; and third, blood monocytes from both septic mice and patients with

155 t stress lowers the threshold of activation; blood monocytes from CAPS patients maintain the high lev

156 monstrate that macrophages from CGD mice and blood monocytes from CGD patients display minimal recrui

157 Blood monocytes from children with systemic lupus erythe

158 uantitate in vivo macrophage tracking (i.e., blood monocytes from donor mice) labeled ex vivo with fl

159 P on DC differentiation in vitro, peripheral blood monocytes from healthy donors were cultured in the

160 Using primary human blood monocytes from healthy donors, we identified ANXA1

161 iation was investigated in vitro using human blood monocytes from healthy subjects and patients with

162 Functionally, we found that peripheral blood monocytes from healthy subjects homozygous for the

163 of cytokines, in freshly isolated peripheral blood monocytes from LGMD2B patients and in the SJL dysf

164 Circulating blood monocytes from macFoxp1tg mice have reduced expres

165 Blood monocytes from patients affected by NLRP3-mediated

166 mic, functional, and mechanistic analysis of blood monocytes from patients during sepsis and after re

167 ynovial macrophages compared with peripheral blood monocytes from RA or healthy controls.

168 Blood monocytes from sepsis patients demonstrated an upr

169 rventions can reduce the number of available blood monocytes, from which macrophages and most DCs and

170 Recent studies have shown that blood monocytes harbor human immunodeficiency virus type

171 We found blood monocytes harboring viruses with multiple phenotyp

172 Compared with primary blood monocytes, high levels of IL-32 are constitutively

173 equally low levels in Ly6C(hi) and Ly6C(lo) blood monocytes, however expression is increased over 2-

174 The number of circulating blood monocytes impacts atherosclerotic lesion size, and

175 tion of IL-1beta protein by human peripheral blood monocytes in a manner that requires neither the pr

176 Moreover, peripheral blood monocytes in humans with type 2 diabetes and poorl

177 acterise the role of regulatory receptors on blood monocytes in patients with sarcoidosis.

178 ty complex class II expression on peripheral blood monocytes in patients.

179 tered distribution of tissue macrophages and blood monocytes in the absence of Galpha(i2) but not Gal

180 eg), generated from cytokine-mobilized donor blood monocytes in vitamin D3 and IL-10, on renal allogr

181 nd growth factors by normal human peripheral blood monocytes in vitro and to examine whether conditio

182 n CD34(+) hematopoietic progenitor cells and blood monocytes in vitro.

183 icer, both in MM6 cells and in primary human blood monocytes, indicating a specific proteolytic degra

184 early phase of NASH development by promoting blood monocyte infiltration through the production of IP

185 both in the MM6 cell line and of peripheral blood monocytes, inhibit an apparently constitutive Dice

186 rdiac macrophages self-renew with negligible blood monocyte input.

187 d the metabolic stress-induced conversion of blood monocytes into a proatherogenic phenotype.

188 Mo-DCs are recruited from blood monocytes into lymph nodes by lipopolysaccharide a

189 The differentiation of human blood monocytes into macrophages is a caspase-dependent

190 g in the differentiation of pro-inflammatory blood monocytes into noninflammatory intestinal macropha

191 e requirement of NOTCH1 for the migration of blood monocytes into the liver and subsequent M1 differe

192 -induced B7-2 expression on human peripheral blood monocytes is dependent on JNK signaling, indicatin

193 How LCs arise from blood monocytes is not fully understood.

194 In conclusion, the recruitment of blood monocytes is the principle mechanism of Mphi accum

195 Normal human peripheral blood monocytes isolated by Ficoll-Hypaque gradient cent

196 Peripheral blood monocytes isolated from 10 AS patients and 10 heal

197 Blood monocytes isolated from diabetic mice showed a 55%

198 nse (within 4 h) in primary human peripheral blood monocytes isolated from healthy donors.

199 Peripheral blood monocytes isolated from individuals with polymorph

200 In peripheral blood monocytes isolated from patients with alcoholic he

201 Peripheral blood monocytes isolated from patients with bone metasta

202 nce believed to depend on the recruitment of blood monocytes, it is now clear that the accumulation o

203 hese mice had similar plasma cholesterol and blood monocyte levels but increased atherosclerosis comp

204 ata emphasise the functional consequences of blood monocyte lipid accumulation and reveal important i

205 A longitudinal analysis of blood monocyte markers revealed that as early as 28 days

206 at a pattern of surface marker expression in blood monocytes may serve as a marker for central nervou

207 the roles of Rho signaling in primary human blood monocytes migrating in chemotactic gradients and i

208 DNA-me was also profiled in blood monocytes (Monos) of the same patients obtained du

209 Blood monocytes, myeloid dendritic cells (mDC), and mono

210 LR2, TLR4, and CD86 expression on peripheral blood monocytes, natural killer (NK) cells, and NK T cel

211 major changes in macrophage polarization or blood monocyte numbers.

212 he effect of VacA on autophagy in peripheral blood monocytes obtained from subjects with different ge

213 pression in THP-1 cells and human peripheral blood monocytes occurred via the activation of PI3K, NAD

214 uced by HIV-1 peptide-loaded DC derived from blood monocytes of HIV-1-negative adults and neonates (m

215 f inflammatory gene expression in peripheral blood monocytes of MZ twin pairs (n = 10 pairs) discorda

216 tion of efferocytic function was observed in blood monocytes of obese participants.

217 s, we showed that mitophagy was inhibited in blood monocytes of patients with sepsis as compared with

218 th solid tumor xenografts but not peripheral blood monocytes or inflammatory macrophages in animal mo

219 tosis did not trigger a respiratory burst in blood monocytes or monocyte-derived macrophages (MDM), a

220 Of note, in primary human blood monocytes, p300-c-Myc binding was strongly correla

221 e differential immunomodulatory responses of blood monocytes, particularly during pregnancy, on infec

222 Peripheral blood monocyte (PBMC) expression of mRNA for TLRs 2, 3,

223 nd Mf2, phenotypically similar to peripheral blood monocytes (PBMos), were largely replaced within 3

224 sured the expression of p66Shc in peripheral blood monocytes (PBMs) and renal biopsy tissues from DN

225 lated cell death in primary human peripheral blood monocytes (PBMs).

226 We now show that an M2-like blood monocyte phenotype is significantly associated wit

227 Peripheral blood monocytes play a role in sarcoidosis inflammation.

228 Blood monocytes play an important role in both processes

229 We studied membrane TNF (mTNF) expression on blood monocytes, polarization into macrophages, miR-155

230 the infarct were again independent from the blood monocyte pool, returning to the steady-state situa

231 ugh which uric acid selectively lowers human blood monocyte production of the natural inhibitor IL-1

232 did not affect the number of bone marrow or blood monocytes, proliferation of precursors, or recruit

233 Our system of infected primary human blood monocytes provides us with an opportunity to answe

234 Blood monocytes recognize Gram-negative bacteria through

235 macrophages are continuously replenished by blood monocytes recruited to the peritoneal and pleural

236 from a resident prenatal population or from blood monocyte recruitment and that IL-4 can increase ma

237 Depleting Ly6C(hi) blood monocytes reduces DC accumulation and the skin cha

238 Here we demonstrate that human blood monocytes release processed IL-1beta after a one-t

239 TUNEL, suggesting a continuous transition of blood monocytes replacing IMs undergoing apoptosis.

240 d macrophage depletion in CD11b(DTR/+) mice, blood monocytes replenished heart macrophages.

241 These data suggest that HIV-1 circulating in blood monocytes represents diverse HIV-1 with multiple p

242 Thus the blood monocyte reservoir becomes the dominant presenting

243 of cells including progenitors, circulating blood monocytes, resident tissue macrophages, and dendri

244 6(+) monocyte subsets comprise 85 and 15% of blood monocytes, respectively, and are thought to repres

245 peritoneal macrophages and human peripheral blood monocytes, respectively, by flow cytometry.

246 le data sets for samples of human peripheral blood monocytes resulted in discovery of 3438 EERs in th

247 Here, the authors examine CD14+ blood monocyte's transcriptome and epigenome signatures

248 f 30-2000 pg/ml in both tumor and peripheral blood monocyte samples.

249 rum C-reactive protein levels and peripheral blood monocyte secretion of IL-6 and TNF-alpha.

250 Blood monocytes serve as the first line of host defense

251 thors generate transcriptome data from human blood monocytes stimulated with various immune stimuli a

252 Blood monocyte subpopulations and serum cytokines were a

253 The lineage relationship between blood monocyte subsets and the various phenotypes and fu

254 Selective labeling of blood monocyte subsets indicates that non-classical mono

255 c insights into CCR2-mediated recruitment of blood monocyte subsets into damaged tissue, the dynamics

256 airway monocytes were less inflammatory than blood monocytes, suggesting a more tolerant nature.

257 Circulating blood monocytes supply peripheral tissues with macrophag

258 ease in the expression of CD14 on peripheral blood monocytes that correlated with IL-23 expression an

259 h are believed to be derived from peripheral blood monocytes that have infiltrated the central nervou

260 Instead, it derives from blood monocytes that rapidly enter the PerC after stimul

261 y and reactivation in circulating peripheral blood monocytes that was developed in order to study the

262 n in the NF-kappaB signaling and function of blood monocytes, the exclusive source of intestinal macr

263 Blood monocytes then enter the inflamed peritoneum and d

264 Unlike human blood monocytes, these melanoma cells require no exogeno

265 ssion of CD44v6 on the 2 main populations of blood monocytes--those that express either low or high l

266 rrently based on the elevation of peripheral blood monocytes to >1 x 10(9)/L, measured for >/=3 month

267 In order for peripheral blood monocytes to facilitate viral dissemination, HCMV

268 ispensable for a potent cytokine response of blood monocytes to group B Streptococcus, although monoc

269 ctant protein-1 (MCP-1) directs migration of blood monocytes to inflamed tissues.

270 s by airway (induced sputum) macrophages and blood monocytes to markers of monocyte programming, in v

271 land and non-New Zealand honeys to stimulate blood monocytes to release tumour necrosis factor (TNF)-

272 cally involved in directing the migration of blood monocytes to sites of inflammation.

273 short-term model system in human peripheral blood monocytes to study the immunological relevance of

274 taxis is involved in enhanced trafficking of blood monocytes to the gut.

275 ation of resident microglia and infiltrating blood monocytes toward an M2 or "alternatively" activate

276 ement of CCR2 and CCR6 in the recruitment of blood monocytes toward the lung upon exposure to DEPs.

277 Here we present the CD14+ blood monocyte transcriptome and epigenome signatures as

278 ils, airway epithelial cells, and peripheral blood monocytes treated with plasma purified A1AT protei

279 Human peripheral blood monocytes treated with the TLR2 agonist Pam2CSK4 s

280 e reported previously that a high peripheral blood monocyte turnover rate was predictive for the onse

281 +)] CD163(+)), suggesting that the increased blood monocyte turnover was required to rapidly replenis

282 he expression of ARHGEF1 by human peripheral blood monocytes varies between individuals and inversely

283 pression in either THP-1 cells or peripheral blood monocytes was reduced upon expression of microRNA-

284 Fresh human blood monocytes were compared with monocyte-derived macr

285 Human THP-1 monocytes and peripheral blood monocytes were differentiated into macrophages and

286 Peripheral blood monocytes were differentiated into macrophages, an

287 Peripheral blood monocytes were isolated from asymptomatic and symp

288 Human blood monocytes were isolated from CF (F508del/F508del)

289 Blood monocytes were labeled by intravenous injection of

290 cA (WT-EcA), although nonleukemic peripheral blood monocytes were not affected.

291 Peripheral blood monocytes were treated with GM-CSF and IL-4 to yie

292 broblasts, small airway epithelial cells and blood monocytes were treated with IL-1beta or cigarette

293 lves antibody binding its cognate antigen on blood monocytes, which generates MIP-2 chemokine product

294 ellular responses in normal human peripheral blood monocytes, which may participate in the developmen

295 ELVs are taken up by peripheral blood monocytes, which then differentiate into activated

296 The depletion of blood monocytes with clodronate liposomes reduced neutro

297 rophages were derived by incubation of human blood monocytes with granulocyte-macrophage colony-stimu

298 on by natural killer cells, and expansion of blood monocytes with less proinflammatory cytokine respo

299 we demonstrate that activation of peripheral blood monocytes with P. acnes in vitro induced their dif

300 uman THP-1 monocytic cells and primary human blood monocytes with RvD2 and LPS to evaluate modulation