ライフサイエンスコーパス: blood neutrophil

1 ondensed chromatin in HL-60 granulocytes and blood neutrophils.

2 nd PU.1 proteins in the patient's peripheral blood neutrophils.

3 , and its receptor was identified on patient blood neutrophils.

4 regulation of ribosomal proteins observed in blood neutrophils.

5 ease in the production of IL-8 by peripheral blood neutrophils.

6 quantitation of images of nuclei from human blood neutrophils.

7 sorders, resulting in the lack of peripheral blood neutrophils.

8 transporters, SLC and ABC, in resting human blood neutrophils.

9 without dexamethasone to mobilize peripheral blood neutrophils.

10 n of LPS to the inhibitory effect of GC-w on blood neutrophils.

11 8), enabling them to recruit APRIL-producing blood neutrophils.

12 o be cell type-dependent and is cytosolic in blood neutrophils.

13 e by finding ehrlichia morulae in peripheral blood neutrophils.

14 rated morulae in the cytoplasm of peripheral blood neutrophils.

15 physiological state of the host by profiling blood neutrophils.

16 oup, the HR was 1.30 (95% CI, 1.06-1.61) for blood neutrophils.

17 expression of CD11b compared to splenic and blood neutrophils.

18 d eosinophils, and 2-hydroxybutyric acid for blood neutrophils.

19 atopy, lung function, blood eosinophils, and blood neutrophils.

20 ted in NP neutrophils compared to peripheral blood neutrophils.

21 are enriched in the enhancers of peripheral blood neutrophils.

22 ed pipeline to the study of human peripheral blood neutrophils.

23 from ex vivo-isolated G-CSF-mobilized human blood neutrophils.

24 -21 induced MRSA killing by human peripheral blood neutrophils.

25 treatment decreased Glut1 and PiT2 levels in blood neutrophils.

26 ced l-selectin shedding and rise in CD11b of blood neutrophils.

27 Obese patients had significantly higher blood neutrophils.

28 many eukaryotic cells, regulate polarity of blood neutrophils?

29 ular hydrogen peroxide content in peripheral blood neutrophils 5 hrs after burn.

30 In resting human and rat peripheral blood neutrophils, 5-LO is localized to the cytoplasm; i

31 erized by extremely low levels of peripheral blood neutrophils, a maturation block of bone marrow pro

32 onal data on threshold-associated peripheral blood neutrophil abundance and peanut-specific serum IgE

33 We show that fetal blood neutrophils acquire the ability to roll and adhere

34 ere significantly correlated with peripheral blood neutrophil activation patterns (r = 0.75, p = 0.00

35 receptor usage, and differential peripheral blood neutrophil activation that could contribute to HCM

36 ear 5-lipoxygenase was also evident in human blood neutrophils after adherence to a variety of surfac

37 Comparison of peripheral blood neutrophils after completion of 5 weekly treatment

38 and MEK1/2 were not activated in peripheral blood neutrophils after hemorrhage or endotoxemia.

39 nding protein were activated in lung but not blood neutrophils after hemorrhage or endotoxemia.

40 to upregulate CD18 expression on peripheral blood neutrophils also appeared in milk at this time.

41 itatively similar to UV-irradiated apoptotic blood neutrophils, although the signaling pathway for th

42 e characterized by an increase in peripheral blood neutrophils, an increase in myeloid progenitor pop

43 rtmentalize TLR4 signaling, we characterized blood neutrophil and cytokine responses, NF-kappaB1 acti

44 ents on protease secretion in isolated human blood neutrophils and BAL-derived macrophages.

45 Blood neutrophils and cultured human mast cells, monocyt

46 e, Prg4 mutant mice had increased peripheral blood neutrophils and decreased marrow B220(+) (B-lympho

47 Blood neutrophils and eosinophils were isolated on 2 sep

48 neutrophils (TANs) differ significantly from blood neutrophils and have a prolonged lifespan and immu

49 ammatory effects in severe asthma peripheral blood neutrophils and HBECs stimulated with lipopolysacc

50 ponent-specific antibody in human peripheral blood neutrophils and HL-60 cells.

51 (IL)-17 and IL-1 coexpressed gene modules in blood neutrophils and in microglia of cognitively impair

52 an autoperfused flow chamber assay of whole blood neutrophils and intravital microscopy of the infla

53 , patients with lupus have subpopulations of blood neutrophils and low-density granulocytes with simi

54 MicroRNA expression in peripheral blood neutrophils and lymphocytes and in situ hybridizat

55 ents showed prompt leukocytosis with maximum blood neutrophils and lymphocytes at 6-12 hours.

56 Analysis of blood neutrophils and monocytes and a microglial cell li

57 tively with FR-beta expression in peripheral blood neutrophils and monocytes and also in KG-1 (AML) c

58 CLECSF8 is primarily expressed by peripheral blood neutrophils and monocytes and weakly by several su

59 In septic peritonitis, blood neutrophils and monocytes are rapidly recruited in

60 in L-selectin expression on both peripheral blood neutrophils and monocytes but has no effect on lym

61 Our findings of increased blood neutrophils and monocytes support the immune hypot

62 In Study 3 peripheral blood neutrophils and monocytes were isolated by using F

63 ee mice showed reduced numbers of peripheral blood neutrophils and monocytes.

64 gene transcripts in unstimulated peripheral-blood neutrophils and mononuclear cells from patients wi

65 Only blood neutrophils and myeloid cell lines expressed this

66 iC1INH enhanced the bactericidal activity of blood neutrophils and peritoneal exudate leukocytes.

67 fluid and accelerated killing of bacteria by blood neutrophils and peritoneal macrophages.

68 arkers of active infection with higher total blood neutrophils and serum C-reactive protein, but no d

69 ding, reduced sugar intake, lowest levels of blood neutrophils and serum inflammatory markers, and el

70 In this study, we used human peripheral blood neutrophils and transfected cells expressing alpha

71 DE plus allergen increased blood neutrophils and was associated with persistent eos

72 is important for resistance of GBS to human blood, neutrophils and oxidative stress.

73 With HL-60 cells, fresh human peripheral blood neutrophils, and a cell line devoid of the fucosyl

74 and CD8 T-cell responses, CMV viral load in blood neutrophils, and allograft rejection during the fi

75 etention of Ox nucleoids is a feature of SLE blood neutrophils, and autoantibodies against Ox mtDNA a

76 due to a failure of bone marrow progenitors, blood neutrophils, and bronchoalveolar lavage cells to i

77 In this study, we examined peripheral blood neutrophil apoptosis and showed it to be accelerat

78 Alveolar and blood neutrophil apoptosis, phagocytosis, and adhesion m

79 d spleen; however, the numbers of peripheral blood neutrophils appear to be independently modulated a

80 Alveolar macrophages are primed and blood neutrophils are down-regulated for production of M

81 Consistently, serum DEL-1 and blood neutrophils are elevated in septic adult and neona

82 Healthy blood neutrophils are functionally quiescent in the bloo

83 Quantitative measurements of C5aR on blood neutrophils are highly predictive of survival or d

84 ore often nasal polyps, and higher levels of blood neutrophils as compared to patients who experience

85 or extracellular release by human peripheral blood neutrophils, as measured by a luminol-dependent ch

86 -C chemokine receptors 2 and 4 on peripheral blood neutrophils, as well as actin polymerization and m

87 rbol myristate acetate-stimulated peripheral blood neutrophils at 4 weeks and 2.6% +/- 1.0% at 14 wee

88 These glycolipids are expressed on human blood neutrophils at densities exceeding those required

89 r by pharmacologic means in human peripheral blood neutrophils attenuated phagocytosis of opsonized K

90 cytes from the thymus and spleen, peripheral blood neutrophils began to recover within 24 hours after

91 nic, and there was no difference in absolute blood neutrophils between groups.

92 erase was negative in the presence of intact blood neutrophils but became positive when neutrophils w

93 Reducing the influx of blood neutrophils by anti-Ly6G treatment increased propo

94 Blood neutrophils can be stimulated to express and rapid

95 s for the first time that drastic changes in blood neutrophils can originate from alternative reservo

96 nd restoration of innate immune functions of blood neutrophils (chemotaxis and reactive oxygen specie

97 The CIT concentration was associated with blood neutrophils (coef.: 0.02; 95% CI: 0.01, 0.03; p <

98 The aim is to review normal blood neutrophil concentrations and the clinical approac

99 This study investigated whether peripheral blood neutrophil concentrations in these mice are elevat

100 Here, we show that all blood neutrophils constitutively secrete APRIL, and infl

101 In contrast to these results, peripheral blood neutrophils contained both leukotriene A(4) hydrol

102 activated human CD4(+) or CD8(+) T cells, or blood neutrophils, could be demonstrated.

103 ), C-reactive protein level (P = .0006), and blood neutrophil count (P = .0024).

104 s in USF1 were significantly associated with blood neutrophil count (P = 2.18 x 10(-7)); rare variant

105 chest infiltrate, C-reactive protein levels, blood neutrophil count and theophylline maintenance use.

106 ion and sham groups in the mean increment in blood neutrophil count at 8 hours (6.16 x 10(9)/L and 6.

107 take was sustained longer than the period of blood neutrophil count elevation.

108 and independently associated with peripheral blood neutrophil count in contacts of patients diagnosed

109 bone marrow was enhanced, and the peripheral blood neutrophil count was also substantially elevated i

110 The blood neutrophil count was modestly elevated.

111 E-selectin, TNFalpha, and MPO and peripheral blood neutrophil count were higher in patients with RA t

112 elevated C-reactive protein level, elevated blood neutrophil count, and greater asthma symptoms.

113 ology Group performance status, haemoglobin, blood neutrophil count, and treatment.

114 ay mortality and biomarkers of inflammation (blood neutrophil count, urea, and creatinine concentrati

115 followed by theophylline maintenance use and blood neutrophil count, were most frequently associated

116 ric oxide levels (38 vs 27 ppb, P = .02) and blood neutrophil counts (5.3 vs 4.0 10(9)/L, P </= .001)

117 and whether age, FEV1 percent predicted, and blood neutrophil counts accurately predict sputum neutro

118 Blood neutrophil counts also increased during G-CSF (med

119 an interleukin-6 receptor inhibitor, reduced blood neutrophil counts and infarct size to a greater ex

120 Blood neutrophil counts and percentages, BAL eosinophil

121 ounts in alveolar air spaces, despite normal blood neutrophil counts and survival of emigrated neutro

122 Blood neutrophil counts are determined by the differenti

123 Recovery occurred even though blood neutrophil counts began to rise 48 hours after the

124 We conclude that IL-17A regulates blood neutrophil counts by inducing G-CSF production mai

125 Likewise, age, asthma duration, and blood neutrophil counts correctly predicted 64% of sputu

126 It also elevated peripheral blood neutrophil counts in mice.

127 Blood neutrophil counts in patients receiving these larg

128 relevant to maintain the normal set point of blood neutrophil counts in vivo.

129 ges, whereas age, FEV1 percent predicted, or blood neutrophil counts might predict sputum neutrophil

130 Here, we demonstrated that blood neutrophil counts post-MI were higher in male than

131 tion-induced testosterone deficiency reduced blood neutrophil counts to the level in females and incr

132 Blood neutrophil counts were elevated (4.8x), whereas ly

133 Age, FEV1 percent predicted, and blood neutrophil counts were similarly unsatisfactory fo

134 with older age, lower serum albumin, higher blood neutrophil counts, and greater prevalence of chron

135 is brief review summarizes the regulation of blood neutrophil counts, which is in part controlled by

136 cells had the greatest effects on regulating blood neutrophil counts.

137 with the clinical symptoms of arthritis and blood neutrophil counts.

138 In human peripheral blood neutrophils, cross-linking of L-selectin induced a

139 Peripheral blood neutrophils cultured with 50% RA synovial fluid we

140 Peripheral blood neutrophils derived from 24 hours post-hemorrhage,

141 and integrin expression on human peripheral blood neutrophils, despite differences in affinity for t

142 Like blood neutrophils, dHL60 cells respond to a uniform conc

143 but that before embryonic day (E) 15, fetal blood neutrophils display little ability to roll or adhe

144 lopment of MOF and the accompanying onset of blood neutrophil dysfunction.

145 Blood neutrophil elastase, histone-DNA, myeloperoxidase-

146 e marrow and highly significant increases in blood neutrophils, eosinophils, and basophils.

147 Freshly isolated human peripheral blood neutrophils exhibited relatively low plasminogen b

148 Human and murine peripheral blood neutrophils expressed HIF-2alpha, with expression

149 ignificant decrease in marrow and peripheral blood neutrophils, far greater than that seen in either

150 These observations suggest that decreased blood neutrophil FcgammaRIII expression without increase

151 ut not in other immunopathological features, blood neutrophils, FeNO, FEV(1), microbiome or DNA methy

152 Similar changes could be induced in vitro on blood neutrophils following contact with phorbol ester o

153 ted in up to approximately 80% of peripheral blood neutrophils for at least 28 to 35 weeks after tran

154 ent delays the apoptosis of human peripheral blood neutrophils for its advantage, peripheral blood gr

155 Peripheral blood neutrophils form highly decondensed chromatin stru

156 Compared with peripheral blood neutrophils from +/+ mice, db/db neutrophils demon

157 rescence in situ hybridization in post-G-CSF blood neutrophils from 4 of 6 patients but was also pres

158 PP2A activity was measured in peripheral blood neutrophils from A1AT-deficient (PiZZ) and healthy

159 yS was detected on the surface of peripheral blood neutrophils from both RA patients and healthy cont

160 expression nor glucose uptake was altered on blood neutrophils from CF patients compared with healthy

161 Blood neutrophils from CLP rats demonstrated defective p

162 Freshly isolated peripheral blood neutrophils from controls did not express surface

163 utrophils from control mice or in peripheral blood neutrophils from endotoxemic, hemorrhaged, or cont

164 Peripheral blood neutrophils from human immunodeficiency virus-nega

165 BAL and blood neutrophils from infants with RSV disease, but not

166 proteins and mRNA transcripts within BAL and blood neutrophils from infants with severe RSV bronchiol

167 Peripheral blood neutrophils from mature animals killed B. dermatit

168 erized by remarkably regular oscillations of blood neutrophils from near normal to extremely low leve

169 Peripheral blood neutrophils from normal mice strongly expressed A1

170 Peripheral blood neutrophils from patients (n = 19), before and 3 m

171 take of whole virus; 17 of 20 BAL and 8 of 9 blood neutrophils from patients expressed RSV N mRNA.

172 Peripheral blood neutrophils from patients with RA but not healthy

173 Freshly isolated peripheral blood neutrophils from patients with RA expressed mRNA,

174 In peripheral blood neutrophils from rats, 5-lipoxygenase was exclusiv

175 in bronchial epithelial cells and peripheral blood neutrophils from severe asthma patients compared w

176 (IEC-18) were carried out in the presence of blood neutrophils from sham, B, EF, or B+EF rats.

177 human bronchial epithelial cells (HBEC) and blood neutrophils from uncontrolled severe asthma (n = 2

178 production because peritoneal and peripheral blood neutrophils from uninfected animals contained IL-1

179 Peripheral blood neutrophils from untreated septic animals and sept

180 ecific response, we used isolated peripheral blood neutrophils from wild-type or CD18-null mice and s

181 The migratory accuracy of blood neutrophils from young (aged <35 yr) and old (aged

182 Peripheral blood neutrophils from young pigs expressed PR-39 and pr

183 om Chiang Mai, Thailand, on human peripheral blood neutrophil functions, including LPS-induced migrat

184 eling in healthy humans is consistent with a blood neutrophil half-life of less than 1 day.

185 il production without significantly altering blood neutrophil half-life or margination.

186 centration of A23187 for activation than did blood neutrophils (half-maximal response, 160 versus 52

187 on of NF-kappaB in LPS-stimulated peripheral blood neutrophils has been shown to be associated with m

188 Blood neutrophil homeostasis is essential for successful

189 tutively exposed on the surface of quiescent blood neutrophils in a proteolytically inactive form.

190 RSV proteins were found in BAL and blood neutrophils in infants with RSV disease but not in

191 duce OSM production (P < .001) in peripheral blood neutrophils in vitro.

192 Human peripheral blood neutrophils, in contrast to a neutrophil-like cell

193 Blood neutrophils increased from preexposure (34.4% +/-

194 the beta(1) and beta(2) integrin content on blood neutrophils increased in a nontranscriptional mann

195 ith higher BMI (P = 0.03), and the number of blood neutrophils increased less in obese than in lean p

196 In this study, peripheral blood neutrophils incubated with RSV (multiplicity of in

197 ated with reduced infiltration of peripheral blood neutrophils into infarcted tissue and with impaire

198 ecies production between oral and peripheral blood neutrophils isolated from patients with chronic pe

199 All the experiments (60+ whole blood neutrophil isolations across 36 blood donors) are

200 Whereas blood neutrophil levels and LPS-elicited tissue neutroph

201 During these events, blood neutrophils lost their chemotactic responsiveness

202 tly higher, and the percentage of peripheral blood neutrophils lower, in infected IL-3 KO mice than i

203 ence interval: 1.47-2.04) and log peripheral blood neutrophil lymphocyte ratio (odds ratio: 1.71, 95%

204 Menin loss modestly impaired blood neutrophil, lymphocyte, and platelet counts.

205 lammation (hepatic neutrophil frequency) and blood neutrophil-lymphocyte ratio on chronic plus binge

206 ung metastases, serum lactate dehydrogenase, blood neutrophil-lymphocyte ratio, therapy (monotherapy/

207 ophil activation in the liver and diminished blood neutrophil-lymphocyte ratio.

208 Analyses included peripheral blood neutrophil mass spectrometry, neutrophil functiona

209 gnificantly, delayed apoptosis of peripheral blood neutrophils (mean suppression 45.7% +/- SD 22.3).

210 blood neutrophils (PBNs) or mouse peripheral blood neutrophils (MPBNs) but markedly greater than did

211 Like mature peripheral blood neutrophils, myeloblasts expressed glucosylceramid

212 Thus, PSGL-1 is expressed on essentially all blood neutrophils, NK cells, B cells, T cells, and monoc

213 Human blood neutrophils normally express two FcgammaRs (Fcgamm

214 These data show that peripheral blood neutrophil numbers are regulated by a feedback loo

215 Although blood neutrophil numbers in inbred mouse strains and ind

216 circulating neutrophil numbers, we measured blood neutrophil numbers in p40-deficient (IL12b-/-) mic

217 rophilic inflammation, with dysregulation in blood neutrophil numbers, frequencies, and functions.

218 ated with NF-kappaB activation in peripheral blood neutrophils obtained over the pre-LPS exposure per

219 ic regression analysis showed that increased blood neutrophil (odds ratio, 10.9; P = .002) and sputum

220 , and the effect of gremubamab in peripheral blood neutrophil opsonophagocytic killing (OPK) assays a

221 nophore-stimulated (A23187; 1-2.5 muM) human blood neutrophils or differentiated HL-60 cells, vitamin

222 an or rat C5a, incubation at pH 7.4 of human blood neutrophils or rat alveolar macrophages (AMs) with

223 veal profound differences in mouse and human blood neutrophils, particularly their granule contents.

224 CD89hiCD32loCD64lo peripheral blood neutrophils (PBN) and TAN stimulated tumor cell gr

225 Peripheral blood neutrophils (PBNs) from mice with meningitis exhib

226 vities, similar to those of human peripheral blood neutrophils (PBNs) or mouse peripheral blood neutr

227 Blood neutrophils peaked at 6-12 hours, increasing from

228 Blood neutrophils perform an essential host-defense func

229 D4 in the 1 to 100 nM range stimulated whole-blood neutrophil phagocytosis of Escherichia coli.

230 Blood neutrophils (PMN) are usually unresponsive to CC c

231 Human peripheral blood neutrophils (PMN) plated onto fibrinogen and activ

232 In comparison with peripheral blood neutrophils (PMNs), the decidual neutrophils expre

233 of M. haemolytica LKT with bovine peripheral blood neutrophils (PMNs).

234 We initially constrained the ratio of the blood neutrophil pool to the marrow precursor pool (rati

235 p < .001) and were negatively correlated to blood neutrophils (r = -0.2881, p = .0384) and neutrophi

236 c oxide in exhaled air (r = 0.48, P = .004), blood neutrophils (r = 0.63, P < .001), and bronchial wa

237 nder the curve for LPS-stimulated peripheral blood neutrophils (r = 0.63, p = 0.01).

238 asal G-CSF correlated closely with increased blood neutrophils (r(s) = 0.69, p < 0.005), whereas nasa

239 In addition, the genome-wide response to blood neutrophils (rather than total WBC) was also not w

240 Blood neutrophils recruited to cystic fibrosis (CF) airw

241 ne cells, with preferential association with blood neutrophils relative to other particles.

242 We demonstrate that CF blood neutrophils release less secondary and tertiary gr

243 Under the same conditions, C5a binding to blood neutrophils remained intact; in tandem, in vitro c

244 ty, although most plasma cytokine levels and blood neutrophil responses were not key components.

245 et, virtually all major plasma cytokines and blood neutrophil responses were related to marrow-derive

246 Late blood neutrophil responses were related to the presence

247 Consistent patterns of peripheral blood neutrophil responses, as determined by LPS-induced

248 nd phenotypic characterization of peripheral blood neutrophils revealed more mature and responsive ne

249 ecruitment.(1) They report that murine fetal blood neutrophil rolling, adhesion, and extravasation fr

250 Human blood neutrophils rolling on E- or P-selectin reduced th

251 that L-selectin (CD62L) on human peripheral blood neutrophils serves as an E-selectin ligand.

252 hysiologically relevant in vitro human whole blood neutrophil shape change assay, an aminopyrazine se

253 We demonstrate that peripheral blood neutrophils shift from relatively immature (Nh0) c

254 After CLP, blood neutrophils showed a reduction in C5aR followed by

255 LP receptor (TSLPR) signaling, had levels of blood neutrophils, spleen myeloid cells, and serum IL-4,

256 The three mature peripheral blood neutrophil subsets arise from distinct maturing bo

257 Moreover, in vitro exocytosis assays of blood neutrophils suggest that surface nutrient transpor

258 can restore a severely neutropenic patient's blood neutrophil supply and neutrophil inflammation resp

259 Compared with blood neutrophils, TANs displayed an activated phenotype

260 n this process is the polarized migration of blood neutrophils through endothelial cells (ECs) lining

261 table patterns in the response of peripheral blood neutrophils to LPS exist in the human population a

262 Allowing the ratio of blood neutrophils to mitotic neutrophil precursors (R) t

263 process, molecular signals guide circulating blood neutrophils to target specific vessels for extrava

264 centration was inversely associated with the blood neutrophil-to-lymphocyte ratio (p = 0.03) and the

265 Baseline peripheral blood neutrophil-to-lymphocyte ratio correlated with res

266 ry host intestinal gene signature, increased blood neutrophil-to-lymphocyte ratio, and unfavorable ou

267 Our in vitro model enables blood neutrophil transepithelial migration into cell-fre

268 monocytoid cells and culturing of peripheral blood neutrophils, treatments which modulate plasminogen

269 in adhesion to Glu-Pg was demonstrable with blood neutrophils, U937 monocytoid cells, and geneticall

270 L-60, the T cell line Jurkat, and peripheral blood neutrophils, undergoing apoptosis induced either w

271 In experimental murine sepsis, pHi of blood neutrophils was analogously alkalinized, which cou

272 rleukin 17A (IL-17) production by peripheral blood neutrophils was examined in patients with fungal k

273 inally, ex vivo adhesion of mouse peripheral blood neutrophils was strongly reduced after oral admini

274 low cytometry was used to characterize whole-blood neutrophils (WBNs) and low-density neutrophils (LD

275 Normal peripheral blood neutrophil were incubated overnight in BALF from n

276 Peripheral blood neutrophils were analyzed for intracellular hydrog

277 Blood neutrophils were cultured for 17 to 20 hours in th

278 Blood neutrophils were isolated from healthy volunteers

279 Peripheral blood neutrophils were isolated from patients with COPD

280 Blood neutrophils were isolated using a standard gradien

281 These higher blood neutrophils were only seen in obese women and not

282 Whole blood neutrophils were pretreated with or without inotro

283 Blood neutrophils were purified and cultured overnight w

284 deed, in 18 CF patients with stable disease, blood neutrophils were readily deficient in the pivotal

285 When normal blood neutrophils were stimulated in vitro with LPS and

286 Isolated peripheral blood neutrophils were stimulated with 19 periodontal ba

287 When human peripheral blood neutrophils were stimulated with parasite Ag, they

288 ause of the finding of morulae in peripheral blood neutrophils were studied for determination of the

289 hemokine receptor 2 is expressed on lung and blood neutrophils, which are increased upon E. coli infe

290 e host, can induce an increase in peripheral blood neutrophils, which are predisposed to NET formatio

291 transient expression and release of OSM from blood neutrophils, which was more rapid than the express

292 8 weeks reduced mitoROS levels in peripheral blood neutrophils, while also restraining plasma cell ex

293 ecretion of beta-glucuronidase in peripheral blood neutrophils with a potency of approximately 1/1000

294 Stimulating human peripheral blood neutrophils with eCIRP also induced APANs, and sti

295 Incubation of peripheral blood neutrophils with RA synovial fluid led to TNFalpha

296 vitro after the culture of human peripheral blood neutrophils with recombinant IL-4 under conditions

297 Incubation of peripheral blood neutrophils with thapsigargin, an inhibitor of the

298 report, we show that treatment of peripheral blood neutrophils with the chemotactic peptide fMLP or w

299 ation, 3 neutrophil subsets are found in the blood: neutrophils with a conventional segmented nucleus

300 9-L) induced a rapid and robust expansion of blood neutrophils, with a concomitant reduction in PBMCs